Relevant bibliographies by topics / Schmorl nodes

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters

Academic literature on the topic 'Schmorl nodes'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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Journal articles on the topic "Schmorl nodes"

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Dar, Gali, Smadar Peleg, Youssef Masharawi, Nili Steinberg, Hila May, and Israel Hershkovitz. "Demographical Aspects of Schmorl Nodes." Spine 34, no. 9 (April 2009): E312—E315. http://dx.doi.org/10.1097/brs.0b013e3181995fc5.

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Niwa, Naoya, Toru Nishiyama, Choichiro Ozu, Yasuto Yagi, and Shiro Saito. "Schmorl Nodes Mimicking Osteolytic Bone Metastases." Urology 86, no. 1 (July 2015): e1-e2. http://dx.doi.org/10.1016/j.urology.2015.03.028.

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Masala, Salvatore, Vincenzo Pipitone, Marco Tomassini, Francesco Massari, Andrea Romagnoli, and Giovanni Simonetti. "Percutaneous Vertebroplasty in Painful Schmorl Nodes." CardioVascular and Interventional Radiology 29, no. 1 (November 24, 2005): 97–101. http://dx.doi.org/10.1007/s00270-005-0153-6.

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Cheung, Kenneth M., Dino Samartzis, Florence P. S. Mok, Jaro Karppinen, Keith D. K. Luk, and Daniel Y. T. Fong. "Schmorl Nodes, Disc Degeneration, and Pain." Global Spine Journal 4, no. 1_suppl (May 2014): s—0034–1376758—s—0034–1376758. http://dx.doi.org/10.1055/s-0034-1376758.

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Pilet, Bernard, Rodrigo Salgado, Tony Van Havenbergh, and Paul M. Parizel. "Development of Acute Schmorl Nodes After Discography." Journal of Computer Assisted Tomography 33, no. 4 (July 2009): 597–600. http://dx.doi.org/10.1097/rct.0b013e318188598b.

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González-Reimers, Emilio, María Mas-Pascual, Matilde Arnay-de-la-Rosa, J. Velasco-Vázquez, and F. Santolaria-Fernández. "Schmorl Nodes: Lack of Relationship between Degenerative Changes and Osteopenia." Radiology 222, no. 1 (January 2002): 293–94. http://dx.doi.org/10.1148/radiol.2221011147.

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Coulier, B. "Giant fatty Schmorl?s nodes: CT findings in four patients." Skeletal Radiology 34, no. 1 (October 15, 2004): 29–34. http://dx.doi.org/10.1007/s00256-004-0858-7.

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Gao, Chang, Min Zong, Wen-tao Wang, Lei Xu, Da Cao, and Yue-fen Zou. "Analysis of risk factors causing short-term cement leakages and long-term complications after percutaneous kyphoplasty for osteoporotic vertebral compression fractures." Acta Radiologica 59, no. 5 (August 7, 2017): 577–85. http://dx.doi.org/10.1177/0284185117725368.

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Background Percutaneous kyphoplasty (PKP) is a common treatment modality for painful osteoporotic vertebral compression fractures (OVCFs). Pre- and postoperative identification of risk factors for cement leakage and follow-up complications would therefore be helpful but has not been systematically investigated. Purpose To evaluate pre- and postoperative risk factors for the occurrence of short-term cement leakages and long-term complications after PKP for OVCFs. Material and Methods A total of 283 vertebrae with PKP in 239 patients were investigated. Possible risk factors causing cement leakage and complications during follow-up periods were retrospectively assessed using multivariate analysis. Cement leakage in general, three fundamental leakage types, and complications during follow-up period were directly identified through postoperative computed tomography (CT). Results Generally, the presence of cortical disruption ( P = 0.001), large volume of cement ( P = 0.012), and low bone mineral density (BMD) ( P = 0.002) were three strong predictors for cement leakage. While the presence of intravertebral cleft and Schmorl nodes ( P = 0.045 and 0.025, respectively) were respectively identified as additional risk factors for paravertebral and intradiscal subtype of cortical (C-type) leakages. In terms of follow-up complications, occurrence of cortical leakage was a strong risk factor both for new VCFs ( P = 0.043) and for recompression ( P = 0.004). Conclusion The presence of cortical disruption, large volume of cement, and low BMD of treated level are general but strong predictors for cement leakage. The presence of intravertebral cleft and Schmorl nodes are additional risk factors for cortical leakage. During follow-up, the occurrence of C-type leakage is a strong risk factor, for both new VCFs and recompression.
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Luk, K., T. Law, M. Portia-Anthony, and D. Samartzis. "Assessment of the Neurocentral Synchondrosis in Pediatric Spines and the “Developmental” Etiology of Schmorl Nodes." Global Spine Journal 4, no. 1_suppl (May 2014): s—0034–1376576—s—0034–1376576. http://dx.doi.org/10.1055/s-0034-1376576.

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Ma, Heather Ting, James F. Griffith, Yixiang Wang, Anthony W. L. Kwok, Ping Chung Leung, Jason Leung, and David K. W. Yeung. "Is there any relationship between lumbar endplate changes (Modic changes, Schmorl nodes) and bone mineral density?" Bone 43 (October 2008): S66. http://dx.doi.org/10.1016/j.bone.2008.08.078.

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Dissertations / Theses on the topic "Schmorl nodes"

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McNaught, Janet ??? "A clinical and archaeological study of Schmorl’s nodes : using clinical data to understand the past." Thesis, Durham University, 2006. http://etheses.dur.ac.uk/2689/.

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This study of Schmorl 's nodes in British populations past and present attempts to reach a better understanding of the origins and aetiology of Schmorl' s nodes and their impact upon the vertebral column in relation to age, sex, pre-existing skeletal lower limb asymmetry, and work patterns. Georg Schmorl has been accepted as the leading authority on the nodes he named. Eighty years later modem diagnostic radiographic imaging and macroscopic studies are used to reach informed answers to the questions of prevalence, relationship to other degenerative joint diseases of the spine, and the possibility of Schmorl's node recognition by clinicians when presented with specific pain location. The clinical samples studied were from Pinderfields Hospital, Wakefield, and the Royal Hospital for Sick Children, Edinburgh. The archaeological samples studied were from Captain's Cabin, Dunbar, The Hirsel, Coldstream and Whithom Priory, Galloway, all in Scotland, and St. Andrew, Fishergate, York, and Tanners Row, Pontefract in England, and dated to between the 6th and 16th centuries AD. The archaeological samples were from military and monastic settlements in urban and rural contexts. Congenital disorders and acute trauma affecting the spine, and alterations to the normal primary and secondary vertebral curves were studied in an attempt to prove or disprove a relationship to Schmorl's nodes. Macroscopic study of the vertebral columns from the archaeological samples, and radiographs of the clinical samples, were analysed, along with clinical case histories and known background information of the demography, settlement type and, the geographic and topographic locations for each sample. No weak area was found in the vertebral end plate. Positive proof for symptomatic Schmorl's nodes was found in the clinical samples. The greatest numbers of Schmorl' s nodes were not formed in the first two decades of life, but in the third and fourth decades. Scheuermann's disease may be indirectly attributable to subadult Schmorl's nodes. However, Schmorl's nodes may or may not precede scoliosis. Males and females Of similar ages from the same settlements did not produce. Similar frequencies, nor were similar patterns observed between samples. Further studies using larger and better-preserved samples, with good contextual evidence may help to provide clearer positive or, negative evidence for work related Schmorl's nodes.
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Law, Tsz-kwun, and 羅子冠. "Ultrashort time-to-echo MRI of the cartilaginous endplate and relationship to disc degeneration and Schmorl's nodes, andretrospective study of paediatric spines and the neurocentralsynchondrosis." Thesis, The University of Hong Kong (Pokfulam, Hong Kong), 2011. http://hub.hku.hk/bib/B47869987.

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Background: An association between cartilaginous endplate (CEP) defects and intervertebral disc (IVD) degeneration has been previously suggested in animal and cadaveric studies. CEP defects may also be involved in Schmorl’s nodes (SN). There have been no previous reports in the literature that describe the use of ultrashort time-to-echo (UTE) MRI to assess the CEP in humans in vivo. In chapter 5 of this thesis, a retrospective study of paediatric spines and the neurocentral synchondrosis (NCS) was singled out to report the incidence of NCS and to raise the hypothesis of NCS as a precursor of SN. Purpose: To assess the feasibility of detecting CEP defects in live humans using UTE MRI, and to assess their relationship with IVD degeneration and SN. Subjects and Methods: A total number of 22 subjects underwent T2-weighted (T2W) and UTE MRI to assess for the presence and severity of IVD degeneration, the presence of SN and for the presence of CEP defects. SN and IVD degeneration were confirmed by assessing T2W images and IVD degeneration was graded according to the Schneiderman classification. CEP defects were defined as discontinuity of high signal over 4 consecutive images and were independently assessed by two raters. Results: Analyses of CEP defects between IVD degeneration and SN were performed separately. For the study of CEP defects and IVD degeneration, subjects with SN were excluded. 37 out of 108 (34.3%) CEPs had defects, which mainly occurred at T12/L1, L1/L2 and L4/L5 (p=0.008). Inter-rater reliability was substantial (Kappa statistic= 0.67, p<0.001). Multivariate logistic regression revealed that lower BMI (p=0.009) and younger (p=0.034) individuals had a decreased likelihood of having CEP defects. A statistically significant association was found to exist between the presence of cartilaginous endplate defects and intervertebral disc degeneration (p=0.036). Degenerated discs with CEP defects were found in L4/5 and L5/S1, while degenerated discs with no CEP defects were found throughout the whole lumbar region. Mean degeneration scores of L4/5 and L5/S1 levels with CEP defects were higher than that of L4/5 and L5/S1 levels without. For the study of CEP defects and SN, with all 22 subjects assessed, 125 out of 264 (47.3%) CEPs had defects. 40 SN were found, and among those, 35 SN had CEP defects (87.5%). 125 CEPs had the presence of CEP defects; among them, a large number of CEP defects did not have SN underneath (92 out of 125, 73.2%). Conclusion: The studies demonstrate the feasibility of using UTE MRI in live humans to assess the integrity of the CEP. Longitudinal studies may reveal the diagnosis of CEP defects to be clinically beneficial for assessment of IVD degeneration and SN.
published_or_final_version
Diagnostic Radiology
Master
Master of Philosophy
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Müllerová, Soňa. "Degenerativní změny kloubních spojení u velkomoravské populace z lokality Mikulčice - Valy." Master's thesis, 2017. http://www.nusl.cz/ntk/nusl-367676.

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This diploma thesis had two primary objectives. The first one was to examine if the incidence and development of primary degenerative joint disease (DJD) of the joints of the vertebral column and the limbs in an early medieval population from a cemetery by the sixth church in Mikulčice were affected mainly by the skeletal age of the deceased, or if other factor of physical loads was more significant. A set of 83 skeletal remains was evaluated primarily by Waldron's method (2009), but methods by Stloukal and Vyhnánek (1976); Buikstra and Ubelaker (1994); and Sager (1969) were also used. Schmorl nodes and eburnation were tracked too. Evaluated were vertebral joint (both intervertebral and apophyseal), jaw and limb joints. The results indicate that the incidence of degenerative joint disease in intervertebral joints, temporomandibular joint and appendicular joints is significantly affected by age, with the onset of degeneration being after the 40th year of skeletal age. No correlation of incidence of Schmorl nodes with age was present, and only a few cases of eburnace were noted. The relationship between the presence of DJD and the sex of the skeletons was not approved. I have compared these results with those from similar studies. I have deemed the primary evaluation by Waldron's method to be good...
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Book chapters on the topic "Schmorl nodes"

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"Schmorl Node." In Imaging in Neurology, 366. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-44781-2.50283-7.

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"Schmorl Node." In Imaging in Spine Surgery, 211. Elsevier, 2017. http://dx.doi.org/10.1016/b978-0-323-48554-8.50115-9.

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"Schmorl Node." In Diagnostic Imaging: Spine, 466–69. Elsevier, 2015. http://dx.doi.org/10.1016/b978-0-323-37705-8.50128-0.

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"Schmorl’s nodes." In Dictionary of Rheumatology, 194. Vienna: Springer Vienna, 2009. http://dx.doi.org/10.1007/978-3-211-79280-3_1010.

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Czervionke, Leo F. "Schmorl's Node." In Imaging Painful Spine Disorders - Expert Consult, 482–87. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4160-2904-5.00064-1.

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"Schmorl's Node." In Imaging of Pain, 87–88. Elsevier, 2011. http://dx.doi.org/10.1016/b978-1-4377-0906-3.00034-1.

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Weiss, Elizabeth. "Stress Fractures." In Reading the Bones. University Press of Florida, 2017. http://dx.doi.org/10.5744/florida/9780813054988.003.0005.

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Stress fractures (or fatigue fractures) are covered in this chapter. Stress fractures are assumed to occur due to microcracks from repetitive forces that accumulate to result in a complete bone break, but these fractures can result from a single traumatic event. The most common location for stress fractures is the vertebral column; two common vertebral stress fractures include spondylolysis and clay-shoveler’s fractures. Spondylolysis fractures, which occur on the lumbar vertebrae, are the most often reported stress fractures. Non-activity causes of stress fractures and the closely related stress hernias, called Schmorl’s nodes, have been found in twin studies; these fractures and hernias seem to be correlated with normal anatomical variation found in the vertebral column. These anatomical variations are likely determined by genes.
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Weiss, Elizabeth. "Conclusions." In Reading the Bones. University Press of Florida, 2017. http://dx.doi.org/10.5744/florida/9780813054988.003.0007.

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This chapter concludes with the major theme that ran through the previous chapters; how much of each of these skeletal activity reconstruction features are a result of environmental influences (i.e., activities) and how much of the variation in these features are a result of genes. Biological confounds, which are largely genetic, have been found in all of the skeletal features covered in the previous chapters. For example, evolutionary body type rules (i.e., Bergmann’s and Allen’s Rules) affect measures of cross-sectional geometries. Plus, age is known to increase entheseal change scores. Furthermore, twin studies have revealed hereditary etiologies for osteoarthritis and Schmorl’s nodes. Yet, not all of the variance is genetic and, thus, the question remains whether skeletal indicators of activity can still be used to reconstruct activity patterns. Methods that avoid circular reasoning and aim to use only skeletal features with predictive validity should be the ultimate goal for those studying skeletal remains. If skeletal indicators of activity cannot be used to reconstruct what people did in the past, then perhaps these skeletal features can help in other ways, such as improving age estimates or drawing better conclusions about biological relatedness.
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