Relevant bibliographies by topics / Salmonella Enteritidi

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Salmonella Enteritidi'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Salmonella Enteritidi"

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Wu, Daichao, Da Teng, Xiumin Wang, Changsong Dai, and Jianhua Wang. "Saccharomyces boulardii prevention of the hepatic injury induced by Salmonella Enteritidis infection." Canadian Journal of Microbiology 60, no. 10 (2014): 681–86. http://dx.doi.org/10.1139/cjm-2014-0259.

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Salmonella enterica subsp. enterica serovar Enteritidis (Salmonella Enteritidis) is the predominant cause of serovar-associated food-borne outbreaks in many countries and causes significant clinical symptoms of liver injury, enteritis, and diarrheal diseases. Saccharomyces boulardii is used in clinical application for prophylaxis and the treatment of a variety of diseases caused by bacterial infection. We used a mouse model of Salmonella Enteritidis infection, which included pretreatment with S. boulardii, to reveal the protection mechanisms of S. boulardii against Salmonella Enteritidis infection, including the translocation of Salmonella Enteritidis to the liver 10 days after Salmonella Enteritidis challenge, and the colonisation of Salmonella Enteritidis and the formation of hepatic tissue lesions in mice after Salmonella Enteritidis challenge on the 10th day. Compared with Salmonella Enteritidis infection in mice, S. boulardii decreased Salmonella Enteritidis translocation to the liver by 96%, and 99% of Salmonella Enteritidis colonised the cecum on the 10th day. Saccharomyces boulardii also abated hepatic tissue injury caused by the infiltration of neutrophilic granulocytes, lymphocytes, and plasmocytes by decreasing the translocation of Salmonella to the liver. These findings demonstrated that S. boulardii is an effective agent in the prevention of the hepatic injury induced by Salmonella Enteritidis infection in a mouse model.
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Poppe, C., R. J. Irwin, S. Messier, G. G. Finley, and J. Oggel. "The prevalence of Salmonella enteritidis and other Salmonella spp. among Canadian registered commercial chicken broiler flocks." Epidemiology and Infection 107, no. 1 (1991): 201–11. http://dx.doi.org/10.1017/s0950268800048822.

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SUMMARYA nation-wide survey was conducted to estimate the prevalence of Salmonella enteritidis and other salmonellas among Canadian commercial broiler flocks. Environmental (litter and/or water) samples from 226 of 294 (76·9%) randomly selected flocks were contaminated with salmonellas. Litter samples were more often contaminated with salmonellas than water samples (47·4 ν 12·3%). Fifty different salmonella serovars were isolated. The most prevalent serovars were S. hadar, S. infantis, and S. schwarzengrund; they were isolated from samples of 98/294 (33·3%), 26/294 (8·8%), and 21/294 (7·1%) flocks, respectively. Feed samples of 39/290 (13·4%) flocks were contaminated with salmonellas. Salmonella enteritidis was isolated from the environmental samples of 9/294 (3·1%) flocks. Salmonella enteritidis phage type (PT) 8 was isolated from seven flocks, and PT 13a from two flocks.
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Poppe, C., R. J. Irwin, C. M. Forsberg, R. C. Clarke, and J. Oggel. "The prevalence of Salmonella enteritidis and other Salmonella spp. among Canadian registered commercial layer flocks." Epidemiology and Infection 106, no. 2 (1991): 259–70. http://dx.doi.org/10.1017/s0950268800048408.

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SUMMARYA survey was conducted to estimate the prevalence of Salmonella enteritidis and other salmonellas among Canadian commercial egg producing flocks. Environmental (faecal and eggbelt) samples from 152 of 295 (52·9%) randomly selected flocks were contaminated with salmonellas. Thirty-five different salmonella serovars were isolated. Eggbelt samples were more often contaminated with salmonellas than faecal samples (25·7 v. 10·1 %). The most prevalent serovars were S. heidelberg, S. infantis, S. hadar, and S. schwarzengrund; they were isolated from samples of 59/295 (20%), 18/295 (6·1%), 17/295 (5·8%), and 15/295 (5·1%) flocks, respectively. Feed samples of 21/295 (7·2%) flocks were contaminated with salmonellas. Salmonella enteritidis was isolated from the environmental samples of 8/295 (2·7%) flocks. Salmonella enteritidis phage type (PT) 8 was isolated from 5 flocks, PT 13a from 2 flocks, and PT 13 from 1 flock.
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SHIROTA, KAZUTOSHI, HIROMITSU KATOH, TOSHIYUKI MURASE, TOSHIHIRO ITO, and KOICHI OTSUKI. "Monitoring of Layer Feed and Eggs for Salmonella in Eastern Japan between 1993 and 1998." Journal of Food Protection 64, no. 5 (2001): 734–37. http://dx.doi.org/10.4315/0362-028x-64.5.734.

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In order to investigate contamination of chicken farms with Salmonella, feed and eggs were sampled from 16 commercial layer farms in eastern Japan between 1993 and 1998 and cultured for salmonellae. Salmonella enterica subsp. enterica isolates belonging to 19 serovars were obtained from the feed. Six of the 19 serotypes, including Salmonella serovar Enteritidis, were observed in isolates recovered from the eggs. Salmonella serovar Enteritidis strains obtained from a feed sample and egg contents in a layer farm showed pulsed-field gel electrophoresis patterns that were genetically related and belonged to a single phage type, suggesting that the contamination of the farms was linked to the occurrence of salmonellae in feed.
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JERNGKLINCHAN, JAOWAPA, CHAILAI KOOWATANANUKUL, KRIENGSAG DAENGPROM, and KRIENGSAG SAITANU. "Occurrence of Salmonellae in Raw Broilers and Their Products in Thailand." Journal of Food Protection 57, no. 9 (1994): 808–10. http://dx.doi.org/10.4315/0362-028x-57.9.808.

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A study was conducted to determine the presence of salmonellae in raw chicken meat, giblets (liver, heart, gizzard) and cooked chicken products (meatballs and sausages) in Bangkok. A total of 1,135 samples, collected from nine open markets, nine supermarkets and four poultry processing plants, were examined. Salmonellae were isolated from 467 (66%) of 705 chicken meat samples, 190 (86%) of 221 samples of giblets and 21 (10%) of 209 cooked products. Out of 678 tested isolates, 46 serotypes and one rough strain were found. The five most common serotypes isolated from chicken meat were Salmonella blockley, Salmonella virchow, Salmonella enteritidis, Salmonella hadar and Salmonella paratyphi B; these accounted for 14, 12, 12, 9 and 9%, respectively, of the strains isolated in this study. The major isolates from giblets were S. virchow, Salmonella Kentucky, S. enteritidis, Salmonella agona and S. blockley (15, 13, 12, 12 and 11%, respectively). Salmonella derby (33%) was the serotype most often isolated from the cooked poultry products.
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SARLIN, LAURA L., ERIC T. BARNHART, RANDLE W. MOORE, DONALD E. CORRIER, LARRY H. STANKER, and BILLY M. HARGIS. "Comparison of Enrichment Methods for Recovery and Chick Infectivity of Chlorine-lnjured Salmonella enteritidis." Journal of Food Protection 61, no. 11 (1998): 1504–6. http://dx.doi.org/10.4315/0362-028x-61.11.1504.

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In recent years, several preenrichment media have been shown to be effective for use in the recovery of sublethally injured Salmonella organisms. Selective enrichment without preenrichment has resulted in a lower recovery of organisms, particularly with regard to injured or stressed salmonellae. The present experiments compared the ability of nonselective preenrichment followed by selective enrichment or direct selective enrichment alone to recover chlorine-injured Salmonella organisms. Additionally, the Salmonella detection limits of the two enrichment methods were compared with minimal infectious dose in neonatal chicks. In three experiments, Salmonella enteritidis cells were exposed to chlorine for specific times and subsequently cultured by using preenrichment followed by selective enrichment or selective enrichment alone. Simultaneously, neonatal chicks were orally challenged with S. enteritidis cells from each exposure time to chlorine. The results indicated a marginal, but significantly (P < 0.05) higher level of recovery of sublethally injured salmonellae by using nonselective preenrichment followed by selective enrichment, as compared to selective enrichment alone. Interestingly, both culture methods were capable of detecting injured S. enteritidis cells at levels incapable of infecting neonatal chicks.
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Jaki Tkalec, Vesna, Željko Cvetnić, Marina Mikulić, et al. "Učestalost serovarova Salmonella spp. u pilećem mesu s područja sjeverozapadne Hrvatske." Veterinarska stanica 52, no. 4 (2021): 387–96. http://dx.doi.org/10.46419/vs.52.4.11.

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Salmoneloza je jedna od najučestalijijh zoonoza koja se prenosi hranom, a najčešći izvor zaraze za ljude je kontaminirano meso i prerađevine od mesa peradi. Tijekom petogodišnjeg razdoblja od 2016. do 2020. godine provedeno je istraživanje tijekom kojeg je na Salmonellu spp., pretraženo 2457 uzoraka pilećeg mesa koje je uzorkovano u klaonicama i mesnicama na području Međimurske, Varaždinske, Koprivničko- križevačke, Bjelovarsko-bilogorske i Zagrebačke županije. Salmonella spp. je izdvojena iz 136 (5,5 %) obrađenih uzoraka. Godine 2016. ustvrđena je u 5 (6 %) pretraženih uzoraka, 2017. godine u 41 (4,7 %) uzorku, 2018. godine u 33 (6,1 %) uzorka, 2019. godine u 26 (6,6 %) uzoraka i u 2020. godini u 31 (5,4 %) uzoraka. Serološkom tipizacijom S. Infantis je identificirana u 86 (63,2 %) izdvojenih izolata; S. Typhimurium u 8 (5,9 %) izolata; a S. Enteritidis je tipizirana u 3 (2,2 %) izdvojena izolata. Tipizirani su i slijedeći serovarovi salmonela: S. Corvallis - 5 izolata (3,7 %), S. Isaszeg - 5 izolata (3,7 %), S. Derby- 3 izolata (2,2 %), S. Give - 2 izolata (2,2 %), S. Indiana - 2 izolata (2,2 %), i po 1 izolat 7 (5,1 %) serovara (S. Schwarzengrund, S. Goldcoast, S. Chester, S. Bredeney, S. Mbandaka, S.Newport, S. Saintpaul). U 15 (11 %) izolata tipizacija nije izvršena. S. Infantis je tijekom svih godina bila najčešće potvrđeni serovar. Salmoneloza predstavlja znatan gospodarski problem zbog šteta u intenzivnoj proizvodnji, ali i kao zoonoza koja se mesom i mesnim prouzvodima od mesa peradi širi na ljude. Provedbom odgovarajućih higijenskih mjera i dobre higijenske prakse od peradarskih farmi i klaonica do prodajnih mjesta moglo bi se doprinijeti manjoj kontaminaciji pilećeg mesa različitim serovarovima Salmonella spp.
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Cohen, Noah D., Deeann E. Wallis, Holly L. Neibergs, and Billy M. Hargis. "Detection of Salmonella Enteritidis in Equine Feces using the Polymerase Chain Reaction and Genus-Specific Oligonucleotide Primers." Journal of Veterinary Diagnostic Investigation 7, no. 2 (1995): 219–22. http://dx.doi.org/10.1177/104063879500700209.

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Salmonella was identified in feces from horses, using the polymerase chain reaction (PCR) and genus-specific oligonucleotide primers. Feces from healthy horses were determined to be culture negative and PCR negative for Salmonella. Fecal samples were inoculated with known numbers of colony-forming units (CFU) of S. enteritidis. The fecal samples were enriched overnight in tetrathionate broth, and then DNA was extracted and amplified by PCR using genus-specific primers. Sensitivity of the assay extended to 100 CFU Salmonella enteritidis/g feces; sensitivity of microbiologic culture with enrichment extended to 100 CFU Salmonella enteritidis/g feces. Feces that were not inoculated with S. enteritidis were negative by the PCR. Detection of salmonellae in feces was possible using the PCR within 24 hours from the time of submission of samples. Because samples were enriched, isolates were available for determining antibiograms and serologic grouping or typing.
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CARLI, K. TAYFUN, AYSEGUL EYIGOR, and VILDAN CANER. "Prevalence of Salmonella Serovars in Chickens in Turkey." Journal of Food Protection 64, no. 11 (2001): 1832–35. http://dx.doi.org/10.4315/0362-028x-64.11.1832.

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In this study, 151 (18.6%) of 814 ceca obtained during in-line processing of 28 broiler (Hybro G, Avian, Arbor acres, and Cobb breeds) and 5 layer (Ross, Tetra SL, Isa Brown, and Brown Nick breeds) flocks in Turkey were found to be contaminated with four different Salmonella serovars. Only Salmonella enterica subsp. enterica Serovar Enteritidis (Salmonella Enteritidis) was recovered from layer birds, whereas Salmonella Enteritidis (81.5%), Salmonella Agona (7.6%), Salmonella Thompson (10.1%), and Salmonella Sarajane (0.8%) were isolated from broiler birds. Isolations of Salmonella Agona and Salmonella Thompson from poultry are reported for the first time in Turkey. The isolation of Salmonella Sarajane from chickens is the first report in the world. The standard method of National Poultry Improvement Plan, U.S. Department of Agriculture, was used to detect Salmonella from chicken cecal samples. Primary and delayed secondary enrichments (PE and DSE) were done in tetrathionate-Hajna broth (TTHB). Two different agar media, xylose lysine tergitol 4 (XLT4) and brilliant green with novobiocin (BGN) were used to observe, and compared for their isolation and selective differentiation of, Salmonella-suspected colonies. Isolated salmonellae were then biotyped and serotyped. Ninety-one and 151 salmonellae were isolated with XLT4 agar after PE and DSE, respectively. From the same samples, BGN agar was able to detect only 50 and 131 Salmonella after PE and DSE, respectively. The isolation rate with XLT4 was 11.2% (P < 0.01) with PE, and this rate increased to 18.6% after DSE. Also, the PE isolation rate (11.2%) with XLT4 agar was significantly higher (P < 0.01) than PE with BGN agar (6.1%). Salmonella was isolated from 39.3% (11 of 28) of the broiler flocks and from 60.0% (3 of 5) of the layers. The detection sensitivity of the isolation method was determined as 1 CFU g−1 experimentally. These data demonstrate the presence of Salmonella Enteritidis, Salmonella Thompson, Salmonella Agona, and Salmonella Sarajane in chicken flocks in Turkey.
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METZER, E., V. AGMON, N. ANDOREN, and D. COHEN. "Emergence of multidrug-resistant Salmonella enterica serotype Typhimurium phage-type DT104 among salmonellae causing enteritis in Israel." Epidemiology and Infection 121, no. 3 (1998): 555–59. http://dx.doi.org/10.1017/s0950268898001526.

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The relative frequency of salmonella strains isolated from hospitalized and non-hospitalized patients in Southern Israel changed during the period, 1994–6. Salmonella enterica serotype Typhimurium definitive phage-type 104 (DT104) appeared in Israel in 1994 and became the most prevalent strain in 1996. An outbreak of enteritis due to Salmonella enterica serotype Agona occurred in Israel, in October 1994 and lasted for 4 months. The relative frequency of Salmonella enterica serotype Enteritidis remained almost constant during these years, with seasonal fluctuations only.The importance of the increase in the prevalence of Typhimurium DT104 has been the epidemic spread of a multiresistant strain of R-type ACT (A, ampicillin; C, chloramphenicol; T, tetracycline) belonging to this phage-type. Since 1995 the frequency of Typhimurium DT104 isolates that possess, in addition to the above R-type, a chromosomally encoded resistance to the quinolone drug, nalidixic acid, increased tenfold. In 1996, 27% of the Typhimurium DT104 isolates were of R-type ACTN. S. Enteritidis exhibited over 95% susceptibility to at least eight of the most commonly used antibiotic drugs, and none of the isolates was resistant to quinolone or fluoroquinoline.
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Dissertations / Theses on the topic "Salmonella Enteritidi"

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Silva, Oyama Rodrigues da. "Modelo teórico de estimativa de risco de Salmonella Enteritidis em sistema integrado de produção de frango de corte e tipagem molecular de Salmonella spp. oriundas de aves e rações submetidas a diferentes tratamentos com ácido." Universidade de São Paulo, 2007. http://www.teses.usp.br/teses/disponiveis/42/42132/tde-28082008-160245/.

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O presente trabalho objetivou identificar os fatores de risco para a presença de S. Enteritidis no sistema de produção de frangos de corte, avaliar, qualificar e quantificar as variáveis encontradas e elaborar um modelo teórico de estimativa de risco deste sorovar em frangos criados em sistema de integração. Os dados foram obtidos de trabalhos recentes realizados por alguns autores e deram subsídios à realização de uma análise de riscos microbiológicos. Para caracterização molecular foram utilizadas 42 cepas de Salmonella isoladas de frangos e rações inoculados experimentalmente com uma cepa de S. Typhimurium. A inoculação da bactéria foi realizada na ração e a mesma tratada com diferentes concentrações dos ácidos propílico, fórmico e acético sendo, então, fornecida para consumo ad libitum até os 21 dias de idade, quando as aves foram sacrificadas. Foram obtidos diferentes perfis genéticos com o uso do ERIC e BOX-PCR, que se mostraram eficientes para discriminação das cepas em estudo.<br>The aim of this work was identify the risk factors for S. Enteritidis in the production system of broiler chickens, to evaluate, qualify and quantify the variables studied and to make a theoretical model of risk assessment of this serovar in broilers in integration system. Therefore, the data was obtain from works of some authors and supported the proposed model of microbiological risk analysis. For molecular characterization were included 42 Salmonella spp. strains isolated from chicks and feed experimentally inoculated with S. Tiphimurium. After inoculation of feed with the specific dose of strain, it was submitted to treatment with propilic, formic and acetic acids in several concentrations and it was given to birds ad libitum until 21 days old, when they were sacrificed. It was obtained different patterns through the ERIC and BOX-PCR techniques, which showed good discrimination power for the strains analyzed.
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Machado, Taís Raquel Marcon. "Avaliação da aderência ao aço inoxidável e ao polietileno por três sorovares de Salmonella e da capacidade de desinfecção dessas superfícies." reponame:Biblioteca Digital de Teses e Dissertações da UFRGS, 2007. http://hdl.handle.net/10183/32453.

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No Rio Grande do Sul, a Salmonella Enteritidis vem se destacando como o principal microrganismo causador de surtos de doenças transmitidas por alimentos. A capacidade desse sorovar em aderir ao aço inoxidável e ao polietileno, assim como a resistência a desinfetantes comumente utilizados nas indústrias de alimentos foi avaliada e comparada com outros dois sorovares de Salmonella. Para mensurar a aderência bacteriana, corpos de prova de aço inoxidável (2 x 2 x 0,1cm) e de polietileno (2 x 2 x 0,7cm) permaneceram em contato com as culturas bacterianas por 15, 30 e 60 minutos e então ultrasonicados, para a realização das contagens de células aderidas. A resistência bacteriana aos desinfetantes foi avaliada através do teste de suspensão como preconizado pela legislação brasileira. Para desinfecção das superfícies, os corpos de prova permaneceram por 15 minutos em contato com as culturas bacterianas e, após dez minutos de exposição aos desinfetantes, o número de células sobreviventes foi determinado. A aderência bacteriana após os tempos de exposição indicou que a S. Typhimurium aderiu significativamente mais ao aço inoxidável que ao polietileno, enquanto que a S. Bredeney aderiu mais ao polietileno. Entretanto, não houve diferenças significativas nos níveis de aderência do sorovar S. Enteritidis, mesmo que as análises de microscopia eletrônica de varredura e de hidrofobicidade tenham indicado diferenças significativas entre os materiais. Foi observada a produção de bioemulsificante pelos três sorovares de Salmonella, sendo que a S. Enteritidis e a S. Bredeney produziram quantidades bem maiores que a S. Typhimurium. A resistência aos desinfetantes ácido peracético, hipoclorito de sódio e quaternário de amônio, quando avaliados pelo teste de suspensão, demonstraram que, nas concentrações indicadas pelo fabricante, os três compostos foram capazes de inativar os três sorovares de Salmonella. Entretanto, na concentração de 200ppm de hipoclorito de sódio comumente utilizada no Brasil, o sorovar S. Enteritidis foi o mais resistente, uma vez que sobreviveu até 15 minutos de exposição. Nenhum desinfetante conseguiu inativar todas as células aderidas (aproximadamente 5log UFC/cm2) em aço inoxidável e polietileno, exceto o quaternário de amônio que eliminou totalmente a S. Enteritidis do aço inoxidável. Tendo em vista a importância desses microrganismos como patógenos alimentares, cuidados especiais devem ser tomados nos processos de desinfecção e contaminação cruzada por Salmonella.<br>In Rio Grande do Sul, southern Brazil, Salmonella Enteritidis have been considered the principal microorganism responsible for foodborne disease. The ability of this sorovar in adhering to stainless steel and polyethylene, as well as the resistance to biocides commonly used in food industries was evaluated and compared with other two serovars of Salmonella. To measure the bacterial adherence, coupons of stainless steel (2 x 2 x 0,1cm) and polyethylene (2 x 2 x 0,7cm) remained in contact with the bacterial cultures for periods of 15, 30 e 60 minutes and so that ultrassonicated for count the adherent cells. The bacterial resistance to biocides was evaluated through the suspension test as praised for the Brazilian legislation. For surfaces disinfection, the coupons remained for 15 minutes in contact with bacterial cultures and after ten minutes of exposition to biocides, the surviving cells were determined. Bacterial adherence after 15, 30, and 60 minutes of exposure indicated that S. Typhimurium adhered significantly more to stainless steel than to polyethylene, whereas S. Bredeney adhered more to polyethylene than to stainless steel. However, there was no significative difference in adherence levels of S. Enteritidis, even when analysis of sacanning electronic microscopy has indicated expressive differences between adherence to the materials. The production of bioemulsifier by Salmonella serovars was observed, being that S. Enteritidis and S. Bredeney produced larger amounts than S. Typhimurium. The resistance to peracetic acid, and quaternary ammonium biocides when evaluated using suspension test in the presence of organic matter, demonstrated that in the concentrations indicated by manufacturers, all of the three biocides were able to inactivate the three serovars. However, using 200ppm of sodium hypochlorite, commonly used in Brazil, S. Enteritidis showed to be the most resistant serovar, since it has survived for up to 15 minutes of exposure. None of the biocides inactivate all the cells adhered (approximately 5log CFU/cm2) in stainless steel and polyethylene, except the quaternary ammonium which totally eliminated S. Enteritidis on the stainless steel surface. In view of the importance of these micorrganismos as alimentary pathogens, special cares of disinfection processes and cross-contamination for Salmonella must be taken.
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Alexandrou, Olga. "Sub-lethal injury to Salmonella enteritidis." Thesis, University of Surrey, 1997. http://epubs.surrey.ac.uk/843803/.

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The effect of acids on the growth, survival and detection of Salmonella enteritidis PT4 is particularly important in view of the number of outbreaks in which mayonnaise has been implicated as a vehicle. Capacitance measurement was compared with colony counting procedures for the enumeration and determination of sub-lethal injury of Salmonella enteritidis during storage under varied conditions of pH, acidulant and temperature. Capacitance monitoring was shown to offer an improved technique for the measurement of sub-lethal injury in cell populations. Higher levels of sub-lethal injury were detected by the extension of capacitance detection time than were indicated by differential colony counts on selective and non-selective media. The extension of detection time noted with sub-lethally injured cell populations was shown to be due to an extended lag phase when cells were placed in the capacitance growth medium and not the result of delayed detection of the growth of a small, uninjured sub-population. Plots of percentage survival and extension of detection time in survivors gave similar curves for acetic and lactic acid. These acids showed both greater lethality and greater ability to inflict sub-lethal injury than the stronger citric or hydrochloric acid. Sub-lethal injury and lethality were not simply related, as little sub-lethal injury was observed with the stronger acids even under conditions that were ultimately highly lethal. The results indicate that weak organic acids cause more reversible damage to cellular sites prior to death: an observation that has implications for choice of resuscitation procedures when examining acidified foods. Injured cells were found to contain lower levels of ATP than healthy unstressed cells. Inhibition with chloramphenicol did not appear to increase injury and total protein patterns for injured and uninjured cells were similar, suggesting that protection afforded by the synthesis of stress proteins is not a significant factor in this case. The recovery of sub-lethally injured cells in various pre-enrichment and selective enrichment media using capacitance detection times and colony counts on selective and non selective media was determined. Buffered peptone water appeared more effective in recovering injured Salmonella compared to Lactose broth. Additionally, selenite cystine was shown to recover cells faster than the other two selective broths tested; the Muller-Kauffmann Tetrathionate broth and the Rappaport Vassiliadis enrichment broth. In this study, different injury conditions were applied; these included acid stress, heating and freezing. According to the lag phases of injured cell populations, short pre-enrichment is not recommended in the present study.
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White, Aaron Paul. "A novel, fimbrial-based heterologous Salmonella vaccine system." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 2000. http://www.collectionscanada.ca/obj/s4/f2/dsk1/tape2/PQDD_0009/NQ52778.pdf.

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Fardini, Yann Velge Philippe. "Étude du contrôle de l'expression des systèmes de sécrétion de type III, généré par l'inactivation du gène yfgL codant une lipoprotéine de la membrane externe, chez Salmonella Enteritidis." aTours : Service commun de documentation, 2008. http://theses.abes.fr/2008TOUR3301.

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Howard, Zoe R. "Invasion of avian reproductive tissues by Salmonella typhimurium and Salmonella enteritidis." Texas A&M University, 2003. http://hdl.handle.net/1969/275.

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Tam, Connie Kwai Ping. "Rapid inversion of the salmonella enterica shufflon : a new molecular mechanism for control of pathogenesis /." View abstract or full-text, 2005. http://library.ust.hk/cgi/db/thesis.pl?BICH%202005%20TAM.

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Leiva, Araya Lorenzo Eugenio. "Contribución del sistema de secreción tipo VI codificado en la isla genómica SPI-6 a los mecanismos de virulencia de Salmonella entérica serovar typhimurium." Tesis, Universidad de Chile, 2013. http://www.repositorio.uchile.cl/handle/2250/113484.

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Magíster en Bioquímica área de Especialización en Bioquímica de Proteínas Recombinantes<br>Memoria de título de bioquímico<br>Los sistemas de secreción tipo VI (T6SS) corresponden a un mecanismo de interacción célula-célula ampliamente distribuido entre bacterias Gram negativo. Si bien inicialmente al T6SS se le atribuyó un papel en la virulencia de los microorganismos, estudios posteriores dieron cuenta de su versatilidad, indicando que el sistema también toma parte en relaciones mutualistas o comensales entre bacterias y eucariontes, además de relaciones de competencia interbacteriana. Salmonella Typhimurium codifica un T6SS en la isla de patogenicidad SPI-6 (T6SSSPI-6), sin embargo el rol que cumple en la patogénesis de Salmonella aún no ha sido aclarado. Resultados obtenidos en nuestro laboratorio indican que mutantes de este sistema presentan una menor colonización de órganos internos, tanto en ratones BALB/c como en pollos White Leghorn infectados oralmente. Considerando que los componentes celulares del sistema inmune son la principal puerta de entrada de Salmonella para el desarrollo de la infección sistémica, se planteó como hipótesis de este trabajo que “el Sistema de Secreción Tipo VI codificado en la isla genómica SPI-6 de Salmonella enterica serovar Typhimurium se expresa en el interior de macrófagos de origen murino y aviar, favoreciendo la supervivencia bacteriana en estas células”. Para probar esta hipótesis el objetivo fue evidenciar la expresión, funcionalidad y contribución del T6SS durante la interacción de S. Typhimurium con macrófagos murinos y aviares. Para determinar la expresión del T6SS durante la infección de macrófagos, se construyó el vector pLZ01 que permitio la generación de fusiones transcripcionales y traduccionales a la proteína fluorescente verde (GFP) en Salmonella, mediante recombinación homóloga de productos de PCR. De esta manera, se fusionaron componentes estructurales del T6SSSPI-6 (VgrG, Hcp-1, Hcp-2) a GFP y se evaluó su transcripción y traducción en ensayos de infección in vitro mediante microscopía de epifluorescencia. Por otra parte, para determinar si el sistema es translocado al citoplasma de macrófagos durante la infección de S. Typhimurium, se estudió la translocación de una fusión traduccional de VgrG a la β-lactamasa TEM1, construida en el plasmidio pFlagTEM1. La translocación de las fusiones fue determinada mediante un ensayo de pérdida de FRET de la cefalosporina CCF2, observado mediante microscopía de epifluorescencia y cuantificado mediante fluorometría. Finalmente, para determinar la contribución del T6SS en los procesos de internalización y supervivencia en macrófagos se realizaron ensayos de protección con gentamicina. En ellos se comparó la capacidad de la cepa silvestre para invadir y sobrevivir en el interior de macrófagos, versus mutantes que carecen de todo el T6SSSPI-6 o poseen un T6SSSPI-6 no funcional debido a la mutación de clpV, ATPasa esencial para este sistema. Todos los experimentos se realizaron en líneas de macrófagos murinos (RAW264.7) y aviares (HD11), utilizando cepas derivadas de S. Typhimurium 14028s. Los resultados mostraron que ninguno de los componentes estructurales estudiados (VgrG, Hcp-1, Hcp-2) del T6SSSPI-6 de S. Typhimurium se transcribe y traduce en el medio de cultivo celular, sin embargo su transcripción y traducción es gatillada al infectar tanto macrófagos murinos como aviares. A pesar de observar la transcripción y traducción de VgrG, no se detectó su translocación al citoplasma de las células infectadas. Contrariamente a lo esperado, se observó que la presencia del T6SSSPI-6 no contribuye a la supervivencia en el interior de macrófagos murinos o aviares, pero sí tendría una implicancia en la etapa de internalización de Salmonella, puesto que al utilizar mutantes con un T6SSSPI-6 no funcional se observó un fenotipo de mayor internalización en ambos modelos celulares. Estos resultados permiten aceptar una parte de la hipótesis planteada, ya que el Sistema de Secreción Tipo VI codificado en la isla genómica SPI-6 de Salmonella enterica serovar Typhimurium se expresa en el interior de macrófagos de origen murino y aviar, y rechazar una segunda parte de la hipótesis, pues este sistema no tendría un rol en la supervivencia bacteriana en estas células. No obstante, el aumento en la capacidad de internalización de mutantes del T6SS indica que el sistema tendría un rol durante la infección de los macrófagos.<br>Type VI Secretion Systems (T6SS) correspond to a widely distributed cell-cell interaction mechanism in Gram-negative bacteria. Although initially the T6SS was attributed a role in the virulence of microorganisms, subsequent studies realized its versatility, indicating that this system also takes part in comensal or mutualistic relationships between bacteria and eukaryotes, as well as interbacterial competition. Salmonella Typhimurium encodes a T6SS in the pathogenicity island SPI-6 (T6SSSPI-6), however the role of this island in the pathogenesis of Salmonella has not been clarified. Results obtained in our laboratory indicate that mutants of this system generate a phenotype of reduced colonization of internal organs, both in orally infected BALB/c mice and White Leghorn chicken. Because the initial contact of Salmonella with cellular components of the immune system is the main gateway for the development of systemic infection of Salmonella, the objective of this work was to determine the expression, functionality and contribution of the T6SS during S. Typhimurium interaction with murine and avian macrophages. The vector pLZ01was built to determine the expression of the T6SS during infection of macrophages. This plasmid enables the generation of transcriptional and translational fusions to the green fluorescent protein (GFP) reporter in Salmonella by homologous recombination of PCR products. In this way, structural components of the T6SSSPI-6 (VgrG, Hcp-1, Hcp-2) were merged to GFP and their transcription and translation were assessed by in vitro infection assays and epifluorescence microscopy. On the other hand, to determine whether the system is translocated to the cytoplasm of macrophages during infection of S. Typhimurium, translocation of VgrG was studied using a translational fusion of VgrG to the β-lactamase TEM1, built in the pFlagTEM1 plasmid. The translocation of the β-lactamase fusion was determined by processing of the CCF2/AM fluorescence substrate, detected by epifluorescence microscopy and quantified using fluorometry. Finally, gentamicin protection assays were performed to determine the contribution of the T6SS in the processes of internalization and survival in macrophages. In these experiments, invasion and survive inside macrophages at the wild type strain was compared to a deletion mutant of the T6SS gene cluster and a mutant on the clpV gene, which encodes the ATPase essential for the functioning of the system, All experiments were carried out in murine (RAW264.7) and avian (HD11) macrophage cell-lines, using strains derived from the sequenced wild-type S. Typhimurium 14028s strain. The results showed that none of the studied structural components (VgrG, Hcp-1, Hcp-2) of T6SSSPI-6 of S. Typhimurium are produced in cell culture media, but their transcription and translation are triggered when murine or avian macrophages are infected. Despite observing transcription and translation of VgrG, translocation of this protein into the cytoplasm of infected cells could not be detected. Contrary to expectations, it was observed that the presence of the T6SSSPI-6 did not contribute to Salmonella survival within murine or avian macrophages. However, internalization experiments showed that non-functional T6SSSPI-6 mutants showed a greater uptake into both cellular models. These results indicate that the T6SSSPI-6 of S. Typhimurium is expressed during infection of murine and avian macrophages (the first part of the hypothesis is true), however it did not have an impact on the ability of S. Typhimurium to survive inside murine or avian macrophages (the second part of the hypothesis is false). However, the increase in the internalization of the T6SS mutants suggests a novel role for the T6SS during infection of macrophages.<br>Fondecyt
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Mardones, Acuña Paula Carolina. "Identificación global de genes de Salmonella enterica serovar Gallinarum requeridos para la colonización sistémica de un hospedero murino." Tesis, Universidad de Chile, 2013. http://www.repositorio.uchile.cl/handle/2250/115384.

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Memoria para optar al Título Profesional de Bioquímico<br>Autorizada por el autor, pero con restricción para ser publicada a texto completo en el Portal de Tesis Electrónicas, hasta diciembre de 2014<br>Salmonella enterica es un patógeno intracelular Gram negativo capaz de infectar un amplio rango de hospederos. En particular, S. enterica serovar Gallinarum infecta aves de corral causando una enfermedad sistémica que puede provocar la muerte. Una vez que entra al organismo, Salmonella utiliza una serie de factores de virulencia que le permiten sobrevivir al pH ácido del estómago, resistir a sales biliares y péptidos antimicrobiales, invadir y traspasar la barrera epitelial intestinal, sobrevivir y replicarse dentro de macrófagos y diseminarse dentro de los órganos del hospedero, principalmente a nivel del bazo e hígado. Hasta el momento, se desconoce gran parte de los factores de virulencia que utiliza S. Gallinarum para infectar un hospedero. Es por eso que en este trabajo se propuso identificar los genes de S. Gallinarum involucrados en la colonización sistémica en un modelo murino a través de un análisis global de mutantes bajo selección negativa in vivo. Para ello, se utilizó una genoteca de ~48.000 mutantes por inserción del transposón EZ-Tn5<T7/KAN-2>, la que se inyectó en ratones BALB/c por vía intraperitoneal. La comparación entre la población de bacterias inyectadas (input) y la población de bacterias recuperadas a partir del bazo de los animales (output) mediante hibridaciones competitivas en un microarray genómico nos permitió obtener una base de datos en la que identificamos 280 mutantes bajo selección negativa in vivo. La lista de mutantes bajo selección negativa incluye genes descritos previamente como necesarios para la virulencia de S. enterica, como los sistemas de secreción tipo III codificados en la SPI-1 y SPI-2, genes que codifican efectores de estos sistemas (sseB, sseE), genes relacionados a la síntesis y modificación del LPS (rfaL, rfaJ, rfbK, rfbM), genes que codifican reguladores globales de la virulencia (phoP, phoQ, ompR, envZ), genes que codifican proteínas de respuesta a estrés (oxyR, rpoE, htrA), entre otros. La lista también incluye genes no reportados previamente como necesarios para la virulencia de S. Gallinarum, pero si para la virulencia de otros serovares de S. enterica, como tatB y tatC que codifican proteínas del sistema Twin-Arginine Transport; y genes con funciones desconocidas como la región génica STM3118 a STM3121 perteneciente a la SPI-13. El sistema Twin-Arginine Transport es un sistema que transporta proteínas plegadas hacia el periplasma de bacterias Gram negativo. Por otra parte, aún se desconoce la función exacta de SPI-13, pero se ha visto que es necesaria para la replicación de S. Typhimurium dentro de macrófagos murinos. A través de ensayos de competencia y complementación in vivo entre la cepa silvestre y las mutantes ΔtatABC o ΔSPI-13, se confirmó la participación de estas regiones génicas en la colonización sistémica de ratones BALB/c. Cabe destacar que la lista de mutantes bajo selección negativa in vivo no incluye ciertos genes previamente descritos como necesarios para la virulencia de S. enterica, como el gen aroA que codifica una proteína involucrada en la síntesis de compuestos aromáticos. Se realizó un ensayo de competencia con una mutante ΔaroA y se determinó que presenta una colonización sistémica deficiente en ratones BALB/c, confirmando la participación de aroA en la virulencia de esta bacteria. Finalmente, mediante este análisis global de mutantes bajo selección negativa in vivo logramos identificar genes de S. Gallinarum requeridos para la colonización sistémica eficiente de un hospedero murino, comprobando de forma independiente la participación del operón tatABC, la isla de patogenicidad SPI-13 y el gen aroA en este proceso. El análisis individual de los genes identificados en esta base de datos permitirá ampliar el conocimiento sobre los mecanismos de patogenicidad de S. Gallinarum<br>Salmonella is a Gram negative intracelular pathogen able to infect a broad range of hosts. Specifically, S. enterica serovar Gallinarum infects poultry leading to a systemic illness that may cause death. Once Salmonella enters the organism, it uses a variety of virulence factors which allows it to survive in the gastric acid, resist bile salts and antimicrobial peptides, invade and cross the intestinal epithelium, survive and grow within macrophages, and colonize internal organs of the host, mainly spleen and liver. The main virulence factors that S. Gallinarum uses to infect a host remained unknown until know. In this work we proposed to identify genes involved in the systemic colonization of S. Gallinarum in the murine model through a genome-wide screening of mutants under negative selection in vivo. To accomplish this, we used a pool of ~48.000 mutants generated by random insertion of the EZ-Tn5<T7/KAN-2> transposon to inoculate BALB/c mice intraperitoneally. The comparison between the pool of inoculated bacteria (input) and the pool of bacteria recovered from the spleen of the animals (output) through high-throughput microarray-based screening of mutants allowed us to obtain a database of 280 mutants under negative selection in vivo. Within this database we found mutants in several genes known to be required for Salmonella enterica virulence, like those related to the type III secretion system encoded in SPI-1 and SPI-2, genes encoding efectors secreted by these systems (sseB, sseE), genes related to LPS synthesis and modification (rfaL, rfaJ, rfbK, rfbM), genes encoding global virulence regulators (phoP, phoQ, ompR, envZ), and genes encoding proteins involved in response to stress (oxyR, rpoE, htrA), among others. We also found genes not previously reported as required for S. Gallinaum virulence, like tatB and tatC, encoding components of the Twin-Arginine Transport system; and genes with unknown function like the genetic region comprised by STM3118 to STM3121, belonging to SPI-13. The Twin-Arginine Transport system transports a number of folded proteins to the periplasma of Gram-negative bacteria. Besides, the exact function of SPI-13 is still unknown, but has been seen that is necessary for the growth of S. Typhimurium inside murine macrophages. Through in vivo competition and complementation assays between wild type and ΔtatABC or ΔSPI-13 mutants, we were able to confirm the important role of these genes in the systemic colonization of BALB/c mice by S. Gallinarum. Noteworthly, there were genes that we didn’t observe on our database that have been reported as required S. enterica virulence like aroA, a gene involved in the synthesis of aromatic coumpounds. Using an in vivo competition assay we observed a systemic colonization defect for the ΔaroA mutant in BALB/c mice, indicating that this gene is indeed required for S. Gallinarum virulence in this host. Overall, our genome-wide screening of mutants under negative selection in vivo allowed us to identify 280 genes of S. Gallinarum required for the systemic colonization of a murine host. Also, we confirmed the role played by the tatABC operon, SPI-13 and aroA in the systemic colonization by this serovar. The in-depth analysis of the genes identified in our screening will expand our current knowledge on the mechanisms of S. Gallinarum pathogenesis<br>FONDECYT
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Mmolawa, Princess Tlou. "Molecular analysis of temperate phages in Salmonella enterica serovar Typhimurium DT 64 isolated in Australia." Title page, contents and summary only, 2001. http://web4.library.adelaide.edu.au/theses/09PH/09phm6855.pdf.

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Files on accompanying CD-ROM: Appendix III Phages ST64T and ST64B sequences, are in rtf format. Bibliography: leaves 279-324. System requirements for accompanying CD-ROM: IBM or compatible ; Microsoft Word or compatible to read rtf files.
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Books on the topic "Salmonella Enteritidi"

1

Salmonella Enteritidis Task Force for Research. Report. Cooperative State Research Service?, 1988.

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United States. Animal and Plant Health Inspection Service. Fact sheet: Salmonella enteritidis, pathogen of people and animals. USDA, APHIS, Veterinary Services, 1990.

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Blumenthal, Dale. Salmonella enteritidis: From the chicken to the egg. Dept. of Health and Human Services, Public Health Service, Food and Drug Administratrion, Office of Public Affairs, 1991.

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Parker, James N. The Official Patient's Sourcebook on Salmonella Enteritidis Infection. ICON Group International Inc., 2002.

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United States. Congress. House. Committee on Energy and Commerce. Subcommittee on Oversight and Investigations. Salmonella poisoning in food: Hearing before the Subcommittee on Oversight and Investigations of the Committee on Energy and Commerce, House of Representatives, One Hundred First Congress, second session, July 20, 1990. U.S. G.P.O., 1990.

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United States. Congress. House. Committee on Energy and Commerce. Subcommittee on Oversight and Investigations. Salmonella poisoning in food: Hearing before the Subcommittee on Oversight and Investigations of the Committee on Energy and Commerce, House of Representatives, One Hundred First Congress, second session, July 20, 1990. U.S. G.P.O., 1990.

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United States. Congress. House. Committee on Energy and Commerce. Subcommittee on Oversight and Investigations. The outbreak of salmonella in eggs: Hearing before the Subcommittee on Oversight and Investigations of the Committee on Energy and Commerce, House of Representatives, One Hundred Eleventh Congress, second session, September 22, 2010. U.S. Government Printing Office, 2013.

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Nicholas, R. Studies on the development and application of an elisa for the detection of antibody to salmonella enteritidis in chickens and their eggs. Brunel University, 1986.

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Dyckman, Lawrence J. Food safety: Overview of Food Safety and Inspection Service and Food and Drug Administration expenditures : statement of Lawrence J. Dyckman, Director, Food and Agriculture Issues, Resources, Community, and Economic Development Division, before the Committee on Agriculture, Nutrition, and Forestry, U.S. Senate. The Office, 2000.

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Dyckman, Lawrence J. Food safety: U.S. needs a consistent farm-to-table approach to egg safety : statement of Lawrence J. Dyckman, Director, Food and Agriculture Issues, Resources, Community, and Economic Development Division, before the Subcommittee on Oversight of Government Management, Restructuring and the District of Columbia, Committee on Governmental Affairs, U.S. Senate. The Office, 1999.

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Book chapters on the topic "Salmonella Enteritidi"

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Cooper, Gerard L., Lindsay M. Venables, Robin A. J. Nicholas, Gavin A. Cullen, and Carlos E. Hormaeche. "The Application of Salmonella Enteritidis aroA Vaccines in Chickens." In Biology of Salmonella. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2854-8_52.

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Woodward, Martin J., Christopher J. Thorns, and Claude Turcotte. "Fimbriae of Salmonella Enteritidis: Molecular Analysis of SEF14 and Vaccine Development Potential." In Biology of Salmonella. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2854-8_9.

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Halavatkar, Homayoun, and Paul A. Barrow. "Preliminary Characterization of TnphoA Mutants of Salmonella Enteritidis with Reduced Invasiveness in vivo." In Biology of Salmonella. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2854-8_46.

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Tauxe, Robert V. "Salmonella Enteritidis and Salmonella Typhimurium DT104: Successful Subtypes in the Modern World." In Emerging Infections 3. ASM Press, 2014. http://dx.doi.org/10.1128/9781555818418.ch3.

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Maddox, Carol W., Suzanne E. Baker, Patricia A. Dunn, and Anthony E. Castro. "Immunoglobulin Response to Salmonella Enteritidis Outer Membrane Proteins." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1828-4_13.

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Kogut, Michael, Guillermo Tellez, Edward McGruder, Billy Hargis, and John DeLoach. "Immunoprophylaxis of Salmonella Gallinarum Infection by Salmonella Enteritidis-Immune Lymphokines in Broiler Chicks." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1828-4_64.

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Tschäpe, H., and H. Kühn. "Virulenz und Verbreitung der Enteritis-Salmonellen." In Ökosystem Darm V. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-642-78733-1_2.

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Cacemiro, Maira Costa, Milena Sobral Espíndola, Leonardo Judson Galvão-Lima, et al. "Immune Response Against Salmonella Enteritidis Is Unsettled by HIV Infection." In Advances in Experimental Medicine and Biology. Springer International Publishing, 2017. http://dx.doi.org/10.1007/5584_2017_40.

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Allen-Vercoe, E., S. Cawthraw, D. G. Newell, and M. J. Woodward. "Expression of Campylobacter Jejuni flaA Epitopes within a Modified Salmonella Flagellin Expressed in Salmonella Enteritidis." In Campylobacters, Helicobacters, and Related Organisms. Springer US, 1996. http://dx.doi.org/10.1007/978-1-4757-9558-5_126.

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Collinson, S. Karen, Sharon C. Clouthier, James L. Doran, Pamela A. Banser, and William W. Kay. "Characterization of the AgfBA Fimbrial Operon Encoding Thin Aggregative Fimbriae of Salmonella Enteritidis." In Advances in Experimental Medicine and Biology. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1828-4_37.

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Conference papers on the topic "Salmonella Enteritidi"

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Ferreira, A. A., R. C. S. Mendonça, H. M. Hungaro, M. M. Carvalho, and J. A. M. Pereira. "Bacteriophages actions on Salmonella Enteritidis biofilm." In Proceedings of the International Conference on Antimicrobial Research (ICAR2010). WORLD SCIENTIFIC, 2011. http://dx.doi.org/10.1142/9789814354868_0026.

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Gomes, Izabela Alves, Erika Fraga de Souza, Ana Paula de Oliveira Ribeiro, Vanessa Fiuza de Mello, Simone Duarte de Oliveira Costa, and Janine Passos Lima da Silva. "Sobrevivência de Salmonella Enteritidis em Maionese Caseira." In XII Latin American Congress on Food Microbiology and Hygiene. Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-267.

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Boshara, P., and M. Kousha. "Nontyphoidal Salmonella Empyema as a Complication of Primary Salmonella Enteritidis Bacteremia." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3895.

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Hayakawa, S., S. Naknukool, T. Uno, and M. Ogawa. "Antimicrobial Activity of Duck Egg Lysozyme against Salmonella enteritidis." In 13th World Congress of Food Science & Technology. EDP Sciences, 2006. http://dx.doi.org/10.1051/iufost:20060308.

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Sousa, Ronnie de, and Marcelo Brocchi. "Construção de Salmonella Sorovar Enteritidis atenuada para imunização de mamíferos." In Congresso de Iniciação Científica UNICAMP. Universidade Estadual de Campinas, 2019. http://dx.doi.org/10.20396/revpibic2720191977.

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Wessling, Claudia Regina, Cibeli Viana, Vanessa Mendonça Soares, et al. "Penetration of Salmonella Enteritidis in Chicken Breasts Stored Under Refrigeration Temperatures." In XII Latin American Congress on Food Microbiology and Hygiene. Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-117.

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Vernace, J., T. Smith, J. M. Galvis, A. Gearhart, and U. A. Gauhar. "A Fowl Effusion: A Rare Case of Empyema Secondary to Salmonella Enteritidis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6848.

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Bhunia, Arun K., Tao Geng, Amanda Lathrop, Angela Valadez, and Mark T. Morgan. "Optical immunosensors for detection of Listeria monocytogenes and Salmonella enteritidis from food." In Optical Technologies for Industrial, Environmental, and Biological Sensing, edited by Bent S. Bennedsen, Yud-Ren Chen, George E. Meyer, Andre G. Senecal, and Shu-I. Tu. SPIE, 2004. http://dx.doi.org/10.1117/12.515900.

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Nene Meltem Keklik, Ali Demirci, Virendra M Puri, and Paul H Heinemann. "Modeling the inactivation of Salmonella Typhimurium, Listeria monocytogenes, and Salmonella Enteritidis on poultry products exposed to pulsed UV-light." In 2011 Louisville, Kentucky, August 7 - August 10, 2011. American Society of Agricultural and Biological Engineers, 2011. http://dx.doi.org/10.13031/2013.37183.

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Elias, Susana, Anderson Sant’Ana, and Eduardo Tondo. "Validation of the Secondary Predictive Model Developed for Salmonella Enteritidis Se86 Growth on Homemade Mayonnaise Salad." In XII Latin American Congress on Food Microbiology and Hygiene. Editora Edgard Blücher, 2014. http://dx.doi.org/10.5151/foodsci-microal-129.

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Reports on the topic "Salmonella Enteritidi"

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Coble, Derrick, Erin Sandford, Tieming Ji, and Susan J. Lamont. Impacts of Salmonella Enteritidis Infection on Liver Transcriptome in Broilers. Iowa State University, 2012. http://dx.doi.org/10.31274/ans_air-180814-57.

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Cheeseman, Jennifer, Nyssa Levy, and Susan J. Lamont. Chemokine mRNA Expression in the Cecum of Chicks Infected with Salmonella enteritidis. Iowa State University, 2007. http://dx.doi.org/10.31274/ans_air-180814-841.

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Coble, Derrick, Sarah Beth Redmond, Ben Hale, and Susan J. Lamont. The Effect of Salmonella Enteritidis on Immune Genes in Three Different Lines of Chickens. Iowa State University, 2010. http://dx.doi.org/10.31274/ans_air-180814-603.

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Ciraci, Ceren, Michael G. Kaiser, and Susan J. Lamont. Chicken Antibody Response to Salmonella enteritidis Vaccine in Advanced Intercross Lines and Parental Lines. Iowa State University, 2009. http://dx.doi.org/10.31274/ans_air-180814-8.

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Mateva, Gergana, Karl Pedersen, Gitte Sorensen, Mia Torpdahl, and Hristo Daskalov. Genetic Polymorphism and Antimicrobial Resistance of Salmonella Enterica Serovar Enteritidis Isolates from Food Chain Sources. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2021. http://dx.doi.org/10.7546/crabs.2021.07.04.

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Redmond, Sarah Beth, and Susan J. Lamont. Genetic Differences in Chicken Heterophil mRNA Expression in Response to In-Vitro Stimulation with Salmonella enteritidis. Iowa State University, 2009. http://dx.doi.org/10.31274/ans_air-180814-755.

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Lamont, Susan J., Michael G. Kaiser, and Jennifer H. Cheeseman. Genetic Line Differences in Cytokine mRNA Expression of Peripheral Blood Leukocytes Exposed to Salmonella enteritidis In-Vitro. Iowa State University, 2005. http://dx.doi.org/10.31274/ans_air-180814-1064.

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Abasht, Behnam, Michael G. Kaiser, Jan van der Pool, and Susan J. Lamont. Toll-Like Receptor Gene Expression in Cecum and Spleen of Chicks Challenged with Salmonella Enterica Serovar Enteritidis. Iowa State University, 2008. http://dx.doi.org/10.31274/ans_air-180814-145.

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Hassena, Amal ben, Hanen Sellami, Abdelkader Bougarech, Morsi Gdoura, Caroline Amiel, and Radhouane Gdoura. Differentiation of the Salmonella enterica Serovars Enteritidis and Kentucky Using Transmittance and Reflectance FTIR Spectroscopies and Multivariate Data Analysis. "Prof. Marin Drinov" Publishing House of Bulgarian Academy of Sciences, 2021. http://dx.doi.org/10.7546/crabs.2021.04.14.

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EPIDEMIOLOGICAL CHARACTERISTIC OF SALMONELLОSIS CAUSED BY SALMONELLA ENTERITIDIS TYPE PLASMID 38:3,0:1,4 MDA. НИИ эпидемиологии и микробиологии им. Г.П. Сомова, 2016. http://dx.doi.org/10.18411/hmes.d-2016-073.

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