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Journal articles on the topic 'Response induction'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Kumar, K., S. N. Shukla, and S. Bhandekar S. K. Singh P. Inwati. "Fertility Response Following Induction of Lactation in Infertile Dairy Cows." International Journal of Trend in Scientific Research and Development Volume-2, Issue-2 (2018): 1302–4. http://dx.doi.org/10.31142/ijtsrd9615.

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2

Baskaran, Charulatha, Anthiyur Subramaniyam Allirathinam, Sundaram Senthil Priya, and Shankar Radhakrishnan. "Ovarian Volume an Indicator of Ovarian Response following Ovulation Induction." Indian Journal of Obstetrics and Gynecology 5, no. 3 (2017): 387–94. http://dx.doi.org/10.21088/ijog.2321.1636.5317.12.

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3

Gurdon, J. B. "The localization of an inductive response." Development 105, no. 1 (1989): 27–33. http://dx.doi.org/10.1242/dev.105.1.27.

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Combinations of tissues from Xenopus blastulae have been used to identify several mechanisms that limit the number of animal cells forming muscle after induction by vegetal cells. The results disagree with a model in which direct physical contact or very close proximity between animal and vegetal cells restricts the number of cells that receive the inductive signal. Rather it seems that a diffusible inducer is released by vegetal cells, and spreads through 4–8 animal cell diameters, equivalent to a distance of 80 microns, from the nearest vegetal cells. Several factors seem to cooperate to prevent the further spread of the mesoderm-forming induction. These include the slow diffusion and/or instability of the inducer, the time of loss of competence of animal cells to respond to induction, and the amount of vegetal tissue that releases inducer for a limited time. The combination of these, and perhaps other, processes seems to ensure that a consistent minority of animal cells are induced to form muscle, thereby leaving other animal cells available to form the nervous system and epidermis.

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4

Russell, M. W., and J. Mestecky. "Induction of the Mucosal Immune Response." Clinical Infectious Diseases 10, Supplement 2 (1988): S440—S446. http://dx.doi.org/10.1093/cid/10.supplement_2.s440.

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5

Marland, Gill, Alexander B. Bakker, Gosse J. Adema, and Carl G. Figdor. "Dendritic Cells in Immune Response Induction." Stem Cells 14, no. 5 (1996): 501–7. http://dx.doi.org/10.1002/stem.140501.

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6

Calder, AA, AD Loughney, CJ Weir, and JW Barber. "Author response to: Induction of labour." BJOG: An International Journal of Obstetrics & Gynaecology 116, no. 3 (2009): 463–64. http://dx.doi.org/10.1111/j.1471-0528.2008.02043.x.

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7

Lohse, A. W., T. W. Spahn, T. Wölfel, J. Herkel, I. R. Cohen, and K. H. Meyer zum Büschenfelde. "Induction of the anti-ergotypic response." International Immunology 5, no. 5 (1993): 533–39. http://dx.doi.org/10.1093/intimm/5.5.533.

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8

Billard, V., F. Moulla, J. L. Bourgain, A. Megnigbeto, and D. R. Stanski. "Hemodynamic Response to Induction and Intubation." Anesthesiology 81, no. 6 (1994): 1384–93. http://dx.doi.org/10.1097/00000542-199412000-00013.

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9

Leiria, Andreia, Pedro Nunes, Atef Morched, and M. Teresa Correia de Barros. "Induction motor response to voltage dips." Electric Power Systems Research 76, no. 8 (2006): 676–80. http://dx.doi.org/10.1016/j.epsr.2005.12.013.

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10

kanchan, Dr Nisha, and Dr Latha B. Dr. Latha.B. "Ovarian Volume by Transvaginal Sonography in the Prediction of Ovarian Response to Ovulation Induction." International Journal of Scientific Research 3, no. 4 (2012): 314–15. http://dx.doi.org/10.15373/22778179/apr2014/109.

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11

Reichard, John F., Timothy P. Dalton, Howard G. Shertzer, and Alvaro Puga. "Induction of Oxidative Stress Responses by Dioxin and other Ligands of the Aryl Hydrocarbon Receptor." Dose-Response 3, no. 3 (2005): dose—response.0. http://dx.doi.org/10.2203/dose-response.003.03.003.

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TCDD and other polyhalogenated aromatic hydrocarbon ligands of the aryl hydrocarbon receptor (AHR) have been classically considered as non-genotoxic compounds because they fail to be directly mutagenic in either bacteria or most in vitro assay systems. They do so in spite of having repeatedly been linked to oxidative stress and to mutagenic and carcinogenic outcomes. Oxidative stress, on the other hand, has been used as a marker for the toxicity of dioxin and its congeners. We have focused this review on the connection between oxidative stress induction and the toxic effects of fetal and adult dioxin exposure, with emphasis on the large species difference in sensitivity to this agent. We examine the roles that the dioxin-inducible cytochromes P450s play in the cellular and toxicological consequences of dioxin exposure with emphasis on oxidative stress involvement. Many components of the health consequences resulting from dioxin exposure may be attributable to epigenetic mechanisms arising from prolonged reactive oxygen generation.

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12

Mendoza-Duarte, J. M., F. C. Robles-Hernandez, I. Estrada-Guel, C. Carreño-Gallardo, and R. Martínez-Sánchez. "Aluminum Composites Reinforced With Graphite: a Densification and Mechanical Response Study." MRS Advances 2, no. 50 (2017): 2847–55. http://dx.doi.org/10.1557/adv.2017.521.

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ABSTRACTAluminum-Graphite composites were prepared from pure aluminum and natural graphite the mixture was processed by the mechanical milling (MM) technique. The microstructural characteristics of prepared composites were investigated by X-rays diffraction (XRD), optical microscopy (OM) and scanning electron microscopy (SEM). The mechanical response of samples was evaluated by compression and hardness tests. To obtain solid samples from milled powders we used an alternative sintering process based on induction heating. Although this method is frequently used for refractory ceramics fabrication (>1500°C), it has not been properly evaluated for materials processing at low temperature (<500°C). This work presents a comparative study of Al-Gr composites sintered by using two routes: conventional pressure-less and high frequency induction heating. After the inductive sintering, is noticeable a pronounced reduction of porosity and increase on the mechanical response of induction sintered specimens, compared with the processed by conventional route. Also, yield strength and hardness increases with graphite addition and induction sintering reaching an increase of 50 and 90%, respectively (compared with an Alp-2h blank).

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13

HOLUBEC, LUBOS, JIRI POLIVKA, MARTIN SAFANDA, MICHAL KARAS, and VACLAV LISKA. "The Role of Cetuximab in the Induction of Anticancer Immune Response in Colorectal Cancer Treatment." Anticancer Research 36, no. 9 (2016): 4421–26. http://dx.doi.org/10.21873/anticanres.10985.

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14

Barabasz, Arreed, and Marianne Barabasz. "Induction Technique: Beyond Simple Response to Suggestion." American Journal of Clinical Hypnosis 59, no. 2 (2016): 204–13. http://dx.doi.org/10.1080/00029157.2016.1209456.

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15

NOVOTNY, D. W., and J. A. A. MELKEBEEK. "Dynamic Response of Voltage-Driven Induction Machines." Electric Machines & Power Systems 10, no. 2-3 (1985): 149–76. http://dx.doi.org/10.1080/07313568508909116.

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16

Valent, Jason, Christy Samaras, Debbie Hastings, et al. "Response Adapted Induction Therapy for Multiple Myeloma." Blood 126, no. 23 (2015): 3327. http://dx.doi.org/10.1182/blood.v126.23.3327.3327.

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Abstract Background: Bortezomib (bort), lenalidomide (len), cyclophosphamide (cy), and dexamethasone (dex) combinations are acceptable initial treatment options for multiple myeloma (MM). At least 2 of these agents are generally used in initial treatment. Continuous len /dex treatment in older patients (pts) may be superior to shorter courses of 3 drug therapy (Benboubker L et al. N Engl J Med 2014; 371:906-917). Standardized treatment programs for cancer pts are important in a changing healthcare reimbursement system. Response adapted therapy for multiple myeloma has the potential to reduce toxicity, limit multi-drug resistant subclone development thus improving survival, and provide cost savings. Methods: The Cleveland Clinic multiple myeloma carepath initiates 2 drug response adapted therapy with len/dex (bort/dex if there is evidence of myeloma related kidney disease or unacceptable len cost to pt). This is an update to preliminary data of Narkhede et al 2014 ASH abstract 2620. Response assessment per current IMWG criteria is obtained with each cycle of therapy, although bone marrow biopsy is not required to confirm complete response. Pts with progressive disease (PD) after one cycle or lack of partial response (PR) after 2 cycles have either bort or len added to the initial 2 drug therapy. If needed, sequential addition of cy and liposomal doxorubicin occurs until at least PR is obtained. Pts achieving PR after 2 cycles are continued on 2 drug therapy. All transplant eligible pts are offered high dose melphalan and autologous stem cell transplant if they achieve PR followed by maintenance therapy. Pts not eligible for transplant are continued on effective therapy for up to 3 years as tolerated. Supportive care with bisphosphonates, vaccinations, zoster prophylaxis, and thromboembolism prophylaxis are outlined in this carepath. Results: From October 2012 to March 2015 carepath therapy was used in 53 pts not eligible for or unwilling to participate in clinical trials. Outcome data was attained through July 31, 2015 and median follow up was 16 months. 10 pts are off carepath treatment due to death (n=4), drug induced anorexia (n=1), or disease progression (n=5). 5 of these 10 pts were treated with ≥ 3 drugs. 3 of the 4 deaths occurred in pts high risk by FISH or MyPRS analysis. For all pts, response rates of VGPR or better were 55% in pts treated with 2 drugs and 41% in pts treated with ≥ 3 drugs. Of the 43 pts remaining on carepath based therapy, 26 pts have required only 2 drugs during the induction phase of treatment with 58% achieving at least very good partial response (VGPR) prior to either transplant or consolidation maintenance therapy. 17 pts required 3 or more drugs during the induction phase with 35% achieving at least VGPR prior to transplant or consolidation maintenance. Grade 3 or higher toxicity included hyperglycemia (n=4) due to dex, neutropenia (n=4) due to len, febrile neutropenia (n=2) due to len, insomnia (n=2) due to dex, and upper respiratory infection (n=2). One episode each of acute coronary syndrome due to dex (pt not on len), pancreatitis due to dex, anorexia due to len, sensory peripheral neuropathy due to bort, and tumor lysis syndrome due to len/dex occurred. Conclusion: Response adapted carepath based therapy provides effective disease control for the majority of MM pts treated while limiting cost due to the lower utilization of 3 drug therapy. While overall survival data is immature, the primary goal of response adapted therapy is to improve overall survival of MM pts by limiting early development of multi-drug resistant subclones. 55% of our pts required only 2 drug induction therapy to achieve a VGPR or better and 84% of these patients remain on carepath based therapy. Estimated cost savings on drug alone is over $4000 per cycle in the 2 drug treated pts. Cost savings may be underestimated as 73% of pts received len/dex as induction therapy and thus incurring less infusion related cost. Disclosures Valent: Takeda/Millennium: Speakers Bureau; Celgene: Speakers Bureau. Off Label Use: cyclophosphamide for the treatment of multiple myeloma. Faiman:Amgen/Onyx: Consultancy; Celgene: Consultancy, Speakers Bureau; Takeda/Millennium: Consultancy. Hamilton:Takeda/Millennium: Speakers Bureau. Smith:celegene, spectrum, genentech: Honoraria. Reu:Takeda/Millennium: Research Funding; Novartis: Research Funding; Celgene: Research Funding.

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17

Ghoshal, Kalpana, Yijie Wang, John F. Sheridan, and Samson T. Jacob. "Metallothionein Induction in Response to Restraint Stress." Journal of Biological Chemistry 273, no. 43 (1998): 27904–10. http://dx.doi.org/10.1074/jbc.273.43.27904.

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18

Kristian Storm, F., Robert M. Elashoff, Harvey W. Baker, et al. "Thermal dose-response of magnetic-induction thermoradiotherapy." Journal of Surgical Oncology 39, no. 2 (1988): 79–83. http://dx.doi.org/10.1002/jso.2930390203.

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19

Corry, David B., and Farrah Kheradmand. "Induction and regulation of the IgE response." Nature 402, S6760 (1999): 18–23. http://dx.doi.org/10.1038/35037014.

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20

SAREEN, JITENDER, ROBERT J. HUDSON, MORLEY ROSENBLOOM, and IAN R. THOMSON. "Dose-Response to Anaesthetic Induction with Sufentanil." Survey of Anesthesiology 61, no. 4 (1997): 196. http://dx.doi.org/10.1097/00132586-199708000-00002.

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21

Wang, Tsili, Liming Yu, and Otto Fanini. "Multicomponent induction response in a borehole environment." GEOPHYSICS 68, no. 5 (2003): 1510–18. http://dx.doi.org/10.1190/1.1620624.

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Field and numerically simulated data show that coplanar induction measurements (σxx and σyy) are affected by both conductive and resistive muds. The effect is, for a resistivity contrast of 10 or less between the formation and mud, approximately proportional to the resistivity contrast. The borehole effect is generally stronger for a decentralized tool. In the perpendicular eccentricity mode, the eccentricity effect on σxx or σyy can be greater than 100% of the true formation responses. We demonstrate that dual‐frequency processing can reduce the eccentricity effect by an order of magnitude. We investigate the effect of invasion for both circular and elliptic invasions. For circular invasion, the effect on σxx, σyy, or the coaxial measurement (σzz) is characterized by a critical invasion thickness. For invasion thinner than the critical thickness, the effect is minimal. The effect increases rapidly as invasion thickness increases beyond the critical thickness. For elliptic invasion, both the σxx and σzz responses are similar to those of the corresponding inner circular invasion. Beyond a long‐to‐short‐axis ratio of three, an elliptic invasion behaves like a long, thin fracture.

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22

Erhardt, Annette, and Gisa Tiegs. "Tolerance Induction in Response to Liver Inflammation." Digestive Diseases 28, no. 1 (2010): 86–92. http://dx.doi.org/10.1159/000282069.

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23

Kovács, Krisztina, Ferenc Somogyvári, Bratko Filipic, Imre Mécs, and Sándor Tóth. "INDUCTION OF ANTIVIRAL RESPONSE BY ELECTRIC PULSES." Electro- and Magnetobiology 19, no. 3 (2000): 279–87. http://dx.doi.org/10.1081/jbc-100102119.

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24

Garabedian, Matthew, Christy Pearce, Wendy Hansen, and Kristine Lain. "306: Obesity attenuates response to labor induction." American Journal of Obstetrics and Gynecology 201, no. 6 (2009): S123. http://dx.doi.org/10.1016/j.ajog.2009.10.321.

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25

Elsharif, Mohamed, and Tareq Elgargani. "QUICK-RESPONSE FUZZY-CONTROLLED INDUCTION MOTOR DRIVE." International Journal of Advances in Signal and Image Sciences 6, no. 2 (2020): 20. http://dx.doi.org/10.29284/ijasis.6.2.2020.20-28.

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26

Helmann, John D., Ming Fang Winston Wu, Phil A. Kobel, et al. "Global Transcriptional Response of Bacillus subtilis to Heat Shock." Journal of Bacteriology 183, no. 24 (2001): 7318–28. http://dx.doi.org/10.1128/jb.183.24.7318-7328.2001.

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ABSTRACT In response to heat stress, Bacillus subtilisactivates the transcription of well over 100 different genes. Many of these genes are members of a general stress response regulon controlled by the secondary sigma factor, ςB, while others are under control of the HrcA or CtsR heat shock regulators. We have used DNA microarrays to monitor the global transcriptional response to heat shock. We find strong induction of known ςB-dependent genes with a characteristic rapid induction followed by a return to near prestimulus levels. The HrcA and CtsR regulons are also induced, but with somewhat slower kinetics. Analysis of DNA sequences proximal to newly identified heat-induced genes leads us to propose ∼70 additional members of the ςB regulon. We have also identified numerous heat-induced genes that are not members of known heat shock regulons. Notably, we observe very strong induction of arginine biosynthesis and transport operons. Induction of several genes was confirmed by quantitative reverse transcriptase PCR. In addition, the transcriptional responses measured by microarray hybridization compare favorably with the numerous previous studies of heat shock in this organism. Since many different conditions elicit both specific and general stress responses, knowledge of the heat-induced general stress response reported here will be helpful for interpreting future microarray studies of other stress responses.

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Jaworski, E., L. A. Gharzai, J. M. Pakela, et al. "Imaging response versus operative laryngoscopy assessment of induction chemotherapy response in an induction bioselection approach to larynx cancer." International Journal of Radiation Oncology*Biology*Physics 106, no. 5 (2020): 1120–21. http://dx.doi.org/10.1016/j.ijrobp.2019.11.374.

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Chen, C. Y., L. W. Forman, and D. V. Faller. "Calcium-dependent immediate-early gene induction in lymphocytes is negatively regulated by p21Ha-ras." Molecular and Cellular Biology 16, no. 11 (1996): 6582–92. http://dx.doi.org/10.1128/mcb.16.11.6582.

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The induction of immediate-early (IE) response genes, such as egr-1, c-fos, and c-jun, occurs rapidly after the activation of T lymphocytes. The process of activation involves calcium mobilization, activation of protein kinase C (PKC), and phosphorylation of tyrosine kinases. p21(ras), a guanine nucleotide binding factor, mediates T-cell signal transduction through PKC-dependent and PKC-independent pathways. The involvement of p21(ras) in the regulation of calcium-dependent signals has been suggested through analysis of its role in the activation of NF-AT. We have investigated the inductions of the IE genes in response to calcium signals in Jurkat cells (in the presence of activated p21(ras)) and their correlated consequences. The expression of activated p21(ras) negatively regulated the induction of IE genes by calcium ionophore. This inhibition of calcium-activated IE gene induction was reversed by treatment with cyclosporin A, suggesting the involvement of calcineurin in this regulation. A later result of inhibition of this activation pathway by p21(ras) was down-regulation of the activity of the transcription factor AP-1 and subsequent coordinate reductions in IL-2 gene expression and protein production. These results suggest that p2l(ras) is an essential mediator in generating not only positive but also negative modulatory mechanisms controlling the competence of T cells in response to inductive stimulations.

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Woolard, Stacie N., and Uday Kumaraguru. "Viral Vaccines and CTL Response." Journal of Biomedicine and Biotechnology 2010 (2010): 1–6. http://dx.doi.org/10.1155/2010/141657.

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Immune induction by successful vaccine formulations seems to involve stimulation of both humoral and cellular arms of immunity. Nevertheless, CD8+ CTLs are of critical relevance in the context of intracellular infection and tumor for many reasons. The task of exerting antipathogen activity by CD8+ T cells, which principally function to control and eradicate intracellular pathogens, is enabled by constitutive expression of MHC class-I molecules on all tissue types. CTL induction offers hope for vaccines against pathogens that are resistant to neutralizing activity. This review discusses the mechanism of immune induction by some successful vaccines and based on the accrued evidence suggests ideas for improved design of CTL-inducing vaccines.

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30

Wang, Rong, Peihua Feng, Yongchen Fan, and Ying Wu. "Spontaneous Electromagnetic Induction Modulating the Neuronal Dynamical Response." International Journal of Bifurcation and Chaos 29, no. 01 (2019): 1950005. http://dx.doi.org/10.1142/s0218127419500056.

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Spontaneous electromagnetic induction originating from neuronal electrical activity is believed to reflect the memory ability in the neural system and significantly modulates neural information transmission, but its fundamental effect on the neuronal dynamic properties is still not well understood. In this paper, we use a memristor to couple neuronal electrical activity and magnetic fields and study how the spontaneous electromagnetic induction modulates the neuronal dynamical response to external stimulation. It is found that the negative feedback of electromagnetic induction on the neuron significantly reduces the dynamical response range, decreases the oscillation amplitude and induces a higher firing frequency. Meanwhile, the memory effect on electromagnetic induction can induce two kinds of bistability, including the coexistence of a stable limit cycle and a fixed point, and the coexistence of two stable limit cycles. Furthermore, high electric driving for electromagnetic induction produces complex firing patterns with single, double and multiple frequencies. Our results not only further confirm the efficacy of spontaneous electromagnetic induction in modulating the neuronal dynamical properties but also provide insights into the possibilities of choosing suitable parameter spaces in studying the effects of external magnetic induction on brain functions.

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Cheshire, J. L., J. H. Evans, and L. R. Keller. "Ca2+ signaling in the Chlamydomonas flagellar regeneration system: cellular and molecular responses." Journal of Cell Science 107, no. 9 (1994): 2491–98. http://dx.doi.org/10.1242/jcs.107.9.2491.

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In response to certain extracellular stimuli, Chlamydomonas reinhardtii cells excise their flagella, induce expression of more than 200 different flagellar mRNAs, and assemble a new flagellar pair. Normally, flagellar excision, gene induction and outgrowth are tightly coupled temporally. Our previous studies showed that uncoupling the cellular response of flagellar excision from flagellar outgrowth resulted in submaximal flagellar gene induction, and led us to propose that normal flagellar gene induction is a composite response. The present study extends these observations by measuring flagellar gene induction in Chlamydomonas cells stimulated under conditions where both flagellar excision and flagellar outgrowth are blocked. We find that the flagellar genes are induced in a Ca(2+)-dependent manner in response to stimulation in the absence of flagellar excision and outgrowth. Flagellar gene induction is therefore independent of flagellar excision and outgrowth but sensitive to extracellular Ca2+ levels. Thus, flagellar excision, flagellar outgrowth and flagellar gene induction are three responses to a common stimulus that are related by their requirement for extracellular Ca2+.

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32

Gianzero, Stan, and Shey‐Min Su. "The response of an induction dipmeter and standard induction tools to dipping beds." GEOPHYSICS 55, no. 9 (1990): 1128–40. http://dx.doi.org/10.1190/1.1442929.

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This theoretical investigation presents the responses in dipping beds of an induction dipmeter and standard induction tools, neglecting the influence of the borehole and invaded zones as well as the mandrel and coil size. The analysis is sufficiently general to determine response characteristics of an induction coil arbitrarily oriented in an arbitrary number of beds. In an induction dipmeter log (IDL) the most useful information is obtained from the out‐of‐phase voltage induced in the receiver coils when they are orthogonally oriented to the transmitter coils. A simple algorithm is developed to convert the crosscoupling signals into the apparent dip and strike information. The agreement between the actual and apparent values is nearly perfect for small transmitter‐to‐receiver spacings, i.e., less than 10 cm. The theoretical results for standard induction tools were consistent with prior work indicating the influence of dip is strongly dependent on the shoulder effect of the device in question. Consequently, dipping beds have less influence on the standard ILm because its shoulder effect is less than the standard ILd. These conclusions are supported by numerous simulations consistent with conditions encountered in the field. Finally, a simple no‐skin‐effect theory is developed for the case of a single dipping interface separating two infinitely thick beds. The results with this approach agree with the exact theory in resistive formations. This simplified theory is a generalization of Doll’s geometrical factor theory.

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Schantz, S. P., H. E. Savage, T. Racz, et al. "Immunologic determinants of head and neck cancer response to induction chemotherapy." Journal of Clinical Oncology 7, no. 7 (1989): 857–64. http://dx.doi.org/10.1200/jco.1989.7.7.857.

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Various measures of immune response were assessed prior to induction chemotherapy (intravenous [IV] cisplatin, fluorouracil [5-FU], and bleomycin) in 43 previously untreated head and neck cancer patients to derive a clinical response prediction model. These were parameters of functional cellular immunity (natural killer [NK] cell activity, lymphocyte blastogenesis response to mitogens), total lymphocyte and lymphocyte subset numbers and percentages, and circulating humoral immunity (total immunoglobulin, immunoglobulin classes, and C1q binding activity [C1q BA]). The C1q BA may reflect levels of circulating immune complexes within peripheral blood. The objective primary tumor response rate was 65% (16 complete responses and 12 partial responses). Univariate logistic regression analysis showed that failure to respond to therapy was significantly related to higher value (vis-à-vis response) of humoral immune parameters total immunoglobulin (Ig), P less than .01; IgG, P less than .01; and C1q BA, P less than .001. No association between cellular immune response measurements and response to chemotherapy was identified. By multivariate logistic regression analysis, only C1q BA levels were predictive of drug therapy responsiveness (P less than .001). Results extend our previous investigations regarding C1q BA measurement in head and neck cancer patients, and show that C1q BA levels add accuracy of prediction of subsequent chemotherapy response to that based solely on standard staging criteria and other parameters of immune status.

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Okamura, Maro, Yosuke Takano, Nobuhiko Hiramatsu, et al. "Suppression of cytokine responses by indomethacin in podocytes: a mechanism through induction of unfolded protein response." American Journal of Physiology-Renal Physiology 295, no. 5 (2008): F1495—F1503. http://dx.doi.org/10.1152/ajprenal.00602.2007.

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We found that, in murine podocytes, expression of monocyte chemoattractant protein 1 ( MCP- 1) in response to TNF-α was suppressed by indomethacin but not by ibuprofen. This anti-inflammatory potential was correlated with induction of 78-kDa glucose-regulated protein ( GRP78), a marker of unfolded protein response (UPR). Indomethacin, but not ibuprofen, also triggered expression of CHOP, another endogenous indicator of UPR, as well as repression of endoplasmic reticulum stress-responsive alkaline phosphatase, an exogenous indicator of UPR. Like ibuprofen, other nonsteroidal anti-inflammatory drugs including aspirin and sulindac also did not induce UPR, indicating that the induction of UPR by indomethacin was independent of cyclooxygenase inhibition. The induction of UPR by indomethacin was observed similarly in other cells including mesangial cells and tubular epithelial cells. In tumor necrosis factor (TNF)-α-treated cells, suppression of MCP-1 by indomethacin was inversely correlated with induction of UPR, and other inducers of UPR including tunicamycin, thapsigargin, and A23187 reproduced the suppressive effect. Reporter assays showed that indomethacin as well as thapsigargin attenuated activation of NF-κB by TNF-α, and it was associated with enhanced degradation of TNF receptor-associated factor 2 (TRAF2) and blunted degradation of IκBβ. Subsequent experiments revealed that acute ablation of GRP78 protein by AB5 subtilase cytotoxin caused reinforcement of MCP-1 induction and NF-κB activation by TNF-α and that transfection with GRP78 significantly suppressed the cytokine-induced activation of NF-κB. These results suggested that indomethacin suppressed the response of podocytes to TNF-α via UPR and that UPR-triggered induction of GRP78 and degradation of TRAF2 may be responsible, at least in part, for the suppressive effect of indomethacin.

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35

Sparrow, Miles P., Konstantinos Papamichael, Mark G. Ward, et al. "Therapeutic Drug Monitoring of Biologics During Induction to Prevent Primary Non-Response." Journal of Crohn's and Colitis 14, no. 4 (2019): 542–56. http://dx.doi.org/10.1093/ecco-jcc/jjz162.

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Abstract Biologic therapies have revolutionized the management of inflammatory bowel disease [IBD], but primary and secondary non-responses occur in a significant proportion of patients. Therapeutic drug monitoring [TDM] now has an established role in the treatment algorithm for managing secondary loss of response to anti-tumour necrosis factor [anti-TNF] agents during maintenance therapy. Data to support the use of TDM in the management of secondary loss of response to vedolizumab and ustekinumab are emerging. The potential to prevent primary non-response to biologic agents during induction is of equal, and potentially greater, clinical importance. Again, most data supporting the use of ‘proactive' TDM during induction pertains to the use of anti-TNF agents, but signals of efficacy for the use of TDM during induction with other biologic classes are now appearing. This review aims to summarize data on the use of TDM during induction to prevent pharmacokinetic primary non-response to all three classes of biologic therapy currently available for the treatment of IBD.

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Balmelli, Carole, Stéphane Demotz, Hans Acha-Orbea, Pierre De Grandi, and Denise Nardelli-Haefliger. "Trachea, Lung, and Tracheobronchial Lymph Nodes Are the Major Sites Where Antigen-Presenting Cells Are Detected after Nasal Vaccination of Mice with Human Papillomavirus Type 16 Virus-Like Particles." Journal of Virology 76, no. 24 (2002): 12596–602. http://dx.doi.org/10.1128/jvi.76.24.12596-12602.2002.

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ABSTRACT Vaccination by the nasal route has been successfully used for the induction of immune responses. Either the nasal-associated lymphoid tissue (NALT), the bronchus-associated lymphoid tissue, or lung dendritic cells have been mainly involved. Following nasal vaccination of mice with human papillomavirus type 16 (HPV16) virus-like-particles (VLPs), we have previously shown that interaction of the antigen with the lower respiratory tract was necessary to induce high titers of neutralizing antibodies in genital secretions. However, following a parenteral priming, nasal vaccination with HPV16 VLPs did not require interaction with the lung to induce a mucosal immune response. To evaluate the contribution of the upper and lower respiratory tissues and associated lymph nodes (LN) in the induction of humoral responses against HPV16 VLPs after nasal vaccination, we localized the immune inductive sites and identified the antigen-presenting cells involved using a specific CD4+ T-cell hybridoma. Our results show that the trachea, the lung, and the tracheobronchial LN were the major sites responsible for the induction of the immune response against HPV16 VLP, while the NALT only played a minor role. Altogether, our data suggest that vaccination strategies aiming to induce efficient immune responses against HPV16 VLP in the female genital tract should target the lower respiratory tract.

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Rawal, Prabhat, and Uday Bajracharya. "Hemodynamic response to Sevoflurane and Propofol induction: a comparative study." Journal of Society of Anesthesiologists of Nepal 2, no. 1 (2015): 2–7. http://dx.doi.org/10.3126/jsan.v2i1.13549.

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Background: Induction of anesthesia is a critical event and hemodynamic stability is an important factor during this period. Propofol is a commonly used intravenous anesthetic and Sevoflurane is a newly introduced inhalational anesthetic in the context of a developing country. This study compared the hemodynamics on induction of anesthesia with Propofol and Sevoflurane.Methods: A total of 108 American Society of Anesthesiologists physical status I patients undergoing elective surgical procedures under general anesthesia were randomized into two groups. Group ‘P’ patients were induced with intravenous 1% Propofol and Group ‘S’ patients were induced with inhalation of 8% Sevoflurane. Mean arterial pressures and heart rates were recorded at baseline, before induction, during induction and at 1, 3 and 5 minutes after induction of anesthesia before endotracheal intubation.Results: The two groups were comparable with respect to demographics and baseline hemodynamic parameters. There was a significant decrease in mean arterial pressure and heart rate from pre-induction values within both groups during and after induction. The reduction in mean arterial pressure was significantly more in Propofol group transiently during induction. The reduction in heart rate was significantly more in Sevoflurane group at 1, 3 and 5 minutes after induction (P < 0.05).Conclusion: Induction of anesthesia with Propofol demonstrated a greater decrease in mean arterial pressure whereas induction with Sevoflurane was associated with greater reduction in heart rate.Journal of Society of Anesthesiologists of Nepal 2015; 2(1): 2-7

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Falkson, G., R. Gelman, J. Glick, C. I. Falkson, and J. Harris. "Reinduction with the same cytostatic treatment in patients with metastatic breast cancer: an Eastern Cooperative Oncology Group study." Journal of Clinical Oncology 12, no. 1 (1994): 45–49. http://dx.doi.org/10.1200/jco.1994.12.1.45.

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PURPOSE To investigate the therapeutic value of reinduction with the same cytostatic treatment that had been used in induction treatment for women with metastatic breast cancer. MATERIALS AND METHODS One hundred six women with metastatic breast cancer were given dibromodulcitol (mitolactol), doxorubicin, vincristine, tamoxifen, and fluoxymesterone (DAVTH) for 6 months of induction treatment, then randomized to receive one of two chemotherapy regimens if they had obtained an induction partial response (PR) or no change (NC), or to receive observation versus chemotherapy if they had obtained an induction complete response (CR). Patients were then retreated with DAVTH reinduction after relapse. RESULTS Seventy-four patients were eligible or had minor reasons for ineligibility. Severe or life-threatening toxicity was documented in 46%, and lethal toxicity in 4%. Eighteen percent had a response on reinduction (zero of 16 induction nonresponders, 15% induction PR, 44% induction CR). The median time to treatment failure (TTF) from reinduction was 3 months, and the median survival duration from reinduction was 14 months. In a logistic model, factors associated with more reinduction responses were observation after induction CR (P = .002) and age greater than 55 years (P = .04). Time since induction was not significant. CONCLUSION Reinduction of response after treatment failure remains a therapeutic problem. The need for better salvage treatment underlines the importance of developing new regimens.

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Kim, Hyun Kyung, Young Ho Jang, and Jung Kil Cheung. "Parental Response after Participation in Pediatric Anesthetic Induction." Korean Journal of Anesthesiology 44, no. 3 (2003): 365. http://dx.doi.org/10.4097/kjae.2003.44.3.365.

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Owens, Trevor. "Immune response induction in the central nervous system." Frontiers in Bioscience 7, no. 1-3 (2002): d427. http://dx.doi.org/10.2741/owens.

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Aalberts, Marian, Edita Sostaric, Richard Wubbolts, et al. "Spermatozoa recruit prostasomes in response to capacitation induction." Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics 1834, no. 11 (2013): 2326–35. http://dx.doi.org/10.1016/j.bbapap.2012.08.008.

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Skarman, Petra Jerström, Afsar Rahbar, Xun Xie, and Cecilia Söderberg-Nauclér. "Induction of polymorphonuclear leukocyte response by human cytomegalovirus." Microbes and Infection 8, no. 6 (2006): 1592–601. http://dx.doi.org/10.1016/j.micinf.2006.01.017.

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Liu, Ying, Jing Wang, Yuxin Yi, et al. "Induction of KLF4 in response to heat stress." Cell Stress & Chaperones 11, no. 4 (2006): 379. http://dx.doi.org/10.1379/csc-210.1.

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Jyot, J., and A. Ghosh. "Induction of heat shock response in Vibrio cholerae." Microbiology 141, no. 9 (1995): 2101–9. http://dx.doi.org/10.1099/13500872-141-9-2101.

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Ammeur, M. Den, P. Coriat, D. Bruere, J. F. Baron, M. Bertrand, and P. Viars. "HEMODYNAMICS AND LV FUNCTION RESPONSE TO PROPOFOL INDUCTION." Anesthesiology 69, no. 3A (1988): A568. http://dx.doi.org/10.1097/00000542-198809010-00568.

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MEDZHITOV, R., and C. A. JANEWAY. "Innate Immune Induction of the Adaptive Immune Response." Cold Spring Harbor Symposia on Quantitative Biology 64 (January 1, 1999): 429–36. http://dx.doi.org/10.1101/sqb.1999.64.429.

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AUN, C. S. T., S. M. SHORT, D. H. Y. LEUNG, and T. E. OH. "INDUCTION DOSE-RESPONSE OF PROPOFOL IN UNPREMEDICATED CHILDREN." British Journal of Anaesthesia 68, no. 1 (1992): 64–67. http://dx.doi.org/10.1093/bja/68.1.64.

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NAGUIB, M., A. SARI-KOUZEL, M. SERAJ, M. EL-GAMMAL, and M. GOMMA. "INDUCTION DOSE-RESPONSE STUDIES WITH PROPOFOL AND THIOPENTONE." British Journal of Anaesthesia 68, no. 3 (1992): 308–10. http://dx.doi.org/10.1093/bja/68.3.308.

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Mabaera, Rodwell, and Christopher H. Lowrey. "Response: 5-azacytidine induction of human fetal hemoglobin." Blood 111, no. 4 (2008): 2486. http://dx.doi.org/10.1182/blood-2007-11-125070.

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Owens, Trevor. "Immune response induction in the central nervous system." Frontiers in Bioscience 7, no. 4 (2002): d427–438. http://dx.doi.org/10.2741/a786.

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