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Books on the topic 'Rickettsiosi'

Author: Grafiati

Published: 9 March 2023

Last updated: 11 March 2023

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1

E, Hechemy Karim, and New York Academy of Sciences, eds. Century of rickettsiology: Emerging, reemerging rickettsioses, molecular diagnostics, and emerging veterinary rickettsioses. Boston, Mass: Blackwell Pub. on behalf of the New York Academy of Sciences, 2006.

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2

Řeháček, Josef. Acari-borne rickettsiae & rickettsioses in Eurasia. Bratislava: Veda Publishing House of the Slovak Academy of Sciences, 1988.

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3

E, Hechemy Karim, and New York Academy of Sciences., eds. Rickettsioses: From genome to proteome, pathobiology, and rickettsiae as an international threat. New York, N.Y: New York Academy of Sciences, 2005.

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4

Organization, Pan American Health. Zoonosis y enfermedades transmisibles comunes al hombre y a los animales, Volume 2: Volumen 2: Clamidiosis, rickettsiosis, y virosis. 3rd ed. Washington: Pan American Health Organization, 2001.

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5

Burdmann, Emmanuel A., and Vivekanad Jha. Rickettsiosis. Edited by Vivekanand Jha. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0193.

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Rickettsiae are obligate intracellular bacteria transmitted by arthropods to a vertebrate host. Clinically relevant rickettsioses have a similar clinical pattern, manifesting as an acute febrile disease accompanied by headache, articular and muscle pain, and malaise.Epidemic typhus is a worldwide distributed disease caused by the Rickettsia prowazekii, with a human louse as a vector. Data on epidemic typhus-related renal injury is extremely scarce.Murine typhus is caused by the Rickettsia typhi and has a rodent flea as the vector. It is one of the most frequent rickettsioses, and is usually a self-limited febrile illness. Proteinuria, haematuria, elevations in serum creatinine (SCr) and/or blood urea nitrogen (BUN) and AKI have been reported. The real frequency of renal involvement in murine typhus is unknown. Renal abnormalities recover after the infectious disease resolution.Scrub typhus, caused by the Orientia tsutsugamushi, has the Leptotrombidium mite larva as vector. It is endemic in the Tsutsugamushi triangle delimited by Japan, Australia, India, and Siberia. It can manifest either as a self-limiting disease or as a severe, life-threatening multiorgan illness. Early administration of adequate antibiotics is essential to prevent adverse outcomes. Proteinuria, haematuria, and acute kidney injury (AKI) are frequent.Tick-borne rickettsioses are caused by bacteria from the spotted fever group and have ticks as vectors. Rocky Mountain spotted fever (RMSF) is caused by Rickettsia rickettsii. It is the most severe of the spotted fever rickettsial diseases, causing significant morbidity and lethality. RMSF occurs in North, Central, and South America. Renal impairment is frequent in severe forms of RMSF. Mediterranean spotted fever is caused by Rickettsia conorii, and is endemic in the Mediterranean area. It is usually a benign disease, but may have a severe course, clinically similar to RMSF. Haematuria, proteinuria, increased serum creatinine, and AKI may occur. Japanese spotted fever is caused by Rickettsia japonica. Lethal cases are reported yearly and AKI has occurred in the context of multiple organ failure.

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6

Mohr, Werner, Egbert Asshauer, and Otto Gsell. Rickettsiosen und Protozoenkrankheiten. Springer, 2013.

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7

Blanco, José R. Century of Rickettsiology: Emerging, Reemerging Rickettsioses, Molecular Diagnostics, and Emerging Veterinary Rickettsioses (Annals of the New York Academy of Sciences). Blackwell Publishing Limited, 2006.

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8

Nasemann, T., Theodor Nasemann, and Alfred Marchionini. Die Viruskrankheiten der Haut: Und Die Hautsymptome Bei Rickettsiosen und Bartonellosen. Springer, 2012.

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9

Nasemann, T., Theodor Nasemann, and Alfred Marchionini. Die Viruskrankheiten der Haut: Und Die Hautsymptome Bei Rickettsiosen und Bartonellosen. Springer London, Limited, 2013.

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10

Nathan, Neil, writer of foreword, ed. Healing Lyme: Natural healing of Lyme borreliosis and the coinfections chlamydia and spotted fever rickettsioses. 2015.

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11

Rickettsiology: Present and Future Directions (Annals of the New York Academy of Sciences, V. 990). New York Academy of Sciences, 2003.

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12

Cannabis, Healing. CBD Oil for Lyme Disease: Natural Healing of Lyme Borreliosis and the Coinfections Chlamydia and Spotted Fever Rickettsiosis. Independently Published, 2019.

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13

Zoonosis y enfermedades transmisibles comunes al hombre y a los animales, 3a edición. Vol. II Clamidiosis, rickettsiosis y virosis. 3rd ed. Organización Panamericana de la Salud, 2003.

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14

Hechemy, Karim E., Spain) International Conference on Rickettsiae and Rickettsial Diseases (4th : 2005 : Logrono, and International Conference on Rickettsiae. Rickettsioses: From Genome to Proteome, Pathobiology, And Rickettsiae As an International Threat (Annals of the New York Academy of Sciences). New York Academy of Sciences, 2006.

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15

Blanco, José R., Jose A. Oteo, Didier Raoult, and David J. Silverman. Rickettsioses: From Genome to Proteome, Pathobiology, and Rickettsiae as an International Threat (Annals of the New York Academy of Sciences). Blackwell Publishing Limited, 2005.

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16

Paho. Zoonoses and Communicable Diseases Common to Man and Animals, Vol. II: Chlamydioses, Rickettsioses, and Viroses, Third Edition (Scientific and Technical Publication). 3rd ed. Pan American Health Organization, 2003.

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17

Pagan, Eli Fernando. Detection of Complement-Fixing Antibodies for Rickettsia Rickettsii in the Serum of Lepus Californicus Melanotis, Mearns (Black-tailed Jack Rabbit). Creative Media Partners, LLC, 2021.

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18

Tuddenham, Susan. Ehrlichia, Anaplasma, and Rickettsia. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0051.

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Rickettsia, Ehrlichia, and Anaplasma are infections primarily transmitted by ticks (but, in the case of certain Rickettsial species, are transmitted by other vectors as well), which can cause an abrupt, febrile, and flu-like illness often associated with headache, nausea, vomiting, abdominal pain, rash, elevated liver function tests, and thrombocytopenia. Disease can be severe, particularly when patients are infected with Rickettsia rickettsii (Rocky Mountain Spotted Fever); patients may develop central nervous system involvement, shock, and multiorgan failure. Diagnostic tests are imperfect, and prompt empiric treatment should be initiated if disease is suspected. Doxycycline is the treatment of choice, and coinfection with other vector-borne pathogens may need to be considered.

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19

Ecological Relationships and Evolution of Rickettsiae. Taylor & Francis Group, 2017.

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20

Marchette, Nyven J. Ecological Relationships and Evolution of Rickettsiae: Volume II. Taylor & Francis Group, 2018.

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21

Ecological Relationships and Evolution of Rickettsiae. Taylor & Francis Group, 2017.

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22

Marchette, Nyven J. Ecological Relationships and Evolution of Rickettsiae: Volume I. Taylor & Francis Group, 2018.

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23

Marchette, Nyven J. Ecological Relationships and Evolution of Rickettsiae: Volume II. Taylor & Francis Group, 2018.

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24

Marchette, Nyven J. Ecological Relationships and Evolution of Rickettsiae: Volume II. Taylor & Francis Group, 2018.

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25

Fletcher, Tom, and Nick Beeching. Rickettsial infection. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0314.

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Rickettsial infections are caused by a variety of obligate intracellular, Gram-negative bacteria from the genera Rickettsia, Orientia, Ehrlichia, and Anaplasma. Rickettsia is further subdivided into the spotted fever group and the typhus group. Bartonella and Coxiella burnetii bacteria are similar to rickettsiae and cause similar diseases. The range of recognized spotted fever group infections is rapidly expanding, complementing long-recognized examples such as Rocky Mountain spotted fever (Rickettsia rickettsii) in the US, and Australian tick typhus (Rickettsia australis), as well as those in southern Europe and Africa. Animals are the predominant reservoir of infection, and transmission to people is usually through ticks, mites, fleas, or lice, during blood-feeding or from scarification of faeces deposited on the skin. This chapter focuses on the two of the most relevant infections encountered in UK practice: African tick typhus, and Q fever.

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26

Cuttle, Lisa. Dermatologic Manifestations of Infectious Disease. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0044.

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Toxic infectious exfoliative conditions include staphylococcal toxic shock syndrome (TSS), streptococcal toxic shock syndrome (STSS), and staphylococcal scalded skin syndrome (SSSS). All three are mediated by bacterial toxin production and are considerations in the differential diagnosis of a febrile, hypotensive patient with a rash. Meningococcemia is potentially fatal and extremely contagious with a short incubation period. Disseminated gonococcal infection (DGI) presents with tenosynovitis, dermatitis, and polyarthralgias without purulent arthritis or with purulent arthritis but without skin lesions. Ecthyma gangrenosum (EG) is a cutaneous manifestation of Pseudomonas aeruginosa infection. Rocky Mountain Spotted Fever (RMSF) is caused by Rickettsia rickettsii, most commonly transmitted by the American dog tick. Patients present with nonspecific symptoms, such as fever, headache, myalgias, arthralgias, nausea, vomiting, and abdominal pain. Finally, vibrio vulnificus is a gram-negative bacterium that causes serious wound infections, sepsis, and diarrhea in patients exposed to shellfish or marine water.

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27

Sun, Lisa, and Michael V. Johnston. Rickettsial Diseases. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199937837.003.0157.

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Tick-borne rickettsioses are emerging as more important health problems throughout the world. The spotted fever group including Rickettsia rickettsia can cause encephalopathy, meningitis and brain damage by selectively targeting capillary endothelial cells in the brain, and stimulating inflammation, capillary leakage, hemorrhage, and intravascular coagulation. Rickettsia are are arthropod-borne gram-negative coccobacilli bacteria and are obligate intracellular organisms that do not survive in artificial medium. In North and South America, the most common rickettsial disorder is rocky mountain spotted fever (RMSF) transmitted by the dog tick Dermacentor variabilis or the wood tick Dermacentor andersoni. A characteristic “starry sky” pattern can be seen on MRI imaging of the brain in some patients with RMSF encephalopathy and is thought to reflect the organisms targeting of brain endothelial cells in capillaries the white matter. Early treatment with doxycycline is curative and reverses signs of encephalopathy if given within a few day of onset, but delayed treatment can be associated with permanent neurological disability. The typhus group of rickettsia bacteria include R. prowazekii, which causes epidemic typhus and R. typhi, which causes murine typhus (endemic) typhus in tropical and subtropical parts of the world. Flying squirrels and humans carry R prowazekii and rats are carry R. typhi. Q fever caused by the rickettsia organism Coxiella burnetti is transmitted from farm animals including sheep and is seen throughout the world including the United States.

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28

Angelakis, Emmanouil, and Didier Raoult. Tick-borne rickettsial diseases. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198570028.003.0010.

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Bacteria of the genus Rickettsia belong to the family Rickettsiaceae in the order Rickettsiales and have for long been described simply as short, Gram-negative, strict intracellular rods that retain basic fuchsin when stained by the method of Gimenez (Raoult and Roux 1997). These bacteria are associated with ticks, lice, fleas or mites. To date the Rickettsia genus contains 24 recognized species classified into three groups based on their antigenic, morphological, and ecologic patterns: 1) the typhus group, 2) the spotted fever group and 3) Rickettsia bellii (Fournier and Raoult 2007). Most spotted fever group (SFG) rickettsiae are closely associated with ticks belonging to the family Ixodidae (also called “hard” ticks) (Parola et al. 2005). Ticks can act as vectors, reservoirs, and/or amplifiers of SFG rickettsiae and require optimal environmental conditions which determine the geographic distribution of the vectors and consequently the risk areas for rickettsioses. Many Rickettsia species are strictly associated with one genus of ticks and the transmittion to people is made through the tick bite, which generally implies that the Rickettsia can localize to their salivary glands. Therefore, since larvae, nymphs, and adults may all be infective for susceptible vertebrate hosts, the ticks must be regarded as the main reservoir host of rickettsiae. Humans are not considered as good reservoirs for Rickettsiae, as they are seldom infested with ticks for long periods and rickettsiaemia has normally short duration, especially with antibiotic intervention.

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29

(Editor), Burt Anderson, Herman Friedman (Editor), and Mauro Bendinelli (Editor), eds. Microorganisms and Bioterrorism (Infectious Agents and Pathogenesis). Springer, 2006.

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30

Friedman, Herman, Mauro Bendinelli, and Burt Anderson. Microorganisms and Bioterrorism. Springer, 2010.

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