Relevant bibliographies by topics / Sarcoptes scabiei vulpes

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Academic literature on the topic 'Sarcoptes scabiei vulpes'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Sarcoptes scabiei vulpes"

1

Bates, P. "Sarcoptic mange (Sarcoptes scabiei var vulpes) in a red fox (Vulpes vulpes) population in north-west Surrey." Veterinary Record 152, no. 4 (January 11, 2003): 112–14. http://dx.doi.org/10.1136/vr.152.4.112.

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2

Rousseau, Julieta, Mónia Nakamura, Helena Rio-Maior, Francisco Álvares, Rémi Choquet, Luís Madeira de Carvalho, Raquel Godinho, and Nuno Santos. "Non-Invasive Molecular Survey of Sarcoptic Mange in Wildlife: Diagnostic Performance in Wolf Faecal Samples Evaluated by Multi-Event Capture–Recapture Models." Pathogens 10, no. 2 (February 20, 2021): 243. http://dx.doi.org/10.3390/pathogens10020243.

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Sarcoptic mange is globally enzootic, and non-invasive methods with high diagnostic specificity for its surveillance in wildlife are lacking. We describe the molecular detection of Sarcoptes scabiei in non-invasively collected faecal samples, targeting the 16S rDNA gene. We applied this method to 843 Iberian wolf Canis lupus signatus faecal samples collected in north-western Portugal (2006–2018). We further integrated this with serological data (61 samples from wolf and 20 from red fox Vulpes vulpes, 1997–2019) in multi-event capture–recapture models. The mean predicted prevalence by the molecular analysis of wolf faecal samples from 2006–2018 was 7.2% (CI95 5.0–9.4%; range: 2.6–11.7%), highest in 2009. The mean predicted seroprevalence in wolves was 24.5% (CI95 18.5–30.6%; range: 13.0–55.0%), peaking in 2006–2009. Multi-event capture–recapture models estimated 100% diagnostic specificity and moderate diagnostic sensitivity (30.0%, CI95 14.0–53.0%) for the molecular method. Mange-infected individually identified wolves showed a tendency for higher mortality versus uninfected wolves (ΔMortality 0.150, CI95 −0.165–0.458). Long-term serology data highlights the endemicity of sarcoptic mange in wild canids but uncovers multi-year epidemics. This study developed and evaluated a novel method for surveying sarcoptic mange in wildlife populations by the molecular detection of S. scabiei in faecal samples, which stands out for its high specificity and non-invasive character.
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3

Bornstein, S., G. Zakrisson, and P. Thebo. "Clinical Picture and Antibody Response to Experimental Sarcoptes scabiei var. vulpes Infection in Red Foxes (Vulpes vulpes)." Acta Veterinaria Scandinavica 36, no. 4 (December 1995): 509–19. http://dx.doi.org/10.1186/bf03547665.

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4

Scott, Dawn M., Rowenna Baker, Alexandra Tomlinson, Maureen J. Berg, Naomi Charman, and Bryony A. Tolhurst. "Spatial distribution of sarcoptic mange (Sarcoptes scabiei) in urban foxes (Vulpes vulpes) in Great Britain as determined by citizen science." Urban Ecosystems 23, no. 5 (March 24, 2020): 1127–40. http://dx.doi.org/10.1007/s11252-020-00985-5.

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Abstract Urban areas may support high densities of wild carnivores, and pathogens can strongly influence carnivore populations. Red foxes (Vulpes vulpes) are hosts of sarcoptic mange (Sarcoptes scabiei), which infects numerous species, and transmission can be density dependent. In Great Britain, urban red foxes (Vulpes vulpes) have recently increased in population density and undergone range expansions. Here we investigate corresponding changes in urban fox mange prevalence. We predicted a higher prevalence closer to historic epi/enzootics and lower prevalence where urban features reduce fox density and movements, i.e. large areas of public green space, and fragmented habitat, as measured by road length and urban perimeter shape complexity. We visually assessed mange symptoms from georeferenced images of urban foxes submitted online by the public, thus surveying private land on a national scale. We measured the proportion of foxes apparently showing mange and used SATSCAN to identify spatial clusters of high infection risk. Landscape features were extracted from urban layers in GIS to determine associations. Although mange was widespread, we identified a single cluster of high prevalence (37.1%) in Northwest and Central England, which exceeded double mean prevalence overall (15.1%) and mirrors the northward expansion of urban fox distribution. Prevalence was positively correlated with perimeter shape complexity and negatively correlated with distance to the nearest city with mange, although the latter association was weak. Our findings show that citizen science can effectively monitor diseases with highly visible symptoms and suggest that fox movements are influential in explaining spatial patterns of prevalence.
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5

Martínez, Ileana Z., Álvaro Oleaga, Irene Sojo, María José García-Iglesias, Claudia Pérez-Martínez, Juan F. García Marín, and Ana Balseiro. "Immunohistochemical Assessment of Immune Response in the Dermis of Sarcoptes scabiei—Infested Wild Carnivores (Wolf and Fox) and Ruminants (Chamois and Red Deer)." Animals 10, no. 7 (July 6, 2020): 1146. http://dx.doi.org/10.3390/ani10071146.

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Sarcoptic mange is caused by the mite Sarcoptes scabiei and has been described in several species of domestic and wild mammals. Macroscopic lesions are predominantly hyperkeratotic (type I hypersensitivity) in fox, chamois and deer, but alopecic (type IV hypersensitivity) in wolf and some fox populations. To begin to understand the immune processes underlying these species differences in lesions, we examined skin biopsies from wolves (Canis lupus), foxes (Vulpes vulpes), chamois (Rupicapra rupicapra) and red deer (Cervus elaphus) naturally infested with S. scabiei. Twenty skin samples from five animals per species were used. Sections were immuno-stained with primary antibodies against Iba1 to detect macrophages, lambda chain to detect plasma cells, CD3 to detect T lymphocytes and CD20 to detect B lymphocytes. Skin lesions contained significantly more inflammatory cells in the fox than in the wolf and chamois. Macrophages were the most abundant inflammatory cells in the lesions of all the species studied, suggesting a predominantly innate, non-specific immune response. Lesions from the wolf contained higher proportions of macrophages than the other species, which may reflect a more effective response, leading to alopecic lesions. In red deer, macrophages were significantly more abundant than plasma cells, T lymphocytes and B lymphocytes, which were similarly abundant. The fox proportion of plasma cells was significantly higher than those of T and B lymphocytes. In chamois, T lymphocytes were more abundant than B lymphocytes and plasma cells, although the differences were significant only in the case of macrophages. These results suggest that all the species examined mount a predominantly innate immune response against S. scabiei infestation, while fox and chamois may also mount substantial humoral and cellular immune responses, respectively, with apparently scarce effectiveness that lead to hyperkeratotic lesions.
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6

Davidson, Rebecca K., Set Bornstein, and Kjell Handeland. "Long-term study of Sarcoptes scabiei infection in Norwegian red foxes (Vulpes vulpes) indicating host/parasite adaptation." Veterinary Parasitology 156, no. 3-4 (October 2008): 277–83. http://dx.doi.org/10.1016/j.vetpar.2008.05.019.

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7

Loredo, Ariel Irene, Jamie Leann Rudd, Janet Elizabeth Foley, Deana Louise Clifford, and Brian Leslie Cypher. "CLIMATIC SUITABILITY OF SAN JOAQUIN KIT FOX (VULPES MACROTIS MUTICA) DENS FOR SARCOPTIC MANGE (SARCOPTES SCABIEI) TRANSMISSION." Journal of Wildlife Diseases 56, no. 1 (January 6, 2020): 126. http://dx.doi.org/10.7589/2019-02-035.

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8

BORNSTEIN, SET, and GORAN ZAKRISSON. "Humoral Antibody Response to Experimental Sarcoptes scabiei var. vulpes Infection in the Dog." Veterinary Dermatology 4, no. 3 (September 1993): 107–10. http://dx.doi.org/10.1111/j.1365-3164.1993.tb00202.x.

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9

BORNSTEIN, SET. "Experimental Infection of Dogs with Sarcoptes scabiei Derived from Naturally Infected Wild Red Foxes (Vulpes vulpes): Clinical Observations." Veterinary Dermatology 2, no. 3-4 (September 1991): 151–59. http://dx.doi.org/10.1111/j.1365-3164.1991.tb00126.x.

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10

Jakubek, Eva-Britt, Roland Mattsson, Torsten Mörner, Jens G. Mattsson, and Dolores Gavier-Widén. "Potential application of serological tests on fluids from carcasses: detection of antibodies against Toxoplasma gondii and Sarcoptes scabiei in red foxes (Vulpes vulpes)." Acta Veterinaria Scandinavica 54, no. 1 (2012): 13. http://dx.doi.org/10.1186/1751-0147-54-13.

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Dissertations / Theses on the topic "Sarcoptes scabiei vulpes"

1

Hörnfeldt, Birger. "Cycles of voles, predators, and alternative prey in boreal Sweden." Doctoral thesis, Umeå universitet, Ekologi och geovetenskap, 1991. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-100711.

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Bank voles, grey-sided voles, and field voles had synchronous 3-4 year density cycles with variable amplitudes which averaged about 200-fold in each species. Cycles of vole predators (red fox and Tengmalm's owl), and their (foxes') alternative prey (mountain hare and forest grouse) lagged behind the vole cycles. The nomadic Tengmalm's owl responded with a very rapid and strong numerical increase to the initial cyclic summer increase of voles (the owl’s staple food). Owl breeding densities in the springs were highly correlated with vole supply in the previous autumns. This suggested that the number of breeding owls was largely determined in the autumn at the time of the owl's nomadic migrations, and that immigration was crucial for the rapid rise in owl numbers. The owl's numerical response was reinforced by the laying of earlier and larger clutches when food was plentiful. In addition, the owl has an early maturation at one year of age. The transition between subsequent vole cycles was characterized by a distinct shift in rate of change in numbers from low to high or markedly higher values in both summer and winter. Regulation increased progressively throughout the cycle since the rate of change decreased continuously in the summers. Moreover, there was a similar decrease of the rate of change in winter. Rate of change was delayed density-dependent. The delayed density-dependence had an 8 month time-lag in the summers and a 4 month time-lag in the winters relative to the density in previous autumns and springs, respectively. These findings suggest that vole cycles are likely to be generated by a time-lag mechanism. On theoretical grounds, it has been found that a delayed density- dependence of population growth rate with a 9 month time-lag caused stable limit cycles with a period between 3 and 4 years. Some mechanisms for the delayed density-dependence are suggested and discussed. The mechanisms are assumed to be related to remaining effects of vole populations past interactions with predators, food supplies, and/or diseases. Unlike the other voles, the bank vole had regular and distinct seasonal declines in density over winter. These declines are proposed to be due to predation, mainly by Tengmalm's owl. Supranivean foraging for epiphytic tree lichens and conifer seeds most likely explains why this species was frequently taken by the owl under snow-rich conditions. The alternative prey hypothesis predicts that a reduction of predator numbers should increase the number of alternative prey. Alternative prey should be less effectively synchronized to the vole cycle by predation at declining and low vole (main prey) densities; they may also lose their 3-4 year cyclicity. The appearance of sarcoptic mange among foxes in northern Sweden in the mid 1970s provided an opportunity to "test" these ideas, and these were found to be supported. In areas with highest mange infection rates, foxes declined markedly from the late 1970s to mid 1980s, whereas hare numbers rose rapidly and appeared non-cyclic.

Diss. (sammanfattning) Umeå : Umeå universitet, 1991, härtill 7 uppsatser


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