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Books on the topic 'Rheumatic fever and rheumatic heart disease'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

All India Symposium on Rheumatic Fever/Rheumatic Heart Disease Complex in India (1983 New Delhi, India). Rheumatic fever and rheumatic heart disease complex in India, present and future: Proceedings from the All India Symposium on Rheumatic Fever/Rheumatic Heart Disease Complex in India, December, 1983. Jaypee Bros., 1987.

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2

Rheumatic fever and rheumatic heart disease: Report of a WHO Expert Consultation, Geneva, 29 October - 1 November, 2001. World Health Organization, 2004.

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3

1938-, Lever John V., ed. Rheumatic diseases and the heart. Springer-Verlag, 1988.

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4

Rheumatic fever and streptococcal infection: Unraveling the mysteries of a dread disease. Francis A. Countway Library of Medicine, 1997.

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5

Cosh, John A. Rheumaticdiseases and the heart. Springer-Verlag, 1988.

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6

Rheumatic Fever and Rheumatic Heart Disease. Jaypee Brothers Medical Publishers (P) Ltd., 2013. http://dx.doi.org/10.5005/jp/books/11741.

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7

SATPATHY, SATPATHY, and SATPATHY. Rheumatic Fever and Rheumatic Heart Disease. JAYPEE, 2013.

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8

Acute Rheumatic Fever and Rheumatic Heart Disease. Elsevier, 2021. http://dx.doi.org/10.1016/c2017-0-03010-0.

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9

Wilson, Nigel, Scott Dougherty, Bongani Mayosi, and Jonathan Carapetis. Acute Rheumatic Fever and Rheumatic Heart Disease. Elsevier, 2020.

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10

World Health Organization (WHO). Rheumatic Fever and Rheumatic Heart Disease (Technical Reports). World Health Organization, 1988.

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11

Acute Rheumatic Fever and Chronic Rheumatic Heart Disease. Jaypee Brothers Medical Publishers (P) Ltd., 2011. http://dx.doi.org/10.5005/jp/books/11212.

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12

Lawrenson, John. Pathogenesis: from acute rheumatic fever to rheumatic heart disease. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0280.

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Rheumatic heart disease is considered to be an autoimmune disease; the trigger of the process is a streptococcal throat infection which then initiates both a humeral and a cellular immune response in environmentally and genetically susceptible individuals.

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13

WHO Study Group on Rheumatic Fever and Rheumatic Heart Disease., ed. Rheumatic fever and rheumatic heart disease: Report of a WHO study group. World Health Organization, 1988.

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14

O, W. H. Rheumatic Fever and Rheumatic Heart Disease : Report of a WHO Study Group. B.R. Publishing Corporation, 1991.

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15

Kaplan, Edward L., and Shanthi Mendis. Rheumatic Fever and Rheumatic Heart Disease (Technical Report Series, No. 923) (Technical Report Series). World Health Organization, 2004.

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16

Ramrakha, Punit, and Jonathan Hill, eds. Valvular heart disease. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199643219.003.0003.

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General considerations 144Acute rheumatic fever 146Mitral stenosis: clinical features 150Mitral stenosis: investigations 152Mitral stenosis guidelines 156Mitral regurgitation 158Mitral regurgitation guidelines 161Mitral valve prolapse 162Aortic stenosis 164Management of aortic stenosis 168Aortic regurgitation 170Aortic regurgitation guidelines ...

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17

Popescu, Bogdan A., Shantanu P. Sengupta, Niloufar Samiei, and Anca D. Mateescu. Heart valve disease (mitral valve disease): mitral stenosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0035.

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The most common cause of mitral stenosis (MS) is rheumatic fever followed by degenerative MS. Echocardiography is the key method to diagnose and evaluate MS. Echocardiographic findings are closely related to aetiology. In rheumatic disease echocardiography shows thickening of leaflet tips with restricted opening caused by commissural fusion resulting in ‘doming’ of the mitral valve in diastole. Quantitation of MS severity includes measuring mitral valve area (MVA) by planimetry (anatomical area, by two-/three-dimensional echo), or by the pressure half-time (PHT) method (functional area, by Doppler), and the mean pressure gradient. Planimetry is considered the reference method to determine MVA as it is relatively load independent. The PHT method is widely used due to its simplicity, but different factors influence the relationship between PHT and MVA. Other indices of MS severity are rarely used in clinical practice. Echocardiography also helps in the assessment of consequences of MS, and of associated valvular lesions. Exercise Doppler is recommended when there is discrepancy between the resting echocardiography findings and the clinical picture. Echocardiography is crucial in determining the timing and type of intervention in patients with MS. When considering percutaneous mitral commissurotomy (PMC) valve morphology should be comprehensively evaluated for mobility, thickness, calcifications, and subvalvular apparatus. The echo findings may determine the suitability for PMC, guide the procedure, and assess its results.

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18

Fye, W. Bruce. Surgeons Begin Trying to Treat Heart Disease. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199982356.003.0009.

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The development of heart surgery lagged behind operations on other organs. In the 1920s surgeons in Boston and in Europe attempted to open mitral valves that had become obstructed as a complication of rheumatic fever. Most of their patients died, and the operation was abandoned until after World War II. Operations to treat children with specific types of congenital heart disease were developed between 1938 and 1944. But these procedures involved the blood vessels outside the heart rather than structures within it. After the war, surgeons in Boston, Philadelphia, and London showed that it was safe to operate on patients with severe mitral stenosis. Without surgery, these individuals would die of heart failure. Mid-century optimism about the potential of treating patients with heart disease was fueled by the discovery of so-called miracle drugs, such as penicillin and cortisone (for which two Mayo staff members shared the Nobel Prize in 1950).

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19

Macleod, Dr Donald C., Dr Ian Scott, Professor Calum Archibald Macrae, et al. Cardiac diseases and resuscitation. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.0004.

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Chapter 4 discusses cardiac diseases and resuscitation, including symptoms, signs, and diagnostic investigations in cardiac disease, adult cardiopulmonary resuscitation, cardiovascular risk assessment, heart failure, acute coronary syndromes, arrhythmias, hypertension and hypertensive emergencies, thromboembolic disease, valvular disease, infective endocarditis, cardiomyopathies, congenital heart disease, heart disease in pregnancy, diseases of arteries and veins, rheumatic fever, pericarditis, ST segment elevation, and myocardial infarction.

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20

Devlin, Hugh, and Rebecca Craven. Heart and blood supply. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198759782.003.0009.

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The heart in relation to dentistry is the topic of this chapter. Heart physiology is described with respect to the cardiac cycle, control of contraction, ECG, and arrhythmias. Control of the cardiovascular system is next considered and the clinical application of this in fainting, shock, and blood loss. Atherosclerosis, angina, and myocardial infarction are described. This leads to a discussion of heart failure and drugs commonly used in cardiovascular disease. Infective endocarditis and rheumatic fever are discussed and the associations between oral bacteria and cardiovascular disease. The concluding section deals with stroke (cerebrovascular accident or CVA), transient ischaemic attacks (TIA) and vascular dementia.

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21

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Rheumatic fever. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.1838_update_002.

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22

Cosh, John A. Rheumatic Diseases and the Heart. Springer, 2011.

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23

Rheumatic Diseases and the Heart. Springer, 2011.

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24

Katritsis, Demosthenes G., Bernard J. Gersh, and A. John Camm. Pseudoaneurysms of the heart. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199685288.003.1884.

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25

Knaggs, Henry Valentine. Rheumatism and other Allied Ailments - Short Chapters on Rheumatism , The Uric Acid Theory of Disease, Rheumatic Fever and Arthritis. Pomona Press, 2006.

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26

Basso, Cristina, Gaetano Thiene, and Siew Yen Ho. Heart valve disease (aortic valve disease): anatomy and pathology of the aortic valve. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0031.

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The gross features of the aortic valve apparatus, consisting of three semilunar leaflets, three interleaflet triangles, three commissures, and the aortic wall, are discussed both in terms of normal and pathological anatomy. The concept of aortic annulus and the relationship of the aortic valve with the coronary arteries, the membranous septum, and conduction system and the mitral valve are addressed. When dealing with pathology, the chapter focuses on the main distinctive features of aortic valve stenosis and aortic valve incompetence. Regarding the former, the abnormalities reside in the cusps, either two or three in number, with cusp thickening, and calcification with or without commissural fusion (thus distinguishing senile and chronic rheumatic valve disease); in the latter, the gross changes can affect either the cusps (infective endocarditis with tissue perforation/laceration and rheumatic valve disease with tissue retraction) or the aortic wall (ascending aorta aneurysm either inflammatory or degenerative). The distinctive gross abnormalities in the various conditions are illustrated.

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27

Adebajo, Ade, and Lisa Dunkley. Polyarticular disease. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0009.

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Polyarticular disease is a commonly encountered musculoskeletal problem which regularly confronts clinicians as a diagnostic dilemma. Polyarticular disease is a musculoskeletal presentation in which more than four joints are affected by the disease. The classical rheumatological condition which presents as polyarticular disease is rheumatoid arthritis, although even this condition can very occasionally present with a mono- or oligoarticular onset. Polyarticular disease includes a wide range of musculoskeletal conditions including such disorders as polyarticular gout, the seronegative spondyloarthropathies, rheumatic fever, and systemic lupus erythematosus. This chapter emphasizes how through a careful history, thorough clinical examination and appropriate investigations, a definitive diagnosis can be made in a patient presenting clinically with polyarticular disease. General management principles for patients with polyarticular disease are also provided.

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28

Adebajo, Ade, and Lisa Dunkley. Polyarticular disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0009_update_001.

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Polyarticular disease is a commonly encountered musculoskeletal problem which regularly confronts clinicians as a diagnostic dilemma. Polyarticular disease is a musculoskeletal presentation in which more than four joints are affected by the disease. The classical rheumatological condition which presents as polyarticular disease is rheumatoid arthritis, although even this condition can very occasionally present with a mono- or oligoarticular onset. Polyarticular disease includes a wide range of musculoskeletal conditions including such disorders as polyarticular gout, the seronegative spondyloarthropathies, rheumatic fever, and systemic lupus erythematosus. This chapter emphasizes how through a careful history, thorough clinical examination and appropriate investigations, a definitive diagnosis can be made in a patient presenting clinically with polyarticular disease. General management principles for patients with polyarticular disease are also provided.

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29

Shirodaria, Cheerag, and Jim Newton. Cardiac infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0108.

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This chapter discusses endocarditis and acute rheumatic fever, including definitions of the disease, etiology, typical symptoms, uncommon symptoms, demographics, natural history, complications, diagnostic approaches, other diagnoses that should be considered, prognosis, and treatment.

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30

Hagendorff, Andreas, Elie Chammas, and Mohammed Rafique Essop. Diseases with a main influence on heart valves. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0058.

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The high spatial and temporal resolution, widespread availability, and non-invasive nature of echocardiography make it the imaging modality of choice for assessment of cardiac valvular disease. Echocardiography allows not only detailed evaluation of valve morphology, but also makes possible assessment of the haemodynamic consequences and impact on left and right ventricular size and function. Based on this data, a more informed decision may be made on the nature and timing of surgical or percutaneous intervention. A wide variety of diseases may afflict the cardiac valves. In some such as rheumatic heart disease and degenerative disease, abnormality of valve function is the most important manifestation. Many systemic diseases, however, may affect the cardiac valves and not infrequently, echocardiography may be the first clue to a systemic illness. The salient points of diseases affecting mainly the cardiac valves are discussed in this chapter.

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31

Foster, Brogan, and Paul A. Brogan. Infection and immunization. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198738756.003.0006.

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This chapter provides detailed reviews of the musculoskeletal manifestations of tuberculosis and mycobacterial disease; other bone and joint infections; infections in immunocompromised paediatric patients; and guidance for the workup of pyrexia of unknown origin. Updated guidance on the management of rheumatic fever and Lyme disease is provided, including detailed antibiotic regimens. Management algorithms for immunocompromised patients exposed to VZV are described, as well as treatment of VZV should it occur in an immunocompromised patient. It also provides detailed guidance on immunization schedules for the immunocompromised. A highlight of the second edition is a section on HIV, with emphasis on rheumatological manifestations and their management.

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32

Iung, Bernard. Epidemiology and physiopathology. Edited by Gilbert Habib. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0389.

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The incidence of infective endocarditis (IE) is estimated at between 15 and 60 cases per million inhabitants per year from population-based studies in industrialized countries. The presentation of IE has changed since patients are getting older and Staphylococcus is now becoming the microorganism most frequently responsible, which is partly attributable to healthcare-associated infections. The incidence of IE is higher in patients with heart valve prosthesis, previous endocarditis, and complex congenital heart disease. In developing countries, IE occurs in younger patients with a majority of rheumatic valve disease and is most frequently due to streptococci. IE is the consequence of bacteraemia on a diseased native valve or foreign material, leading to vegetation or tissue destruction, or both of these. The main consequences of these lesions are heart failure due to valvular regurgitation, embolic events due to vegetation migration, and perivalvular abscesses.

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33

Hagendorff, Andreas, and Laura Ernande. Diseases with a main influence on pericardium. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0059.

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The high spatial and temporal resolution, widespread availability, and non-invasive nature of echocardiography make it the imaging modality of choice for assessment of cardiac valvular disease. Echocardiography allows not only detailed evaluation of valve morphology, but also makes possible assessment of the haemodynamic consequences and impact on left and right ventricular size and function. Based on this data, a more informed decision may be made on the nature and timing of surgical or percutaneous intervention. A wide variety of diseases may afflict the cardiac valves. In some such as rheumatic heart disease and degenerative disease, abnormality of valve function is the most important manifestation. Many systemic diseases, however, may affect the cardiac valves and not infrequently, echocardiography may be the first clue to a systemic illness. The salient points of diseases affecting mainly the cardiac valves are discussed in this chapter.

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34

Deen, Jason F., and Karen K. Stout. Causes and diagnosis of valvular problems. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0158.

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While valvular heart disease encountered in developing countries is primarily rheumatic in aetiology, in industrialized countries it is largely comprised of degenerative valvular disease. Although less prevalent than ischaemic heart disease, its prevalence increases with older age and increased life expectancy, and therefore represents significant disease burden in aging populations. Transthoracic echocardiography remains the imaging modality of choice for timely delineation of the anatomy and severity of the lesion,although, once identified, may not correlate with symptoms due to clinical latency of disease onset to disease manifestation. Variations of disease severity, which may not meet criteria for intervention, lead to chronicity of disease, while clinically silent lesions may remain undiagnosed—both of these situations may lead to acute illness requiring intensive care management. Stabilization through medical intervention may be required, although many patients with severe disease will need emergent surgical repair, therefore collaborative involvement between intensivists, cardiologists, and cardiovascular surgeons is needed to minimize patient mortality and morbidity.

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35

Grossman, Jonah, Tanzila Shams, and Cathy Sila. Neurological Complications of Infective Endocarditis. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0167.

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Infective endocarditis is the fourth leading cause of life-threatening infections, accounting for 40,000 annual U.S. hospital admissions. Due to decline in rheumatic heart disease, a shift in causative organisms from viridans streptococci to S. aureus, Group D Streptococcus, and multidrug-resistant species has been observed. The spectrum of neurological complications ranges widely from cerebrovascular pathologies-including septic embolization, mycotic aneurysms, and intracerebral hemorrhages-to seizures, meningitis, cerebritis, and abscess. Transthoracic echocardiogram remains the standard for initial investigation whereas CT scans, MRI with DWI sequence, and cerebral angiograms are useful for exploring neurological complications. Antibiotic regimens, tailored to culprit organisms, should be initiated early after obtaining blood cultures and continued for 4 to 6 weeks. Antithrombotic treatment may pose increased risk for intracerebral hemorrhage, even in the absence of mycotic aneurysms (MA). Unruptured MA must be treated according to risk of rupture and overall health of the patient. MAs either at risk or previously ruptured should be secured by neurosurgical or endovascular means. Early cardiac surgery is a viable option for prevention of septic embolization for high-risk cardiac diseases such as perivalvular abscess and infection with resistant organisms, but may increase mortality rates for those with decompensated heart failure.

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