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Books on the topic 'Rheumatoid Arthritis (ra)'

Author: Grafiati

Published: 4 June 2021

Last updated: 26 July 2025

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1

Alan, Sarokhan, ed. Understanding rheumatoid arthritis (RA) ; and, Understanding osteoarthritis (OA). T. Peters, 1996.

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2

Porges, Geoff. Biotechnology: Biologicals for RA - Amgen vs. Johnson & Johnson, again! Bernstein Research, 2004.

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3

Griffiths, Bridget. Rheumatoid arthritis in the two most prevalent racial groups living in the UK: A clinical serological, radiological and genetic comparison of North Indian and Pakistani RA patients with Northern European RA patients. University of Birmingham, 1999.

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4

Miller, Aaron E., and Teresa M. DeAngelis. Rheumatoid Arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199732920.003.0012.

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Rheumatoid arthritis (RA) is a systemic inflammatory disease that is characterized principally by a polyarthritis, but can result in several neurologic complications involving both the central and peripheral nervous system. In addition, several immunotherapies used to treat RA have been associated with neurological complications. In this chapter, we review the characteristic neurological sequelae of RA as well as the possible neurological consequences of its therapeutic regimens.

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5

Bawa, Sandeep, and Paul Wordsworth. Rheumatoid arthritis. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.010002.

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♦ Rheumatoid arthritis (RA) is one of the most common disabling chronic diseases of the Western world.♦ New biologic treatments have provided great benefit in the management of this condition, reducing morbidity and mortality.♦ RA diagnosis requires careful clinical, biochemical, and radiological assessment♦ Rheumatoid factor is not a reliable diagnostic test for rheumatoid arthritis (RA)♦ The core of its management requires careful input from all members of the multidisciplinary team.

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6

Aletaha, Daniel, and Helga Radner. Rheumatoid arthritis—diagnosis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0110.

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Rheumatoid arthritis (RA) is among the most disabling form of chronic inflammatory joint disease. Not all forms of arthritis develop into RA; on the contrary, it may be very challenging to differentiate RA from cases of arthritis that are self-limiting or caused by another disease. Evaluation of early arthritis includes some basic steps, such as excluding trauma, crystal, or infectious-related disease, as well as considering additional features that may guide towards a specific diagnosis. If no specific diagnosis can then be made, the presentation can be labelled as undifferentiated arthritis.

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7

Deighton, Chris. Rheumatoid arthritis—management. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0112.

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Influential guidelines on rheumatoid arthritis (RA) management agree on most key recommendations. Early diagnosis of persistent synovitis, and identification of poor prognostic markers, is essential. Rapid intervention is vital with drugs to suppress inflammation, slow down damaging disease components, and prevent disability. The label of RA covers a broad spectrum of disease severity, and there is controversy on: • whether the same interventions are needed for all patients • whether monotherapy or combination treatment is appropriate • the role of steroids in RA • the appropriate introduction

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8

Eyre, Steve, and Jane Worthington. Genetics of rheumatoid arthritis. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0040.

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A range of epidemiological studies have clearly established that susceptibility to rheumatoid arthritis (RA) is determined by both genetic and environmental factors. Studies over the last five decades have used a variety of approaches to identify the genetic variants associated with disease. HLA DRB1 was the first RA susceptibility locus to be discovered and has the largest effect size. We describe current understanding of the complexities of HLA association for RA. Linkage and small-scale association studies prior to 2007 provided convincing evidence for only one more RA susceptibility locus,

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9

Eyre, Steve, Jane Worthington, and Sebastien Viatte. Genetics of rheumatoid arthritis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0040_update_003.

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A range of epidemiological studies have clearly established that susceptibility to rheumatoid arthritis (RA) is determined by both genetic and environmental factors. Studies over the last five decades have used a variety of approaches to identify the genetic variants associated with disease. HLA DRB1 was the first RA susceptibility locus to be discovered and has the largest effect size. We describe current understanding of the complexities of HLA association for RA. Linkage and small-scale association studies prior to 2007 provided convincing evidence for only one more RA susceptibility locus,

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10

Feist, Eugen, and Gerd-R. Burmester. Rheumatoid arthritis—clinical features. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0111.

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Rheumatoid arthritis (RA) presents with variable clinical features, making this most frequent chronic systemic autoimmune disease with characteristic joint involvement a diagnostic and therapeutic challenge. This chapter describes in detail the different clinical, laboratory and imaging findings in patients with RA. In addition to the characteristic arthritic involvement, which can lead to severe joint changes with progressive destruction and loss of function, other systemic disease manifestations as well as an increased risk for cardiovascular events and non-Hodgkin's lymphoma with relevance

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11

Clark, David. Rheumatoid arthritis of the elbow. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.05.04.

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♦ 50% of RA patients have elbow involvement♦ Females affected 3 times more than males♦ Peak incidence 60-70 years of age♦ Radiological severity assessed using the Larson radiological grading system♦ No single test used to diagnose RA♦ The management of RA requires a multidisciplinary approach♦ Anti-TNF drugs are used when disease-modifying agent combinations have failed to control symptoms♦ Intra-articular and intramuscular cortisone is an effective way of controlling fl are-ups♦ Total elbow arthroplasty is indicated in severe RA where there is failure of medical management to control symptoms

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12

Ra Rheumatoid Arthritis. Managing Rheumatoid Arthritis. How to Effectively Cope with Rheumatoid Arthritis: Pain Relief, Treatment, Diet and Remedies. . IMB Publishing, 2015.

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13

Rheumatoid Arthritis Ra. Managing Rheumatoid Arthritis. How to Effectively Cope with Rheumatoid Arthritis: Pain Relief, Treatment, Diet and Remedies. IMB Publishing, 2014.

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14

Scott, David L., James Galloway, Andrew Cope, Arthur Pratt, and Vibeke Strand, eds. Oxford Textbook of Rheumatoid Arthritis. Oxford University Press, 2020. http://dx.doi.org/10.1093/med/9780198831433.001.0001.

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This new textbook provides a comprehensive overview of the scientific and clinical aspects of rheumatoid arthritis (RA). Split into eight sections—history, diagnosis, and epidemiology; pathogenesis; clinical presentation; disease assessment; impact on life; non-drug treatments; drug treatments; and management and outcomes—it collects the contemporary ideas about RA and explains the revolutionary changes that have taken place over the past two decades, and indicates areas of future research. Witten by leading clinicians and scientists in the field, each chapter gives a detailed background, key

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15

Back2lifeagain, Back2lifeagain Back2life. Rheumatoid Arthritis Treatment: Or How I Overcame RA Without Pills. Independently Published, 2019.

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16

Aggarwal, Monica, and Jyothi Rao. Reversing Rheumatoid Arthritis: A Drug-Free RA Treatment That Works. Square One Publishers, 2019.

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17

Stothard, J. Rheumatoid arthritis of the hand and wrist. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.006005.

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♦ RA is common♦ Medical treatment is the mainstay and newer anti-TNF drugs are reducing morbidity and thus referral for surgery♦ Assessment is primarily clinical♦ Investigations – Primarily radiographs♦ Treatment• Non-operative• Steroid injections are often very useful• Operative• Site and condition specific♦ In general• DRUJ – excision ulna head• Wrist – partial fusion, arthrodesis, arthroplasty• MP joints – synvestomy, arthroplasty• PIP joints – soft tissue rebalancing, arthrodesis• DIP joints – arthrodesis• Thumb MP and IP joints – arthrodesis

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18

Scammell, Henry. The New Arthritis Breakthrough: The Only Medical Therapy Clinically Proven to Produce Long-term Improvement and Remission of RA, Lupus, Juvenile RS, Fibromyalgia, ... & Other Inflammatory Forms of Arthritis. M. Evans and Company, Inc., 1998.

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19

K. Gautam, Rupesh, Lokesh Deb, and Kamal Dua, eds. Natural Products for the Management of Arthritic Disorders. BENTHAM SCIENCE PUBLISHERS, 2022. http://dx.doi.org/10.2174/97898150507761220101.

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Rheumatoid arthritis (RA) is the most common inflammatory complication and affects approximately 1 % of the global population. It affects three times more women than men. RA is an autoimmune disorder elicited by exposure of genetic factors from the host to unknown antigens causing arthritogenic complaints. It also includes the activation of lymphocytes as well as CD4+ helper T cells along with local release of chronic inflammatory mediators and cytokines like tumor necrosis factor (TNF α) and various cytokines like interleukins (IL) that enormously affect the joints. The available allopathic t

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20

Sanders, Allan. Rheumatoid Arthritis Cookbook: Dietary Guide for Managing RA with Anti-Inflammatory Recipes to Fight Flares and Fatigue. Independently Published, 2021.

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21

Merkesdal, Sonja, and Wilfried Mau. Health economics. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0031.

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The economic burden of rheumatic diseases for society, various payers, and last but not least the individual patient has been increasingly recognized. In addition to the well-known impact of back pain and osteoarthritis, the upcoming new and expensive therapies have made this issue especially intriguing in the treatment of rheumatoid arthritis (RA). A mean international estimate of the total annual costs of RA, mainly consisting of direct resource consumption and indirect costs due to productivity losses relating to paid work, comes to about €5600. Other inflammatory rheumatologic diseases (an

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22

Wiffen, Philip, Marc Mitchell, Melanie Snelling, and Nicola Stoner. Therapy-related issues: musculoskeletal diseases. Oxford University Press, 2012. http://dx.doi.org/10.1093/med/9780199603640.003.0025.

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Rheumatoid arthritis 548Gout 556Rheumatoid arthritis (RA) is an autoimmune disease which causes joints lined with synovium to become inflamed, swollen, stiff, and painful, and leads to joint erosion. It is a multisystem disorder which can affect many organs including the eyes, lungs, heart, and blood vessels. The aim of treatment is to decrease pain and inflammation, prevent joint damage, and ultimately induce remission of disease....

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23

Klingenberg, Roland, and Ulf Müller-Ladner. Mechanisms of inflammation. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198784906.003.0270.

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This chapter provides a brief summary of the immune pathogenesis of atherosclerosis, highlighting shared features with inflammatory pathways in rheumatoid arthritis (RA) described in detail in Chapter 25.4. RA constitutes a prototype autoimmune disease primarily affecting the joints but also the heart and vessels associated with increased cardiovascular mortality. Recent years have produced a wealth of novel insights into the diversity of immune cell types which either propagate or dampen inflammation in atherogenesis. Expansion of this inherent anti-inflammatory component carried by regulator

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24

Dörner, Thomas, and Peter E. Lipsky. Cellular side of acquired immunity (B cells). Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0050.

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B cells have gained interest in rheumatoid arthritis (RA) beyond being the precursors of antibody-producing plasma cells since they are also a broader component of the adaptive immune system. They are capable of functioning as antigen-presenting cells for T-cell activation and can produce an array of cytokines. Disturbances of peripheral B-cell homeostasis together with the formation of ectopic lymphoid neogenesis within the inflamed synovium appears to be a characteristic of patients with RA. Enhanced generation of memory B cells and autoreactive plasma cells producing IgM-RF and ACPA-IgG ant

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25

Strand, Vibeke, Jeremy Sokolove, and Alvina D. Chu. Design of clinical trials in rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0030.

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Development of new therapies for rheumatic diseases requires a series of randomized controlled trials (RCTs) progressing from phase 1, 'first-in-human' to generate initial safety, pharmacokinetic (PK) and pharmacodynamic (PD) data; to phase 2, proof of concept for efficacy with safety and PK/PD data; and phase 3, designed to demonstrate definitive efficacy and safety to support regulatory approval. Important aspects of RCT designs include sample size estimations, treatment allocation, rescue, blinding, and statistical analyses of prespecified endpoints to preserve trial integrity. Over the pas

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26

Strand, Vibeke, Jeremy Sokolove, and Alvina D. Chu. Design of clinical trials in rheumatology. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199642489.003.0030_update_001.

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Development of new therapies for rheumatic diseases requires a series of randomized controlled trials (RCTs) progressing from phase 1, ’first-in-human’ to generate initial safety, pharmacokinetic (PK) and pharmacodynamic (PD) data; to phase 2, proof of concept for efficacy with safety and PK/PD data; and phase 3, designed to demonstrate definitive efficacy and safety to support regulatory approval. Important aspects of RCT designs include sample size estimations, treatment allocation, rescue, blinding, and statistical analyses of prespecified endpoints to preserve trial integrity. Over the pas

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27

Strand, Vibeke, Jeremy Sokolove, and Alvina D. Chu. Design of clinical trials in rheumatology. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199642489.003.0030_update_002.

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Development of new therapies for rheumatic diseases requires a series of randomized controlled trials (RCTs) progressing from phase 1, ’first-in-human’ to generate initial safety, pharmacokinetic (PK) and pharmacodynamic (PD) data; to phase 2, proof of concept for efficacy with safety and PK/PD data; and phase 3, designed to demonstrate definitive efficacy and safety to support regulatory approval. Important aspects of RCT designs include sample size estimations, treatment allocation, rescue, blinding, and statistical analyses of prespecified endpoints to preserve trial integrity. Over the pas

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28

Plants for joints: A plant-based diet, physical activity, and stress management for rheumatic diseases. eigen beheer, 2024.

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29

Pitzalis, Costantino, Frances Humby, and Michael P. Seed. Synovial pathology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0052.

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Synovial pathology is seen in a variety of disease states, including rheumatoid arthritis (RA), osteoarthritis (OA), psoriatic arthritis, and systemic lupus erythmatosus (SLE). This chapter highlights recent advances that characterize the cellular composition of these tissues according to surface markers and chemokine and cytokine expression, and describes synovial functional status and response to therapeutics. In RA, after initiation, pannus migrates over and under cartilage, and into subchondral bone, in a destructive process. Cartilage-pannus junction (CPJ) is characterized as invasive or

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30

Isaacs, John D., and Philip M. Brown. Rituximab and abatacept. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0083.

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Two biologics that target cells have been licensed to treat rheumatoid arthritis (RA). Rituximab is a chimeric monoclonal antibody (mAb) against CD20 that depletes B cells; abatacept is a soluble form of CTLA-4 that blocks costimulation and interferes with T-cell function. Both drugs alleviate signs and symptoms of RA and have been shown to retard radiographic progression. Rituximab is licensed for use following failure of tumour necrosis factor (TNF) blockade whereas abatacept's licence permits it use as a first-line biologic. In the United Kingdom, however, the National Institute for Health

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31

Malaviya, Anand, and Ashok Kumar. Axial spondyloarthritis in India. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198734444.003.0030.

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Spondyloarthritis (SpA) is among the commonest rheumatological conditions in India, second only to rheumatoid arthritis (RA). This is consistent with the fact that the prevalence of SpA is linked to the prevalence of HLA-B27 in that population. Its prevalence in different regions of the country has been reported to be approximately 6%. That would put India prominently on the SpA map of the world, reflected in the large number of publications from the late 1970s onwards, dispelling any earlier misconceptions that it is largely a ‘white man’s disease’. This chapter summarizes the various aspects

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32

cronley, shane. RA Warrior: One Man's Fight and Struggle Against Rheumatoid Arthritis, Spinal Atrophy, Osteoporosis and Degenerative Disc Disease While Maintaining a Family and Working As a Firefighter/EMT. Independently Published, 2018.

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33

Fleischmann, Roy. Signalling pathway inhibitors. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0081.

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Oral, small-molecule signalling pathway inhibitors, including ones that inhibit the JAK and SyK pathways, are currently in development for the treatment of rheumatoid arthritis (RA). Tofacitinib is an orally administered small-molecule inhibitor that targets the intracellular Janus kinase 3 and 1 (JAK1/3) molecules to a greater extent than JAK2 while baricitinib (formerly INCB028050) predominantly inhibits JAK1/2. Many of the proinflammatory cytokines implicated in the pathogenesis of RA utilize cell signalling that involves the JAK-STAT pathways and therefore inhibition of JAK-STAT signalling

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34

Hoyles, Rachel K., and Athol U. Wells. Respiratory system. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0020.

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Pulmonary involvement is common in the connective tissue diseases (CTDs) and is associated with significant morbidity and mortality. Improved management of systemic disease has led to increasing numbers of surviving patients with clinically significant pulmonary disease. Screening for pulmonary complications highlights the frequency of subclinical involvement. In this chapter, the pulmonary manifestations of the more common CTDs are detailed, including rheumatoid arthritis (RA), systemic sclerosis (SSc), systemic lupus erythematosus (SLE), polymyositis/dermatomyositis (PM/DM), Sjögren's syndro

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35

Splitthoff, Adrian Jan Oliver. 3 Tesla-Magnestresonanztomographie (MRT) der Rheumatoiden Arthritis (RA): Morphologie und dynamisches Kontrastmittelverhalten. 2005.

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36

Lewis, Myles, and Tim Vyse. Genetics of connective tissue diseases. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0042.

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The advent of genome-wide association studies (GWAS) has been an exciting breakthrough in our understanding of the genetic aetiology of autoimmune diseases. Substantial overlap has been found in susceptibility genes across multiple diseases, from connective tissue diseases and rheumatoid arthritis (RA) to inflammatory bowel disease, coeliac disease, and psoriasis. Major technological advances now permit genotyping of millions of single nucleotide polymorphisms (SNPs). Group analysis of SNPs by haplotypes, aided by completion of the Hapmap project, has improved our ability to pinpoint causal ge

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