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1

Newman, Judie, Patricia Ward D'Itri, Norman Lavers, Patrick O'Donnell, and Robert R. Dutton. "Damon Runyon." Yearbook of English Studies 16 (1986): 356. http://dx.doi.org/10.2307/3507868.

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2

Arthur Vogelsang. "Curson, Cahuenga, Runyon, Nichols." Hopkins Review 3, no. 4 (2010): 496–97. http://dx.doi.org/10.1353/thr.2010.0015.

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3

Geyer, Alan. "Theology, Politics, and Peace. Theodore Runyon." Journal of Religion 71, no. 4 (October 1991): 609–10. http://dx.doi.org/10.1086/488749.

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4

RUNYON, JUSTIN B. "Nematode-induced demasculinization of Nearctic Dolichopodidae (Diptera) with five new synonyms." Zootaxa 5092, no. 5 (January 24, 2022): 545–58. http://dx.doi.org/10.11646/zootaxa.5092.5.3.

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Demasculinization, the reduction of male sexual characters due to parasitic nematode infection, is described in Nearctic Dolichopodidae. Five species are confirmed to be demasculinized (3 species of Dolichopus Latreille and 1 species each of Rhaphium Meigen and Tachytrechus Haliday) by nematodes in two families (apparent Mermithidae and Parasitylenchidae). The following nematode-infected males were inadvertently described as new species and are here proposed as synonyms: Dolichopus hurleyi Runyon, 2008 = D. dorycerus Loew, 1864, syn. nov.; Dolichopus frosti Runyon, 2008 = D. sincerus Melander, 1900, syn. nov.; Tachytrechus boharti Harmston, 1968 = T. sanus Osten Sacken, 1877, syn. nov.; Tachytrechus duplicatus Harmston, 1972 = T. sanus Osten Sacken, 1877, syn. nov.; Tachytrechus mchughi Harmston, 1972 = T. sanus Osten Sacken, 1877, syn. nov. A conspectus is presented of the scope of taxonomic problems caused by nematodes in Nearctic Dolichopus and Tachytrechus, which suggests that ten additional names in Dolichopus could be based on infected specimens. Guidelines are provided for recognizing nematode infection to avoid erroneous species descriptions.
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5

Stempel, Guido H. "Book Review: Winchell and Runyon: The Untold Story." Journalism & Mass Communication Quarterly 89, no. 1 (February 16, 2012): 170–71. http://dx.doi.org/10.1177/1077699011431141.

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6

RUNYON, JUSTIN B. "Two new species of Hurleyella Runyon & Robinson (Diptera: Dolichopodidae), with the first record from the Neotropics." Zootaxa 4568, no. 3 (March 21, 2019): 548. http://dx.doi.org/10.11646/zootaxa.4568.3.8.

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Two new species of the long-legged fly genus Hurleyella Runyon & Robinson, 2010 are described and illustrated: Hurleyella belizensis sp. nov. from Belize and Hurleyella salina sp. nov. from alkali areas of the Northern Rockies of the USA (Idaho, Montana, Wyoming). The discovery of these new species greatly extends the known distribution of Hurleyella northward in the Nearctic and southward into the Neotropics. Notes, photos of habitats, a distribution map, and a key to the four known species of Hurleyella are provided.
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7

Stone, Gregory B. "Order in Disorder: Intratextual Symmetry in Montaigne’s Essais by Randolph Paul Runyon." French Review 89, no. 3 (2016): 229–30. http://dx.doi.org/10.1353/tfr.2016.0402.

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8

Larsson, C. E., E. H. Delayte, A. C. Balda, N. S. Michalany, S. R. Pinheiro, M. Otsuka, and E. Roxo. "Dermatite micobacteriana atípica em gato: relato de caso." Arquivo Brasileiro de Medicina Veterinária e Zootecnia 58, no. 6 (December 2006): 1092–98. http://dx.doi.org/10.1590/s0102-09352006000600018.

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Relata-se, pela primeira vez no Brasil, um caso de dermatite decorrente de infecção por micobactéria atípica do complexo Mycobacterium fortuitum-peregrinum, em espécie felina, sem raça definida, fêmea, com cinco anos de idade. Há oito meses, evoluía com lesões maculares equimóticas, nodulares, erosadas, ulceradas, acompanhadas de fístulas exsudativas, com intenso prurido e algia. Evidenciou-se a presença de micobactéria do complexo Mycobacterium fortuitum-peregrinum (grupo IV de Runyon ) identificada após evidenciação histopatológica, cultivo bacteriano e por testes bioquímicos. Após dois meses de terapia sistêmica com enrofloxacina (5mg/kg/Bid/VO) e tópica (triclosan e rifamicina) houve involução das lesões, com efeitos colaterais discretos.
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9

Owa, Maxima, Fransiskus Maria Separ, and Febe F. Irawati Wanggai. "CARL’S SURVIVAL MOTIVATION IN UP MOVIE." Lantern: Journal of Language and Literature 7, no. 2 (September 17, 2021): 130–39. http://dx.doi.org/10.37478/lantern.v7i2.1246.

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Penelitian ini bertujuan untuk menemukan jenis-jenis motivasi dan cara untuk bertahan hidup dalam film Up. Dalam penelitian ini, metode deskriptif kualitatif digunakan untuk menggambarkan ucapan dan tindakan dari tokoh. Pendekatan psikologis diterapkan untuk menganalisis jenis-jenis motivasi dan cara untuk bertahan hidup. Teori yang digunakan untuk menganalisis jenis-jenis motivasi adalah teori yang diusulkan oleh Runyon (1984), sedangkan untuk menganalisis cara untuk bertahan hidup, teori yang diusulkan oleh McClleand (1987) yang dipakai. Hasil penelitian menunjukkan bahwa ada empat jenis motivasi yaitu motivasi intrinsik, motivasi ekstrinsik, motivasi kontributif, dan motivasi relasional. Sedangkan cara untuk bertahan hidup di film Up, yaitu kebutuhan akan prestasi, kebutuhan akan afilisasi, dan kebutuhan akan kekuatan
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10

Schliesser, Th, and A. Weber. "Untersuchungen über die Tenazität von Mykobakterien der Gruppe III nach Runyon in Sägemehleinstreu." Zentralblatt für Veterinärmedizin Reihe B 20, no. 9 (May 13, 2010): 710–14. http://dx.doi.org/10.1111/j.1439-0450.1973.tb01168.x.

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11

LOWRY, JAMES K., ALAN A. MYERS, and TAKAFUMI NAKANO. "Replacement names for four preoccupied talitrid genus-group names proposed by Lowry & Myers in 2019 (Crustacea, Amphipoda, Senticaudata)." Zootaxa 4615, no. 2 (June 13, 2019): 395. http://dx.doi.org/10.11646/zootaxa.4615.2.11.

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A recent comprehensive review of the generic-level classification of the amphipod family Talitridae established 37 new genera (Lowry & Myers 2019). Among the 37 genus-group names, however, four names—Fleuria Lowry & Myers, 2019, Hurleyella Lowry & Myers, 2019, Tasmanella Lowry & Myers, 2019 and Wairua Lowry & Myers, 2019—are respectively preoccupied by the dipteran Fleuria Kieffer, 1924, the dipteran Hurleyella Runyon & Robinson, 2010, the brachiopod Tasmanella Laurie, 1991, and the arachnid Wairua Forster in Forster et al., 1990. Accordingly, new replacement names for each of the talitrid genus-group names are proposed herein. Additionally, the authorship of the superfamily Talitroidea cited in Lowry & Myers (2019) as Bulycheva, 1957, should be cited as Rafinesque, 1815.
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12

Kraus, Joe. "Broadway Boogie Woogie: Damon Runyon and the Making of New York City Culture (review)." MFS Modern Fiction Studies 50, no. 3 (2004): 752–53. http://dx.doi.org/10.1353/mfs.2004.0072.

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13

HURLEY, RICHARD L., and JUSTIN B. RUNYON. "A review of Erebomyia (Diptera: Dolichopodidae), with descriptions of three new species." Zootaxa 2054, no. 1 (March 27, 2009): 38–48. http://dx.doi.org/10.11646/zootaxa.2054.1.2.

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The genus Erebomyia is reviewed and a key is provided for the four known species, three of which are described here: Erebomyia aetheoptera n. sp., E. akidoptera n. sp., and E. ramseyensis n. sp. The type species, E. exalloptera Runyon & Hurley, is redescribed and the male genitalia illustrated. Erebomyia is known from Arizona (3 spp.) and California (1 sp.), and males are notable for their modified wings, most exceptionally those of E. exalloptera whose left wing is of a different shape and size than the right wing. The occurrence of directional wing asymmetry in insects is reviewed to place the unique wings of E. exalloptera in context. Observations of Erebomyia courtship behavior are provided, and the potential adaptive significance of asymmetrical and modified wings of Erebomyia is discussed.
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14

Raymond, J. T., L. Tell, M. Bush, D. K. Nichols, F. Y. Schulman, and R. J. Montali. "Subcutaneous Atypical Mycobacteriosis in Captive Tiger Quolls (Dasyurus maculatus)." Veterinary Pathology 37, no. 2 (March 2000): 137–42. http://dx.doi.org/10.1354/vp.37-2-137.

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From July 1989 to October 1998, 9/37 (24%) adult captive tiger quolls ( Dasyurus maculatus) were diagnosed with atypical mycobacterial infection involving the subcutis and skin. Females were more often affected than males (seven females, two males). Grossly, lesions presented as focal thickenings, plaques, and abscesses within the subcutis, often with fistulous tracts. The subcutis and skin overlying cervical and thoracic regions were the primary sites of infection. Cytology of subcutaneous impression smears from all nine affected tiger quolls revealed pyogranulomatous inflammation admixed with several acid-fast bacilli. Histologically, all tiger quolls had nodular to diffuse pyogranulomatous panniculitis and cellulitis. Small numbers of acid-fast bacilli were noted histologically in 7/9 (78%) animals. Skin cultures from seven tiger quolls were positive for one or more different Runyon group IV mycobacteria. The disease described in these tiger quolls is similar to subcutaneous atypical mycobacteriosis of humans and domestic animals.
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15

Afifah, Salma, and Gazi Saloom. "DUKUNGAN SOSIAL TEMAN SEBAYA DAN SELF-EFFICACY DALAM PENYESUAIAN DIRI SANTRI BARU." Dialog 41, no. 2 (February 10, 2020): 139–50. http://dx.doi.org/10.47655/dialog.v41i2.309.

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Penelitian ini dilakukan untuk mengetahui pengaruh dukungan sosial teman sebaya dan self-efficacy terhadap penyesuaian diri santri baru di pondok pesantren. Pengambilan sampel yang dilakukan menggunakan non probability sampling. Alat ukur yang digunakan dalam penelitian ini terdiri dari Psychological Adjustment Scale (PAS), yang dikembangkan oleh Haber dan Runyon (dalam Mahmood dkk., 2015), The Social Provision Scale (SPS) yang dikembangkan oleh Cutrona dan Russel (1987), dan General Self-Efficacy Scale 12 (GSES-12), yang dikembangkan oleh Bosscher dan Smit (1998). Uji validitas alat ukur menggunakan teknik Confirmatory Factor Analysis (CFA) dan teknik analisis data menggunakan teknik analisis regresi berganda. Hasil penelitian menunjukkan bahwa ada pengaruh yang signifikan dukungan sosial teman sebaya dan self-efficacy terhadap penyesuaian diri sebesar 35.4%. Artinya, proporsi varians dari penyesuaian diri yang dijelaskan secara bersama-sama oleh dukungan sosial teman sebaya dan self-efficacy adalah sebesar 35.4% sementara 64.6% lainnya dipengaruhi oleh variabel lain di luar penelitian ini.
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16

Dotson, Rand. "The Assault on Elisha Green: Race and Religion in a Kentucky Community by Randolph Paul Runyon." Register of the Kentucky Historical Society 119, no. 4 (September 2022): 423–25. http://dx.doi.org/10.1353/khs.2022.0003.

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17

Zhang, Yi, Ashley R. Sandy, Jina Wang, Koji Kato, Shan He, Ivy Tran, Gloria Tran Shan, et al. "Notch Signaling Is a Critical Regulator of Allogeneic T Cell Responses Mediating Graft-Versus-Host Disease." Blood 114, no. 22 (November 20, 2009): 230. http://dx.doi.org/10.1182/blood.v114.22.230.230.

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Abstract Abstract 230 Allogeneic hematopoietic stem cell transplantation (HSCT) provides powerful therapeutic activity through elimination of cancer cells by donor T cells. However, its success is limited by life-threatening graft-versus-host disease (GVHD). Novel immunomodulatory approaches are needed to effectively control GVHD. Notch signaling is a highly conserved cell-cell communication that plays an essential role in T cell development and can influence differentiation and function of mature T cells in a context-dependent fashion. Using several mouse models of allogeneic HSCT, we report that inactivation of Notch signaling in donor T cells inhibits the induction of severe GVHD while preserving significant graft-versus-leukemia (GVL) activity. To block canonical Notch signaling in mature T cells without interfering with T cell development, we used conditional expression of the pan-Notch inhibitor DNMAML (ROSADNMAMLf × Cd4-Cre mice). DNMAML CD4+ T cells derived from donor C57BL/6 (B6) mice had normal initial activation, proliferation and expansion, but failed to differentiate into effector T cells mediating severe GVHD in lethally irradiated major histocompatibility complex-mismatched BALB/c recipient mice. Notably, Notch-deprived alloreactive T cells retained potent GVL activity, leading to improved overall survival of the recipients. Alloreactive DNMAML T cells showed impaired production of IL-2 and multiple inflammatory cytokines (e.g. TNFα, IFNγ and IL-17), as well as defective induction of selected effector molecules (e.g. granzyme B and perforin). The specificity of these findings was independently validated using donor CD4+ T cells with conditional inactivation of Rbpj, encoding CSL/RBP-Jk, a transcription factor that is absolutely required for Notch-mediated transcriptional activation. To further evaluate the effects of Notch signaling in both CD4+ and CD8+ alloreactive T cells, we infused control B6 or DNMAML B6 T cells into unirradiated haploidentical B6xDBA/2 F1 and irradiated minor histocompatibility antigen (miHA)-mismatched BALB/b recipients. Donor DNMAML CD4+ and CD8+ T cells both showed significantly reduced ability to induce severe GVHD in these CD4+ help-dependent CD8+ T cell-mediated GVHD mouse models. These results indicate that Notch is a novel critical signaling pathway regulating CD4+ and CD8+ T cell responses in multiple mouse models of allogeneic HSCT. Given the beneficial immunomodulatory effects of Notch inhibition in alloreactive donor T cells, Notch signaling appears as a promising therapeutic target to limit the harmful effects of GVHD while preserving beneficial GVL activity after allogeneic HSCT. Disclosures: Zhang: University of Michigan Comprehensive Cancer Center: Research Funding; Damon Runyon Cancer Research Foundation: Research Funding. Maillard:University of Michigan Comprehensive Cancer Center: Research Funding; Damon Runyon Cancer Foundation: Research Funding; ASH Scholar Awards : Research Funding.
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18

Bonin, Kate M. "Intratextual Baudelaire: The Sequential Fabric of the Fleurs du mal and Spleen de Paris by Randolph Paul Runyon." French Review 85, no. 1 (2011): 183–84. http://dx.doi.org/10.1353/tfr.2011.0139.

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19

Kreuzpaintner, Georg, Philip Kirschner, Axel Wallnert, Richard K??lble, Rudolf Hesterberg, Lutz Thomas, and Franz Borchard. "Mycobacteria of Runyon groups I, II and IV do not play an aetiological role in Crohn??s disease." European Journal of Gastroenterology & Hepatology 12, no. 7 (December 1995): 1177–82. http://dx.doi.org/10.1097/00042737-199512000-00009.

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20

Sim, Stuart. "John Bunyan’s Master Story: The Holy War as Battle Allegory in Religious and Biblical Context by Daniel Runyon." Scriblerian and the Kit-Cats 41, no. 2 (2009): 239–41. http://dx.doi.org/10.1353/scb.2009.0008.

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21

BROCK, J. A., LAUREN K. NAKAGAWA, and R. J. SHIMOJO. "Infection of a cultured freshwater prawn, Macrobrachium rosenbergii de Man (Crustacea: Decapoda), by Mycobacterium spp., Runyon Group II." Journal of Fish Diseases 9, no. 4 (July 1986): 319–24. http://dx.doi.org/10.1111/j.1365-2761.1986.tb01021.x.

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22

Niemi-Pynttäri, Risto, Vesa Haapala, Tiina Käkelä-Puumala, and Outi Oja. "Arvostelut." AVAIN - Kirjallisuudentutkimuksen aikakauslehti, no. 1 (October 1, 2004): 75–90. http://dx.doi.org/10.30665/av.74603.

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Risto Niemi-Pynttäri Kuinka sovitan itseni tieteelliseen tekstiin Johanna Latvala, Eeva Peltonen ja Tuija Saresma (toim.): Tutkija kertojana, Tunteet, tutkimusprosessi ja kirjoittaminen Vesa Haapala Runot ajassaan ja paikassaan Sakari Katajamäki ja Johanna Pentikäinen (toim.): Runosta runoon. Suomalaisen runon yhteyksiä länsimaiseen kirjallisuuteen antiikista nykyaikaan Tiina Käkelä-Puumala Narratologia sateenkaaren päässä Samuli Hägg: Narratologies of Gravity’s Rainbow Outi Oja Väitöskirja Bo Carpelanin kirjallisuuskäsityksestä Anna Hollsten: ”Ei kattoa, ei seiniä” – Näkökulmia Bo Carpelanin kirjallisuuskäsitykseen
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23

Patel, Trisha, Leslie Scroggins-Markle, and Brent Kelly. "A Dermal Piercing Complicated byMycobacterium fortuitum." Case Reports in Dermatological Medicine 2013 (2013): 1–3. http://dx.doi.org/10.1155/2013/149829.

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Background. Dermal piercings have recently become a fashion symbol. Common complications include hypertrophic scarring, rejection, local infection, contact allergy, and traumatic tearing. We report a rare case ofMycobacterium fortuitumfollowing a dermal piercing and discuss its medical implications and treatments.Case. A previously healthy 19-year-old woman presented complaining of erythema and edema at the site of a dermal piercing on the right fourth dorsal finger. She was treated with a 10-day course of trimethoprim-sulfamethoxazole and one course of cephalexin by her primary care physician with incomplete resolution. The patient stated that she had been swimming at a local water park daily. A punch biopsy around the dermal stud was performed, and cultures with sensitivities revealedMycobacterium fortuitum. The patient was treated with clarithromycin and ciprofloxacin for two months receiving full resolution.Discussion.Mycobacterium fortuitumis an infrequent human pathogen. This organism is a Runyon group IV, rapidly growing nontuberculous mycobacteria, often found in water,soil, and dust. Treatment options vary due to the size of the lesion. Small lesions are typically excised, while larger lesions require treatment for 2–6 months with antibiotics. We recommend a high level of suspicion for atypical mycobacterial infections in a piercing resistant to other therapies.
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24

Kaczorowski, Robert J. "The Enforcement Provisions of the Civil Rights Act of 1866: A Legislative History in Light of Runyon v. McCrary." Yale Law Journal 98, no. 3 (January 1989): 565. http://dx.doi.org/10.2307/796630.

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25

Watkins, Andrea S. "The Mentelles: Mary Todd Lincoln, Henry Clay, and the Immigrant Family Who Educated Antebellum Kentucky by Randolph Paul Runyon." Journal of the Early Republic 40, no. 1 (2020): 171–73. http://dx.doi.org/10.1353/jer.2020.0021.

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26

Neels, Mark A. "The Mentelles: Mary Todd Lincoln, Henry Clay, and the Immigrant Family Who Educated Antebellum Kentucky by Randolph Paul Runyon." Journal of Southern History 85, no. 2 (2019): 440–41. http://dx.doi.org/10.1353/soh.2019.0151.

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27

HUMAIRA, HUMAIRA, and RISANA RACHMATAN. "PERBEDAAN PENYESUAIAN DIRI PENSIUNAN YANG MENDAPATKAN TRAINING PRA PENSIUN DENGAN YANG TIDAK MENDAPATKAN TRAINING PRA-PENSIUN." Jurnal Ecopsy 4, no. 1 (May 2, 2017): 1. http://dx.doi.org/10.20527/ecopsy.v4i1.3409.

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ABSTRAK Masa pensiun merupakan masa dimana individu kehilangan aktivitas bekerja di kantor dan mulai mencari kegiatan baru untuk mengganti aktivitas formal tersebut. Dibutuhkan penyesuaian diri untuk melalui masa-masa tersebut, salah satunya dengan mengikuti training pra pensiun. Training pra pensiun merupakan sebuah pelatihan yang diberikan kepada karyawan yang akan memasuki masa pensiun. Melalui training pra pensiun, pensiunan dapat merubah sikap menjadi lebih positif dalam menghadapi masa pensiun. Penyesuaian diri pensiunan yang baik adalah menerima keterbatasan-keterbatasan yang tidak bisa berubah dan secara aktif memodifikasi keterbatasan yang masih bisa diubah. Berdasarkan latar belakang tersebut penelitian ini bertujuan untuk mengetahui perbedaan penyesuaian diri pensiunan pada pensiunan yang mendapatkan training pra pensiun dengan yang tidak mendapatkan training pra pensiun. Hipotesis penelitian adalah adanya perbedaan penyesuaian diri pensiunan yang mendapatkan training pra pensiun dengan yang tidak mendapatkan training pra pensiun. Sampel penelitian adalah pensiunan yang mendapatkan training pra pensiun, yaitu PT PLN Persero yang berjumlah 24 orang, dan 24 orang PT POS Indonesia dan PT Kimia Farma yang tidak mendapatkan training pra pensiun. Pengumpulan data dilakukan dengan menggunakan skala Penyesuaian Diri Pensiunan yang disusun berdasarkan teori Habber dan Runyon (1984). Hasil analisis data menggunakan teknik komparasi Independent Sample T-Test menunjukkan terdapat perbedaan penyesuaian diri pensiunan yang signifikan antara pensiunan yang mendapatkan training pra pensiun dengan yang tidak mendapatkantraining pra pensiun. Kata kunci : Penyesuaian Diri, Pensiunan, Karyawan, Training Pra Pensiun ABSTRACT Retirement is a period in which the individual loses work activity in the office and starts to look the new activities to replace the formal activity. Adjustment is needed to get through this time, one of it is by following the pre-retirement training. Pre-retirement training is a training given to employees who will retire. Through pre-retirement training, retirees will have more positive attitude toward retirement by following the retirement preparation seminars. An effective adjustment of retirees is to accept the limitations that cannot be changed and to modify actively the limitations that can be changed. Based on this background, the aim of this study is to determine the difference of adjustment between retirees who receive pre-retirement training and did not receive pre-retirement training. Hypothesis of this research is there is the difference of adjustment between retirees who receive pre-retirement training and did not receive pre-retirement training. The participant were 24 retirees who get pre-retirement training of PT PLN Persero and 24 retirees of PT POS Indonesia and PT Kimia Farma who did not receive pre-retirement training. The data was collected using Retirees Adjustment scale which is based on Habber and Runyon (1984) theory. The results of the data analysis using comparative techniques Independent Sample T-Test showed there is a significant difference of adjustment between retirees who receive pre-retirement training and did not receive pre-retirement training. Keywords: Adjustment, Retireess, Employees, Pre-Retirement Training.
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28

Shan, Gloria T., Ivy Tran, Ashley R. Sandy, Ann Friedman, Yi Zhang, and Ivan Maillard. "Inhibition of Notch Signaling in T Cells Prevents Immune-Mediated Bone Marrow Failure." Blood 114, no. 22 (November 20, 2009): 180. http://dx.doi.org/10.1182/blood.v114.22.180.180.

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Abstract Abstract 180 Aplastic anemia is a severe bone marrow disorder characterized by the loss of hematopoietic stem cells (HSC). HSC destruction is thought to be T cell-mediated in a majority of patients with aplastic anemia. Global immunosuppression and HSC transplantation can induce disease remission, but these treatments are not effective in all patients and can promote life-threatening complications. Thus, novel immunomodulatory approaches are needed in this disorder. Notch is a conserved cell-cell communication pathway that can regulate T cell differentiation and function with context-dependent effects. To study the role of Notch signaling in pathogenic T cells causing immune-mediated bone marrow failure, we inhibited canonical Notch signaling in mature T cells through conditional expression of the pan-Notch inhibitor DNMAML (ROSA-DNMAMLf × Cd4-Cre mice). We used two complementary mouse models of immune-mediated bone marrow failure that mimic features of aplastic anemia: administration of C57BL/6 (B6) T cells into sublethally irradiated (500 rads) minor histocompatibility antigen mismatched BALB/b recipients (Chen et al., J Immunol 2007; 178:4159), or infusion of B6 lymphocytes into unirradiated MHC-mismatched B6×DBA F1 recipients. In contrast to control B6 T cells which led to lethal bone marrow failure in virtually all recipients, DNMAML-expressing Notch-deprived T cells were profoundly deficient at inducing HSC loss in both disease models, leading to markedly improved long-term survival (>90%). Notch-deficient T cells showed a modest decrease in overall expansion within secondary lymphoid organs, but their accumulation in the target bone marrow was preserved. Upon restimulation with anti-CD3 and anti-CD28 antibodies, DNMAML T cells had decreased production of IL-2 and interferon gamma. Activated CD4+ and CD8+ DNMAML T cells had reduced interferon gamma, granzyme B, and perforin transcripts despite preserved induction of the master transcription factors Tb×21 (encoding T-bet) and Eomes. In vivo infusion of CFSE-labeled host-type target cells revealed a decreased cytotoxicity in DNMAML as compared to control B6 T cell recipients. These observations point to a novel spectrum and mechanism of Notch action in mature T cells. Since we have shown recently that canonical Notch signaling is dispensable for the maintenance of adult HSCs (Maillard et al., Cell Stem Cell 2008, 2:356), our findings suggest that Notch inhibition could represent a novel therapeutic modality to target the T cell response and reverse immune-mediated HSC destruction in aplastic anemia. Disclosures: Shan: American Society of Hematology: Research Funding. Zhang:University of Michigan Comprehensive Cancer Center: Research Funding; Damon Runyon Cancer Research Foundation: Research Funding. Maillard:Damon Runyon Cancer Research Foundation: Research Funding; American Society of Hematology: Research Funding; University of Michigan Comprehensive Cancer Center: Research Funding.
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29

Jarzembowski, Jason A., and Michael B. Young. "Nontuberculous Mycobacterial Infections." Archives of Pathology & Laboratory Medicine 132, no. 8 (August 1, 2008): 1333–41. http://dx.doi.org/10.5858/2008-132-1333-nmi.

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Abstract Context.—Nontuberculous mycobacteria include numerous acid-fast bacilli species, many of which have only recently been recognized as pathogenic. The diagnosis of mycobacterial disease is based on a combination of clinical features, microbiologic data, radiographic findings, and histopathologic studies. Objective.—To provide an overview of the clinical and pathologic aspects of nontuberculous mycobacteria infection, including diagnostic laboratory methods, classification, epidemiology, clinical presentation, and treatment. Data Sources.—Review of the pertinent literature and published methodologies. Conclusions.—Nontuberculous mycobacteria include numerous acid-fast bacilli species, many of which are potentially pathogenic, and are classified according to the Runyon system based on growth rates and pigment production. Their slow growth hinders cultures, which require special medium and prolonged incubation. Although such methods are still used, newer nucleic acid–based technologies (polymerase chain reaction and hybridization assays) can rapidly detect and speciate some mycobacteria—most notably, distinguishing Mycobacterium tuberculosis from other species. Infections caused by these organisms can present as a variety of clinical syndromes, not only in immunocompromised patients but also in immunocompetent hosts. Most common among these are chronic pulmonary infections, superficial lymphadenitis, soft tissue and osteoarticular infections, and disseminated disease. Treatment of nontuberculous mycobacterial infections is difficult, requiring extended courses of multidrug therapy with or without adjunctive surgical intervention.
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Li, Jing, Herbert E. Cohn, Charles J. Yeo, and Scott W. Cowan. "John Y. Templeton III: Pioneer of Modern Cardiothoracic Surgery." American Surgeon 78, no. 11 (November 2012): 1201–3. http://dx.doi.org/10.1177/000313481207801124.

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John Young Templeton III was born in 1917 in Portsmouth, Virginia, and graduated from Jefferson Medical College in 1941. He completed his residency training under Dr. John H. Gibbon, Jr., and was the first resident who worked on Gibbon's heart–lung machine. After his training, he remained at Jefferson as an American Cancer Society fellow and Damon Runyon fellow and went on to become the fourth Samuel D. Gross Professor and Chair of the Department of Surgery in 1967. Dr. Templeton was the recipient of numerous grants and published over 80 papers in the field of cardiothoracic surgery. As a teacher and mentor, he was a beloved figure who placed great faith in his residents. He participated in over 60 professional societies, serving as president to many such as the Philadelphia Academy of Surgery and the Pennsylvania Association of Thoracic Surgery. He was also recognized through his many awards, in particular the John Y. Templeton III lectureship established in 1980 at Jefferson of whom Denton Cooley was the first lecturer. Dr. Templeton retired from practice in 1987. He is forever remembered as an important model of a modern surgeon evident in numerous academic achievements, the admiration and affection of his trainees, and the lives of patients that he had touched.
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Obar, Joshua, Kamal M. Khanna, Quynh-Mai Pham, and Leo Lefrancois. "Endogenous naive CD8 T cell precursor frequency and infection type control CD62L gene expression (85.6)." Journal of Immunology 178, no. 1_Supplement (April 1, 2007): S119. http://dx.doi.org/10.4049/jimmunol.178.supp.85.6.

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Abstract CD62L is essential for homing of lymphocytes to lymph nodes. Moreover, CD62L expression identifies distinct lineages of memory CD8 T cells. However, the factors regulating memory cell lineage differentiation are ill defined. Our data show that CD62L gene regulation is controlled by the input number of transferred antigen-specific CD8 T cells. To enable analysis of endogenous antigen-specific CD8 T cells, we have now utilized a method to enrich naïve antigen-specific CD8 T cells using MHC class I tetramers in conjunction with magnetic bead separation. Of three specificities tested thus far, naïve precursor frequencies ranged from ~100–1000 cells/mouse. Interestingly, following infection with recombinant vesicular stomatitis viruses, the naïve precursor frequency inversely correlated with the time required for the resulting memory population to shift to CD62L+ cells. Remarkably, in the case of ova-specific CD8 T cells, the shift toward CD62L+ ova-specific memory cells occurred substantially faster following infection with Listeria monocytogenes expressing ovalbumin than after VSV-ova infection. This system will allow us to examine parameters of the endogenous CD8 T cell response to infection from initial activation to memory development. This work was supported by NIH grants AI41576 and DK45260 (LL) and a Damon Runyon Foundation fellowship (KMK).
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Kulalert, Warakorn, Alexandria Wells, Michel Enamorado, Verena Link, Juraj Kabat, Olena Kamenyeva, and Yasmine Belkaid. "Functional tuning of commensal-specific lymphocytes by nociceptive sensory neurons." Journal of Immunology 208, no. 1_Supplement (May 1, 2022): 113.21. http://dx.doi.org/10.4049/jimmunol.208.supp.113.21.

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Abstract The somatosensory nervous system surveils external stimuli at barrier tissues, regulating innate immune cells under infection and inflammation. The roles of nociceptive nerves in homeostatic adaptive immunity to the microbiota, however, remain elusive. Here, we identified a novel mechanism for direct neuro-immune interaction between commensal-specific T lymphocytes and skin-innervating nociceptive neurons through secretion of calcitonin gene-related peptide (CGRP). We observed upregulation of the CGRP co-receptor, receptor activity modifying protein 1 (RAMP1), in T lymphocytes induced by topical colonization of the human skin commensal Staphylococcus epidermidis, but not in T cell populations elicited by cutaneous infection or inflammation. Utilizing intravital imaging, we observed that commensal-specific T cells interact intimately and dynamically with skin nociceptive nerves in a CGRP-dependent manner. Importantly, CGRP secreted from nociceptors acts directly on commensal-specific T cells via RAMP1 to regulate their accumulation in the tissues, cytokine production and motility in vivo. Activation or inhibition of nociceptive neurons functionally tune commensal-specific T cells and optimize their responses to ensuing heterologous infections or damaging challenges in the skin. The ability of the somatosensory neurons to participate in the highly specific and potentially long-lasting adaptive immune response to the microbiota underscores the functional versatility and plasticity of commensal-specific T lymphocytes that can be swiftly tuned to diverse arrays of sensory modalities ranging from thermosensation to noxious pain under steady state and pathology. Supported by Damon Runyon Cancer Research Foundation
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Jarjour, Nicholas N., Kelsey M. Wanhainen, Changwei M. Peng, Henrique Borges da Silva, Ryan J. Martinez, Talia S. Dalzell, Matthew A. Huggins, Joseph F. Urban, Sara E. Hamilton Hart, and Stephen C. Jameson. "Common gamma chain-dependent cytokines drive antigen-independent proliferation of circulating and resident memory CD8+ T cells." Journal of Immunology 208, no. 1_Supplement (May 1, 2022): 45.03. http://dx.doi.org/10.4049/jimmunol.208.supp.45.03.

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Abstract Memory CD8+ T cells are found throughout the body and can locally control reinfection at barrier sites. Vaccines often fail to elicit durable tissue-resident memory (TRM) CD8+ T cells, likely contributing to unsuccessful efforts to vaccinate against several important pathogens. TRMs locally self-renew by poorly understood mechanisms; a better understanding of this process could help to enhance TRM immunity. We propose that proliferating TRMs flexibly use available members of the common gamma chain-(γC)-dependent cytokine family to persist and as bystanders during other immune responses. We have found that γC-dependent cytokines can mediate cell-intrinsic signaling to drive proliferation of both circulating and resident memory CD8+ T cells, in contrast to naïve T cells. Using adoptive transfer as well as tetramer staining of endogenous populations, we have confirmed that cytokine-stimulated proliferation of memory T cells of known antigen specificities elicited by different pathogens can occur without cognate antigen. Via bulk and single-cell RNA-Seq of resting and in vivo cytokine-stimulated memory CD8+ T cells, we are elucidating the downstream signature of cytokine-driven TRM proliferation. We have also found that memory CD8+ T cells proliferate comparably in response to exogenous cytokines or unrelated pathogens, consistent with pathogen-elicited γC-dependent cytokine signals driving memory CD8+ T cell proliferation. These data support a conserved ability of TRM and circulating memory T cells to use infection-elicited γC-dependent cytokines for enhanced proliferation, likely to promote maintenance, and that treatment with γC-dependent cytokines can drive rapid proliferation without a need to identify antigens. N. N. J. Damon Runyon Cancer Research Foundation Fellow DRG-2427-21 K. M. W. NCI F30 H. B. d. S. CRI/Paul Shiverick Fellow; NIAID K99/R00 AI139381 S. C. J. NIAID R01 AI038903
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Cauley, Linda S., Kamal M. Khanna, and Leo Lefrancois. "In situ visualization of CD8 T cell-DC interactions reveals distinct features of the early and late response to antigen after influenza virus infection (87.27)." Journal of Immunology 178, no. 1_Supplement (April 1, 2007): S133. http://dx.doi.org/10.4049/jimmunol.178.supp.87.27.

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Abstract The anatomical characteristics of T cell activation following infection are poorly described. Using transferred CD8 T cells and confocal microscopy we show that small clusters of virus-specific T cells formed transiently in the lung-draining lymph nodes (DLN) early in the response to influenza virus infection. Several weeks after infection naïve nucleoprotein (NP)-specific CD8 T cells still responded to antigen in the DLN, but remained predominantly in large stable clusters that included CD11c+ APCs. Many activated cells upregulated CD69 and PD-1, but did not lose CD62L-expression and remained clustered with CD11c+ APCs in the paracortex of the DLN until at least 7 days after transfer. Some central/memory cells also entered the DLN after transfer, but did not colocalize with the clustered naïve cells or show any signs of a response to the antigen. These data indicated a change in the response to antigen stimulation in the DLN as the time interval after infection increased and suggested that naïve and memory F5 CD8 T cells used different mechanisms to locate antigen-bearing APCs within the pLN. Some endogenous NP366-374/Db-specific CD8 T cells were also retained in the MLN and showed signs of a continuing response to antigen stimulation, while PA324-332/Db-specific cells were predominantly found in non-lymphoid tissues. Overall all the data revealed a dichotomy in the response to late antigen presentation which could influence epitope selection during reinfection by favoring preferential expansion of the NP366-374/Db-specific cells in the DLN. This work was supported by the American Lung Association RG1119 (L.S.C.), the Damon Runyon foundation (K.M.K.) and National Institutes of Health grants; R21-AI065895 (L.S.C.) and DK45260, AI41576 and AI56172 (LL).
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LINDNER, CHRISTOPH. "Daniel R. Schwartz, Broadway Boogie Woogie: Damon Runyon and the Making of New York City Culture (New York: Palgrave Macmillan, 2003, £25.00). Pp. 346. ISBN 0 312 23948 3." Journal of American Studies 38, no. 3 (December 2004): 528–29. http://dx.doi.org/10.1017/s0021875804508770.

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36

Scott, Clive. "Book Reviews: Intratextual Baudelaire: The Sequential Fabric of the Fleurs du mal and Spleen de Paris. By Randolph Paul Runyon. Columbus: Ohio State University Press, 2010. Pp. 282. $57.95 (hbk)." Journal of European Studies 41, no. 1 (February 3, 2011): 85–86. http://dx.doi.org/10.1177/00472441110410010507.

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Thompson, Craig B. "Targeting Metabolic Inputs into Epigenetic Regulations of Acute Leukemia." Blood 122, no. 21 (November 15, 2013): SCI—26—SCI—26. http://dx.doi.org/10.1182/blood.v122.21.sci-26.sci-26.

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Abstract An essential feature of acute myeloid leukemia (AML) is the profound block to differentiation exhibited by leukemic cells. This AML-associated arrest in the normal process of cellular differentiation is not observed in more chronic forms of leukemia. Chronic forms of leukemia and lymphoma are marked by the increased propensity to enter the cell cycle as the malignant cells move to more mature states of differentiation and the increased resistance to apoptosis these cells exhibit. Considerable recent evidence has suggested that profound epigenetic remodeling occurs as either a result or a cause of cellular differentiation. Altered DNA methylation has now been established as a hallmark of acute leukemia and yet very little is known concerning the mechanisms through which this occurs. In 2010, in collaboration with others, we found that neomorphic mutations of the citrate metabolism genes IDH1 and IDH2 induce DNA hypermethylation and impair differentiation in hematopoietic cells. IDH mutations create a block to DNA and histone demethylation as a result of the production of 2-hydroxyglutarate (2HG). 2HG acts as a competitive inhibitor of α-ketoglutarate-dependent enzymes. The epigenetic effects of 2HG are caused in part though inhibition of TET2, a DNA demethylase enzyme also mutated in leukemia. IDH 1/2- and TET2-mutant primary AML cells displayed a similar defect in epigenetic programming consisting of global hypermethylation and a gene-specific methylation signature. This work identifies IDH1/2- and TET2-mutant leukemias as a biologically distinct disease subtype, and links cancer metabolism with epigenetic control of gene expression. The implications of this work and the identification of additional metabolic genes involved in epigenetic deregulation in leukemia will be discussed. Disclosures: Thompson: NIH: Research Funding; Keystone Symposia: Consultancy; NCI: Research Funding; Damon Runyon : Research Funding; Ludwig: Membership on an entity’s Board of Directors or advisory committees; HHMI: Membership on an entity’s Board of Directors or advisory committees; Merck: Equity Ownership, Membership on an entity’s Board of Directors or advisory committees; Agios: Equity Ownership, Membership on an entity’s Board of Directors or advisory committees; MSKCC: Employment; Pfizer: Patents & Royalties; BMS: Patents & Royalties; Repligen: Patents & Royalties.
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Larson, Rebecca, Michael Kann, Stefanie Bailey, Nicholas Haradhvala, Kai Stewart, Amanda Bouffard, Irene Scarfo, et al. "Loss of IFNgR1 signaling glioblastoma drives resistance to CAR T cell binding avidity and cytotoxicity due to lower downstream expression of ICAM-1." Journal of Immunology 208, no. 1_Supplement (May 1, 2022): 176.18. http://dx.doi.org/10.4049/jimmunol.208.supp.176.18.

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Abstract Though CAR T cell therapy has had success treating hematologic malignancies, these treatments have shown only modest efficacy in solid tumors. Little is known about the necessary molecular components required for CAR T cell cytotoxicity, especially in the context of solid tumors. We investigated the role of IFNgR signaling on solid tumors and discovered across tumor types (including glioblastoma, pancreatic, ovarian and lung) loss of IFNgR1 signaling resulted lower CAR T cell cytotoxicity in vitro and that this was irrespective of target antigen (including endogenous EGFR, IL13Ra2, mesothelin, and exogenous CD19). We also validated this in orthotopic xenograft models of glioblastoma and pancreatic cancer in vivo. CAR T cells exposed to wildtype (WT) or IFNgR1 KO tumor had similar transcriptional profiles, but the tumor cells had different signatures in response to CAR T cell co-culture. Further investigation showed marked upregulation of cell adhesion pathways in WT cells compared to IFNgR1 KO. We utilized microscopy with acoustic force and discovered CAR T cells had lower cell binding avidity to cells lacking IFNgR1 compared to WT cells. Following CAR T cell exposure, Intercellular Adhesion Molecule 1 (ICAM-1) was strongly upregulated at both the transcriptional and protein levels in WT cells but not IFNgR1 KO cells. We overexpressed ICAM-1 on IFNgR1 KO tumor cells and observed that CAR T cell binding avidity and cytotoxicity was restored to that of WT levels. This work highlights the importance of CAR T cell binding avidity and adhesion for optimal cytotoxicity. Better understanding of CAR T cell function will inform future construct design for successful therapies against solid tumors. RCL was supported by T32 GM007306, T32 AI007529, and the Richard N. Cross Fund. ML was supported by T32 2T32CA071345-21A1. SRB was supported by T32CA009216-38. NJH was supported by the Landry Cancer Biology Fellowship. JJ is supported by a NIH F31 fellowship (1F31-MH117886). GG was partially funded by the Paul C. Zamecnik Chair in Oncology at the Massachusetts General Hospital Cancer Center and NIH R01CA 252940. MVM and this work is supported by the Damon Runyon Cancer Research Foundation, Stand Up to Cancer, NIH R01CA 252940, R01CA238268, and R01CA249062.
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Kato, Koji, Shuaiying Cui, Rork Kuick, Shin Mineishi, Elizabeth Hexner, James LM Ferrara, Victor E. Marquez, Stephen G. Emerson, and Yi Zhang. "Alloreactive CD8+ Effector T Cells Proliferate and Persist Upon Chronic Exposure to Alloantigens through Reactivation of Stem Cell Transcriptional Programs." Blood 114, no. 22 (November 20, 2009): 2444. http://dx.doi.org/10.1182/blood.v114.22.2444.2444.

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Abstract Abstract 2444 Poster Board II-421 Alloreactive effector T cells are the central to graft-versus-host disease (GVHD), a life-threatening complication after allogeneic hematopoietic stem cell transplantation (HSCT). In GVHD host antigens are never cleared and alloreactive effector T cells are continuously generated over a period of several months or longer, but their suppression and control have proven to be difficult in practice. Using mouse models of GVHD directed against minor histocompatibility antigens (miHAs), we demonstrate that alloreactive effector T cells proliferate and persist upon chronic exposure to alloantigens via reactivation of stem cell transcriptional programs normally expressed in embryonic stem cells and neural stem cells. Many activated stem cell genes in effector T cells were distinct from those in memory T cells and were maintained at high levels upon T cell receptor activation, suggesting a specific role in chronically activated effector T cells. One of these genes, Ezh2, encodes a chromatin modifying enzyme essential to the proliferation, survival and differentiation of stem cells, was upregulated in CD8+ effector T cells upon antigenic stimulation and downregulated when the antigen was withdrawn. Pharmacologically inactivation of EZH2 with 3-Deazaneplanocin A inhibited effector T cell proliferation and survival. Silencing Ezh2 independently validated that Ezh2 was important for regulating effector T cell proliferation and expression of many stem cell genes. To further evaluate whether alloreactive CD8+ effector T cells obtained stem cell-like properties, e.g. the ability to self-renew to continually generate effector cells, we adoptively transferred highly purified miHA H60-specific (H60+) CD8+ effector T cells into secondary allogeneic and congenic recipients, respectively. As compared to congenic recipients, allogeneic recipients had 80-fold more proliferating H60+CD8+ effector T cells. These donor H60+CD8+ effector T cells expressed high levels of CD122, CD69, CXCR3, PD1, IFNγ and Granzyme B, required miHA H60 stimulation to sustain their replication with effector function and expression of stem cell genes, and caused severe GVHD in secondary allogeneic recipients. These results indicate that stem cell transcriptional programs expressed in embryonic and neural stem cells may play important roles in effector T cells. Among these stem cell genes, Ezh2 emerges as an important therapeutic target in modulating alloreactive T cell-mediated GVHD. Disclosures: Zhang: University of Michigan Comprehensive Cancer Center: Research Funding; Damon Runyon Cancer Research Foundation: Research Funding.
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40

Lummaa, Karoliina, and Siru Kainulainen. "Runon puheenomaisuudesta." AVAIN - Kirjallisuudentutkimuksen aikakauslehti, no. 3 (September 1, 2008): 64–68. http://dx.doi.org/10.30665/av.74742.

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41

Elovaara, Raili. "Koukussa Tavaststiernan runoon." AVAIN - Kirjallisuudentutkimuksen aikakauslehti, no. 3 (September 1, 2006): 65–67. http://dx.doi.org/10.30665/av.74668.

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42

Gillison, M. L., B. S. Glisson, E. O’Leary, B. A. Murphy, M. A. Levine, M. S. Kies, D. Chan, and A. A. Forastiere. "Phase II trial of trastuzumab (T), paclitaxel (P) and cisplatin (C) in metastatic (M) or recurrent (R) head and neck squamous cell carcinoma (HNSCC): Response by tumor EGFR and HER2/neu status." Journal of Clinical Oncology 24, no. 18_suppl (June 20, 2006): 5511. http://dx.doi.org/10.1200/jco.2006.24.18_suppl.5511.

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5511 Background: EGFR expression is associated with poor prognosis and resistance of HNSCC to therapy. EGFR/Her2/neu heterodimers may potentiate receptor signaling and therapy resistance. T enhances cytotoxic effects of C and P in Her2/neu+ cell lines. Methods: A phase II trial evaluated the response rate (RR) of HNSCC pts to TPC as a function of Her2/neu and EGFR expression by immunohistochemistry (IHC). Secondary outcomes included toxicity, one-year progression-free (PFS) and overall (OS) survival. Eligible pts had M or R HNSCC, ECOG PS 0–1, CrCl >50 ml/min, and ANC >1500. Chemotherapy regimen consisted of P (175 mg/m2) and C (75 mg/m2) IV on day 1 and T (4 mg/kg day 1, cycle 1, 2 mg/kg subsequent) IV on day 1, 8 and 15 of 21. Paraffin embedded tumor was analyzed for Her2/neu (HercepTest) and EGFR (Zymed Lab) expression by IHC and for Her2/neu amplification by FISH (−/+) at LabCorp. Two-stage Simon design had 80% power for a 15% improvement in RR over 35% and required response in ≥ 14/42 pts by ECOG response criteria. Results: 61 pts (55 R HNSCC) received a median of 4 cycles (range 1–14) of TPC. Membrane staining of ≥ 10% of cells was observed for Her2/neu in 4/58 (6.9%, 95% CI 2–17) and for EGFR in 41/57 (72%, 95% CI 58–83). Her2/neu amplification was absent in 55/55 (0%, one-sided 97.5% CI 0–6.7) tumors. A RR of 36% (95% CI 24–50) was observed in 58 evaluable pts. RR was lower in pts with ≥ 10% staining by EGFR (25% versus 62.5%, p = 0.01). Her2/neu expression had no effect on RR (p = 0.75). Toxicities included two grade 5 dehydration/hypokalemia, and grade 3–4 neutropenia, fatigue, infection, nausea, vomiting, and neuropathy were common. Median follow-up was 4.2 yrs. For all 61 pts: median TTP was 4.3 mos, PFS 19.8% (95% CI 10.6–30.9) and OS 44% (95% CI 31.6–56.2) at 1 yr. Pts with <10% EGFR membrane staining had improved median PFS (6.7 vs 3.1 mos, p = 0.003) and OS (16.1 vs 7.4 mos, p = 0.005) when compared to patients with tumors with ≥ 10% EGFR. Conclusions: T did not improve RR to PC, likely because Her2/neu gene amplification and expression was rare. Tumor EGFR status significantly affected RR, PFS, and OS to the underlying regimen of PC. Sponsored by Bristol-Myers Squibb, Genentech, and Damon Runyon Cancer Research Foundation (MG). [Table: see text]
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43

Stuart, Anthony J., and Adrian M. Lister. "Introduction: The West Runton Freshwater Bed and the West Runton Mammoth." Quaternary International 228, no. 1-2 (December 2010): 1–7. http://dx.doi.org/10.1016/j.quaint.2010.07.035.

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44

Seutu, Katja. "Puheenomaisen runon rakenteesta." AVAIN - Kirjallisuudentutkimuksen aikakauslehti, no. 2 (June 1, 2006): 21–36. http://dx.doi.org/10.30665/av.74657.

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45

Crapanzano, Vincent. "Crapanzano's Response to Runyan." American Anthropologist 88, no. 1 (March 1986): 183. http://dx.doi.org/10.1525/aa.1986.88.1.02a00230.

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46

Sejati, Irfanda Rizki Harmono, and Ghazian Arka Fikry. "Proses Kreativitas Aransemen Lagu Lir-Ilir Oleh Grup Musik Rungon Wresthi." Musikolastika: Jurnal Pertunjukan dan Pendidikan Musik 4, no. 1 (June 25, 2022): 10–17. http://dx.doi.org/10.24036/musikolastika.v4i1.79.

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Creativity is a thought or idea that exists in a person to create new things that have never been thought of by others, including in terms of arrangement. In the arrangement there is a process of creation, because the music to be arranged is the basic material, while the music that has been arranged must contain overall plus values ​​both in chords, melodies, accompaniment patterns, fillers and counter melodies. Rungon Wresthi is one of the music groups located in the city of Semarang. This musical group has its own uniqueness and charm in presenting its musical creativity. This study uses a qualitative research method with descriptive exposure, with the aim of revealing the creative method and background of the Rungon Wresthi music group in composing songs. The results of this study can be seen from the four elements of creativity from Rhodes' theory which show how Rungon Wresthi has creativity in creating works which in this context are in the form of Lir-Ilir song arrangements.
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Stuart, A. J., and A. M. Lister. "The West Runton Freshwater Bed and the West Runton Mammoth: Summary and conclusions." Quaternary International 228, no. 1-2 (December 2010): 241–48. http://dx.doi.org/10.1016/j.quaint.2010.07.033.

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48

Newsom, Brent. "Tania Runyan, Second Sky: Poems." Christianity & Literature 65, no. 1 (November 10, 2015): 124–28. http://dx.doi.org/10.1177/0148333115601626.

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Price, Sally, and Richard Price. "Rundown." New West Indian Guide / Nieuwe West-Indische Gids 67, no. 1-2 (January 1, 1993): 101–8. http://dx.doi.org/10.1163/13822373-90002677.

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[First paragraph]Our 1993 rundown of books that have not, for one reason or another, been reviewed in the NWIG follows the culinary metaphors of its precedents, "Caribbean pepper-pot" (NWIG 58:89-98) and "Callaloo" (NWIG 66:95-99). Cassidy & Le Page's Dictionary ofJamaican English (Cambridge University Press, 1967) offers, s.v. rundown:A kind of sauce made by boiling coconut down till it becomes like custard (but stops short of becoming oil). In it may be cooked salt or pickled fish, banana, or other ingredients.It is served in a bowl in the middle of the table, into which one dips one's bread-kind. See dip-and-come-back.Under "dip-and-come-back," we find thirty-six alternative terms for the dish, including: dip-and-fall-back, dip-and-shake-off, assistant, bread-fruit remedy, dip-dip, dividen-an-flabub, duck-and-shake-back, elbow-grease, frigasi, johnny run-down, kobijong, kuochi waata, malongkontong, mulgrave, pakassa, plaba, plomi, rege, round-the-road, stew-down, swimmerdown, tap-i-a-paas....
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Becker, H. F., G. Mayer, and T. Penzel. "Schlafst�rungen und schlafbezogene Atmungsst�rungen." Der Internist 45, no. 1 (January 1, 2004): 57–84. http://dx.doi.org/10.1007/s00108-003-1108-0.

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