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Books on the topic 'Septic shock'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 July 2024

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1

Vincent, Jean Louis, and Lambertus G. Thijs, eds. Septic Shock. Berlin, Heidelberg: Springer Berlin Heidelberg, 1987. http://dx.doi.org/10.1007/978-3-642-83108-9.

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2

K, Root Richard, Sande Merle A. 1939-, and University of California, San Francisco. Dept. of Medicine. Continuing Medical Education Division., eds. Septic shock. New York: Churchill Livingstone, 1985.

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3

J, Sibbald William, and Sprung Charles L, eds. Perspectives on sepsis and septic shock. Fullerton, Calif: Society of Critical Care Medicine, 1986.

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4

Rietschel, E. T., and Hermann Wagner, eds. Pathology of Septic Shock. Berlin, Heidelberg: Springer Berlin Heidelberg, 1996. http://dx.doi.org/10.1007/978-3-642-80186-0.

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5

L, Vincent J., and Thijs L. G. 1938-, eds. Septic shock: European view. Berlin: Springer-Verlag, 1987.

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6

Eichacker, Peter Q., and Jérôme Pugin, eds. Evolving Concepts in Sepsis and Septic Shock. Boston, MA: Springer US, 2001. http://dx.doi.org/10.1007/978-1-4615-1581-4.

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7

Redl, Heinz, and Günther Schlag, eds. Cytokines in Severe Sepsis and Septic Shock. Basel: Birkhäuser Basel, 1999. http://dx.doi.org/10.1007/978-3-0348-8755-7.

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8

Heinz, Redl, and Schlag Günther, eds. Cytokines in severe sepsis and septic shock. Basel: Birkhäuser Verlag, 1998.

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9

Evans, Thomas J. Septic Shock Methods and Protocols. New Jersey: Humana Press, 1999. http://dx.doi.org/10.1385/1592592163.

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10

Sakorafas, George H. Septic shock: Current pathogenetic concepts, optimal management, and future perspectives. New York: Nova Biomedical Books, 2004.

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11

1945-, Proctor R. A., ed. Clinical aspects of endotoxin shock. Amsterdam: Elsevier, 1986.

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12

A, Neugebauer Edmund, and Holaday John W, eds. Handbook of mediators in septic shock. Boca Raton: CRC Press, 1993.

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13

K, Reinhart, Eyrich K, and International Steglitz Symposium (2nd : 1987 : Berlin, Germany), eds. Sepsis: An interdisciplinary challenge. Berlin: Springer-Verlag, 1989.

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14

Hans, Cottier, and Kraft R, eds. Gut-derived infectious-toxic shock (GITS): A major variant of septic shock. Basel: Karger, 1992.

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15

Bergdoll, Merlin S. Toxic shock syndrome. Boca Raton: CRC Press, 1991.

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16

C, Reithmann, and Werdan Karl, eds. Cytokines and the heart: Molecular mechanisms of septic cardiomyopathy. New York: Springer, 1996.

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17

J, Jackson Simon, ed. Endotoxins and septic shock: The antibiotic connection. Newbury: Bayer, 1995.

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18

Mittag, Hans-Christian. Toxic shock syndrome and the other staphylococcal toxicoses. Stuttgart: New York, 1988.

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19

Mittag, Hans-Christian. Toxic shock syndrome and the other staphylococcal toxicoses. Stuttgart: Schattauer, 1988.

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20

Wiggers Bernard Conference (2nd 1990 Dürnstein, Austria). Shock, sepsis, and organ failure: Second Wiggers Bernard Conference, May 27-30, 1990, Schloss Dürnstein. Berlin: Springer-Verlag, 1991.

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21

L, Roth Bryan, Nielsen Thor B, and Liss Alan R, eds. Molecular and cellular mechanisms of septic shock: Proceedings of a conference held in Bethesda, Maryland, February 29 - March 1, 1988. New York: Alan R. Liss, 1988.

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22

European Conference on Toxic Shock Syndrome (1997 London). European conference on toxic shock syndrome: Proceedings of a symposium sponsored by Procter & Gamble Ltd. ... [et al.], held at the Royal Society of Medicine in London, 10-12 September 1997. London: Royal Society of Medicine Press, 1998.

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23

M, Tellado J., Forse R. Armour, and Solomkin J. S, eds. Modulation of the inflammatory response in severe sepsis. Basel: Karger, 1995.

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24

Müller-Werdan, Ursula. Cytokines and the heart: Molecular mechanisms of septic cardiomyopathy. New York: Springer, 1996.

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25

Wiggers Bernard Conference (1st 1989 Schloss Fuschl, Austria). Shock, sepsis, and organ failure. Berlin: Springer-Verlag, 1990.

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26

L, Roth Bryan, Nielsen Thor B, and McKee Adam E, eds. Molecular and cellular mechanisms of septic shock: Proceedings of a conference held in Bethesda, Maryland, February 29-March 1, 1988. New York: Liss, 1989.

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27

Jordi, Rello, and Restrepo Marcos I, eds. Sepsis: New strategies for management. Berlin: Springer, 2008.

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28

Wiggers Bernard Conference (4th 1994 Krumbach, Austria). Shock, sepsis, and organ failure--nitric oxide: Fourth Wiggers Bernard Conference 1994. Berlin: Springer-Verlag, 1995.

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29

1932-, Levin Jack, and International Endotoxin Society Congress, eds. Endotoxin and sepsis: Molecular mechanisms of pathogenesis, host resistance, and therapy. New York: Wiley-Liss, 1998.

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30

Artero1, Arturo, Marilena Galdiero1, Jolanta Korsak, and Sebastian Lucas. Sepsis and Septic Shock. DI Press, 2022.

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31

Litell, John M., and Nathan I. Shapiro. Pathophysiology of septic shock. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0297.

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The pathophysiology of sepsis is the result of a dysregulated host response to infection. Interactions between conserved pathogenic signals and host recognition systems initiate a systemic reaction to local infection. Pro- and anti-inflammatory intermediates and associated coagulatory abnormalities lead to altered macrovascular, microvascular, and mitochondrial function. Uncorrected, these processes yield similar patterns of failure in multiple organ systems. Mortality increases with successive organ failures. Although commonly thought to be a manifestation of impaired renal circulation, septic acute kidney injury may be due primarily to non-haemodynamic factors. Pulmonary parenchymal dysfunction in sepsis also contributes to failures in other organ systems. Sepsis involves complex alterations in myocardial function, vascular tone, and capillary integrity, which are mediated by elevated concentrations of inflammatory cytokines, inducible nitric oxide, and reactive oxygen species, among others. Gut hypomotility and translocation of enteric flora likely contribute to a persistent inflammatory response. This perpetuates the pathophysiological pattern of sepsis, and can lead to the delayed onset of these features in patients with other types of critical illness. The neurological manifestations of sepsis include acquired delirium, which is also probably due to circulatory and inflammatory abnormalities, as well as alterations in cerebral amino acid metabolism. Critical illness-related corticosteroid insufficiency and derangements in glucose metabolism are among the endocrine abnormalities commonly seen in septic patients. Restoration of homeostasis requires early haemodynamic resuscitation and aggressive infectious source control.

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32

Septic Shock and Awe. Blurb, 2017.

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33

Septic Shock: European View. Springer, 2011.

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34

Wagner, Hermann, and Ernst T. Rietschel. Pathology of Septic Shock. Springer London, Limited, 2011.

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35

Wagner, Hermann, and Ernst T. Rietschel. Pathology of Septic Shock. Springer London, Limited, 2012.

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36

Wise, Matt, and Paul Frost. ICU treatment of sepsis and septic shock. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0152.

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Bacteria are the most frequent causes of severe sepsis and septic shock, while viruses, fungi, and parasites are implicated less often. Positive cultures are found in only 60% of cases; this may be the result of previous antibiotic therapy or inadequate sampling or testing. The etiology of sepsis is constantly changing; whereas Gram-negative organisms used to make up the majority of cases, Gram-positive bacteria now predominate. Sepsis due to fungal disease has also seen a dramatic rise. These changes may be explained by alterations in patient demographics, such as an increasingly elderly population with multiple comorbidities; an increased frequency of indwelling catheters or devices; and greater numbers of patients with immunosuppression as a result of disease or drug therapy. This chapter covers symptoms, demographics, diagnosis, investigation, prognosis, and treatment within the ITU environment.

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37

Cytokines in Severe Sepsis and Septic Shock. Birkhauser Verlag, 2013.

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38

Evolving Concepts in Sepsis and Septic Shock. Springer, 2011.

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39

Redl, H. Cytokines in Severe Sepsis and Septic Shock. Birkhauser, 2012.

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40

Eichacker, Peter Q., and Jérôme Pugin. Evolving Concepts in Sepsis and Septic Shock. Springer London, Limited, 2012.

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41

Eichacker, Peter Q., and Jerome Pugin. Evolving Concepts in Sepsis and Septic Shock. Springer, 2012.

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42

Optimizing Antimicrobial Therapy Of Sepsis And Septic Shock. W.B. Saunders Company, 2010.

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43

J, Evans Thomas. Septic Shock Methods and Protocols. Humana Press, 2010.

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44

Septic Shock (Critical Care Management). HARCOURT PUBLISHERS LIMITED, 2000.

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45

Septic Shock Methods and Protocols. Humana Press, 2000.

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46

Sakorafas, George H. Septic Shock: Current Pathogenetic Concepts, Optimal Management, And Future Perspectives (Nova Biomedical). Nova Biomedical Books, 2006.

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47

Nichols, Dane. Optimizing Hemodynamic Support in Severe Sepsis and Septic Shock. Elsevier - Health Sciences Division, 2010.

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48

Fernandez, Ricardo, ed. Severe Sepsis and Septic Shock - Understanding a Serious Killer. InTech, 2012. http://dx.doi.org/10.5772/1311.

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49

Eyrich, K., and K. Reinhart. Sepsis: An Interdisciplinary Challenge. Springer, 2011.

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50

Reinhart, K. Sepsis: An Interdisciplinary Challenge. Springer, 1989.

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