Relevant bibliographies by topics / Severe anemia

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Academic literature on the topic 'Severe anemia'

Author: Grafiati
Published: 5 June 2025
Last updated: 24 June 2025

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Journal articles on the topic "Severe anemia"

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Shih, Angela, Ruby E. Kassanoff, and Basel Altrabulsi. "Severe Anemia." Baylor University Medical Center Proceedings 14, no. 3 (2001): 289–93. http://dx.doi.org/10.1080/08998280.2001.11927775.

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Akpinar, Cetin Kursad, Erdem Gurkas, and Emrah Aytac. "Moderate to Severe Anemia Is Associated with Poor Functional Outcome in Acute Stroke Patients Treated with Mechanical Thrombectomy." Interventional Neurology 7, no. 1-2 (2017): 12–18. http://dx.doi.org/10.1159/000480642.

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Background: Anemia will negatively affect cerebral collaterals and penumbra. Eventually, it may cause worse clinical outcomes and even increase mortality rates in stroke patients. Anemia has recently been suggested to be an independent risk factor for ischemic stroke. Therefore, we aimed to investigate the effects of the presence of anemia on clinical outcomes in ischemic stroke patients undergoing mechanical thrombectomy. Methods: This was a retrospective study involving the prospectively and consecutively collected data of 90 adult patients between January 2015 and August 2016. Hemoglobin (Hb) cutoff levels were accepted as 12 g/dL for women and 13 g/dL for men. Patients having anemia were further divided into three subgroups as severe anemia (Hb <8 g/dL for both genders), moderate anemia (Hb <10 g/dL for both genders), and mild anemia (Hb <13 g/dL for men and Hb <12 g/dL for women). Results: Forty of the subjects (44.4%) had anemia. Moderate anemia was detected in 14 out of 90 patients (15.5%) and severe anemia was found in only four of them (4.4%). Poor functional outcome (mRS 3-6) was similar in both anemic and non-anemic patients (37.5% vs. 38%, respectively, p = 0.08), but poor functional outcome was found to be statistically significant with severe anemic group (Hb <8 mg/dL) (p = 0.003). In multiple logistic regression analysis, moderate and severe anemia has been found to increase the mortality (p = 0.032). Conclusions: Our study demonstrated a poor functional outcome only in moderate to severe anemic patients. Clinicians should keep in mind the negative effect of moderate to severe anemia in the clinical course of acute stroke patients treated with mechanical thrombectomy.
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Chen, Chong, Wenhui Zhou, Wenliang Fan, et al. "Association of anemia and COVID-19 in hospitalized patients." Future Virology 16, no. 7 (2021): 459–66. http://dx.doi.org/10.2217/fvl-2021-0044.

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Aim: COVID-19 is a major threat to public health worldwide. A large proportion of COVID-19 patients is proved to develop anemia. Herein, we investigate the association between anemia and severe pneumonia. Materials & methods: 137 of COVID-19-confirmed patients admitted to Wuhan Union Hospital (Wuhan, China) from 13 February to 17 March 2020 were included. Their clinical characteristics and laboratory data were studied, and multivariable logistic regression analyses were performed. Results: The anemic patients were less likely to develop fever in the early stage of COVID-19. Elevated IL-6 levels were found in anemic COVID-19 patients compared with those without anemia. COVID-19 patients with anemia had an 8.2 times greater possibility of developing severe pneumonia compared with their counterparts without anemia. Conclusion: This study comprehensively describes the clinical characteristics of anemic patients with ordinary, severe and critical COVID-19 and demonstrates the close relationship between the anemia and severe COVID-19.
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Yadav, Meetu, Pooja T, Sumit Dagar, Arti Dhingra, and Divya Mangla. "Outcome of newborns born to anaemic mother in a rural tertiary care hospital, comparative study." Indian Journal of Public Health Research & Development 16, no. 2 (2025): 191–98. https://doi.org/10.37506/8rd7y767.

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Background: Anemia is one of the most common nutritional deficiency issues in pregnant women. Maternal anemia has been linked (IUGR), LBW, and (PPH). Prematurity, poor Apgar scores, and intrauterine foetal mortality are more likely in anemic pregnant women than in nonanemic pregnant women.5 Maternal anemia increases the odds of preterm delivery and low birth weight of the newborns, which increases foetal morbidity and death.Hence, this study was aimed to assess neonatal outcomes among babies born to anemic mothers in comparison to Non-anemic and to find the correlation between maternal and neonatal hemoglobin and serum ferritin levels. Methods: The records of 100 anemic and 30 non anemic mothers ,and newborns over a period of one year (October 2020 to April 2021) were reviewed, and a statistical comparison of various variables were made. Results: Pre-term birth was significantly more among severe anemia compared to mild and moderate anemia. Low birth weight was significantly more among subjects with severe anemia (81.8%) compared to moderate anemia (59.2%) which was significantly more than mild anemia (37.5%). APGAR score at 1 minute < 7 was significantly more among severe anemia (100.0%) compared to moderate anemia (61.2%) which was significantly more than mild anemia (82.5%). APGAR score at 5 minutes < 7 was significantly more among severe anemia (100.0%) compared to moderate and mild anemia (2.5% and 6.1% respectively).There was a significantly positive association of maternal and newborn hemoglobin levels.There was a significantly positive association of maternal and newborn Ferritin levels.Antenatal visits >3 were significantly more among Non-Anemic mothers compared to Anemic mothers.The mean Maternal Hb, Maternal Ferritin, Hb of newborn and Serum Ferritin of Newborn was significantly more among Non-Anemic group compared to Anemic group. Conclusion: Anaemia in pregnancy definitely has a very poor outcome on the newborn with increase in severity of anaemia in terms of low birth weight, poor APGAR score.
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Bansal, Priyanka, and Anand Singhal. "Women with Severe Anemia in Labor: Fetomaternal Outcomes." International Journal of Health Sciences and Research 12, no. 1 (2022): 1–6. http://dx.doi.org/10.52403/ijhsr.20220101.

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Background: Anaemia in pregnancy is the leading medical disorder in developing countries like India. Women with severe anemia in labor is a challenging situation for the obstetrician with increased maternal and perinatal morbidity and mortality. It has multifactorial etiology, Main cause of anemia in obstetrics is iron deficiency. There are specific risks for the mother like preterm labour pain, IUGR baby, preeclampsia, antepartum haemorrhage, sepsis etc and the fetus such as prematurity, lowbirth weight, birth asphyxia etc. Aim and Objectives: To determine the socio-demographic variables and also the maternal and perinatal outcomes of pregnant women admitted to labour room with severe anemia (Hb <7gm%) late in pregnancy. Results: It was seen that out of 50 severely anemic women, 82% had Hb between 4-7 gm% and 18% had Hb < 4gm%. Severe anemia is seen in 82% unbooked patients and only in 18% booked patients. There was increased incidence of preterm delivery, PPH, preeclampsia, eclampsia, mortality in anemic group as compared to non anemic group. Among the adverse fetal outcomes, there was increased incidence of intrauterine deaths, intrauterine growth restriction, NICU admission, low birth weight among the anemic group as compared to non anemic group. Conclusions: Severely anemic women reporting in labor had significantly high maternal and perinatal morbidity and mortality. Even a minor blood loss is much devastating for such patients. Extrapolating our observations, it can be said that a close vigilance, anticipation of complications and appropriate care and interventions during labor and puerperium will help in improving outcomes in these severely anemic women. Awareness and education on early antenatal bookings, regular iron intake and continuous antenatal care should be the goal in tackling anemia in pregnancy. Key words: Anaemia, Iron deficiency, Intrauterine growth retardation, prematurity, maternal outcome, neonatal outcome.
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Liu, Pin-Chen, and Bi-Ling Su. "CAUSES OF CANINE ANEMIA IN TAIWAN: A FIVE-YEAR RETROSPECTIVE SURVEY." Taiwan Veterinary Journal 41, no. 01 (2015): 31–37. http://dx.doi.org/10.1142/s1682648515500031.

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Anemia is a common hematologic disorder in dogs, however, few data are available regarding epidemiology and causes in Taiwan. To investigate the causes of anemia, 3174 anemic cases (hematocrit < 37%) collected between January 2008 and December 2012 at National Taiwan University Veterinary Hospital (NTUVH) were analyzed. Most dogs (48.5%, n = 1540) presented with a mild form (30% ≤ hematocrit < 37%), which was followed by a moderate form (20% ≤ hematocrit < 30%; 36.4%, n = 1155), a severe form (13% ≤ hematocrit < 20%; 10.5%, n = 332) and a very severe form (hematocrit < 13%; 4.6%, n = 147). Among the 2037 dogs with identifiable causes, 70.4% (1435 dogs) were induced by single cause, whereas 29.6% (602 dogs) by multiple causes. Cancer-related anemia (CRA, n = 460), infectious pathogens-related anemia (n = 287), renal disease-related anemia (n = 251) and post-surgery/trauma-related anemia (n = 182) account for 32.1%, 20.0%, 17.6% and 12.7% of single-cause cases, respectively. Furthermore, 255 of the total (17.8.0%) presented with severe and very severe anemia. 72 dogs with very severe anemia primarily resulted from infectious disease-related anemia (59.7%), followed by immune-mediated hemolytic anemia (13.9%), and other disease-related anemia (9.7%). Of the 43 infectious disease-related very severe anemic dogs, the most commonly diagnosed pathogen was Babesia gibsoni (83.7%, n = 36), followed by Ehrlichia canis (11.6%, n = 5), Babesia canis (2.3%, n = 1) and Leptospira spp. (2.3%, n = 1). Taken together, cancer, infectious diseases, and renal failure are the most frequent causes of canine anemia in Taiwan, and B. gibsoni appeared to be the most important infectious pathogen causing very severe anemia.
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Singh, Ruchika, Mamta Gupta, and Vandana Saini. "Pregnant women with severe anemia reporting in labor: prevalence, socio-demographic and obstetric determinants." International Journal Of Community Medicine And Public Health 6, no. 2 (2019): 701. http://dx.doi.org/10.18203/2394-6040.ijcmph20190194.

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Background: Severely anemic women reporting in labor remains one of the most important challenging situation for the obstetrician as well as for the mother and her family due to its adverse feto-maternal outcome. Various socio-demographic and obstetric conditions need to be explored which are important to tackle them, for primary prevention of anemia. The aim and objectives of the study were to estimate prevalence of severe anemia in pregnant women reporting in labor in a tertiary hospital of Delhi and to evaluate various socio-economic and associated obstetric factors associated.Methods: This is a hospital based, prospective, case contol study. Hemoglobin was estimated at the time of labor room admission. Fifty consecutive antenatal women with severe anemia (Group A) and 50 non-anemic women (Group B) were enrolled in early labor. Socio-demographic and obstetric factors, were recorded and analyzed.Results: Prevalence of severe anemia was estimated to be 2.23%. Determinants of severe anemia were found to be socio-economic status (p value 0.001), education (p value 0.001), rural living (p value 0.016), calorie intake (p value 0.001), BMI (p value 0.046), booking status of pregnancy (p value 0.001), gravida (p value 0.024), inter-conception interval (p value 0.002) and regular iron-folic acid intake (p value 0.001).Conclusions: Primary prevention of anemia by targeting these factors at the community/state/ national level, by the policy makers is important. Early booking and screening for anemia in antenatal clinics, providing iron supplements to anemic women for secondary prevention of severe anemia is recommended so that no woman reports with severe anemia in labor.
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Gold, Eric. "Severe Fetal Anemia." Laboratory Medicine 28, no. 1 (1997): 21–24. http://dx.doi.org/10.1093/labmed/28.1.21.

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Mir, Naseer Yousuf, Bashir U. Zaman, Jawad Nazir Wani, Mohammad Imran Malik, Muzaffar Jan, and Kaiser Ahmad. "Non-nutritional causes of severe anemia in Kashmiri children." International Journal Of Community Medicine And Public Health 10, no. 3 (2023): 1256–60. http://dx.doi.org/10.18203/2394-6040.ijcmph20230649.

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Background: Anemia is one of the leading causes of mortality and morbidity in children. The etiology of anemia is multifactorial including nutritional anemia, hemolytic anemia, aplastic anemia, storage disorders, hematological malignancies, anemia secondary to some chronic diseases. Methods: The study is an observational study. Physical signs of severe anemia were looked in patients attending outpatient department or admitted in hospital. Children with hemoglobin levels <7 g/dl and in the age group 6 months to 6 years were recruited in study. The aim is to look for non-nutritional etiology of severe anemia in children in Kashmir. Results: Out of 192 children, 97 (50.52%) females and 95 (49.48%) males, with a male, female ratio of around 1:1. A total of 147 (76.56%) children in our study were having nutritional and 45 (23.44%) were having non nutritional causes. Among non-nutritional anemia 30 cases were hemolytic anemia, 11 hypoplastic anemia, 3 leukemia and 1 patient were diagnosed as Osteopetrosis. Conclusions: Though nutritional deficiency anemias are predominant; it may not always be the case. Hemolytic anemia and leukemias should be ruled out while evaluating a patient of severe anemia and pancytopenia. Bone marrow failure syndrome, storage disorders should be ruled out in patients with skeletal anomalies.
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Djimde, Moussa, Henk D. F. H. Schallig, Mohamed Bougoury Traoré, et al. "Severe Anemia among Blood Donors in an Area Where Blood Donation Is Sought to Save the Lives of Children with Severe Malaria—Anemia in Blood Donors." African Journal of Parasitology, Mycology and Entomology 1, no. 1 (2023): 1. http://dx.doi.org/10.35995/ajpme1010005.

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Introduction: Severe anemia caused by severe malaria remains concentrated in children under 5 years old in Africa. In addition to blood-transfusion-related infections that pose a health concern, transfusion of severely anemic blood to a patient with severe anemia is another important concern. Method: Following the observation of a case of severe anemia in a blood donor, we conducted a secondary data analysis from a previous study to assess the frequency of anemia in blood donors in San, Mali. Results: In total, out of 140 volunteers, 13 (9%, 95% CI: 5.43–15.41) blood donors had moderate to severe anemia, based on Mali hemoglobin (Hb) normal values (10.5–16.5 g/dL, MRTC-GLP laboratory). In particular, we observed one case of severe anemia with an Hb level of 6.5 g/dL. Conclusions: To the best of our knowledge, this is the first report of severe anemia in blood donors in a malaria endemic area in Mali.
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Dissertations / Theses on the topic "Severe anemia"

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Pirker-Krassnig, Daniela Karoline Polrat Wilairatana. "Hematological changes in patients with severe malaria from Bangkok Hospital for tropical diseases /." Abstract, 2006. http://mulinet3.li.mahidol.ac.th/thesis/2549/cd388/4838786.pdf.

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Thematic Paper ((M.C.T.M. Clinical Tropical Medicine))--Mahidol University, 2006.<br>LICL has E-Thesis 0012 ; please contact computer services. LIRV has E-Thesis 0012 ; please contact circulation services.
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Schinagl, Carina [Verfasser], and Orsolya [Akademischer Betreuer] Genzel-Boroviczény. "Microcirculatory assessment of red blood cell transfusion in children with severe anemia / Carina Schinagl. Betreuer: Orsolya Genzel-Boroviczény." München : Universitätsbibliothek der Ludwig-Maximilians-Universität, 2015. http://d-nb.info/1079477241/34.

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Wildig, James William. "The contribution of Human parvovirus B19 infection to cases of severe anaemia among young children in Malaria endemic areas." Thesis, The University of Sydney, 2008. https://hdl.handle.net/2123/28139.

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Human Parvovirus BI9 (BI9) is a common virus with worldwide distribution. Acute infection causes cessation of erythropoiesis for 5 - 7 days. This viral suppression of bone marrow leads to a decrease in the haemoglobin level of the infected individual. Cases of severe anaemia induced by B19 infection have been described in people with underlying haematological disorders such as Sickle Cell disease, as well as children with malaria and iron deficiency. Among young children in malaria endemic areas anaemia is extremely common. The aetiology of this is complex with multiple contributing factors, including malaria, iron deficiency and protein-energy malnutrition. In 1990 it was suggested that B19 may have a role in a significant number of the cases of severe anaemia that occur among young children in malaria endemic areas (144) . The studies presented in this thesis were undertaken to investigate the contribution of parvovirus B l 9 to cases of severe anaemia among young children in malaria endemic areas.
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Nguyen, The Trong [Verfasser], and Benjamin [Akademischer Betreuer] Mordmüller. "Development and implementation of molecular methods to genotype Plasmodium falciparum in uncomplicated malaria and severe malarial anemia in Gabon / The Trong Nguyen ; Betreuer: Benjamin Mordmüller." Tübingen : Universitätsbibliothek Tübingen, 2020. http://d-nb.info/1206933755/34.

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Santos-Ciminera, Patricia Dantas Ciminera Patricia Dantas Santos Santos Patricia. "Molecular epidemiology of epidemic severe malaria caused by Plasmodium vivax in the state of Amazonas, Brazil /." Download the dissertation in PDF, 2005. http://www.lrc.usuhs.mil/dissertations/pdf/Santos2005.pdf.

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Yenque, Aguilar María Angélica. "Anemia Aplásica Severa: Perfil clínico, Enero 2001 – Diciembre 2004, H. N. ERM – ESSALUD." Bachelor's thesis, Universidad Ricardo Palma, 2006. http://cybertesis.urp.edu.pe/handle/urp/212.

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La anemia aplásica es un trastorno hematopoyético caracterizado por la presencia de pancitopenia asociada a hipocelularidad de médula ósea y sin presencia de células malignas ni fibrosis. Podemos hablar de anemia aplásica cuando a una médula ósea hipocelular, se asocia como mínimo en sangre periférica: < 10 grs. de Hemoglobina, plaquetas en < 50 000 x mm3 y neutrófilos en < 1500 x mm3 10 .Según el grado de hipocelularidad en médula ósea y parámetros en sangre periférica tenemos aplasias leves, como la arriba descrita y las severas definidas como: < 7grs de HB, < 20 000 plaquetas y < 500 neutrófilos PMN. Existen formas moderadas (parámetros sanguíneos entre ambos rangos) y extremadamente severas (neutrófilos PMN 200) La Anemia aplásica severa es conocida por tener diversas etiologías: virales, farmacológicas, inmunológicas, por sustancias químicas. Un buen porcentaje se definen como idiopáticas, de origen y mecanismo desconocido. El objetivo de este trabajo es determinar el perfil clínico de los pacientes adultos diagnosticados de Anemia Aplásica Severa durante el período comprendido 2001-2004.
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Gomes, Isabella Azevedo. "Deficiência de ferro como indicador de risco para cárie de estabelecimento precoce severa em crianças de 24 a 36 meses." Universidade Federal do Maranhão, 2009. http://tedebc.ufma.br:8080/jspui/handle/tede/1124.

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Made available in DSpace on 2016-08-19T18:15:58Z (GMT). No. of bitstreams: 1 ISABELLA AZEVEDO GOMES.pdf: 354561 bytes, checksum: 1b4455b424873020a4e21715db35372b (MD5) Previous issue date: 2009-11-18<br>The aim was evaluate the association between iron deficiency and severe early childhood caries in children between 24 and 36 months; rising hematological indicators that can be related to early establishment of the disease. At intraoral exam the caries index (def) praised by WHO, and the presence of active white spots (AWP) were evaluated. All the children were submitted to a bloody collection, being the samples frozen and sent to processing at Hemomar Analysis Laboratory (São Luís, Maranhão, Brazil), for the evaluation of hemoglobin, ferritin and serumal iron levels. The obtained information was consolidated in data bank of STATA 9.0 program. The Poisson regression model was applied to the association between dependent variables (def and AWP) and others, being the prevalence ratio and confidence interval (IC 95%). It was certified that the higher the number of caries cavities in children, the lower are the hemoglobin levels in serum. About caries activity (AWP), it was observed that the higher the number of initial lesions, the lower is the levels in serum of hemoglobin, ferritin and serumal iron. The data show that the levels in serum of the marked variables of iron deficiency anemia are lower when the number of caries lesions in children with severe early childhood caries is higher.<br>O objetivo foi avaliar a associação entre deficiência de ferro e cárie de estabelecimento precoce severa em crianças de 24 a 36 meses; levantando-se indicadores de riscos hematológicos que possam estar relacionados ao estabelecimento precoce da doença. No exame clínico intra-bucal foram avaliados os índices de cárie (ceo), preconizado pela OMS, e presença de manchas brancas ativas (MBA).Todas as crianças foram submetidas a uma coleta de sangue , sendo as amostras mantidas refrigeradas e enviadas para processamento no Laboratório de Análises do Hemomar (São Luís-MA), para avaliação dos níveis de hemoglobina, ferritina e ferro sérico.As informações obtidas foram consolidadas em bancos de dados do programa STATA 9.0.Foi aplicada uma regressão de Poisson para associação entre as variáveis dependentes (ceo e MBA) e demais variáveis, sendo a razão das prevalências (IR) e intervalo de confiança (IC 95%).Foi observado que quanto maior o número de cavidades de cárie nas crianças, menores foram os níveis séricos da hemoglobina. Em se tratando da atividade da doença cárie (MBA), foi encontrado que quanto maior o número de lesões iniciais, menores foram os níveis séricos de hemoglobina, ferritina e ferro sérico. Os dados mostram que os níveis séricos das variáveis marcadoras da anemia ferropriva são menores quanto maior o número de lesões cariosas em crianças com cárie precoce severa.
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Lorenzato, Claudia Santos. "Análise do crescimento e desenvolvimento de crianças e adultos jovens com anemia aplástica severa submetidos a transplante de medula." reponame:Repositório Institucional da UFPR, 2011. http://hdl.handle.net/1884/25458.

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Resumo: A Anemia Aplástica (AA) é uma doença hematológica rara, de elevada letalidade, caracterizada por pancitopenia no sangue periférico. O transplante de células tronco hematopoiéticas (SCT) está indicado e regimes de condicionamento atualmente utilizados são feitos com ciclofosfamida (CFA) isolada ou associada ao bussulfano (BU). Os efeitos tardios do SCT e do regime de condicionamento com BU e CFA sobre o crescimento e alguns órgãos endócrinos ainda não são totalmente conhecidos. Objetivo: avaliar o crescimento, desenvolvimento gonadal e função tireoidiana em pacientes submetidos ao SCT por AA, onde a doença de base aparentemente não tem efeito deletério sobre estas funções. Pacientes e Métodos: pacientes atendidos no Serviço de Transplante de Medula Óssea do Hospital de Clínicas da UFPR. Os dados de história e exame físico do momento do transplante foram obtidos retrospectivamente. A avaliação pós-transplante compreendeu anamnese, exame físico e exames laboratoriais para avaliar as funções tireoidiana, gonadal e crescimento. Resultados: Foram avaliados 26 pacientes (13 do sexo masculino), com idade média no SCT de 10,6 ± 4,3 anos, idade média na reavaliação de 17,96 ± 2,9 anos; sendo que 12 pacientes foram condicionados com CFA e 14 com CFA+BU. Quanto ao crescimento, não foi encontrada diferença significativa no escore Z da estatura entre os pacientes que foram submetidos a SCT antes e depois dos 10 anos de idade. Também não foi observada diferença significativa quanto ao tipo de condicionamento utilizado. Entretanto, em 3 pacientes houve um avanço maior da idade óssea comparado à idade cronológica. Hipotireoidismo subclínico (TSH aumentado) em 3 dos pacientes. Quanto a função gonadal, 3 pacientes apresentaram algum tipo de alteração, necessitando de reposição hormonal. Uma paciente desenvolveu hipogonadismo hipergonadotrófico. Nenhum paciente do sexo masculino apresentou alterações nos níveis de hormônios sexuais. Conclusão: a maioria dos pacientes submetidos a SCT por AA não apresentou alterações do crescimento, da função gonadal e da função tireoidiana. Entretanto, hipotireoidismo subclínico e alterações da função gonadal podem ocorrer nestes pacientes e devem ser monitorados.
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Scheinberg, Phillip. "Parâmetros preditivos de resposta hematológica, recidiva, evolução clonal e sobrevida em pacientes com anemia aplástica severa tratados com terapia imunossupressora." Universidade de São Paulo, 2018. http://www.teses.usp.br/teses/disponiveis/5/5141/tde-06112018-153339/.

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A anemia aplástica severa (AAS) pode ser tratada com sucesso na maioria dos casos com terapia imunossupressora (IS) ou transplante alogenêico de medula óssea (TMO). Os principais fatores que determinam a escolha da modalidade terapêutica são a idade e a disponibilidade de um doador HLA-histocompatível. Em pacientes mais jovens, o TMO de um doador aparentado é preferível, enquanto que em pacientes acima de 40-50 anos, a terapia IS é a modalidade terapêutica de escolha. Resposta hematológica é obtida em 60-75% dos casos com terapia IS na AAS, o que correlaciona com melhor sobrevida. Recidivas ocorrem em aproximadamente um terço dos respondentes e evolução clonal para mielodisplasia em 10-15% ao longo termo. A doença do enxerto-versus-hospedeiro (GVHD) agudo ocorre em 30-40% dos casos sendo a forma crônica presente em 40-50%. Infecções são frequentes e podem complicar o transplante. Portanto, a refratariedade à terapia IS, recidivas e evolução clonal limitam o sucesso da terapia IS na AAS, enquanto rejeição do enxerto, GVHD, e infecções limitam o sucesso do TMO na clínica. Fatores preditivos dessas complicações seriam de grande valor na clínica, uma vez que poder-se-iam realizar decisões terapêuticas com base mais racional, onde pacientes fossem alocados a diferentes tratamentos com base no seu perfil de risco. Ou seja, pacientes com alta probabilidade de resposta e baixo risco de recidiva e evolução clonal se beneficiariam de terapia IS, enquanto àqueles com baixa probabilidade de resposta e alto risco de recidiva e/ou evolução clonal teriam mais benefícios do TMO, por exemplo. Com base nessa premissa, desenvolvemos estudos para investigar fatores que pudessem estar associados ao sucesso da terapia IS na AAS. Os principais achados de 3 análises distintas sobre o tema evidenciou: 1) crianças (< 18 anos) apresentam alta taxa de resposta à terapia IS (em torno de 75%) com uma excelente sobrevida geral em pacientes respondentes; 2) o número absoluto de reticulócitos e de linfócitos pré-tratamento correlaciona com resposta hematológica aos seis meses após terapia IS; e 3) o comprimento telomérico não está associado à resposta hematológica, porém, está associado a probabilidade de recidiva, evolução clonal, e sobrevida geral após terapia IS. Esses parâmetros identificados nesses estudos podem servir de base em algoritmos futuros onde faz-se estratificação de risco de cada paciente, a fim de alocar a modalidade terapêutica mais apropriada com base no perfil individual de risco. No que diz respeito ao comprimento telomérico, é provável que esse marcador biológico não só esteja associado ao processo de evolução clonal na AAS, mas que também participe na biologia da instabilidade genômica de células na medula óssea levando a aberrações cromossômicas e o desenvolvimento de mielodisplasia e leucemias.<br>Severe aplastic anemia (SAA) can be treated successfully in the majority of cases with immunosuppressive therapy (IST) or allogeneic bone marrow transplantation (BMT). The principal factors that determine the choice of treatment modalities are age and availability of an HLA-histocompatible donor. In younger patients, BMT from a related donor is preferred, while in patients over 40-50 years of age, IST is often employed. Hematologic response is achieved in 60-75% of cases with IST, which correlates with better survival. Relapses occur in approximately one third of responders and clonal evolution to myelodysplasia occurs in 10-15% of cases long-term. Acute graft-versus-host disease (GVHD) occurs in 30-40% of cases and chronic GVHD in 40-50%. Infections are common and complicate transplant outcomes. Therefore, refractoriness, relapses and clonal evolution limit the success of IST in SAA, while graft rejection, GVHD, and infections limited the success of BMT in the clinic. Predictors for these complications would be of great value in the clinic since one could make more rational treatment decisions where patients were allocated to different treatment modalities based on their risk profile. For example, patients with high probability of response and low risk of relapse and clonal evolution would benefit more from IST, while those with low probability of hematologic response and high risk of recurrence and/or clonal evolution most likely to benefit from BMT. Based on this premise, we developed studies to investigate factors that could be associated with the success of IST in SAA. The main findings of three separate analysis on the subject showed: 1) children ( < 18 years) have a high response rate to IST (around 75%) with an excellent long-term survival rate among responders; 2) the absolute number of reticulocytes and lymphocytes pre-treatment correlates with hematologic response at 6 months after IST, and 3) telomere length is not associated with hematologic response, but, associated with the likelihood of relapse, clonal evolution, and overall survival after IST. These parameters may serve as a basis for future algorithms allowing for risk stratification for each individual patient allowing for better treatment allocation. With respect to the telomere length, it is likely that it not only represents a biological marker but that it is involved in the process of clonal evolution contributing to genomic instability in bone marrow cells leading to the development of myelodysplasia and leukemia
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Quiroga, Márcia Regina Silva. "Avaliação do quimerismo em pacientes com anemia aplástica severa adquirida, após 18 meses do transplante de células-tronco hematopoiéticas, submetidos a diferentes regimes de condicionamento." reponame:Repositório Institucional da UFPR, 2014. http://hdl.handle.net/1884/36311.

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Orientador: Prof. Dr. Ricardo Pasquini<br>Co-orientadora: Drª. Noemi Farah Pereira<br>Dissertação (mestrado) - Universidade Federal do Paraná, Setor de Ciências da Saúde, Programa de Pós-Graduação em Medicina Interna. Defesa : Curitiba, 01/08/2014<br>Inclui referências<br>Resumo: O resultado do transplante de células-tronco hematopoéticas (TCTH) é avaliado por meio da recuperação hematológica e da análise do quimerismo. Este estudo tem como objetivo avaliar os níveis de quimerismo em pacientes com Anemia Aplástica Severa (AAS) Adquirida com mais de 18 meses de acompanhamento pós-TCTH, que apresentaram recuperação hematológica parcial ou completa no sangue periférico. Foram analisados 104 pacientes com AAS transplantados no Hospital de Clínicas da Universidade Federal do Paraná e monitorados no período de 1987 a 2012. Os pacientes foram divididos em dois grupos, conforme o regime de condicionamento. Os pacientes do grupo I (n=55) receberam ciclofosfamida (CFA) isolada, 200 mg/Kg de peso corpóreo e os do grupo II (n=49) CFA, 120 mg/Kg de peso corpóreo, associada ao bussulfano (BUS), 12 mg/Kg de peso corpóreo. Cada grupo foi subdividido de acordo com os seguintes níveis de quimerismo em relação às células do doador: ?50%, entre 51 e 90% e >90%. A análise de quimerismo foi realizada pela amplificação de locos VNTRs/STRs com detecção dos fragmentos em gel de poliacrilamida e coloração com sais de prata (84 pacientes) até julho de 2009 e após esse período por eletroforese capilar em Analisador Genético de DNA (20 pacientes). Os níveis de quimerismo foram correlacionados com as variáveis pré-transplante (idade e sexo do paciente e intervalo entre o diagnóstico e o TCTH), variáveis do transplante (idade e sexo do doador e número de células infundidas) e variáveis pós-transplante (número de neutrófilos, plaquetas e dosagem de hemoglobina após 18 meses do transplante). A análise dos resultados mostrou associação entre os níveis de quimerismo e os tipos de condicionamento empregados (p<0,001). A recuperação autóloga (quimerismo ?50%) foi encontrada em 36,4% dos pacientes do grupo I e em nenhum dos pacientes do grupo II. Quimerismo entre 51 e 90% foi identificado em 11 pacientes (20,0%) do grupo I e em 5 pacientes (10,2%) do grupo II. Quimerismo >90% foi mais frequente no grupo II (89,8%) quando comparado ao grupo I (43,6%). As características pré-transplante (idade e sexo do paciente e intervalo entre o diagnóstico e o TCTH) e do transplante (idade e sexo do doador) não mostraram associação ao quimerismo em nenhum dos grupos. O maior número de células infundidas mostrou associação com níveis de quimerismo mais elevados nos pacientes do grupo I (p=0,013) e não apresentou associação nos do grupo II. A análise multivariada mostrou que o nível de quimerismo >90% está associado ao condicionamento com CFA+BUS (p<0,001) e ao maior número de células infundidas (p=0,009). A recuperação hematológica, avaliada com base no último hemograma disponível, mostrou associação entre o número mais elevado de neutrófilos (p=0,003) e de plaquetas (p<0,001) e maior grau de quimerismo nos pacientes do grupo I. A dosagem de hemoglobina não mostrou associação com o quimerismo nos dois regimes de condicionamento. Este estudo sugere que o condicionamento com CFA associada ao BUS e o maior número de células infundidas são fatores preditivos da evolução do enxerto alogênico de células-tronco hematopoéticas. Os resultados deste estudo reforçam que a recuperação autóloga da hematopoese depende da intensidade da imunossupressão exigida para cada caso e que a função imunossupressora da CFA isolada pode induzir a regeneração hematológica autóloga. Palavras-chave: Anemia Aplástica Severa. Transplante de células-tronco hematopoéticas. Condicionamento. Quimerismo.<br>Abstract: The outcome of hematopoietic stem cell transplantation is evaluated (HSCT) by hematologic recovery and chimerism analysis. The aim of this study is to assess the levels of chimerism in Acquired Severe Aplastic Anemia (SAA) patients with post-transplant follow up of 18 months or more, and that have showed partial or complete hematologic recovery in peripheral blood. A total of 104 SAA patients transplanted at the Hospital de Clínicas of the Universidade Federal do Paraná and monitored from 1987 to 2012 were analyzed. Patients were divided into two groups on the basis of the conditioning regimen. Group I (n=55) received only cyclophosphamide (CY) at 200mg/kg of body weight, while those of group II (n=49) received CY at 120mg/kg of body weight associated with busulfan (BUS) at 12mg/kg of body weight. Each group was then subdivided according to the following levels of chimerism in relation to the donor's cells: ?50%, from 51 to 90%, and >90%. Chimerism analysis was performed by amplification of VNTR/STR loci followed by detection of fragments in polyacrylamide silver stained gels (84 patients) until July of 2009 and, from then on, by capillary electrophoresis in DNA Genetic Analyser (20 patients). Levels of chimerism were correlated to pre-transplant (patient's age, sex, and time elapsed from diagnosis to HSCT), transplant (donor's age, sex, and number of infused cells) and post-transplant (number of neutrophils and platelets, and hemoglobin levels from18 months on after transplant) variables. Data analysis showed an association between the levels of chimerism and the different conditioning regimens (p<0.001). Autologous recovery (chimerism ?50%) was achieved by 36.4% of patients from group I and by none of those from group II. Chimerism ranging from 51 to 90% was identified in 11 patients (20.0%) from group I and in 5 patients (10.2%) from group II. Levels of chimerism >90% were more frequent in group II (89.8%) than in group I (43.6%). Pre-transplant (patient's age, sex, and time elapsed from diagnosis to HSCT) and transplant (donor's age and sex) characteristics did not show association with chimerism in either one of the groups. The largest number of infused cells showed association with higher levels of chimerism in patients from group I (p=0.013), but not with those in group II. Multivariate analysis indicated that the chimerism level >90% is associated with the conditioning regimen CY+BUS (p<0.001) and with the highest number of infused cells (p=0.009). Hematologic recovery evaluated on the basis of the last available blood count indicated an association of the largest number of neutrophils (p=0.003) and platelets (p<0.001) with the highest level of chimerism in patients from group I. Hemoglobin levels did not show any association with chimerism in either one of the groups. This study suggests that the conditioning regimen with CY and BUS as well as the highest number of infused cells are predictive factors of the establishment of hematopoietic stem cell allogeneic graft. The results of this study corroborate that hematopoietic autologous recovery relies on the intensity of immunosuppression needed for each case, and that the immunosuppressive function of CY alone can induce autologous hematologic recovery. Keywords: Severe Aplastic Anemia. Hematopoietic stem cell transplantation. Conditioning regimen. Chimerism.
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Books on the topic "Severe anemia"

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Two cases of severe anæmia with absence of hydrochloric acid in the gastric juice. s.n., 1985.

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White, Robert Allen. An inherited erythroid ankyrin deficiency in normoblastosis: A mutation causing severe hemolytic anemia in the mouse. 1989.

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Severe Anemia in Pregnancy: Report of a Workshop Held at the Institute of Child and Mother Health in Dhaka, Bangladesh. International Development Research Centre, 2000.

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Purandare, Amol, and Barbara A. Jantausch. Parvovirus. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190604813.003.0012.

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Parvovirus B19 is a common infection in humans that occurs worldwide. Parvovirus B19 is transmitted through exposure to respiratory droplets, blood, and blood products, and through mother-to-child transmission (MTCT) in utero. Intrauterine parvovirus B19 infection is a rare occurrence during pregnancy but can result in significant morbidity and mortality for the fetus, including severe fetal anemia and nonimmune fetal hydrops (NIFH). Intrauterine transfusion can be successful in treating fetal anemia. Neurodevelopmental impairment has been reported in infants with congenital infection who have received intrauterine transfusion (IUT). Future research on the development of antiviral agents for the treatment of parvovirus B19 infection in pregnant women is needed, along with the development of a parvovirus B19 vaccine. Longitudinal studies to evaluate neurodevelopmental outcome of infants with a history of congenital parvovirus B19 infection are needed in order to facilitate the optimal evaluation and management of these infants.
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Ng, Ann, and Erin S. Williams. Sickle Cell Disease. Edited by Erin S. Williams, Olutoyin A. Olutoye, Catherine P. Seipel, and Titilopemi A. O. Aina. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190678333.003.0033.

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Sickle cell anemia (sickle cell disease) is a common hemoglobinopathy with anywhere from 90,000 to 100,000 Americans affected. This chronic condition has a predominance in populations of African descent, occurring in approximately 1 out of 365 African American births, compared to 1 out of 16,300 Hispanic births. The sickle cell trait can be detected in 1 of 13 African American births. One of the most common complications associated with sickle cell anemia, vaso-occlusive crises by sickled cells, results in severe pain. Other issues associated with this condition include acute chest syndrome, lung infections, end organ damage, and stroke. With improvements in the management and prevention of pain crises, infection, and other systemic involvement, these patients are living longer, thus increasing the potential for surgical needs. Whether it is for routine surgeries or surgeries that are due to the natural history of the disease; the pediatric anesthesiologist must be knowledgeable of the management of these patients in order to prevent morbidity and mortality.
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Ng, Dominic S. Familial Lecithin Cholesterol Acyl Transferase Deficiency Syndromes. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199972135.003.0034.

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Lecithin cholesterol ester transferase (LCAT) is the sole enzyme in the circulation that mediates the esterification of free cholesterol (FC) to cholesterol ester (CE) in lipoproteins. Mutations in the LCAT gene result in one of two clinical syndromes: complete LCAT deficiency syndrome, and “fish eye disease.” The former is characterized by a broad spectrum of clinical features, including profound high-density lipoprotein (HDL) deficiency, hypertriglyceridemia, corneal opacities, anemia, neuropathies, and nephropathy. In contrast, fish eye disease patients develop severe HDL deficiency and severe corneal opacities, but the nervous system and kidneys are typically unaffected. Whether there is a predisposition to accelerated coronary heart disease with LCAT deficiency remains controversial. Currently, severe corneal opacities may be treated with corneal transplant. Only anecdotal evidence is available for preventive measures of progressive renal complications. LCAT replacement therapies are under investigation.
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Kocher, Bharati. Diverticulitis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199976805.003.0033.

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Diverticular disease includes two conditions: diverticulosis and diverticulitis. Both involve saclike protrusions of the mucosal and submucosal walls, typically in the colon. Diverticulosis is the presence of multiple diverticula, which may or may not be symptomatic. Symptomatic diverticulosis presents with indistinct symptoms and, less commonly, with severe symptoms, such as slow bleeding (causing anemia) or rapid bleeding (causing frank hematochezia and even hemodynamic instability). Diverticulitis is acute or chronic inflammation of the diverticula, possibly leading to abscesses and even perforation. Classic diverticulitis includes fever, leukocytosis, and left-sided abdominal pain, with localized tenderness and guarding. Incidence of disease increases with age. CT is the gold standard for diagnosis, which rules out other abdominal pathology and detects any diverticular complications. An outpatient course of oral antibiotics is prescribed for uncomplicated diverticulitis in an immunocompetent patient. Indications for surgery are generalized peritonitis or large abscesses that cannot be drained.
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Supply of Blood for Transfusion in Latin America and Caribbean Countries 2016-2017. Organización Panamericana de la Salud, 2020. http://dx.doi.org/10.37774/9789275121719.

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Blood transfusions are necessary to improve or save the lives of children with severe anemia, mothers in obstetric emergencies, patients with hemoglobinopathies, cancer patients, transplant patients, patients with chronic age-related diseases, such as bleeding caused by vascular problems and orthopedic surgeries, people injured in accidents, among other causes. Supply and access to safe blood for transfusions are closely related to the organization and degree of development of blood services, with the governance and participation of society through voluntary unpaid donation. Since 2004, the Pan American Health Organization (PAHO) has been collecting and publishing indicators related to blood supply in the countries of Latin America and the Caribbean. In 2014, the countries of the Region of the Americas reaffirmed their commitment to universal health through the approval of the Action Plan for Universal Access to Safe Blood 2014-2019, approved by the 53rd Directing Council held in October 2014 (CD53.6), this plan promotes universal access to safe blood for transfusion in the region, through unpaid voluntary donations, the organization of blood services, the implementation of quality and safety standards and the implementation of governance actions. The data presented here allows monitoring and reporting on progress and limitations in the implementation of the Action Plan for Universal Access to Safe Blood. Furthermore, it is hoped that these data will promote the analysis and evaluation of the indicators at the national and subregional levels, and that strategies that improve blood safety and accessibility to transfusions will be strengthened or modified. The information was provided by the authorities of the countries and corresponds to the years 2016 and 2017.
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Bricker, Leanne, Julian N. Robinson, and Baskaran Thilaganathan, eds. Management of Multiple Pregnancies. Cambridge University Press, 2022. http://dx.doi.org/10.1017/9781108915038.

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Multiple pregnancies are associated with higher risks for both mother and babies. Women with multiple pregnancies have an increased risk of miscarriage, anemia, hypertensive disorders, haemorrhage, and postnatal illness. These pregnancies are more likely to need an operative delivery, and maternal mortality is generally 2.5 times that of singleton births. Fetuses are at increased risk for anatomic and genetic anomalies, growth abnormalities, prematurity, and several physiological problems related to monochorionicity. This book provides a much needed, up-to-date guide to the management of multiple pregnancies. Presented with a uniform approach to all chapters, information is easily navigable, evidence-based, and highly practical. Heavily illustrated, particularly with ultrasound images – the cornerstone of management of multiple pregnancies - this book will appeal to obstetricians and specialists in maternal-fetal medicine, midwives and ultrasonographers and will improve outcomes for mothers and babies.
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Reed, Wornie L. Health and Medical Care of African-Americans. Praeger, 1993. http://dx.doi.org/10.5040/9798400662249.

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African-Americans suffer disproportionately to their percentage of the population from ill health and poor or non-existent medical care. Because health and illness are related to social status, only a change in economic and social policies will remedy the sorrowful health and care situation carefully detailed and substantiated in this book. Reed and his colleagues identify and show the enormity of the several health problems of particular concern to the black population. These include adverse birth outcomes, increasing cancer rates, lead poisoning, and the high rate of homicide among African-Americans. Sickle Cell Anemia with its special problems for the black community is addressed, as is the widespread use of alcohol and other substances. Finally, the AIDS epidemic's impact on the African-American population is described and the implications stated. The authors leave no doubt that decisive action is warranted on a large scale.
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Book chapters on the topic "Severe anemia"

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Blatt, Julie, April M. Evans, and Jessica Benjamin-Eze. "Severe Anemia." In Manual for Night-Time Emergencies for Pediatric Hematology-Oncology Fellows. CRC Press, 2024. http://dx.doi.org/10.1201/9781003473701-13.

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Chandekar, Sushama A., Kusum D. Jashnani, and Girish Rajadhyaksha. "Severe Anemia and Pregnancy." In Maternal Mortality - Lessons Learnt from Autopsy. Springer Nature Singapore, 2022. http://dx.doi.org/10.1007/978-981-19-3420-9_34.

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Le Roux, Peter. "Treatment of Anemia." In Controversies in Severe Traumatic Brain Injury Management. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-89477-5_12.

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de Latour, Régis Peffault, Antonio Risitano, and Carlo Dufour. "Severe Aplastic Anemia and PNH." In The EBMT Handbook. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-030-02278-5_77.

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Storb, R., and K. Doney. "Therapy of Severe Aplastic Anemia." In 11th Annual meeting of the EBMT. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-662-40457-7_42.

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Singh, Kavita N., and Jitendra Bhargava. "Severe Anemia in Critically Ill Obstetric Patients." In Principles of Critical Care in Obstetrics. Springer India, 2016. http://dx.doi.org/10.1007/978-81-322-2686-4_15.

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Sharma, Sanjeev Kumar. "Stem Cell Transplant for Severe Aplastic Anemia." In Basics of Hematopoietic Stem Cell Transplant. Springer Nature Singapore, 2023. http://dx.doi.org/10.1007/978-981-19-5802-1_27.

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Beliaev, A. M., and S. J. Allen. "4. Management and outcomes in severe anemia." In Human Health Handbooks. Wageningen Academic Publishers, 2016. http://dx.doi.org/10.3920/978-90-8686-822-3_4.

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Hanna, Rabi. "Hematopoietic Cell Transplant for Severe Aplastic Anemia." In Blood and Marrow Transplant Handbook. Springer International Publishing, 2021. http://dx.doi.org/10.1007/978-3-030-53626-8_23.

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Stoute, José A. "Role of Complement in Severe Malarial Anemia." In Complement Activation in Malaria Immunity and Pathogenesis. Springer International Publishing, 2018. http://dx.doi.org/10.1007/978-3-319-77258-5_3.

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Conference papers on the topic "Severe anemia"

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Lampl, C., and P. M. Joseph. "Not Always the Antibiotics: Severe Hemolytic Anemia in Cystic Fibrosis." In American Thoracic Society 2020 International Conference, May 15-20, 2020 - Philadelphia, PA. American Thoracic Society, 2020. http://dx.doi.org/10.1164/ajrccm-conference.2020.201.1_meetingabstracts.a3157.

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Aydın, İlayda, and Üule Ünal Cangül. "PP-061 Severe vitamin B12 deficiency presenting with hemolytic anemia." In 11th Europaediatrics Congress, Antalya, Türkiye, 17 – 21 April 2024. BMJ Publishing Group Ltd, 2024. http://dx.doi.org/10.1136/bmjpo-2024-epac.205.

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Landim, Joaquim Ivo Vasques Dantas, Heitor Furlan Giordano, Victor Caires Tadeu, et al. "Severe glucocorticoid resistant autoimmune hemolytic anemia secondary to systemic lupus erythematosus." In XXXIX Congresso Brasileiro de Reumatologia. Sociedade Brasileiro de Reumatologia, 2022. http://dx.doi.org/10.47660/cbr.2022.2071.

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Umrani, Rohan, Hiral Amin, Ashley Pinckney, Charles Yang, and Daniel Tran. "Mixed Beri’s: High Output Heart Failure from Severe Anemia and Thiamine Deficiency." In 27th Annual Rowan-Virtua Research Day. Rowan University Libraries, 2023. https://doi.org/10.31986/issn.2689-0690_rdw.stratford_research_day.12_2023.

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Background: Severe anemia and thiamine deficiency can independently result in high output heart failure (HOHF) through different mechanisms. Data on the threshold and timeframe at which these etiologies may precipitate HOHF is unclear. Case: A 63-year-old male with alcohol use disorder consuming 18 drinks/week presented with progressive shortness of breath and lower extremity edema for a few months. Physical exam revealed tachycardia, anasarca, jugular venous distension, Lancisi’s sign, and a midsystolic murmur. Echocardiogram demonstrated EF of 30-35%, dilated left ventricle, and cardiac index of 4.2L/min/m2. Decision-Making: Heart failure can independently precipitate anemia while severe alcoholism can induce myelosuppression and promote risk for bleeding. Thus, iron studies and endoscopy are crucial to elucidating the underlying pathophysiology to further direct therapy. Labs demonstrated hemoglobin 3.0 g/dL (baseline 11 g/dL 7 months prior), ferritin 5 ng/mL, thiamine 26 nmol/L, and hemoccult positive stool while colonoscopy revealed bleeding diverticulosis and hemorrhoids, thereby confirming critical iron deficiency anemia from lower GI bleed and thiamine deficiency. Cardiac catheterization ruled out ischemic cause of his new onset cardiomyopathy, supporting the diagnosis of HOHF in the setting of severe anemia and concomitant cardiac Beri Beri. Our patient was treated with parental thiamine infusions, intravenous iron sucrose, blood transfusions, and gradually diuresed with bumetanide. Repeat evaluation of his left ventricular function 3 months later showed recovery of EF to 50-55%. Conclusion: HOHF can generally be reversed with etiology-directed treatments. In chronic alcoholic patients, we advocate for routine testing of whole blood thiamine to assess for occult vitamin deficiency to determine whether supplementation may assist in therapy. The relative independent contributions or synergistic effects of iron deficiency and thiamine deficiency on HOHF remains unknown, but thorough investigations are essential to ensure recovery of cardiac function.
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Halas, Ryan, Garrett Koon, Pediatrics Resident, and Patrick Jones. "Severe Iron Deficiency Anemia from a Rare Cause in a Pediatric Patient." In Selection of Abstracts From NCE 2016. American Academy of Pediatrics, 2018. http://dx.doi.org/10.1542/peds.141.1_meetingabstract.702.

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Biegelmeyer, Erika, Julia Boechat Farani, Evelise Mileski do Amaral Berlet, Marcel Mathias Villaça, Marília Voges de Souza, and Cristina Ferreira Rabelo. "Intestinal vasculitis, mesenteritis, microangiopathic anemia and alveolar hemorrhage: a severe presentation of dermatomyositis." In SBR 2021 Congresso Brasileiro de Reumatologia. Sociedade Brasileira de Reumatologia, 2021. http://dx.doi.org/10.47660/cbr.2021.2089.

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Reed, Anna, and Christine Karwowski. "Child with Pulmonary Hemorrhage and Severe Anemia: An Unusual Presentation of Celiac Disease." In AAP National Conference & Exhibition Meeting Abstracts. American Academy of Pediatrics, 2021. http://dx.doi.org/10.1542/peds.147.3_meetingabstract.858.

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Zahra, Sayyeda, Negar Moheb, and Christopher Melinosky. "Bilateral Ischemic Optic Neuropathy as the First Manifestation of Severe Anemia (P1-9.008)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000204295.

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Amlie-Lefond, Catherine, and Kristin Maher. "Cow’s Milk Anemia In Childhood Manifesting As Severe Cerebral Venous Sinus Thrombosis (P14-9.004)." In 2023 Annual Meeting Abstracts. Lippincott Williams & Wilkins, 2023. http://dx.doi.org/10.1212/wnl.0000000000202304.

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Mönkemüller, K., K. M. Pawlak, T. Kröner, L. Fry, and A. Martinez-Alcalá. "Small Bowel B-Cell Lymphoma Presenting as Autoimmune Hemolytic Anemia and Severe Obscure Gastrointestinal Bleeding." In ESGE Days 2023. Georg Thieme Verlag KG, 2023. http://dx.doi.org/10.1055/s-0043-1765967.

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Reports on the topic "Severe anemia"

1

Harris, Juliana V. The Fate of the Red Cells: Insights from Two Models of Severe Malarial Anemia. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ad1013332.

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2

Schat, Karel Antoni, Irit Davidson, and Dan Heller. Chicken infectious anemia virus: immunosuppression, transmission and impact on other diseases. United States Department of Agriculture, 2008. http://dx.doi.org/10.32747/2008.7695591.bard.

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1. Original Objectives. The original broad objectives of the grant were to determine A) the impact of CAV on the generation of cytotoxic T lymphocytes (CTL) to reticuloendotheliosis virus (REV) (CU), B). the interactions between chicken anemia virus (CAV) and Marek’s disease virus (MDV) with an emphasis on horizontal spread of CAV through feathers (KVI), and C) the impact of CAV infection on Salmonella typhimurium (STM) (HUJI). During the third year and the one year no cost extension the CU group included some work on the development of an antigen-antibody complex vaccine for CAV, which was partially funded by the US Poultry and Egg Association. 2. Background to the topic. CAV is a major pathogen causing clinical disease if maternal antibody-free chickens are infected vertically or horizontally between 1 and 14 days of age. Infection after 3 weeks of age when maternal antibodies are not longer present can cause severe subclinical immunosuppression affecting CTL and cytokine expression. The subclinical immunosuppression can aggravate many diseases including Marek’s disease (MD) and several bacterial infections. 3. Major conclusions and achievements. The overall project contributed in the following ways to the knowledge about CAV infection in poultry. As expected CAV infections occur frequently in Israel causing problems to the industry. To control subclinical infections vaccination may be needed and our work indicates that the development of an antigen-antibody complex vaccine is feasible. It was previously known that CAV can spread vertically and horizontally, but the exact routes of the latter had not been confirmed. Our results clearly show that CAV can be shed into the environment through feathers. A potential interaction between CAV and MD virus (MDV) in the feathers was noted which may interfere with MDV replication. It was also learned that inoculation of 7-day-old embryos causes growth retardation and lesions. The potential of CAV to cause immunosuppression was further examined using CTL responses to REV. CTL were obtained from chickens between 36 and 44 days of age with REV and CAV given at different time points. In contrast to our earlier studies, in these experiments we were unable to detect a direct impact of CAV on REV-specific CTL, perhaps because the CTL were obtained from older birds. Inoculation of CAV at one day of age decreased the IgG antibody responses to inactivated STM administered at 10 days of age. 4. Scientific and Agricultural Implications The impact of the research was especially important for the poultry industry in Israel. The producers have been educated on the importance of the disease through the many presentations. It is now well known to the stakeholders that CAV can aggravate other diseases, decrease productivity and profitability. As a consequence they monitor the antibody status of the breeders so that the maternal antibody status of the broilers is known. Also vaccination of breeder flock that remain antibody negative may become feasible further reducing the negative impact of CAV infection. Vaccination may become more important because improved biosecurity of the breeder flocks to prevent avian influenza and Salmonella may delay the onset of seroconversion for CAV by natural exposure resulting in CAV susceptible broilers lacking maternal antibodies. Scientifically, the research added important information on the horizontal spread of CAV through feathers, the interactions with Salmonella typhimurium and the demonstration that antigen-antibody complex vaccines may provide protective immunity.
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3

Wang, Yan, Zhenzhen Wang, Jie Xu, et al. Efficacy and safety of haploidentical hematopoietic stem cell transplantation on severe aplastic anemia using busulfan-based myeloablative regimenA protocol for a Bayesian network meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.1.0116.

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4

Rosenblatt, David, Diether Beuermann, Sylvia Dohnert, et al. aribbean Economics Quarterly: Volume 13, Issue 3: Innovation for Faster Economic Growth in the Caribbean: Are We There Yet? Inter-American Development Bank, 2024. https://doi.org/10.18235/0013347.

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As discussed in several editions of the Caribbean Economics Quarterly, the Caribbean has seen anemic growth, high volatility, and low productivity over the past few decades. This edition of the Caribbean Economics Quarterly focuses on innovation which has been proven to be a key driver of productivity growth across economies. The underlying dataset for the analysis is the Innovation, Firm Performance, and Gender (IFPG) Survey conducted by the Compete Caribbean Partnership Facility in 2020. The survey was administered to 1,979 formal firms across 13 Caribbean countries. The report summarizes information from the innovation chapter of the recently released IDB book Are We There Yet? The Path toward Sustainable Private Sector Development in the Caribbean, and it provides a unique country-specific perspective for each of the six countries of the IDBs Caribbean Department - The Bahamas, Barbados, Guyana, Jamaica, Suriname and Trinidad and Tobago.
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5

Lazonick, William. Investing in Innovation: A Policy Framework for Attaining Sustainable Prosperity in the United States. Institute for New Economic Thinking Working Paper Series, 2022. http://dx.doi.org/10.36687/inetwp182.

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“Sustainable prosperity” denotes an economy that generates stable and equitable growth for a large and growing middle class. From the 1940s into the 1970s, the United States appeared to be on a trajectory of sustainable prosperity, especially for white-male members of the U.S. labor force. Since the 1980s, however, an increasing proportion of the U.S labor force has experienced unstable employment and inequitable income, while growing numbers of the business firms upon which they rely for employment have generated anemic productivity growth. Stable and equitable growth requires innovative enterprise. The essence of innovative enterprise is investment in productive capabilities that can generate higher-quality, lower-cost goods and services than those previously available. The innovative enterprise tends to be a business firm—a unit of strategic control that, by selling products, must make profits over time to survive. In a modern society, however, business firms are not alone in making investments in the productive capabilities required to generate innovative goods and services. Household units and government agencies also make investments in productive capabilities upon which business firms rely for their own investment activities. When they work in a harmonious fashion, these three types of organizations—household units, government agencies, and business firms—constitute “the investment triad.” The Biden administration’s Build Back Better agenda to restore sustainable prosperity in the United States focuses on investment in productive capabilities by two of the three types of organizations in the triad: government agencies, implementing the Infrastructure Investment and Jobs Act, and household units, implementing the yet-to-be-passed American Families Act. Absent, however, is a policy agenda to encourage and enable investment in innovation by business firms. This gaping lacuna is particularly problematic because many of the largest industrial corporations in the United States place a far higher priority on distributing the contents of the corporate treasury to shareholders in the form of cash dividends and stock buybacks for the sake of higher stock yields than on investing in the productive capabilities of their workforces for the sake of innovation. Based on analyzes of the “financialization” of major U.S. business corporations, I argue that, unless Build Back Better includes an effective policy agenda to encourage and enable corporate investment in innovation, the Biden administration’s program for attaining stable and equitable growth will fail. Drawing on the experience of the U.S. economy over the past seven decades, I summarize how the United States moved toward stable and equitable growth from the late 1940s through the 1970s under a “retain-and-reinvest” resource-allocation regime at major U.S. business firms. Companies retained a substantial portion of their profits to reinvest in productive capabilities, including those of career employees. In contrast, since the early 1980s, under a “downsize-and-distribute” corporate resource-allocation regime, unstable employment, inequitable income, and sagging productivity have characterized the U.S. economy. In transition from retain-and-reinvest to downsize-and-distribute, many of the largest, most powerful corporations have adopted a “dominate-and-distribute” resource-allocation regime: Based on the innovative capabilities that they have previously developed, these companies dominate market segments of their industries but prioritize shareholders in corporate resource allocation. The practice of open-market share repurchases—aka stock buybacks—at major U.S. business corporations has been central to the dominate-and-distribute and downsize-and-distribute regimes. Since the mid-1980s, stock buybacks have become the prime mode for the legalized looting of the business corporation. I call this looting process “predatory value extraction” and contend that it is the fundamental cause of the increasing concentration of income among the richest household units and the erosion of middle-class employment opportunities for most other Americans. I conclude the paper by outlining a policy framework that could stop the looting of the business corporation and put in place social institutions that support sustainable prosperity. The agenda includes a ban on stock buybacks done as open-market repurchases, radical changes in incentives for senior corporate executives, representation of workers and taxpayers as directors on corporate boards, reform of the tax system to reward innovation and penalize financialization, and, guided by the investment-triad framework, government programs to support “collective and cumulative careers” of members of the U.S. labor force. Sustained investment in human capabilities by the investment triad, including business firms, would make it possible for an ever-increasing portion of the U.S. labor force to engage in the productive careers that underpin upward socioeconomic mobility, which would be manifested by a growing, robust, and hopeful American middle class.
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6

Iron supplementation: Knowledge, perceptions, and usage among pregnant women in rural India. Population Council, 1997. http://dx.doi.org/10.31899/rh1997.1021.

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In India, severe anemia has been one of the causes of high maternal mortality and death of newborns and infants due to low birthweight. The prevalence of anemia among women ages 15–44 is extremely high. The Government of India has provided iron and folic acid tablets (IFA) as a prophylaxis against nutritional anemia among pregnant women as part of the Child Survival and Safe Motherhood program and will continue to do so in the Reproductive and Child Health package. This paper presents findings of both qualitative and quantitative research conducted among pregnant women to investigate the extent of distribution and use, information provision, and knowledge and perceptions regarding IFA tablets and reasons for nonuse. The study shows that consumption rate of IFA tablets is high if women are knowledgeable and have positive experiences after taking the tablets. Findings suggest that more accurate and complete information should be provided to pregnant women while distributing IFA tablets along with health and nutritional education messages. At the same time, follow-up visits and counseling are essential to address symptoms that are not related to IFA.
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