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Relevant bibliographies by topics / RNA stem loop / Dissertations / Theses

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Dissertations / Theses on the topic 'RNA stem loop'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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1

Sabir, Tara. "Single molecule studies of the MS2 TR RNA stem loop." Thesis, University of Leeds, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.424024.

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2

Crombie, Catriona Ann. "Histone hairpin binding protein, an RNA binding protein, essential for development." Thesis, University of Aberdeen, 2003. http://digitool.abdn.ac.uk/R?func=search-advanced-go&find_code1=WSN&request1=AAIU602058.

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Histones are proteins found in the nuclei of eukaryotic cells where they are complexed to DNA in chromatin. Rephcation-dependent histones are expressed only during S-phase. Regulation of expression of replication-dependent histone genes requires a highly conserved hairpin RNA element in the 3' untranslated region of histone mRNAs. Replication-dependent histone mRNAs are not polyadenylated; their 3' end is formed by an endonucleolytic cleavage event, 3' of a hairpin element, which is recognised by the Hairpin Binding Protein, HBP (also known as Stem-Loop Binding Protein, SLBP). This protein-RNA

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3

Striggles, John. "The RNA worldview and selecting aptamers against the P5.1 stem-loop of B.subtilis RNase P /." Free to MU Campus, others may purchase, 2003. http://wwwlib.umi.com/cr/mo/fullcit?p1418067.

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4

Hellmund, Christopher James. "Investigating the role of stem-loop 1 in the assembly process of HIV-1." Thesis, University of Cambridge, 2019. https://www.repository.cam.ac.uk/handle/1810/288541.

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An important step in the production of infectious HIV-1 particles is maturation of the virus core. This process is completed by cleavage of the capsid (CA) domain of Gag, from its precursor, CA-SP1, by the viral protease. Large deletions in stem-loop 1 (SL1) in the 5' untranslated region (UTR) of HIV-1 genomic RNA (gRNA) delay CA-SP1 processing. SL1 harbours the dimerisation initiation site (DIS) palindrome suggesting that efficient Gag processing may be linked to gRNA dimerization as shown in HIV-2. However, a dimerisation mutant with normal Gag processing was identified. Gag processing defec

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5

Cojocaru, Vlad. "Molecular motions at the 5 stem-loop of U4 snRNA: Implications for U4/U6 snRNP assembly." Doctoral thesis, [S.l.] : [s.n.], 2005. http://webdoc.sub.gwdg.de/diss/2005/cojocaru.

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6

Denham, Elizabeth. "The Effects of Relocating the Ku-binding Stem-loop of Telomerase RNA on Telomere Healing Events." Thesis, Boston College, 2008. http://hdl.handle.net/2345/528.

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Thesis advisor: Anne E. Stellwagen<br>Thesis advisor: Clare O'Connor<br>In most eukaryotes, the enzyme telomerase adds telomeric DNA repeats to the 3' ends of chromosomes in order to stabilize them and protect them from degradation. In the budding yeast Saccharomyces cerevisiae, telomerase is a ribonucleoprotein complex consisting of multiple protein subunits and an approximately 1.3 kb RNA component termed TLC1. Among the various proteins involved in telomerase, Ku is a heterodimer that binds both to double-stranded DNA and to a 48 nucleotide stem loop on the TLC1 RNA. Beyond its function of

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7

Clingman, Carina C. "A Feedback Loop Couples Musashi-1 Activity to Omega-9 Fatty Acid Biosynthesis: A Dissertation." eScholarship@UMMS, 2014. http://escholarship.umassmed.edu/gsbs_diss/718.

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All living creatures change their gene expression program in response to nutrient availability and metabolic demands. Nutrients and metabolites can directly control transcription and activate second-‐messenger systems. In bacteria, metabolites also affect post-‐transcriptional regulatory mechanisms, but there are only a few isolated examples of this regulation in eukaryotes. Here, I present evidence that RNA-‐binding by the stem cell translation regulator Musashi-‐1 (MSI1) is allosterically inhibited by 18-‐22 carbon ω-‐9 monounsaturated fatty acids. The fatty acid binds to the N-‐termi

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8

Gangapatnam, Girija Lakshmi. "A NOVEL ROLE FOR dADAR ISOFORMS IN DROSOPHILA rnp-4f 5'-UTR INTRON SPLICING REGULATION." Miami University / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=miami1335963918.

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9

Lu, Jian. "The Kluyveromyces lactis killer toxin is a transfer RNA endonuclease." Doctoral thesis, Umeå : Department of Molecular Biology, Umeå Univ, 2007. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-1092.

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10

Seleem, Mohamed N. "Ochrobactrum anthropi: a soil bacterium for the study of Brucella virulence." Diss., Virginia Tech, 2006. http://hdl.handle.net/10919/28818.

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The species of Brucella were isolated and characterized almost 120 years ago and their genomes sequenced for almost 4 years. Compared to other bacterial pathogens relatively, little is known about the factors contributing to their persistence in hosts and multiplication within phagocytic cells. Also, many aspects of the interactions between Brucella and its host remain unclear. Molecular characterization of intracellular survival processes of Brucella will provide guidance for additional prevention and control measures. One of the features that distinguishes Brucella is that they do not expres

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11

Emara, Mohamed Maged. "Analysis of the Cellular Proteins, TIA-1 and TIAR, and their Interaction with the West Nile Virus (WNV) 3' SL Minus-Strand RNA." Digital Archive @ GSU, 2008. http://digitalarchive.gsu.edu/biology_diss/38.

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The 3' terminal stem loop of the WNV minus-strand [WNV3'(-) SL] RNA was previously shown to bind the cell protein, T-cell intracellular antigen-1 (TIA-1), and the related protein, TIAR. These two proteins are known to bind AU-rich sequences in the 3' UTRs of some cellular mRNAs. AU stretches are located in three single-stranded loops (L1, L2, and L3) of the WNV3'(-) SL RNA. The RNA binding activity of both proteins was reduced when L1 or L2, but not L3, AU sequences were deleted or substituted with Cs. Deletion or substitution with Cs of the entire AU-rich sequence in either L1 or L2 in a WNV

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12

Schnorrer, Frank, Pavel Tomancak, Cornelia Schönbauer, Radoslaw K. Ejsmont, and Christoph C. H. Langer. "In Vivo RNAi Rescue in Drosophila melanogaster with Genomic Transgenes from Drosophila pseudoobscura." Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2015. http://nbn-resolving.de/urn:nbn:de:bsz:14-qucosa-185372.

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Background Systematic, large-scale RNA interference (RNAi) approaches are very valuable to systematically investigate biological processes in cell culture or in tissues of organisms such as Drosophila. A notorious pitfall of all RNAi technologies are potential false positives caused by unspecific knock-down of genes other than the intended target gene. The ultimate proof for RNAi specificity is a rescue by a construct immune to RNAi, typically originating from a related species. Methodology/Principal Findings We show that primary sequence divergence in areas targeted by Drosophila melanogaste

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13

Schnorrer, Frank, Pavel Tomancak, Cornelia Schönbauer, Radoslaw K. Ejsmont, and Christoph C. H. Langer. "In Vivo RNAi Rescue in Drosophila melanogaster with Genomic Transgenes from Drosophila pseudoobscura." PloS, 2010. https://tud.qucosa.de/id/qucosa%3A29009.

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Background Systematic, large-scale RNA interference (RNAi) approaches are very valuable to systematically investigate biological processes in cell culture or in tissues of organisms such as Drosophila. A notorious pitfall of all RNAi technologies are potential false positives caused by unspecific knock-down of genes other than the intended target gene. The ultimate proof for RNAi specificity is a rescue by a construct immune to RNAi, typically originating from a related species. Methodology/Principal Findings We show that primary sequence divergence in areas targeted by Drosophila melanogaste

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14

Fetherson, Rebecca A. "An experimental and genomic approach to the regulation of alternative pre-mRNA splicing in Drosophila rnp-4f." Connect to this document online, 2005. http://rave.ohiolink.edu/etdc/view?acc%5Fnum=miami1114713461.

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Thesis (M.S.)--Miami University, Dept. of Zoology, 2005.<br>Title from first page of PDF document. Document formatted into pages; contains [1], ix, 75 p. : ill. Includes bibliographical references (p. 69-75).

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15

Declercq, Marion. "Host RNA degradation pathways and influenza A virus interplay : identification of a major role of the cellular exonuclease ERI1 in the influenza A virus life cycle." Thesis, Université de Paris (2019-....), 2019. https://theses.md.univ-paris-diderot.fr/DECLERCQ_Marion_va1.pdf.

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Les mécanismes de dégradation de l'ARN représentent un processus cellulaire central. En effet, ils contrôlent la stabilité et la qualité de l'ARN et, par conséquent, régulent l'expression des gènes. D’une part, la régulation de la stabilité des transcrits est un élément essentiel au maintien de l’homéostasie cellulaire mais aussi à l’établissement d’une réponse cellulaire adaptée en cas d’infection virale. D’autre part, le succès de l’infection virale dépend fortement de la capacité du virus à prendre le contrôle des machineries d’expression géniques cellulaires. De ce fait, les virus doivent

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16

Rakotondrafara, Aurélie Mamisoa. "Mechanism of cap-independent translation by long distance kissing stem-loop interactions in plant viral RNAs." [Ames, Iowa : Iowa State University], 2007.

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17

McManus, Charles Joel. "Functions of the yeast U6 RNA internal stem-loop during pre-messenger RNA splicing." 2007. http://www.library.wisc.edu/databases/connect/dissertations.html.

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18

HONG, DENNIS, and 洪瑞陽. "The characterization of stem-loop RNA binding protein-ribosomal protein P0." Thesis, 2000. http://ndltd.ncl.edu.tw/handle/11811307484196569079.

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碩士<br>國立成功大學<br>生物科技研究所<br>88<br>We obtained a mutant m4(-) derived from E.coli strain XL1-Blue MRF'' with a mutation in priB gene. The mutated priB gene contains a point mutation and leads to a single amino acid substitution from Phe to Val at position 77. However, no direct evidence has been described for the role of PriB in replication in vivo. The mutant strain m4(-) significantly reduces the copy number of the Δrop ColE1-type plasmid. With the feature of reducing plasmid copy number, we suppose PriB is a conserved protein in evolution. Using this mutant strain, we tried find ou

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19

Tan, Dazhi. "Molecular Basis for the Recognition of the Regulatory Stem-loop Structures in Eukaryotic Messenger RNAs." Thesis, 2014. https://doi.org/10.7916/D8FN14B4.

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Apart from carrying genetic information, RNAs also act as effectors of cellular processes through folding into intricate secondary and tertiary structures. The ubiquitous RNA structures in eukaryotic mRNAs, in collaboration with specific RNA-binding proteins, control many aspects of the post-transcriptional regulation of gene expression. However, the molecular bases for the recognition of these mRNA structures by their protein partners remain poorly understood due to the lack of structural information. This dissertation presents our structural studies on two protein-RNA complexes that b

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20

Fortner, David Michael. "A stem/loop in U6 RNA defines a conformational switch required for pre-mRNA splicing." 1995. http://catalog.hathitrust.org/api/volumes/oclc/32621470.html.

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Thesis (Ph. D.)--University of Wisconsin--Madison, 1994.<br>Typescript. eContent provider-neutral record in process. Description based on print version record. Includes bibliographical references (leaves 110-119).

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21

Liu, Hsuan. "Further Analysis of the Interaction of the Cellular Protein TIAR with the 3' Terminal Stem-Loop of the West Nile Virus (WNV) Minus-Strand RNA." 2013. http://scholarworks.gsu.edu/biology_diss/138.

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Cellular T-cell intracellular antigen-1 related protein (TIAR) binds to the 3' terminal stem-loop of the West Nile virus minus-strand RNA [WNV 3'(-) SL RNA]. TIAR binding sites were previously mapped on loop 1 (L1) and loop 2 (L2) of the 3' (-) SL RNA and mutations of these sites in a WNV infectious clone inhibited virus replication. In the present study, data from in vitro binding assays suggested that multiple TIAR proteins bind to each WNV 3′ (-) SL RNA in a positively cooperative manner. The tertiary structure of WNV 3′ (-) SL RNA was predicted and it was suggested that L2 forms an exposed

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22

Mardaryev, Andrei N., N. Meier, Krzysztof Poterlowicz, et al. "Lhx2 differentially regulates Sox9, Tcf4 and Lgr5 in hair follicle stem cells to promote epidermal regeneration after injury." 2011. http://hdl.handle.net/10454/6079.

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The Lhx2 transcription factor plays essential roles in morphogenesis and patterning of ectodermal derivatives as well as in controlling stem cell activity. Here, we show that during murine skin morphogenesis, Lhx2 is expressed in the hair follicle (HF) buds, whereas in postnatal telogen HFs Lhx2(+) cells reside in the stem cell-enriched epithelial compartments (bulge, secondary hair germ) and co-express selected stem cell markers (Sox9, Tcf4 and Lgr5). Remarkably, Lhx2(+) cells represent the vast majority of cells in the bulge and secondary hair germ that proliferate in response to skin injury

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