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Journal articles on the topic 'RETN'

Author: Grafiati

Published: 4 June 2021

Last updated: 8 February 2022

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1

Schwartz, Daniel R., Erika R. Briggs, Mohammed Qatanani, et al. "Human Resistin in Chemotherapy-Induced Heart Failure in Humanized Male Mice and in Women Treated for Breast Cancer." Endocrinology 154, no. 11 (2013): 4206–14. http://dx.doi.org/10.1210/en.2013-1399.

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Resistin is a circulating mediator of insulin resistance mainly expressed in human monocytes and responsive to inflammatory stimuli. Recent clinical studies have connected elevated resistin levels with the development and severity of heart failure. To further our understanding of the role of human resistin in heart failure, we studied a humanized mouse model lacking murine resistin but transgenic for the human Retn gene (Hum-Retn mice), which exhibits basal and inflammation-stimulated resistin levels similar to humans. Specifically, we explored whether resistin underlies acute anthracycline-in

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2

Ibrahim, Sherine M., and Afaf A. Bastawy. "The Relevance of Single-nucleotide Polymorphism +62 G>A to the Expression of Resistin Gene Affecting Serum Resistin Levels in Metabolic Syndrome in the Egyptian Population." Current Pharmaceutical Biotechnology 21, no. 7 (2020): 626–34. http://dx.doi.org/10.2174/1389201021666191210122851.

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Background: Metabolic Syndrome (MS) is a clinical condition consisting of risk factors associated with type two diabetes and developing cardiovascular disease. It has been suggested that resistin is a linkage between obesity, inflammation and type two diabetes. This study aims to investigate whether Resistin Gene (RETN) polymorphism (+62G>A) is linked to MS and resistin levels among the Egyptian population. Methods: This study was performed with 310 Egyptian volunteers: 160 MS subjects and 150 controls. Anthropometric parameters and biochemical variables were determined. The RETN +62G>A

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3

Suriyaprom, Kanjana, Rungsunn Tungtrongchitr, and Pisit Namjuntra. "Associations of Resistin Levels with Resistin Gene Polymorphism and Metabolic Syndrome in Thais / Asocijacija Nivoa Rezistina Sa Polimorfizmom Gena Za Rezistin I Metaboličkim Sindromom Kod Tajlanđana." Journal of Medical Biochemistry 34, no. 2 (2015): 170–78. http://dx.doi.org/10.2478/jomb-2014-0034.

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Summary Background: Metabolic syndrome (MS) is a clinical constellation comprising risk factors associated with developing cardiovascular disease and type 2 diabetes. Resistin has been suggested as a linkage between obesity, inflammation and type 2 diabetes. This study aimed to investigate resistin concentrations and hematological-biochemical parameters in MS subjects and controls, and to determine whether two resistin gene (RETN) polymorphisms (-420C>G & +299G>A) are linked to resistin levels and MS among Thais. Methods: This case-control study was performed with 322 Thai volunteers

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4

Mahmoudi, Touraj, Khatoon Karimi, Maral Arkani, et al. "Resistin -420C>G Promoter Variant and Colorectal Cancer Risk." International Journal of Biological Markers 29, no. 3 (2014): 233–38. http://dx.doi.org/10.5301/jbm.5000079.

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Purposes Obesity is associated with an increased risk of colorectal cancer (CRC), and ghrelin (GHRL) and resistin (RETN) are thought to be related to obesity. Our aim was to investigate whether GHRL and RETN gene variants are associated with CRC risk. Materials and Methods All 414 subjects, including 197 cases with CRC and 217 controls, were genotyped for the GHRL (rs26802) and RETN (rs1862513) or -420C>G gene variants using the PCR-RFLP method. Results Our findings indicated that the RETN -420C>G “CC” genotype, compared with the “GG” and “GC” genotypes, was a marker of decreased CRC sus

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5

Pravenec, Michal, Petr Mlejnek, Václav Zídek, et al. "Autocrine effects of transgenic resistin reduce palmitate and glucose oxidation in brown adipose tissue." Physiological Genomics 48, no. 6 (2016): 420–27. http://dx.doi.org/10.1152/physiolgenomics.00122.2015.

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Resistin has been originally identified as an adipokine that links obesity to insulin resistance in mice. In our previous studies in spontaneously hypertensive rats (SHR) expressing a nonsecreted form of mouse resistin ( Retn) transgene specifically in adipose tissue (SHR- Retn), we have observed an increased lipolysis and serum free fatty acids, ectopic fat accumulation in muscles, and insulin resistance. Recently, brown adipose tissue (BAT) has been suggested to play an important role in the pathogenesis of metabolic disturbances. In the current study, we have analyzed autocrine effects of t

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6

Al-Hilali, Hammadi A., and Mohammed Jaafar AL-Anssari. "Resistin (RETN) Gene rs1862513 Polymorphisms and Acne vulgaris Patients." International Journal of Current Microbiology and Applied Sciences 5, no. 12 (2016): 415–22. http://dx.doi.org/10.20546/ijcmas.2016.512.045.

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7

Wang, Chao-Qun, Chih-Hsin Tang, Huey-En Tzeng, et al. "Impacts of RETN genetic polymorphism on breast cancer development." Journal of Cancer 11, no. 10 (2020): 2769–77. http://dx.doi.org/10.7150/jca.38088.

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8

de Luis, Daniel Antonio, Olatz Izaola, David Primo, Beatriz de la Fuente, Ines Mulero, and Rocío Aller. "The rs1862513 Variant in Resistin Gene-Modified Insulin Resistance and Insulin Levels after Weight Loss Secondary to Hypocaloric Diet." Annals of Nutrition and Metabolism 69, no. 3-4 (2016): 256–62. http://dx.doi.org/10.1159/000453676.

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Background/Aims: Polymorphisms of a single nucleotide in RETN have been associated with indexes of insulin resistance. Our aim was to analyze the effects of the rs1862513 RETN gene polymorphism on insulin resistance, insulin levels, and resistin levels changes after 3 months of a low-fat hypocaloric diet. Design: A Caucasian population of 133 obese patients was analyzed before and after 3 months on a low-fat hypocaloric diet. Results: Fifty-six patients (42.1%) had the genotype GG (wild group) and 77 (57.9%) patients had the other genotypes; GC (59 patients, 44.4%) or CC (18 patients, 13.5%; m

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9

Kawamura, Ryoichi, Yasuharu Tabara, Akiko Tsukada, et al. "Genome-wide association study of plasma resistin levels identified rs1423096 and rs10401670 as possible functional variants in the Japanese population." Physiological Genomics 48, no. 11 (2016): 874–81. http://dx.doi.org/10.1152/physiolgenomics.00040.2016.

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Resistin is a cytokine inducing insulin resistance in mice. We previously identified single nucleotide polymorphisms (SNPs) at −420 (rs1862513) and −358 (rs3219175) located in the human resistin gene ( RETN) promoter as strong determinants for circulating resistin in the Japanese population. The objective was to identify additional functional variants for circulating resistin. We conducted a genome-wide association study in 448 Japanese subjects. A peak association signal was found on chromosome 19 where RETN is located. The top-hit SNP was SNP −358 G>A, followed by rs1423096 C>T, SNP −4

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10

Elkhattabi, Lamiae, Imane Morjane, Hicham Charoute, et al. "In Silico Analysis of Coding/Noncoding SNPs of Human RETN Gene and Characterization of Their Impact on Resistin Stability and Structure." Journal of Diabetes Research 2019 (May 20, 2019): 1–9. http://dx.doi.org/10.1155/2019/4951627.

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Resistin (RETN) is a gene coding for proinflammatory adipokine called resistin secreted by macrophages in humans. Single nucleotide polymorphisms (SNPs) in RETN are linked to obesity and insulin resistance in various populations. Using dbSNP, 78 nonsynonymous SNPs (nsSNPs) were retrieved and tested on a PredictSNP 1.0 megaserver. Among these, 15 nsSNPs were predicted as highly deleterious and thus subjected to further analyses, such as conservation, posttranscriptional modifications, and stability. The 3D structure of human resistin was generated by homology modeling using Swiss model. Root-me

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11

Miyamoto, Yuji, Sebastian Stintzing, Wu Zhang, et al. "Polymorphisms in adipokine-related genes to predict treatment outcomes in patients (pts) with metastatic colorectal cancer (mCRC) treated with bevacizumab-based chemotherapy." Journal of Clinical Oncology 35, no. 4_suppl (2017): 600. http://dx.doi.org/10.1200/jco.2017.35.4_suppl.600.

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600 Background: The molecular effects of obesity are mediated by alterations in the levels of adipokines, which are produced mainly by adipose tissue. Adipokines have multiple effects, including insulin sensitivity, cell proliferation, inflammation, and angiogenesis. We previously reported that single-nucleotide polymorphisms (SNPs) in obesity-related genes were associated with the recurrence probability of CRC treated with 5-FU based adjuvant chemotherapy. This study aimed to evaluate whether SNPs in adipokine-related genes may predict clinical outcomes in bevacizumab treated mCRC pts. Method

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Uchiyama, Tomoko, Asako Itaya-Hironaka, Akiyo Yamauchi та ін. "Intermittent Hypoxia Up-Regulates CCL2, RETN, and TNFα mRNAs in Adipocytes via Down-regulation of miR-452". International Journal of Molecular Sciences 20, № 8 (2019): 1960. http://dx.doi.org/10.3390/ijms20081960.

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Sleep apnea syndrome (SAS), characterized by recurrent episodes of oxygen desaturation and reoxygenation (intermittent hypoxia [IH]), is a risk factor for insulin resistance. Recently, IH is considered to independently cause adipose tissue inflammation/dysfunction, leading to worsening insulin resistance; however, the detailed mechanism remains unknown. We exposed mouse 3T3-L1 and human SW872 adipocytes to experimental IH or normoxia for 24 h, and analyzed mRNA expression of several adipokines. We found that the mRNA levels of RETN, TNFα, and CCL2 in SW872 and 3T3-L1 adipocytes were significan

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13

Wu, Xiaoyan, Elise R. den Boer, Manon Vos-Loohuis, et al. "Investigation of Genetic Modifiers of Copper Toxicosis in Labrador Retrievers." Life 10, no. 11 (2020): 266. http://dx.doi.org/10.3390/life10110266.

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Copper toxicosis is a complex genetic disorder in Labrador retrievers characterized by hepatic copper accumulation eventually leading to liver cirrhosis. The variation of hepatic copper levels in Labrador retrievers has been partly explained by mutations in ATP7A c.980C>T and ATP7B c.4358G>A. To further elucidate the genetic background of this disease, we used targeted Next Generation Sequencing (NGS) in a cohort of 95 Labrador retrievers to analyze 72 potential modifier genes for variations associated with hepatic copper levels. Variants associated with copper levels were subsequently e

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14

Alharithy, Rowyda N. "Polymorphisms in RETN gene and susceptibility to colon cancer in Saudi patients." Annals of Saudi Medicine 34, no. 4 (2014): 334–39. http://dx.doi.org/10.5144/0256-4947.2014.334.

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15

Pechlivanis, Sonali, Justo Lorenzo Bermejo, Barbara Pardini, et al. "Genetic variation in adipokine genes and risk of colorectal cancer." European Journal of Endocrinology 160, no. 6 (2009): 933–40. http://dx.doi.org/10.1530/eje-09-0039.

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ObjectiveObesity has been related to an increased risk of colorectal cancer (CRC). Adipokines produced by the adipose tissue are directly linked to obesity and may thus contribute to the pathogenesis of CRC. We hypothesized that potentially functional polymorphisms in the adipokine genes leptin (LEP), leptin receptor (LEPR), resistin (RETN), and adiponectin (ADIPOQ) may be associated with CRC.Design and methodsWe studied the association of four putatively functional single nucleotide polymorphisms (SNPs) with CRC risk using a hospital-based study design with 702 cases and 752 controls from the

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16

Mahmoudi, Touraj, Keivan Majidzadeh-A, Khatoon Karimi, et al. "Gly972Arg Variant of Insulin Receptor Substrate 1 Gene and Colorectal Cancer Risk in Overweight/Obese Subjects." International Journal of Biological Markers 31, no. 1 (2016): 68–72. http://dx.doi.org/10.5301/jbm.5000159.

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Background Given the major role of obesity and insulin resistance (IR) in colorectal cancer (CRC), we investigated whether genetic variants in ghrelin ( GHRL), resistin ( RETN) and insulin receptor substrate 1 ( IRS1) were associated with CRC risk. Methods This study was conducted as a case-control study, and 750 subjects, including 438 controls and 312 patients with CRC, were enrolled and genotyped using the PCR-RFLP method. Results No significant differences were observed for GHRL (rs696217), RETN (rs3745367) and IRS1 (rs1801278, Gly972Arg or G972R) gene variants between the cases and contro

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17

Chang, Ming-Ling, Su-Wei Chang, Shiang-Chi Chen, et al. "Genetic Association of Hepatitis C-Related Mixed Cryoglobulinemia: A 10-Year Prospective Study of Asians Treated with Antivirals." Viruses 13, no. 3 (2021): 464. http://dx.doi.org/10.3390/v13030464.

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Genetic profiles of hepatitis C virus (HCV)-associated mixed cryoglobulinemia (MC) in Asians remain elusive. A 10-year prospective cohort study was conducted with 1043 consecutive HCV Ab-positive Taiwanese surveyed with 13 single nucleotide polymorphisms (SNPs). Of 1043, 589 (56.5%) had baseline MC, 934 (89.5%) had positive HCV RNA, 796 completed anti-HCV therapy, and 715 had sustained virological responses (SVRs). SNP associations were surveyed withgenotypic, allelic, trend, permutation and multivariate analyses. At baseline, higher male sex and MC rates were noted in HCV RNA-positive than RN

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Hastuti, Pramudji, Tasmini Tasmini, Rizki Fajar Utami, Meirlin Rambu Kaita Riwa, Steven Steven, and Ahmad Hamim Sadewa. "Variation of Resistin Gene Is Correlated with Insulin Resistance in Obese People of Indonesia." Open Access Macedonian Journal of Medical Sciences 7, no. 12 (2019): 1891–95. http://dx.doi.org/10.3889/oamjms.2019.456.

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BACKGROUND: Obesity is considered associated with an increase of resistin levels that plays a role in the regulation of energy and maintaining fasting blood glucose. Polymorphism of resistin is thought to be correlated with the levels of resistin and insulin resistance.AIM: This study aimed to examine the association of +299G > A and -420C > G resistin (RETN) gene with resistin level and insulin resistance in obese people of Indonesia.METHODS: We examined 142 healthy unrelated subjects consisting of 71 obese and 71 controls. Fasting blood glucose was measured by the enzymatic method whil

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19

Menzaghi, Claudia, Angelo Coco, Lucia Salvemini, et al. "Heritability of Serum Resistin and Its Genetic Correlation with Insulin Resistance-Related Features in Nondiabetic Caucasians." Journal of Clinical Endocrinology & Metabolism 91, no. 7 (2006): 2792–95. http://dx.doi.org/10.1210/jc.2005-2715.

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Abstract Context: Serum levels of resistin are believed to modulate insulin resistance in humans. Objective: The aim of this study was to investigate whether serum resistin levels are genetically controlled and whether this control is shared with other insulin resistance traits. Design and Methods: The study cohort included 264 nondiabetic probands, Caucasian from Italy, and their 473 adult family members. Phenotypic characterization included anthropometric variables, blood pressure, fasting glucose and insulin, lipid profile, and resistin levels. Genotypes were determined at position g.−420C→

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Beckers, Sigri, Fenna de Freitas, Doreen Zegers, et al. "Identification and functional characterization of a missense mutation in resistin in two patients with severe obesity and insulin resistance." European Journal of Endocrinology 164, no. 6 (2011): 927–36. http://dx.doi.org/10.1530/eje-10-1080.

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ObjectiveIn this study, we hypothesized that mutations in the resistin encoding gene, RETN, may cause a monogenic form of obesity.Design/methodsWe screened the coding region of RETN in 81 morbidly obese adults, 263 overweight and obese children/adolescents, and 116 healthy lean subjects. In vitro experiments include qPCR, ELISA, and western blot for WT and mutant resistin transfected into 3T3-L1 adipocytes.ResultsMutation analysis identified five sequence variants in our patient populations: 3′-UTR +87 G/A, 3′-UTR +100 A/G, T73T, IV3-61 C/A, and C78S. In our control population, we only found t

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Abd-Elmonsef, Marwa M. E., Naglaa F. Ghoname, Abeer Shahba, Maha M. Hagras, Lamees M. Dawood, and Mohamed M. E. Abd-Elmonsef. "Association of the RETN –420C˃G Polymorphism with Rheumatoid Arthritis in an Egyptian Population." International Journal of Current Microbiology and Applied Sciences 5, no. 11 (2016): 183–95. http://dx.doi.org/10.20546/ijcmas.2016.511.021.

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22

Utami, Rizki Fajar, Pramudji Hastuti, and Ahmad Hamim Sadewa. "RETN rs3745368 polymorphism and resistin level in Javanese ethnic Indonesian obese: a case control study." Jurnal Teknologi Laboratorium 8, no. 1 (2019): 41–49. http://dx.doi.org/10.29238/teknolabjournal.v8i1.164.

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 Obesity has become a global public health problem. It occurs due to a positive energy balance leading to adipose tissue expansion. White adipose tissue was an endocrine organ which secreted resistin. Resistin also produced by immune cells due to low chronic level inflammation might cause higher resistin level in obese people. Polymorphism +62G>A RETN gene was reported has a relationship with low resistin level and A allele as a protective allele. This study aimed to determine genotype and allele frequency distribution concerning resistin level. Another objective aimed to know the cor

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Cieslak, Jakub, Joanna Nowacka-Woszuk, Magdalena Bartz, et al. "Association studies on the porcine RETN, UCP1, UCP3 and ADRB3 genes polymorphism with fatness traits." Meat Science 83, no. 3 (2009): 551–54. http://dx.doi.org/10.1016/j.meatsci.2009.07.001.

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Li, Yahui, Huaixing Li, Shiyuan Dong, Chao Yu, Yu Jiang, and Shuhan Sun. "Influence and related mechanism of Retn gene expression on glucose uptake in 3T3-L1 cells." Frontiers of Medicine in China 1, no. 3 (2007): 269–73. http://dx.doi.org/10.1007/s11684-007-0051-1.

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Chi, Shuguang, Cailian Lan, Sizhong Zhang, et al. "Association of −394C>G and −420C>G polymorphisms in the RETN gene with T2DM and CHD and a new potential SNP might be exist in exon 3 of RETN gene in Chinese." Molecular and Cellular Biochemistry 330, no. 1-2 (2009): 31–38. http://dx.doi.org/10.1007/s11010-009-0097-2.

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Hernández-Romero, D., E. Orenes-Piñero, A. García-Honrubia, et al. "Involvement of the −420C>G RETN polymorphism in myocardial fibrosis in patients with hypertrophic cardiomyopathy." Journal of Internal Medicine 278, no. 1 (2015): 50–58. http://dx.doi.org/10.1111/joim.12334.

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Joyce, Brian T., Huikun Liu, Leishen Wang, et al. "Novel epigenetic link between gestational diabetes mellitus and macrosomia." Epigenomics 13, no. 15 (2021): 1221–30. http://dx.doi.org/10.2217/epi-2021-0096.

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Background & objectives: Examine maternal gestational diabetes mellitus (GDM), macrosomia and DNA methylation in candidate genes IGF1, IGF2, H19, ARHGRF11, MEST, NR3C1, ADIPOQ and RETN. Materials & methods: A total of 1145 children (572 GDM cases and 573 controls) from the Tianjin GDM study, including 177 with macrosomia, had blood DNA collection at median age 5.9 (range: 3.1–10.0). We used logistic regression to screen for associations with GDM and model macrosomia on 37 CpGs, and performed mediation analysis. Results: One CpG was associated with macrosomia at false discovery rate (FD

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Hivert, M. F., A. K. Manning, J. B. McAteer, et al. "Association of Variants in RETN With Plasma Resistin Levels and Diabetes-Related Traits in the Framingham Offspring Study." Diabetes 58, no. 3 (2008): 750–56. http://dx.doi.org/10.2337/db08-1339.

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Onuma, Hiroshi, Yasuharu Tabara, Ryoichi Kawamura, et al. "Dual Effects of a RETN Single Nucleotide Polymorphism (SNP) at –420 on Plasma Resistin: Genotype and DNA Methylation." Journal of Clinical Endocrinology & Metabolism 102, no. 3 (2016): 884–92. http://dx.doi.org/10.1210/jc.2016-2417.

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Kumar, Vikas, Jaswinder Singh, Ashish Aneja, and Jasbir Singh. "Association of RETN gene polymorphism at +299 G>A with type 2 diabetes mellitus: a meta-analysis." International Journal of Diabetes in Developing Countries 40, no. 1 (2019): 12–20. http://dx.doi.org/10.1007/s13410-019-00746-x.

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Kartodihardjo, Hariadi, and Chalid Muhammad. "Mengatasi Persoalan Institusional Pengelolaan Sumber Daya Alam (PSDA): Pembelajaran dari Kasus Revitalisasi Ekosistem Tesso Nilo (RETN) di Provinsi Riau." Jurnal Hukum Lingkungan Indonesia 5, no. 2 (2019): 253. http://dx.doi.org/10.38011/jhli.v5i2.95.

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UCHIYAMA, TOMOKO, ASAKO ITAYA-HIRONAKA, AKIYO YAMAUCHI та ін. "1771-P: Upregulation of RETN, TNFα, and CCL2 mRNAs in Adipocytes by Intermittent Hypoxia and Its MicroRNA-Mediated Mechanism". Diabetes 68, Supplement 1 (2019): 1771—P. http://dx.doi.org/10.2337/db19-1771-p.

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Arráiz, Nailet, Carolina Escalona, Carem Prieto, et al. "Polimorfismo 3’UTR +62G>A del gen RETN codificante de resistina y asociación con componentes del síndrome metabólico." Medicina Clínica 141, no. 8 (2013): 325–31. http://dx.doi.org/10.1016/j.medcli.2012.07.020.

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Saleh, Razwa, Zahidul Islam Zahid, Mohammad Atikur Rahman та ін. "Prevalence of PPAR-γ2 (rs1801282), RETN (rs3745367) and ADIPOQ (rs2241766) SNP markers in the Bangladeshi type 2 diabetic population". Meta Gene 10 (грудень 2016): 100–107. http://dx.doi.org/10.1016/j.mgene.2016.08.002.

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Gao, L., E. Coe, M. Campbell, et al. "Polymorphisms in Resistin (RETN) and Resistin-like Beta (RETNLB) and Risk of Asthma in Independent Populations of African Descent." Journal of Allergy and Clinical Immunology 123, no. 2 (2009): S166. http://dx.doi.org/10.1016/j.jaci.2008.12.623.

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NAWAZ, Yasar, Sumbla GHAZANVI, Nadia RASHEED, Shah JAHAN, and Muhammad Ikram ULLAH. "Association of Serum Resistin Level and Resistin (RETN) Gene (-420C>G) Polymorphism in Pakistani Women with Polycystic Ovarian Syndrome." Turkish Journal of Endocrinology and Metabolism 24, no. 1 (2020): 16–22. http://dx.doi.org/10.25179/tjem.2019-66784.

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Xia, Qing Lin, Liu Xian Pan, Yuan Dong Peng, et al. "First Principles Investigation of Electronic Structure, Chemical Bonding, Elastic and Optical Properties of Novel Rhenium Nitrides." Key Engineering Materials 512-515 (June 2012): 883–89. http://dx.doi.org/10.4028/www.scientific.net/kem.512-515.883.

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we investigate the electronic structure, chemical bonding, optical and elastic properties of the novel rhenium nitrides, hexagonal phase re3n and re2n by using density-functional theory (dft) within generalized gradient approximation (gga). the calculated equilibrium lattice constants of both re3n and re2n are in reasonable agreement with the experimental results. the band structure along the higher symmetry axes in the Brillouin zone, the density of states (dos) and the partial density of states (pdos) are presented. the calculated energy band structures and dos show that re3n and re2n are me

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López-Quintero, A., A. G. García-Zapién, S. E. Flores-Martínez, et al. "Contribution of polymorphisms in the LEP, LEPR and RETN genes on serum leptin and resistin levels in young adults from Mexico." Cellular and Molecular Biology 63, no. 8 (2017): 10. http://dx.doi.org/10.14715/cmb/2017.63.8.3.

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Zhou, Qiang, Bo Chen, Tianxing Ji, Miaoshan Luo, and Jiandong Luo. "Association of genetic variants in RETN, NAMPT and ADIPOQ gene with glycemic, metabolic traits and diabetes risk in a Chinese population." Gene 642 (February 2018): 439–46. http://dx.doi.org/10.1016/j.gene.2017.10.084.

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40

Wang, Li, Zhou, et al. "Association of Twelve Candidate Gene Polymorphisms with the Intramuscular Fat Content and Average Backfat Thickness of Chinese Suhuai Pigs." Animals 9, no. 11 (2019): 858. http://dx.doi.org/10.3390/ani9110858.

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The present study aimed to identify the molecular markers for genes that influence intramuscular fat content (IFC), but not average backfat thickness (ABT). A total of 330 Suhuai pigs were slaughtered, and measurements of IFC and ABT were obtained. Phenotypic and genetic correlations between IFC and ABT were calculated. Thirteen single nucleotide polymorphisms (SNPs) among 12 candidate genes for IFC were analyzed, including FABP3, LIPE, IGF1, IGF2, LEP, LEPR, MC4R, PHKG1, RETN, RYR1, SCD, and UBE3C. Associations of the evaluated SNPs with IFCIFC and ABT were performed. Our results showed that

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Yang, Wei-Hung, Shoou-Jyi Wang, Yung-Sen Chang, Chen-Ming Su, Shun-Fa Yang, and Chih-Hsin Tang. "Association of Resistin Gene Polymorphisms with Oral Squamous Cell Carcinoma Progression and Development." BioMed Research International 2018 (October 14, 2018): 1–8. http://dx.doi.org/10.1155/2018/9531315.

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Oral squamous cell carcinoma (OSCC) accounts for over 90% of malignant neoplasms of the mouth. In Taiwan, OSCC is the fourth most common male cancer and the fourth leading cause of male cancer death. Resistin (RETN) is an adipokine that is associated with obesity, inflammation, and various cancers. Here, we examine the association between four single nucleotide polymorphisms (SNPs) of theRETNgene (rs3745367, rs7408174, rs1862513, and rs3219175) and OSCC susceptibility as well as clinical outcomes in 935 patients with OSCC and in 1200 cancer-free healthy controls. We found that, in 1465 smokers

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Elkhattabi, L., I. Morjane, H. Charoute, R. Saile, and A. Barakat. "Computational screening and analysis of the functional and structural impact of SNPS of the human RETN gene associated to type 2 diabetes." Atherosclerosis 315 (December 2020): e199-e200. http://dx.doi.org/10.1016/j.atherosclerosis.2020.10.622.

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Butera, Federico, and Fernando Alberti. "Il governo delle reti inter-organizzative per la competitivitŕ." STUDI ORGANIZZATIVI, no. 1 (December 2012): 77–111. http://dx.doi.org/10.3280/so2012-001004.

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I policy maker sono costantemente alla ricerca delle forme e degli strumenti per contribuire ad aumentare la prosperitŕ economica e sociale del proprio territorio. Gli studi a livello internazionale ci dicono che la prosperitŕ di un territorio č direttamente riconducibile alla sua competitivitŕ, e quindi in primis al livello di produttivitŕ e innovazione del sistema delle imprese. Come verrŕ ampiamente illustrato in questo articolo, le reti inter-organizzative - nella varietŕ di forme che l'evidenza empirica ci suggerisce - attraverso una flessibilitŕ senza precedenti, una piů veloce circolazi

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Deligoz, E., K. Colakoglu, H. B. Ozisik, and Y. O. Ciftci. "Vibrational properties of Re2N and Re3N compounds." Solid State Communications 151, no. 17 (2011): 1122–27. http://dx.doi.org/10.1016/j.ssc.2011.05.028.

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Alberti, Fernando G. "Il contratto di rete: una rassegna." STUDI ORGANIZZATIVI, no. 2 (April 2013): 176–97. http://dx.doi.org/10.3280/so2012-002007.

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Il presente articolo intende essere una breve nota tesa a fare il punto dello stato di adozione e di elaborazione dei contratti di rete. Con il contratto di rete, il Legislatore ha voluto introdurre una nuova fattispecie giuridica finalizzata a favorire l'aggregazione tra imprese di minore dimensione, aumentando cosě le dimensioni aziendali per meglio competere nei mercati internazionali e supportare l'innovazione. Il contratto di rete si differenzia nella ratio da altre forme burocratiche di reti inter-organizzative esistenti, per maggiore flessibilitŕ sia nella definizione degli scopi e dei

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Wang, Juanjuan, Mengsi Xu, Xinhua Wang, et al. "Comparative Proteome Analysis Reveals Lipid Metabolism-Related Protein Networks in Response to Rump Fat Mobilization." International Journal of Molecular Sciences 19, no. 9 (2018): 2556. http://dx.doi.org/10.3390/ijms19092556.

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Altay is a typical fat-tailed sheep breed displaying the unique ability to rapidly mobilize fat, which is vital for maintaining a normal metabolism that facilitates its survival in lengthy winter conditions. However, the physiological, biochemical, and molecular mechanisms underlying fat mobilization remain to be elucidated. In this study, the monitoring of rump fat adipocyte sizes disclosed a positive correlation between cell size and fat deposition ability. In addition, we subjected sheep to persistent starvation to imitate the conditions that trigger rump fat mobilization and screened 112 d

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Vashi, Neeti, Carolina Stryjecki, Jesus Peralta-Romero, et al. "Genetic markers of inflammation may not contribute to metabolic traits in Mexican children." PeerJ 4 (June 23, 2016): e2090. http://dx.doi.org/10.7717/peerj.2090.

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Background:Low-grade chronic inflammation is a common feature of obesity and its cardio-metabolic complications. However, little is known about a possible causal role of inflammation in metabolic disorders. Mexico is among the countries with the highest obesity rates in the world and the admixed Mexican population is a relevant sample due to high levels of genetic diversity.Methods:Here, we studied 1,462 Mexican children recruited from Mexico City. Six genetic variants in five inflammation-related genes were genotyped: rs1137101 (leptin receptor (LEPR)), rs7305618 (hepatocyte nuclear factor 1

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Geraci, Salvatore, and Chiara Bodini. "Rete di reti per la salute degli immigrati." SALUTE E SOCIETÀ, no. 2 (November 2011): 129–40. http://dx.doi.org/10.3280/ses2011-002010.

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TIMIRCI-KAHRAMAN, OZLEM, UMIT YILMAZ, NESIBE YILMAZ, et al. "A Study of Short- and Long-term mRNA Levels of the Retn, Iapp, and Drd5 Genes in Obese Mice Induced with High-fat Diet." In Vivo 32, no. 4 (2018): 813–17. http://dx.doi.org/10.21873/invivo.11312.

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Martínez-Paz, Pedro, Marta Aragón-Camino, Esther Gómez-Sánchez, et al. "Gene Expression Patterns Distinguish Mortality Risk in Patients with Postsurgical Shock." Journal of Clinical Medicine 9, no. 5 (2020): 1276. http://dx.doi.org/10.3390/jcm9051276.

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Nowadays, mortality rates in intensive care units are the highest of all hospital units. However, there is not a reliable prognostic system to predict the likelihood of death in patients with postsurgical shock. Thus, the aim of the present work is to obtain a gene expression signature to distinguish the low and high risk of death in postsurgical shock patients. In this sense, mRNA levels were evaluated by microarray on a discovery cohort to select the most differentially expressed genes between surviving and non-surviving groups 30 days after the operation. Selected genes were evaluated by qu

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