Relevant bibliographies by topics / Sarcomagenesis

Contents

  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Sarcomagenesis'

Author: Grafiati
Published: 5 June 2025
Last updated: 2 August 2025

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Journal articles on the topic "Sarcomagenesis"

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Cardona, Andrés Felipe, Jairo Zuluaga, Hernán Carranza, Jorge Miguel Otero, and Carlos Vargas. "Sarcomagenesis." Revista Colombiana de Hematología y Oncología 1, no. 4 (2012): 30–38. http://dx.doi.org/10.51643/22562915.321.

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Los sarcomas representan un número heterogéneo de neoplasias que surgen de la transformación de algunas células mesenquimales primitivas. La evidencia ha aumentado de forma considerable respecto de las células pluripotenciales que dan origen a estos tumores y que parecen ser responsables de la iniciación, el mantenimiento, la diferenciación y la proliferación del osteosarcoma, sarcoma sinovial, rabdomiosarcoma y del sarcoma de Ewing. Se han adoptado diferentes métodos para la identificación de células primitivas en los sarcomas, tales como el uso de marcadores de superficie, la citometría de f
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Matushansky, Igor, and Robert G. Maki. "Mechanisms of Sarcomagenesis." Hematology/Oncology Clinics of North America 19, no. 3 (2005): 427–49. http://dx.doi.org/10.1016/j.hoc.2005.03.006.

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Hayashi, Takuma. "Genomic Analysis of Sarcomagenesis." Journal of Gynecology and Obstetrics Bulletin 1, no. 1 (2016): 1–2. http://dx.doi.org/10.24218/jgob.2016.01.

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Underhill, T. Michael. "Abstract IA004: Mesenchymal progenitors and sarcomagenesis." Clinical Cancer Research 28, no. 18_Supplement (2022): IA004. http://dx.doi.org/10.1158/1557-3265.sarcomas22-ia004.

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Abstract Synovial sarcoma (SS) is an aggressive soft-tissue malignancy that can arise in any area of the body, and most frequently affects adolescents and young adults. The 10-year survival rate has been estimated at 50%. SS is characterized by a pathognomonic t(X;18)(p11.2;q11.2) translocation which produces a fusion oncogene named SS18-SSX. In mouse models, expression of SS18-SSX is sufficient to drive the development of SS-like tumors. Despite recent advancements in our understanding of SS biology, the cell of origin remains undefined. In most instances, it is likely mesenchymal in nature.
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Alba-Castellón, Lorena, Raquel Batlle, Clara Francí, et al. "Snail1 Expression Is Required for Sarcomagenesis." Neoplasia 16, no. 5 (2014): 413–21. http://dx.doi.org/10.1016/j.neo.2014.05.002.

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Esperança-Martins, Miguel, Iola F.Duarte, Mara Rodrigues, et al. "On the Relevance of Soft Tissue Sarcomas Metabolic Landscape Mapping." International Journal of Molecular Sciences 23, no. 19 (2022): 11430. http://dx.doi.org/10.3390/ijms231911430.

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Soft tissue sarcomas (STS) prognosis is disappointing, with current treatment strategies being based on a “fit for all” principle and not taking distinct sarcoma subtypes specificities and genetic/metabolic differences into consideration. The paucity of precision therapies in STS reflects the shortage of studies that seek to decipher the sarcomagenesis mechanisms. There is an urge to improve STS diagnosis precision, refine STS classification criteria, and increase the capability of identifying STS prognostic biomarkers. Single-omics and multi-omics studies may play a key role on decodifying sa
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Rodriguez, Rene, Ruth Rubio, and Pablo Menendez. "Modeling sarcomagenesis using multipotent mesenchymal stem cells." Cell Research 22, no. 1 (2011): 62–77. http://dx.doi.org/10.1038/cr.2011.157.

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Radons, Jürgen. "Inflammatory stress and sarcomagenesis: a vicious interplay." Cell Stress and Chaperones 19, no. 1 (2013): 1–13. http://dx.doi.org/10.1007/s12192-013-0449-4.

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Miller, Henry E., Aparna Gorthi, Nicklas Bassani, Liesl A. Lawrence, Brian S. Iskra, and Alexander J. R. Bishop. "Reconstruction of Ewing Sarcoma Developmental Context from Mass-Scale Transcriptomics Reveals Characteristics of EWSR1-FLI1 Permissibility." Cancers 12, no. 4 (2020): 948. http://dx.doi.org/10.3390/cancers12040948.

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Ewing sarcoma is an aggressive pediatric cancer of enigmatic cellular origins typically resulting from a single translocation event t (11; 22) (q24; q12). The resulting fusion gene, EWSR1-FLI1, is toxic or unstable in most primary tissues. Consequently, attempts to model Ewing sarcomagenesis have proven unsuccessful thus far, highlighting the need to identify the cellular features which permit stable EWSR1-FLI1 expression. By re-analyzing publicly available RNA-Sequencing data with manifold learning techniques, we uncovered a group of Ewing-like tissues belonging to a developmental trajectory
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Kannan, Sarmishta, Ian Lock, Benjamin B. Ozenberger, and Kevin B. Jones. "Genetic drivers and cells of origin in sarcomagenesis." Journal of Pathology 254, no. 4 (2021): 474–93. http://dx.doi.org/10.1002/path.5617.

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Dissertations / Theses on the topic "Sarcomagenesis"

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Hall, Sarah Lynne. "Characterizing the contribution of hippo pathway dysregulation to sarcomagenesis." Thesis, University of Iowa, 2017. https://ir.uiowa.edu/etd/5769.

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Sarcomas are cancers of mesenchymal origin. Though they comprise 15-20% of childhood cancers, and have a 5-year survival rate of 16% for metastatic disease, few targeted therapies exist, and the underlying mechanisms of their development are poorly understood. Transcriptional coactivators TAZ and YAP promote cell growth and proliferation, and are constitutively activated in a number of carcinomas. Accompanying TAZ/YAP activation in these cancers is decreased expression of Hippo pathway kinases MST1/2 and LATS1/2. As the Hippo pathway is the primary negative regulator of TAZ/YAP, this provides
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Siddiqi, Sara. "The Role of Hiwi in Stem Cell Maintenance and Sarcomagenesis." Thesis, 2012. https://doi.org/10.7916/D8K64R57.

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Sarcomas are cancers of connective tissues, such as bone, adipose and cartilage, and are thought to arise from the aberrant development of the mesenchyme. As such, mesenchymal stem cells are thought to be the cell of origin for sarcomas. Genetic or epigenetic lesions at particular points during the differentiation of a mesenchymal stem cell into its terminal mesenchymal cell type are able to give rise to specific subtypes of sarcomas. Recently, a number of reports have identified elevated expression of the human Piwi homolog--called Hiwi--in a variety of human cancers, including gastric cance
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Hsueh, Cheng-Shun, та 薛丞舜. "Investigation of the Role of Nuclear Factor-kappa B (NF-κB) on Sarcomagenesis of Feline Injection Site Sarcoma". Thesis, 2019. http://ndltd.ncl.edu.tw/handle/m6zc3w.

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碩士<br>國立臺灣大學<br>分子暨比較病理生物學研究所<br>107<br>Feline injection site sarcomas (FISSs) are malignant entities of mesenchymal origin. The disease has been proven to be associated with a vaccine adjuvant, aluminum, which serves as a stimulus, continuously inducing exaggerated inflammatory and immunologic reactions. Chronic inflammation has been implicated in sarcomagenesis. Among various factors, the activation of the nuclear factor-kappa B (NF-κB) signaling pathway has been documented to promote genes associated with tumor progression, and to up-regulate the expression of tumor-promoting cytokines and
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Books on the topic "Sarcomagenesis"

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Siddiqi, Sara. The Role of Hiwi in Stem Cell Maintenance and Sarcomagenesis. [publisher not identified], 2012.

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Book chapters on the topic "Sarcomagenesis"

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Cardona, Andrés Felipe, Jairo Zuluaga, Hernán Carranza, Jorge Miguel Otero, Carlos Vargas, and León Darío Ortiz. "Sarcomagenesis." In Stem Cells in Cancer: Should We Believe or Not? Springer Netherlands, 2014. http://dx.doi.org/10.1007/978-94-017-8754-3_12.

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Cortner, J., G. F. Vande Woude, and S. Rong. "The Met-HGF/SF autocrine signaling mechanism is involved in sarcomagenesis." In Experientia Supplementum. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-9070-0_6.

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Conference papers on the topic "Sarcomagenesis"

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Jones, Kevin B. "Abstract IA20: Autophagy in alveolar soft part sarcomagenesis." In Abstracts: Advances in Sarcomas: From Basic Science to Clinical Translation; May 16-19, 2017; Philadelphia, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3265.sarcomas17-ia20.

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Mater, David Van, Leonor Ano, Jordan Blum, and David G. Kirsch. "Abstract A64: A role for injury in sarcomagenesis." In Abstracts: AACR Special Conference: Pediatric Cancer at the Crossroads: Translating Discovery into Improved Outcomes; November 3-6, 2013; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.pedcan-a64.

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Ye, Shuai, Koreana Pak, Jennifer Shah, et al. "Abstract 3342: Hippo signaling promotes sarcomagenesis through the NF-kB pathway." In Proceedings: AACR Annual Meeting 2017; April 1-5, 2017; Washington, DC. American Association for Cancer Research, 2017. http://dx.doi.org/10.1158/1538-7445.am2017-3342.

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Jones, Kevin B., Benjamin Illum, Huifeng Jin, et al. "Abstract C12: Beta-catenin enhances synovial sarcomagenesis and provides a therapeutic target." In Abstracts: AACR-NCI-EORTC International Conference: Molecular Targets and Cancer Therapeutics--Oct 19-23, 2013; Boston, MA. American Association for Cancer Research, 2013. http://dx.doi.org/10.1158/1535-7163.targ-13-c12.

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Ye, Shuai, Koreana Pak, Jennifer Shah, et al. "Abstract B07: Regaining epigenetic control of the Hippo pathway to inhibit sarcomagenesis." In Abstracts: Advances in Sarcomas: From Basic Science to Clinical Translation; May 16-19, 2017; Philadelphia, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3265.sarcomas17-b07.

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Lu, Chao, Siddhant Jain, Dominik Hoelper, et al. "Abstract PR12: Histone H3K36 mutations promote sarcomagenesis through altered histone methylation landscape." In Abstracts: Advances in Sarcomas: From Basic Science to Clinical Translation; May 16-19, 2017; Philadelphia, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3265.sarcomas17-pr12.

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Eisinger, T. S. Karin. "Abstract IA12: Epigenetic deregulation of the Hippo pathway mediates NF-κB driven sarcomagenesis". У Abstracts: Advances in Sarcomas: From Basic Science to Clinical Translation; May 16-19, 2017; Philadelphia, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3265.sarcomas17-ia12.

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Singer, Samuel. "Abstract IA18: Integrin-α10 drives metastasis and sarcomagenesis in mxyofibrosarcoma and undifferentiated pleomorphic sarcoma". У Abstracts: Advances in Sarcomas: From Basic Science to Clinical Translation; May 16-19, 2017; Philadelphia, PA. American Association for Cancer Research, 2018. http://dx.doi.org/10.1158/1557-3265.sarcomas17-ia18.

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Kendall, Genevieve C., Sarah Watson, Lin Xu, et al. "Abstract 872: Genetic models reveal that the novelVGLL2-NCOA2fusion oncogene leverages embryonic programs for sarcomagenesis." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.sabcs18-872.

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Kendall, Genevieve C., Sarah Watson, Lin Xu, et al. "Abstract 872: Genetic models reveal that the novelVGLL2-NCOA2fusion oncogene leverages embryonic programs for sarcomagenesis." In Proceedings: AACR Annual Meeting 2019; March 29-April 3, 2019; Atlanta, GA. American Association for Cancer Research, 2019. http://dx.doi.org/10.1158/1538-7445.am2019-872.

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Journal articles
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