Relevant bibliographies by topics / ROC1

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'ROC1'

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Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "ROC1"

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Donaldson, Timothy D., Maher A. Noureddine, Patrick J. Reynolds, William Bradford, and Robert J. Duronio. "Targeted Disruption of Drosophila Roc1b Reveals Functional Differences in the Roc Subunit of Cullin-dependent E3 Ubiquitin Ligases." Molecular Biology of the Cell 15, no. 11 (November 2004): 4892–903. http://dx.doi.org/10.1091/mbc.e04-03-0180.

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Cullin-dependent ubiquitin ligases regulate a variety of cellular and developmental processes by recruiting specific proteins for ubiquitin-mediated degradation. Cullin proteins form a scaffold for two functional modules: a catalytic module comprised of a small RING domain protein Roc1/Rbx1 and a ubiquitin-conjugating enzyme (E2), and a substrate recruitment module containing one or more proteins that bind to and bring the substrate in proximity to the catalytic module. Here, we present evidence that the three Drosophila Roc proteins are not functionally equivalent. Mutation of Roc1a causes lethality that cannot be rescued by expression of Roc1b or Roc2 by using the Roc1a promoter. Roc1a mutant cells hyperaccumulate Cubitus interruptus, a transcription factor that mediates Hedgehog signaling. This phenotype is not rescued by expression of Roc2 and only partially by expression of Roc1b. Targeted disruption of Roc1b causes male sterility that is partially rescued by expression of Roc1a by using the Roc1b promoter, but not by similar expression of Roc2. These data indicate that Roc proteins play nonredundant roles during development. Coimmunoprecipitation followed by Western or mass spectrometric analysis indicate that the three Roc proteins preferentially bind certain Cullins, providing a possible explanation for the distinct biological activities of each Drosophila Roc/Rbx.
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Wu, Kenneth, Serge Y. Fuchs, Angus Chen, Peilin Tan, Carlos Gomez, Ze'ev Ronai, and Zhen-Qiang Pan. "The SCFHOS/β-TRCP-ROC1 E3 Ubiquitin Ligase Utilizes Two Distinct Domains within CUL1 for Substrate Targeting and Ubiquitin Ligation." Molecular and Cellular Biology 20, no. 4 (February 15, 2000): 1382–93. http://dx.doi.org/10.1128/mcb.20.4.1382-1393.2000.

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ABSTRACT We describe a purified ubiquitination system capable of rapidly catalyzing the covalent linkage of polyubiquitin chains onto a model substrate, phosphorylated IκBα. The initial ubiquitin transfer and subsequent polymerization steps of this reaction require the coordinated action of Cdc34 and the SCFHOS/β-TRCP-ROC1 E3 ligase complex, comprised of four subunits (Skp1, cullin 1 [CUL1], HOS/β-TRCP, and ROC1). Deletion analysis reveals that the N terminus of CUL1 is both necessary and sufficient for binding Skp1 but is devoid of ROC1-binding activity and, hence, is inactive in catalyzing ubiquitin ligation. Consistent with this, introduction of the N-terminal CUL1 polypeptide into cells blocks the tumor necrosis factor alpha-induced and SCF-mediated degradation of IκB by forming catalytically inactive complexes lacking ROC1. In contrast, the C terminus of CUL1 alone interacts with ROC1 through a region containing the cullin consensus domain, to form a complex fully active in supporting ubiquitin polymerization. These results suggest the mode of action of SCF-ROC1, where CUL1 serves as a dual-function molecule that recruits an F-box protein for substrate targeting through Skp1 at its N terminus, while the C terminus of CUL1 binds ROC1 to assemble a core ubiquitin ligase.
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Maeda, Ichiro, Tomohiko Ohta, Hirotaka Koizumi, and Mamoru Fukuda. "In vitro ubiquitination of cyclin D1 by ROC1-CUL1 and ROC1-CUL3." FEBS Letters 494, no. 3 (April 10, 2001): 181–85. http://dx.doi.org/10.1016/s0014-5793(01)02343-2.

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Fukuchi, Minoru, Takeshi Imamura, Tomoki Chiba, Takanori Ebisawa, Masahiro Kawabata, Keiji Tanaka, and Kohei Miyazono. "Ligand-dependent Degradation of Smad3 by a Ubiquitin Ligase Complex of ROC1 and Associated Proteins." Molecular Biology of the Cell 12, no. 5 (May 2001): 1431–43. http://dx.doi.org/10.1091/mbc.12.5.1431.

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Smads are signal mediators for the members of the transforming growth factor-β (TGF-β) superfamily. Upon phosphorylation by the TGF-β receptors, Smad3 translocates into the nucleus, recruits transcriptional coactivators and corepressors, and regulates transcription of target genes. Here, we show that Smad3 activated by TGF-β is degraded by the ubiquitin–proteasome pathway. Smad3 interacts with a RING finger protein, ROC1, through its C-terminal MH2 domain in a ligand-dependent manner. An E3 ubiquitin ligase complex ROC1-SCFFbw1a consisting of ROC1, Skp1, Cullin1, and Fbw1a (also termed βTrCP1) induces ubiquitination of Smad3. Recruitment of a transcriptional coactivator, p300, to nuclear Smad3 facilitates the interaction with the E3 ligase complex and triggers the degradation process of Smad3. Smad3 bound to ROC1-SCFFbw1a is then exported from the nucleus to the cytoplasm for proteasomal degradation. TGF-β/Smad3 signaling is thus irreversibly terminated by the ubiquitin–proteasome pathway.
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Furukawa, Manabu, Yanping Zhang, Joseph McCarville, Tomohiko Ohta, and Yue Xiong. "The CUL1 C-Terminal Sequence and ROC1 Are Required for Efficient Nuclear Accumulation, NEDD8 Modification, and Ubiquitin Ligase Activity of CUL1." Molecular and Cellular Biology 20, no. 21 (November 1, 2000): 8185–97. http://dx.doi.org/10.1128/mcb.20.21.8185-8197.2000.

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ABSTRACT Members of the cullin and RING finger ROC protein families form heterodimeric complexes to constitute a potentially large number of distinct E3 ubiquitin ligases. We report here that the highly conserved C-terminal sequence in CUL1 is dually required, both for nuclear localization and for modification by NEDD8. Disruption of ROC1 binding impaired nuclear accumulation of CUL1 and decreased NEDD8 modification in vivo but had no effect on NEDD8 modification of CUL1 in vitro, suggesting that ROC1 promotes CUL1 nuclear accumulation to facilitate its NEDD8 modification. Disruption of NEDD8 binding had no effect on ROC1 binding, nor did it affect nuclear localization of CUL1, suggesting that nuclear localization and NEDD8 modification of CUL1 are two separable steps, with nuclear import preceding and required for NEDD8 modification. Disrupting NEDD8 modification diminishes the IκBα ubiquitin ligase activity of CUL1. These results identify a pathway for regulation of CUL1 activity—ROC1 and the CUL1 C-terminal sequence collaboratively mediate nuclear accumulation and NEDD8 modification, facilitating assembly of active CUL1 ubiquitin ligase. This pathway may be commonly utilized for the assembly of other cullin ligases.
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Furukawa, Manabu, and Yue Xiong. "BTB Protein Keap1 Targets Antioxidant Transcription Factor Nrf2 for Ubiquitination by the Cullin 3-Roc1 Ligase." Molecular and Cellular Biology 25, no. 1 (January 1, 2005): 162–71. http://dx.doi.org/10.1128/mcb.25.1.162-171.2005.

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ABSTRACT The concentrations and functions of many eukaryotic proteins are regulated by the ubiquitin pathway, which consists of ubiquitin activation (E1), conjugation (E2), and ligation (E3). Cullins are a family of evolutionarily conserved proteins that assemble by far the largest family of E3 ligase complexes. Cullins, via a conserved C-terminal domain, bind with the RING finger protein Roc1 to recruit the catalytic function of E2. Via a distinct N-terminal domain, individual cullins bind to a protein motif present in multiple proteins to recruit specific substrates. Cullin 3 (Cul3), but not other cullins, binds directly with BTB domains to constitute a potentially large number of BTB-CUL3-ROC1 E3 ubiquitin ligases. Here we report that the human BTB-Kelch protein Keap1, a negative regulator of the antioxidative transcription factor Nrf2, binds to CUL3 and Nrf2 via its BTB and Kelch domains, respectively. The KEAP1-CUL3-ROC1 complex promoted NRF2 ubiquitination in vitro and knocking down Keap1 or CUL3 by short interfering RNA resulted in NRF2 protein accumulation in vivo. We suggest that Keap1 negatively regulates Nrf2 function in part by targeting Nrf2 for ubiquitination by the CUL3-ROC1 ligase and subsequent degradation by the proteasome. Blocking NRF2 degradation in cells expressing both KEAP1 and NRF2 by either inhibiting the proteasome activity or knocking down Cul3, resulted in NRF2 accumulation in the cytoplasm. These results may reconcile previously observed cytoplasmic sequestration of NRF2 by KEAP1 and suggest a possible regulatory step between KEAP1-NRF2 binding and NRF2 degradation.
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Trupkin, Santiago A., Santiago Mora-García, and Jorge J. Casal. "The cyclophilin ROC1 links phytochrome and cryptochrome to brassinosteroid sensitivity." Plant Journal 71, no. 5 (July 6, 2012): 712–23. http://dx.doi.org/10.1111/j.1365-313x.2012.05013.x.

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Furukawa, Manabu, Yizhou Joseph He, Christoph Borchers, and Yue Xiong. "Targeting of protein ubiquitination by BTB–Cullin 3–Roc1 ubiquitin ligases." Nature Cell Biology 5, no. 11 (October 5, 2003): 1001–7. http://dx.doi.org/10.1038/ncb1056.

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Chen, Angus, Kenneth Wu, Serge Y. Fuchs, Peilin Tan, Carlos Gomez, and Zhen-Qiang Pan. "The Conserved RING-H2 Finger of ROC1 Is Required for Ubiquitin Ligation." Journal of Biological Chemistry 275, no. 20 (March 13, 2000): 15432–39. http://dx.doi.org/10.1074/jbc.m907300199.

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Huang, J., and J. Chen. "VprBP targets Merlin to the Roc1-Cul4A-DDB1 E3 ligase complex for degradation." Oncogene 27, no. 29 (March 10, 2008): 4056–64. http://dx.doi.org/10.1038/onc.2008.44.

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Dissertations / Theses on the topic "ROC1"

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Nai, Gisele Alborghetti [UNESP]. "O possível papel da proteína ROC1 na expressão da proteína ciclina D1 em melanomas cutâneos." Universidade Estadual Paulista (UNESP), 2009. http://hdl.handle.net/11449/104578.

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Fundação de Amparo à Pesquisa do Estado de São Paulo (FAPESP)
O aumento da expressão de ciclina D1, demonstrado em melanomas cutâneos, provavelmente está relacionado ao potencial invasivo do tumor. A diminuição da proteína ROC1, envolvida na degradação da ciclina D1, pode constituir uma alternativa para explicar o aumento desta proteína na ausência de superexpressão gênica. O objetivo deste estudo foi avaliar a relação da proteína ROC1 com a expressão de ciclina D1 em melanomas cutâneos. Foram estudados 62 casos de melanomas primários de pele e 58 nevos melanocíticos compostos. Realizou-se imuno-histoquímica com marcação para os anticorpos ciclina D1 e ROC1, e hibridação “in situ” fluorescente para avaliação da expressão do gene CCND1. Em 87,9% dos nevos melanocíticos, a expressão da proteína ROC1 foi evidenciada em mais de 50% das células, enquanto nos melanomas ocorreu em 45,2% dos casos (p=0,0014). A correlação entre a expressão da proteína ROC1 e da proteína ciclina D1 foi significativa e negativa em todos os casos estudados (p=0,0008985). Nos nevos melanocíticos, o aumento de expressão de ROC1 em relação à ciclina D1 ocorreu em 86,2% dos casos e nos melanomas em 45,2% (p<0,001). A relação ROC1/ciclina D1 não está associada à medida de Breslow (p=0,166), nem ao tipo histológico de melanoma (p=0,605). Entre os melanomas não amplificados, 50% daqueles que apresentaram expressão de ciclina D1 em mais de 50% das células mostraram expressão de proteína ROC1 em menos de 25% delas. A expressão da proteína ROC1 está correlacionada negativamente à expressão da proteína ciclina D1 nos melanomas, mostrando sua importância para degradação da ciclina D1 nestas neoplasias.
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Hu, Jian Xiong Yue. "Cell cycle and cell growth regulation by the CUL4-DDB1-ROC1 ubiquitin ligases." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2007. http://dc.lib.unc.edu/u?/etd,980.

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Thesis (Ph. D.)--University of North Carolina at Chapel Hill, 2007.
Title from electronic title page (viewed Dec. 18, 2007). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Biochemistry and Biophysics." Discipline: Biochemistry and Biophysics; Department/School: Medicine.
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Loos, Trina Jane. "Determining the Function of Nuclear Bmp4." BYU ScholarsArchive, 2010. https://scholarsarchive.byu.edu/etd/2586.

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Bone morphogenetic protein 4 (Bmp4) is a well known growth factor that regulates gene expression through the SMAD signaling pathway. Bmp4 is involved in many developmental processes and has been identified as an important factor in several cancers, including melanoma, ovarian cancer, and colon cancer. Madoz-Gurpide et al. recently observed Bmp4 in the nuclei of a minor percentage of cells in colon cancer tissues. In addition, our lab has recently discovered a nuclear variant of Bmp2 (nBmp2), the TGF-β family member most closely related to Bmp4. These observations led us to hypothesize that a nuclear variant of Bmp4 (nBmp4) also exists. The results of chapter one report the existence of a nuclear variant of Bmp4. nBmp4 is translated from an alternative start codon downstream of the signal peptide sequence which allows a bipartite nuclear localization signal to direct translocation of nBmp4 to the nucleus. Chapter 2 and 3 further report that nBmp4 interacts with several subunits in the SCF E3 ubiquitin ligase, namely two Regulator of Cullins (ROC) proteins, five Cullin proteins, and two F-box proteins. Due to the known role of the SCF E3 ubiquitin ligase in regulating the cell cycle, the effect of nBmp4 on cell cycle progression was analyzed and the results show that nBmp4 affects the cell cycle by causing cells to accumulate in G0/G1. The association of nBmp4 and the SCF E3 ubiquitin ligase components and the affect that nBmp4 has on the cell cycle suggest that nBmp4 functions in the nucleus by inhibiting the SCF E3 ubiquitin ligase from ubiquitinating target proteins that are involved in regulating cell cycle progression. Finally, the initial stages in the generation of an nBmp4 over-expression mouse are described. The results of this research clearly change the traditional paradigm that Bmp4 performs all of its functions via extracellular signaling and introduce the existence of a nuclear variant that is involved in cell cycle regulation.
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Nai, Gisele Alborghetti. "O possível papel da proteína ROC1 na expressão da proteína ciclina D1 em melanomas cutâneos /." Botucatu : [s.n.], 2009. http://hdl.handle.net/11449/104578.

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Orientador: Mariângela Esther Alencar Marques
Banca: Luciana Patrícia Fernandes Abbade
Banca: Luís Fernando Barbisan
Banca: Mírian Nacagami Sotto
Banca: Cleverson Teixeira Soares
Resumo: O aumento da expressão de ciclina D1, demonstrado em melanomas cutâneos, provavelmente está relacionado ao potencial invasivo do tumor. A diminuição da proteína ROC1, envolvida na degradação da ciclina D1, pode constituir uma alternativa para explicar o aumento desta proteína na ausência de superexpressão gênica. O objetivo deste estudo foi avaliar a relação da proteína ROC1 com a expressão de ciclina D1 em melanomas cutâneos. Foram estudados 62 casos de melanomas primários de pele e 58 nevos melanocíticos compostos. Realizou-se imuno-histoquímica com marcação para os anticorpos ciclina D1 e ROC1, e hibridação "in situ" fluorescente para avaliação da expressão do gene CCND1. Em 87,9% dos nevos melanocíticos, a expressão da proteína ROC1 foi evidenciada em mais de 50% das células, enquanto nos melanomas ocorreu em 45,2% dos casos (p=0,0014). A correlação entre a expressão da proteína ROC1 e da proteína ciclina D1 foi significativa e negativa em todos os casos estudados (p=0,0008985). Nos nevos melanocíticos, o aumento de expressão de ROC1 em relação à ciclina D1 ocorreu em 86,2% dos casos e nos melanomas em 45,2% (p<0,001). A relação ROC1/ciclina D1 não está associada à medida de Breslow (p=0,166), nem ao tipo histológico de melanoma (p=0,605). Entre os melanomas não amplificados, 50% daqueles que apresentaram expressão de ciclina D1 em mais de 50% das células mostraram expressão de proteína ROC1 em menos de 25% delas. A expressão da proteína ROC1 está correlacionada negativamente à expressão da proteína ciclina D1 nos melanomas, mostrando sua importância para degradação da ciclina D1 nestas neoplasias.
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Harris, William B. "The geologic history of Rock Canyon, Utah : a virtual trip /." CLICK HERE for online access, 2002. http://www.geology.byu.edu/faculty/rah/slides/Rock%20Canyon/Home.htm.

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Thesis (M.S.)--Brigham Young University. Dept. of Geology, 2002.
Web site works as of 02/10/03. Consult BYU Dept of Geology for URL changes in future. Includes bibliographical references (leaves 8-9). Also available via Internet.
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Papaliangas, T. T. "Shear behaviour of rock discontinuities and soil-rock interfaces." Thesis, University of Leeds, 1996. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.435675.

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Gatto, Vinicius Delangelo Martins. "Rock Progressivo e Punk Rock : Uma análise sociológica da mudança na vanguarda estética do campo do Rock." reponame:Repositório Institucional da UnB, 2011. http://repositorio.unb.br/handle/10482/9724.

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Dissertação (mestrado)—Universidade de Brasília, Instituto de Ciências Sociais, Departamento de Sociologia, 2011.
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Esta dissertação possui como objetivo explicar sociologicamente a transição na vanguarda estética do campo do Rock. Entre 1967 e 1977, o campo do Rock conheceu os períodos de maior sucesso comercial e de prestígio primeiramente do Rock Progressivo e em um segundo momento do Punk Rock. Que motivos sociológicos explicam esta transição? Considera-se aqui que a arte não é uma esferada ilhada e separada do restante da sociedade. Por outro lado, a arte pode possuir, sim, um aspecto mais autonônomo entretanto os desenvolvimentos nos campos artísticos são mediados pelos desenvolvimentos sociais mais gerais. É fundamental para este trabalho a noção de campo de Bourdieu. Se as transições nos campos artísticos são perpassados pelos desenvolvimentos sociais mais gerais isto quer dizer que cada movimento ou vanguarda artística tem de lidar necessariamente com o seu período histórico e com as condições materias objetivas de produção e do capital. Esta preocupação com a estrutura econômica é tratada com base no trabalho do téorico Fredric Jameson. Há uma preocupação em demonstrar desenvolvimentos micro sociais, entretanto estes desenvolvimentos são reconectados aos desenvolvimentos sociais mais gerais. Portanto as sociaibilidades, valores e sentimentos que se desenvolvem entre os atores do campo do Rock e que são analisados neste trabalho são limitados por uma situação objetiva comum de cada tempo histórico. Como conclusão procura-se demonstrar que cada tempo histórico, Modernidade e Pós-Modernidade e cada lógica cultural correspondente modernismo e pós-modernismo mediaram os desenvolvimentos no campo do Rock. ______________________________________________________________________________ ABSTRACT
This work has as main objective to explain in sociological terms the transition in the aesthetics vanguard of the Rock field. The Rock field experienced between 1967 and 1977, first the apogee of Progressive Rock and then the apogee of Punk Rock. Which sociological concepts do explain this transiton? It´s considered in this work that art is not a separated island from the rest of society. The fields of art may be autonomous in a certain way but, this autonomy is not self complet, this means that the developments in the artistic fields are mediated by the social developments in general. It is fundamental for this work the notion of field as conceived by Bourdieu. If the developments in the artistic field are transpassed by the more general social developments, this means that every artistic vanguard has to deal, necessarily, with his historic time and with the material condition of a certain given time. This concern about the economic structure is analysed according the work of the theorist Fredric Jameson. There is a concern about micro social developments, but these developments are reconected to the social developments in capital and society. So, the sociabilitys, values and feelings among the actors of the Rock field are limitades by a given situation of each historic time. As conclusion, I intend to demonstrate that each historic time, Modernity and Post Modernity and each corresponding cultural logic, modernism and post modernism have mediated the developments in the Rock field.
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Melendez, Elisa M. "For Those About to Rock: Gender Codes in the Rock Music Video Games Rock Band and Rocksmith." FIU Digital Commons, 2018. https://digitalcommons.fiu.edu/etd/3685.

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This dissertation explores gender codes within the intersection of two American pop culture staples, video games and rock music, by conducting a feminist analysis of two video games (Rock Band and Rocksmith). Both video games and rock music have had their share of feminist academic critique: Musicologists point out how lack of canonical inclusion, gendered attitudes towards instruments, and messages from supporting media create an unwelcome environment for women to pursue a rock music career. Game studies scholars have examined similar attitudes, including a lack of women represented in both the video games and the studios that create them. Through a mix of creator and player interviews, participant observation, content analysis, and autoethnography, I look at the intersection of these two literatures (the rock music video game) to see how gender is hard-coded into the game, and what opportunities, if any, exist for subversion of societal and industry gender norms. Through not just looking at the game as text, I present a more “thick description” of a video game that takes into account the creators of the games, the players that play them, and a researcher that occupies multiple identities within the space. I argue that, in an effort to replicate an authentic rock musician experience in a video game, Rock Band and Rocksmith often replicate a lot of these gendered messages. The games’ text and set list emphasize a male-centric rock music canon. Rocksmith’s original whiskey-soaked visual design and marketing skew heavily towards an older male demographic. However, resistances to these codes exist in both the players who defy expectations by showing up to perform and compete, as well as the creators who actively work to make these games more inclusive via changes to future games as well as inclusive hiring practices, marketing, and music sourcing (with varying degrees of success).
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Konoske, Ashley Anderson. "The archaeology and rock art of Rock Creek, northwestern Nevada /." abstract and full text PDF (free order & download UNR users only), 2006. http://0-gateway.proquest.com.innopac.library.unr.edu/openurl?url_ver=Z39.88-2004&rft_val_fmt=info:ofi/fmt:kev:mtx:dissertation&res_dat=xri:pqdiss&rft_dat=xri:pqdiss:1436190.

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Thesis (M.A.)--University of Nevada, Reno, 2006.
"May, 2006." Includes bibliographical references (leaves 241-257). Library also has microfilm. Ann Arbor, Mich. : ProQuest Information and Learning Company, [2006]. 1 microfilm reel ; 35 mm. Online version available on the World Wide Web.
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Pine, R. J. "Rock joint and rock mass behaviour during pressurised hydraulic injections." Thesis, University of Exeter, 1986. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.374872.

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The hydro-mechanical effects of high pressure fluid injections into jointed rock are considered mostly in the context of Hot Dry Rock (HDR) geothermal energy systems. In Part I, the mai n aspects ari sing from the HDR research at the "Camborne School of Mines (CSM) and Los Alamos Nat iana 1 Laboratory (LANL) projects are reviewed. Previous approaches to fluid-rock interacti ons at these projects and important observed phenomena are highlighted. Fundamental aspects of rock joint geometry, mechanical behaviour and flow regimes within jointed rock are also reviewed. These aspects are then related to possible conditions in HDR systems. The role of in situ stress conditions is of great significance in this study and is reviewed theoretically and in detail for both the CSM and LANL project sites. The revi ew incl udes a comprehensi ve seri es of measurements, by different techniques, organised and interpreted by the author at the CSM project. In Part II, model development, the emphasis is on intermediate fluid pressures which are too high for simple diffusion alone and too low for tensile hydraulic fracturing. The dominant mechanical activity is one of joint shear. Strike-slip shearing due to fluid injection is examined in two dimensions with the numerical model FRIP, which has been extended by the author. Similar behaviour is examined in three dimensions with an analytical model which is linked to microseismic observations. This model explains the observed phenomenon of downward shear growth. Joint distribution and mechanical properties, and their effect on fluid diffusivity, are examined and used in analytical models of fluid pressure pulse propagation, tracer transport, and rock stress increment transfer. All models are used to help interpret field data, mainly from the CSM project. The models are also of potential application to hydrocarbon reservoir stimulation, liquid waste disposal and leakage from high pressure water tunnels.
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Books on the topic "ROC1"

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ida, Miroslav Str. C eskoslovenska geograficka literatura v roce 1985: Bibliograficka roc enka. Brno: C eskoslovenska Akademie ve dGeograficky U stav, 1986.

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Rock Island rock. Las Vegas, NV: Thomas & Mercer, 2013.

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Chorao, Kay. Rock, rock, my baby. [New York]: Random House, 1993.

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Rock. Edina, Minn: ABDO Pub. Co., 2011.

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Mayer, Cassie. Rock. Chicago, Ill: Heinemann Library, 2008.

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Mayer, Cassie. Rock. Chicago, Ill: Heinemann Library, 2008.

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Moore, Tom. Rock. [S. l: s. n., 2000.

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Vega, Angel de. Rocs. Barcelona: Editorial Empúries, 2001.

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Vega, Àngel de. Rocs. Barcelona: Editorial Empúries, 2001.

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Rock. Chicoutimi [Québec]: JCL, 1988.

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Book chapters on the topic "ROC1"

1

Van Valin Jr., Robert D. "Role and reference grammar." In Handbook of Pragmatics, 1–13. Amsterdam: John Benjamins Publishing Company, 2009. http://dx.doi.org/10.1075/hop.13.rol1.

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Hecken, Thomas. "Rock." In Handbuch Popkultur, 35–43. Stuttgart: J.B. Metzler, 2017. http://dx.doi.org/10.1007/978-3-476-05601-6_6.

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Martemyanov, Kirill A., Pooja Parameswaran, Irene Aligianis, Mark Handley, Marga Gual-Soler, Tomohiko Taguchi, Jennifer L. Stow, et al. "ROCK1." In Encyclopedia of Signaling Molecules, 1690. New York, NY: Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101199.

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Dorf, Samuel N., Heather MacLachlan, and Julia Randel. "Rock." In Anthology to Accompany Gateways to Understanding Music, 416–20. New York : Routledge, 2021.: Routledge, 2020. http://dx.doi.org/10.4324/9781003041542-52.

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Arndt, Nicholas. "Rock." In Encyclopedia of Astrobiology, 1479. Berlin, Heidelberg: Springer Berlin Heidelberg, 2011. http://dx.doi.org/10.1007/978-3-642-11274-4_1384.

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Spunt, Barry. "Rock." In Heroin and Music in New York City, 65–101. New York: Palgrave Macmillan US, 2014. http://dx.doi.org/10.1057/9781137314291_3.

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Wiersam, Dirk J. "Rock." In Magic of Minerals and Rocks, 24–43. Berlin, Heidelberg: Springer Berlin Heidelberg, 2004. http://dx.doi.org/10.1007/978-3-642-18695-0_3.

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Arndt, Nicholas. "Rock." In Encyclopedia of Astrobiology, 1. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27833-4_1384-3.

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Samuel, Michael S., and Michael F. Olson. "ROCK." In Encyclopedia of Signaling Molecules, 4746–51. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_328.

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Arndt, Nicholas. "Rock." In Encyclopedia of Astrobiology, 2212. Berlin, Heidelberg: Springer Berlin Heidelberg, 2015. http://dx.doi.org/10.1007/978-3-662-44185-5_1384.

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Conference papers on the topic "ROC1"

1

"ROCS Workshop 2005 (formerly the GaAs REL Workshop) Proceedings." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201543.

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"ROCS Workshop 2005 (formerly the GaAs REL Workshop) Proceedings." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201544.

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"Copyright page." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201545.

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"Committee." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201546.

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"2005 ROCS workshop program." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201548.

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"[Breaker page]." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201550.

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"[Breaker page]." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201554.

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"[Breaker page]." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201558.

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van der Wel, P. J., R. A. van den Heuvel, H. J. F. Peuscher, Y. Li, J. G. Gommans, F. van Rijs, P. Bron, and S. J. C. H. Theeuwen. "Application of aluminium metallisation in ldmos RF power applications." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201561.

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"[Breaker page]." In ROCS Workshop, 2005. IEEE, 2005. http://dx.doi.org/10.1109/rocs.2005.201562.

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Reports on the topic "ROC1"

1

Grosjean, E., D. S. Edwards, Z. Hong, N. Jinadasa, and T. Buckler. TOC and Rock-Eval pyrolysis data from the well Roc 2, Roebuck Basin, Australia. Geoscience Australia, 2019. http://dx.doi.org/10.11636/record.2019.012.

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Lyons, Anthony P. Scattering from Rock and Rock Outcrops. Fort Belvoir, VA: Defense Technical Information Center, September 2014. http://dx.doi.org/10.21236/ada618019.

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Tweddale, Scott A. Rock River Geographic Information System: ROCK-GIS (User Manual). Fort Belvoir, VA: Defense Technical Information Center, April 2004. http://dx.doi.org/10.21236/ada430945.

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Ghosh, A., S. M. Hsiung, and A. H. Chowdhury. Seismic response of rock joints and jointed rock mass. Office of Scientific and Technical Information (OSTI), June 1996. http://dx.doi.org/10.2172/270673.

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Myers, Mary, and Allison Arnold. Black Rock Sanctuary. Landscape Architecture Foundation, 2012. http://dx.doi.org/10.31353/cs0350.

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Wang, H. F. Poroelasticity of rock. Office of Scientific and Technical Information (OSTI), March 1992. http://dx.doi.org/10.2172/5598301.

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C. Lum. Rock Properties Model. Office of Scientific and Technical Information (OSTI), September 2004. http://dx.doi.org/10.2172/838644.

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G.H. Nieder-Westermann. ROCK PROPERTIES MODEL. Office of Scientific and Technical Information (OSTI), February 2005. http://dx.doi.org/10.2172/841284.

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Das, B. Rock characterization microhardness technique. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1986. http://dx.doi.org/10.4095/304894.

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Choquet, P. Rock bolting practical guide. Natural Resources Canada/ESS/Scientific and Technical Publishing Services, 1991. http://dx.doi.org/10.4095/305079.

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