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Relevant bibliographies by topics / RNA viruses Plant viruses

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Academic literature on the topic 'RNA viruses Plant viruses'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

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Journal articles on the topic "RNA viruses Plant viruses"

1

Roossinck, Marilyn J. "Lifestyles of plant viruses." Philosophical Transactions of the Royal Society B: Biological Sciences 365, no. 1548 (June 27, 2010): 1899–905. http://dx.doi.org/10.1098/rstb.2010.0057.

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The vast majority of well-characterized eukaryotic viruses are those that cause acute or chronic infections in humans and domestic plants and animals. However, asymptomatic persistent viruses have been described in animals, and are thought to be sources for emerging acute viruses. Although not previously described in these terms, there are also many viruses of plants that maintain a persistent lifestyle. They have been largely ignored because they do not generally cause disease. The persistent viruses in plants belong to the family Partitiviridae or the genus Endornavirus . These groups also have members that infect fungi. Phylogenetic analysis of the partitivirus RNA-dependent RNA polymerase genes suggests that these viruses have been transmitted between plants and fungi. Additional families of viruses traditionally thought to be fungal viruses are also found frequently in plants, and may represent a similar scenario of persistent lifestyles, and some acute or chronic viruses of crop plants may maintain a persistent lifestyle in wild plants. Persistent, chronic and acute lifestyles of plant viruses are contrasted from both a functional and evolutionary perspective, and the potential role of these lifestyles in host evolution is discussed.

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Elena, S. F., P. Agudelo-Romero, P. Carrasco, F. M. Codoñer, S. Martín, C. Torres-Barceló, and R. Sanjuán. "Experimental evolution of plant RNA viruses." Heredity 100, no. 5 (February 6, 2008): 478–83. http://dx.doi.org/10.1038/sj.hdy.6801088.

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Goldbach, R. W. "Molecular Evolution of Plant RNA Viruses." Annual Review of Phytopathology 24, no. 1 (September 1986): 289–310. http://dx.doi.org/10.1146/annurev.py.24.090186.001445.

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ISHIKAWA, Masayuki. "Special issue: Plant viruses. Studies on the replication mechanisms of plant RNA viruses." Uirusu 44, no. 1 (1994): 3–10. http://dx.doi.org/10.2222/jsv.44.3.

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Wang, Ming-Bo, Chikara Masuta, Neil A. Smith, and Hanako Shimura. "RNA Silencing and Plant Viral Diseases." Molecular Plant-Microbe Interactions® 25, no. 10 (October 2012): 1275–85. http://dx.doi.org/10.1094/mpmi-04-12-0093-cr.

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RNA silencing plays a critical role in plant resistance against viruses, with multiple silencing factors participating in antiviral defense. Both RNA and DNA viruses are targeted by the small RNA-directed RNA degradation pathway, with DNA viruses being also targeted by RNA-directed DNA methylation. To evade RNA silencing, plant viruses have evolved a variety of counter-defense mechanisms such as expressing RNA-silencing suppressors or adopting silencing-resistant RNA structures. This constant defense–counter defense arms race is likely to have played a major role in defining viral host specificity and in shaping viral and possibly host genomes. Recent studies have provided evidence that RNA silencing also plays a direct role in viral disease induction in plants, with viral RNA-silencing suppressors and viral siRNAs as potentially the dominant players in viral pathogenicity. However, questions remain as to whether RNA silencing is the principal mediator of viral pathogenicity or if other RNA-silencing-independent mechanisms also account for viral disease induction. RNA silencing has been exploited as a powerful tool for engineering virus resistance in plants as well as in animals. Further understanding of the role of RNA silencing in plant–virus interactions and viral symptom induction is likely to result in novel anti-viral strategies in both plants and animals.

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Rao, A. L. N. "Genome Packaging by Spherical Plant RNA Viruses." Annual Review of Phytopathology 44, no. 1 (September 2006): 61–87. http://dx.doi.org/10.1146/annurev.phyto.44.070505.143334.

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Lai, M. M. "RNA recombination in animal and plant viruses." Microbiological Reviews 56, no. 1 (1992): 61–79. http://dx.doi.org/10.1128/mmbr.56.1.61-79.1992.

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Lai, M. M. "RNA recombination in animal and plant viruses." Microbiological Reviews 56, no. 1 (1992): 61–79. http://dx.doi.org/10.1128/mr.56.1.61-79.1992.

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Chare, Elizabeth R., and Edward C. Holmes. "Selection pressures in the capsid genes of plant RNA viruses reflect mode of transmission." Journal of General Virology 85, no. 10 (October 1, 2004): 3149–57. http://dx.doi.org/10.1099/vir.0.80134-0.

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To determine the selection pressures faced by RNA viruses of plants, patterns of nonsynonymous (d N) and synonymous (d S) substitution in the capsid genes of 36 viruses with differing modes of transmission were analysed. This analysis provided strong evidence that the capsid proteins of vector-borne plant viruses are subject to greater purifying selection on amino acid change than those viruses transmitted by other routes and that virus–vector interactions impose greater selective constraints than those between virus and plant host. This could be explained by specific interactions between capsid proteins and cellular receptors in the insect vectors that are necessary for successful transmission. However, contrary to initial expectations based on phylogenetic relatedness, vector-borne plant viruses are subject to weaker selective constraints than vector-borne animal viruses. The results suggest that the greater complexity involved in the transmission of circulative animal viruses compared with non-circulative plant viruses results in more intense purifying selection.

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Garcia-Ruiz, Hernan. "Susceptibility Genes to Plant Viruses." Viruses 10, no. 9 (September 10, 2018): 484. http://dx.doi.org/10.3390/v10090484.

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Plant viruses use cellular factors and resources to replicate and move. Plants respond to viral infection by several mechanisms, including innate immunity, autophagy, and gene silencing, that viruses must evade or suppress. Thus, the establishment of infection is genetically determined by the availability of host factors necessary for virus replication and movement and by the balance between plant defense and viral suppression of defense responses. Host factors may have antiviral or proviral activities. Proviral factors condition susceptibility to viruses by participating in processes essential to the virus. Here, we review current advances in the identification and characterization of host factors that condition susceptibility to plant viruses. Host factors with proviral activity have been identified for all parts of the virus infection cycle: viral RNA translation, viral replication complex formation, accumulation or activity of virus replication proteins, virus movement, and virion assembly. These factors could be targets of gene editing to engineer resistance to plant viruses.

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Dissertations / Theses on the topic "RNA viruses Plant viruses"

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Chare, Elizabeth R. "Recombination in RNA viruses and plant virus evolution." Thesis, University of Oxford, 2005. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.433381.

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Keese, Paul Konrad. "Structures of viroids and virusoids and their functional significance." Title page, contents and summary only, 1986. http://web4.library.adelaide.edu.au/theses/09PH/09phk268.pdf.

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Afsharifar, Alireza. "Characterisation of minor RNAs associated with plants infected with cucumber mosaic virus." Title page, table of contents and abstract only, 1997. http://web4.library.adelaide.edu.au/theses/09PH/09pha2584.pdf.

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Bibliography: leaves 127-138. This thesis studies the minor double stranded RNAs (dsRNA) and single stranded RNAs (ssRNA) which are consistently associated with plants infected with Q strain of cucumber mosaic virus (Q-CMV). The investigations are focused on the structural elucidation of new RNAs which have been observed in single stranded and double stranded RNA profiles of Q strain of CMV.

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Jeffries, Alex Craig. "The study at the molecular level of the New Zealand isolate of Lucerne transient streak sobemovirus and its satellite RNA." Title page, contents and summary only, 1993. http://web4.library.adelaide.edu.au/theses/09PH/09phj47.pdf.

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Naylor, Martin. "The effects of salicylic acid on RNA plant viruses." Thesis, University of Cambridge, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.624519.

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Rohozinski, J. "Studies of velvet tobacco mottle virus RNA replication by enzyme-template complexes in extracts from infected leaves /." Title page, contents and summary only, 1985. http://web4.library.adelaide.edu.au/theses/09PH/09phr738.pdf.

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Liu, Yuan Yi. "A study of a satellite RNA from arabis mosaic nepovirus." Thesis, University of Oxford, 1992. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.335830.

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Bustamante-Gallardo, Pedro. "Molecular studies on Rice hoja blanca virus (RHBV)." Thesis, University of East Anglia, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.338096.

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Sheldon, Candice Claire. "Hammerhead mediated self-cleavage of plant pathogenic RNAs /." Title page, contents and summary only, 1992. http://web4.library.adelaide.edu.au/theses/09PH/09phs544.pdf.

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Fei, Yue. "Investigating RNA silencing-mediated epigenetic modifications in virus-infected plants." Thesis, University of Edinburgh, 2018. http://hdl.handle.net/1842/33125.

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Plant viruses can cause many plant diseases, which result in substantial damage to crop production. To overcome viral infections, plants evolved RNA silencing which can recognise viral RNAs during their replications and slice them into small RNA (sRNA) using antiviral nucleases called DICER or Dicer-like (DCL). The resulting virus-derived small interfering RNA (vsiRNA, 21-24 nucleotides) then guides effector nucleases, namely ARGONAUTE (AGO), to cleave viral RNAs in the cytoplasm in a nucleotide-specific manner. However, the activity of vsiRNA is not restricted to the control of viral RNA accumulation. Virus-derived sRNAs can regulate host gene expression if host mRNAs share sequence complementarity with vsiRNAs. Interestingly, vsiRNAs are also able to target and methylate homologous DNA sequences in the nucleus indicating that vsiRNAs have potential to regulate endogenous genes at transcriptional level by modifying the epigenetic status of gene promoter sequences. This mechanism is referred to as transcriptional gene silencing (TGS). Thus, RNA silencing opens up new strategies to stably and heritably alter gene expression in plants. However, the mechanisms and efficacy of plant virus-induced TGS are largely unknown. The aim of my PhD was to investigate the molecular and environmental factors that are involved in virus-induced epigenetic modifications in the infected plants and in their progeny. First, I examined the required sequence complementary between sRNAs and their nuclear target sequence. I demonstrated for the first time that nuclear-imported vsiRNAs can induce RNA-directed DNA methylation (RdDM) and subsequently heritable virus-induced transcriptional gene silencing (ViTGS) even when they do not share 100% nucleotide sequence complementarity with the target DNA. This finding reveals a more dynamic interaction between viral RNAs and the host epigenome than previously thought. Secondly, I explored how environmental stimuli such as light and temperature can affect the efficacy of ViTGS. I found that ViTGS is greatly inhibited at high temperature. Using RNA-seq, I established that inefficient ViTGS at high temperature is due to the limited production of secondary sRNAs that may limit the initiation, amplification and spreading of virus-induced DNA methylation to neighbouring cells and down generations. Lastly, I studied the link between the viral suppressors of RNA silencing (VSRs): viral proteins that can interfere with plant RNA silencing and ViTGS. I established that VSRs of certain viruses can impair TGS in infected tissues, suggesting that viruses may alter the epigenome and consequently plant gene expression in the infected plants and their progeny. Collectively, my work reveals how viruses can re-program the epigenome of infected plants, and deepens our knowledge of how we can harness pathogens to modify the epigenome for plant breeding.

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Books on the topic "RNA viruses Plant viruses"

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Mandahar, C. L. Multiplication of RNA Plant Viruses. Dordrecht: Springer Netherlands, 2006. http://dx.doi.org/10.1007/1-4020-4725-8.

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Brinton, Margo A., Charles H. Calisher, and Roland Rueckert, eds. Positive-Strand RNA Viruses. Vienna: Springer Vienna, 1994. http://dx.doi.org/10.1007/978-3-7091-9326-6.

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Luo, Ming. Negative strand RNA virus. Singapore: World Scientific, 2011.

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Bridgen, Anne, ed. Reverse Genetics of RNA Viruses. Chichester, UK: John Wiley & Sons, Ltd, 2012. http://dx.doi.org/10.1002/9781118405338.

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Perez, Daniel R., ed. Reverse Genetics of RNA Viruses. New York, NY: Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-6964-7.

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Holland, John J., ed. Genetic Diversity of RNA Viruses. Berlin, Heidelberg: Springer Berlin Heidelberg, 1992. http://dx.doi.org/10.1007/978-3-642-77011-1.

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Francki, R. I. B., ed. The Plant Viruses. Boston, MA: Springer US, 1985. http://dx.doi.org/10.1007/978-1-4684-4937-2.

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Harrison, B. D., and A. F. Murant, eds. The Plant Viruses. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-1772-0.

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Van Regenmortel, M. H. V., and Heinz Fraenkel-Conrat, eds. The Plant Viruses. Boston, MA: Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-7026-0.

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Milne, Robert G., ed. The Plant Viruses. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4684-7038-3.

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Book chapters on the topic "RNA viruses Plant viruses"

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Koenig, R. "Polyhedral Plant Viruses with Monopartite RNA Genomes." In The Plant Viruses, 1–12. Boston, MA: Springer US, 1988. http://dx.doi.org/10.1007/978-1-4613-0921-5_1.

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Harrison, B. D., and A. F. Murant. "Plant Viruses with Bipartite RNA Genomes and Polyhedral Particles." In The Plant Viruses, 1–15. Boston, MA: Springer US, 1996. http://dx.doi.org/10.1007/978-1-4899-1772-0_1.

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Héricourt, François, Isabelle Jupin, and Anne-Lise Haenni. "Genome of RNA Viruses." In Molecular Biology of Plant Viruses, 1–28. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-5063-1_1.

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Nagy, Peter D. "Recombination in Plant RNA Viruses." In Plant Virus Evolution, 133–56. Berlin, Heidelberg: Springer Berlin Heidelberg, 2008. http://dx.doi.org/10.1007/978-3-540-75763-4_8.

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Morozov, Sergey, and Andrey Solovyev. "Genome Organization in RNA Viruses." In Molecular Biology of Plant Viruses, 47–98. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-5063-1_3.

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Ricci, Angela, Silvia Sabbadini, Laura Miozzi, Bruno Mezzetti, and Emanuela Noris. "Host-induced gene silencing and spray-induced gene silencing for crop protection against viruses." In RNAi for plant improvement and protection, 72–85. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789248890.0072.

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Abstract Since the beginning of agriculture, plant virus diseases have been a strong challenge for farming. Following its discovery at the very beginning of the 1990s, the RNA interference (RNAi) mechanism has been widely studied and exploited as an integrative tool to obtain resistance to viruses in several plant species, with high target-sequence specificity. In this chapter, we describe and review the major aspects of host-induced gene silencing (HIGS), as one of the possible plant defence methods, using genetic engineering techniques. In particular, we focus our attention on the use of RNAi-based gene constructs to introduce stable resistance in host plants against viral diseases, by triggering post-transcriptional gene silencing (PTGS). Recently, spray-induced gene silencing (SIGS), consisting of the topical application of small RNA molecules to plants, has been explored as an alternative tool to the stable integration of RNAi-based gene constructs in plants. SIGS has great and innovative potential for crop defence against different plant pathogens and pests and is expected to raise less public and political concern, as it does not alter the genetic structure of the plant.

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Ricci, Angela, Silvia Sabbadini, Laura Miozzi, Bruno Mezzetti, and Emanuela Noris. "Host-induced gene silencing and spray-induced gene silencing for crop protection against viruses." In RNAi for plant improvement and protection, 72–85. Wallingford: CABI, 2021. http://dx.doi.org/10.1079/9781789248890.0008.

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Abstract Since the beginning of agriculture, plant virus diseases have been a strong challenge for farming. Following its discovery at the very beginning of the 1990s, the RNA interference (RNAi) mechanism has been widely studied and exploited as an integrative tool to obtain resistance to viruses in several plant species, with high target-sequence specificity. In this chapter, we describe and review the major aspects of host-induced gene silencing (HIGS), as one of the possible plant defence methods, using genetic engineering techniques. In particular, we focus our attention on the use of RNAi-based gene constructs to introduce stable resistance in host plants against viral diseases, by triggering post-transcriptional gene silencing (PTGS). Recently, spray-induced gene silencing (SIGS), consisting of the topical application of small RNA molecules to plants, has been explored as an alternative tool to the stable integration of RNAi-based gene constructs in plants. SIGS has great and innovative potential for crop defence against different plant pathogens and pests and is expected to raise less public and political concern, as it does not alter the genetic structure of the plant.

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Bujarski, Jozef J. "Molecular Basis of Genetic Variability in RNA Viruses." In Molecular Biology of Plant Viruses, 121–41. Boston, MA: Springer US, 1999. http://dx.doi.org/10.1007/978-1-4615-5063-1_5.

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Goldbach, R., J. Wellink, J. Verver, A. Kammen, D. Kasteel, and J. Lent. "Adaptation of positive-strand RNA viruses to plants." In Positive-Strand RNA Viruses, 87–97. Vienna: Springer Vienna, 1994. http://dx.doi.org/10.1007/978-3-7091-9326-6_10.

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Truve, E., M. Kelve, A. Aaspôllu, A. Kuusksalu, P. Seppänen, and M. Saarma. "Principles and background for the construction of transgenic plants displaying multiple virus resistance." In Positive-Strand RNA Viruses, 41–50. Vienna: Springer Vienna, 1994. http://dx.doi.org/10.1007/978-3-7091-9326-6_5.

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Conference papers on the topic "RNA viruses Plant viruses"

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Tang, Ya-bing, Da Xing, De-bin Zhu, and Xiao-ming Zhou. "High sensitive method detection of plant RNA viruses by electrochemiluminescence reverse transcription PCR." In SPIE Proceedings, edited by Qingming Luo, Lihong V. Wang, Valery V. Tuchin, and Min Gu. SPIE, 2007. http://dx.doi.org/10.1117/12.741573.

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Belov, George. "COUPLING POLIOVIRUS RNA REPLICATION TO CELLULAR MEMBRANES." In Viruses: Discovering Big in Small. TORUS PRESS, 2019. http://dx.doi.org/10.30826/viruses-2019-12.

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Taliansky, Michael E., Jane Shaw, Antonida Makhotenko, Andrew J. Love, Natalia O. Kalinina, and Stuart MacFarlane. "PLANT-VIRUS INTERACTIONS: THE ROLE OF SUBNUCLEAR STRUCTURES." In Viruses: Discovering Big in Small. TORUS PRESS, 2019. http://dx.doi.org/10.30826/viruses-2019-11.

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Solovyev, Andrey. "NOVEL TRANSPORT MODULE IN A PLANT VIRUS GENOME INCLUDES HELICASE AND HYDROPHOBIC PROTEIN GENES." In Viruses: Discovering Big in Small. TORUS PRESS, 2019. http://dx.doi.org/10.30826/viruses-2019-05.

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Shimada, Takaharu, Tatsuya Hazemoto, Shunsuke Makino, Kouichi Hirata, and Kimihito Ito. "Finding correlated mutations among RNA segments in H3N2 influenza viruses." In 2012 Joint 6th Intl. Conference on Soft Computing and Intelligent Systems (SCIS) and 13th Intl. Symposium on Advanced Intelligent Systems (ISIS). IEEE, 2012. http://dx.doi.org/10.1109/scis-isis.2012.6505354.

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Yang, J., L. Kaphalia, W. J. Calhoun, and A. Brasier. "Long Noncoding RNA Regulates IRF1/IFNL Responses to Respiratory Viruses." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6168.

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Alvarez, Juan Manuel. "Management options for arthropod-transmitted plant viruses and limitations." In 2016 International Congress of Entomology. Entomological Society of America, 2016. http://dx.doi.org/10.1603/ice.2016.93163.

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Pershin, S. M., N. V. Tcherniega, A. F. Bunkin, E. K. Donchenko, O. V. Karpova, A. D. Kudryavtseva, T. V. Mironova, M. A. Strokov, M. A. Shevchenko, and K. I. Zemskov. "Laser Excitation of Coherent Gigahertz Vibrations in Plant Viruses." In 2018 International Conference Laser Optics (ICLO). IEEE, 2018. http://dx.doi.org/10.1109/lo.2018.8435836.

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Brewer, Wesley H., Franzine D. Smith, and John C. Sanford. "Information Loss: Potential for Accelerating Natural Genetic Attenuation of RNA Viruses." In Proceedings of the Symposium. WORLD SCIENTIFIC, 2013. http://dx.doi.org/10.1142/9789814508728_0015.

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Naumenko, Krystyna, Polina Zaremba, Svitlana Zagorodnya, and Yurii Shermolovych. "Antiviral activity of fluorinated compounds against DNA- and RNA-containing viruses." In 5th International Electronic Conference on Medicinal Chemistry. Basel, Switzerland: MDPI, 2019. http://dx.doi.org/10.3390/ecmc2019-06353.

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Reports on the topic "RNA viruses Plant viruses"

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Morris, T. J., and A. O. Jackson. Characterization of defective interfering RNAs associated with RNA plant viruses. Office of Scientific and Technical Information (OSTI), January 1993. http://dx.doi.org/10.2172/6880107.

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Morris, T. J., and A. O. Jackson. Characterization of defective interfering RNAs associated with RNA plant viruses. Progress report. Office of Scientific and Technical Information (OSTI), April 1993. http://dx.doi.org/10.2172/10139870.

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Davison, Michelle, Ruonan Wu, Vincent Danna, and Iobani Godinez. Uncovering novel RNA viruses in permafrost. Office of Scientific and Technical Information (OSTI), October 2020. http://dx.doi.org/10.2172/1776877.

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Sette, Alesandro, Bjoern Peters, and Martin Blythe. Predicting the Interplay of Epitope Recognition and Evolution in RNA Viruses Under Immune Pressure. Fort Belvoir, VA: Defense Technical Information Center, April 2008. http://dx.doi.org/10.21236/ada500852.

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ARIZONA STATE UNIV TEMPE CANCER RESEARCH INST. Discovery and Development of Therapeutic Drugs Against Lethal Human RNA Viruses: A Multidisciplinary Assault. Fort Belvoir, VA: Defense Technical Information Center, March 1992. http://dx.doi.org/10.21236/ada251561.

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Pettit, George R. Discovery and Development of Therapeutic Drugs against Lethal Human RNA Viruses: a Multidisciplinary Assault. Fort Belvoir, VA: Defense Technical Information Center, July 1991. http://dx.doi.org/10.21236/ada239742.

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Pettit, George R. Discovery and Development of Therapeutic Drugs against Lethal Human RNA- Viruses: A Multidisciplinary Assault. Fort Belvoir, VA: Defense Technical Information Center, February 1990. http://dx.doi.org/10.21236/ada219393.

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