Relevant bibliographies by topics / Secretory granula

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers
  6. Reports

Academic literature on the topic 'Secretory granula'

Author: Grafiati
Published: 4 June 2021
Last updated: 1 February 2022

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Journal articles on the topic "Secretory granula"

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von Zglinicki, Thomas, and Godfried M. Roomans. "X-Ray Microanalysis of the Intestine: Identification of Electrolyte Secreting Cells." Proceedings, annual meeting, Electron Microscopy Society of America 48, no. 2 (1990): 342–43. http://dx.doi.org/10.1017/s0424820100135319.

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Frozen dried cryosections of mouse jejunum were investigated. Besides Paneth and Goblet cells, two different types of crypt enterocytes could be distinguished according to clearly different electrolyte concentrations and to the presence (type A) or absence (type B) of small secretion granula in the cytoplasm.Secretion was stimulated by an intraperitoneal injection of either isoproterenol or pilocarpine. In some experiments, isoproterenol stimulation was blocked by alloxan, a potent inhibitor of the adenylate cyclase. Changes of cytoplasmic element concentrations were measured in frozen dried c
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Cossu, M., M. S. Lantini, and R. Puxeddu. "Immunocytochemical localization of Lewis blood group antigens in human salivary glands." Journal of Histochemistry & Cytochemistry 42, no. 8 (1994): 1135–42. http://dx.doi.org/10.1177/42.8.8027532.

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We demonstrated the immunohistochemical distribution of Le-a and Le-b blood group antigens in human major and minor salivary glands at the ultrastructural level by applying a post-embedding immunogold staining method. In secretors' glands, a faint Le-a reactivity was found only in mucous droplets, whereas Le-b antigen was intensely stained in secretory granules of most mucous cells, in those of intercalated duct cells, in the pale granular matrix of some serous cells, and, when osmication was omitted, in cytoplasmatic vesicles and cell surfaces of striated ducts. In the submandibular gland of
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Skott, O. "Do osmotic forces play a role in renin secretion?" American Journal of Physiology-Renal Physiology 255, no. 1 (1988): F1—F10. http://dx.doi.org/10.1152/ajprenal.1988.255.1.f1.

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Secretory granules swell during exocytosis. Swelling may follow fusion and assist in extrusion of the granular content, or swelling may cause granular fusion with the plasmalemma. A granular proton gradient has been suggested to be involved in such preexocytic granular swelling. Exocytosis of renin from juxtaglomerular cells of isolated preparations is very sensitive to changes in the extracellular osmolality. Extracellular hyposmolality causes swelling of secretory granules, fusions between peripherally located granules and plasmalemma, and an increased number of release episodes. Induction o
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Barg, Sebastian, Ping Huang, Lena Eliasson, et al. "Priming of insulin granules for exocytosis by granular Cl− uptake and acidification." Journal of Cell Science 114, no. 11 (2001): 2145–54. http://dx.doi.org/10.1242/jcs.114.11.2145.

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ATP-dependent priming of the secretory granules precedes Ca2+-regulated neuroendocrine secretion, but the exact nature of this reaction is not fully established in all secretory cell types. We have further investigated this reaction in the insulin-secreting pancreatic B-cell and demonstrate that granular acidification driven by a V-type H+-ATPase in the granular membrane is a decisive step in priming. This requires simultaneous Cl− uptake through granular ClC-3 Cl− channels. Accordingly, granule acidification and priming are inhibited by agents that prevent transgranular Cl− fluxes, such as 4,
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Kemter, Elisabeth, Andreas Müller, Martin Neukam, et al. "Sequential in vivo labeling of insulin secretory granule pools in INS-SNAP transgenic pigs." Proceedings of the National Academy of Sciences 118, no. 37 (2021): e2107665118. http://dx.doi.org/10.1073/pnas.2107665118.

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β cells produce, store, and secrete insulin upon elevated blood glucose levels. Insulin secretion is a highly regulated process. The probability for insulin secretory granules to undergo fusion with the plasma membrane or being degraded is correlated with their age. However, the molecular features and stimuli connected to this behavior have not yet been fully understood. Furthermore, our understanding of β cell function is mostly derived from studies of ex vivo isolated islets in rodent models. To overcome this translational gap and study insulin secretory granule turnover in vivo, we have gen
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Burgoyne, Robert D., and Alan Morgan. "Secretory Granule Exocytosis." Physiological Reviews 83, no. 2 (2003): 581–632. http://dx.doi.org/10.1152/physrev.00031.2002.

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Regulated exocytosis of secretory granules or dense-core granules has been examined in many well-characterized cell types including neurons, neuroendocrine, endocrine, exocrine, and hemopoietic cells and also in other less well-studied cell types. Secretory granule exocytosis occurs through mechanisms with many aspects in common with synaptic vesicle exocytosis and most likely uses the same basic protein components. Despite the widespread expression and conservation of a core exocytotic machinery, many variations occur in the control of secretory granule exocytosis that are related to the spec
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Tooze, John. "Secretory granule formation." Cell Biophysics 19, no. 1 (1991): 117–30. http://dx.doi.org/10.1007/bf02989885.

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Möhn, H., V. Le Cabec, S. Fischer, and I. Maridonneau-Parini. "The src-family protein-tyrosine kinase p59hck is located on the secretory granules in human neutrophils and translocates towards the phagosome during cell activation." Biochemical Journal 309, no. 2 (1995): 657–65. http://dx.doi.org/10.1042/bj3090657.

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The src-family protein-tyrosine kinase p59hck is mainly expressed in neutrophils; however, its functional role in these cells is unknown. Several other src-family members are localized on secretory vesicles and have been proposed to regulate intracellular traffic. We have established here the subcellular localization of p59hck in human neutrophils. Immunoblotting of subcellular fractions showed that approx. 60% of the p59hck per cell is localized on the secretory granules; the other 40% is distributed equally between non-granular membranes and the cytosol. Immunofluorescence of neutrophils and
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Alvarez de Toledo, G., and J. M. Fernandez. "Patch-clamp measurements reveal multimodal distribution of granule sizes in rat mast cells." Journal of Cell Biology 110, no. 4 (1990): 1033–39. http://dx.doi.org/10.1083/jcb.110.4.1033.

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Using patch-clamp techniques, we have followed the attributes of the secretory granules of peritoneal mast cells obtained from rats of different ages. The granule attributes were determined by following the step increases in the cell surface membrane area caused by the exocytosis of the granules in GTP gamma S stimulated mast cells. Our data show that the amount of granule membrane available for exocytosis depends exponentially on the weight (age) of the donor rat, reaching a maximum at approximately 300 g. The data are consistent with an exponential growth in the number of granules contained
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Sano, K., S. Waguri, N. Sato, E. Kominami, and Y. Uchiyama. "Coexistence of renin and cathepsin B in secretory granules of granular duct cells in male mouse submandibular gland." Journal of Histochemistry & Cytochemistry 41, no. 3 (1993): 433–38. http://dx.doi.org/10.1177/41.3.8429206.

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Cathepsin B, a representative lysosomal cysteine proteinase, has been demonstrated to coexist with renin in secretary granules of rat pituitary LH/FSH cells and renal juxtaglomerular cells. We investigated immunocytochemically the localization of cathepsins B, H, and L in the submandibular gland of male mice, in which active renin is also produced. By light microscopy, granular immunodeposits for cathepsin B were detected in epithelial cells of the gland, particularly in granular duct cells and interstitial cells. Immunoreactivity for cathepsins H and L was mainly found in interstitial cells,
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Dissertations / Theses on the topic "Secretory granula"

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Schubert, Sandra. "The Role of [beta]2-Syntrophin Phosphorylation in Secretory Granule Exocytosis." Doctoral thesis, Saechsische Landesbibliothek- Staats- und Universitaetsbibliothek Dresden, 2006. http://nbn-resolving.de/urn:nbn:de:swb:14-1146851994562-42414.

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The trafficking of insulin secretory granules(SGs) of pancreatic b-cells is a tightly controlled complex network. Increasing evidence indicates that the cortical actin cytoskeleton modulates the mobility and exocytosis of SGs,yet the mechanisms anchoring SGs to the cytoskeleton is not completely understood.It has been shown by Ort et al.(2000,2001) that the cytoplasmic tail of an intrinsic membrane protein of the SGs named ICA512/IA-2 binds the PDZ domain of b2-syntrophin,which in turn binds to the F-actin-binding protein utrophin. These data also indicate that stimulation of SG exocytosis aff
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Schubert, Sandra. "The Role of [beta]2-Syntrophin Phosphorylation in Secretory Granule Exocytosis." Doctoral thesis, Technische Universität Dresden, 2005. https://tud.qucosa.de/id/qucosa%3A23710.

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The trafficking of insulin secretory granules(SGs) of pancreatic b-cells is a tightly controlled complex network. Increasing evidence indicates that the cortical actin cytoskeleton modulates the mobility and exocytosis of SGs,yet the mechanisms anchoring SGs to the cytoskeleton is not completely understood.It has been shown by Ort et al.(2000,2001) that the cytoplasmic tail of an intrinsic membrane protein of the SGs named ICA512/IA-2 binds the PDZ domain of b2-syntrophin,which in turn binds to the F-actin-binding protein utrophin. These data also indicate that stimulation of SG exocytosis aff
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Guest, Paul C. "Insulin secretory granule biogenesis." Thesis, University of Cambridge, 1991. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.304963.

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Bennett, Deborah Louise. "Subtilisin-related proteases of the insulin secretory granule." Thesis, University of Cambridge, 1993. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.319496.

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Urbe, Sylvie. "Molecular mechanisms of secretory granule biogenesis in neuroendocrine cells." Thesis, University College London (University of London), 1998. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.286744.

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Ahras, Malika. "Synaptotagmin IV function in secretory granule maturation in neuroendocrine cells." Thesis, University College London (University of London), 2005. http://discovery.ucl.ac.uk/1445250/.

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In neuroendocrine cells, immature secretory granules (ISGs) bud from the trans Golgi network (TGN), and then undergo a maturation process that includes ISG-ISG homotypic fusion, removal of excess membranes via clathrin-coated vesicles (CCVs) and acidification of the granules. These mature secretory granules (MSGs) can then fuse with the plasma membrane upon receiving an external signal, and finally release their content in the extracellular space. In this thesis, I will present evidence that Synaptotagmin IV (Syt IV) is one of the components involved in the regulation of granule maturation in
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Wasmeier, Christina. "Molecular cloning of phogrin, a novel insulin secretory granule membrane protein." Thesis, University of Cambridge, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627383.

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Holroyd, Phillip. "Secretory granule exo-endocytosis studied in the neuroendocrine cell line PC12." Thesis, University of Bath, 2002. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.398430.

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Arden, Catherine. "Compartmentation and function of glucokinase in insulin secretory granules." Thesis, University of Newcastle Upon Tyne, 2004. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.407841.

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Pryde, James Grant. "Biogenesis of secretory granules in the bovine adrenal medulla." Thesis, University of Edinburgh, 1987. http://hdl.handle.net/1842/24238.

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Books on the topic "Secretory granula"

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Bacon, Mark. Como Hacer Marketing Directo: Secretos Para LA Pequena Empresa (Management (Granica)). Ediciones Granica, S.A., 1996.

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Oklopcic, Zoran. Nephos, Scopos, Algorithm. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198799092.003.0005.

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Chapter 5 moves beyond the two most politically consequential understandings of the right to self-determination: attributed to Demos and Ethnos respectively. While normative theorists are not sure how to evoke these figures, this chapter treats them as ensembles that are extracted from Nephos; an even fuzzier and more granular political ‘aerosol’. Against it as a backdrop, the discrete locations of territorial rights will also appear more fuzzified—not as identifiable locations, but rather as Scopos; visual effects of concealed, but nevertheless contestable scopic regimes. Once its holders and
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Book chapters on the topic "Secretory granula"

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Pavelka, Margit, and Jürgen Roth. "Secretory Granules." In Functional Ultrastructure. Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_45.

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Pavelka, Margit, and Jürgen Roth. "Secretory Granule Types." In Functional Ultrastructure. Springer Vienna, 2010. http://dx.doi.org/10.1007/978-3-211-99390-3_46.

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Tooze, Sharon A. "Maturation of Secretory Granules." In Molecular Mechanisms of Membrane Traffic. Springer Berlin Heidelberg, 1993. http://dx.doi.org/10.1007/978-3-662-02928-2_30.

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Fujita, Tsuneo, Tomio Kanno, and Shigeru Kobayashi. "Secretions and Secretory Granules." In The Paraneuron. Springer Japan, 1988. http://dx.doi.org/10.1007/978-4-431-68066-6_3.

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Kögel, Tanja, and Hans-Hermann Gerdes. "Maturation of Secretory Granules." In Results and Problems in Cell Differentiation. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/400_2009_31.

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Gerasimenko, Oleg. "Ca2+ Measurements in Secretory Granules." In Measuring Calcium and Calmodulin Inside and Outside Cells. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-56851-0_10.

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Hutton, J. C., M. Peshavaria, H. W. Davidson, K. Grimaldi, R. Pogge Von Strandmann, and K. Siddle. "The Insulin Secretory Granule: Features and Functions in Common with Other Endocrine Granules." In Advances in Experimental Medicine and Biology. Springer US, 1986. http://dx.doi.org/10.1007/978-1-4684-5314-0_36.

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Bowman, Grant R., Andrew T. Cowan, and Aaron P. Turkewitz. "Biogenesis of Dense-Core Secretory Granules." In Trafficking Inside Cells. Springer New York, 2009. http://dx.doi.org/10.1007/978-0-387-93877-6_10.

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Miwa, I., Y. Toyoda, and S. Yoshie. "Glucokinase in β-Cell Insulin-Secretory Granules." In Frontiers in Diabetes. KARGER, 2004. http://dx.doi.org/10.1159/000079029.

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Guest, Paul C. "Biogenesis of the Insulin Secretory Granule in Health and Disease." In Reviews on Biomarker Studies of Metabolic and Metabolism-Related Disorders. Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-12668-1_2.

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Conference papers on the topic "Secretory granula"

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morqenstern, E., and H. Patscheke. "THE SECRETORY PATHWAY IN PLATELETS STUDIED BY CRYO-FIXATION." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643491.

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It is widely held, that the constituents packed in the a -granules are released by stimulated platelets via the surface connected system (SCS). By means of the fast-freezing and freeze substitution technique (which allow the investigation of membrane fusion) we found a secretory pathway in platelets (compound exocytosis) without an involvement of the SCS during the release of a-granules. To study the process of a-granule secretion human platelets concentrated in citrated blood plasm were stimulated with thrombin or collagen. 20 - 120 seconds after stimulation the platelets were rapidly frozen
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Koike, Kazuo, and Holm Holmsen. "REQUIREMENT FOR THROMBIN RECEPTOR OCCUPANY DURING PLATELET SECRETION UNDER AGGREGATING CONDITIONS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644469.

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We have previously showed that hirudin abruptly arrests thrombin-induced secretion of acid hydrolase at any stage of its progress, whereas it only affects dense granule secretion only at its initial stages; these results have been interpreted to show that acid hydrolase secretion requires sustained while dense granule secreion ony requires a brief receptor occupancy (Holmsen et al. JBC 256(1981)9393). The requirement for receptor occupancy in thrombin-induced α-granule secretion and secretion during aggregation have been studied. Increasing concentrations of thrombin were added to gel-fitered
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Patscheke, H., and G. Mathieu. "MONITORING OF THE PLATELET ALPHA-GRANULE SECRETION IN THE AGGREGOMETER." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643492.

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If platelets are stimulated to secrete and the aggregation is prevented by EDTA and no stirring, the optical density (OD) decreases as a consequence of secretion (Patscheke et al. Thromb. Res. 33: 314, 1984). The purpose of this study was to determine which secretory compartment causes the change in OD and to analyze the quantitative relationship between decrease in OD and granule discharge. Human washed platelets were stimulated with thrombin and A 23187 in a Lumi-Aggregometer (Chrono-Log) which permitted simultaneous recording of the change in OD and of the ATP release from dense granules. A
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Malmgren, R. "LUMI-AGGREGOMETER STUDIES OF THE INITIAL ATP-SECRETION FROM COLLAGEN-ADHERENT PLATELETS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643550.

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We have earlier, with the use of a lumi-aggregometer and sub-aggregating doses of collagen (0.2-0.8 ug/ml PRP), been able to detect the initial, aspirin-insensitive secretion of ATP from the collagen-adherent platelets, and to correlate this secretion to the doses of collagen, and onset and degree of subsequent shape change of non-adherent platelets (Malmgren, Thromb Res 4:445, 1986). The present study shows, that 200 ATU of hirudin,which reduced near-maximal aggregation and ATP-secretion induced by high collagen doses (2.5 ug/ml PRP) from 3.35 ± 0.2 uM to 2.85 ± 0.1 uM, did neither reduce the
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Kingston, P. R., K. R. Bruckdorfer, and R. A. Hutton. "AGONIST INDUCED CALCIUM MOBILISATION IN PLATELETS OF PATIENTS WITH "ASPIRIN-LIKE" DEFECTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644570.

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A group of patients have been described with a condition often referred to as an'Aspirin-like" defect, which is characterised by easy bruising and prolonged bleeding following dental extraction or surgery. Initial studies eliminated a deficiency in coagulation factors, plasma factors, platelet glycoproteins or platelet storage granules as being the cause of this condition and demonstrated a diminished aggregatory and/or secretory response in platelet-rich-plasma to ADP and collagen. Aggregation responses in isolated platelets to thrombin, arachidonic acid and ionomycin are within the normal ra
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Blockmans, D., E. Van Houtte, J. Arnout, P. Mombaerts, D. Collen, and J. Vermylen. "TISSUE PLASMINOGEN ACTIVATOR INHIBITS THROMBIN-INDUCED AGGREGATION AND SHAPE CHANGE,BUT FACILITATES SECRETION,IN GEL-FILTERED PLATELETS ONLY." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1643763.

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Prolonged administration of tissue plasminogen activator (t-PA) has caused bleeding problems in some patients, that did not necessarily correlate with a significant drop of fibrinogen levels. We have therefore evaluated the effect of t-PA on platelet function in vitro.Incubation of gel-filtered platelets for one hour at 37°C with 180 μg/ml plasminogen and increasing concentrations of t-P(50-1600 ng/ml) significantly inhibited shapechange and aggregation induced by thrombinand the thromboxane mimetic U 46619 in a dose-dependent manner. In an EDTA milieu, whichbolishes aggregation, a dual effect
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Kajiwara, Y., M. Sakon, T. Tsujinaka, J. Kambayashi, T. Mori, and T. Murachi. "STUDIES ON ROLE OF CALPAIN IN PLATELET REACTION, UTILIZING NEWLY SYNTHETIZED PEPTIDE ANTAGONISTS." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642823.

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The system of calpain (Ca2+-activated protease) and its inhibitor calpastatin in platelets have been well characterized in our laboratory but the role of the system has not been fully elucidated yet, though various endogenous substrates have been identified. Employing SH inhibitors as calpain antagonists, We have proposed a possible role of platelet calpain in myosin light chain (20K) phosphorylation (Biochem.Int. 6,767,1986) but more specific calpain antagonists were required to draw conclusion. Recentry, series of peptide calpain antagonists(PCAs) were syn-thetized such as Ac-Leu-Leu-Nle.al(
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Nachman, R. L., R. L. Silverstein, and A. S. Asch. "THROMBOSPONDIN: CELL BIOLOGY OF AN ADHESIVE GLYCOPROTEIN." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644653.

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Thrombospondin (TSP), a multifunctional 450 KD glycoprotein is a secretory product of thrombin stimulated platelets. It is a major component of the platelets alpha granule constituting approximately 3% of total platelet protein. Thrombospondin does not circulate in appreciable concentrations ∽0 100 ng/ml); however, the tissue distribution is broad. In addition to its expression on the membrane of activated platelets, the protein is synthesized by fibroblasts endothelial cells, glial cell smooth muscle cells alveolar pneumocytes mononuclear phagocytes and various tumor cells. TSP is a major con
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Reports on the topic "Secretory granula"

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Bonilla-Mejía, Leonardo, and Erika Londoño-Ortega. Geographic Isolation and Learning in Rural Schools. Banco de la República, 2021. http://dx.doi.org/10.32468/be.1169.

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Rural schools are usually behind in terms of learning, and part of this could be related to geographical isolation. We explore this hypothesis, assessing the effect of distance between rural schools and local governments on learning in Colombia. We use spatial discontinuous regression models based on detailed administrative records from the education system and granular geographic information. Results indicate that distance to towns and Secretary of Education has significant negative effects on students’ standardized test scores. We evaluated alternative mechanisms, finding that the effect of
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