Academic literature on the topic 'Scar remodeling'
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Journal articles on the topic "Scar remodeling"
Costa, Andréa Monte Alto, Simone Peyrol, Luís Cristóvão Pôrto, Jean-Pierre Comparin, Jean-Louis Foyatier, and Alexis Desmoulière. "Mechanical Forces Induce Scar Remodeling." American Journal of Pathology 155, no. 5 (November 1999): 1671–79. http://dx.doi.org/10.1016/s0002-9440(10)65482-x.
Full textLudwig, Irene H., and Alan Y. Chow. "Scar remodeling after strabismus surgery." Journal of American Association for Pediatric Ophthalmology and Strabismus 4, no. 6 (December 2000): 326–33. http://dx.doi.org/10.1067/mpa.2000.107899.
Full textSingh, Yashbir, Deepa Shakyawar, and Weichih Hu. "Non-ischemic endocardial scar geometric remodeling toward topological machine learning." Proceedings of the Institution of Mechanical Engineers, Part H: Journal of Engineering in Medicine 234, no. 9 (July 10, 2020): 1029–35. http://dx.doi.org/10.1177/0954411920937221.
Full textTepper, Ronnie, Yoram Beyth, Zvi Klein, and Rami Aviram. "Postmyomectomy sonographic imaging: uterus remodeling and scar repair." Archives of Gynecology and Obstetrics 280, no. 3 (February 14, 2009): 509–11. http://dx.doi.org/10.1007/s00404-009-0967-6.
Full textLee, Sang-jun, Dong-Hye Suh, Ji Min Lee, Kye-Yong Song, and Hwa Jung Ryu. "Dermal Remodeling of Burn Scar by Fractional CO2 Laser." Aesthetic Plastic Surgery 40, no. 5 (August 12, 2016): 761–68. http://dx.doi.org/10.1007/s00266-016-0686-x.
Full textDuerr, Georg D., Naziha Elhafi, Toktam Bostani, Joerg Ellinger, Louay Swieny, Elvis Kolobara, Armin Welz, and Oliver Dewald. "Comparison of Myocardial Remodeling between Cryoinfarction and Reperfused Infarction in Mice." Journal of Biomedicine and Biotechnology 2011 (2011): 1–10. http://dx.doi.org/10.1155/2011/961298.
Full textHolmes, J. W., H. Yamashita, L. K. Waldman, and J. W. Covell. "Scar remodeling and transmural deformation after infarction in the pig." Circulation 90, no. 1 (July 1994): 411–20. http://dx.doi.org/10.1161/01.cir.90.1.411.
Full textAstakhova, M. I., E. S. Golovneva, L. V. Astakhova, I. A. Astakhov, and E. N. Ignatieva. "REGULATION OF MICROCIRCULATION DURING REMODELING OF SKIN SCAR UNDER INFLUENCE OF LASER." Medical academic journal 19, no. 1S (December 15, 2019): 144–45. http://dx.doi.org/10.17816/maj191s1144-145.
Full textSun, Daniel, and Tatjana C. Jakobs. "Structural Remodeling of Astrocytes in the Injured CNS." Neuroscientist 18, no. 6 (October 7, 2011): 567–88. http://dx.doi.org/10.1177/1073858411423441.
Full textSnider, J. Caleb, Lance A. Riley, Noah T. Mallory, Matthew R. Bersi, Prachi Umbarkar, Rekha Gautam, Qinkun Zhang, et al. "Targeting 5-HT 2B Receptor Signaling Prevents Border Zone Expansion and Improves Microstructural Remodeling After Myocardial Infarction." Circulation 143, no. 13 (March 30, 2021): 1317–30. http://dx.doi.org/10.1161/circulationaha.120.051517.
Full textDissertations / Theses on the topic "Scar remodeling"
Nelson, Charles A. L. "Relationship between the transmural distribution of myocardial scar and ventricular function /." [St. Lucia, Qld.], 2004. http://www.library.uq.edu.au/pdfserve.php?image=thesisabs/absthe18564.pdf.
Full textQurashi, Abrar Ahmad. "Neuronal remodeling in Drosophila melanogaster with WAVE/SCAR complex and its implication in cognitive functions." Université Louis Pasteur (Strasbourg) (1971-2008), 2006. http://www.theses.fr/2006STR13112.
Full textNeuronal morphogenesis and plasticity during development as well as in cognitive functions rely on actin cytoskeleton remodeling in response to extra-cellular signals that are interpreted by Rho family of small GTPases. The key subject of my thesis is to understand how signaling pathways downstream of Rac proteins, members of the Rho GTPase family, are utilized to orchestrate distinct aspects of neuronal morphogenesis and structural plasticity. WAVE/SCAR complex, an evolutionarily-conserved assembly of five proteins: WAVE (SCAR), PIR121 (CYFIP), Hem-2 (Kette), Abi and HSPC300 has emerged as a critical link between Rac1 and Arp2/3, molecular complex triggering actin nucleation. During my thesis I have used Drosophila melanogaster as a model system to understand the physiological significance of WAVE/SCAR complex. We have elucidated its role in neuronal actin remodeling underlying axon as well synapse development. Specifically, I have isolated mutations in the HSPC300 gene, and present its detailed characterization both at genetic and biochemical level. My thesis work provides evidence that in Drosophila melanogater SCAR, CYFIP, Kette and HSPC300 associate together to form a complex. All these proteins are highly expressed in the embryonic nervous system and show strong accumulation in central and motor neurons. Interestingly, in many processes examined, there are striking similarities between the phenotypes resulting from the mutations in any member of the complex, for example defects in axon path-finding, axon growth and Neuromuscular Junction (NMJ) morphology, thus demonstrating their pivotal roles for precise neuronal development. By biochemical and genetic experiments, we demonstrated that loss of any of the complex components leads to instability in other components. Therefore, the results provide an unequivocal reason for the common pathological condition noticed in single mutation of the WAVE/SCAR complex. Interestingly, mutation in individual components of the complex not only affects the stability of other complex components but also affects the multiple downstream pathways associated with them. For example, mutation in any component of the complex has an impact on CYFIP signaling to the Fragile X Mental Retardation Protein (FMRP) implicated in learning and memory in humans. Thus, our results identify the Drosophila WAVE/SCAR complex as a multifunctional unit orchestrating different pathways and aspects of neuronal connectivity and support an emerging theme: different aspects of xv morphogenesis may involve the regulation of common core signaling pathways. Additionally, my thesis also demonstrates the interaction of all three Racs (Rac1, Rac2 and Mtl) with CYFIP and suggests their requirement during NMJ growth and plasticity
Aubin, Marie-Claude. "Étude de la fonction vasculaire et du remodelage cardiaque avant l’établissement de l’obésité et de la dyslipidémie chez les rats femelles Sprague-Dawley recevant une diète riche en gras." Thèse, 2009. http://hdl.handle.net/1866/3569.
Full textKnowledge is insufficient regarding cardiovascular modifications occurring prior to the development of overt obesity and dyslipidemia. In this regard, the present project aimed at testing the hypothesis stipulating that the administration of a high fat diet for an 8-week period in female rats can adversely influence cardiac function and remodeling prior to the development of overt obesity, and in the absence of hyperlipidemia and hyperglycaemia. To directly examine these issues, normal female Sprague-Dawley rats were fed a standard (SD; 12.5% lipid, kcal) or a high-fat diet (HF; 42% lipids, kcal) for 8 weeks. This regimen was insufficient to induce a significant gain in body mass in HF rats (329±14g) as compared to SD rats (300±10g), or any variation in the lipid profile. By contrast, systemic arterial hypertension developed in high fat fed rats (130±4mmHg versus SD, 108±6mmHg, p<0.05), additionally to a significant decrease in acetylcholine-mediated maximal relaxation of isolated aortic rings (HF, 22±5%) compared to rats fed a standard diet (53±8%, p<0.05). Chronic oral administration of the antioxidant resveratrol (RES; 20 mg·kg-1·d-1) prevented the development of both pathological alterations, attesting to the implication of oxidative stress. However, it failed to attenuate the perivascular fibrosis that developed following the administration of the high-fat diet. Following ischemia/reperfusion injury, scar length (SD, 0.29±0.09 versus HF, 0.32±0.13 cm), thickness (SD, 0.05±0.02 versus HF, 0.06±0.01 cm) and collagen α1 type 1 content (SD, 0.21±0.04 versus HF, 0.20±0.04 arbitrary units/mm2) in the infarcted heart of rats fed a high fat diet were similar to infarcted normal rats. Despite these findings, the rate of death was significantly increased (p<0.05) in female rats fed a high fat diet (56%) compared to rats fed a standard diet (5%). An electrophysiology approach revealed that normal female rats fed a high fat diet had an increased incidence (p<0.05) of induced ventricular extrasystoles. In addition, these hearts presented a functional sympathetic hyperinnervation, as reflected by the increased density of neurofilament-M immunoreactive fibres (SD, 2020±260 versus HF, 2830±250 μm2/mm2; p<0.05) and increased protein expression of tyrosine hydroxylase. The gap junction function was also impaired, characterized by lateralization and internalization of connexine 43, and a decreased expression of connexine 40 in intercalated discs of rats fed a high fat diet. Thus, prior to the development of overt obesity and dyslipidemia, female rats with modest hypertension exhibit an arrhythmogenic cardiac phenotype due in part to sympathetic hyperinnervation and a concomitant aberrant pattern of gap junctional protein expression and distribution. The lack of significant clinical manifestations of obesity in the present study does not enable clinicians to suspect the increased susceptibility to ventricular arrhythmias. Hence, in presence of modest hypertension in a non-obese patient, evaluation of the sympathetic activity by the assessment of circulating catecholamine could be helpful in detecting patients at high risk for sudden death.
Proulx, Cindy. "L’implication du Stromal Cell-derived Factor-1 alpha dans le remodelage cardiaque une semaine après un infarctus du myocarde." Thèse, 2008. http://hdl.handle.net/1866/2707.
Full textThe injection of bone marrow stem cells in the infarcted heart was shown to improve ventricular function and scar remodelling. The chemokine Stromal Cell-derived factor-1 alpha (SDF-1 alpha) is implicated in the migration of inflammatory and bone marrow derived stem cells to damaged region. Despite a local increase of SDF-1 alpha expression in the damaged myocardium, the biological impact of the chemokine during the acute phase of remodelling in the ischemic heart remains undefined. Therefore, the present study examined the role of SDF-1 alpha on scar expansion, cardiac hypertrophy and ventricular function in rats following myocardial infarction (MI) via administration of AMD3100 (1 mg/kg, 24 hours post-MI and continued for 6 days) a selective antagonist of the SDF-1 alpha receptor, CXCR4. This receptor is coupled to a G alpha i protein and induced migration and proliferation of cells. In 1-week post-MI rats, chemokine expression was detected in smooth muscle and endothelial cells of blood vessels residing in the infarcted region. An SDF-1 alpha gradient towards the infarcted region was detected in the post-MI rat heart. In 1-week post-MI rats, AMD3100 therapy reduced scar size, concomitantly improved left ventricular function and partially supressed the elevated expression of ANP mRNA in the non-infarcted left ventricule. Preliminary studies on mice showed that the reduced infarct size in AMD3100-treated post-MI mice was associated with an attenuation of neutrophil infiltration in the ischemic region. These data highlight the novel observation that pharmacological antagonism of the SDF-1 alpha/CXCR4 axis during the acute phase of repartive fibrosis post-MI attenuated scar expansion and improved ventricular function in part via attenuation of the inflammatory response.
Books on the topic "Scar remodeling"
Dilsizian, Vasken, Ines Valenta, and Thomas H. Schindler. Myocardial Viability Assessment. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199392094.003.0021.
Full textBook chapters on the topic "Scar remodeling"
Weber, Karl T. "Infarct Scar. Living Tissue." In Cardiac Remodeling and Failure, 333–43. Boston, MA: Springer US, 2003. http://dx.doi.org/10.1007/978-1-4419-9262-8_23.
Full textOgawa, Rei. "Mechanobiology of Cutaneous Scarring." In Textbook on Scar Management, 11–18. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44766-3_2.
Full textLiu, Wei. "Emerging Technologies in Scar Management: Remodeling of Post-surgical Linear Scar Using Microplasma Radiofrequency." In Textbook on Scar Management, 465–73. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44766-3_53.
Full textOgawa, Rei. "Ideal Wound Closure Methods for Minimizing Scarring After Surgery." In Textbook on Scar Management, 185–91. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44766-3_21.
Full textMoortgat, Peter, Mieke Anthonissen, Ulrike Van Daele, Jill Meirte, Tine Vanhullebusch, and Koen Maertens. "Shock Wave Therapy for Wound Healing and Scar Treatment." In Textbook on Scar Management, 485–90. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44766-3_55.
Full textDarby, Ian A., and Alexis Desmoulière. "Scar Formation: Cellular Mechanisms." In Textbook on Scar Management, 19–26. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-44766-3_3.
Full text"Chapter 18 SCAR REMODELING WITH FAT GRAFTING AFTER BURN INJURY." In Fat Injection, edited by Sydney R. Coleman, Riccardo F. Mazzola, and Lee L. Q. Pu. Stuttgart: Georg Thieme Verlag, 2018. http://dx.doi.org/10.1055/b-0038-149554.
Full textAbboud, Nicolas, and Marwan Abboud. "Power-Assisted Liposuction Mammaplasty (PALM): A Short Scar Mammaplasty in Gigantomastia." In Enhanced Liposuction - New Perspectives and Techniques [Working Title]. IntechOpen, 2021. http://dx.doi.org/10.5772/intechopen.98816.
Full textMankiewicz, Kimberly A., and Leonard K. Seibold. "Wound Healing In Glaucoma." In Complications of Glaucoma Surgery. Oxford University Press, 2013. http://dx.doi.org/10.1093/oso/9780195382365.003.0013.
Full textCastelvecchio, Serenella, Raffaella Molfetta, Andrea Garatti, and Lorenzo Menicanti. "Coronary artery bypass grafting with surgical ventricular reconstruction." In State of the Art Surgical Coronary Revascularization, edited by Naresh Trehan and Yasir Abu-Omar, 431–34. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780198758785.003.0074.
Full textConference papers on the topic "Scar remodeling"
Nagel, Thomas, and Daniel J. Kelly. "Compaction and Anisotropy Induced by Remodeling of the Collagen Network’s State of Tension-Compression Transition." In ASME 2011 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2011. http://dx.doi.org/10.1115/sbc2011-53399.
Full textRichardson, William J., and Jeffrey W. Holmes. "Do Infarcts Really Expand or Compact? Relationship Between Changing Material Properties and Apparent Infarct Remodeling." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14411.
Full textShehata, Monda L., Jan Skrok, Dirk Lossnitzer, Sukhminder Singh, Danielle Boyce, Noah Lechtzin, Stephen C. Mathai, et al. "Myocardial Scar In Pulmonary Hypertension: Relationship To Pulmonary Hemodynamics, Right Ventricular Function And Remodeling." In American Thoracic Society 2010 International Conference, May 14-19, 2010 • New Orleans. American Thoracic Society, 2010. http://dx.doi.org/10.1164/ajrccm-conference.2010.181.1_meetingabstracts.a5536.
Full textFomovsky, Gregory M., and Jeffrey W. Holmes. "Evolution of Scar Mechanical Properties During Myocardial Infarct Healing in Rat." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176422.
Full textTrubelja, Alen, John W. MacArthur, Joseph J. Sarver, Jeffrey E. Cohen, Yasuhiro Shudo, Alexander S. Fairman, Jay Patel, William Hiesinger, Pavan Atluri, and Y. Joseph Woo. "Bioengineered SDF-1a Analogue Delivered as an Angiogenic Therapy Significantly Normalizes Elastic and Viscoelastic Material Properties of Infarcted Cardiac Muscle." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14602.
Full textDischer, Dennis, and Adam Engler. "Mesenchymal Stem Cell Injection After Myocardial Infarction Improves Myocardial Compliance." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-176754.
Full textTang, Dalin, Chun Yang, Tal Geva, Glenn Gaudette, and Pedro J. del Nido. "Regenerated Contracting Myocardium May Improve Post-Surgery Right Ventricle Function: Patch Comparison Using MRI-Based Two-Layer Anisotropic Models of Human Right and Left Ventricles." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19067.
Full textRajabi-Jaghargh, Ehsan, Mahesh K. Krishnamoorthy, and Rupak K. Banerjee. "Longitudinal Effect of Pressure Drop on the Intima-Media Thickening of the Venous Segment of the Arteriovenous Fistula." In ASME 2013 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2013. http://dx.doi.org/10.1115/sbc2013-14478.
Full textScotti, Christine M., Ender A. Finol, Siddharth Viswanathan, Aleksandr Shkolnik, Elena S. DiMartino, David A. Vorp, and Cristina H. Amon. "Computational Fluid Dynamics and Solid Mechanics Analyses of a Patient-Specific AAA Pre- and Post-EVAR." In ASME 2004 International Mechanical Engineering Congress and Exposition. ASMEDC, 2004. http://dx.doi.org/10.1115/imece2004-62352.
Full textFan, Rong, Michael S. Sacks, Ahmed Bayoumi, John E. Mayer, Christopher M. Hobson, and William R. Wagner. "Optimization of Engineered Ovine Pulmonary Heart Valve Leaflet Tissue Shape for Single Leaflet Replacement." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80824.
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