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Journal articles on the topic 'Rheumatoid arthritis Rheumatoid arthritis Osteoarthritis Osteoarthritis Hydrocortisone'

Author: Grafiati

Published: 4 June 2021

Last updated: 5 February 2022

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1

Krug, Barbara. "Rheumatoid Arthritis and Osteoarthritis." Orthopaedic Nursing 16, no. 5 (September 1997): 73???75. http://dx.doi.org/10.1097/00006416-199709000-00017.

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2

Starodubtseva, I. A., and L. V. Vasilyeva. "SECONDARY OSTEOARTHRITIS IN RHEUMATOID ARTHRITIS." Clinician 9, no. 1 (May 15, 2015): 24. http://dx.doi.org/10.17650/1818-8338-2015-1-24-29.

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3

Jessop, David S., Alexander Fassold, Christine Wolff, Rafael Hofbauer, Antonio Chover-Gonzalez, Louise J. Richards, and Rainer H. Straub. "Endomorphins in rheumatoid arthritis, osteoarthritis, and experimental arthritis." Annals of the New York Academy of Sciences 1193, no. 1 (April 2010): 117–22. http://dx.doi.org/10.1111/j.1749-6632.2009.05294.x.

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4

Nevins, A., and S. McManigal. "Understanding Osteoarthritis and Understanding Rheumatoid Arthritis." Gerontologist 35, no. 5 (October 1, 1995): 719. http://dx.doi.org/10.1093/geront/35.5.719.

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5

Mella, Lucas Francisco Botequio, Manoel Barros Bértolo, and Paulo Dalgalarrondo. "Depressive symptoms in rheumatoid arthritis." Revista Brasileira de Psiquiatria 32, no. 3 (August 13, 2010): 257–63. http://dx.doi.org/10.1590/s1516-44462010005000021.

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OBJECTIVE: To determine the prevalence of depressive and anxiety symptoms in patients with rheumatoid arthritis (a chronic inflammatory disease) in comparison to a control group with osteoarthritis (a chronic non-inflammatory degenerative disease) and to identify the sociodemographic and clinical variables associated with depressive symptoms in these patients. METHOD: Sixty-two rheumatoid arthritis patients and 60 osteoarthritis patients participated in the study. Sociodemographic and clinical data were collected and the Hospital Anxiety and Depression Scale and the Disability Index of the Health Assessment Questionnaire were applied. RESULTS: The prevalence of depressive symptoms was of 53.2% in rheumatoid arthritis and 28.3% in osteoarthritis (p = 0.005). The prevalence of anxiety symptoms was of 48.4% in rheumatoid arthritis and 50.0% in osteoarthritis (p = 0.859). The mean (and standard deviation) scores in the Disability Index of the Health Assessment Questionnaire were 1.4 (0.8) in rheumatoid arthritis and 1.4 (0.6) in osteoarthritis (p = 0.864). Rheumatoid arthritis patients with depressive symptoms had lower education and higher disease activity and functional disability. CONCLUSION: Although these two rheumatic diseases are similar in terms of the pain and functional disability that they cause, a significantly higher prevalence of depressive symptoms was found in rheumatoid arthritis patients. This difference might be explained by the hypothesis of a neuroimmunobiological mechanism related to cytokines in inflammatory diseases, which has been considered as a candidate to the development of depressive symptoms.

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6

Lark, Michael W., Ellen K. Bayne, and L. Stefan Lohmander. "Aggrecan degradation in osteoarthritis and rheumatoid arthritis." Acta Orthopaedica Scandinavica 66, sup266 (January 1995): 92–97. http://dx.doi.org/10.3109/17453679509157660.

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7

Ehrlich, G. E. "Concurrence of interphalangeal osteoarthritis and rheumatoid arthritis." Rheumatology 43, no. 4 (January 6, 2004): 536. http://dx.doi.org/10.1093/rheumatology/keh085.

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8

Lin, Hai-Shu, Roland Baron, and Philippe Clement-Lacroix. "Aberrant epigenetics in rheumatoid arthritis and osteoarthritis." Future Rheumatology 2, no. 3 (June 2007): 257–60. http://dx.doi.org/10.2217/17460816.2.3.257.

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9

ROSS, CLARE. "A Comparison of Osteoarthritis and Rheumatoid Arthritis." Nurse Practitioner 22, no. 9 (September 1997): 20???41. http://dx.doi.org/10.1097/00006205-199709000-00003.

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10

Kofoed, H., and T. S. Sørensen. "Ankle arthroplasty for rheumatoid arthritis and osteoarthritis." Journal of Bone and Joint Surgery. British volume 80-B, no. 2 (March 1998): 328–32. http://dx.doi.org/10.1302/0301-620x.80b2.0800328.

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11

Marino, Catherine, and Elizabeth McDonald. "Osteoarthritis and rheumatoid arthritis in elderly patients." Postgraduate Medicine 90, no. 5 (October 1991): 237–43. http://dx.doi.org/10.1080/00325481.1991.11701087.

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12

Clayton, Elizabeth Smelter, and Marc C. Hochberg. "Osteoporosis and Osteoarthritis, Rheumatoid Arthritis and Spondylarthropathies." Current Osteoporosis Reports 11, no. 4 (October 2, 2013): 257–62. http://dx.doi.org/10.1007/s11914-013-0172-1.

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13

Semble, Elliott L., Richard F. Loeser, and Christopher M. Wise. "Therapeutic exercise for rheumatoid arthritis and osteoarthritis." Seminars in Arthritis and Rheumatism 20, no. 1 (August 1990): 32–40. http://dx.doi.org/10.1016/0049-0172(90)90092-t.

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14

Beasley, Jeanine. "Osteoarthritis and Rheumatoid Arthritis: Conservative Therapeutic Management." Journal of Hand Therapy 25, no. 2 (April 2012): 163–72. http://dx.doi.org/10.1016/j.jht.2011.11.001.

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15

Jessop, David S. "Endomorphins in rheumatoid arthritis, osteoarthritis and experimental arthritis: clinical prospects." International Journal of Clinical Rheumatology 4, no. 4 (August 2009): 429–35. http://dx.doi.org/10.2217/ijr.09.27.

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16

Topf, Yvonne. "Video Review: Rheumatoid Arthritis; Osteoarthritis; Arthritis: Feeling Good with it." Canadian Journal of Occupational Therapy 60, no. 5 (December 1993): 291. http://dx.doi.org/10.1177/000841749306000518.

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17

Acar, M., E. Tonga, A. Daskapan, M. Karataş, and A. Tekindal. "Comparison of Physical Activity Levels in Rheumatic Diseases." Aktuelle Rheumatologie 42, no. 04 (June 29, 2016): 329–35. http://dx.doi.org/10.1055/s-0042-100614.

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Abstract Objectives: The aim of this study was to evaluate levels of physical activity in patients with rheumatoid arthritis, osteoarthritis and fibromyalgia and to compare the results with both healthy subjects and each other. Methods: A group of 50 rheumatoid arthritis patients, 95 osteoarthritis patients, 82 fibromyalgia patients and 110 healthy subjects were included in this study. Physical activity levels were assessed by the International Physical Activity Questionnaire (IPAQ). Results: There were significant differences in walking and total physical activity scores in IPAQ between the rheumatoid arthritis (RA), osteoarthritis (OA), fibromyalgia (FMS) patients and the control group (p<0.05). 36.6% of the fibromyalgia group, 28.4% of the osteoarthritis group, 38% of the rheumatoid arthritis group and 22.7% of the healthy subjects were found to be inactive. 45.1% of the fibromyalgia group, 42.1% of the osteoarthritis group, 46% of the rheumatoid arthritis group and 36.4% of the healthy subjects were found to be insufficiently active. Conclusion: As a result, when compared to healthy people, physical activity levels were significantly decreased in patients with rheumatoid arthritis, osteoarthritis, fibromyalgia. The decreases in the physical activity levels were clearer in patients with rheumatoid arthritis than in patients with osteoarthritis and fibromyalgia. Recommending regular physical activity should be integral to rheumatic disease management and walking offers a potentially accessible, inexpensive, and acceptable physical activity intervention.

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18

Flynn, Barbara L. "Rheumatoid Arthritis and Osteoarthritis: Current and Future Therapies." American Pharmacy 34, no. 11 (November 1994): 31–42. http://dx.doi.org/10.1016/s0160-3450(15)30253-1.

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19

Xing, Lian-ping. "Molecular imaging technologies for osteoarthritis/rheumatoid arthritis studies." Journal of Orthopaedic Translation 2, no. 4 (October 2014): 202–3. http://dx.doi.org/10.1016/j.jot.2014.07.124.

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20

Verstraeten, A., J. Dequeker, and P. Geusens. "Metabolic bone disease in rheumatoid arthritis and osteoarthritis." Annals of the Rheumatic Diseases 44, no. 4 (April 1, 1985): 283–84. http://dx.doi.org/10.1136/ard.44.4.283.

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21

Kaltsas, D. S., and D. J. Klugman. "Total Knee Replacement in Osteoarthritis and Rheumatoid Arthritis." Journal of the Royal Society of Medicine 78, no. 7 (July 1985): 552–56. http://dx.doi.org/10.1177/014107688507800707.

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22

Ko, Sang Hun. "β-glucuronidase Activity in Rheumatoid Arthritis and Osteoarthritis." Ewha Medical Journal 20, no. 4 (1997): 385. http://dx.doi.org/10.12771/emj.1997.20.4.385.

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23

&NA;. "Chlortenoxicam: appears effective in osteoarthritis and rheumatoid arthritis." Inpharma Weekly &NA;, no. 767 (December 1990): 7–8. http://dx.doi.org/10.2165/00128413-199007670-00018.

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24

Wai Ying, Kathryn Ning, and Alison While. "Pain relief in osteoarthritis and rheumatoid arthritis: TENS." British Journal of Community Nursing 12, no. 8 (August 2007): 364–71. http://dx.doi.org/10.12968/bjcn.2007.12.8.24366.

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25

SHANRELD, STEWART, PATRICIA CAMPBELL, MORRIS BAUMGARTEN, ROY BLOEBAUM, and AUGUSTO SARMIENTO. "Synovial Fluid Osmolality in Osteoarthritis and Rheumatoid Arthritis." Clinical Orthopaedics and Related Research &NA;, no. 235 (October 1988): 289???295. http://dx.doi.org/10.1097/00003086-198810000-00029.

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26

Sanchez-Sotelo, Joaquin. "(i) Shoulder arthroplasty for osteoarthritis and rheumatoid arthritis." Current Orthopaedics 21, no. 6 (December 2007): 405–14. http://dx.doi.org/10.1016/j.cuor.2007.11.002.

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27

MacDonald, Iona J., Chien-Chung Huang, Shan-Chi Liu, Yen-You Lin, and Chih-Hsin Tang. "Targeting CCN Proteins in Rheumatoid Arthritis and Osteoarthritis." International Journal of Molecular Sciences 22, no. 9 (April 21, 2021): 4340. http://dx.doi.org/10.3390/ijms22094340.

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The CCN family of matricellular proteins (CYR61/CCN1, CTGF/CCN2, NOV/CCN3 and WISP1-2-3/CCN4-5-6) are essential players in the key pathophysiological processes of angiogenesis, wound healing and inflammation. These proteins are well recognized for their important roles in many cellular processes, including cell proliferation, adhesion, migration and differentiation, as well as the regulation of extracellular matrix differentiation. Substantial evidence implicates four of the proteins (CCN1, CCN2, CCN3 and CCN4) in the inflammatory pathologies of rheumatoid arthritis (RA) and osteoarthritis (OA). A smaller evidence base supports the involvement of CCN5 and CCN6 in the development of these diseases. This review focuses on evidence providing insights into the involvement of the CCN family in RA and OA, as well as the potential of the CCN proteins as therapeutic targets in these diseases.

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28

Falsetti, P., B. Frediani, A. Fioravanti, C. Acciai, F. Baldi, G. Filippou, and R. Marcolongo. "Sonographic study of calcaneal entheses in erosive osteoarthritis, nodal osteoarthritis, rheumatoid arthritis and psoriatic arthritis." Scandinavian Journal of Rheumatology 32, no. 4 (August 2003): 229–34. http://dx.doi.org/10.1080/03009740310003721.

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29

Kuranobu, Koji, Kichizo Yamamoto, Ryota Teshima, Hideaki Kishimoto, and Touru Okano. "Iliac Bone Histomorphometric Study in Osteoarthritis and Rheumatoid Arthritis." Orthopedics & Traumatology 41, no. 1 (1992): 37–40. http://dx.doi.org/10.5035/nishiseisai.41.37.

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30

Kim, Jae Do, Jeong Hyeon Jo, and Sung Koun Heo. "Total Knee Replacement Arthroplasty in Rheumatoid Arthritis and Osteoarthritis." Journal of the Korean Orthopaedic Association 25, no. 5 (1990): 1438. http://dx.doi.org/10.4055/jkoa.1990.25.5.1438.

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31

Brouwers, Hilde, Joost von Hegedus, René Toes, Margreet Kloppenburg, and Andreea Ioan-Facsinay. "Lipid mediators of inflammation in rheumatoid arthritis and osteoarthritis." Best Practice & Research Clinical Rheumatology 29, no. 6 (December 2015): 741–55. http://dx.doi.org/10.1016/j.berh.2016.02.003.

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32

Macauley, David. "Worldwide experience with diclofenac in rheumatoid arthritis and osteoarthritis." Seminars in Arthritis and Rheumatism 15, no. 2 (November 1985): 68–72. http://dx.doi.org/10.1016/s0049-0172(85)80014-7.

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33

Broadbent, Mark R., and Michael J. Hayton. "The hand and wrist in rheumatoid arthritis and osteoarthritis." Surgery (Oxford) 28, no. 2 (February 2010): 89–94. http://dx.doi.org/10.1016/j.mpsur.2009.10.018.

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34

Kettunen, Jyrki A., and Urho M. Kujala. "Exercise therapy for people with rheumatoid arthritis and osteoarthritis." Scandinavian Journal of Medicine and Science in Sports 14, no. 3 (June 2004): 138–42. http://dx.doi.org/10.1111/j.1600-0838.2004.00396.x.

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35

Inoue, K., K. Shichikawa, J. Nishioka, and S. Hirota. "Older age onset rheumatoid arthritis with or without osteoarthritis." Annals of the Rheumatic Diseases 46, no. 12 (December 1, 1987): 908–11. http://dx.doi.org/10.1136/ard.46.12.908.

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36

Martinez, A., J. Varade, J. R. Lamas, M. Fernandez-Arquero, J. A. Jover, E. G. de la Concha, B. Fernandez-Gutierrez, and E. Urcelay. "GDF5 Polymorphism associated with osteoarthritis: risk for rheumatoid arthritis." Annals of the Rheumatic Diseases 67, no. 9 (November 30, 2007): 1352–53. http://dx.doi.org/10.1136/ard.2007.085050.

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37

Juni, P. "Systematic review of celecoxib for osteoarthritis and rheumatoid arthritis." BMJ 326, no. 7384 (February 8, 2003): 334. http://dx.doi.org/10.1136/bmj.326.7384.334.

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38

Spector, T. D., L. Woodward, G. M. Hall, A. Hammond, A. Williams, M. G. Butler, I. T. James, D. J. Hart, P. W. Thompson, and D. L. Scott. "Keratan sulphate in rheumatoid arthritis, osteoarthritis, and inflammatory diseases." Annals of the Rheumatic Diseases 51, no. 10 (October 1, 1992): 1134–37. http://dx.doi.org/10.1136/ard.51.10.1134.

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39

Blackburn, Warren D. "Management of osteoarthritis and rheumatoid arthritis: Prospects and possibilities." American Journal of Medicine 100, no. 2 (February 1996): 24S—30S. http://dx.doi.org/10.1016/s0002-9343(97)89543-5.

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40

Ronai, Peter, Paul Sorace, and Tom LaFontaine. "Resistance Training for Persons With Osteoarthritis and Rheumatoid Arthritis." Strength and Conditioning Journal 30, no. 2 (April 2008): 32–34. http://dx.doi.org/10.1519/ssc.0b013e318166c2b5.

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41

Ruchlin, Hirsch S., Elena B. Elkin, and Stephen A. Paget. "Assessing cost-effectiveness analyses in rheumatoid arthritis and osteoarthritis." Arthritis Care & Research 10, no. 6 (December 1997): 413–21. http://dx.doi.org/10.1002/art.1790100609.

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42

Kaneta, Tomohiro, Takashi Hakamatsuka, Takayuki Yamada, Kei Takase, Akihiro Sato, Shuichi Higano, Hiroshi Ito, Hiroshi Fukuda, Shoki Takahashi, and Shogo Yamada. "Atlantoaxial Osteoarthritis in Rheumatoid Arthritis: FDG PET/CT Findings." Clinical Nuclear Medicine 31, no. 4 (April 2006): 209. http://dx.doi.org/10.1097/01.rlu.0000204124.79497.bb.

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43

V??ZQUEZ-VELA, GONZALO, EDUARDO V??ZQUEZ-VELA, and FIDEL GARCIA DOBARGANES. "The Baternan Bipolar Prosthesis in Osteoarthritis and Rheumatoid Arthritis." Clinical Orthopaedics and Related Research &NA;, no. 251 (February 1990): 82???86. http://dx.doi.org/10.1097/00003086-199002000-00013.

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44

Vergunst, C. E., M. G. H. van de Sande, M. C. Lebre, and P. P. Tak. "The role of chemokines in rheumatoid arthritis and osteoarthritis." Scandinavian Journal of Rheumatology 34, no. 6 (January 2005): 415–25. http://dx.doi.org/10.1080/03009740500439159.

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45

Scott, D. L., and D. A. Houssien. "Clinical and Laboratory Assessments in Rheumatoid Arthritis and Osteoarthritis." Rheumatology 35, suppl 3 (January 1, 1996): 6–9. http://dx.doi.org/10.1093/rheumatology/35.suppl_3.6.

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46

Gao, Ningyang, Li Ding, Jian Pang, Yuxin Zheng, Yuelong Cao, Hongsheng Zhan, and Yinyu Shi. "Metabonomic-Transcriptome Integration Analysis on Osteoarthritis and Rheumatoid Arthritis." International Journal of Genomics 2020 (January 2, 2020): 1–9. http://dx.doi.org/10.1155/2020/5925126.

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Purpose. This study is aimed at exploring the potential metabolite/gene biomarkers, as well as the differences between the molecular mechanisms, of osteoarthritis (OA) and rheumatoid arthritis (RA). Methods. Transcriptome dataset GSE100786 was downloaded to explore the differentially expressed genes (DEGs) between OA samples and RA samples. Meanwhile, metabolomic dataset MTBLS564 was downloaded and preprocessed to obtain metabolites. Then, the principal component analysis (PCA) and linear models were used to reveal DEG-metabolite relations. Finally, metabolic pathway enrichment analysis was performed to investigate the differences between the molecular mechanisms of OA and RA. Results. A total of 976 DEGs and 171 metabolites were explored between OA samples and RA samples. The PCA and linear module analysis investigated 186 DEG-metabolite interactions including Glycogenin 1- (GYG1-) asparagine_54, hedgehog acyltransferase- (HHAT-) glucose_70, and TNF receptor-associated factor 3- (TRAF3-) acetoacetate_35. Finally, the KEGG pathway analysis showed that these metabolites were mainly enriched in pathways like gap junction, phagosome, NF-kappa B, and IL-17 pathway. Conclusions. Genes such as HHAT, GYG1, and TRAF3, as well as metabolites including glucose, asparagine, and acetoacetate, might be implicated in the pathogenesis of OA and RA. Metabolites like ethanol and tyrosine might participate differentially in OA and RA progression via the gap junction pathway and phagosome pathway, respectively. TRAF3-acetoacetate interaction may be involved in regulating inflammation in OA and RA by the NF-kappa B and IL-17 pathway.

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47

Reines, Brandon P. "Is rheumatoid arthritis premature osteoarthritis with fetal-like healing?" Autoimmunity Reviews 3, no. 4 (June 2004): 305–11. http://dx.doi.org/10.1016/j.autrev.2003.11.002.

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48

ABRAMSON, S., and Y. YAZICI. "Biologics in development for rheumatoid arthritis: Relevance to osteoarthritis☆." Advanced Drug Delivery Reviews 58, no. 2 (May 20, 2006): 212–25. http://dx.doi.org/10.1016/j.addr.2006.01.008.

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49

Symons, J. A., J. F. McCulloch, N. C. Wood, and G. W. Duff. "Soluble CD4 in patients with rheumatoid arthritis and osteoarthritis." Clinical Immunology and Immunopathology 60, no. 1 (July 1991): 72–82. http://dx.doi.org/10.1016/0090-1229(91)90113-o.

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50

Baysal, Ozlem, Tamer Baysal, Ahmet Sigirci, Yuksel Ersoy, and Zuhal Altay. "Atlanto-odontoid osteoarthritis in rheumatoid arthritis: dynamic CT findings." Clinical Rheumatology 23, no. 5 (April 20, 2004): 390–94. http://dx.doi.org/10.1007/s10067-004-0895-6.

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