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1

Miller, Laurie C. "Rheumatic fever and rheumatic heart disease." Current Opinion in Rheumatology 1, no. 3 (1989): 257–61. http://dx.doi.org/10.1097/00002281-198901030-00003.

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2

WAALER, ERIK. "Rheumatic Fever and Rheumatic Heart Disease." Acta Medica Scandinavica 160, no. 4 (2009): 281–92. http://dx.doi.org/10.1111/j.0954-6820.1958.tb10354.x.

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3

WAALER, ERIK. "Rheumatic Fever and Rheumatic Heart Disease." Acta Medica Scandinavica 160, no. 4 (2009): 293–303. http://dx.doi.org/10.1111/j.0954-6820.1958.tb10355.x.

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4

Saggi, Manpreet, and Raman Kalia. "Rheumatic Fever and Rheumatic Heart Disease." Asian Journal of Nursing Education and Research 10, no. 3 (2020): 360. http://dx.doi.org/10.5958/2349-2996.2020.00076.2.

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5

Lawrence, Joanna G., Jonathan R. Carapetis, Kalinda Griffiths, Keith Edwards, and John R. Condon. "Acute Rheumatic Fever and Rheumatic Heart Disease." Circulation 128, no. 5 (2013): 492–501. http://dx.doi.org/10.1161/circulationaha.113.001477.

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6

Cilliers, Antoinette Myrna. "Rheumatic fever and rheumatic heart disease in Africa." South African Medical Journal 105, no. 5 (2015): 361. http://dx.doi.org/10.7196/samj.9433.

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7

KAWAKITA, seiichi. "Rheumatic fever and rheumatic heart disease in Japan." Japanese Circulation Journal 50, no. 12 (1986): 1241–45. http://dx.doi.org/10.1253/jcj.50.1241.

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8

Muhamed, Babu, Tom Parks, and Karen Sliwa. "Genetics of rheumatic fever and rheumatic heart disease." Nature Reviews Cardiology 17, no. 3 (2019): 145–54. http://dx.doi.org/10.1038/s41569-019-0258-2.

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9

Yacoub, Magdi, Bongani Mayosi, Ahmed ElGuindy, Alain Carpentier, and Salim Yusuf. "Eliminating acute rheumatic fever and rheumatic heart disease." Lancet 390, no. 10091 (2017): 212–13. http://dx.doi.org/10.1016/s0140-6736(17)31608-2.

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10

Khanal, Namrata. "PREVENTION OF RHEUMATIC HEART DISEASE AND RHEUMATIC FEVER." Journal of Nepal Medical Association 42, no. 145 (2003): 64. http://dx.doi.org/10.31729/jnma.793.

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Arvind, Balaji, and Sivasubramanian Ramakrishnan. "Rheumatic Fever and Rheumatic Heart Disease in Children." Indian Journal of Pediatrics 87, no. 4 (2020): 305–11. http://dx.doi.org/10.1007/s12098-019-03128-7.

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12

Groves, Alison M. M. "Rheumatic Fever and Rheumatic Heart Disease: An Overview." Tropical Doctor 29, no. 3 (1999): 129–32. http://dx.doi.org/10.1177/004947559902900301.

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13

Agarwal, Ajit K. "Rheumatic fever and rheumatic heart disease in Arabia." International Journal of Cardiology 43, no. 3 (1994): 229–30. http://dx.doi.org/10.1016/0167-5273(94)90200-3.

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14

Alam, Muhammad Badrul, Sania Hoque, Amiruzzaman Khan, Md Zakir Hossain, and Khondoker Asaduzzaman. "A Lady with Systemic Lupus Erythematosus and Mitral Stenosis." Bangladesh Heart Journal 31, no. 2 (2017): 109–12. http://dx.doi.org/10.3329/bhj.v31i2.32383.

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Mitral stenosisis a valvular heart disease caused by a number of diseases. Chronic rheumatic fever is the most important cause. Among rare causes, some rheumatoid diseases like SLE may involve cardiovascular system causing libman- sacks endocarditis,pericardial diseases and other valvular lesions mostly associated with positive antiphospholipid and anticardiolipin antibody.Here, we presented a case of rheumatic mitral valvular heart disease having systemic lupus erythromatosus but negative antiphospholipid and anticardiolipin antibody.Bangladesh Heart Journal 2016; 31(2) : 109-112
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15

Cleonice de Carvalho Coelho Mota and Zilda Maria Alves Meira. "Rheumatic fever." Cardiology in the Young 9, no. 3 (1999): 239–48. http://dx.doi.org/10.1017/s1047951100004893.

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AbstractIn recent years, rheumatic fever has been declining in the industrialized countries, and has became a rare disease. In developing areas, nonetheless, the sequels of its cardiac involvement have important implications from the stance of public health, and this disease is still the main cause of heart disease in children and young adults. From a historical perspective, the long-term prevention and the control of socioeconomic problems have been proven to be effective, and it is these measures which have contributed to the decline of the disease in developed countries. But, due to its present high prevalence around the world, and the potential for his resurgence, this disease remains an unsolved problem. Furthermore, the complete understanding of its pathophysiology remains a challenge. In this presentation, we will discuss our experience with epidemiological, clinical and prophylatic aspects of this enigmatic disease, and the strategies we have developed to study and control heumatic fever in Minas Gerais, Brazil.
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16

Guilherme, Luiza, Kellen Faé, Sandra E. Oshiro, and Jorge Kalil. "Molecular pathogenesis of rheumatic fever and rheumatic heart disease." Expert Reviews in Molecular Medicine 7, no. 28 (2005): 1–15. http://dx.doi.org/10.1017/s146239940501015x.

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Molecular mimicry between streptococcal and human proteins has been proposed as the triggering factor leading to autoimmunity in rheumatic fever (RF) and rheumatic heart disease (RHD). This article summarises studies on genetic susceptibility markers involved in the development of RF/RHD. It also focuses on the molecular mimicry in RHD mediated by the responses of B and T cells of peripheral blood, and T cells infiltrating heart lesions, against streptococcal antigens and human tissue proteins. The molecular basis of T-cell recognition is assessed through the definition of heart-crossreactive antigens. The production of cytokines from peripheral and heart-infiltrating mononuclear cells suggests that T helper 1 (Th1)-type cytokines are the mediators of RHD heart lesions. An insufficiency of interleukin 4 (IL-4)-producing cells in the valvular tissue might contribute to the maintenance and progression of valve lesions.
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17

Hajar, Rachel. "Rheumatic fever and rheumatic heart disease a historical perspective." Heart Views 17, no. 3 (2016): 120. http://dx.doi.org/10.4103/1995-705x.192572.

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18

Guilherme, L., R. Ramasawmy, and J. Kalil. "Rheumatic Fever and Rheumatic Heart Disease: Genetics and Pathogenesis." Scandinavian Journal of Immunology 66, no. 2-3 (2007): 199–207. http://dx.doi.org/10.1111/j.1365-3083.2007.01974.x.

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19

Muhamed, Babu, Gasnat Shaboodien, and Mark E. Engel. "Genetic variants in rheumatic fever and rheumatic heart disease." American Journal of Medical Genetics Part C: Seminars in Medical Genetics 184, no. 1 (2020): 159–77. http://dx.doi.org/10.1002/ajmg.c.31773.

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20

Catarino, Sandra Jeremias, Fabiana Antunes Andrade, Lorena Bavia, Luiza Guilherme, and Iara Jose Messias-Reason. "Ficolin-3 in rheumatic fever and rheumatic heart disease." Immunology Letters 229 (January 2021): 27–31. http://dx.doi.org/10.1016/j.imlet.2020.11.006.

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21

Prajapati, Dipanker, Deewakar Sharma, Prakash Raj Regmi, et al. "Epidemiological survey of Rheumatic fever, Rheumatic heart disease and Congenital heart disease among school children in Kathmandu valley of Nepal." Nepalese Heart Journal 10, no. 1 (2014): 1–5. http://dx.doi.org/10.3126/njh.v10i1.9738.

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Background and Aims: Rheumatic Fever, Rheumatic Heart Disease and Congenital Heart disease are the most common cardiac problems in school children. Prior studies have reported different prevalence rate of Rheumatic Heart Disease among different groups of population of Nepal. The aim of this study was to estimate the prevalence of Rheumatic Fever, Rheumatic Heart Disease and Congenital Heart Disease among school children in Kathmandu Valley of Nepal. Methods: Cardiac screening of 34,876 school children from 115 randomly selected public schools from two cities of Kathmandu Valley (Kathmandu and Lalitpur) was done. Cases with abnormal findings in auscultation underwent echocardiography and the diagnosis was confirmed. Results: The prevalence of Congenital Heart Disease was noted to be 1 per thousand and prevalence of Rheumatic Heart Disease was found to be 0.90 per thousand (in the age group 5-16 years) with the most common lesion being Mitral Regurgitation. No significant statistical difference was noted between male and female students in both the cases of Rheumatic Heart Disease and Congenital Heart Disease. No cases of Acute Rheumatic Fever were noted. Conclusion: The prevalence of Rheumatic Heart Disease among school children in Kathmandu valley was noted to be lower than reported in similar previous studies. Primary and secondary prevention programs of RF/RHD have been effective in Nepal and are needed to be strengthened and expanded to further reduce the burden of these diseases. Nepalese Heart Journal | Volume 10 | No.1 | November 2013| Pages 1-5 DOI: http://dx.doi.org/10.3126/njh.v10i1.9738
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22

Hasab, Aly A., Ali Jaffer, and Abdulla M. Riyami. "Rheumatic heart disease among Omani schoolchildren." Eastern Mediterranean Health Journal 3, no. 1 (1997): 17–23. http://dx.doi.org/10.26719/1997.3.1.17.

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Screening of 9904 Omani schoolchildren from different regions in Oman gave a prevalence rate of rheumatic heart disease of 8 per 10 000 with no significant difference by sex or level of education. Follow-up of the sample for three months gave an estimated annual incidence of rheumatic fever of 4 per 10 000. The positive predictive value of definite murmurs for diagnosis of cardiovascular disease was 35.21% for school health physicians and 86.67% for regional physicians. The results show that rheumatic fever and rheumatic heart disease are not major public health problems in Oman. The study recommends integration of the management and control of the diseases within the primary health care system
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23

PADMAVATI, S. "Rheumatic heart disease: prevalence and preventive measures in the Indian subcontinentKeywords: rheumatic heart disease; rheumatic fever." Heart 86, no. 2 (2001): 127. http://dx.doi.org/10.1136/hrt.86.2.127.

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24

Goswami, Ravinder, Pankaj Shah, C. S. Bal, Balbir Singh, A. C. Ammini, and K. K. Talwar. "Thyrotoxicosis, rheumatic heart disease and fever." International Journal of Cardiology 47, no. 1 (1994): 31–35. http://dx.doi.org/10.1016/0167-5273(94)90130-9.

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25

Rahman, Mohammad Arifur, Afzalur Rahman, Syed Nasir Uddin, et al. "Rheumatic Fever and Rheumatic Heart Diseases in Bangladesh: Challenges and Remedies." Cardiovascular Journal 10, no. 2 (2018): 206–11. http://dx.doi.org/10.3329/cardio.v10i2.36294.

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In the 21st century, Rheumatic fever (RF) and Rheumatic heart disease (RHD) are neglected diseases of marginalized communities. Globally, RHD remains the most-common cardiovascular disease in young people aged <25 years. Although RF and RHD have been almost eradicated in areas with established economies, migration from low-income to high-income settings might be responsible for a new burden of RHD in high-income countries. Globally, the prevalence of rheumatic fever (RF) and rheumatic heart disease (RHD) has declined sharply but, in developing countries, RF is still aleading cause of heart disease and, consequently, death in children and young adults. In 2005, it was estimated that over 2.4 million children aged 5-14 years were having RHD globally and 79% of all these cases were from lessdeveloped countries.Cardiovasc. j. 2018; 10(2): 206-211
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26

Islam, AKM Monwarul, and HI Lutfur Rahman Khan. "Diagnosis of Rheumatic Fever and Rheumatic Heart Disease in Evolution." Bangladesh Heart Journal 30, no. 1 (2016): 1–4. http://dx.doi.org/10.3329/bhj.v30i1.28125.

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27

Neupane, Ganesh Prasad, R. Makaju, and R. Koju. "Rheumatic Fever and Antibody Response to Group A Streptococcal Infections." Nepalese Heart Journal 6, no. 1 (2017): 58–61. http://dx.doi.org/10.3126/njh.v6i1.18597.

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Rheumatic fever is non-suppurative sequel of group A streptococcal infection. It is a multifocal inflammatory disease, affecting primarily the heart, joints, skin and central nervous system occurring in 0.1% to 3% after untreated pharyngitis. It was a major cause of death and a common cause of chronic structural heart disease in children until 1960. It has declined in developed countries due to advent of penicillin and improved social conditions. Rheumatic fever is very common disease among children in developing countries till date. Rheumatic fever and its clinically significant sequel, rheumatic heart disease, continue to be a major health problem in developing countries like Nepal. Rheumatic fever is still a major cause of death and heart disease.
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28

Millard-Bullock, D. "The Rheumatic Fever and Rheumatic Heart Disease Control Programme – Jamaica." West Indian Medical Journal 61, no. 4 (2012): 361–64. http://dx.doi.org/10.7727/wimj.2012.134.

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29

Carapetis, JonathanR, and LieslJ Zühlke. "Global research priorities in rheumatic fever and rheumatic heart disease." Annals of Pediatric Cardiology 4, no. 1 (2011): 4. http://dx.doi.org/10.4103/0974-2069.79616.

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30

Steer, Andrew C., and Jonathan R. Carapetis. "Acute Rheumatic Fever and Rheumatic Heart Disease in Indigenous Populations." Pediatric Clinics of North America 56, no. 6 (2009): 1401–19. http://dx.doi.org/10.1016/j.pcl.2009.09.011.

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31

Al-Munibari, Abdul Nasser, Thabet Mohsen Nasher, Siddig Ahmed Ismail, and El-Daw Ahmed Mukhtar. "Prevalence of Rheumatic Fever and Rheumatic Heart Disease in Yemen." Asian Cardiovascular and Thoracic Annals 9, no. 1 (2001): 41–44. http://dx.doi.org/10.1177/021849230100900111.

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32

Essop, Mohammed R., and Ferande Peters. "Contemporary Issues in Rheumatic Fever and Chronic Rheumatic Heart Disease." Circulation 130, no. 24 (2014): 2181–88. http://dx.doi.org/10.1161/circulationaha.114.009857.

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33

Bhatia, R., J. Narula, K. S. Reddy, et al. "Lymphocyte subsets in acute rheumatic fever and rheumatic heart disease." Clinical Cardiology 12, no. 1 (1989): 34–38. http://dx.doi.org/10.1002/clc.4960120106.

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34

Islam, A. K. M. Monwarul, and A. A. S. Majumder. "Rheumatic fever and rheumatic heart disease in Bangladesh: A review." Indian Heart Journal 68, no. 1 (2016): 88–98. http://dx.doi.org/10.1016/j.ihj.2015.07.039.

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35

Sanderson, J. E., and K. S. Woo. "Rheumatic fever and rheumatic heart disease — declining but not gone." International Journal of Cardiology 43, no. 3 (1994): 231–32. http://dx.doi.org/10.1016/0167-5273(94)90201-1.

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36

Medeiros, Caio César Jorge, Alvaro Villela de Moraes, Rachel Snitcowsky, et al. "Echocardiographic diagnosis of rheumatic fever and rheumatic valvar disease." Cardiology in the Young 2, no. 3 (1992): 236–39. http://dx.doi.org/10.1017/s1047951100000962.

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SummaryWe studied 56 children echocardiographically, ages four to 15 years (mean nine years) with acute rheumatic fever to determine the extent of cardiac involvement. All had a clinical diagnosis of carditis. Mitral regurgitation was always present and was associated with aortic regurgitation in 53.6% and tricuspid regurgitation in 32.1%. Mitral valvar prolapse was observed in 18 patients 5(32.1%) and could not be differentiated from myxomatous prolapse in 10 (17.8%). Cordal rupture was detected in seven patients (12.5%), three of whom required surgical treatment. Other findings concerning the mitral valve were vegetations in two patients (3.6%), commissural fusion without stenosis in 36(64.3%), and mitral stenosis in four (7.1%). Heart failure was usually secondary to valvitis rather than myocarditis, and led to surgical treatment in seven patients. The myocardial function was depressed in only two patients. In both, the ejection function returned to normal after medical treatment. Echocardiography provides important information on the involvement of the heart in acute rheumatic fever, helping to determine prognosis and the results of treatment.
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37

Santalova, G. V., P. A. Lebedev, A. A. Garanin, and M. E. Kuzin. "Problems of chronic rheumatic heart disease diagnosis at the present stage." Clinical Medicine (Russian Journal) 99, no. 4 (2021): 259–65. http://dx.doi.org/10.30629/0023-2149-2021-99-4-259-265.

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The review refl ects modern data on the epidemiology of acute rheumatic fever and chronic rheumatic heart disease in Russia and the world at present, as well as the dynamics of the prevalence of these diseases over the past decades. Much attention is paid to the issues of modern diagnostics of these conditions by physical, laboratory and instrumental methods. The focus is on the Jones criteria in the diagnosis of acute rheumatic fever in accordance with their revision by the American Heart Association experts in 2015. Taking into account the fact that damage to the valvular apparatus of the heart in acute rheumatic fever is the main disabling outcome of carditis at the present stage, a special place in the article is devoted to the discussion of echocardiographic criteria for valvulitis. The recommendations of the International Expert Council of the World Heart Federation aimed at detecting chronic rheumatic heart disease in patients without a history of acute rheumatic fever diagnosed by ultrasound imaging are also given. Criteria for pathological aortic and mitral regurgitation are presented. The authors believe that extrapolation of modern principles of ultrasound diagnostics of chronic rheumatic heart disease in Russia and their use as screening programs in young people and adolescents will contribute to its early detection and timely selection of patients for secondary prevention of benzathine with benzylpenicillin.
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38

Ketheesan, Natkunam. "Update on rheumatic fever – new insights into the pathogenesis of rheumatic fever and rheumatic heart disease." Pathology 49 (February 2017): S53. http://dx.doi.org/10.1016/j.pathol.2016.12.130.

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39

Karataş, Zehra, Tamer Baysal, Fatih Şap, Hayrullah Alp, and Idris Mehmetoğlu. "Increased ischaemia-modified albumin is associated with inflammation in acute rheumatic fever." Cardiology in the Young 24, no. 3 (2013): 430–36. http://dx.doi.org/10.1017/s1047951113000516.

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AbstractIntroduction: Ischaemia-modified albumin, a novel biochemical marker for tissue ischaemia, was found to be associated with oxidative stress. The purpose of this study was to assess the role of ischaemia-modified albumin in the diagnosis of acute rheumatic fever and also to evaluate the ischaemia-modified albumin levels in children with heart valve disease. Methods: The study groups, aged 5–18 years, consisted of 128 individuals – 40 with acute rheumatic fever, 35 with congenital heart valve disease, 33 with chronic rheumatic heart disease, and 20 healthy control subjects. Results: The ischaemia-modified albumin, erythrocyte sedimentation rate, and C-reactive protein levels of the acute rheumatic fever group were significantly higher than those in the chronic rheumatic heart disease, congenital heart valve disease, and control groups, separately (p < 0.001). The ischaemia-modified albumin levels in both carditis and isolated arthritis subgroups of children with acute rheumatic fever were significantly higher than in the control group (p < 0.001, p < 0.01, respectively). However, there was no statistically significant difference between the chorea subgroup and control subjects. In addition, significant correlations were observed between ischaemia-modified albumin and acute phase reactants of patients with acute rheumatic fever (p < 0.001 for both erythrocyte sedimentation rate and C-reactive protein). The ischaemia-modified albumin levels of chronic rheumatic heart disease, congenital heart valve disease, and control subjects were similar. Conclusions: The increased level of ischaemia-modified albumin in children with acute rheumatic fever seems to be associated with inflammation. However, further studies are needed to provide stronger evidence.
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40

Shrestha, PN, A. Das, A. Rayamajhi, C. Mahaseth, and UK Shrestha. "Rheumatic fever and rheumatic heart disease: how often we suspect infective endocarditis." Journal of Institute of Medicine Nepal 34, no. 3 (2013): 17–20. http://dx.doi.org/10.3126/jiom.v34i3.8911.

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Introduction: Rheumatic fever and rheumatic heart disease is a common problem in developing countries. Rheumatic valvular heart disease is one of the important risk factor for infective endocarditis. Methods: Retrospective study was conducted among 56 children admitted in cardiology ward of Kanti children hospital with the diagnosis of rheumatic fever or rheumatic heart disease during May 2008 to May 2010. Results: We found male to female ratio 2: 1, median age 12 (5-14yrs) with the most affected age group of 11-12 years. Common symptoms were fever (66%), dyspnea (68%), joint pain (50%), palpitation (30%), cough (16%) and chest pain (12%). Only 9% of patients had positive history of sore throat. Sixty percent patients were presented with features of congestive cardiac failure (CCF). A SO positive was found in 25 (44.6%) cases. The most common valvular lesion was mitral regurgitation (MR). Moderate to severe mitral regurgitation was found in 89% of cases. Tricuspid regurgitation (T R) was found in 57% of patients and aortic regurgitation (AR) in 55% of cases. Thirty-two patients (57%} had PAH in which 8 patients has severe PAH and 24 patients had mild to moderate PAH. Infective endocarditis was diagnosed in twenty patients (35.7%). Out of 20 patients 14 (70%)had vegetation in echocardiography. The mortality was 11% in this study. Conclusion: Infective endocarditis is the major complication of rheumatic heart disease among children of Nepal and so clinical suspecian is important whenever patients with rheumatic heart disease present with fever. DOI: http://dx.doi.org/10.3126/joim.v34i3.8911 Journal of Institute of Medicine, December, 2012; 34:17-20
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41

Watson, Gabriella, Bintou Jallow, Kirsty Le Doare, Kuberan Pushparajah, and Suzanne T. Anderson. "Acute rheumatic fever and rheumatic heart disease in resource-limited settings." Archives of Disease in Childhood 100, no. 4 (2015): 370–75. http://dx.doi.org/10.1136/archdischild-2014-307938.

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Poststreptococcal complications, such as acute rheumatic fever (ARF) and rheumatic heart disease (RHD), are common in resource-limited settings, with RHD recognised as the most common cause of paediatric heart disease worldwide. Managing these conditions in resource-limited settings can be challenging. We review the investigation and treatment options for ARF and RHD and, most importantly, prevention methods in an African setting.
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42

Ahmed, Jasimuddin, M. Mostafa Zaman, and M. M. Monzur Hassan. "Prevalence of rheumatic fever and rheumatic heart disease in rural Bangladesh." Tropical Doctor 35, no. 3 (2005): 160–61. http://dx.doi.org/10.1258/0049475054620879.

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A community-based study was done on 5923 rural Bangladeshi children aged 5-15 years to determine the prevalence of rheumatic fever (RF) and rheumatic heart disease (RHD). The prevalence was found to be 1.2 (95% confidence interval 0.3-2.1) per 1000 for RF defined by revised Jones criteria and 1.3 (0.4-2.2) per 1000 for Doppler echocardiography-confirmed RHD.
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43

Hirachan, Anish, Reeju Manandhar, Madhu Roka, and Deewakar Sharma. "Acute Rheumatic Activity in a 49 year old Nepalese adult with established Rheumatic heart Disease: a case report." Nepalese Heart Journal 13, no. 2 (2016): 39–40. http://dx.doi.org/10.3126/njh.v13i2.15564.

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Rheumatic fever presenting late in adult beyond 25 years of age is a rare but common phenomena and noted in various case reports .The diagnosis of rheumatic activity is based on the same modified Jones criteria for rheumatic fever and rheumatic heart disease. Here we describe a 49 year old male , known rheumatic heart disease with severe aortic regurgitation and post aortic valve replacement who presented with fever and migratory polyarthritis along with history of preceding sore throat 2 weeks prior to this illness . He was managed with high dose of aspirin therapy along with oral penicillin after which he had dramatic improvement in his symptomatology and was discharged with good recovery.Nepalese Heart Journal 2016; 13(2): 39-40
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44

WATANABE, NOBUO, and YUKIYOSHI NAKAMURA. "Estimation of antistreptococcal esterase in rheumatic fever and rheumatic heart disease." Japanese Circulation Journal 49, no. 12 (1985): 1262–64. http://dx.doi.org/10.1253/jcj.49.1262.

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45

Abella-Reloza, Asuncion. "Rheumatic fever and rheumatic heart disease in Asia: a global concern." Progress in Pediatric Cardiology 9, no. 1 (1998): 53–54. http://dx.doi.org/10.1016/s1058-9813(98)00038-1.

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46

VEASY, L. GEORGE, and HARRY R. HILL. "Immunologic and clinical correlations in rheumatic fever and rheumatic heart disease." Pediatric Infectious Disease Journal 16, no. 4 (1997): 400–407. http://dx.doi.org/10.1097/00006454-199704000-00012.

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47

de Dassel, Jessica L., Anna P. Ralph, and Jonathan R. Carapetis. "Controlling acute rheumatic fever and rheumatic heart disease in developing countries." Current Opinion in Pediatrics 27, no. 1 (2015): 116–23. http://dx.doi.org/10.1097/mop.0000000000000164.

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48

He, Vincent Y. F., John R. Condon, Anna P. Ralph, et al. "Long-Term Outcomes From Acute Rheumatic Fever and Rheumatic Heart Disease." Circulation 134, no. 3 (2016): 222–32. http://dx.doi.org/10.1161/circulationaha.115.020966.

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49

ERTUG, M. H. "HLA -- DR Antigens in Rheumatic Fever and Rheumatic Heart Disease Patients." Rheumatology 32, no. 4 (1993): 347–48. http://dx.doi.org/10.1093/rheumatology/32.4.347.

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Reddy, K. S., Jagat Narula, Ravi Bhatia, et al. "Immunologic and immunogenetic studies in rheumatic fever and rheumatic heart disease." Indian Journal of Pediatrics 57, no. 5 (1990): 693–700. http://dx.doi.org/10.1007/bf02728716.

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