Relevant bibliographies by topics / Selective deficiency of IgA (sDIgA)

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  1. Journal articles
  2. Dissertations / Theses
  3. Book chapters
  4. Conference papers

Academic literature on the topic 'Selective deficiency of IgA (sDIgA)'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 February 2022

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Journal articles on the topic "Selective deficiency of IgA (sDIgA)"

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Cunningham-Rundles, C. "Selective IgA Deficiency." Journal of Pediatric Gastroenterology and Nutrition 7, no. 4 (July 1988): 482–83. http://dx.doi.org/10.1097/00005176-198807000-00002.

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Yel, Leman. "Selective IgA Deficiency." Journal of Clinical Immunology 30, no. 1 (January 2010): 10–16. http://dx.doi.org/10.1007/s10875-009-9357-x.

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Gambaro, Giovanni. "Primary Selective IgA Deficiency." Annals of Internal Medicine 120, no. 8 (April 15, 1994): 694. http://dx.doi.org/10.7326/0003-4819-120-8-199404150-00022.

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Rubinstein, Israel, Gerald L. Baum, and Yehuda Hiss. "Selective IgA Deficiency and Sarcoidosis." Chest 88, no. 1 (July 1985): 160. http://dx.doi.org/10.1378/chest.88.1.160-b.

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RAMSAHOYE, B. H., R. EVELY, W. MUMAR-BASHI, and S. H. LIM. "Selective IgA deficiency and hypoplenism." Clinical & Laboratory Haematology 16, no. 4 (June 28, 2008): 375–77. http://dx.doi.org/10.1111/j.1365-2257.1994.tb00438.x.

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Liblau, Roland S., and Jean-François Bach. "Selective IgA Deficiency and Autoimmunity." International Archives of Allergy and Immunology 99, no. 1 (1992): 16–27. http://dx.doi.org/10.1159/000236330.

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Catassi, C., A. Guerrieri, G. Natalini, F. Busco, and P. L. Giorgi. "Macroamylasaemia and selective IgA deficiency." Archives of Disease in Childhood 61, no. 7 (July 1, 1986): 704–6. http://dx.doi.org/10.1136/adc.61.7.704.

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Wang, Ning, Nan Shen, Timothy J. Vyse, Vidya Anand, Iva Gunnarson, Gunnar Sturfelt, Solbritt Rantapää-Dahlqvist, et al. "Selective IgA Deficiency in Autoimmune Diseases." Molecular Medicine 17, no. 11-12 (August 4, 2011): 1383–96. http://dx.doi.org/10.2119/molmed.2011.00195.

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Guaitolini, Bruna, Priscilla Santos, and Davisson Tavares. "Selective deficiency of IgA: case series." Residência Pediátrica 9, no. 1 (2019): 23–28. http://dx.doi.org/10.25060/residpediatr-2019.v9n1-03.

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Remolina López, A. J., C. Uribe Rueda, L. Patrucco, J. I. Rojas, and E. Cristiano. "Selective IgA deficiency and multiple sclerosis." Neurología (English Edition) 26, no. 6 (2011): 375–77. http://dx.doi.org/10.1016/s2173-5808(11)70087-6.

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Dissertations / Theses on the topic "Selective deficiency of IgA (sDIgA)"

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Schönhage, Kai Oliver. "Particle Gel Immuno Assay (ID-PaGIA) zum Nachweis von anti-IgA Antikörpern." Doctoral thesis, Humboldt-Universität zu Berlin, Medizinische Fakultät - Universitätsklinikum Charité, 2005. http://dx.doi.org/10.18452/15265.

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Anti-IgA Antikörper werden häufig als Ursache nicht-hämolytischer Transfusionsreaktionen angesehen. Die Inzidenz solcher Reaktionen schwankt zwischen 1:17.000 bis 1:770.000 und beruht größtenteils auf Fallberichten. Die Bedeutung dieser Antikörper, obwohl mit einer Prävalenz von 1: 18 bis 1:1.250 relativ häufig vorkommend, konnte in den circa vierzig Jahren seit ihrer Entdeckung nicht eindeutig geklärt werden; verschiedene Spezifitäten der Antikörper mit unterschiedlichen Reaktionen erschweren die Diagnose und eine klare Schematisierung. Ein Nachteil war bisher das Fehlen einer schnellen und unkomplizierten Nachweismethode, die in vielen Laboratorien angewandt werden kann. Die Ende der sechziger Jahre entwickelte Die Ende der 1960’er Jahre entwickelte Passive Hämagglutination (PHA) ist oft ungenau und unterliegt starken Schwankungen, kann aber relativ einfach durchgeführt werden und ist deshalb die Hauptmethode in der Diagnose von anti-IgA gewesen. Neuere und genauere Methoden wie Radio Immuno Assay (RIA) und Enzyme Linked Immunosorbent Assay (ELISA) sind weder schnell durchzuführen noch in vielen Laboratorien verfügbar. In dieser Arbeit wird eine neue Agglutinationsmethode, Particle Gel Immuno Assay (PaGIA) evaluiert und mit der PHA verglichen. Im ersten Teil wurden die Seren 105 gesunder Spender untersucht: 70 führten zu Reaktionen im PHA mit Titern bis 1:80 während keines im PaGIA reagierte, was die Spezifität des PaGIA unterlegt. Anschließend wurden elf Seren von Patienten mit selektivem IgA Mangel (sDIgA) und 23 Seren von Patienten mit variablem Immundefektsyndrom (CVID) auf das Vorliegen eines anti-IgA Antikörpers untersucht. Fünf Seren beider Patientengruppen führten in beiden Tests zu Agglutinationen und ein Serum (sDIgA) reagierte mit einem Titer von 1:1 in der PHA aber nicht im PaGIA. Die hier gefundenen Prävalenz (22% sDIgA, 45% CVID) und Größe der Titer (sDIgA>CVID) von anti-IgA stimmt mit den bisherigen Erkenntnissen überein. Weitere Untersuchungen konnten die Stabilität des PaGIA bzw. dessen Beads und Reproduzierbarkeit der Ergebnisse über mehrere Monate als auch die Möglichkeit subklassenspezifisches anti-IgA nachzuweisen darlegen. Der PaGIA stellt einen schnell und einfach durchzuführenden Test dar, mit dem anti-IgA Antikörper verschiedener Spezifität verläßlich bestimmt werden können, um Untersuchungen im großen Rahmen durchzuführen, die die Bedeutung der anti-IgA Antikörpern erhellen.
Anti-IgA antibodies are thought to be responsible for non-hemolytic transfusion reactions in one in 17,000 to one in 770,000 number of cases. This incidence is mainly supported by case reports. Despite their relative frequency of one in 18 to one in 1,250, since their discovery approximately forty years ago, the true significance of these antibodies has not yet been determined. Several specificities of these antibodies resulting in different reaction patterns make diagnosis and categorization difficult. Until recently, the lack of a fast and reliable laboratory test was a drawback. This test needed to be easily performed, fast, accurate, reproducible and accessible to many practitioners in many laboratories. The Passive Hemagglutination Assay (PHA), developed in the late 1960’s, is neither precise nor reliable but easy to perform and therefore has been the mainstay in diagnosis of anti-IgA. While newer methods, such as Radio Immuno Assay (RIA) and Enzyme Linked Immunosorbent Assay (ELISA), are neither fast nor easily performed but very precise. This thesis studies and evaluates a new agglutination assay, the Particle Gel Immuno Assay (PaGIA), and compares it to the PHA. In the first part of our study we established the specificity of PaGIA. Sera of 105 healthy blood donors were tested: 70 led to positive reactions with the PHA with titers up to 1:80 while none reacted with the PaGIA. Subsequently, eleven sera of patients with selective deficiency of IgA (sDIgA) and 23 sera of those with Common Variable Immunodeficiency (CVID) were tested for the presence of anti-IgA antibodies. Five sera in each group led to agglutinations in both assays and one serum reacted with a titer of 1:1 in the PHA but not in the PaGIA. The prevalence (22% sDIgA, 45% CVID) and strength of the titers (sDIgA>CVID) of anti-IgA corresponds with current knowledge. Further tests demonstrated the PaGIA’s and its beads stability and reproducibility over several months as well as the possibility for detection of subclass-specific anti-IgA. The PaGIA is a fast and easily performed assay which reliably detects anti-IgA antibodies of different specificities, thereby providing a tool for large scale studies to shed more light on the significance of anti-IgA antibodies.
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BAN, NOBUTARO, MASAHIRO YAMAMURA, JUICHI SATO, TOMIO SUZUKI, NAOZUMI HASHIMOTO, TAKAFUMI ANDO, MOTOKI SATO, et al. "Selective IgA Deficiency Mimicking Churg-Strauss Syndrome and Hypereosinophilic Syndrome: A Case Report." Nagoya University School of Medicine, 2013. http://hdl.handle.net/2237/17611.

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Klartag, Ayelet. "Acquired selective IgA deficiency induced by dietary bovine IgA." 2007. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.16612.

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Book chapters on the topic "Selective deficiency of IgA (sDIgA)"

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Leung, Alexander K. C., Cham Pion Kao, Andrew L. Wong, Alexander K. C. Leung, Thomas Kolter, Ute Schepers, Konrad Sandhoff, et al. "Selective IgA Deficiency." In Encyclopedia of Molecular Mechanisms of Disease, 1912–13. Berlin, Heidelberg: Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_929.

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Sofia Appelberg, K., Hassan Abolhassani, and Lennart Hammarström. "Selective IgA Deficiency." In Humoral Primary Immunodeficiencies, 201–15. Cham: Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-91785-6_16.

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Agarwal, Shradha. "Selective IgA Deficiency." In Allergy and Clinical Immunology, 348–54. Chichester, UK: John Wiley & Sons, Ltd, 2015. http://dx.doi.org/10.1002/9781118609125.ch41.

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Gershwin, Laurel J. "Case 6: Selective IgA Deficiency." In Case Studies in Veterinary Immunology, 23–26. New York, NY : Garland Science, [2017]: Garland Science, 2017. http://dx.doi.org/10.4324/9781315165462-6.

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Norhagen, G., P. E. Engström, L. Hammarström, M. Jonsson, and C. I. E. Smith. "IgM and IgG levels in serum and saliva do not correlate to susceptibility of upper respiratory tract infections of HLA in individuals with selective IgA deficiency." In Advances in Mucosal Immunology, 502–3. Dordrecht: Springer Netherlands, 1990. http://dx.doi.org/10.1007/978-94-009-1848-1_150.

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"Deficiency of IgA, selective." In Dictionary of Rheumatology, 54. Vienna: Springer Vienna, 2009. http://dx.doi.org/10.1007/978-3-211-79280-3_299.

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Spickett, Gavin P. "Primary immunodeficiency." In Oxford Handbook of Clinical Immunology and Allergy, 1–80. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199603244.003.0001.

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Introduction Classification of immunodeficiency Clinical features of immunodeficiency Investigation of immunodeficiency Laboratory investigation Major B-lymphocyte disorders Rare antibody deficiency syndromes X-linked agammaglobulinaemia (Bruton’s disease) Common variable immunodeficiency (CVID) CVID 2: complications and treatment Selective IgA deficiency IgG subclass deficiency Specific antibody deficiency with normal serum immunoglobulins...
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Conference papers on the topic "Selective deficiency of IgA (sDIgA)"

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Akbiek, M., A. Hadeh, A. Daya, A. Gandhi, and K. Patel. "Selective IgA Deficiency and Bronchiectasis." In American Thoracic Society 2019 International Conference, May 17-22, 2019 - Dallas, TX. American Thoracic Society, 2019. http://dx.doi.org/10.1164/ajrccm-conference.2019.199.1_meetingabstracts.a6389.

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Toti, F., A. Stierlé, M. L. Wiesel, A. Schwartz, J. M. Freyssinet, and J. P. Cazenave. "PRODUCTION OF ANTIBODIES TO HUMAN VON WILLEBRAND FACTOR IN LAYING HENS. ISOLATION OF IMMUNOGLOBULINS AND APPLICATIONS TO THE DETECTION OF MOLECULAR DEFECTS OF VON WILLEBRAND FACTOR." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644084.

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Von Willebrand disease (vWD) is an inherited disorder of primary hemostasis caused by deficiency or structural abnormalities of von Willebrand factor (vWF). VWF circulates in plasma and is also present in platelets. Plasma vWF, the carrier protein for factor VIII, is a large multimeric glycoprotein composed of identical subunits linked by disulfide bridges. Plasma and platelet vWF display distinct multimeric electrophoretic patterns. The different vWD subtypes can be classified either by the determination of vWFantigen (vWFíAg) and/or by multimer distribution. Antibodies to human vWF were raised in laying hens by intramuscular injections of purified human vWF. Immunoglobulins were isolated from egg yolks by selective polyethylene glycol and ammonium sulfate precipitations. These antibodies appeared to be monospecific, as they did not react with the plasma proteins of a patient with severe vWD. The pullets received weekly 50 μg vWF for 4 weeks and then had monthly injections. The antibodies occurred as early as the third injection, the yield being 300 to 500 mg of immunoglobulin per week (6-7 eggs). The titre could be constant over periods greater than 1 year. These immunoglobulins to vWF were tested in vWFíAg electroimmunoassays and for the multimer analysis of plasma and platelet vWF by electrophoresis and immunoblotting techniques. In no case could a difference be detected between assays performed with rabbit monospecific antiserum or with yolk immunoglobulins to human vWF. Ten to 12 multimers could be revealed for normal plasma vWF and up to 12 to 14 bands for normal platelet vWF (1.7% agarose). In the case of vWD, the electrophoresis patterns were identical with both antibodies. Thus, antibodies to vWF raised in laying hens are a suitable tool to detect and to characterize vWD. Although they do not interact with protein A, yolk antibodies are certainly advantageous to produce, as they do not contain IgM or IgA. Immunoglobulin fractions can contain up to 10 % of specific antibodies. Since they are available in larger quantities and are easy to isolate, larger homogeneous batches of antibodies can be obtained. This method may easily be applied to develop antibodies to a variety of antigens.
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