Academic literature on the topic 'Serdab'

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Journal articles on the topic "Serdab"

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Grover, A. K., S. E. Samson, and C. M. Misquitta. "Sarco(endo)plasmic reticulum Ca2+ pump isoform SERCA3 is more resistant than SERCA2b to peroxide." American Journal of Physiology-Cell Physiology 273, no. 2 (August 1, 1997): C420—C425. http://dx.doi.org/10.1152/ajpcell.1997.273.2.c420.

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Sarco(endo)plasmic reticulum Ca2+ pumps are encoded by genes SERCA1, SERCA2, and SERCA3. Most tissues express SERCA2 Ca2+ pumps (splice SERCA2b) which are inactivated by reactive oxygen. In contrast, SERCA3 is expressed in tissues such as tracheal epithelium, mast cells, lymphoid cells, and aortic endothelium, which are frequently exposed to oxidative stress. Therefore, we compared SERCA3 and SERCA2b proteins for their sensitivity to oxidation. We isolated microsomes from HEK-293 cells overexpressing SERCA3 or SERCA2b. We incubated the microsomes with different concentrations of hydrogen peroxide and then determined Ca2+ pump activities in them in the following three assay systems: ATP-dependent oxalate-stimulated azide-insensitive 45Ca2+ uptake by the microsomal vesicles, Ca(2+)-Mg(2+)-ATPase, and Ca(2+)-dependent acylphosphate formation. Peroxide inhibited the pump activities in microsomes with half-maximal inhibitory concentration (IC50) values of 69 +/- 14, 66 +/- 13, and 84 +/- 15 microM for the 45Ca2+ uptake, Ca(2+)-Mg(2+)-ATPase, and the acylphosphate formation reactions, respectively. However, for microsomes from SERCA3-expressing cells, the corresponding values of IC50 for peroxide were 274 +/- 47, 857 +/- 110, and 746 +/- 40 microM. Thus, in each assay system, the resistance to inactivation by peroxide was significantly (P < 0.05) higher for the SERCA3 protein than for SERCA2b. The SERCA3 resistance to oxidants may aid the cells expressing it to function during exposure to oxidative stress.
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Simons, Frank. "Innovation in Serdab Decoration in the Late Sixth Dynasty." Journal of Egyptian Archaeology 102, no. 1 (January 2016): 196–203. http://dx.doi.org/10.1177/030751331610200115.

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Baines, John, and Christina Riggs. "Archaism and Kingship: A Late Royal Statue and its Early Dynastic Model." Journal of Egyptian Archaeology 87, no. 1 (December 2001): 103–18. http://dx.doi.org/10.1177/030751330108700110.

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Publication of British Museum EA 941, a Late Period or early Ptolemaic royal statue in travertine whose model is the Early Dynastic statue of Djoser (Cairo JE 49158) from the serdab on the north side of his Step Pyramid, or another statue of the same type. We present the British Museum statue, compare it with the Djoser statue, and argue for the former's likely dating and Saqqara provenance. Both statues are significant for their iconography of divine kingship and mortuary transfiguration.
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Chandrasekera, P. Charukeshi, Margaret E. Kargacin, Julie P. Deans, and Jonathan Lytton. "Determination of apparent calcium affinity for endogenously expressed human sarco(endo)plasmic reticulum calcium-ATPase isoform SERCA3." American Journal of Physiology-Cell Physiology 296, no. 5 (May 2009): C1105—C1114. http://dx.doi.org/10.1152/ajpcell.00650.2008.

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The sarco(endo)plasmic reticulum Ca2+-ATPases (SERCAs) play a crucial role in regulating free cytosolic Ca2+ concentration in diverse cell types. It has been shown that recombinant SERCA3, when measured in heterologous systems, exhibits low apparent affinity for Ca2+; however, Ca2+ affinity of native SERCA3 in an endogenous setting has not been examined. Such a measurement is complicated, because SERCA3 is always coexpressed with the housekeeping isoform SERCA2b. We used a fluorescence-based assay for monitoring continuous Ca2+ uptake into microsomes to examine the properties of endogenous human SERCA3 and SERCA2b. The kinetic parameters were derived using a cooperative two-component uptake model for Ca2+ activation, and the values assigned to SERCA3 were confirmed using the highly specific human SERCA3 inhibitory antibody PL/IM430. First, using recombinant human SERCA3 and SERCA2b proteins transiently expressed in HEK-293 cells, we confirmed the previously observed low apparent Ca2+ affinity for SERCA3 compared with SERCA2b (1.10 ± 0.04 vs. 0.26 ± 0.01 μM), and using mixtures of recombinant protein isoforms, we validated the two-component uptake model. Then we determined apparent Ca2+ affinity for SERCA proteins present endogenously in cultured Jurkat T lymphocytes and freshly isolated human tonsil lymphocytes. The apparent Ca2+ affinity in these two preparations was 1.04 ± 0.07 and 1.1 ± 0.2 μM for SERCA3 and 0.27 ± 0.02 and 0.26 ± 0.01 μM for SERCA2b, respectively. Our data demonstrate, for the first time, that affinity for Ca2+ is inherently lower for SERCA3 expressed in situ than for other SERCA isoforms.
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Pacifico, F., L. Ulianich, S. De Micheli, S. Treglia, A. Leonardi, P. Vito, S. Formisano, E. Consiglio, and B. Di Jeso. "The expression of the sarco/endoplasmic reticulum Ca2+-ATPases in thyroid and its down-regulation following neoplastic transformation." Journal of Molecular Endocrinology 30, no. 3 (June 1, 2003): 399–409. http://dx.doi.org/10.1677/jme.0.0300399.

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Maintaining a high Ca(2+) concentration in the lumen of the endoplasmic reticulum (ER), by the action of sarco/endoplasmic reticulum Ca(2+)-ATPases (SERCAs), is important in many cellular processes, such as Ca(2+)-mediated cytosolic signaling in response to extracellular stimuli, cell growth and proliferation, and synthesis, processing and folding of ER-translated proteins. In the thyroid gland, SERCAs have not been studied yet, and there is little information available on general problems such as the expression of SERCAs following neoplastic transformation. In this study we investigated the expression of SERCA2b and SERCA3 in rat thyroid tIssue and, in addition, in normal and transformed rat thyroid cell lines. RT-PCR and Northern blot assays showed that SERCA2b is the SERCA form preferentially expressed in the thyroid. In rat thyroid, SERCA2b mRNA was expressed at a higher level than that of other non-muscle tIssues such as liver or spleen, but at much lower level than in brain. On the other hand, SERCA3 mRNA was not detected in thyroid by Northern blot analysis, or barely detected by RT-PCR assays. We also studied the SERCA2b expression pattern in PC Cl3 thyroid cells transformed by several oncogenes that induce different degrees of malignancy and dedifferentiation. RT-PCR and Northern blot assays showed that SERCA2b mRNA expression dramatically decreased in highly tumorigenic thyroid cells, while expression of glyceraldehyde-3-phosphate dehydrogenase mRNA, a housekeeping gene used as internal control, exhibited no variations. The dramatic down-regulation of SERCA2b expression in fully transformed thyroid cells was also evident by Western blot analysis. Also, following neoplastic transformation of thyroid cells, the enzymatic activity of SERCA2b was reduced in a measure which correlated with the mRNA and protein levels. Therefore, rat thyrocytes expressed intermediate levels of SERCAs, mostly the SERCA2b isoform. This pattern of expression was basically reproduced in fully differentiated thyroid cells in culture and was sensitive to neoplastic transformation.
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LACABARATZ-PORRET, Christine, Sophie LAUNAY, Elisabeth CORVAZIER, Raymonde BREDOUX, Béla PAPP, and Jocelyne ENOUF. "Biogenesis of endoplasmic reticulum proteins involved in Ca2+ signalling during megakaryocytic differentiation: an in vitro study." Biochemical Journal 350, no. 3 (September 8, 2000): 723–34. http://dx.doi.org/10.1042/bj3500723.

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The endoplasmic reticulum (ER) plays a key role in Ca2+ signalling through Ca2+ release via inositol 1,4,5-trisphosphate receptors (InsP3-Rs) and Ca2+ uptake by sarco/endoplasmic reticulum Ca2+-ATPases (SERCAs). Here, we investigated the organization of platelet ER and its biogenesis during megakaryocytopoiesis. First, erythro/megakaryoblastic MEG 01, UT7, M-O7e and CHRF 288-11 cell lines, platelets and thrombopoietin-induced UT7-Mpl cells were selected for the study of SERCA2b and SERCA3 proteins by Western blotting using the antibodies IID8 and PL/IM430, respectively. As judged by platelet glycoprotein IIIa (GPIIIa) expression, an increase in SERCA3 proteins was observed while that of SERCA2b remained unchanged throughout maturation. Second, these studies were extended to the newly described alternatively spliced SERCA3a–c RNAs and InsP3-Rs using the in vitro model of PMA-induced differentiation of MEG 01 cells. Time-course and dose–response studies showed a maximal approx. 4-fold up-regulation of SERCA3 proteins using 10-8 M PMA for 3 days, which paralleled induction of GPIIIa expression. SERCA3 induction was found to occur at the level of mRNA. The modulation of the different SERCA3 species (i.e. 3a, 3b and 3c) was isoform-specific: while SERCA3a was slightly increased, an approx. 3-fold induction of SERCA3b, and a 4-fold induction of SERCA3c, was observed after 24h of PMA treatment. Isoform-specific Western blotting and/or reverse transcriptase PCR studies showed that InsP3-R types I, II and III are expressed in MEG 01 cells, as well as in platelets. Study of the expression of these InsP3-R types in PMA-induced MEG 01 cells revealed that: (i) InsP3-RI protein and mRNA showed no changes; (ii) InsP3-RII mRNA was up-regulated and peaked at hour 48 and (iii) InsP3-RIII mRNA and protein showed a transitory maximal 3- and 2.3-fold increase at hours 6 and 30, respectively. Upon PMA treatment of CHRF 288-11 cells, in which GPIIIa is not induced upon treatment, a similar pattern of regulation of InsP3-R types II and III was seen, but a distinct pattern of SERCA3 regulation was observed. These results suggest a profound reorganization of ER-protein patterns during megakaryocytopoiesis and underline the role of SERCA3 gene regulation in the control of Ca2+-dependent platelet functions.
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Zarain-Herzberg, Angel, and Georgina Alvarez-Fernandez. "Sarco(endo)plasmic Reticulum Ca2+-ATPase-2 Gene: Structure and Transcriptional Regulation of the Human Gene." Scientific World JOURNAL 2 (2002): 1469–83. http://dx.doi.org/10.1100/tsw.2002.228.

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The sarco(endo)plasmic reticulum Ca2+-ATPases (SERCAs) belong to a family of active calcium transport enzymes encoded by the SERCA1, 2, and 3 genes. In this study, we describe the complete structure of the human SERCA2 gene and its 5’ -regulatory region. The hSERCA2 gene is located in chromosome 12 position q24.1 in Contig NT_009770.8, spans 70 kb, and is organized in 21 exons intervened by 20 introns. The last two exons of the pre-mRNA produce by alternatively splicing the cardiac/slow-twitch muscle-specific SERCA2a isoform and the ubiquitous SERCA2b isoform. The sequence of the proximal 225-bp regulatory region of the SERCA2 genes is 80% G+C-rich and is conserved among human, rabbit, rat, and mouse species. It contains a TATA-like-box, an E-box/USF sequence, a CAAT-box, four Sp1 binding sites, and a thyroid hormone responsive element (TRE). There are two other conserved regulatory regions located between positions -410 to -661 bp and from -919 to -1410 bp. Among the DNA cis-elements present in these two regulatory regions there are potential binding sites for: GATA-4, -5, -6, Nkx-2.5/Csx, OTF-1, USF, MEF-2, SRF, PPAR/RXR, AP-2, and TREs. Upstream from position -1.5 kb, there is no significant homology among the SERCA2 genes cloned. In addition, the human gene has several repeated sequences mainly of the Alu and L2 type located upstream from position -1.7 kb, spanning in a continuous fashion for more than 40 kb. In this study, we report the cloning of 2.4 kb of 5’-regulatory region and demonstrate that the proximal promoter region is sufficient for expression in cardiac myocytes, and the region from -225 to -1232 bp contains regulatory DNA elements which down-regulate the expression of the SERCA2 gene in neonatal cardiomyocytes.
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Wu, K. D., W. S. Lee, J. Wey, D. Bungard, and J. Lytton. "Localization and quantification of endoplasmic reticulum Ca(2+)-ATPase isoform transcripts." American Journal of Physiology-Cell Physiology 269, no. 3 (September 1, 1995): C775—C784. http://dx.doi.org/10.1152/ajpcell.1995.269.3.c775.

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The Ca(2+)-adenosinetriphosphatase pump of the sarcoplasmic or endoplasmic reticulum (SERCA) plays a critical role in Ca2+ signaling and homeostasis in all cells and is encoded by a family of homologous and alternatively spliced genes. To understand more clearly the role the different isoforms play in cell physiology, we have undertaken a quantitative and qualitative assessment of the tissue distribution of transcripts encoding each SERCA isoform. SERCA1 expression is restricted to fast-twitch striated muscles, SERCA2a to cardiac and slow-twitch striated muscles, whereas SERCA2b is ubiquitously expressed. SERCA3 is expressed most abundantly in large and small intestine, thymus, and cerebellum and at lower levels in spleen, lymph node, and lung. In situ hybridization analyses revealed SERCA3 transcripts in cells of the intestinal crypt, the thymic cortex, and Purkinje cells in cerebellum. In addition, SERCA3 was expressed abundantly in isolated rat spleen lymphocytes, in various murine lymphoid cell lines, and in primary cultured microvascular endothelial cells. This analysis demonstrates that SERCA3 is expressed selectively in cells in which Ca2+ signaling plays a critical and sensitive role in regulating physiological processes.
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Chami, Mounia, Devrim Gozuacik, David Lagorce, Marisa Brini, Pierre Falson, Gérard Peaucellier, Paolo Pinton, et al. "Serca1 Truncated Proteins Unable to Pump Calcium Reduce the Endoplasmic Reticulum Calcium Concentration and Induce Apoptosis." Journal of Cell Biology 153, no. 6 (June 11, 2001): 1301–14. http://dx.doi.org/10.1083/jcb.153.6.1301.

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By pumping calcium from the cytosol to the ER, sarco/endoplasmic reticulum calcium ATPases (SERCAs) play a major role in the control of calcium signaling. We describe two SERCA1 splice variants (S1Ts) characterized by exon 4 and/or exon 11 splicing, encoding COOH terminally truncated proteins, having only one of the seven calcium-binding residues, and thus unable to pump calcium. As shown by semiquantitative RT-PCR, S1T transcripts are differentially expressed in several adult and fetal human tissues, but not in skeletal muscle and heart. S1T proteins expression was detected by Western blot in nontransfected cell lines. In transiently transfected cells, S1T homodimers were revealed by Western blot using mildly denaturing conditions. S1T proteins were shown, by confocal scanning microscopy, to colocalize with endogenous SERCA2b into the ER membrane. Using ER-targeted aequorin (erAEQ), we have found that S1T proteins reduce ER calcium and reverse elevation of ER calcium loading induced by SERCA1 and SERCA2b. Our results also show that SERCA1 variants increase ER calcium leakage and are consistent with the hypothesis of a cation channel formed by S1T homodimers. Finally, when overexpressed in liver-derived cells, S1T proteins significantly induce apoptosis. These data reveal a further mechanism modulating Ca2+ accumulation into the ER of nonmuscle cells and highlight the relevance of S1T proteins to the control of apoptosis.
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Reinhardt, Timothy A., and Ronald L. Horst. "Ca2+-ATPases and their expression in the mammary gland of pregnant and lactating rats." American Journal of Physiology-Cell Physiology 276, no. 4 (April 1, 1999): C796—C802. http://dx.doi.org/10.1152/ajpcell.1999.276.4.c796.

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The transcellular Ca2+fluxes required for milk production must be rigorously regulated to maintain the low cytosolic Ca2+concentrations critical to cell function. Ca2+-ATPases play a critical role in the maintenance of this cellular Ca2+ homeostasis. Using RT-PCR and sequencing, we identified six Ca2+pumps in lactating mammary tissue. Three plasma membrane Ca2+-ATPases (PMCAs) were found (PMCA1b, PMCA2b, and PMCA4b). Two sarco (endo)plasmic reticulum Ca2+-ATPases (SERCAs) were identified (SERCA2 and SERCA3), and the rat homologue to the yeast Golgi Ca2+-ATPase RS-10 was also found. The pattern of mRNA expression of each of these pumps was examined in rat mammary tissue from the 7th day of pregnancy to the 21st day of lactation. Northern blots revealed increased mRNA expression for all Ca2+ pumps by the 14th day of lactation, and transcripts continued to increase through the 18th day of lactation. PMCA1b, PMCA4b, SERCA2, and SERCA3 showed the lowest levels of expression. RS-10 transcripts were more abundant than SERCA2, SERCA3, PMCA1b, and PMCA4b. RS-10 was the only pump to increase in expression before parturition. PMCA2b was the most abundant transcript found in lactating mammary tissue. At peak lactation, expression of PMCA2b approached that of actin. The high expression, high affinity for Ca2+, and high activity at low calmodulin concentrations exhibited by PMCA2b suggest that it is uniquely suited for maintenance of Ca2+ homeostasis in the lactating mammary gland. The pattern of expression and abundance of RS-10 suggest that it is a candidate for the Golgi Ca2+-ATPase shown to be important in maintaining the Golgi Ca2+concentration required for casein synthesis and micelle formation.
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Dissertations / Theses on the topic "Serdab"

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Lehmann, Katja [Verfasser], and Jan [Akademischer Betreuer] Assmann. "Der Serdab in den Privatgräbern des Alten Reiches / Katja Lehmann ; Betreuer: Jan Assmann." Heidelberg : Propylaeum, 2002. http://d-nb.info/1226471137/34.

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Holst, Anna. "Singular serial." Thesis, Konstfack, Inredningsarkitektur & Möbeldesign, 2006. http://urn.kb.se/resolve?urn=urn:nbn:se:konstfack:diva-2703.

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Projektet handlar om formgivning och konsumtion, om hur mycket vi förbrukar (varför) och om produkternas livslängd. Våra produkter är kanske mer miljövänliga nu än för ett par decennier sedan, men vi köper fler. På en kort tid har vårt sätt att konsumera ändrats oerhört. En förutsättning för vår konsumtion är att både material och arbetskraft är billigt, vilket är ohållbart i ett längre perspektiv. Materialen blir dyrare ju mindre som finns kvar, arbetskraften är bara billig så länge landet den finns i är fattigt. Som formgivare kan man känna en problematik i att formge "en sak till". I examensarbetet vill jag ta ställning till detta och leta efter lösningar. Hur vill jag arbeta som formgivare?
Magisterexamen 2006
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Ayhan, Serdal [Verfasser]. "Hochgenaue radarbasierte Abstandsmessung mit geführter Wellenausbreitung / Serdal Ayhan." Karlsruhe : KIT Scientific Publishing, 2016. http://www.ksp.kit.edu.

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Sharma, Neena. "SERIAL PROTOCOL BRIDGE." University of Cincinnati / OhioLINK, 2012. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1352403332.

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Çekin, Mesut Serdar [Verfasser]. "Offenlegungs- und Mitteilungspflichten nach § 67 AktG / Mesut Serdar Çekin." Frankfurt : Peter Lang GmbH, Internationaler Verlag der Wissenschaften, 2012. http://d-nb.info/1042414203/34.

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Blignaut, Christiaan Johannes. "Ketamine-butorphanol-medetomidine versus butorphanol-midazolam-medetomidine immobilisation of serval (Leptailurus serval)." Diss., University of Pretoria, 2020. http://hdl.handle.net/2263/77438.

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Objective To compare ketamine-butorphanol-medetomidine (KBM) to butorphanolmidazolam- medetomidine (BMM) for chemical capture (immobilisation) of serval (Leptailurus serval). Study design Blinded, randomised immobilisation trial. Animals 23 free-ranging captures (KBM: 5 females, 6 males; mean weight 10.7 kg; BMM: 10 females, 2 males; mean weight 9.6 kg). Methods Free-ranging serval were cage trapped and then immobilised using the randomly assigned drug combination delivered via a blow dart into the gluteal muscles. Prior to darting, a stress score was assigned (0: Calm; to 3: markedly stressed). The drug combinations were dosed based on estimated body weights: KBM - 8.0, 0.4 and 0.08 mg kg-1, respectively; BMM - 0.4, 0.3 and 0.08 mg kg-1, respectively. Time to first handling, duration of anaesthesia and recovery times were recorded. Physiological variables were recorded at five-minute intervals and arterial blood was sampled 20 minutes after instrumentation for arterial blood gas analysis. Atipamezole (5 mg kg-1 medetomidine) and naltrexone (2 mg kg-1 butorphanol) were administered intramuscularly for recovery. Data, presented using mean ± standard deviation values, were analysed using student t-test, general linear mixed model and Spearman’s rank correlation. Results The dose based on actual body weights were 8.7 ± 1.5, 0.4 ± 0.08 and 0.09 ± 0.02 mg kg-1 for KBM; and 0.5 ± 0.07, 0.4 ± 0.01 and 0.09 ± 0.05 mg kg-1 for BMM. Time to first handling was 611 ± 165 seconds for KBM and 800 ± 228 seconds for BMM (p = 0.033). Both combinations produced a physiological stable immobilisation that lasted for at least 35 minutes. Recovery was rapid and calm overall, but ataxia was noted in KBM. Stress score was positively and strongly correlated to blood glucose (r2 = 0.788; p = 0.001) and temperature (r2 = 0.634; p = 0.015). Conclusion and clinical relevance Both combinations produce similar effective immobilisation that were physiologically stable in serval. Overall, BMM is recommended because it is fully antagonisable. It is essential to provide a calm, quiet environment before drug administration to avoid capture-induced hyperglycaemia and hyperthermia.
Dissertation (MSc (Veterinary Science))--University of Pretoria, 2019.
Companion Animal Clinical Studies
MSc (Veterinary Science)
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Robb, Simon. "Reading the serial killer /." Title page, contents and introduction only, 1994. http://web4.library.adelaide.edu.au/theses/09AR/09arr631.pdf.

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Lewis-Åkerman, Erik. "Rapid Serial Visual Presentation." Thesis, Malmö universitet, Fakulteten för teknik och samhälle (TS), 2018. http://urn.kb.se/resolve?urn=urn:nbn:se:mau:diva-20800.

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Rapid Seriell Visuell Presentation (RSVP) är ett koncept som har möjligheten att påverka hur en användare konsumerar sin dagliga läsning. Denna metod att läsa skiljer sig från traditionell läsning på så sätt att enbart ett (ibland flera) ord visas i varje bildsekvens i motsats till traditionell läsning då läsaren har full överblick över den statiska texten. Eftersom denna metod att läsa skiljer sig i sådan utsträckning från traditionell läsning så finns det ett behov av att utveckla RSVP till att bli en allt mer intuitiv upplevelse för läsaren. Sex testpersoner intervjuas, varvid resultaten analyseras och utvecklas till en prototyp.
Rapid Serial Visual Presentation (RSVP) is a concept that changes every day reading into an efficient way of gathering information. Due to this innovative new way of reading, the importance of interaction that allows the user to interact with RSVP in an intuitive manner is needed to help the reader reach its full reading potential. This thesis puts existing RSVP programs to the test with the help of six participants in different ages. Through in-depth interviews with the participants, this thesis presents a possible new way of displaying information and interacting with RSVP. From analysing the results of the in-depth interviews, a prototype is created.
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Hu, Yi Ni. "Serial killers in the People's Republic of China :the origins underlying the serial killing." Thesis, University of Macau, 2016. http://umaclib3.umac.mo/record=b3534658.

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Silva, Felipe Henriques da 1978. "Serial Annotator = managing annotations of time series = Serial Annotator: gerenciando anotações em séries temporais." [s.n.], 2013. http://repositorio.unicamp.br/jspui/handle/REPOSIP/275622.

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Orientador: Claudia Maria Bauzer Medeiros
Dissertação (mestrado) - Universidade Estadual de Campinas, Instituto de Computação
Made available in DSpace on 2018-08-23T21:42:10Z (GMT). No. of bitstreams: 1 Silva_FelipeHenriquesda_M.pdf: 3283921 bytes, checksum: 6875b168c728390c5cbeb2e32389cb99 (MD5) Previous issue date: 2013
Resumo: Séries temporais são sequências de valores medidos em sucessivos instantes de tempo. Elas são usadas em diversos domínios, tais como agricultura, medicina e economia. A análise dessas séries é de extrema importância, fornecendo a especialistas a capacidade de identificar tendências e prever possíveis cenários. A fim de facilitar sua análise, especialistas frequentemente associam anotações com séries temporais. Tais anotações também podem ser usadas para correlacionar séries distintas, ou para procurar por séries específicas num banco de dados. Existem muitos desafios envolvidos no gerenciamento destas anotações - desde encontrar estruturas adequadas para associá-las com as séries, até organizar e recuperar séries através das anotações associadas a estas. Este trabalho contribui para o trabalho em gerenciamento de séries temporais. Suas principais contribuições são o projeto e desenvolvimento de um arcabouço para o gerenciamento de múltiplas anotações associadas com uma ou mais séries em um banco de dados. Este arcabouço também fornece meios para o controle de versão das anotações, de modo que os estados anteriores de uma anotação nunca sejam perdidos. Serial Annotator é uma aplicação desenvolvida para a plataforma Android. Ela foi usada para validar o arcabouço proposto e foi testada com dados reais envolvendo problemas do domínio agrícola
Abstract: Time series are sequences of values measured at successive time instants. They are used in several domains such as agriculture, medicine and economics. The analysis of these series is of utmost importance, providing experts the ability to identify trends and forecast possible scenarios. In order to facilitate their analyses, experts often associate annotations with time series. Such annotations can also be used to correlate distinct series, or look for specific series in a database. There are many challenges involved in managing annotations - from finding proper structures to associate them with series, to organizing and retrieving series based on annotations. This work contributes to the work in management of time series. Its main contributions are the design and development of a framework for the management of multiple annotations associated with one or multiple time series in a database. The framework also provides means for annotation versioning, so that previous states of an annotation are never lost. Serial Annotator is an application implemented for the Android smart phone platform. It has been used to validate the proposed framework and has been tested with real data involving agriculture problems
Mestrado
Ciência da Computação
Mestre em Ciência da Computação
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Books on the topic "Serdab"

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Soekirman. Onderneming van Sergai: Perkembangan kebun-kebun di Kabupaten Serdang Bedagai. Yogyakarta: Pustaka Raja, 2014.

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Serial. México, D.F: Consejo Nacional para la Cultura y las Artes. Dirección General de Publicaciones, 2011.

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Serial. New York: Pinnacle Books, 2011.

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Gurban, Durdymukhammet. Serdar: Tăze tu̇rkmen tarykhyna syn. Ashgabat: "Tu̇rkmenistan" RNChB, 1992.

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Waters, John, John Fiedler, and Mark Tarlov. Serial mom. Universal City, CA: Universal Studios Home Entertainment, 2008.

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Chudori-Soerjoatmojo, Rain. Serdadu kumbang. Yogyakarta: Gradien Mediatama, 2011.

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Brinton, Alexander. Serial blood. New York: Zebra Books, 1992.

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Serial bride. Toronto: Harlequin, 2006.

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Martínez, Carlos Dámaso. Serial: Novela. Córdoba, Argentina: Ediciones del Copista, 2006.

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Carmen Serdán. Puebla, Pue., México: Benemérita Universidad Autónoma de Puebla, Dirección de Fomento Editorial, 2010.

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Book chapters on the topic "Serdab"

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Weik, Martin H. "serial." In Computer Science and Communications Dictionary, 1552. Boston, MA: Springer US, 2000. http://dx.doi.org/10.1007/1-4020-0613-6_17024.

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Hale, Kenneth L. "Misumalpan Verb Sequencing Constuctions." In Serial Verbs, 1. Amsterdam: John Benjamins Publishing Company, 1991. http://dx.doi.org/10.1075/ssls.8.02hal.

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Lefebvre, Claire. "TakeSerial Verb Constructions in Fon." In Serial Verbs, 37. Amsterdam: John Benjamins Publishing Company, 1991. http://dx.doi.org/10.1075/ssls.8.03lef.

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Baker, Mark C. "On the Relation of Serialization to Verb Extensions." In Serial Verbs, 79. Amsterdam: John Benjamins Publishing Company, 1991. http://dx.doi.org/10.1075/ssls.8.04bak.

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Li, Yafei. "On Deriving Serial Verb Constructions." In Serial Verbs, 103. Amsterdam: John Benjamins Publishing Company, 1991. http://dx.doi.org/10.1075/ssls.8.05li.

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Givón, T. "Some Substantive Issues Concerning Verb Serialization." In Serial Verbs, 137. Amsterdam: John Benjamins Publishing Company, 1991. http://dx.doi.org/10.1075/ssls.8.06giv.

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Larson, Richard K. "Some Issues in Verb Serialization." In Serial Verbs, 185. Amsterdam: John Benjamins Publishing Company, 1991. http://dx.doi.org/10.1075/ssls.8.07lar.

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Stamant, Nicole. "Introduction." In Serial Memoir, 1–27. London: Palgrave Macmillan UK, 2014. http://dx.doi.org/10.1057/9781137410337_1.

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Stamant, Nicole. "Mary McCarthy’s archival performance as a “perfect execution of the idea”." In Serial Memoir, 28–52. London: Palgrave Macmillan UK, 2014. http://dx.doi.org/10.1057/9781137410337_2.

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Stamant, Nicole. "Alternate archives." In Serial Memoir, 53–84. London: Palgrave Macmillan UK, 2014. http://dx.doi.org/10.1057/9781137410337_3.

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Conference papers on the topic "Serdab"

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Elgamal, Tarek, and Klara Nahrstedt. "Serdab: An IoT Framework for Partitioning Neural Networks Computation across Multiple Enclaves." In 2020 20th IEEE/ACM International Symposium on Cluster, Cloud and Internet Computing (CCGRID). IEEE, 2020. http://dx.doi.org/10.1109/ccgrid49817.2020.00-41.

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Rashdan, Mostafa, Fahmi El-Sayed, and Mohammad Salman. "Performance Comparison between SerDes and Time-Based Serial Links." In 2020 7th International Conference on Electrical and Electronics Engineering (ICEEE). IEEE, 2020. http://dx.doi.org/10.1109/iceee49618.2020.9102626.

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Roosevelt, Gary, Weston Roper, and Thomas Romanko. "Optimizing high speed serial communication using Honeywell Rad Hard SerDes." In 2011 NASA/ESA Conference on Adaptive Hardware and Systems (AHS). IEEE, 2011. http://dx.doi.org/10.1109/ahs.2011.5963938.

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Roosevelt, Gary, David Bueno, Jamal Haque, Weston Roper, and Thomas Romanko. "Rad-Hard high speed serial communication using Honeywell SerDes macros." In 2009 IEEE Aerospace conference. IEEE, 2009. http://dx.doi.org/10.1109/aero.2009.4839504.

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Melikyan, Vazgen Sh, Arshavir V. Matevosyan, Arman S. Petrosyan, Armen A. Martirosyan, Karen T. Khachikyan, Ruben H. Musayelyan, Arman S. Trdatvan, and David K. Hakobyan. "High Quality Factor 5.0 Gbps CTLE Circuit for SERDES Serial Links." In 2018 IEEE East-West Design & Test Symposium (EWDTS). IEEE, 2018. http://dx.doi.org/10.1109/ewdts.2018.8524731.

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Gumaste, Ashwin. "Towards a Transponder for Serial 100 Gigabit Ethernet using a Novel Optical SERDES." In National Fiber Optic Engineers Conference. Washington, D.C.: OSA, 2009. http://dx.doi.org/10.1364/nfoec.2009.jwa63.

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Landy, Aaron, and Greg Stitt. "Doubling FPGA Throughput via a Soft SerDes Architecture for Full-Bandwidth Serial Pipelining (Abstract Only)." In FPGA'16: The 2016 ACM/SIGDA International Symposium on Field-Programmable Gate Arrays. New York, NY, USA: ACM, 2016. http://dx.doi.org/10.1145/2847263.2847301.

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Cogliati, Paolo. "Serial taxi." In SIGGRAPH Asia 2013 Computer Animation Festival. New York, New York, USA: ACM Press, 2013. http://dx.doi.org/10.1145/2542398.2542438.

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Serban, Emanuel. "Emanuel Serban." In 2008 11th International Conference on Optimization of Electrical and Electronic Equipment (OPTIM). IEEE, 2008. http://dx.doi.org/10.1109/optim.2008.4602301.

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Pagliari, Daniele Jahier, Enrico Macii, and Massimo Poncino. "Serial T0." In DAC '16: The 53rd Annual Design Automation Conference 2016. New York, NY, USA: ACM, 2016. http://dx.doi.org/10.1145/2897937.2898089.

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Reports on the topic "Serdab"

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Kandasamy, A. Serial interface controller. Office of Scientific and Technical Information (OSTI), April 1995. http://dx.doi.org/10.2172/137308.

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Elz, R., and R. Bush. Serial Number Arithmetic. RFC Editor, August 1996. http://dx.doi.org/10.17487/rfc1982.

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Kingston, Mary Lynn, and Mary Lynn Kingston. NIST serial holdings. Gaithersburg, MD: National Institute of Standards and Technology, 1990. http://dx.doi.org/10.6028/nist.sp.777-1990.

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Kingston, Mary Lynn, and Mary Lynn Kingston. NIST serial holdings. Gaithersburg, MD: National Institute of Standards and Technology, 1991. http://dx.doi.org/10.6028/nist.sp.777-1991.

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Sanders, Susan A., and Susan A. Sanders. NIST serial holdings. Gaithersburg, MD: National Institute of Standards and Technology, 1992. http://dx.doi.org/10.6028/nist.sp.777-1992.

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Sanders, Susan A., and Susan A. Sanders. NIST serial holdings. Gaithersburg, MD: National Institute of Standards and Technology, 1993. http://dx.doi.org/10.6028/nist.sp.777-1993.

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Sanders, Susan A., and Susan A. Sanders. NIST serial holdings. Gaithersburg, MD: National Institute of Standards and Technology, 1994. http://dx.doi.org/10.6028/nist.sp.777-1994.

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Sanders, Susan A., and Susan A. Sanders. NIST serial holdings. Gaithersburg, MD: National Institute of Standards and Technology, 1995. http://dx.doi.org/10.6028/nist.sp.777-1995.

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Sanders, Susan A., and Susan A. Sanders. NIST serial holdings. Gaithersburg, MD: National Institute of Standards and Technology, 1996. http://dx.doi.org/10.6028/nist.sp.777-1996.

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Sanders, Susan A., and Susan A. Sanders. NIST serial holdings. Gaithersburg, MD: National Institute of Standards and Technology, 1997. http://dx.doi.org/10.6028/nist.sp.777-1997.

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