Relevant bibliographies by topics / Salt-inducible kinase

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Salt-inducible kinase'

Author: Grafiati
Published: 4 June 2025
Last updated: 1 August 2025

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Journal articles on the topic "Salt-inducible kinase"

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Lin, Xing-zi, Hiroshi Takemori, Yoshiko Katoh, et al. "Salt-Inducible Kinase Is Involved in the ACTH/cAMP-Dependent Protein Kinase Signaling in Y1 Mouse Adrenocortical Tumor Cells." Molecular Endocrinology 15, no. 8 (2001): 1264–76. http://dx.doi.org/10.1210/mend.15.8.0675.

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Abstract The involvement of salt-inducible kinase, a recently cloned protein serine/threonine kinase, in adrenal steroidogenesis was investigated. When Y1 mouse adrenocortical tumor cells were stimulated by ACTH, the cellular content of salt-inducible kinase mRNA, protein, and enzyme activity changed rapidly. Its level reached the highest point in 1–2 h and returned to the initial level after 8 h. The mRNA levels of cholesterol side-chain cleavage cytochrome P450 and steroidogenic acute regulatory protein, on the other hand, began to rise after a few hours, reaching the highest levels after 8
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Okamoto, Mitsuhiro, Hiroshi Takemori, and Yoshiko Katoh. "Salt-inducible kinase in steroidogenesis and adipogenesis." Trends in Endocrinology & Metabolism 15, no. 1 (2004): 21–26. http://dx.doi.org/10.1016/j.tem.2003.11.002.

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Darling, Nicola J., and Philip Cohen. "Nuts and bolts of the salt-inducible kinases (SIKs)." Biochemical Journal 478, no. 7 (2021): 1377–97. http://dx.doi.org/10.1042/bcj20200502.

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The salt-inducible kinases, SIK1, SIK2 and SIK3, most closely resemble the AMP-activated protein kinase (AMPK) and other AMPK-related kinases, and like these family members they require phosphorylation by LKB1 to be catalytically active. However, unlike other AMPK-related kinases they are phosphorylated by cyclic AMP-dependent protein kinase (PKA), which promotes their binding to 14-3-3 proteins and inactivation. The most well-established substrates of the SIKs are the CREB-regulated transcriptional co-activators (CRTCs), and the Class 2a histone deacetylases (HDAC4/5/7/9). Phosphorylation by
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Ozanne, James, Alan R. Prescott, and Kristopher Clark. "The clinically approved drugs dasatinib and bosutinib induce anti-inflammatory macrophages by inhibiting the salt-inducible kinases." Biochemical Journal 465, no. 2 (2015): 271–79. http://dx.doi.org/10.1042/bj20141165.

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We have discovered that bosutinib and dasatinib, which are protein tyrosine kinase inhibitors used in the clinic to treat human cancer, induce anti-inflammatory but block pro-inflammatory cytokine production by inhibiting the serine/threonine kinases known as the salt-inducible kinases.
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Takemori, Hiroshi, Yoshiko Katoh, Nanao Horike, Junko Doi, and Mitsuhiro Okamoto. "ACTH-induced Nucleocytoplasmic Translocation of Salt-inducible Kinase." Journal of Biological Chemistry 277, no. 44 (2002): 42334–43. http://dx.doi.org/10.1074/jbc.m204602200.

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Tesch, Roberta, Marcel Rak, Monika Raab, et al. "Structure-Based Design of Selective Salt-Inducible Kinase Inhibitors." Journal of Medicinal Chemistry 64, no. 12 (2021): 8142–60. http://dx.doi.org/10.1021/acs.jmedchem.0c02144.

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Katoh, Yoshiko, Hiroshi Takemori, Junko Doi, and Mitsuhiro Okamoto. "IDENTIFICATION OF THE NUCLEAR LOCALIZATION DOMAIN OF SALT-INDUCIBLE KINASE." Endocrine Research 28, no. 4 (2002): 315–18. http://dx.doi.org/10.1081/erc-120016802.

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Takemori, Hiroshi, and Mitsuhiro Okamoto. "Regulation of CREB-mediated gene expression by salt inducible kinase." Journal of Steroid Biochemistry and Molecular Biology 108, no. 3-5 (2008): 287–91. http://dx.doi.org/10.1016/j.jsbmb.2007.09.006.

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Robinson, Emily, Matthew Care, Reuben Tooze, and Gina Doody. "Salt-Inducible kinases are critical regulators of terminal B-cell differentiation." Journal of Immunology 200, no. 1_Supplement (2018): 171.3. http://dx.doi.org/10.4049/jimmunol.200.supp.171.3.

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Abstract The generation of antibody-secreting plasma cells is a step-wise process involving initial B-cell activation and expansion, followed by the persistence of end-stage effector cells that reside in supportive niches. The transition through these stages requires reprogramming for high levels of secretion and adaptation to the bioenergetic demands of producing immunoglobulin. To improve our understanding of the pathways involved, we applied gene co-expression network analysis to temporal data derived from in vitro differentiating human plasma cells. We identified salt-inducible kinase 1 (S
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Heap, Rachel E., Anthony G. Hope, Lesley-Anne Pearson, et al. "Identifying Inhibitors of Inflammation: A Novel High-Throughput MALDI-TOF Screening Assay for Salt-Inducible Kinases (SIKs)." SLAS DISCOVERY: Advancing the Science of Drug Discovery 22, no. 10 (2017): 1193–202. http://dx.doi.org/10.1177/2472555217717473.

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Matrix-assisted laser desorption/ionization time-of-flight (MALDI TOF) mass spectrometry has become a promising alternative for high-throughput drug discovery as new instruments offer high speed, flexibility and sensitivity, and the ability to measure physiological substrates label free. Here we developed and applied high-throughput MALDI TOF mass spectrometry to identify inhibitors of the salt-inducible kinase (SIK) family, which are interesting drug targets in the field of inflammatory disease as they control production of the anti-inflammatory cytokine interleukin-10 (IL-10) in macrophages.
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Dissertations / Theses on the topic "Salt-inducible kinase"

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Mannion, David Leslie. "Identifying novel substrates of salt-inducible kinase 2." Thesis, University of Oxford, 2016. https://ora.ox.ac.uk/objects/uuid:269df8a2-04a4-4653-9aab-0facf4cbf12f.

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Salt-inducible kinase 2 (SIK2) has previously been identified as a potential therapeutic target in high-grade serous ovarian cancer. Depletion of SIK2 sensitises ovarian cancers to paclitaxel treatment both in vitro and in xenograft models. In patients, increased tumour SIK2 expression is associated with paclitaxel resistance and correlates with increased risk of cancer progression following paclitaxel treatment. Although high-grade serous tumours are highly chemosensitive, 80-90% of patients with advanced stage disease eventually relapse due to recurrent chemotherapy-resistant disease, so the
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Patel, Kashyap. "The role of salt-inducible kinase in the control of hepatic gluconeogenesis." Thesis, University of Dundee, 2014. https://discovery.dundee.ac.uk/en/studentTheses/531ab35e-3d37-4706-a771-ec518f775d04.

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Regulation of hepatic gluconeogenesis by hormones insulin and glucagon is central to glucose homeostasis. Recent work has proposed that amongst the salt inducible kinase isoforms (SIK1, 2 and 3), members of the AMPK-related kinase family, the SIK2 isoform may play a role as signalling mediator in the control of insulin- and glucagon-regulated hepatic gluconeogenesis. However, the mechanisms of the hormonal-regulation of SIK2 in liver remain controversial, with much of the data based on the studies in non-hepatic tissues/cells. Therefore, the exact molecular regulation of SIK2 by these hormones
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Gao, Weiwei, and 高蔚为. "Salt-inducible kinases function as a host restriction to human T-cell leukemia virus type 1 transcription." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2012. http://hub.hku.hk/bib/B4818309X.

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Human T-cell leukemia virus type 1 (HTLV-1) is the causative agent of adult T-cell leukemia (ATL) and HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP). HTLV-1 Tax is the major viral transactivator and transforming protein centrally involved in the proviral transcription, transformation and proliferation of infected T-cells as well as progression of diseases caused by HTLV-1 infection. Salt-inducible kinases (SIKs) are serine/threonine protein kinases belonging to the AMPK-related kinase (AMPK-RK) family. SIK subfamily consists of three isoforms named SIK1, SIK2 and SIK3 res
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Yu-LingLiang and 梁玉玲. "Salt-inducible kinase 3 (SIK3) in prediction the prognosis of epithelial ovarian cancer." Thesis, 2017. http://ndltd.ncl.edu.tw/handle/4hb7tc.

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Matias, Emanuel Fernando Pinto Guedes Lopes. "Effects of salt-inducible kinase 1 (SIK1) ablation on intestinal ion transport modulation." Master's thesis, 2019. https://hdl.handle.net/10216/120654.

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Matias, Emanuel Fernando Pinto Guedes Lopes. "Effects of salt-inducible kinase 1 (SIK1) ablation on intestinal ion transport modulation." Dissertação, 2019. https://hdl.handle.net/10216/120654.

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Yi-HuaChen and 陳怡華. "Attenuation of Salt-inducible kinase 3 (SIK3) expression reprograms glucose metabolism in pancreatic duct adenocarcinoma." Thesis, 2018. http://ndltd.ncl.edu.tw/handle/8vaq8f.

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碩士<br>國立成功大學<br>分子醫學研究所<br>106<br>Pancreatic duct adenocarcinoma (PDAC) is known as a highly-malignant cancer, and the patients have the lowest five-year survival rate among a variety of cancers. Obviously, discovery of early diagnostic markers and development of effective anticancer drugs are urgently needed. Since PDAC has been considered as a metabolic cancer type, many lines of evidence suggest that targeting its metabolic dependency may be a potential strategy for cancer treatment in future. Salt-inducible kinase (SIK) subfamily belongs to the AMPK-related kinases (AMPK-RK). In this sub
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Chen, Wei-Hao, and 陳韋豪. "Functional interaction between Salt-inducible kinase 2 and p97/VCP regulates ER-associated degradation and application of nanodiamond in proteomic research." Thesis, 2007. http://ndltd.ncl.edu.tw/handle/27548052248400576294.

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博士<br>國立臺灣大學<br>分子醫學研究所<br>95<br>The first part of this study is to investigate function and regulation of salt-inducible kinase-2 (SIK2) in ER-associated degradation (ERAD). Here we identified p97/VCP, an ATPase associated with a variety of cellular activities, as an SIK2 interacting protein. We found that p97/VCP is an in vitro substrate of SIK2, and the ATPase activity of p97/VCP was enhanced when phosphorylated by SIK2. Furthermore, SIK2 and p97/VCP were colocalized to the ER membrane, suggesting their functional interaction may be associated with events in the ER. Expression of wild-type
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Chen, Wei-Hao. "Functional interaction between Salt-inducible kinase 2 and p97/VCP regulates ER-associated degradation and application of nanodiamond in proteomic research." 2007. http://www.cetd.com.tw/ec/thesisdetail.aspx?etdun=U0001-2507200703511100.

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Chen, Wei-Yao, and 陳煒堯. "Functional Characterization of Salt-Inducible Kinases in Secretory Glands." Thesis, 2013. http://ndltd.ncl.edu.tw/handle/96235418621824823822.

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碩士<br>國立臺灣大學<br>口腔生物科學研究所<br>101<br>AMPK is involved in the maintenance of energy balance in human body. Salt-inducible kinases (SIK1/2/3 isoforms) belong to the AMP-activated protein kinase (AMPK) family, and were first cloned from the adrenal glands of rats fed with high salt diet. Through phosphorylation and subsequent cytoplasmic sequestration of the coactivator TORC2, SIKs can down-regulate transcriptional activation of the cAMP-response element-binding protein CREB. This SIK-dependent transcriptional regulation has been shown to be an important determinant for survival and protein, lipid
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Book chapters on the topic "Salt-inducible kinase"

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Desroy, Nicolas, Christophe Peixoto, and Steve De Vos. "SALT-INDUCIBLE KINASES: AN EMERGING TARGET CLASS WITH BROAD THERAPEUTIC POTENTIAL." In Medicinal Chemistry Reviews. MEDI, Inc. Published by American Chemical Society., 2023. http://dx.doi.org/10.1021/mc-2023-vol58.ch09.

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Yoon, Sung-Hee, Cheng-Chia Tang, and Marc N. Wein. "Salt inducible kinases and PTH1R action." In Vitamins and Hormones. Elsevier, 2022. http://dx.doi.org/10.1016/bs.vh.2022.04.008.

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Conference papers on the topic "Salt-inducible kinase"

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Facchi, Cecilia, Xin Wang, Elizabeth Cartwright, and Delvac Oceandy. "BS24 Investigating the role of salt-inducible kinase 2 (SIK2) in heart." In British Cardiovascular Society Virtual Annual Conference, ‘Cardiology and the Environment’, 7–10 June 2021. BMJ Publishing Group Ltd and British Cardiovascular Society, 2021. http://dx.doi.org/10.1136/heartjnl-2021-bcs.222.

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Hsu, Keng-Fu, Yu-Ling Liang, Chin-Han Wu, and Neng-Yao Shih. "Abstract 639: Salt-inducible kinase 3 expression identifies long-term survivors of serous ovarian cancer." In Proceedings: AACR Annual Meeting 2018; April 14-18, 2018; Chicago, IL. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1538-7445.am2018-639.

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Facchi, Cecilia, Min Zi, Sukhpal Prehar, et al. "BS28 Salt-inducible kinase 2 (SIK2) as a new putative therapeutic target for heart failure." In British Cardiovascular Society Annual Conference, ‘100 years of Cardiology’, 6–8 June 2022. BMJ Publishing Group Ltd and British Cardiovascular Society, 2022. http://dx.doi.org/10.1136/heartjnl-2022-bcs.208.

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Miranda, Fabrizio, Shujuan Liu, Sandra Herrero-Gonzalez, David Mannion, Stefan Knapp, and Ahmed A. Ahmed. "Abstract 4111: The salt inducible kinase 2 (SIK2) links lipid metabolism to survival of ovarian cancer metastasis." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-4111.

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Maser, Ilona-Petra, Sonja Lacher, Marisa Stebegg, et al. "60 Development of a gene signature to predict the anti-tumor response of the salt-inducible kinase (SIK) inhibitor OMX-0407." In SITC 38th Annual Meeting (SITC 2023) Abstracts. BMJ Publishing Group Ltd, 2023. http://dx.doi.org/10.1136/jitc-2023-sitc2023.0060.

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Xin, Ling, Chang Liu, Yinhua Liu, et al. "Abstract PS4-41: Salt-inducible kinases suppress tumour function and regulate drug resistance in breast cancer." In Abstracts: 2020 San Antonio Breast Cancer Virtual Symposium; December 8-11, 2020; San Antonio, Texas. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.sabcs20-ps4-41.

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