Academic literature on the topic 'RORα'

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Journal articles on the topic "RORα"

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Kang, Hong Soon, Martin Angers, Ju Youn Beak та ін. "Gene expression profiling reveals a regulatory role for RORα and RORγ in phase I and phase II metabolism". Physiological Genomics 31, № 2 (2007): 281–94. http://dx.doi.org/10.1152/physiolgenomics.00098.2007.

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Retinoid-related orphan receptors alpha (RORα) and gamma (RORγ) are both expressed in liver; however, their physiological functions in this tissue have not yet been clearly defined. The RORα1 and RORγ1 isoforms, but not RORα4, show an oscillatory pattern of expression during circadian rhythm. To obtain insight into the physiological functions of ROR receptors in liver, we analyzed the gene expression profiles of livers from WT, RORα-deficient staggerer (sg) mice ( RORα sg/sg), RORγ−/−, and RORα sg/sg RORγ−/− double knockout (DKO) mice by microarray analysis. DKO mice were generated to study fu
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Park, Su, Il-Geun Park, Hyunkyung Kim та Ji Lee. "N-Terminal Domain Mediated Regulation of RORα1 Inhibits Invasive Growth in Prostate Cancer". International Journal of Molecular Sciences 20, № 7 (2019): 1684. http://dx.doi.org/10.3390/ijms20071684.

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Four members of the retinoic acid-related orphan receptor α (RORα) family (RORα1, RORα2, RORα3 and RORα4) are transcription factors that regulate several processes including circadian rhythm, lipid metabolism, cerebellar development, immune function, and cancer. Only two isoforms, RORα1 and 4, are specifically co-expressed in the murine and human. In the present study, we identified a specific N-terminal domain (NTD) of RORα1 that potentiated the downregulation of target genes involved in tumor progression and proliferation, based on results from RORα-deficient mouse embryonic fibroblasts and
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Jetten, Anton M. "Retinoid-Related Orphan Receptors (RORs): Critical Roles in Development, Immunity, Circadian Rhythm, and Cellular Metabolism." Nuclear Receptor Signaling 7, no. 1 (2009): nrs.07003. http://dx.doi.org/10.1621/nrs.07003.

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The last few years have witnessed a rapid increase in our knowledge of the retinoid-related orphan receptors RORα, -β, and -γ (NR1F1-3), their mechanism of action, physiological functions, and their potential role in several pathologies. The characterization of ROR-deficient mice and gene expression profiling in particular have provided great insights into the critical functions of RORs in the regulation of a variety of physiological processes. These studies revealed that RORa plays a critical role in the development of the cerebellum, that both RORα and RORβ are required for the maturation of
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Wada, Taira, Hong Soon Kang, Anton M. Jetten та Wen Xie. "The Emerging Role of Nuclear Receptor RORα and Its Crosstalk with LXR in Xeno- and Endobiotic Gene Regulation". Experimental Biology and Medicine 233, № 10 (2008): 1191–201. http://dx.doi.org/10.3181/0802-mr-50.

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Retinoid-related orphan receptors (RORs), including the α, β and γ isoforms (NR1F1–3), are orphan nuclear receptors that have been implicated in tissue development, immune responses, and circadian rhythm. Although RORα and RORγ have been shown to be expressed in the liver, the hepatic function of these two RORs remains unknown. We have recently shown that loss of RORα and/or RORγ can positively or negatively influence the expression of multiple Phase I and Phase II drug metabolizing enzymes and transporters in the liver. Among ROR responsive genes, we identified oxysterol 7α-hydroxylase (Cyp7b
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CHAUVET, Caroline, Brigitte BOIS-JOYEUX та Jean-Louis DANAN. "Retinoic acid receptor-related orphan receptor (ROR) α4 is the predominant isoform of the nuclear receptor RORα in the liver and is up-regulated by hypoxia in HepG2 human hepatoma cells". Biochemical Journal 364, № 2 (2002): 449–56. http://dx.doi.org/10.1042/bj20011558.

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The retinoic acid receptor-related orphan receptor α (RORα) is critically involved in many physiological functions in several organs. We find that the main RORα isoform in the mouse liver is the RORα4 isoform, in terms of both mRNA and protein levels, while the RORα1 isoform is less abundant. Because hypoxia is a major feature of liver physiology and pathology, we examined the effect of this stress on Rora gene expression and RORα transcriptional activity. HepG2 human hepatoma cells were cultured for 24h under normoxia (20% O2) or hypoxia (10, 2, and 0.1% O2) and the abundance of the Rora tran
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Nematisouldaragh, Darya, Eryn Kirshenbaum, Michael Uzonna, Lorrie Kirshenbaum, and Inna Rabinovich-Nikitin. "The Role of Retinoic-Acid-Related Orphan Receptor (RORs) in Cellular Homeostasis." International Journal of Molecular Sciences 25, no. 21 (2024): 11340. http://dx.doi.org/10.3390/ijms252111340.

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Retinoic-acid-related orphan receptors (RORs) are transcription factors belonging to the nuclear receptor subfamily consisting of RORα, RORβ, and RORγ. By binding to the ROR response elements (ROREs) on target gene promoters, RORs regulate a wide variety of cellular processes, including autophagy, mitophagy, oxidative stress, and inflammation. The regulatory roles of RORs are observed in cardiac cells, hepatocytes, pulmonary epithelial cells, renal cells, immune cells, and cancer cells. A growing body of clinical and experimental evidence suggests that ROR expression levels are markedly reduce
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Koibuchi, Noriyuki, Ying Liu, Harumi Fukuda, Akira Takeshita, Paul M. Yen та William W. Chin. "RORα Augments Thyroid Hormone Receptor-Mediated Transcriptional Activation*". Endocrinology 140, № 3 (1999): 1356–64. http://dx.doi.org/10.1210/endo.140.3.6562.

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Abstract This study is designed to clarify the role of an orphan nuclear hormone receptor, RORα, on thyroid hormone (TH) receptor (TR)-mediated transcription on a TH-response element (TRE). A transient transfection study using various TREs [i.e., F2 (chick lysozyme TRE), DR4 (direct repeat), and palindrome TRE] and TR and RORα1 was performed. When RORα1 and TR were cotransfected into CV1 cells, RORα1 enhanced the transactivation by liganded-TR on all TREs tested without an effect on basal repression by unliganded TR. By electrophoretic mobility shift assay, on the other hand, although RORα bou
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Song, Hyerin, Jung Woong Chu, Su Chan Park та ін. "Isoform-Specific Lysine Methylation of RORα2 by SETD7 Is Required for Association of the TIP60 Coactivator Complex in Prostate Cancer Progression". International Journal of Molecular Sciences 21, № 5 (2020): 1622. http://dx.doi.org/10.3390/ijms21051622.

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The retinoid acid-related orphan receptor α (RORα), a member of the orphan nuclear receptor superfamily, functions as an unknown ligand-dependent transcription factor. RORα was shown to regulate a broad array of physiological processes such as Purkinje cell development in the cerebellum, circadian rhythm, lipid and bone metabolism, inhibition of inflammation, and anti-apoptosis. The human RORα gene encodes at least four distinct isoforms (RORα1, -2, -3, -4), which differ only in their N-terminal domain (NTD). Two isoforms, RORα2 and 3, are not expressed in mice, whereas RORα1 and 4 are express
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Zhang, Yijia, Zixu Wang, Yulan Dong, Jing Cao та Yaoxing Chen. "Melatonin Nuclear Receptors Mediate Green-and-Blue-Monochromatic-Light-Combinations-Inhibited B Lymphocyte Apoptosis in the Bursa of Chickens via Reducing Oxidative Stress and Nfκb Expression". Antioxidants 11, № 4 (2022): 748. http://dx.doi.org/10.3390/antiox11040748.

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Previous studies found that melatonin modulates a combination of green-and-blue-light-induced B-lymphocyte proliferation via its membrane receptors Mel1a and Mel1c. However, in addition to its membrane-bound receptors, melatonin also functions through binding to nuclear receptors RORα/RORβ/RORγ. In this study, we raised 120 chicks under 400–700 nm white (WW), 660 nm red (RR), 560 nm green (GG) and 480 nm blue light (BB) from P0 to P26. From P27 to P42, half of the chickens in green, blue and red were switched to blue (G→B), green (B→G) and red (R→B), respectively. We used immunohistochemistry,
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Qiu, Chun-Hong, Noriaki Shimokawa, Toshiharu Iwasaki, Ishwar S. Parhar та Noriyuki Koibuchi. "Alteration of Cerebellar Neurotropin Messenger Ribonucleic Acids and the Lack of Thyroid Hormone Receptor Augmentation by staggerer-Type Retinoic Acid Receptor-Related Orphan Receptor-α Mutation". Endocrinology 148, № 4 (2007): 1745–53. http://dx.doi.org/10.1210/en.2006-1131.

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The mutant mouse staggerer (sg) harbors a deletion within the gene encoding the retinoic acid receptor-related orphan receptor-α (RORα). Homozygotes show aberrant cerebellar development. However, the mechanisms responsible for the cerebellar defect are still poorly understood. In the present study, the involvement of neurotropins (NTs), including nerve growth factor, brain-derived neurotropic factor, NT-3 and NT-4/5, and their receptors, which play a crucial role in brain development, on the cerebellar defects of sg mice was studied by semiquantitative RT-PCR and in situ hybridization histoche
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Dissertations / Theses on the topic "RORα"

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Nikitin, Artemii. "Role of nuclear receptor RORα in regulatory T cells". Thesis, Université de Lille (2018-2021), 2019. http://www.theses.fr/2019LILUS073.

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Les facteurs de transcription de la superfamille des récepteurs nucléaires jouent de multiples rôles dans le développement et la fonction des lymphocytes T régulateurs (TREG). Les TREG sont des cellules régulatrices/suppressives qui contrôlent les réponses d’autres types cellulaires et l’homéostasie locale des tissus.Comme les TREG sont actives au sein de divers organes, tant à l’homéostasie qu’en conditions inflammatoires,ils doivent répondre à la fois aux contexte local au sein du tissus et à un environnement immunologiquement agressif tout en préservant leurs propriétés tolérogéniques au co
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Boukhtouche, Fatiha. "Rôle du récepteur nucléaire RORα dans la survie et la différenciationneuronale". Phd thesis, Université Pierre et Marie Curie - Paris VI, 2006. http://tel.archives-ouvertes.fr/tel-00080417.

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RORα (Retinoic acid receptor related Orphan Receptor α) est un récepteur nucléaire<br />dont la perte de fonction entraîne chez la souris staggerer - entre autres phénotypes - une<br />sévère ataxie cérébelleuse. RORα a longtemps été considéré comme un récepteur nucléaire<br />orphelin, cependant, le cholestérol (ou un de ses dérivés) semble être un ligand physiologique<br />de ce récepteur.<br />Le mutant staggerer, identifié dès 1962 par Sidman, Lane et Dickie, présente une<br />hypoplasie cérébelleuse liée à l'absence de la grande majorité des cellules de Purkinje, ainsi<br />que des grains
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Journiac, Nathalie. "Etude du rôle du facteur de transcription RORα dans la réaction gliale". Paris 6, 2007. http://www.theses.fr/2007PA066031.

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Notre objectif était de déterminer le rôle de RORalpha dans l’activation des astrocytes associée à une neurodégénérescence. Nous avons utilisé la souris staggerer exprimant une mutation perte de fonction dans le gène Rora à l’origine de la dégénérescence des neurones cérébelleux et réalisé des transfections stables de lignées astrocytaires. Nous avons montré que RORalpha, jusqu’alors considéré comme exclusivement neuronal, est exprimé dans les astrocytes et démontré qu’il module : -l’expression de la GFAP, un marqueur spécifique de l’activation astrocytaire. -la prolifération, avec un effet di
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Boukhtouche, Fatiha. "Rôle du récepteur nucléaire RORα dans la survie et la différentiation des neurones". Paris 6, 2006. http://www.theses.fr/2006PA066007.

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Sermikli, Benan Pelin. "Role of the nuclear receptor RORα expressed by myeloid cells in metabolic diseases". Thesis, Lille 2, 2020. http://www.theses.fr/2020LIL2S019.

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Retinoic acid receptor-related orphan receptor-alpha (RORa) is a transcription factor from the nuclearreceptor superfamily expressed by immune and non-immune cells involved in the regulation of obesity, insulin resistance (IR) and non-alcoholic steatohepatitis (NASH). Cholesterol, cholesterol-sulfate, 7-oxygenated sterols and oxysterols have been identified as potential endogenous RORa ligands. Tobetter understand the mechanisms of macrophage-expressed RORa regulation in obesity and IR, we generated a macrophage-specific RORa-deficient (MKO) mouse line by using a LysM-Cre and floxedRORa mouse
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Ermisch, Michael [Verfasser], Dieter [Akademischer Betreuer] Steinhilber та Rolf [Akademischer Betreuer] Marschalek. "Regulation der nukleären Rezeptoren RORα und REVERBα / Michael Ermisch. Gutachter: Dieter Steinhilber ; Rolf Marschalek". Frankfurt am Main : Univ.-Bibliothek Frankfurt am Main, 2011. http://d-nb.info/1044274638/34.

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Gineste, Romain. "Régulation de l'activité transcriptionnelle des récepteurs nucléaires FXR, RORα et PPARγ par modifications post-traductionnelles". Lille 2, 2008. http://www.theses.fr/2008LIL2S041.

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Les facteurs de transcriptions FXR, RORα et PPARγ appartiennent à la superfamille des récepteurs nucléaires (RN). Ils ont pour fonction de moduler l'expression de nombreux gènes impliqués dans de nombreux processus physiologiques. Leurs rôles dans l'homéostasie glucidique, lipidique et énergétique font de ces récepteurs des cibles pharmacologiques d'intérêt pour le traitement de nombreuses maladies métaboliques comme les dyslipidémies, le diabète de type II, l'athérosclérose, l'obésité et les maladies neuro-dégénératives. Les RN activent leurs gènes cibles en se liant à l'ADN, sur une séquence
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Kadiri, Sarah. "Inflammation du tissu adipeux et dysfonctions de l'homéostasie lipidique : différences spécifiques de dépôts chez l'humain et la souris, et implication RORα". Paris 6, 2013. http://www.theses.fr/2013PA066518.

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Le tissu adipeux (TA) joue un rôle central dans l’homéostasie lipidique par ses capacités de stockage/déstockage des acides gras (AG). La voie de la glycéronéogenèse (GNG), qui permet la ré-estérification des acides gras en période de jeûne, participe à l’homéostasie lipidique en limitant la lipotoxicité des AG et leur localisation ectopique. Nous avons traité des explants de TA sous-cutané (TASC) et viscéral (TAV) issus de femmes minces par des inhibiteurs de la protéase du VIH (en cause dans les lipodystrophies) ou le lipopolysaccharide. Mes travaux ont mis en évidence une atteinte du TASC e
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Kagwiria, Rosebeth [Verfasser], та Lars [Gutachter] Nitschke. "Evaluation of the nuclear receptor Retinoic Acid Receptor related Orphan Receptor α (RORα) as a regulator of osteoclastogenesis in rheumatoid arthritis and osteoporosis / Rosebeth Kagwiria ; Gutachter: Lars Nitschke". Erlangen : Friedrich-Alexander-Universität Erlangen-Nürnberg (FAU), 2017. http://d-nb.info/1140228609/34.

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Boyault, Sandrine. "Modulation des effets et de la production de l'interleukine-1ß par des ligands des récepteurs nucléaires : PPAR, "peroxisome proliferator-activated receptor" et RORα "retinoic acid receptor-related orphan receptor alpha" dans les tissus de l'articulation". Nancy 1, 2003. http://docnum.univ-lorraine.fr/public/SCD_T_2003_0245_BOYAULT.pdf.

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La destruction progressive du cartilage, caractéristique commune des arthropathies inflammatoires et dégénératives, résulte d 'un déséquilibre entre les processus de synthèse et de dégradation matricielle. L'interleukine-l (IL-1) joue un rôle central dans la perte d 'homéostasie du cartilage lors de ces pathologies articulaires. Cette cytokine induit la production de médiateurs de l'inflammation, tels que le monoxyde d'azote (NO) et les prostaglandines ainsi que l'expression d'enzymes protéolytiques. La modulation des effets et/ou de la production d'IL-l par les chondrocytes et les synoviocyte
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Books on the topic "RORα"

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Coghe, Jean-Noël. Rory Gallagher. Mercier Press, 2002.

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Doyle, Roddy. Rory & Ita. Jonathan Cape, 2002.

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Doyle, Roddy. Rory & Ita. Random House Publishing Group, 2008.

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Sardjono, Agnes Yani. Roro Jonggrang. Diva Press, 2013.

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Coghe, Jean Noel. Rory Gallagher. Mercier Press, 2001.

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Chambers, Anne Cox, 1919- publisher, Salsgiver Karen book designer, Bixler Michael typographer printer, et al., eds. Remembering Rory. [Anne Cox Chambers], 1987.

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ill, Fitzgerald Brian 1959, ed. Roaring Rory. Lapwing, 2010.

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John, Haslam, ed. Roaring Rory. Ladybird, 2004.

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Doyle, Roddy. Rory & Ita. Viking, 2002.

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United Church of Papua New Guinea and the Solomon Islands. Hui: Hymn book in the Waima-Roro language of Papua New Guinea. Summer Institute of Linguistics, 1999.

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Book chapters on the topic "RORα"

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Gent, Peter M., and Bruce A. Hamilton. "RORα: An Orphan that Staggers the Mind." In Transcription Factors in the Nervous System. Wiley-VCH Verlag GmbH & Co. KGaA, 2006. http://dx.doi.org/10.1002/3527608036.ch15.

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Morellini, Natalie, Ann M. Lohof, Jean Mariani, and Rachel M. Sherrard. "The Staggerer Mouse: RORα Deficiency Induces Cerebellar Neurodegeneration." In Essentials of Cerebellum and Cerebellar Disorders. Springer International Publishing, 2023. http://dx.doi.org/10.1007/978-3-031-15070-8_67.

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Matsuno, Hiroshi, and Makoto Akashi. "Roles of RORα on Transcriptional Expressions in the Mammalian Circadian Regulatory System." In Advances in Bioinformatics and Computational Biology. Springer International Publishing, 2013. http://dx.doi.org/10.1007/978-3-319-02624-4_2.

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Charles, Kenney. "Rory." In Disrupting the Status Quo. Productivity Press, 2017. http://dx.doi.org/10.1201/9781315157986-7.

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Martemyanov, Kirill A., Pooja Parameswaran, Irene Aligianis, et al. "ROKα." In Encyclopedia of Signaling Molecules. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4419-0461-4_101201.

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Olsen, Alexander Arnfinn. "RORO vessels." In Merchant Ship Types. Routledge, 2023. http://dx.doi.org/10.1201/9781003342366-8.

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Löh, Clara. "Roro-Robo." In Quod erat knobelandum. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-58725-6_21.

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Janz, Bernhard. "Rore, Cipriano de." In Metzler Komponisten Lexikon. J.B. Metzler, 1992. http://dx.doi.org/10.1007/978-3-476-03421-2_251.

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Janz, Bernhard. "Rore, Cipriano de." In Komponisten Lexikon. J.B. Metzler, 2003. http://dx.doi.org/10.1007/978-3-476-05274-2_254.

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Töteberg, Michael. "Wolf, Ror." In Kindlers Literatur Lexikon (KLL). J.B. Metzler, 2020. http://dx.doi.org/10.1007/978-3-476-05728-0_21396-1.

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Conference papers on the topic "RORα"

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Jiang, Zhengping, Yining Lu, Hanjie Chen, Daniel Khashabi, Benjamin Van Durme, and Anqi Liu. "RORA: Robust Free-Text Rationale Evaluation." In Proceedings of the 62nd Annual Meeting of the Association for Computational Linguistics (Volume 1: Long Papers). Association for Computational Linguistics, 2024. http://dx.doi.org/10.18653/v1/2024.acl-long.60.

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Liu, Jun, Zhenglun Kong, Peiyan Dong, et al. "RoRA: Efficient Fine-Tuning of LLM with Reliability Optimization for Rank Adaptation." In ICASSP 2025 - 2025 IEEE International Conference on Acoustics, Speech and Signal Processing (ICASSP). IEEE, 2025. https://doi.org/10.1109/icassp49660.2025.10889613.

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Moreno-Smith, Myrthala, Ling Tao, Ronald Bernardi та ін. "Abstract 1951: RORα activation opposes MYCN signaling and restores the circadian clock in neuroblastoma". У Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-1951.

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Xiong, GaoFeng, та Ren Xu. "Abstract 2701: RORα suppresses breast tumor progression by inducing cell polarization and inhibiting cell invasion". У Proceedings: AACR 103rd Annual Meeting 2012‐‐ Mar 31‐Apr 4, 2012; Chicago, IL. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.am2012-2701.

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Zeng, Zhao-lei, Ying-nan Wang, Dan-yun Ruan, Zi-xian Wang та Kai Yu. "IDDF2020-ABS-0179 Cholesterol-Rorα/Γ axis promotes colorectal cancer progression through c-myc stabilization". У Abstracts of the International Digestive Disease Forum (IDDF), 22–23 November 2020, Hong Kong. BMJ Publishing Group Ltd and British Society of Gastroenterology, 2020. http://dx.doi.org/10.1136/gutjnl-2020-iddf.31.

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Henry, Claire E., Neville F. Hacker, and Caroline E. Ford. "Abstract 106: Shock and ROR! Targeting ROR1 and ROR2 in a preclinical patient-derived model of ovarian cancer." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-106.

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Ackerman, D. A., M. I. Dahbura, Y. Shani, et al. "Compact hybrid resonant-optical reflector lasers with very narrow linewidths." In Integrated Photonics Research. Optica Publishing Group, 1990. http://dx.doi.org/10.1364/ipr.1990.wd3.

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Lasers with submegahertz linewidths have importance in various applications, such as in coherent optical communications, interferometric sensors, and resonant-optical gyroscopes. For several years our laboratory has investigated a hybrid Bragg reflector laser with typical linewidths of less than 1 MHz and a narrowest linewidth of 100 kHz. The narrow line results from operating the laser at the steeply rising edge of the Bragg reflection. Understanding this mechanism led to the development1 of resonant-optical reflectors (RORs), integrated-optical elements capable of providing feedback with a r
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O'Connell, Michael P., Janelle L. Nelson, Jennifer L. Fiori, Sarah S. Subaran, Fred E. Indig, and Ashani T. Weeraratna. "Abstract 2346: Switching from an ROR1-positive phenotype to an ROR2-positive phenotype promotes melanoma progression." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2346.

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Bieber, Gerald, Wencke Chodan, Rainer Bader, Bernd Hölle, Peter Herrmann, and Ingo Dreher. "RoRo." In PETRA '19: The 12th PErvasive Technologies Related to Assistive Environments Conference. ACM, 2019. http://dx.doi.org/10.1145/3316782.3322779.

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WITTE, R. "NOISE EMISSION RORO TERMINALS." In Euronoise 2009. Institute of Acoustics, 2023. http://dx.doi.org/10.25144/17394.

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Reports on the topic "RORα"

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Wright, Alec, Susan Miranda, and Gustavo Miranda-Carboni. The Co-expression of ROR1 and FZD2 in WNT5B Signaling in Osteosarcoma. University of Tennessee Health Science Center, 2024. http://dx.doi.org/10.21007/com.lsp.2024.0022.

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Aubry, Philippe, Nicolas Boileau, Marie Briandy, Marie-Christine Chauvat, Sandrine Conin, and Éric Min-Tung. Rapport sur le chantier IdRef en Normandie. Normandie Université, 2023. http://dx.doi.org/10.51203/rapport.nu.000003.

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Suivant le développement des systèmes d’information et des plateformes, ouverts ou fermés, les identifiants pour la recherche se sont multipliés. Leur usage ainsi que leur amélioration est un enjeu pour le développement de l’écosystème de la science ouverte, comme l’indique la note d’orientation du Comité pour la science ouverte de 2019. Qu’ils concernent les structures de recherche, les auteurs ou les objets de la recherche, ils sont indispensables pour identifier et être facilement retrouvés sur internet, répondant ainsi au F des principes FAIR. Ces identifiants, nationaux et internationaux,
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Gruson-Daniel, Célya, and Maya Anderson-González. Étude exploratoire sur la « recherche sur la recherche » : acteurs et approches. Ministère de l'enseignement supérieur et de la recherche, 2021. http://dx.doi.org/10.52949/24.

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• Introduction Dans le cadre du deuxième Plan National de la Science Ouverte, le Comité pour la science ouverte a souhaité mener une étude pour la préfiguration d’un Lab de la science ouverte (LabSO) afin de mieux comprendre le périmètre de la recherche sur la recherche (research on research) dans un contexte international. • Objectifs de l’étude : L’étude répond ainsi à trois objectifs : 1. repérer des grands courants de recherche sur la recherche (RoR) dans le paysage plus général de la recherche universitaire en Europe et outre-atlantique, en reconnaître les principaux acteurs institutionne
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Papua New Guinea - Economic Planning - Mekeo / Roro Survey. Reserve Bank of Australia, 2021. http://dx.doi.org/10.47688/rba_archives_2006/04065.

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Use of RORA for Complex Ground-Water Flow Conditions. US Geological Survey, 2004. http://dx.doi.org/10.3133/wri034304.

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