Relevant bibliographies by topics / Rho GTPases Signaling

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Rho GTPases Signaling'

Author: Grafiati
Published: 7 September 2023
Last updated: 31 July 2025

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Journal articles on the topic "Rho GTPases Signaling"

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Mosaddeghzadeh, Niloufar, and Mohammad Reza Ahmadian. "The RHO Family GTPases: Mechanisms of Regulation and Signaling." Cells 10, no. 7 (2021): 1831. http://dx.doi.org/10.3390/cells10071831.

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Much progress has been made toward deciphering Rho GTPase functions, and many studies have convincingly demonstrated that altered signal transduction through Rho GTPases is a recurring theme in the progression of human malignancies. It seems that 20 canonical RHO GTPases are likely regulated by three GDIs, 85 GEFs, and 66 GAPs, and eventually interact with >70 downstream effectors. A recurring theme is the challenge in understanding the molecular determinants of the specificity of these four classes of interacting proteins that, irrespective of their functions, bind to common sites on the s
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Mulloy, James C., Jose A. Cancelas, Marie-Dominique Filippi, Theodosia A. Kalfa, Fukun Guo, and Yi Zheng. "Rho GTPases in hematopoiesis and hemopathies." Blood 115, no. 5 (2010): 936–47. http://dx.doi.org/10.1182/blood-2009-09-198127.

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AbstractRho family GTPases are intracellular signaling proteins regulating multiple pathways involved in cell actomyosin organization, adhesion, and proliferation. Our knowledge of their cellular functions comes mostly from previous biochemical studies that used mutant overexpression approaches in various clonal cell lines. Recent progress in understanding Rho GTPase functions in blood cell development and regulation by gene targeting of individual Rho GTPases in mice has allowed a genetic understanding of their physiologic roles in hematopoietic progenitors and mature lineages. In particular,
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Olayioye, Monilola A., Bettina Noll, and Angelika Hausser. "Spatiotemporal Control of Intracellular Membrane Trafficking by Rho GTPases." Cells 8, no. 12 (2019): 1478. http://dx.doi.org/10.3390/cells8121478.

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As membrane-associated master regulators of cytoskeletal remodeling, Rho GTPases coordinate a wide range of biological processes such as cell adhesion, motility, and polarity. In the last years, Rho GTPases have also been recognized to control intracellular membrane sorting and trafficking steps directly; however, how Rho GTPase signaling is regulated at endomembranes is still poorly understood. In this review, we will specifically address the local Rho GTPase pools coordinating intracellular membrane trafficking with a focus on the endo- and exocytic pathways. We will further highlight the sp
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Voena and Chiarle. "RHO Family GTPases in the Biology of Lymphoma." Cells 8, no. 7 (2019): 646. http://dx.doi.org/10.3390/cells8070646.

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RHO GTPases are a class of small molecules involved in the regulation of several cellular processes that belong to the RAS GTPase superfamily. The RHO family of GTPases includes several members that are further divided into two different groups: typical and atypical. Both typical and atypical RHO GTPases are critical transducers of intracellular signaling and have been linked to human cancer. Significantly, both gain-of-function and loss-of-function mutations have been described in human tumors with contradicting roles depending on the cell context. The RAS family of GTPases that also belong t
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Zubor, Pavol, Zuzana Dankova, Zuzana Kolkova, et al. "Rho GTPases in Gynecologic Cancers: In-Depth Analysis toward the Paradigm Change from Reactive to Predictive, Preventive, and Personalized Medical Approach Benefiting the Patient and Healthcare." Cancers 12, no. 5 (2020): 1292. http://dx.doi.org/10.3390/cancers12051292.

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Rho guanosine triphospatases (GTPases) resemble a conserved family of GTP-binding proteins regulating actin cytoskeleton dynamics and several signaling pathways central for the cell. Rho GTPases create a so-called Ras-superfamily of GTPases subdivided into subgroups comprising at least 20 members. Rho GTPases play a key regulatory role in gene expression, cell cycle control and proliferation, epithelial cell polarity, cell migration, survival, and apoptosis, among others. They also have tissue-related functions including angiogenesis being involved in inflammatory and wound healing processes.
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Fritz, Rafael Dominik, and Olivier Pertz. "The dynamics of spatio-temporal Rho GTPase signaling: formation of signaling patterns." F1000Research 5 (April 26, 2016): 749. http://dx.doi.org/10.12688/f1000research.7370.1.

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Rho GTPases are crucial signaling molecules that regulate a plethora of biological functions. Traditional biochemical, cell biological, and genetic approaches have founded the basis of Rho GTPase biology. The development of biosensors then allowed measuring Rho GTPase activity with unprecedented spatio-temporal resolution. This revealed that Rho GTPase activity fluctuates on time and length scales of tens of seconds and micrometers, respectively. In this review, we describe Rho GTPase activity patterns observed in different cell systems. We then discuss the growing body of evidence that upstre
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Barlow, Haley Rose, and Ondine Cleaver. "Building Blood Vessels—One Rho GTPase at a Time." Cells 8, no. 6 (2019): 545. http://dx.doi.org/10.3390/cells8060545.

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Blood vessels are required for the survival of any organism larger than the oxygen diffusion limit. Blood vessel formation is a tightly regulated event and vessel growth or changes in permeability are linked to a number of diseases. Elucidating the cell biology of endothelial cells (ECs), which are the building blocks of blood vessels, is thus critical to our understanding of vascular biology and to the development of vascular-targeted disease treatments. Small GTPases of the Rho GTPase family are known to regulate several processes critical for EC growth and maintenance. In fact, many of the
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Kjøller, Lars, and Alan Hall. "Signaling to Rho GTPases." Experimental Cell Research 253, no. 1 (1999): 166–79. http://dx.doi.org/10.1006/excr.1999.4674.

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Zhang, Zheng, Ming Liu, and Yi Zheng. "Role of Rho GTPases in stem cell regulation." Biochemical Society Transactions 49, no. 6 (2021): 2941–55. http://dx.doi.org/10.1042/bst20211071.

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The future of regenerative medicine relies on our understanding of stem cells which are essential for tissue/organ generation and regeneration to maintain and/or restore tissue homeostasis. Rho family GTPases are known regulators of a wide variety of cellular processes related to cytoskeletal dynamics, polarity and gene transcription. In the last decade, major new advances have been made in understanding the regulatory role and mechanism of Rho GTPases in self-renewal, differentiation, migration, and lineage specification in tissue-specific signaling mechanisms in various stem cell types to re
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Guo, Daji, Xiaoman Yang, and Lei Shi. "Rho GTPase Regulators and Effectors in Autism Spectrum Disorders: Animal Models and Insights for Therapeutics." Cells 9, no. 4 (2020): 835. http://dx.doi.org/10.3390/cells9040835.

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The Rho family GTPases are small G proteins that act as molecular switches shuttling between active and inactive forms. Rho GTPases are regulated by two classes of regulatory proteins, guanine nucleotide exchange factors (GEFs) and GTPase-activating proteins (GAPs). Rho GTPases transduce the upstream signals to downstream effectors, thus regulating diverse cellular processes, such as growth, migration, adhesion, and differentiation. In particular, Rho GTPases play essential roles in regulating neuronal morphology and function. Recent evidence suggests that dysfunction of Rho GTPase signaling c
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Dissertations / Theses on the topic "Rho GTPases Signaling"

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Fransson, Åsa. "Cell signaling by Rho and Miro GTPases : Studies of Rho GTPases in Cytoskeletal Reorganizations and of Miro GTPases in Mitochondrial Dynamics." Doctoral thesis, Uppsala University, Ludwig Institute for Cancer Research, 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8514.

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<p>The Ras superfamily of GTPases embraces six major branches of proteins: the Ras, Rab, Ran, Arf, Rho and Miro subfamilies. The majority of GTPases function as binary switches that cycle between active GTP-bound and inactive GDP-bound states. This thesis will focus primarily on the biological functions of the Rho and Miro proteins. The Rho GTPases control the organization of the actin cytoskeleton and other associated activities, whereas the Miro GTPases are regulators of mitochondrial movement and morphology. </p><p>A diverse array of cellular phenomena, including cell movement and intracell
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Fransson, Åsa. "Cell signaling by Rho and Miro GTPases : studies of Rho GTPases in cytoskeletal reorganizations and of Miro GTPases in mitochondrial dynamics /." Uppsala : Acta Universitatis Upsaliensis : Universitetsbiblioteket [distributör], 2008. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-8514.

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Chan, Man-lok Mandy, and 陳文樂. "A study of RhoV and PAK4 signaling in hepatocarcinogenesis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47053434.

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Sipes, Nisha Schuler. "Cdc42 signaling in extracellular matrix remodeling in three dimensions." University of Cincinnati / OhioLINK, 2009. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1253622562.

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Picard, Mariêve. "The role of the small Rho GTPases in the signaling mechanisms mediated by the netrin-1 receptor UNC5a." Thesis, McGill University, 2008. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=19255.

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Netrin-1 is a bifunctional chemotropic cue that attracts or repels different classes of axons through the Deleted in Colorectal Cancer (DCC) and the UNC5 receptors (UNC5a, b, c and d). DCC is implicated in mediating both responses whereas UNC5 receptors are strictly involved in the repulsive events. The intracellular molecular mechanisms underlying axon growth and guidance are still unclear but it is known that this process requires remodeling of the actin cytoskeleton. There is now compelling evidence that Rho GTPases, in
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Hoop, Alyssa N. "Rho-Family GTPase Signaling in the Nervous System: An Analysis of the C. elegans RhoGEF UNC-73." University of Toledo / OhioLINK, 2014. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1404733888.

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Saint-Cyr, Proulx Étienne. "Role of the Rho GTPases in the signaling mechanisms regulated by the axon guidance cue Netrin-1 receptors deleted in colorectar cancer and Unc5H1." Thesis, McGill University, 2005. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=98788.

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Chemotropic cues guide the migrating axons of neurons in the developing nervous system. Netrins are bifunctional guidance cues, attracting or repelling different classes of axons. The attraction to netrins is mediated by DCC whereas repulsion is achieved through Unc5H receptors. In this thesis, we demonstrate that Rho GTPases are required for embryonic spinal commissural axon outgrowth induced by Netrin-1. Using N1E-115 neuroblastoma cells, we have found that Rac1 and Cdc42 activities are required for DCC-induced neurite outgrowth. In fibroblasts, DCC was found to trigger actin reorganization
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Silva, Gisele Espinha Teixeira da. "Sinalização da GTPase RhoA nas respostas celulares após estresse genotóxico promovido por radiação ultravioleta." Universidade de São Paulo, 2016. http://www.teses.usp.br/teses/disponiveis/87/87131/tde-19102016-165552/.

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A via de sinalização da GTPase RhoA atua em diversos processos celulares. Para avaliar o comportamento de RhoA, após estresse causado por radiação ultravioleta, foram gerados clones mutantes que expressam RhoA em seu estado constitutivamente ativo e dominante negativo. Após exposição das linhagens à radiação ultravioleta, observou-se uma maior sensibilidade e um maior tempo de recuperação das linhagens quando a atividade de RhoA é reduzida. Estes prejuízos no reparo prejudicaram a proliferação e sobrevivência celular quando da deficiência na atividade de RhoA. Em linhagens deficientes na via d
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Osaki, Juliana Harumi. "O papel de RhoA e Rac1 GTPases nas respostas celulares após danos no DNA induzidos por radiação ionizante gama." Universidade de São Paulo, 2015. http://www.teses.usp.br/teses/disponiveis/46/46131/tde-22092015-075415/.

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O mecanismo pelo qual uma célula responde a algum dano no seu material genético é extremamente importante. Isto ocorre pela rápida ativação da maquinaria de reparo de danos no DNA, a qual é composta por uma rede intrincada de sinalização proteica, culminando no reparo do DNA; porém se o dano for irreparável ocorre ativação de mecanismos de morte celular. RhoA,e Rac1 pertencem a família das pequenas proteínas sinalizadoras Rho GTPases, as quais atuam como interruptores moleculares ciclando entre estado ativo (ligada a GTP) e inativo (ligada a GDP). Os componentes desta família estão relacionado
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Marshall, Andrew Keith. "Signalling through Rho GTPases in cardiomyocytes." Thesis, Imperial College London, 2011. http://hdl.handle.net/10044/1/6962.

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Endothelin-1 (ET-1) promotes changes in gene/protein expression in cardiomyocytes leading to hypertrophy. This results from activation of intracellular signalling pathways including small G proteins that activate protein kinases. Thus, ET-1 activates RhoA that stimulates ROCK and PKN, and Ras that promotes activation of extracellular signal-regulated kinases 1/2 (ERK1/2). Microarrays were used to dissect the roles of ERK1/2 vs RhoA in the cardiomyocyte transcriptomic response to ET-1 using PD184352 and C3 endotoxin from C. botulinum (C3T) for selective inhibition of the ERK1/2 cascade and RhoA
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Books on the topic "Rho GTPases Signaling"

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Ciano-Oliveira, Caterina Di. Signaling pathways linking osmotic stress to adaptive responses: Roles for Rho family GTPases. 2006.

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Book chapters on the topic "Rho GTPases Signaling"

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Karnoub, Antoine E., Emily J. Chenette, and Channing J. Der. "RHO Proteins in RAS Signaling and Transformation." In RAS Family GTPases. Springer Netherlands, 2006. http://dx.doi.org/10.1007/1-4020-4708-8_7.

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Hajicek, Nicole, Barry Kreutz, and Tohru Kozasa. "Signaling through Galpha12/13 and RGS-RhoGEFs." In The Rho GTPases in Cancer. Springer New York, 2009. http://dx.doi.org/10.1007/978-1-4419-1111-7_4.

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Zhu, Shizhen, and Boon Chuan Low. "Using Zebrafish for Studying Rho GTPases Signaling In Vivo." In Methods in Molecular Biology. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-61779-442-1_21.

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Chiariello, Mario, Jose P. Vaqué, Piero Crespo, and J. Silvio Gutkind. "Activation of Ras and Rho GTPases and MAP Kinases by G-Protein-Coupled Receptors." In MAP Kinase Signaling Protocols. Humana Press, 2010. http://dx.doi.org/10.1007/978-1-60761-795-2_8.

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Hu, Bo, Marc Symons, Bodour Salhia, et al. "Rho GTPases and Their Activators, Guanine Nucleotide Exchange Factors (GEFs): Their Roles in Glioma Cell Invasion." In Signaling Pathways and Molecular Mediators in Metastasis. Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-2558-4_6.

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Liu, Jian-Qin, and Katsunori Shimohara. "A Novel Programmable Molecular Computing Method Based on Signaling Pathways Regulated by Rho-GTPases in Living MDCK Epithelial Mammalian Cells." In Lecture Notes in Computer Science. Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-540-30217-9_32.

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Fujita, Yasuyuki, and Vania Braga. "Epithelial Cell Shape and Rho Small GTPases." In Signalling Networks in Cell Shape and Motility. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/047001766x.ch12.

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Aspenström, Pontus. "BAR Domain Proteins Regulate Rho GTPase Signaling." In Protein Reviews – Purinergic Receptors. Springer International Publishing, 2018. http://dx.doi.org/10.1007/5584_2018_259.

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Eisenmann, Kathryn M., Jun Peng, Bradley J. Wallar, and Arthur S. Alberts. "Rho GTPase-Formin Pairs in Cytoskeletal Remodelling." In Signalling Networks in Cell Shape and Motility. John Wiley & Sons, Ltd, 2008. http://dx.doi.org/10.1002/047001766x.ch16.

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Feltrin, Daniel, and Olivier Pertz. "Assessment of Rho GTPase Signaling During Neurite Outgrowth." In Methods in Molecular Biology. Springer New York, 2011. http://dx.doi.org/10.1007/978-1-61779-442-1_13.

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Conference papers on the topic "Rho GTPases Signaling"

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Na, Sungsoo. "Engineering Tools for Studying Coordination Between Biochemical and Biomechanical Activities in Cell Migration." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53709.

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Cell migration is achieved by the dynamic feedback interactions between traction forces generated by the cell and exerted onto the underlying extracellular matrix (ECM), and intracellular mechano-chemical signaling pathways, e.g., Rho GTPase (RhoA, Rac1, and Cdc42) activities [1,2,3]. These components are differentially distributed within a cell, and thus the coordination between tractions and mechanotransduction (i.e, RhoA and Rac1 activities) must be implemented at a precise spatial and temporal order to achieve optimized, directed cell migration [4,5]. Recent studies have shown that focal a
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Sun, Hui-Chuan, Jian-Yang Ao, Zong-Tao Chai, and Yuan-Yuan Zhang. "Abstract 4168: Robo1 promotes angiogenesis through CDC42/Rho GTPases signaling pathway in hepatocellular carcinoma." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4168.

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Shi, Geng-Xian, Ling Jin, Michelle L. Matter, Santosh Kesari, and Joe W. Ramos. "Abstract 1363: RSK2 provokes invasive signaling in glioblastoma through LARG-dependent activation of Rho GTPases." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1363.

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Wan, Qiaoqiao, Eunhye Cho, Seungman Park, Bumsoo Han, Hiroki Yokota, and Sungsoo Na. "Visualizing Chondrocyte Mechanotransduction in 3D." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14484.

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Chondrocytes are the only cell type present in the articular cartilage and their response to mechanical stimuli influences the maintenance and remodeling of the cartilage. Numerous studies have shown that the balance between anabolic and catabolic responses of the chondrocytes to mechanical loading is dependent on the loading intensities (reviewed in ref. [1]). Moderate, physiological loading, for instance, increases synthetic activity of the extracellular matrix (ECM) such as collagen type II, aggrecan, and proteoglycan, while decreasing the catabolic activity of degradative enzymes such as m
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Mansour, Mariam, Sue Haupt, Ai-Leen Chan, et al. "Abstract A72: The E3-ligase E6AP represses breast cancer metastasis through regulation of ECT2-Rho-GTPases signaling." In Abstracts: AACR Special Conference: Advances in Breast Cancer; October 17-20, 2015; Bellevue, WA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1557-3125.advbc15-a72.

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Kurisetty, Vittal, Trinath P. Das, Rama S. Reddy, Jessica Stiles, Brad Bryan, and Chendil Damodaran. "Abstract 5329: The role of miR-301-3P in the regulation of Rho GTPases mediated EMT signaling in castration resistant prostate cancer." In Proceedings: AACR 104th Annual Meeting 2013; Apr 6-10, 2013; Washington, DC. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1538-7445.am2013-5329.

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Balraj, P., N. S. Ambhore, N. A. Borkar, C. M. Pabelick, Y. S. Prakash, and S. Venkatachalem. "Kisspeptin Attenuates Airway Smooth Muscle Cell Migration by Regulating Rho GTPase Signaling Pathway." In American Thoracic Society 2022 International Conference, May 13-18, 2022 - San Francisco, CA. American Thoracic Society, 2022. http://dx.doi.org/10.1164/ajrccm-conference.2022.205.1_meetingabstracts.a1229.

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Little, Andrew C., Pragathi Pathanjeli, Zhifen Wu, et al. "Abstract 4518: IL-4/IL-13 stimulated tumor-associated macrophages enhance breast cancer cell invasion through Rho-GTPase signaling." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-4518.

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Little, Andrew C., Pragathi Pathanjeli, Zhifen Wu, et al. "Abstract 4518: IL-4/IL-13 stimulated tumor-associated macrophages enhance breast cancer cell invasion through Rho-GTPase signaling." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-4518.

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Schrecengost, Randy S., Ashley L. Wilson, Michael S. Guerrero, and Amy H. Bouton. "Abstract 5135: Breast cancer antiestrogen resistance-3 influences breast cancer cell migration by regulating Rac and Rho GTPase signaling." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-5135.

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