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Relevant bibliographies by topics / S100A8

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Academic literature on the topic 'S100A8'

Author: Grafiati

Published: 4 June 2021

Last updated: 25 July 2025

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Journal articles on the topic "S100A8"

1

Mitrović Ajtić, Olivera, Tijana Subotički, Miloš Diklić, et al. "Regulation of S100As Expression by Inflammatory Cytokines in Chronic Lymphocytic Leukemia." International Journal of Molecular Sciences 23, no. 13 (2022): 6952. http://dx.doi.org/10.3390/ijms23136952.

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The calcium-binding proteins S100A4, S100A8, and S100A9 are upregulated in chronic lymphocytic leukemia (CLL), while the S100A9 promotes NF-κB activity during disease progression. The S100-protein family has been involved in several malignancies as mediators of inflammation and proliferation. The hypothesis of our study is that S100A proteins are mediators in signaling pathways associated with inflammation-induced proliferation, such as NF-κB, PI3K/AKT, and JAK/STAT. The mononuclear cells (MNCs) of CLL were treated with proinflammatory IL-6, anti-inflammatory IL-10 cytokines, inhibitors of JAK

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Broome, Ann-Marie, David Ryan, and Richard L. Eckert. "S100 Protein Subcellular Localization During Epidermal Differentiation and Psoriasis." Journal of Histochemistry & Cytochemistry 51, no. 5 (2003): 675–85. http://dx.doi.org/10.1177/002215540305100513.

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S100 proteins are calcium-activated signaling proteins that interact with target proteins to modulate biological processes. Our present studies compare the level of expression, and cellular localization of S100A7, S100A8, S100A9, S100A10, and S100A11 in normal and psoriatic epidermis. S100A7 and S100A11 are present in the basal and spinous layers in normal epidermis. These proteins appear in the nucleus and cytoplasm in basal cells but are associated with the plasma membrane in spinous cells. S100A10 is present in basal and spinous cells, in the cytoplasm, and is associated with the plasma mem

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Peterova, Eva, Jan Bures, Paula Moravkova, and Darina Kohoutova. "Tissue mRNA for S100A4, S100A6, S100A8, S100A9, S100A11 and S100P Proteins in Colorectal Neoplasia: A Pilot Study." Molecules 26, no. 2 (2021): 402. http://dx.doi.org/10.3390/molecules26020402.

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S100 proteins are involved in the pathogenesis of sporadic colorectal carcinoma through different mechanisms. The aim of our study was to assess tissue mRNA encoding S100 proteins in patients with non-advanced and advanced colorectal adenoma. Mucosal biopsies were taken from the caecum, transverse colon and rectum during diagnostic and/or therapeutic colonoscopy. Another biopsy was obtained from adenomatous tissue in the advanced adenoma group. The tissue mRNA for each S100 protein (S100A4, S100A6, S100A8, S100A9, S100A11 and S100P) was investigated. Eighteen biopsies were obtained from the he

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Calaf, Gloria M., Luis N. Ardiles, and Leodan A. Crispin. "Role of Calcium in an Experimental Breast Cancer Model Induced by Radiation and Estrogen." Biomedicines 12, no. 11 (2024): 2432. http://dx.doi.org/10.3390/biomedicines12112432.

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Background: Breast cancer, a global health challenge, significantly impacts women worldwide, causing morbidity, disability, and mortality. Objectives: To analyze the role of genes encoding S100 calcium-binding proteins and their relationship with radiation as possible markers in breast carcinogenesis. Methods: The normal MCF-10F cell line was used to study the role of ionizing radiation and estrogen to induce distinct stages of malignancy giving rise to an in vitro experimental breast cancer model. Results: Analysis of an Affymetrix system revealed that the gene expression levels of the S100 c

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Li, Changyou, Siyuan Li, Changkai Jia, Lingling Yang, Zicheng Song, and Yiqiang Wang. "Low Concentration of S100A8/9 Promotes Angiogenesis-Related Activity of Vascular Endothelial Cells: Bridges among Inflammation, Angiogenesis, and Tumorigenesis?" Mediators of Inflammation 2012 (2012): 1–8. http://dx.doi.org/10.1155/2012/248574.

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Previous studies showed that several members of the S100A family are involved in neovascularization and tumor development. This study checked whether low concentrations of S100A8 or S100A9 has any effect on the behaviour of vascular endothelial cells. A human umbilical vascular endothelial cell (HUVEC) line was used to measure vascular endothelial cell bioactivity related to angiogenesis, such as cell proliferation, migration, and vessel formation. In the low concentration range up to 10 μg/mL, either each alone or in combination, S100A8 and S100A9 proteins promoted proliferation of HUVEC cell

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Wakiya, R., T. Kameda, K. Ueeda, et al. "Hydroxychloroquine modulates elevated expression of S100 proteins in systemic lupus erythematosus." Lupus 28, no. 7 (2019): 826–33. http://dx.doi.org/10.1177/0961203319846391.

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Objectives We investigated the effect of hydroxychloroquine (HCQ) on S100A8 and S100A9 serum levels in systemic lupus erythematosus (SLE) patients with low disease activity receiving immunosuppressants. Methods SELENA-SLEDAI, Cutaneous Lupus Erythematous Disease Area and Severity Index (CLASI) and serum levels of complement factors, anti-dsDNA antibodies, and white blood cell, lymphocyte, and platelet counts were used to evaluate disease activity, cutaneous disease activity, and immunological activity, respectively. Serum S100A8 and S100A9 were measured at HCQ administration and after 3 or 6 m

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Hu, Shao-yan, Ming-ying Zhang, Shui-yan Wu, et al. "High Transcription Levels Of S100A8 and S100A9 In Acute Myeloid Leukemia Are Predictors For Poor Overall Survival." Blood 122, no. 21 (2013): 2610. http://dx.doi.org/10.1182/blood.v122.21.2610.2610.

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Abstract S100A8 and S100A9 are two members of the S100 calcium-binding protein family, preferentially form functional heterodimers of S100A8/S100A9, and have been increasingly recognized as biomarkers in malignancies. Recent proteomic studies revealed that S100A8 and S100A9 played pivotal roles in hematologic malignancies and elevated expression of S100A8/S100A9 implicated in glucocorticoid resistance in MLL-rearranged infant acute lymphoblastic leukemia (ALL). In addition, S100A8 proteomic expression in leukemic cells was reported to predict survival in AML patients. However, information on t

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Barabé, Frédéric, Malika Laouedj, and Philippe Tessier. "Myeloid-Related Protein S100A9 Induces Cellular Differentiation in Acute Myeloid Leukemia through TLR2 and TLR4 Receptors." Blood 126, no. 23 (2015): 3858. http://dx.doi.org/10.1182/blood.v126.23.3858.3858.

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Abstract The myeloid-related proteins S100A8 (MRP8) and S100A9 (MRP14) are endogenous alarmins abundantly and constitutively expressed by myeloid cells (neutrophils, monocytes and immature myeloid cells). S100A8 and S100A9 proteins exist as homodimers but also associate to form the heterodimer calprotectin (S100A8/A9) and are up-regulated in several inflammatory diseases and human cancers. In patients with acute myeloid leukemia (AML), the concentration of S100A8/A9 in serum is elevated and the expression of S100A8 correlates with poor prognosis. However, the role of these proteins in hematolo

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Jukic, A., R. Hilbe, L. Zundel, et al. "DOP028 The resolution of fecal calprotectin configurations and their biological functions in gut inflammation." Journal of Crohn's and Colitis 19, Supplement_1 (2025): i134. https://doi.org/10.1093/ecco-jcc/jjae190.0067.

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Abstract Background Fecal calprotectin (CP) serves as a worldwide established biomarker used for the diagnosis and management of inflammatory bowel diseases (IBD). However, the biological function of CP in the human gut is still unknown and numerous studies obtained contradictory results. Calprotectin is mainly described to be present as a tetrameric protein complex (S100A8/S100A9)2, while the quaternary protein structure (configuration) of S100A8 and S100A9 in the human gut lumen remains enigmatic.1 In this study we resolved the fecal protein configurations of calprotectin and their biologica

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Wolf, Marc, Robiya Joseph, Judith Austermann, et al. "S100A8/S100A9 Integrates F-Actin and Microtubule Dynamics to Prevent Uncontrolled Extravasation of Leukocytes." Biomedicines 11, no. 3 (2023): 835. http://dx.doi.org/10.3390/biomedicines11030835.

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Immune reactions are characterized by the rapid immigration of phagocytes into sites of inflammation. Meticulous regulation of these migratory processes is crucial for preventing uncontrolled and harmful phagocyte extravasation. S100A8/S100A9 is the major calcium-binding protein complex expressed in phagocytes. After release, this complex acts as a proinflammatory alarmin in the extracellular space, but the intracellular functions of these highly abundant proteins are less clear. Results of this study reveal an important role of S100A8/S100A9 in coordinated cytoskeleton rearrangement during mi

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Dissertations / Theses on the topic "S100A8"

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Betz, Christine [Verfasser], and Oliver [Akademischer Betreuer] Einsle. "Structural characterization of the metal-binding ligands S100A8/S100A9 and S100B of the receptor for advanced glycation end products = Strukturelle Charakterisierung der metallbindenden Liganden S100A8/S100A9 und S100B des Rezeptors für Advanced Glycation End Products." Freiburg : Universität, 2013. http://d-nb.info/1115813455/34.

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Leukert, Nadja. "Molekulare Charakterisierung verschiedener Komplexformen der Calcium-bindenden Proteine S100A8 und S100A9." [S.l. : s.n.], 2003. http://deposit.ddb.de/cgi-bin/dokserv?idn=967777062.

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van, Hummel Annika Elise. "The Roles of S100A8 and S100A9 in Cartilage: Degradation and Formation." Thesis, The University of Sydney, 2014. http://hdl.handle.net/2123/11521.

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Osteoarthritis (OA) is a debilitating joint disease that increases in prevalence with age, with latest figures show that around 2.4 million Australians suffer from OA. There are currently no disease-modifying drugs available and due to poor management options, the number of joint replacement surgeries is increasing by nearly 10% per annum in Australia. S100A8 and S100A9 calcium-binding proteins are expressed by immune cells, and are involved in inflammatory situations, including inflammatory diseases such as rheumatoid arthritis, where there is an increase in circulating and local (synovial fl

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Baker, Jonathan Richard. "S100A8 in development." Thesis, University College London (University of London), 2008. http://discovery.ucl.ac.uk/1444146/.

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S100 proteins are a family of Ca2+ binding EF-hand proteins. S100A8 is a cytosolic protein expressed in myeloid cells and epithelia where it forms a stable heterodimer with another S100 protein family member, S100A9. The S100A9 null mouse is viable and has no gross defect whereas the S100A8 null mouse is embryonic lethal. It was originally proposed that the S100A8 null mouse is lethal at E 9.0 in development due to lack of expression at E 6.5 in ectoplacental cone cells. This thesis shows that the S100A8 null phenotype is more complex than originally thought. S100A8 has a role in preimplantati

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Raquil, Marie-Astrid. "Études des rôles pro-inflammatoires et prolifératifs des protéines S100A8 et S100A9." Thesis, Université Laval, 2008. http://www.theses.ulaval.ca/2008/25415/25415.pdf.

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Sikora, Kristin [Verfasser]. "RAGE-abhängige S100A8- und S100A9-Expression in humanen THP-1 Zellen / Kristin Sikora." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2009. http://d-nb.info/1023749920/34.

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Ludwig, Stefan [Verfasser]. "Die S100-Proteine S100A8 und S100A9 sowie der Heterodimerkomplex S100A8/A9 im Serum und Plasma als Marker des Prostatakarzinoms : Untersuchungen zu präanalytischen Einflussfaktoren und zur diagnostischen Differenzierung zwischen benigner Prostatahyperplasie und Prostatakarzinom / Stefan Ludwig." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2008. http://d-nb.info/1023022486/34.

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Laouedj, Malika. "Effets des protéines S100A8 et S100A9 dans la différenciation cellulaire dans la leucémie myéloïde aiguë." Doctoral thesis, Université Laval, 2017. http://hdl.handle.net/20.500.11794/27761.

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Tableau d’honneur de la Faculté des études supérieures et postdoctorales, 2016-2017<br>Les leucémies myéloïdes aiguës (LMA) sont des hémopathies rares, mais très agressives. Elles résultent d’un dérèglement du processus d’hématopoïèse qui se caractérise par une prolifération incontrôlée de cellules sanguines immatures engagées dans la lignée myéloïde. En dépit des traitements actuels qui reposent sur l’utilisation d’agents chimiothérapeutiques ciblant les cellules en prolifération, le pronostic des patients souffrants de LMA est très sombre. En effet, seuls 30% des patients souffrants de LMA s

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Defrêne, Joan. "Fonctions des protéines S100A8 et S100A9 dans la réponse inflammatoire associée aux maladies auto-immunes." Doctoral thesis, Université Laval, 2020. http://hdl.handle.net/20.500.11794/66869.

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De nos jours, les maladies auto-immunes concernent une part grandissante de la population mondiale et s’accompagnent d’une comorbidité importante, mais aussi d’un lourd fardeau économique. Parmi ces maladies les plus courantes, on distingue l'arthrite rhumatoïde et le psoriasis affectant respectivement les articulations et la peau et qui ne possèdent toujours pas de traitements curatifs. Dans la pathogenèse de ces maladies, une forte réponse inflammatoire est notamment générée et entretenue, contribuant ainsi à la dégradation des tissus ciblés. De nombreux marqueurs de l’inflammation sont sécr

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Endoh, Yasumi Medical Sciences Faculty of Medicine UNSW. "New mechanisms modulating S100A8 gene expression." Publisher:University of New South Wales. Medical Sciences, 2008. http://handle.unsw.edu.au/1959.4/42942.

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S100A8 is a highly-expressed calcium-binding protein in neutrophils and activated macrophages, and has proposed roles in myeloid cell differentiation and host defense. Functions of S100A8 are not fully understood, partly because of difficulties in generating S100A8 knockout mice. Attempts to silence S100A8 gene expression in activated macrophages and fibroblasts using RNA interference (RNAi) technology were unsuccessful. Despite establishing validated small interfering RNA (siRNA) systems, enzymaticallysynthesized siRNA targeted to S100A8 suppressed mRNA levels by only 40% in fibroblasts activ

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Books on the topic "S100A8"

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Tirkos, Sam. Investigation of S100A8 and S100A9 as potential genetic modifiers of the pulmonary phenotype in cystic fibrosis mice. National Library of Canada, 2003.

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2

Wicki, Roland. Characterization of the S100 gene cluster on human chromosome 1q21 and analysis of transcriptional control elements of the potential tumor suppressor gene S100A2 in breast epithelial cells. [s.n.], 1997.

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Dubois, Josephine. Untersuchung von linearen und zirkulären Transkripten des TRAM1- und S100A6-Genlokus im Kontext des Harnblasenkarzinoms. Springer Fachmedien Wiesbaden, 2022. http://dx.doi.org/10.1007/978-3-658-40358-4.

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White, Alexander S. Photographer's guide to the canon powershot s100. White Knight Press, 2011.

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Brant, Stephen. Distribution of renal S100 proteins in psysiological and pathological models. University of East London, 2000.

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McKendry, Amanda Jayne. Comparison of HMB45 monoclonal antibody and S100 poprtein in the immunohistochemical diagnosis of melanoma. The Author], 1993.

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Ruiz, Rafael. S100b: Serum Detection, Functions and Clinical Significance. Nova Science Publishers, Incorporated, 2015.

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Dubois, Josephine. Untersuchung Von Linearen und Zirkulären Transkripten des TRAM1- und S100A6-Genlokus Im Kontext des Harnblasenkarzinoms. Springer Fachmedien Wiesbaden GmbH, 2023.

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9

Cuna. S100 Money & Negotiable Instruments. Kendall Hunt Pub Co, 1994.

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World Cup 1994 (LADYBD/S100). Ladybird Books Ltd, 1994.

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Book chapters on the topic "S100A8"

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Sreejit, Gopalkrishna, Sunil Kiran Nooti, Baskaran Athmanathan, and Prabhakara Reddy Nagareddy. "S100A8/A9 in Myocardial Infarction." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9030-6_46.

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Gharibyan, Anna L., Dina Raveh, and Ludmilla A. Morozova-Roche. "S100A8/A9 Amyloidosis in the Ageing Prostate: Relating Ex Vivo and In Vitro Studies." In Methods in Molecular Biology. Humana Press, 2012. http://dx.doi.org/10.1007/978-1-61779-551-0_26.

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Donato, Rosario, Guglielmo Sorci, and Ileana Giambanco. "S100A6." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101531.

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Donato, Rosario, Guglielmo Sorci, and Ileana Giambanco. "S100A6." In Encyclopedia of Signaling Molecules. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101531-1.

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Yatime, Laure. "Structural Analysis of S100A8 Complex with Zinc and Calcium: A General Protocol for the Study of S100 Proteins in the Presence of Divalent Cations by X-Ray Crystallography." In Methods in Molecular Biology. Springer New York, 2019. http://dx.doi.org/10.1007/978-1-4939-9030-6_26.

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Prudovsky, Igor, Thallapuranam Krishnaswamy Suresh Kumar, and Rosario Donato. "S100a13." In Encyclopedia of Signaling Molecules. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-67199-4_101530.

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Prudovsky, Igor, Thallapuranam Krishnaswamy Suresh Kumar, and Rosario Donato. "S100a13." In Encyclopedia of Signaling Molecules. Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4614-6438-9_101530-1.

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Gressner, A. M., and O. A. Gressner. "S100A12-Protein." In Lexikon der Medizinischen Laboratoriumsdiagnostik. Springer Berlin Heidelberg, 2017. http://dx.doi.org/10.1007/978-3-662-49054-9_2726-1.

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Gressner, A. M., and O. A. Gressner. "S100A12-Protein." In Springer Reference Medizin. Springer Berlin Heidelberg, 2019. http://dx.doi.org/10.1007/978-3-662-48986-4_2726.

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House, Reniqua P., Sarah C. Garrett, and Anne R. Bresnick. "Moving Aggressively: S100A4 and Tumor Invasion." In Signaling Pathways and Molecular Mediators in Metastasis. Springer Netherlands, 2011. http://dx.doi.org/10.1007/978-94-007-2558-4_4.

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Conference papers on the topic "S100A8"

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Crowe, L. A. N., M. Akbar, K. Patommel, et al. "AB0068 Alarmins s100a8 and s100a9 modulate the inflammatory microenvironment in early tendinopathy." In Annual European Congress of Rheumatology, EULAR 2018, Amsterdam, 13–16 June 2018. BMJ Publishing Group Ltd and European League Against Rheumatism, 2018. http://dx.doi.org/10.1136/annrheumdis-2018-eular.7019.

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Lee, Eunmi, Maria Ouzounova, Raziye Piranlioglu, et al. "Abstract 2956: Chemical library screen identifies compounds that target S100A8/S100A9 complex and MDSC accumulation." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-2956.

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Nazarov, K. V., Y. A. Shevchenko, R. Y. Perik-Zavodskii, et al. "ACUTE BLOOD LOSS INDUCES INCREASED EXPRESSION OF THE PAMP GENE CLEC5A IN MURINE BONE MARROW ERYTHROID CELLS." In XI МЕЖДУНАРОДНАЯ КОНФЕРЕНЦИЯ МОЛОДЫХ УЧЕНЫХ: БИОИНФОРМАТИКОВ, БИОТЕХНОЛОГОВ, БИОФИЗИКОВ, ВИРУСОЛОГОВ, МОЛЕКУЛЯРНЫХ БИОЛОГОВ И СПЕЦИАЛИСТОВ ФУНДАМЕНТАЛЬНОЙ МЕДИЦИНЫ. IPC NSU, 2024. https://doi.org/10.25205/978-5-4437-1691-6-30.

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Hemorrhage, a condition that accompanies most physical trauma cases, affects all bone marrow lineages, including erythroid nucleated cells. In this study, we studied the immune response genes transcriptome of murine erythroid cells to acute blood loss using NanoString immune transcriptome profiling. We observed that acute blood loss up-regulate pathogen-associated molecular pattern sensing gene Clec5a in post-acute blood loss murine bone marrow erythroid cells. We believe that the up-regulation of the Clec5a gene in bone marrow erythroid cells could help bone marrow erythroid cells detect and

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Ghavami, Saeid, Thomas Vogl, Johannes Roth, Helmut Unruh, and Andrew J. Halayko. "S100A8 And S100A9 Homo-, And Hetero-Dimers Affect Extracellular Matrix In Human Smooth Muscle With Different Down Stream Signalling." In American Thoracic Society 2012 International Conference, May 18-23, 2012 • San Francisco, California. American Thoracic Society, 2012. http://dx.doi.org/10.1164/ajrccm-conference.2012.185.1_meetingabstracts.a6682.

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Rastrygina, V. A., A. S. Kazakov, E. I. Deryusheva, et al. "INTERACTION OF S100P AND S100A6 PROTEINS WITH THE FOUR-HELICAL CYTOKINES." In X Международная конференция молодых ученых: биоинформатиков, биотехнологов, биофизиков, вирусологов и молекулярных биологов — 2023. Novosibirsk State University, 2023. http://dx.doi.org/10.25205/978-5-4437-1526-1-208.

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Recent studies have revealed the ability of S100 proteins to affect cell signaling via direct interaction with cytokines. This work considers interaction of promiscuous representatives of S100 protein family with numerous four-helical cytokines. The binding of ca 71/73 % of the studied cytokines to S100P/S100A6 proteins respectively was established.

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Ghavami, S., T. Hynes, T. Vogl, et al. "A Role for S100A8/A9 in Allergic Airway Inflammation?." In American Thoracic Society 2009 International Conference, May 15-20, 2009 • San Diego, California. American Thoracic Society, 2009. http://dx.doi.org/10.1164/ajrccm-conference.2009.179.1_meetingabstracts.a6326.

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Litus, Ekaterina, Marina Shevelyova, Alisa Vologzhannikova та ін. "S100A8 Interaction with Amyloid-β Peptide Suppresses Its Fibrillation". У IECBM 2024. MDPI, 2024. http://dx.doi.org/10.3390/proceedings2024103001.

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Bahmed, K., C. R. Lin, S. Bolla, N. Marchetti, G. J. Criner, and B. Kosmider. "S100A8-MCM3 Axis in Alveolar Type II Cells in Emphysema." In American Thoracic Society 2024 International Conference, May 17-22, 2024 - San Diego, CA. American Thoracic Society, 2024. http://dx.doi.org/10.1164/ajrccm-conference.2024.209.1_meetingabstracts.a4299.

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Gemicioglu, Bilun, Ozgur Yasar, and Tulay Akcay. "Significance of serum YKL-40, S100A8, S100A9, calprotectin, periostin and LRG1 levels in patients with newly diagnosed, controlled and uncontrolled asthma." In ERS International Congress 2018 abstracts. European Respiratory Society, 2018. http://dx.doi.org/10.1183/13993003.congress-2018.pa2023.

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Liya, LI, Zuo Xiaoxia, Xiao Yizhi, DI Liu, Luo Hui, and Zhu Honglin. "AB0211 NEUTROPHIL-DERIVED EXOSOME S100A8/A9 INHIBITS ANGIOGENESIS IN SYSTEMIC SCLEROSIS." In Annual European Congress of Rheumatology, EULAR 2019, Madrid, 12–15 June 2019. BMJ Publishing Group Ltd and European League Against Rheumatism, 2019. http://dx.doi.org/10.1136/annrheumdis-2019-eular.6484.

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Reports on the topic "S100A8"

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ฤทธิ์ประจักษ์, พัชรี, та ประสิทธิ์ ภวสันต์. ผลของแรงเชิงกลต่อการเกิดการละลายภายในคลองรากฟันและ การพัฒนาแนวทางในการรักษา : รายงานความก้าวหน้าผลการดำเนินงานปีที่ 1. จุฬาลงกรณ์มหาวิทยาลัย, 2014. https://doi.org/10.58837/chula.res.2014.11.

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การบาดเจ็บจากแรงเชิงกลส่งผลให้เกิดการละลายภายในคลองรากฟัน ซึ่งเป็นปัญหาหนึ่งของการสูญเสียฟัน ดังนั้นการศึกษากลไกการเกิดการละลายภายในคลองรากฟันจึงมีผลต่อการพัฒนาแนวทางในการรักษาผู้ป่วยทางคลินิก ในงานวิจัยนี้คณะผู้วิจัยจึงมีวัตถุประสงค์หลักในการศึกษากลไกการเกิดการละลายภายในคลองรากฟัน โดยคณะผู้วิจัยมีสมมุติฐานเบื้องต้นว่าความเครียดจากแรงเชิงกล ส่งผลให้เกิดการอักเสบของเซลล์โพรงฟัน ทำให้เซลล์โพรงฟันหลั่งสารที่มีผลต่อการแปรสภาพและการทำงานของเซลล์สลายกระดูก/สลายฟัน ซึ่งเซลล์ดังกล่าวนี้จะไปทำลายเนื้อฟันและทำให้เกิดลักษณะทางคลินิกของการละลายภายในคลองรากฟัน จากผลการทดลองพบว่าเมื่อเซลล์โพรงฟันได้รับความเ

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2

ฤทธิ์ประจักษ์, พัชรี, та ประสิทธิ์ ภวสันต์. ผลของแรงเชิงกลต่อการเกิดการละลายภายในคลองรากฟันและการพัฒนาแนวทางในการรักษา : รายงานฉบับสมบูรณ์. จุฬาลงกรณ์มหาวิทยาลัย, 2015. https://doi.org/10.58837/chula.res.2015.16.

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การบาดเจ็บจากแรงเชิงกลส่งผลให้เกิดการละลายภายในคลองรากฟัน ซึ่งเป็นปัญหาหนึ่งของการสูญเสียฟัน ดังนั้นการศึกษากลไกการเกิดการละลายภายในคลองรากฟันจึงมีผลต่อการพัฒนาแนวทางในการรักษาผู้ป่วยทางคลินิก ในงานวิจัยนี้คณะผู้วิจัยจึงมีวัตถุประสงค์หลักในการศึกษากลไกการเกิดการละลายภายในคลองรากฟัน โดยคณะผู้วิจัยมีสมมุติฐานเบื้องต้นว่าความเครียดจากแรงเชิงกล ส่งผลให้เกิดการอักเสบของเซลล์โพรงฟัน ทำให้เซลล์โพรงฟันหลั่งสารที่มีผลต่อการแปรสภาพและการทำงานของเซลล์สลายกระดูก/สลายฟัน ซึ่งเซลล์ดังกล่าวนี้จะไปทำลายเนื้อฟันและทำให้เกิดลักษณะทางคลินิกของการละลายภายในคลองรากฟัน จากผลการทดลองของคณะผู้วิจัยพบว่าเมื่อเซลล์โพรง

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3

Emberley, Ethan D., and Peter Watson. The Role of S100A7/RANBPM Interaction in Human Breast Cancer. Defense Technical Information Center, 2001. http://dx.doi.org/10.21236/ada396984.

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4

Emberley, Ethan, and Peter Watson. The Role of S100A7/RANBPM Interaction in Human Breast Cancer. Defense Technical Information Center, 2002. http://dx.doi.org/10.21236/ada412819.

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5

Emberley, Ethan D., and Peter Watson. The Role of S100A7/RANBPM Interaction in Human Breast Cancer. Defense Technical Information Center, 2003. http://dx.doi.org/10.21236/ada418754.

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6

West, Nathan. Exploring and Exploiting the Protein S100A7 as a New Target for Breast Cancer Therapy. Defense Technical Information Center, 2010. http://dx.doi.org/10.21236/ada520729.

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7

Kim, Edward J., and David Helfman. Characterization of Molecular Factors Critical to the S100A4 (A Metastasis-Associated Protein) - Dependent Increase in Motility of Breast Cancer Cells. Defense Technical Information Center, 2004. http://dx.doi.org/10.21236/ada424207.

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8

State Savings Bank of Tasmania - Hobart (Chief Office) - Depositors Ledgers - Hobart - Accounts - S1 - S1000 -1908-1912. Reserve Bank of Australia, 2021. http://dx.doi.org/10.47688/rba_archives_2006/20525.

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9

OVERHANG EFFECT ON WEB CRIPPLING CAPACITY OF COLDFORMED AUSTENITIC STAINLESS STEEL SHS MEMBERS: AN EXPERIMENTAL STUDY. The Hong Kong Institute of Steel Construction, 2022. http://dx.doi.org/10.18057/icass2020.p.343.

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This paper studies the overhang effects on ultimate bearing capacities of cold-formed austenitic stainless steel square hollow section (SHS) members undergoing web crippling between EndTwo-Flange (ETF) and Interior-Two-Flange (ITF) loading conditions. A total of 16 web crippling tests were conducted with specimens covering various overhang lengths. Tensile coupon tests were performed to obtain the material properties of the test specimens. The web crippling capacities obtained from the tests were compared with the nominal capacities predicted by the SEI/ASCE 8-22 Specification for the design o

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10

Government Savings Bank of New South Wales - Balmain - Depositors Ledgers - Friendly Society (Loose Leaf System) Cheque Accounts S1-S100 (incl. Continuation) - 1930-1933. Reserve Bank of Australia, 2021. http://dx.doi.org/10.47688/rba_archives_2006/22603.

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