Relevant bibliographies by topics / Reverse cholesterol transport pathway

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Reverse cholesterol transport pathway'

Author: Grafiati
Published: 2 June 2025
Last updated: 31 July 2025

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Journal articles on the topic "Reverse cholesterol transport pathway"

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Kuypers, Frans A., Sandra Larkin, Jenifer Beckstead, Michael Oda, Kazumitsu Ueda, and Robert O. Ryan. "Red Blood Cells Facilitate Reverse Cholesterol Transport." Blood 104, no. 11 (2004): 1589. http://dx.doi.org/10.1182/blood.v104.11.1589.1589.

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Abstract Lecithin:cholesterol acyl transferase (LCAT)-dependent conversion of cholesterol (CH) to cholesteryl ester (CE), a key component of the reverse cholesterol transport (RCT) pathway, is essential for cholesterol processing. We hypothesized that red blood cells (RBCs) function in this pathway by facilitating phosphatidylcholine (PC) re-generation from LCAT-derived lysophosphatidylcholine (LPC). Addition of 14C-oleate to fresh RBCs resulted in an ATP-dependent incorporation of radiolabel into PC via the Lands pathway. Prior depletion of red cell LPC content reduced the incorporation of 14
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Zhang, Xinyuan, Kaiyue Wang, Ling Zhu, and Qiyun Wang. "Reverse Cholesterol Transport Pathway and Cholesterol Efflux in Diabetic Retinopathy." Journal of Diabetes Research 2021 (October 26, 2021): 1–11. http://dx.doi.org/10.1155/2021/8746114.

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Cholesterol esters, synthesized from cholesterol with long-chain fatty acids, are essential components of plasma lipoproteins and cell membranes that participate in various metabolic processes in the body. Cholesterol can be excreted through the cholesterol reverse transport (RCT) pathway when excessive cholesterol is produced in the extrahepatic cells, which is regulated by the liver X receptor (LXR) and its downstream regulators ATP-binding cassette subfamily A member 1 (ABCA1) and ATP-binding cassette subfamily G member 1 (ABCG1) genes. Abnormal cholesterol metabolism is closely associated
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Moradi, Hamid, Jun Yuan, Zhemin Ni, Keith Norris, and Nosratola D. Vaziri. "Reverse Cholesterol Transport Pathway in Experimental Chronic Renal Failure." American Journal of Nephrology 30, no. 2 (2009): 147–54. http://dx.doi.org/10.1159/000210020.

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Wang, David Q. H., Piero Portincasa, and Patrick Tso. "Transintestinal cholesterol excretion: A secondary, nonbiliary pathway contributing to reverse cholesterol transport." Hepatology 66, no. 4 (2017): 1337–40. http://dx.doi.org/10.1002/hep.29341.

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Alam, Khairul, Robert S. Meidell, and David K. Spady. "Effect of Up-regulating Individual Steps in the Reverse Cholesterol Transport Pathway on Reverse Cholesterol Transport in Normolipidemic Mice." Journal of Biological Chemistry 276, no. 19 (2001): 15641–49. http://dx.doi.org/10.1074/jbc.m010230200.

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Bellanger, Natacha, Alexina Orsoni, Zélie Julia, et al. "Atheroprotective Reverse Cholesterol Transport Pathway Is Defective in Familial Hypercholesterolemia." Arteriosclerosis, Thrombosis, and Vascular Biology 31, no. 7 (2011): 1675–81. http://dx.doi.org/10.1161/atvbaha.111.227181.

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Chao, W. T., and V. C. Yang. "Visualization of Uptake of High-Density Lipoprotein by Rat Aortic Endothelial Cells and Smooth Mus.Cle Cells in Vitro." Microscopy and Microanalysis 6, S2 (2000): 482–83. http://dx.doi.org/10.1017/s1431927600034905.

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The high concentration of low-density lipoprotein in the plasma is the major risk factor of atherosclerosis. On the other hand, another plasma lipoprotein—high-density lipoprotein (HDL) — is inversely correlated with atherosclerosis. Recent studies have demonstrated that HDL mediates the transport of cholesterol from peripheral tissues to the liver through “reverse cholesterol transport” pathway. However there is considerable debate about the mechanisms by which HDL removes excess cholesterol from cells. Two different pathways were suggested: (i) a docking receptor promoting cholesterol transl
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Getz, Godfrey, and Catherine Reardon. "Apoprotein E and Reverse Cholesterol Transport." International Journal of Molecular Sciences 19, no. 11 (2018): 3479. http://dx.doi.org/10.3390/ijms19113479.

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Apoprotein E (apoE) is a multifunctional protein. Its best-characterized function is as a ligand for low-density lipoprotein (LDL) receptor family members to mediate the clearance of apoB-containing atherogenic lipoproteins. Among its other functions, apoE is involved in cholesterol efflux, especially from cholesterol-loaded macrophage foam cells and other atherosclerosis-relevant cells, and in reverse cholesterol transport. Reverse cholesterol transport is a mechanism by which excess cellular cholesterol is transported via lipoproteins in the plasma to the liver where it can be excreted from
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Motte, Alexandre, Julie Gall, Joe-Elie Salem, et al. "Reduced Reverse Cholesterol Transport Efficacy in Healthy Men with Undesirable Postprandial Triglyceride Response." Biomolecules 10, no. 5 (2020): 810. http://dx.doi.org/10.3390/biom10050810.

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Elevation of nonfasting triglyceride (TG) levels above 1.8 g/L (2 mmol/L) is associated with increased risk of cardiovascular diseases. Exacerbated postprandial hypertriglyceridemia (PP–HTG) and metabolic context both modulate the overall efficacy of the reverse cholesterol transport (RCT) pathway, but the specific contribution of exaggerated PP–HTG on RCT efficacy remains indeterminate. Healthy male volunteers (n = 78) exhibiting no clinical features of metabolic disorders underwent a postprandial exploration following consumption of a typical Western meal providing 1200 kcal. Subjects were s
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Weingärtner, Oliver, Ulrich Laufs, Michael Böhm, and Dieter Lütjohann. "An alternative pathway of reverse cholesterol transport: The oxysterol 27-hydroxycholesterol." Atherosclerosis 209, no. 1 (2010): 39–41. http://dx.doi.org/10.1016/j.atherosclerosis.2009.09.015.

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Dissertations / Theses on the topic "Reverse cholesterol transport pathway"

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Catalano, Giovanna. "Role of HDL particles in the reverse cholesterol transport pathway : a clinical need to evaluate HDL functionality." Paris 6, 2008. http://www.theses.fr/2008PA066420.

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Les taux de cholestérol-HDL (C-HDL) représentent un facteur de risque indépendant de maladie cardiovasculaire et des taux élèves de C-HDL sont corrélés à une diminution du risque cardiovasculaire. Dans le contexte du transport inverse du cholestérol, la sortie du cholestérol des cellules et la délivrance des esters de cholestérol au niveau hépatique représentent les étapes majeures impliquant les particules HDL. Au cours des dyslipidémies athérogènes les concentrations plasmatiques de C-HDL ainsi que la distribution du cholestérol au sein des sous populations de HDL sont altérés. La protéine c
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Zhou, Huali. "Reverse cholesterol transport in type 2 diabetes mellitus." Click to view the E-thesis via HKUTO, 2008. http://sunzi.lib.hku.hk/hkuto/record/B39794003.

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Zhou, Huali, and 周華麗. "Reverse cholesterol transport in type 2 diabetes mellitus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2008. http://hub.hku.hk/bib/B39794003.

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Cooke, Conrad Martin. "Investigation of reverse cholesterol transport in humans in vivo." Thesis, Queen Mary, University of London, 2003. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.405731.

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Sabeva, Nadezhda Steliyanova. "REGULATION OF ABCG5 AND ABCG8 STEROL TRANSPORTERS IN BILIARY CHOLESTEROL ELIMINATION, REVERSE CHOLESTEROL TRANSPORT AND DYSLIPIDEMIA." UKnowledge, 2011. http://uknowledge.uky.edu/gradschool_diss/193.

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ATP-binding cassette transporters ABCA1 and ABCG1 initiate reverse cholesterol transport generating HDL particles, whereas ABCG5/G8 promote biliary cholesterol secretion thereby facilitating the last step of reverse cholesterol transport. Mutations in the leptin axis result in obesity and dyslipidemia in ob/ob and db/db mice. These mice have defective HDL clearance, increased plasma cholesterol and decreased biliary cholesterol elimination. My studies demonstrate that ABCG5/G8 protein is low in these animals and can be restored with caloric restriction or leptin replacement. To directly test w
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Teh, Evelyn Mei-lin. "Molecular defects and structure/function studies of proteins in reverse cholesterol transport." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape7/PQDD_0017/NQ57371.pdf.

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Shen, Haiqing. "New candidate gene involved in reverse cholesterol transport (RCT) : the case for phospholipid transfer protein (PLTP): interactions with dietary factors /." Thesis, Connect to Dissertations & Theses @ Tufts University, 2004.

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Thesis (Ph.D.)--Tufts University, 2004.<br>Adviser: Jose Ordovas. Submitted to the School of Nutrition Science and Policy. Includes bibliographical references (leaves 136-137). Access restricted to members of the Tufts University community. Also available via the World Wide Web;
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Sparks, Daniel Leslie. "The effect of variations in HDL composition on components of reverse cholesterol transport." Thesis, University of British Columbia, 1989. http://hdl.handle.net/2429/29290.

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This thesis is concerned with the mechanism which underlies the apparent anti-atherogenic capacity of high density lipoproteins (HDL). Specifically, the relationship between HDL composition, cholesteryl ester transfer protein (CETP) and lecithin: cholesterol acyltransferase (LCAT) was investigated. A novel assay was developed which allowed for the determination of the rate of transfer of [³H]cholesteryl ester from agarose-bound HDL to the endogenous lipoproteins of a plasma sample. Comparison of this assay with an established method, which utilized exogenous lipoprotein substrates, indicated
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Al-Khfajy, Wrood Salim Dawood. "Role Of Transmembrane 141 in Cholesterol Metabolism." Kent State University / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=kent1416142859.

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Mindham, Malcolm A. "Development, validation and application of the isolated perfused rat spleen for the study of reverse cholesterol transport." Thesis, Royal Veterinary College (University of London), 1990. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.522691.

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Books on the topic "Reverse cholesterol transport pathway"

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Insulin Regulation of Reverse Cholesterol Transport. [publisher not identified], 2019.

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Book chapters on the topic "Reverse cholesterol transport pathway"

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van Tol, A. "Reverse Cholesterol Transport." In Recent Developments in Lipid and Lipoprotein Research. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83665-7_11.

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Hoeg, Jeffrey M., and Alan T. Remaley. "Reverse Cholesterol Transport." In Genetic factors in coronary heart disease. Springer Netherlands, 1994. http://dx.doi.org/10.1007/978-94-011-1130-0_25.

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Tauler Riera, Pedro, Maurizio Volterrani, Ferdinando Iellamo, et al. "Reverse Cholesterol Transport (RCT)." In Encyclopedia of Exercise Medicine in Health and Disease. Springer Berlin Heidelberg, 2012. http://dx.doi.org/10.1007/978-3-540-29807-6_2980.

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Favari, Elda, Angelika Chroni, Uwe J. F. Tietge, Ilaria Zanotti, Joan Carles Escolà-Gil, and Franco Bernini. "Cholesterol Efflux and Reverse Cholesterol Transport." In High Density Lipoproteins. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-09665-0_4.

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Chiesa, G., L. Calabresi, G. Franceschini, and C. R. Sirtori. "Drugs Affecting Reverse Cholesterol Transport." In Drugs Affecting Lipid Metabolism. Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1703-6_16.

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Lund-Katz, Sissel, and Michael C. Phillips. "High Density Lipoprotein Structure–Function and Role in Reverse Cholesterol Transport." In Cholesterol Binding and Cholesterol Transport Proteins:. Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-8622-8_7.

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Ghiselli, G., R. Musanti, and A. M. Gotto. "Current concepts in reverse cholesterol transport." In Atherosclerosis and Cardiovascular Disease. Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-0731-7_38.

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Franceschini, Guido, Monica Gomaraschi, and Laura Calabresi. "HDL, Reverse Cholesterol Transport, and Atherosclerosis." In Nutritional and Metabolic Bases of Cardiovascular Disease. Wiley-Blackwell, 2011. http://dx.doi.org/10.1002/9781444318456.ch20.

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Reichl, D. "Initial Stages of Reverse Transport of Cholesterol." In Drugs Affecting Lipid Metabolism. Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-71702-4_42.

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Hersberger, M., and A. von Eckardstein. "Modulation of High-Density Lipoprotein Cholesterol Metabolism and Reverse Cholesterol Transport." In Atherosclerosis: Diet and Drugs. Springer Berlin Heidelberg, 2005. http://dx.doi.org/10.1007/3-540-27661-0_20.

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Conference papers on the topic "Reverse cholesterol transport pathway"

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Hernández Martín, Marina, Rocío Redondo Castillejo, Paula Ortega Menéndez, et al. "Silicon intake reduces hypercholesterolemia facilitating reverse cholesterol transport through intestinal activation of LXR/ABC transporters pathway in type 2 diabetic rats." In 7th International Electronic Conference on Medicinal Chemistry. MDPI, 2021. http://dx.doi.org/10.3390/ecmc2021-11503.

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Wake, Amanda K., Geeta Datta, Mayakonda N. Palgunachari, Vinod K. Mishra, G. M. Anantharamaiah, and C. Roger White. "Apolipoprotein A-I Mimetic Peptide Retains Function After Oxidant Exposure." In ASME 2008 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2008. http://dx.doi.org/10.1115/sbc2008-189660.

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Mechanical integrity of arteries is vital for maintaining compensatory mechanisms under physiologic conditions, and changes in vessel structure (and consequently, vessel mechanical properties) are hallmarks of the progression of pathologic states. Oxidative stress can (1) induce endothelial dysfunction by interfering with nitric oxide (NO) signaling pathways relevant to vasodilation/vasoconstriction of arteries, and (2) alter atherosclerotic plaque composition by oxidation of constituents or by interruption of reverse cholesterol transport from plaques.
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Adak, Asish, Arpita Devi, and Praveen Kumar Gupta. "Effect of reverse cholesterol transport on porous media type ABC fractional atherosclerosis model with memory function." In PROBLEMS IN THE TEXTILE AND LIGHT INDUSTRY IN THE CONTEXT OF INTEGRATION OF SCIENCE AND INDUSTRY AND WAYS TO SOLVE THEM: PTLICISIWS-2. AIP Publishing, 2024. http://dx.doi.org/10.1063/5.0201315.

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Baos, S. Pérez, JI Barrasa, P. Gratal, et al. "FRI0071 Tofacitinib restores the inhibition of reverse cholesterol transport induced by inflammation: understanding the lipid paradox associated with rheumatoid arthritis." In Annual European Congress of Rheumatology, 14–17 June, 2017. BMJ Publishing Group Ltd and European League Against Rheumatism, 2017. http://dx.doi.org/10.1136/annrheumdis-2017-eular.2693.

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Charles-Schoeman, Christina, Thierry Sornasse, and Jeremy Sokolove. "THU0166 TREATMENT WITH UPADACITINIB IS ASSOCIATED WITH IMPROVEMENTS IN REVERSE CHOLESTEROL TRANSPORT IN PATIENTS WITH RHEUMATOID ARTHRITIS: CORRELATION WITH CHANGES IN INFLAMMATION AND HDL LEVELS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.2983.

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Coloma, Mikhail, William M. Buehler, J. David Schaffer, Paul R. Chiarot, and Peter Huang. "Modeling Low Reynolds Number Flows Driven by Forward-Propagating and Reflected Boundary Waves in Concentric Micro-Cylinders." In ASME 2015 13th International Conference on Nanochannels, Microchannels, and Minichannels collocated with the ASME 2015 International Technical Conference and Exhibition on Packaging and Integration of Electronic and Photonic Microsystems. American Society of Mechanical Engineers, 2015. http://dx.doi.org/10.1115/icnmm2015-48463.

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We report on a computational model used to study the reversal of flow direction inside the annular region between concentric micro-cylinders filled with an incompressible Newtonian fluid. The flow is induced by boundary deformations on the inner and outer cylinder surfaces due to forward-propagating transverse waves and their reflections. This microfluidic transport mechanism is postulated as a vital pathway for removal of beta-amyloid from the brain along sub-millimeter cerebral arteries, and failure of this clearance is associated with Alzheimer’s disease. We show that the direction of this
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Villeneuve, Pierre, Claire Bourlieu-Lacanal, David McClements, Eric Decker, and Erwann Durand. "Lipid oxidation in emulsions and bulk oils: A review of the importance of micelles." In 2022 AOCS Annual Meeting & Expo. American Oil Chemists' Society (AOCS), 2022. http://dx.doi.org/10.21748/lzak8107.

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Lipid oxidation is a major cause of quality deterioration in food or cosmetic products. In these matrices, lipids are often present in a bulk or in emulsified forms. In both systems, the rate, extent and pathway of oxidation are highly dependent on the presence of colloidal structures and interfaces because these are the locations where oxidation normally occurs. In bulk oils, reverse micelles (association colloids) are present and are believed to play a crucial role on lipid oxidation. Conversely, in emulsions, surfactant micelles are present that also play a major role in lipid oxidation p
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