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Relevant bibliographies by topics / Retinal degeneration – Prevention – Testing

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Academic literature on the topic 'Retinal degeneration – Prevention – Testing'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "Retinal degeneration – Prevention – Testing"

1

Puntel, Anthony, Akiko Maeda, Marcin Golczak, et al. "Prolonged prevention of retinal degeneration with retinylamine loaded nanoparticles." Biomaterials 44 (March 2015): 103–10. http://dx.doi.org/10.1016/j.biomaterials.2014.12.019.

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Williams, D. S., A. Chadha, R. Hazim, and D. Gibbs. "Gene therapy approaches for prevention of retinal degeneration in Usher syndrome." Gene Therapy 24, no. 2 (2017): 68–71. http://dx.doi.org/10.1038/gt.2016.81.

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Chakrabarti, Subrata, Anders A. F. Sima, W. Jun Tze, and Joseph Tai. "Prevention of diabetic retinal capillary pericyte degeneration and loss by pancreatic islet allograft." Current Eye Research 6, no. 5 (1987): 649–58. http://dx.doi.org/10.3109/02713688709034827.

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Weiss, S., E. Kohn, D. Dadon, et al. "Compartmentalization and Ca2+ Buffering Are Essential for Prevention of Light-Induced Retinal Degeneration." Journal of Neuroscience 32, no. 42 (2012): 14696–708. http://dx.doi.org/10.1523/jneurosci.2456-12.2012.

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Di Carlo, Emiliano, and Albert J. Augustin. "Prevention of the Onset of Age-Related Macular Degeneration." Journal of Clinical Medicine 10, no. 15 (2021): 3297. http://dx.doi.org/10.3390/jcm10153297.

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Age-related macular degeneration (AMD) represents the leading cause of irreversible blindness in elderly people, mostly after the age of 65. The progressive deterioration of visual function in patients affected by AMD has a significant impact on quality of life and has also high social costs. The current therapeutic options are only partially able to slow down the natural course of the disease, without being capable of stopping its progression. Therefore, better understanding of the possibilities to prevent the onset of the disease is needed. In this regard, a central role is played by the ide

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Du, Weiwei, Yuanlong An, Xiangdong He, Donglei Zhang, and Wei He. "Protection of Kaempferol on Oxidative Stress-Induced Retinal Pigment Epithelial Cell Damage." Oxidative Medicine and Cellular Longevity 2018 (November 21, 2018): 1–14. http://dx.doi.org/10.1155/2018/1610751.

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The protection of retinal pigment epithelium (RPE) injury plays an important role in the prevention of or in delaying the pathological progress of retinal degeneration diseases, like age-related macular degeneration (AMD), diabetic retinopathy, and retinitis pigmentosa. Oxidative stress has been identified as a major inducer of RPE injury, which eventually could lead to a loss of vision. Kaempferol is a natural flavonoid widely distributed in many edible plants, fruits, and traditional medicines and has been reported to have antioxidant, anti-inflammatory, anticancer, and antimicrobial activit

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Dang, Kuan-Rong, Yan-Nian Hui, and Hong-Jun Du. "Newly-found functions of metformin for the prevention and treatment of age-related macular degeneration." International Journal of Ophthalmology 14, no. 8 (2021): 1274–80. http://dx.doi.org/10.18240/ijo.2021.08.20.

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Metformin (MET), a first-line oral agent used to treat diabetes, exerts its function mainly by activating adenosine monophosphate-activated protein. The accumulation of oxidized phospholipids in the outer layer of the retina plays a key role in retinal pigment epithelium (RPE) cells death and the formation of choroidal neovascularization (CNV), which mean the development of age-related macular degeneration (AMD). Recent studies have shown that MET can regulate lipid metabolism, inhibit inflammation, and prohibit retinal cell death and CNV formation due to various pathological factors. Here, ne

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Sharma, Ruchi, Vladimir Khristov, Aaron Rising, et al. "Clinical-grade stem cell–derived retinal pigment epithelium patch rescues retinal degeneration in rodents and pigs." Science Translational Medicine 11, no. 475 (2019): eaat5580. http://dx.doi.org/10.1126/scitranslmed.aat5580.

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Considerable progress has been made in testing stem cell–derived retinal pigment epithelium (RPE) as a potential therapy for age-related macular degeneration (AMD). However, the recent reports of oncogenic mutations in induced pluripotent stem cells (iPSCs) underlie the need for robust manufacturing and functional validation of clinical-grade iPSC-derived RPE before transplantation. Here, we developed oncogenic mutation-free clinical-grade iPSCs from three AMD patients and differentiated them into clinical-grade iPSC-RPE patches on biodegradable scaffolds. Functional validation of clinical-gra

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von der Emde, Leon, Maximilian Pfau, Frank G. Holz, et al. "AI-based structure-function correlation in age-related macular degeneration." Eye 35, no. 8 (2021): 2110–18. http://dx.doi.org/10.1038/s41433-021-01503-3.

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AbstractSensitive and robust outcome measures of retinal function are pivotal for clinical trials in age-related macular degeneration (AMD). A recent development is the implementation of artificial intelligence (AI) to infer results of psychophysical examinations based on findings derived from multimodal imaging. We conducted a review of the current literature referenced in PubMed and Web of Science among others with the keywords ‘artificial intelligence’ and ‘machine learning’ in combination with ‘perimetry’, ‘best-corrected visual acuity (BCVA)’, ‘retinal function’ and ‘age-related macular d

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Milligan, Scott C., James G. Alb, Raya B. Elagina, Vytas A. Bankaitis, and David R. Hyde. "The Phosphatidylinositol Transfer Protein Domain of Drosophila Retinal Degeneration B Protein Is Essential for Photoreceptor Cell Survival and Recovery from Light Stimulation." Journal of Cell Biology 139, no. 2 (1997): 351–63. http://dx.doi.org/10.1083/jcb.139.2.351.

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The Drosophila retinal degeneration B (rdgB) gene encodes an integral membrane protein involved in phototransduction and prevention of retinal degeneration. RdgB represents a nonclassical phosphatidylinositol transfer protein (PITP) as all other known PITPs are soluble polypeptides. Our data demonstrate roles for RdgB in proper termination of the phototransduction light response and dark recovery of the photoreceptor cells. Expression of RdgB's PITP domain as a soluble protein (RdgB-PITP) in rdgB2 mutant flies is sufficient to completely restore the wild-type electrophysiological light respons

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Dissertations / Theses on the topic "Retinal degeneration – Prevention – Testing"

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Charvet, Casey Douglas. "Post-Translational Modification By Isolevuglandins: Retinal Detection, Effects, and Prevention." Case Western Reserve University School of Graduate Studies / OhioLINK, 2013. http://rave.ohiolink.edu/etdc/view?acc_num=case1366296569.

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Rojas-Martinez, Julio Cesar. "Strategies of neuroprotection in an in vivo model of retinal degeneration induced by mitochondrial dysfunction." 2009. http://hdl.handle.net/2152/18401.

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Current approaches to treat neurodegenerative disease provide only mild symptomatic relief but do not modify the natural history of these conditions. A large body of evidence suggests that mitochondrial dysfunction is a key event in the pathophysiology of neurodegeneration. Supporting and improving mitochondrial function has a big potential as a strategy for neuroprotection. The goal of this dissertation was to test whether interventions that target mitochondrial function are effective at preventing neurodegeneration induced by mitochondrial failure in vivo. A rodent model of optic neuropathy

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Gupta, Manav. "Differentiation and characterization of cell types associated with retinal degenerative diseases using human induced pluripotent stem cells." Thesis, 2014. http://hdl.handle.net/1805/4839.

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Indiana University-Purdue University Indianapolis (IUPUI)<br>Human induced pluripotent stem (iPS) cells have the unique ability to differentiate into 200 or so somatic cell types that make up the adult human being. The use of human iPS cells to study development and disease is a highly exciting and interdependent field that holds great promise in understanding and elucidating mechanisms behind cellular differentiation with future applications in drug screening and cell replacement studies for complex and currently incurable cellular degenerative disorders. The recent advent of iPS cell technol

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Books on the topic "Retinal degeneration – Prevention – Testing"

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Chet, Cunningham, and Chet Cunningham. The macular degeneration handbook: Natural ways to prevent & reverse it. United Research Publishers, 2000.

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Hiramatsu, Rui. Ōhan hensei·fushu de shitsumei shinai tame ni: Wakariyasui saishin chiryō. 2015.

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The macular degeneration handbook. agora health books, 2003.

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author, Seddon Johanna M., and American Macular Degeneration Foundation, eds. Eat right for your sight: Simple, tasty recipes that help reduce the risk of vision loss from macular degeneration. 2015.

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The Macular Degeneration Handbook: Natural Ways to Prevent & Reverse It. Windsor Health, 2003.

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6

Celesia, Gastone G., and Neal S. Peachey. Visual Evoked Potentials and Electroretinograms. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0041.

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Electrophysiological testing of vision permits the objective assessment of the function of the retina, visual pathways, and cortices. This chapter covers visual evoked potentials (VEPs) and electroretinography (ERG). Flash ERG is useful in evaluating the outer retinal function and specifically helping in the diagnosis of retinal degeneration, monitoring the progress of retinal diseases, monitoring the retinal toxicity of drugs, and understanding the pathophysiology of retinal disorders. VEPs to various stimuli are useful in evaluating macular disorders, diagnosing optic neuropathies, detecting

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Book chapters on the topic "Retinal degeneration – Prevention – Testing"

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Rashid, Khalid, Katharina Dannhausen, and Thomas Langmann. "Testing for Known Retinal Degeneration Mutants in Mouse Strains." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-8669-9_3.

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Wen, Runxia H., Aaron D. Loewen, Ruanne Y. J. Vent-Schmidt, and Orson L. Moritz. "Autophagy Induction by HDAC Inhibitors Is Unlikely to be the Mechanism of Efficacy in Prevention of Retinal Degeneration Caused by P23H Rhodopsin." In Retinal Degenerative Diseases. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-27378-1_66.

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Choudhary, Mayur, and Goldis Malek. "A Review of Pathogenic Drivers of Age-Related Macular Degeneration, Beyond Complement, with a Focus on Potential Endpoints for Testing Therapeutic Interventions in Preclinical Studies." In Retinal Degenerative Diseases. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-27378-1_2.

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Brinton, Daniel A., and Charles P. Wilkinson. "Prevention of Retinal Detachment." In Retinal Detachment. Oxford University Press, 2009. http://dx.doi.org/10.1093/oso/9780195330823.003.0011.

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Retinal detachment is an uncommon disease, affecting approximately 1 in 10,000 people in the general population per year. However, the incidence of retinal breaks is relatively high, affecting 5% to 7% of the population. Obviously, many retinal breaks have minimal, if any, risk for the possible development of a retinal detachment. This includes macular holes that occur as a degenerative process, and asymptomatic, small, round atrophic holes near the ora serrata. However, equatorial horseshoe tears with relevant symptoms progress to retinal detachment in most cases. Probably all surgeons would agree that a large horseshoe tear near the equator in the superior temporal quadrant, with new-onset symptoms of fl ashes and fl oaters and associated vitreous hemorrhage, should be treated prophylactically to avoid retinal detachment. In contrast, most would not advise treatment of a small, round atrophic hole near the inferior ora serrata in an asymptomatic patient with no history of prior detachment. Between these two obvious examples lies a broad spectrum of retinal breaks for which the surgeon must exercise judgment about instituting prophylactic treatment. Most of the breaks reported in surveys of asymptomatic patients or in autopsy series are of the atrophic type, and only a small proportion are horseshoe tears. Although there are no specific rules for the selection of patients for treatment, and each case has to be judged on its own characteristics, the application of evidencebased medicine to this topic has modified the opinions of many regarding the genuine value of prophylactic therapy for most retinal breaks. The American Academy of Ophthalmology has used this approach in developing a Preferred Practice Pattern (PPP) entitled “Posterior Vitreous Detachment, Retinal Breaks, and Lattice Degeneration,” the latest version of which was published in 2008. The evidence base described in this PPP will be employed in the following discussion. Characteristics associated with a relatively high risk of retinal detachment in an eye with visible retinal breaks are listed in Table 6–1. Symptoms and signs of PVD place an eye at particularly high risk. Additional factors include a variety of hereditary, congenital, acquired, and iatrogenic problems.

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Conference papers on the topic "Retinal degeneration – Prevention – Testing"

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Rys, J. P., A. M. Ellingson, D. J. Nuckley, and V. H. Barocas. "Biaxial Mechanical Testing of Single Annulus Fibrosus Layers." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19551.

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The intervertebral disc (IVD), consisting of the inner nucleus pulposus and the outer annulus fibrosus, is subjected to multiaxial stress in vivo. The disc undergoes degenerative changes that account for impairment and disability in middle-aged and older individuals.4 In addition to age-related degeneration, the intervertebral disc is subject to the development of lesions due to partial displacement or rupture of the annulus fibrosus. Such occurrences, typically resulting from physical trauma, can yield disabling effects from impingement on spinal nerve structures. A greater understanding of t

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Ho, Clifford K., Cheryl M. Ghanbari, and Richard B. Diver. "Methodology to Assess Potential Glint and Glare Hazards From Concentrating Solar Power Plants: Analytical Models and Experimental Validation." In ASME 2010 4th International Conference on Energy Sustainability. ASMEDC, 2010. http://dx.doi.org/10.1115/es2010-90053.

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With growing numbers of concentrating solar power systems being designed and developed, glint and glare from concentrating solar collectors and receivers is receiving increased attention as a potential hazard or distraction for motorists, pilots, and pedestrians. This paper provides analytical methods to evaluate the irradiance originating from specularly and diffusely reflecting sources as a function of distance and characteristics of the source. Sample problems are provided for both specular and diffuse sources, and validation of the models is performed via testing. In addition, a summary of

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