Relevant bibliographies by topics / Schizophrenics Schizophrenia Youth

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Academic literature on the topic 'Schizophrenics Schizophrenia Youth'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Schizophrenics Schizophrenia Youth"

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Kaleda, V. G., and M. A. Omelchenko. "The Clinical Course and Prognostic Problems of Youth Depressions with Attenuated Schizophrenic Symptoms." V.M. BEKHTEREV REVIEW OF PSYCHIATRY AND MEDICAL PSYCHOLOGY, no. 1 (April 12, 2021): 42–52. http://dx.doi.org/10.31363/2313-7053-2021-1-42-52.

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Objective Clinical and follow-up verification of Attenuated Schizophrenic Symptoms (ASS) in the first youth depressive episode as early markers of the schizophrenic process, establishing further variants of the course of the disorder and its outcomes.Materials and methods. 124 young inpatients (averaged age 19,6±2,3 years) with the first depressive episode with ASS were examined. The control group consisted of 27 patients with youth depression without ASS. All patients have been tracked for at least five years. The average follow-up period was 7,1±1,6 years. The HDRS, SOPS, SANS and PSP scales were used to assess the symptomatic and functional outcomes. Statistical analysis was carried out using STATISTICA 12.Results. The typological classification of youth depressions (ASD) with ASS has been developed with the identification of three main types: (1) with attenuated positive symptoms (APS), (2) with attenuated negative symptoms (ANS), and (3) with attenuated symptoms of disorganization. Youth depression with ASS, compared to the control group, is more likely to move into chronic forms, has reliably worse functional and symptomatic outcomes, and is more associated with the diagnosis of schizophrenic spectrum disorders at five years follow-up.Conclusion. Attenuated schizophrenic symptoms in the structure of youth depressions have high affinity to each other, indicating a common pathogenic mechanism of their formation, and also have predicate value as risk factors for schizophrenia.
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Jungbauer, Johannes, Jutta Kinzel-Senkbeil, Juliane Kuhn, and Albert Lenz. "Familien mit einem schizophren erkrankten Elternteil: Ergebnisse einer fallrekonstruktiven Familienstudie." Journal of Family Research 23, no. 1 (April 1, 2011): 57–76. http://dx.doi.org/10.20377/jfr-234.

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Objective: This study aims at investigating the impact of a parental schizophrenia on the family members, their everyday life and their relations. For this purpose, we conduct qualitative interviews with mothers and fathers suffering from schizophrenia, their spouses and children. Methods: Interview data is analyzed using casereconstructive as well as content analysis methods. Results: Although results illustrate a great variety of family constellations and burdening circumstances, there are a number of typical patterns: Having children is perceived by affected parents in an ambiguous manner, i.e. as a resource as well as a distress. Relationships of couples and families are often impaired, resulting in a high risk of abandonment of relationships. At the same time, family members strive for normality in everyday life. Normalisation and avoidance strategies can bring about that the schizophrenia becomes a taboo issue within the family. Thus, with regard to their parent’s illness, many of the children are insufficiently informed. Often, the children are overstrained by this situation and, in turn, may develop behaviour disorders, anxiety, or depression. Discussion: In sum, schizophrenia can be considered as a “family disease” as it strongly affects the whole family system. Hence, it is necessary to provide preventive help offers for affected parents, their spouses and children. For delivering support, youth welfare and public health services should cooperate closely. Zusammenfassung Fragestellung: In diesem Beitrag werden Ergebnisse einer fallrekonstruktiven Studie vorgestellt, bei der Familien mit einem schizophren erkrankten Elternteil befragt wurden. Dabei sollte untersucht wurden, wie sich die Schizophrenie auf die Familienmitglieder, ihren Alltag und ihre Beziehungen auswirkt. Methodik: Die Auswertung erfolgte sowohl fall- als auch themenbezogen, wobei inhaltsanalytische und fallrekonstruktive Verfahren eingesetzt wurden. Ergebnisse: Trotz der Vielfalt der familiären Konstellationen und Belastungslagen zeigte sich eine Reihe charakteristischer Muster. Kinder zu haben bedeutet für erkrankte Eltern, sowohl Ressourcen als auch Belastungen zu haben. Paar- und Familienbeziehungen sind oft stark beeinträchtigt und weisen ein hohes Risiko für Beziehungsabbrüche auf. Zugleich wird im Familienalltag eine Normalität jenseits der Erkrankung angestrebt und erlebt. Normalisierungs- und Vermeidungsstrategien können dazu beitragen, dass die Erkrankung zu einem Tabuthema wird. Viele Kinder sind daher unzureichend über die elterliche Schizophrenie informiert. Sie sind in dieser Situation oft überfordert und entwickeln ihrerseits Verhaltensauffälligkeiten, Ängste und Depressionen. Diskussion: Die Schizophrenie kann insofern als „Familienerkrankung“ gedeutet werden, als sie das gesamte Familiensystem beeinflusst, belastet und gefährdet. Aus diesem Grund sollten verstärkt familienorientierte Präventionsangebote bereitgestellt werden, wobei Gesundheitswesen und Jugendhilfe eng miteinander kooperieren sollten.
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Rajji, T. K., Z. Ismail, and B. H. Mulsant. "Age at onset and cognition in schizophrenia: meta-analysis." British Journal of Psychiatry 195, no. 4 (October 2009): 286–93. http://dx.doi.org/10.1192/bjp.bp.108.060723.

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BackgroundThe relationship between cognition and age at onset of schizophrenia is largely unknown.AimsTo compare cognitive deficits in individuals with youth-onset and late-onset schizophrenia with those in adults with first-episode schizophrenia.MethodTwenty-nine databases (including EMBASE, MEDLINE and PsycINFO) were searched from 1980 to 2008. Selected publications had to include healthy controls and analyse separately individuals diagnosed with schizophrenia or a related disorder and individuals with first-episode, youth-onset or late-onset schizophrenia. Descriptive and cognitive data were extracted and the latter aggregated into 22 cognitive measures. Cohen's effect size raw and weighted means of cognitive deficits were generated and compared in the three groups.ResultsIndividuals with youth-onset and first-episode schizophrenia demonstrate large deficits (mean effect size ⩾0.8) on almost all cognitive measures. Individuals with youth-onset schizophrenia demonstrate larger deficits than those with first-episode schizophrenia on arithmetic, executive function, IQ, psychomotor speed of processing and verbal memory. In contrast, those with late-onset schizophrenia demonstrate minimal deficits on arithmetic, digit symbol coding and vocabulary, but larger ones on attention, fluency, global cognition, IQ and visuospatial construction.ConclusionsIndividuals with youth-onset schizophrenia have severe cognitive deficits, whereas those with late-onset schizophrenia have some relatively preserved cognitive functions. This finding supports the view that severity of the disease process is associated with different ages at onset. In addition, the cognitive pattern of people with late-onset schizophrenia suggests that their deficits are specific rather than solely as a result of ageing and related factors.
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Dickson, Hannah, Emily P. Hedges, Shin Y. Ma, Alexis E. Cullen, James H. MacCabe, Matthew J. Kempton, Johnny Downs, and Kristin R. Laurens. "Academic achievement and schizophrenia: a systematic meta-analysis." Psychological Medicine 50, no. 12 (July 20, 2020): 1949–65. http://dx.doi.org/10.1017/s0033291720002354.

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AbstractBackgroundCognitive impairments in childhood are associated with increased risk of schizophrenia in later life, but the extent to which poor academic achievement is associated with the disorder is unclear.MethodsMajor databases were searched for articles published in English up to 31 December 2019. We conducted random-effects meta-analyses to: (1) compare general academic and mathematics achievement in youth who later developed schizophrenia and those who did not; (2) to examine the association between education level achieved and adult-onset schizophrenia; and, (3) compare general academic achievement in youth at-risk for schizophrenia and typically developing peers. Meta-regression models examined the effects of type of academic assessment, educational system, age at assessment, measurement of educational level attained, school leaving age, and study quality on academic achievement and education level among individuals with schizophrenia.ResultsMeta-analyses, comprising data of over four million individuals, found that: (1) by age 16 years, those who later developed schizophrenia had poorer general academic (Cohen's d = −0.29, p ⩽ 0.0001) and mathematics achievement (d = −0.23, p = 0.01) than those who did not; (2) individuals with schizophrenia were less likely to enter higher education (odds ratio = 0.49, p ⩽ 0.0001); and, (3) youth reporting psychotic-like experiences and youth with a family history of schizophrenia had lower general academic achievement (d = −0.54, p ⩽ 0.0001; d = −0.39, p ⩽ 0.0001, respectively). Meta-regression analyses determined no effect modifiers.DiscussionDespite significant heterogeneity across studies, various routinely collected indices of academic achievement can identify premorbid cognitive dysfunction among individuals who are vulnerable for schizophrenia, potentially aiding the early identification of risk in the population.
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Lim, Keane, Max Lam, Hailiang Huang, Jianjun Liu, and Jimmy Lee. "Genetic liability in individuals at ultra-high risk of psychosis: A comparison study of 9 psychiatric traits." PLOS ONE 15, no. 12 (December 2, 2020): e0243104. http://dx.doi.org/10.1371/journal.pone.0243104.

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Individuals at ultra-high risk (UHR) of psychosis are characterised by the emergence of attenuated psychotic symptoms and deterioration in functioning. In view of the high non-psychotic comorbidity and low rates of transition to psychosis, the specificity of the UHR status has been called into question. This study aims to (i) investigate if the UHR construct is associated with the genetic liability of schizophrenia or other psychiatric conditions; (ii) examine the ability of polygenic risk scores (PRS) to discriminate healthy controls from UHR, remission and conversion status. PRS was calculated for 210 youths (nUHR = 102, nControl = 108) recruited as part of the Longitudinal Youth at Risk Study (LYRIKS) using nine psychiatric traits derived from twelve large-scale psychiatric genome-wide association studies as discovery datasets. PRS was also examined to discriminate UHR-Healthy control status, and healthy controls from UHR remission and conversion status. Result indicated that schizophrenia PRS appears to best index the genetic liability of UHR, while trend level associations were observed for depression and cross-disorder PRS. Schizophrenia PRS discriminated healthy controls from UHR (R2 = 7.9%, p = 2.59 x 10−3, OR = 1.82), healthy controls from non-remitters (R2 = 8.1%, p = 4.90 x 10−4, OR = 1.90), and converters (R2 = 7.6%, p = 1.61 x 10−3, OR = 1.82), with modest predictive ability. A trend gradient increase in schizophrenia PRS was observed across categories. The association between schizophrenia PRS and UHR status supports the hypothesis that the schizophrenia polygenic liability indexes the risk for developing psychosis.
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Seidman, Larry J., and Allan F. Mirsky. "Evolving Notions of Schizophrenia as a Developmental Neurocognitive Disorder." Journal of the International Neuropsychological Society 23, no. 9-10 (October 2017): 881–92. http://dx.doi.org/10.1017/s1355617717001114.

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AbstractWe review the changing conceptions of schizophrenia over the past 50 years as it became understood as a disorder of brain function and structure in which neurocognitive dysfunction was identified at different illness phases. The centrality of neurocognition has been recognized, especially because neurocognitive deficits are strongly related to social and role functioning in the illness, and as a result neurocognitive measures are used routinely in clinical assessment of individuals with schizophrenia. From the original definitions of the syndrome of schizophrenia in the early 20th century, impaired cognition, especially attention, was considered to be important. Neurocognitive impairments are found in the vast majority of individuals with schizophrenia, and they vary from mild, relatively restricted deficits, to dementia-like syndromes, as early as the first psychotic episode. Neurocognitive deficits are found in the premorbid phase in a substantial minority of pre-teenage youth who later develop schizophrenia, and they apparently worsen by the prodromal, high-risk phase in a majority of those who develop the illness. While there is limited evidence for reversibility of impairments from pharmacological interventions in schizophrenia, promising results have emerged from cognitive remediation studies. Thus, we expect cognitive interventions to play a larger role in schizophrenia in the coming years. Moreover, because youth at risk for schizophrenia can be identified by an emergent high-risk syndrome, earlier interventions might be applied in a pre-emptive way to reduce disability and improve adaptation. The notion of schizophrenia as a developmental neurocognitive disorder with stages opens up a window of possibilities for earlier interventions. (JINS, 2017, 23, 881–892)
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Jalbrzikowski, Maria, Lambertus Klei, William Foran, Beatriz Luna, and Bernie Devlin. "O11.1. MULTIPLE GENOMIC MEASURES OF SCHIZOPHRENIA RISK ARE RELATED TO CORTICAL THICKNESS IN TYPICALLY DEVELOPING YOUTH AND YOUNG ADULTS." Schizophrenia Bulletin 46, Supplement_1 (April 2020): S27. http://dx.doi.org/10.1093/schbul/sbaa028.061.

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Abstract Background The incidence of psychotic disorders increases in adolescence and young adulthood. Transition to a psychotic disorder is associated with atypical development of brain structures, specifically protracted developmental course. It is unknown how polygenic risk for schizophrenia and gene expression profiles of schizophrenia risk genes affect typical brain development. The goal of the current study is to examine relationships multiple genomic measures associated with schizophrenia risk and structural neuroimaging measures thickness in typically developing youth. Methods We combined structural neuroimaging and genetic data from three different cohorts of typically developing youth (N=994, 5–30 years old): the Philadelphia Neurodevelopmental Cohort, Pediatric Imaging Neurocognition and Genetics Study, and a locally collected sample at the University of Pittsburgh. All youth were free from psychiatric disorders and not taking psychiatric medications. We used Freesurfer to process the T1-weighted structural scans and calculate subcortical volumes, cortical thickness, and surface area measurements. After regressing out study, sex, ancestry eigenvectors, and grey matter signal-to-noise ratio, we ran principal components analysis on all neuroimaging measures (N=156). We calculated a schizophrenia polygenic risk score using genome-wide summary statistics from the Psychiatric Genome Consortium. Using a generalized linear model, each of the top five principal components was evaluated in relation to the risk score. We then used a computational method, Predixcan, to calculate expected gene expression profiles from the genotype data. We selected 125 genes that were associated with schizophrenia in a previous case-control comparison. Elastic net regression was used to determine significant associations between individual gene expression and the principal components. Results Schizophrenia polygenic risk was statistically associated with the 5th principal component (b=-0.10, p=0.001), which consisted of contributions from multiple measures of cortical thickness. Reduced cortical thickness in frontal and temporal regions was associated with increased genetic liability for schizophrenia. Increased cortical thickness in sensory-motor areas was associated with higher schizophrenia polygenic risk scores. This relationship remained when age was included as a predictor of interest and there were no statistically significant interactions between schizophrenia polygenic risk and age. Sixteen unique gene expression profiles were also associated with this principal component, significantly increasing the proportion of variance explained in this measure (from ~1% with the schizophrenia polygenic risk only to ~6% when including the additional gene expression measures). Many of the genes significantly associated with this principal component have important roles during early fetal brain development, including neuronal migration (e.g., SDCCAG8) and DNA repair (e.g., MLH1). Discussion These results suggest that that genetic risk for schizophrenia has a consistent influence on subtle, individual differences in a distinct spatial pattern of cortical thickness across typical development. This spatial pattern of cortical thickness is also associated with schizophrenia risk genes that have important functions during early brain development. Taken together, these findings suggest that increased genetic risk for schizophrenia is related to early subtle alterations during early brain development, setting up individuals with higher risk profiles to have a small biological vulnerability for later developing the illness.
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O'Callaghan, Eadbhard, Conall Larkin, Oonagh Redmond, John Stack, Joseph T. Ennis, and John L. Waddington. "‘Early-Onset Schizophrenia’ After Teenage Head Injury." British Journal of Psychiatry 153, no. 3 (September 1988): 394–96. http://dx.doi.org/10.1192/bjp.153.3.394.

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A 14–year-old youth sustained an Injury to the left frontoparietal area, which was followed by evident change in personality and subsequently by an early-onset schizophrenia-like psychosis. Magnetic resonance imaging revealed ventricular dilatation, slightly more marked in the left hemisphere, and cortical atrophy. Some implications of this case for research on schizophrenia itself are discussed.
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Arsalidou, Marie, Zachary Yaple, Tomas Jurcik, and Vadim Ushakov. "Cognitive Brain Signatures of Youth With Early Onset and Relatives With Schizophrenia: Evidence From fMRI Meta-analyses." Schizophrenia Bulletin 46, no. 4 (January 24, 2020): 857–68. http://dx.doi.org/10.1093/schbul/sbz130.

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Abstract Deficits in cognitive function are a major characteristic of schizophrenia. Many functional magnetic resonance imaging (fMRI) studies examine brain correlates of cognitive function in adults with schizophrenia, showing altered implication of associative areas such as the prefrontal cortex and temporal cortex. fMRI studies also examine brain representation of cognitive function in adolescents with early onset schizophrenia and those at risk of the disorder, yet results are often inconsistent. We compile and analyze data from eligible fMRI studies using quantitative meta-analyses to reveal concordant brain activity associated with adolescent relatives of patients with schizophrenia and those with early onset schizophrenia. Results show similar functional hubs of brain activity (eg, precuneus) yet in opposite hemispheres and clusters in ventrolateral rather than dorsolateral prefrontal cortices. Other areas of altered implication include the middle temporal gyrus, insula, and cerebellum. We discuss the findings in reference to the protracted maturation of the prefrontal cortex and possible effects due to the medication status of the two groups.
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Dalsgaard, S., P. B. Mortensen, M. Frydenberg, C. M. Maibing, M. Nordentoft, and P. H. Thomsen. "Association between Attention-Deficit Hyperactivity Disorder in childhood and schizophrenia later in adulthood." European Psychiatry 29, no. 4 (May 2014): 259–63. http://dx.doi.org/10.1016/j.eurpsy.2013.06.004.

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AbstractPurpose:To estimate the risk of schizophrenia in adulthood among children and adolescents with ADHD compared to the background population.Subjects/materials and methods:Two hundred and eight youths with ADHD (183 boys; 25 girls) were followed prospectively. Diagnoses of schizophrenia were obtained from The Danish Psychiatric Central Register. The relative risk (RR) of schizophrenia for cases with ADHD, compared to the normal population, was calculated as risk ratios. Hazard ratios (HR's) by Cox regression were calculated in the predictor analyses.Results:Mean age for ADHD cases at follow-up was 31.1 years. Schizophrenia diagnoses were given to 3.8% of these cases. Compared to the general population, RR of schizophrenia in cases with ADHD was 4.3 (95% CI 1.9–8.57).Discussion and conclusion:This prospective follow-up study found children with ADHD to be at higher risk of later schizophrenia than controls. If replicated, these results warrant increased focus on the possible emergence symptoms of schizophrenia or schizophreniform psychosis during clinical follow-up of patients with ADHD.
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Dissertations / Theses on the topic "Schizophrenics Schizophrenia Youth"

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Slayton, Kristen A. "A psychoeducational support group for families of youth experiencing symptoms of schizophrenia| A grant proposal." Thesis, California State University, Long Beach, 2013. http://pqdtopen.proquest.com/#viewpdf?dispub=1523103.

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The purpose of this project was to develop a program to assist parents and caregivers of youth experiencing schizophrenia to address the needs and challenges of this population. The function of this grant proposal was to target a funding resource in order to implement a psychoeducational support group at The Guidance Center in Long Beach. The presence of schizophrenia symptoms in youth can create additional challenges to parenting and thus has a recognizable impact on families. The goal of this proposed program is to provide social support, education, coping skills, and decrease the stress of parents and caregivers of youth with schizophrenia to ultimately improve the lives of impacted youth and their families. The California Wellness Foundation was identified as a potential funder for this proposed program. The actual submission and/or funding of this grant proposal were not a requirement for the successful completion of this project.

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孫敏紅 and Man-hung Suen. "The influence of Chinese translations for psychosis on stigma of schizophrenia from youth service providers' views." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2013. http://hdl.handle.net/10722/192973.

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Objectives: The study investigates the effects of different Chinese translations for psychosis on perceptions of youth service providers. The hypothesis is youth service providers believe that to have better understanding and acceptance in name si-jue-shi-tiao rather than others. Participants and methods: 100 youth service providers were recruited and interviewed with 34-item questionnaires. They were presented with a vignette describing a person with jing-shen-fen-lie-zheng/ si-jue-shi-tiao/ jing-shen-bin. Belief of cause, benevolence, separatism, stereotyping, restrictiveness, pessimistic prediction and stigmatization of different labeling were investigated. Results: The study found that si-jue-shi-tiao group has less stigmatization effect compared with jing-shen-fen-lie-zheng group and Jing-shen-bin group but the psychiatric labeling has no statistically significant effect on benevolence, separatism, stereotyping, restrictiveness, pessimistic prediction. Conclusion: The study supported renaming psychosis has an improvement on stigmatization, but not obviously seen to have improvement in the other attitudinal dimensions. People who have religious belief, profession in occupation, having previous contact with people (e.g. friend and client) who have mental illness indeed affected to have positive effect on their views towards person with psychosis.
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Psychological Medicine
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Master of Psychological Medicine
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Greher, Felicia Reynolds. "Neuromotor and Neurocognitive Functioning in the Prediction of Cognition, Behavior Problems, and Symptoms at Two-year Follow-up in Youth with Schizotypal Personality Disorder." Thesis, University of North Texas, 2006. https://digital.library.unt.edu/ark:/67531/metadc5412/.

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Individuals diagnosed with schizotypal personality disorder (SPD) exhibit patterns of cognitive deficits, neuromotor disturbances, and behavior problems similar to individuals with schizophrenia, and thus SPD is thought to represent one point on the continuum of schizophrenia spectrum disorders (SSDs). Deficits in behavior, cognition, and motor functioning have been implicated as childhood precursors of SSDs and appear to also vary as a function of gender and family history of psychopathology. As such, studies of youth may help in further identification of individuals at risk for SSDs. The current study examined the prospective associations between problem behaviors, neuromotor and neurocognitive functioning, as well as SSD symptoms, at baseline and 2-year follow-up in youth meeting criteria for SPD, other personality disorders, or healthy controls. The neuromotor and neurocognitive measures were able to significantly predict SSD symptoms and behavior problems above and beyond baseline predictors. Overall, the findings provide further support for the role of subcortical motor centers operating together with prefrontal cortical areas in the regulation of higher-order cognitive functioning and in producing the psychiatric features of SSDs. Significant correlations between gender, family history of schizophrenia, and history of head injury with symptoms, behavior, cognition, and motor functioning were also found and highlight the importance of examining the effects of these variables in future investigations. In sum, the current study helped in identifying factors that predict the clinical course of schizotypy and may shed light on the disturbed neural circuitry underlying SSDs.
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Wu, Huey-Yuan, and 吳惠媛. "The voice of illness--a hermeneutics study of illness experience of youth schizophrenia:a hermeneutics study of illness experience of youth schizophrenia." Thesis, 1994. http://ndltd.ncl.edu.tw/handle/59904589841966561704.

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Lévesque, Isabelle Sarah. "L’itinérance chez une cohorte de jeunes adultes avec premier épisode psychotique : étude comparative à 2 ans de l’impact de l’itinérance sur l’évolution fonctionnelle et symptomatique." Thèse, 2018. http://hdl.handle.net/1866/22314.

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Books on the topic "Schizophrenics Schizophrenia Youth"

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Eggers, Christian, ed. Schizophrenia and Youth. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7.

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Remschmidt, Helmut, and Martin H. Schmidt, eds. Schizophrenie. Berlin, Germany: Springer Medizin, 2011.

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Characteristics of emotional and behavioral disorders of children and youth. 6th ed. Upper Saddle River, N.J: Merrill, 1997.

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Kauffman, James M. Characteristics of emotional and behavioral disorders of children and youth. 9th ed. Upper Saddle River, N.J: Merrill, 2009.

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My mother's house. Huntsville, Texas: Texas Review Press, 2016.

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Eggers, Christian. Schizophrenia and Youth. Springer, 1991.

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Youth With Juvenile Schizophrenia: The Search for Reality (Helping Youth With Mental, Physical, and Social Disabilities). Mason Crest Publishers, 2007.

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Eggers, Christian. Schizophrenia and Youth: Etiology and Therapeutic Consequences. Springer-Verlag, 1991.

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1938-, Eggers Christian, ed. Schizophrenia and youth: Etiology and therapeutic consequences. Berlin: Springer-Verlag, 1991.

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Ratcliff, Jason Stuart. Rites of Passage: My Schizophrenic Youth in Mosaic. Writers Club Press, 2002.

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Book chapters on the topic "Schizophrenics Schizophrenia Youth"

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Beckmann, H., H. Heinsen, S. Heckers, and H. Jakob. "Morphological Disturbances in the Brains of Schizophrenics." In Schizophrenia and Youth, 88–99. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7_9.

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Benedetti, G., and M. Peciccia. "New Insights in the Psychotherapy of Adolescent Schizophrenics." In Schizophrenia and Youth, 126–37. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7_12.

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Webb, Sala. "Schizophrenia." In Handbook of Mental Health in African American Youth, 249–59. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-25501-9_15.

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Ciompi, L. "Affect Logic and Schizophrenia." In Schizophrenia and Youth, 15–27. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7_3.

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Eggers, C. "Introduction." In Schizophrenia and Youth, 1–2. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7_1.

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Grüsser, O. J. "Impairment of Perception and Recognition of Faces, Facial Expression and Gestures in Schizophrenic Children and Adolescents." In Schizophrenia and Youth, 100–118. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7_10.

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Oades, R. D., and D. Zerbin. "Signs of Differential Stimulus Processing Problems: A Comment on Event-Related Potentials in Young Schizophrenic and Autistic Subjects." In Schizophrenia and Youth, 119–23. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7_11.

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Boscolo, L. "The Systemic Approach to the Therapy of Schizophrenia." In Schizophrenia and Youth, 140–45. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7_13.

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Cierpka, M., and P. Joraschky. "The Development of Boundary Disturbances in Families with a Schizophrenic Adolescent." In Schizophrenia and Youth, 146–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7_14.

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Wynne, L. C. "Systems Consultation for Psychosis: A Biopsychosocial Integration of Systemic and Psychoeducational Approaches." In Schizophrenia and Youth, 158–68. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-662-02684-7_15.

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