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Books on the topic 'Schizophrenia Cognition disorders'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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1

Harvey, Philip D. Cognitive Impairment in Schizophrenia: Characteristics, Assessment and Treatment. Cambridge, England: Cambridge University Press, 2013.

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2

Schizophrenia from a neurocognitive perspective: Probing the impenetrable darkness. Boston: Allyn and Bacon, 1998.

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3

Brain protection in schizophrenia, mood, and cognitive disorders. Dordrecht: Springer, 2010.

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4

Quinlan, Donald M., joint author., ed. Disordered thinking and schizophrenic psychopathology. New York: Gardner Press, 1985.

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5

Harrow, Martin. Disordered thinking and schizophrenic psychopathology. New York: Gardner Press, 1985.

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6

Clare, Reeder, ed. Cognitive remediation therapy for schizophrenia: An introduction. New York: Brunner-Routledge, 2005.

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7

Roder, Volker. Neurocognition and social cognition in schizophrenia patients: Basic concepts and treatment. Basel [Switzerland]: Karger, 2010.

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8

Wykes, Til. Cognitive remediation therapy for schizophrenia: Theory and practice. London: Routledge, 2005.

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9

1953-, Okoniewski L. A., and Lehman M. 1955-, eds. Cognitive synthesis test. Berlin: Springer-Verlag, 1987.

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10

Harvey, Philip D. Understanding and Treating Cognition in Schizophrenia: A Clinician's Handbook. London, England: Dunitz, 2002.

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11

Three papers on the theory of mental health sciences. Stuttgart: Hippokrates, 1987.

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12

Ritsner, Michael S., ed. Brain Protection in Schizophrenia, Mood and Cognitive Disorders. Dordrecht: Springer Netherlands, 2010. http://dx.doi.org/10.1007/978-90-481-8553-5.

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13

Staying well after psychosis: A cognitive interpersonal approach to recovery and relapse prevention. Chichester, UK: John Wiley & Sons, 2006.

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14

Matthias, Schwannauer, ed. Staying well after psychosis: A cognitive interpersonal approach to recovery and relapse prevention. Hoboken, NJ: Wiley, 2006.

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15

Gumley, Andrew. Staying Well After Psychosis. New York: John Wiley & Sons, Ltd., 2006.

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16

Schizophrenia: A neuropsychological perspective. Chichester: J. Wiley, 1996.

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17

(Editor), Christos Pantelis, Hazel E. Nelson (Editor), and Thomas E. Barnes (Editor), eds. Schizophrenia: A Neuropsychological Perspective. John Wiley & Sons Ltd (Import), 1997.

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18

Henrik, Lublin, ed. Cognitive dysfunction in schizophrenia. Copenhagen: Munksgaard, 2001.

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19

T, Beck Aaron, ed. Schizophrenia: Cognitive theory, research, and therapy. New York: Guilford Press, 2009.

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20

Schizophrenia: Cognitive Theory, Research, and Therapy. Guilford Publications, 2011.

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21

Cognitive Neuropsychology of Schizophrenia. Taylor & Francis Group, 2015.

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22

Cognitive Neuropsychology of Schizophrenia. Taylor & Francis Group, 2015.

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23

(Editor), Tonmoy Sharma, and Philip Harvey (Editor), eds. Cognition in Schizophrenia: Impairments, Importance, and Treatment Strategies. Oxford University Press, USA, 2000.

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24

Serper, Mark Richard. Controlled and automatic information processing and positive and negative thought disorder in schizophrenia. 1987.

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25

Ritsner, Michael S. Brain Protection in Schizophrenia, Mood and Cognitive Disorders. Springer, 2014.

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26

Volker, Roder, and Medalia Alice, eds. Neurocognition and social cognition in schizophrenia patients: Comprehension and treatment. Basel: Karger, 2010.

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27

Phenomenology, Language & Schizophrenia. Springer, 2011.

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28

Spitzer, Manfred. Phenomenology, Language & Schizophrenia. Springer, 2011.

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29

Manfred, Spitzer, ed. Phenomenology, language & schizophrenia. New York: Springer-Verlag, 1992.

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30

Neurocognition and social cognition in schizophrenia patients: Basic concepts and treatment. Basel: Karger, 2010.

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31

Tanenbaum, Robbi R. Distractability, reality monitoring, and thought disorder: Their association in mania and schizophrenia. 1987.

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32

Negative Symptom and Cognitive Deficit Treatment Response in Schizophrenia. American Psychiatric Publishing, Inc., 2001.

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33

Frith, Christoph. The Cognitive Neuropsychology Of Schizophrenia (Essays in Cognitive Psychology). Psychology Press, 1995.

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34

The Cognitive Neuropsychology Of Schizophrenia (Essays in Cognitive Psychology). Psychology Press, 1992.

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35

Understanding and Treating Cognition in Schizophrenia: A Clinician's Handbook. Informa Healthcare, 2002.

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36

E, Keefe Richard S., and McEvoy Joseph P. 1948-, eds. Negative symptom and cognitive deficit treatment response in schizophrenia. Washington, DC: American Psychiatric Press, 2001.

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37

Penn, David L., David L. Roberts, and Dennis R. Combs. Social Cognition and Interaction Training: Group Psychotherapy for Schizophrenia and Other Psychotic Disorders, Clinician Guide. Oxford University Press, Incorporated, 2015.

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38

Wells, Adrian, and Gillian Butler. Generalized anxiety disorder. Oxford University Press, 2015. http://dx.doi.org/10.1093/med:psych/9780192627254.003.0007.

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Chapter 7 discusses generalized anxiety disorder (GAD), and argues for an improved conceptual understanding of GAD, based on experimental and clinical observations. It first outlines the nature of the problem, and the development of existing psychological treatments, before focusing on experimental and theoretical work on generalized anxiety and worry. A cognitive model of GAD is discussed, along with its treatment implications, and the concepts used in constructing models of vulnerability to stress in general, and of GAD in particular, are discussed in relation to models of cognition in disorders such as obsessive-compulsive disorder (OCD) and schizophrenia.
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39

Keshavan, Matcheri, and Shaun Eack. Cognitive Enhancement in Schizophrenia and Related Disorders. Cambridge University Press, 2019.

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40

Young, Jared W., Alan Anticevic, and Deanna M. Barch. Cognitive and Motivational Neuroscience of Psychotic Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0016.

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Schizophrenia is a complex neuropsychiatric syndrome presenting with a constellation of symptoms. Clinicians have long recognized that abnormalities in cognitive function and motivated behavior are a key component of psychosis, and of schizophrenia in particular. Here we postulate that these deficits may reflect, at least in part, impairments in the ability to actively maintain and utilize internal representations of emotional experiences, previous rewards, and motivational goals in order to drive current and future behavior in a way that would normally allow individuals to obtain desired outcomes. We discuss the evidence for such impairment in schizophrenia, how it manifests in domains typically referred to as executive control, working memory, and episodic memory, how it may help us understand impairments in reward processing and motivation in schizophrenia, and the animal research consistent with these hypotheses.
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41

Bachman, Peter, and Tyrone D. Cannon. The Cognitive Neuroscience of Thought Disorder in Schizophrenia. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780199734689.013.0034.

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42

McCauley, Robert N., and George Graham. Hearing Voices and Other Matters of the Mind. Oxford University Press, 2020. http://dx.doi.org/10.1093/oso/9780190091149.001.0001.

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This book endorses an ecumenical naturalism toward all cognition, which will illuminate the long-recognized and striking similarities between features of mental disorders and features of religions. The authors emphasize underlying cognitive continuities between familiar features of religiosity, of mental disorders, and of everyday thinking and action. They contend that much religious thought and behavior can be explained in terms of the cultural activation of maturationally natural cognitive systems, which address fundamental problems of human survival, encompassing such capacities as hazard precautions, agency detection, language processing, and theory of mind. The associated skills are not taught and appear independent of general intelligence. Religions’ representations cue such systems’ operations. The authors hypothesize that in doing so they sometimes elicit responses that mimic features of cognition and conduct associated with mental disorders. Both in schizophrenia and in religions some people hear alien voices. The inability of depressed participants to communicate with or sense their religions’ powerful, caring gods can exacerbate their depression. Often religions can domesticate the concerns and compulsions of people with OCD. Religions’ rituals and pronouncements about moral thought-action fusion can temporarily evoke similar obsessions and compulsions in the general population. A chapter is devoted to each of these and to the exception that proves the rule. The authors argue that if autistic spectrum disorder involves theory-of mind-deficits, then people with ASD will lack intuitive insight and find inferences with many religious representations challenging. Ecumenical naturalism’s approach to mental abnormalities and religiosity promises both explanatory and therapeutic understanding.
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43

Cohen, Alex S., Dallas A. Callaway, and Tracey L. Auster. Schizophrenia. Edited by C. Steven Richards and Michael W. O'Hara. Oxford University Press, 2014. http://dx.doi.org/10.1093/oxfordhb/9780199797004.013.011.

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Depressive symptoms commonly occur in individuals with schizophrenia-spectrum disorders. Empirical investigation of this comorbidity has revealed a number of interesting and potentially confusing findings. The purpose of this review is to summarize this literature, focusing on clinical, cognitive, behavioral, phenomenological, and neurobiological processes that are common and potentially disparate to these disorders. Additionally, the review will discuss four depression-related paradoxes that have emerged within the schizophrenia literature. It concludes with a brief summary of treatment considerations for patients with schizophrenia with co-morbid depressive symptoms. It is hoped that this chapter can serve as an organizing framework for future research and can help focus efforts on designing new treatments for ameliorating depression-related symptoms in patients with schizophrenia.
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44

Beauchaine, Theodore P., and Sheila E. Crowell, eds. The Oxford Handbook of Emotion Dysregulation. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780190689285.001.0001.

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Emotion dysregulation—which is often defined as the inability to modulate strong affective states including impulsivity, anger, fear, sadness, and anxiety—is observed in nearly all psychiatric disorders. These include internalizing disorders such as panic disorder and major depression, externalizing disorders such as conduct disorder and antisocial personality disorder, and various other disorders including schizophrenia, autism, and borderline personality disorder. Among many affected individuals, precursors to emotion dysregulation appear early in development, and often predate the emergence of diagnosable psychopathology. Collaborative work by Drs. Crowell and Beauchaine, and work by many others, suggests that emotion dysregulation arises from both familial (coercion, invalidation, abuse, neglect) and extrafamilial (deviant peer group affiliations, social reinforcement) mechanisms. These studies point toward strategies for prevention and intervention. The Oxford Handbook of Emotion Dysregulation brings together experts whose work cuts across levels of analysis, including neurobiological, cognitive, and social, in studying emotion dysregulation. Contributing authors describe how early environmental risk exposures shape emotion dysregulation, how emotion dysregulation manifests in various forms of mental illness, and how emotion dysregulation is most effectively assessed and treated. This is the first text to assemble a highly accomplished group of authors to address conceptual issues in emotion dysregulation research; define the emotion dysregulation construct at levels of cognition, behavior, and social dynamics; describe cutting-edge assessment techniques at neural, psychophysiological, and behavioral levels of analysis; and present contemporary treatment strategies. Conceptualizing emotion dysregulation as a core vulnerability to psychopathology is consistent with modern transdiagnostic approaches to diagnosis and treatment, including the Research Domain Criteria and the Unified Protocol, respectively.
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45

Charakeristische Selbstwahrnehmungen kognitiver Dysfunktionen Schizophrener. Aachen, Germany: Shaker, 2003.

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46

Dienel, Samuel J., and David A. Lewis. Cellular Mechanisms of Psychotic Disorders. Edited by Dennis S. Charney, Eric J. Nestler, Pamela Sklar, and Joseph D. Buxbaum. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190681425.003.0018.

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Cognitive dysfunction in schizophrenia, including disturbances in working memory, is a core feature of the illness and the best predictor of long-term functional outcome. Working memory relies on neural network oscillations in the prefrontal cortex. Gamma-aminobutyric acid (GABA) neurons in the prefrontal cortex, which are crucial for this oscillatory activity, exhibit a number of alterations in individuals diagnosed with schizophrenia. These GABA neuron disturbances may be secondary to upstream alterations in excitatory pyramidal cells in the prefrontal cortex. Together, these findings suggest both a neural substrate for working memory impairments in schizophrenia and therapeutic targets for improving functional outcomes in this patient population.
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47

Medalia, Alice, Tiffany Herlands, Alice Saperstein, and Nadine Revheim. Cognitive Remediation for Psychological Disorders. Oxford University Press, 2017. http://dx.doi.org/10.1093/med-psych/9780190608453.001.0001.

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Individuals with serious and persistent mental illnesses, including schizophrenia and affective disorders, often experience cognitive deficits that make it difficult to perform everyday tasks. For example, they may have difficulty with attention, memory, processing speed, and problem solving, and this may interfere with functioning at work, school, and in social situations. Cognitive remediation is an evidence-based behavioral treatment for people who are experiencing cognitive impairments that interfere with role functioning. This edition contains all the information needed to set up a cognitive remediation program so clients can strengthen the cognitive skills needed for everyday functioning. The program described is called Neuropsychological and Educational Approach to Remediation (NEAR), which is an evidence-based approach to cognitive remediation that uses carefully crafted instructional techniques that reflect an understanding of how people learn best. The goals of NEAR are to provide a positive learning experience, to promote independent learning, and to promote optimal cognitive functioning in daily life. This second edition of the popular 2009 therapist’s guide provides step-by-step instructions on how to implement NEAR techniques with patients to improve their cognitive functioning and quality of life. Guidelines are provided for setting up and running a successful cognitive remediation program. Therapists learn how to choose appropriate cognitive exercises, recruit and work with clients, perform intake interviews, and create treatment plans. This guide comes complete with all the tools necessary for facilitating treatment, including program evaluation forms and client handouts.
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48

Alavi, Abass, and Andrew B. Newberg. Functional Neuroimaging: A Transformative Tool for Integrative Psychiatry. Edited by Anthony J. Bazzan and Daniel A. Monti. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190690557.003.0014.

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Functional neuroimaging with positron emission tomography (PET), single photon emission computed tomography (SPECT), and functional magnetic resonance imaging (fMRI) can be highly useful in the evaluation and management of patients with psychiatric disorders. PET and SPECT imaging typically evaluate cerebral metabolism and blood flow, respectively, and can determine patterns associated with different disorders such as depression or schizophrenia. PET and SPECT imaging can also evaluate neurotransmitter changes such as dopamine or serotonin associated with different psychiatric disorders. fMRI is an excellent tool for studying the effects of psychiatric disorders on specific brain processes related to cognition and mood. fMRI activations studies allow researchers to present various stimuli to a subject in order to determine how the brain reacts and whether psychiatric disorders are associated with different brain reactivity patterns. Functional neuroimaging with PET, SPECT, and fMRI can be highly useful in the investigation of the mechanism of action of integrative therapies for psychiatric disorders.
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49

Cummings, Jeffrey, and Kate Zhong. Promise and Challenges in Drug Development and Assessment for Cognitive Enhancers. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190214401.003.0001.

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Cognitive disturbances are ubiquitous in neurologic and psychiatric disorders. Schizophrenia, depression, developmental disorders, acquired brain disorders (traumatic brain injury and stroke), and neurodegenerative disorders all have cognitive impairment as a manifestation. Cognitive enhancers can improve intellectual function and have been approved for Alzheimer’s dementia, dementia of Parkinson’s disease, and attention deficit hyperactivity disorder. Cognitive enhancers are being developed for other cognitive disorders. There are many advantages for development of symptomatic cognitive enhancers compared to disease-modifying agents. Cognitive enhancers typically modulate transmitter systems. Cross-disease phenotypes such as executive function impairment may represent a development strategy for cognitive enhancing agents. Life cycle management strategies for cognitive enhancers include expanding indications to disorders with related pathophysiology or to different stages of disease severity and development of alternate formulations. Cognitive enhancers can restore essential cognitive capability and are a critical element of optimal care of patients with neurologic and psychiatric disorders.
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50

Prasad, Konasale M. Course, Prognosis, and Outcomes of Schizophrenia and Related Disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199331505.003.0004.

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Course and outcome in schizophrenia and related disorders historically depend on diagnostic conceptualizations, with significant variability even across individuals with the exact same diagnosis. In this chapter, we will review the heterogeneity of course and outcome, providing some context in terms of factors that affect prognosis. Generally speaking, current outcomes are better than previously thought, with three-quarters of individuals having a good prognosis. Although these illnesses cannot be cured, we know that recovery is possible. The best predictors of outcome in schizophrenia are cognitive and negative symptoms (not positive symptoms), along with premorbid functioning, duration of untreated psychosis, and treatment adherence over time. Finally, we will touch on functional outcomes such as risk of violence and suicide, as well as issues around treatment discontinuation.
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