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Dissertations / Theses on the topic 'SARS-Coronavirus'

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Published: 4 June 2021

Last updated: 16 February 2022

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1

You, Jae Hwan. "Characterisation of the SARS-coronavirus nucleocapsid protein." Thesis, University of Leeds, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.489883.

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Coronaviruses are the causative agents of various mammalian diseases which have crucial economic and health-related problems and are mainly respiratory and gastrointestinal pathogens. They are positive strand RNA viruses which may require nuclear functions for replication. The nucleocapsid (N) protein of s~veral members has previously been shown to localise to the nucleus/nucleolus and the cytoplasm during infection and after transient transfection. The coronavirus N protein is a viral RNA binding protein with several functions during the virus life cycle, especially with regard to RNA replication and transcription and controlling cell signalling pathways. In order to localise to the cytoplasm and nucleus/nucleolus N protein must contain appropriate trafficking motif(s). This thesis focused on characterising the newly emerged severe acute respiratory syndrome coronavirus (SARS-CoV) N protein. The sub-cellular localisation of the SARS-CoV N protein was determinedÃ‚Â· in virus infected and transfected cells using antibody labelling and C-terminally tagged fluorescent fusion proteins, respectively. Comparison with the avian coronavirus N protein indicated that in contrast to other coronavirus N proteins, SARS-CoV N protein localised mainly in the cytoplasm with low frequency localisation in the nucleolus. Live cell, confocal microscopy and fluorescence loss in photo-bleaching (FLIP) was used to investigate the presence of any potential trafficking signals. Based on amino acid sequence conservation with the other coronavirus N proteins the SARS-CoV N protein was divided into three regions and mutation and bioinformatic analysis was used to potential nuclear import and export motifs. This approach delineated a cryptic nucleolar localisation signal in the central portion of the protein and a novel nuclear export signal in the C-terminal part, which may be the predominant trafficking signal. These motifs may be exposed by differential phosphorylation. The N protein was expressed in vitro and experiments formally demonstrated that it was a phosphoprotein which could bind viral RNA and an RNA binding region spanned the N-terminal and centralÃ‚Â·part ofthe protein. Together, the data in this study has provided insights into the expression and sub-cellular localisation ofthe SARS Cov N protein.

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2

Sheahan, Timothy Patrick Baric Ralph S. "SARS coronavirus pathogenesis and therapeutic treatment design." Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2008. http://dc.lib.unc.edu/u?/etd,1659.

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Thesis (Ph. D.)--University of North Carolina at Chapel Hill, 2008.
Title from electronic title page (viewed Sep. 16, 2008). "... in partial fulfillment of the requirements for the degree of Doctor of Philosophy in the Department of Microbiology and Immunology." Discipline: Microbiology and Immunology; Department/School: Medicine.

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3

Ivanov, Konstantin. "Charakterisierung der Helikase- und Endonukleaseaktivitäten des Humanen Coronavirus 229E und des SARS-Coronavirus." [S.l.] : [s.n.], 2005. http://deposit.ddb.de/cgi-bin/dokserv?idn=978851749.

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4

Vabret, Astrid. "Coronavirus humains hors-SARS-CoV : veille virologique et étude épidémiologique moléculaire du coronavirus OC43." Caen, 2006. http://www.theses.fr/2006CAEN2012.

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Cinq coronavirus infectent l’homme : HCoVs 229E, OC43, NL63, HKU1 et SARS-CoV. Parmi eux, trois ont été identifiés récemment : le SARS-CoV en 2003, le HCoV-NL63 en 2004, et le HCoV-HKU1 en 2005. Les coronavirus humains (hors SARS-CoV) sont responsables d’infections respiratoires aiguës; ils possèdent également un tropisme digestif et neurologique. Des méthodes de détection et de caractérisation moléculaires ont été développées pour les HCoVs (hors SARS-CoV). Elles ont permis de mettre en évidence une épidémie d’infections respiratoires à HCoV-OC43, la circulation et la diversité génétique des HCoVs NL63 et HKU1. La diversité génétique des HCoV-OC43 a été particulièrement étudiée. L’analyse moléculaire et phylogénique du gène S1 de 7 HCoV-OC43 a montré une grande diversité génétique inter-souches. Nous avons pu mettre en évidence une distribution en quasi-espèces de HCoV-OC43 dans un contexte d'infection respiratoire aiguë. Il existe une importante hétérogénéité virale au sein d’une même population HCoV-OC43. La mise en évidence de ces quasi-espèces permet une meilleure compréhension de l’évolution des coronavirus et de leur capacité pour franchir les barrières d’espèces, s’adapter à leur nouvel hôte, et établir des infections persistantes
Five human coronaviruses have been identified: HCoVs 229E, OC43, NL63, HKU1 and SARS-CoV. Among them, three have been found very recently: SARS-CoV in 2003, HCoV-NL63 in 2004, and HCoV-HKU1 in 2005. Human coronaviruses (except for SARS-CoV) mainly cause acute respiratory tract illnesses. They are also involved in enteric and neurological diseases. We have developed molecular methods to detect and characterize the HCoVs (except for SARS-CoV). These methods allow us to identify an outbreak of HCoV-OC43 respiratory infections, as well as the circulation and the genetic diversity of HCoVs NL63 and HKU1. The genetic diversity of HCoV-OC43 has been the foc us of more elaborate studies. The molecular and phylogenetic analysis of the S1 gene of seven HCoV-OC43 strains has shown a great inter-strain genetic diversity. We have demonstrated the quasispecies organization of the HCoV-OC43 viral population in a context of acute respiratory infection. The intra-strain genetic heterogeneity is very important. The demonstration of quasispecies distribution of HCoV-OC43 could provide a better understanding of the evolution of coronaviruses, especially their capacity to jump species barriers, to adapt to their new host, and to establish persistent infections

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5

Teepe, Carola. "Subzelluläre Lokalisation und Interaktionen der Offenen Leserahmen des SARS Coronavirus." Diss., lmu, 2010. http://nbn-resolving.de/urn:nbn:de:bvb:19-111848.

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6

Mccrory, Sarah Ann. "SARS coronavirus : The nucleocapsid protein and the human immune response." Thesis, University of Reading, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.515794.

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7

Law, Ka-man. "Vaccine development against the severe acute respiratory syndrome-coronavirus (SARS-CoV) using SARS-CoV spike protein." Click to view the E-thesis via HKUTO, 2005. http://sunzi.lib.hku.hk/hkuto/record/B36774480.

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8

Law, Ka-man, and 羅嘉敏. "Vaccine development against the severe acute respiratory syndrome-coronavirus (SARS-CoV) using SARS-CoV spike protein." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2005. http://hub.hku.hk/bib/B36774480.

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9

Chauhan, Vinita Singh. "Molecular characterization of severe acute respiratory syndrome (SARS) coronavirus - nucleocapsid protein." Diss., Manhattan, Kan. : Kansas State University, 2006. http://hdl.handle.net/2097/152.

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10

Schöpf, Julia. "Identifikation und Charakterisierung zellulärer Zielproteine zur antiviralen Therapie der SARS-Coronavirus Infektion." Diss., Ludwig-Maximilians-Universität München, 2011. http://nbn-resolving.de/urn:nbn:de:bvb:19-139194.

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The severe acute respiratory syndrome (SARS) was first observed in the Chinese province Guangdong in November 2002. The disease quickly spread around the globe via air travelling and caused a worldwide epidemic. Several research institutions together with the World Health Organisation (WHO) identified the SARS-coronavirus (SARS-CoV) as the causative agent of this disease. During the epidemic, about 8,000 people were infected with a mortality of approximately 10%. Although no new infections have been observed since the summer of 2003, a recurrence of the pathogen cannot be excluded. Up to now, no specific therapy against the virus have been available. Viruses contain a very compact genome, which does not encode all proteins necessary for independant replication. Thus, viruses necessarily depend on host proteins and have to interact directly with them. The analysis of protein-protein interactions between SARS-CoV and human host cells contributes to a better understanding of the viral replication and pathogenicity. Prior to this work, an automated, genome-wide yeast-two-hybrid (Y2H) screen between all 28 proteins of SARS-CoV and the gene products of three human cDNA libraries had been performed, and approximately 460, mostly new protein-protein interactions had been identified. The aim of this work was to confirm newly identified virus-host SARS-CoV protein interactions and to functionally analyse them to identify new targets for antiviral therapy. 89 newly identified protein-protein interactions were examined via a modified LUMIER binding-assay to confirm individual interactions. 37 out of 89 protein interactions were found to be positive, resulting in a confirmation rate of 42%. In subsequent functional analyses of protein-protein interactions between the SARS-CoV non-structural protein 1 (Nsp1) and proteins of the immunophilin family, two different functional consequences were observed. First, it could be shown that SARS-CoV Nsp1 boosts the expression of genes regulated via the calcineurin/NFAT-signalling cascade. The increased expression of NFAT-regulated genes in SARS-CoV infection may cause the cytokine dysregulation described in SARS patients which leads to severe lung tissue destructions and which correlates with high mortality. The considerably less harmful human coronavirus HCoV-NL63 and mouse coronavirus (MHV) did not boost the expression of NFAT-regulated genes. It was thus hypothesized that the therapy of the cytokine dysregulation with the immunosuppressive drug Cyclosporine A (CspA) might improve the course of the disease. In addition, it could be shown for the first time that the replication of the SARS-CoV can be inhibited by the immunosuppressive drug CspA. Subsequent experiments showed a similar inhibition of the viral replication of the less harmful human coronavirus HCoV-NL63 and HCoV-229E mediated by CspA. In cooperation with several groups of the ”SARS-Zoonose- Verbund”, further inhibition experiments were performed with animal coronaviruses like FCoV, IBV Bd and TGEV PUR46, which showed a similar antiviral effect of CspA. The two cellular proteins Cyclophilin A and FK506 binding-protein 1A were shown to be essential for viral replication of HCoV-NL63. The findings of this work may contribute to a better understanding of the interactions between SARS-CoV and infected host cells and their innate immune response. The application of the general coronaviral inhibitor CspA identified in this study and of non-immunosuppressive CspA analogues like DEBIO-025 procures promising options for anti-coronaviral therapy.
Im November 2002 brach das Schwere Akute Atemwegssyndrom (severe acute respiratory syndrom, SARS) zum ersten Mal in der chinesischen Provinz Guangdong aus. Dieser Erreger verursachte aufgrund des internationalen Flugverkehrs erstmals eine weltweite Epidemie. Verschiedene Forschungseinrichtungen konnten in Zusammenarbeit mit der Weltgesundheitsorganisation (WHO) das SARS-assoziierte Coronavirus (SARS-CoV) als Erreger der schweren Krankheit identifizieren. Insgesamt wurden während der Epidemie etwa 8000 Menschen infiziert, von denen ca. 10 % verstarben. Obwohl seit Sommer 2003 keine Neuinfektionen mehr beobachtet wurden, kann ein erneutes Auftreten dieses Pathogens nicht ausgeschlossen werden. Bis heute steht keine spezifische Therapie gegen SARS-CoV zur Verfügung. Viren haben ein sehr kompaktes Genom, in dem nicht alle notwendigen Proteine kodiert sind, die für einen kompletten Infektionszyklus benötigt werden. Aus diesem Grund sind Viren ausnahmslos abhängig von den Protein-Protein-Interaktionen mit einer lebenden Wirtszelle. Die Analyse von Protein-Protein-Interaktionen zwischen dem SARS-CoV und der humanen Wirtszelle trägt zum besseren Verständnis der viralen Replikation und Pathogenität bei. Im Vorfeld dieser Arbeit wurde ein automatisierter, genomweiter Hefe-Zwei-Hybrid (H2H)-Screen zwischen allen 28 Proteinen des SARS-CoV und den Genprodukten von drei humanen cDNA-Banken durchgeführt, wobei ca. 460, zumeist völlig neue, Protein-Protein-Interaktionen zwischen dem SARS-CoV und dem humanen Wirt identifiziert wurden. Ziel dieser Arbeit war es, die neu identifzierten Protein-Protein-Interaktionen zu bestätigen und funktionelle Analysen ausgewählter Interaktionen durchzuführen, um neue Angriffspunkte für die antivirale Therapie zu finden. 89 Protein-Protein-Interaktionen, die im H2H-Screen neu identifiziert werden konnten, wurden mit Hilfe des modifizierten LUMIER Bindungs-Assays zur Bestätigung der einzelnen Interaktionen untersucht. Von diesen 89 getesteten Protein-Protein-Interaktionen waren 37 Tests positiv, wodurch sich eine Bestätigungsrate von 42 % ergab. In anschließenden funktionellen Analysen der Protein-Interaktionen zwischen dem SARS-CoV Nicht-Strukturprotein 1 (Nsp1) und Proteinen der Immunophilinfamilie konnten zwei Funktionen dieser Interaktionen aufgezeigt werden. Zunächst konnte gezeigt werden, dass das SARS-CoV Nsp1 die Expression von Genen, welche über die Calcineurin/NFAT-Signalkaskade reguliert werden, erhöht. Die SARS-spezifische Erhöhung der Expression NFAT-regulierter Gene kann eine Ursache der in SARS-Patienten beschriebenen Zytokindysregulation sein. Diese Zytokindysregulation führt zu schweren Gewebeschädigungen in der Lunge und trägt maßgeblich zum schlechten Ausgang der Krankheit bei. Das weniger pathogene humane Coronavirus HCoV-NL63 und das Maus-Coronavirus MHV zeigten diese Erhöhung der Expression NFAT-regulierter Gene nicht auf. Es wurde deshalb die Hypothese aufgestellt, dass eine Behandlung der Zytokindysregulation mit dem Immunsuppressivum Cyclosporin A positive Auswirkungen auf den Verlauf der Krankheit hat. Zum zweiten konnte erstmals gezeigt werden, dass die Replikation des SARS-CoV durch das Immunsuppressivum Cyclosporin A gehemmt werden kann. Anschließende Inhibitionsversuche der deutlich harmloseren humanen Coronaviren HCoV-NL63 und HCoV-229E zeigten die gleiche Hemmung der viralen Replikation durch Cyclosporin A. In Kooperation mit verschiedenen Arbeitsgruppen des SARS-Zoonose-Verbunds konnten weitere Inhibitionsversuche mit den Tiercoronaviren FCoV, IBV Bd und TGEV PUR46 durchgeführt werden und ebenfalls ein inhibitorisches Potential des Cyclosporin A auf die virale Replikation dieser Tiercoronaviren gezeigt werden. In weiterführenden Untersuchungen zum Wirkmechanismus der CspA- und FK506-vermittelten Inhibition der Replikation des humanen Coronavirus HCoV-NL63 konnten die beiden zellulären Proteine Cyclophilin A und FK506-Bindeprotein1A (FKBP1A) erstmals als essentielle Proteine für die virale Replikation identifiziert werden. Die Erkenntnisse dieser Arbeit können dazu beitragen, die komplexen Interaktionen zwischen dem SARS-CoV, der infizierten Wirtszelle und der Immunabwehr besser zu verstehen. Weiterhin konnte im Rahmen dieser Arbeit ein allgemeiner, coronaviraler Inhibitor in Form von Cyclosporin A identifiziert werden. Nicht-immunsuppressive Cyclosporin A Analoga wie DEBIO 025 sind deshalb mögliche Kandidaten für die Therapie coronaviraler Infektionen.

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11

Chow, Chun-kin, and 周俊健. "Suppressor of cytokine signaling (SOCS 3) induction in SARS coronavirus infected cells." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2009. http://hub.hku.hk/bib/B42925150.

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12

Chow, Chun-kin. "Suppressor of cytokine signaling (SOCS 3) induction in SARS coronavirus infected cells." Click to view the E-thesis via HKUTO, 2009. http://sunzi.lib.hku.hk/hkuto/record/B42925150.

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13

Chow, Yan-ching Ken, and 周恩正. "Characterization of the apoptotic properties of severe acute respiratory syndrome coronavirus (SARS-CoV) structural proteins." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2004. http://hub.hku.hk/bib/B30105493.

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14

Wong, Hiu-ling Beatrice. "Development of antibody and antigen detection assays and vaccines for SARS associated coronavirus." Click to view the E-thesis via HKUTO, 2007. http://sunzi.lib.hku.hk/hkuto/record/B39634024.

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15

Wong, Hiu-ling Beatrice, and 黃曉靈. "Development of antibody and antigen detection assays and vaccines for SARS associated coronavirus." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2007. http://hub.hku.hk/bib/B39634024.

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16

Mnyamana, Yanga Eddie. "Expression of human coronavirus NL63 and SARS-CoV nucleocapsid proteins for antibody production." University of the Western Cape, 2012. http://hdl.handle.net/11394/2989.

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>Magister Scientiae - MSc
Human Coronaviruses (HCoVs) are found within the family Coronaviridae (genus, Coronavirus) and are enveloped, single-stranded, positive-sense RNA viruses. Infections of humans by coronaviruses are not normally associated with severe diseases. However, the identification of the coronavirus responsible for the outbreak of severe acute respiratory syndrome (SARS-CoV) showed that highly pathogenic coronaviruses can enter the human population. The SARS-CoV epidemic resulted in 8 422 cases with 916 deaths globally (case fatality rate: 10.9%). In 2004 a group 1 Coronavirus, designated Human Coronavirus NL63 (HCoV-NL63), was isolated from a 7 month old Dutch child suffering from bronchiolitis. In addition, HCoV-NL63 causes disease in children (detected in approximately 10% of respiratory tract infections), the elderly and the immunocompromised. This study was designed to express the full length nucleocapsid (N) proteins of HCoV-NL63 and SARS-CoV for antibody production in an animal model. The NL63-N/pFN2A and SARSN/ pFN2A plasmid constructs were used for this study. The presence of the insert on the Flexi ® vector was confirmed by restriction endonuclease digest and sequence verification. The sequenced chromatographs obtained from Inqaba Biotec were consistent with sequences from the NCBI Gen_Bank. Proteins were expressed in a KRX Escherichia coli bacterial system and analysed using 15% SDS-PAGE and Western Blotting. Thereafter, GST-tagged proteins were purified ith an affinity column purification system. Purified fusion proteins were subsequently cleaved with Pro-TEV Plus protease, separated on 15% SDS-PAGE gel and stained with Coomassie Brilliant Blue R250. The viral fusion proteins were subsequently used to immunize Balbc mice in order to produce polyclonal antibodies. A direct ELISA was used to analyze and validate the production of polyclonal antibodies by the individual mice. This is a preliminary study for development of diagnostic tools for the detection of HCoV-NL63 from patient samples collected in the Western Cape.
South Africa

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 29." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651958.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 45." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652066.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 46." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652078.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 51." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652111.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 48." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652085.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 49." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652087.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 50." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652088.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 55." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652146.

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Struck, Anna-Winona [Verfasser], and Bernd [Akademischer Betreuer] Meyer. "Bindungsstudien mit humanen Zellrezeptoren für das SARS-Coronavirus / Anna-Winona Struck. Betreuer: Bernd Meyer." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2011. http://d-nb.info/1020422831/34.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 52." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652127.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 53." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652133.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 2." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651716.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 3." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651717.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 10." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651812.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 4." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651727.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 5." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651740.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 6." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651766.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 7." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651780.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 8." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651790.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 9." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651808.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 11." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651822.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 12." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651833.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 13." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651849.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 27." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651943.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 28." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651950.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 31." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/651968.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 33." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652002.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 35." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652005.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 36." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652007.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 39." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652040.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 37." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652016.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 41." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652060.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 42." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652061.

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ALTAMIRA, Asociación Peruana de Bibliotecas Académicas. "Boletín diario de información científica N° 44." Asociación Peruana de Bibliotecas Académicas ALTAMIRA, 2020. http://hdl.handle.net/10757/652065.

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