Academic literature on the topic 'Siah'

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Journal articles on the topic "Siah"

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Li, Qiang, Ping Wang, Keqiang Ye, and Hua Cai. "Central role of SIAH inhibition in DCC-dependent cardioprotection provoked by netrin-1/NO." Proceedings of the National Academy of Sciences 112, no. 3 (January 5, 2015): 899–904. http://dx.doi.org/10.1073/pnas.1420695112.

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Deleted in colorectal cancer (DCC), a large transmembrane receptor of netrin-1, is critical for mediating netrin-1’s cardioprotective function. In the present study we investigated novel mechanisms underlying netrin-1–induced, rapid, and feed-forward up-regulation of DCC, which is believed to sustain nitric oxide (NO) production to potentiate cardioprotection. Intriguingly, NO markedly reduced expression of the E3 ubiquitin ligase seven in absentia homolog (SIAH) that is specific for regulation of protesome-dependent DCC degradation, resulting in accumulation of DCC. The two SIAH isoforms compensate for each other when one is repressed; inhibition of both SIAH1 and SIAH2 using combined siRNAs significantly reduced infarct size while improving cardiac function after ischemia/reperfusion injury of the heart. This effect was absent in DCC-deficient mice. Moreover, in vivo RNAi inhibition of SIAH1/2 further augmented netrin-1’s cardioprotective function. In summary, these data identify a novel therapeutic target of SIAH in facilitating NO/netrin-1–dependent cardioprotection, using the DCC receptor. Combination of netrin-1 and SIAH RNAi may prove to be a substantially effective therapy for myocardial infarction.
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Frew, Ian J., Ross A. Dickins, Andrew R. Cuddihy, Merci Del Rosario, Christoph Reinhard, Matthew J. O'Connell, and David D. L. Bowtell. "Normal p53 Function in Primary Cells Deficient for Siah Genes." Molecular and Cellular Biology 22, no. 23 (December 1, 2002): 8155–64. http://dx.doi.org/10.1128/mcb.22.23.8155-8164.2002.

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ABSTRACT Overexpression studies have suggested that Siah1 proteins may act as effectors of p53-mediated cellular responses and as regulators of mitotic progression. We have tested these hypotheses using Siah gene knockout mice. Siah1a and Siah1b were not induced by activation of endogenous p53 in tissues, primary murine embryonic fibroblasts (MEFs) or thymocytes. Furthermore, primary MEFs lacking Siah1a, Siah1b, Siah2, or both Siah2 and Siah1a displayed normal cell cycle progression, proliferation, p53-mediated senescence, and G1 phase cell cycle arrest. Primary thymocytes deficient for Siah1a, Siah2, or both Siah2 and Siah1a, E1A-transformed MEFs lacking Siah1a, Siah1b, or Siah2, and Siah1b-null ES cells all underwent normal p53-mediated apoptosis. Finally, inhibition of Siah1b expression in Siah2 Siah1a double-mutant cells failed to inhibit cell division, p53-mediated induction of p21 expression, or cell cycle arrest. Our loss-of-function experiments do not support a general role for Siah genes in p53-mediated responses or mitosis.
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Hu, Gang, and Eric R. Fearon. "Siah-1 N-Terminal RING Domain Is Required for Proteolysis Function, and C-Terminal Sequences Regulate Oligomerization and Binding to Target Proteins." Molecular and Cellular Biology 19, no. 1 (January 1, 1999): 724–32. http://dx.doi.org/10.1128/mcb.19.1.724.

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ABSTRACT The Drosophila seven in absentia (sina) gene was initially discovered because its inactivation leads to R7 photoreceptor defects. Recent data indicate that Sina binds to the Sevenless pathway protein Phyllopod, and together they mediate degradation of Tramtrack, a transcriptional repressor of R7 cell fate. Independent studies have shown that Sina and its highly related mammalian homologues Siah-1 and Siah-2 bind to the DCC (deleted in colorectal cancer) protein and promote its proteolysis via the ubiquitin-proteasome pathway. To determine the roles of mammalian Siahs in proteolysis and their interactions with target proteins, we sought to define Siah-1 domains critical for regulation of DCC. Mutant Siah-1 proteins, harboring missense mutations in the carboxy (C)-terminal domain analogous to those present in Drosophila sinaloss-of-function alleles, failed to promote DCC degradation. Point mutations and deletion of the amino (N)-terminal RING finger domain of Siah-1 abrogated its ability to promote DCC proteolysis. In the course of defining Siah-1 sequences required for DCC degradation, we found that Siah-1 is itself rapidly degraded via the proteasome pathway, and RING domain mutations stabilized the Siah-1 protein. Siah-1 was found to oligomerize with itself and other Sina and Siah proteins via C-terminal sequences. Finally, evidence that endogenous Siah-1 regulates DCC proteolysis in cells was obtained through studies of an apparent dominant negative mutant of Siah-1, as well as via an antisense approach. The data indicate that the Siah-1 N-terminal RING domain is required for its proteolysis function, while the C-terminal sequences regulate oligomerization and binding to target proteins, such as DCC.
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Gupte, Girish R., and R. Prasad. "Geometries and Vibrational Spectra of Small Hydrogenated Silicon Clusters." International Journal of Modern Physics B 12, no. 16n17 (July 10, 1998): 1737–50. http://dx.doi.org/10.1142/s021797929800096x.

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We report a systematic study of ground state structures, vibrational spectra, cohesive energies and HOMO-LUMO gaps of small Si n H clusters (n=3, 10) based on the nonorthogonal tight-binding molecular dynamics scheme. The ground state structures have been obtained by using simulated annealing. In particular, we focus on how the addition of a hydrogen atom affects the ground state geometry and the stability of a Si n cluster. We find that hydrogen either enters into the surface of the cluster or occupies a position outside the cluster. In the first case, it drastically distorts the cluster, while in the latter, there is very little distortion. We find that in some cases Si n H cluster has some resemblance with Si n+1 cluster. We also find that hydrogen can form bonds with more than one silicon atom. Our calculation indicates that SiH, Si3H and Si5H will be more stable and Si4H , Si6H , Si7H , Si9H and Si10H will be less stable clusters.
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Frew, Ian J., Vicki E. Hammond, Ross A. Dickins, Julian M. W. Quinn, Carl R. Walkley, Natalie A. Sims, Ralf Schnall, et al. "Generation and Analysis of Siah2 Mutant Mice." Molecular and Cellular Biology 23, no. 24 (December 15, 2003): 9150–61. http://dx.doi.org/10.1128/mcb.23.24.9150-9161.2003.

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ABSTRACT Siah proteins function as E3 ubiquitin ligase enzymes to target the degradation of diverse protein substrates. To characterize the physiological roles of Siah2, we have generated and analyzed Siah2 mutant mice. In contrast to Siah1a knockout mice, which are growth retarded and exhibit defects in spermatogenesis, Siah2 mutant mice are fertile and largely phenotypically normal. While previous studies implicate Siah2 in the regulation of TRAF2, Vav1, OBF-1, and DCC, we find that a variety of responses mediated by these proteins are unaffected by loss of Siah2. However, we have identified an expansion of myeloid progenitor cells in the bone marrow of Siah2 mutant mice. Consistent with this, we show that Siah2 mutant bone marrow produces more osteoclasts in vitro than wild-type bone marrow. The observation that combined Siah2 and Siah1a mutation causes embryonic and neonatal lethality demonstrates that the highly homologous Siah proteins have partially overlapping functions in vivo.
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Della, N. G., P. V. Senior, and D. D. Bowtell. "Isolation and characterisation of murine homologues of the Drosophila seven in absentia gene (sina)." Development 117, no. 4 (April 1, 1993): 1333–43. http://dx.doi.org/10.1242/dev.117.4.1333.

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The seven in absentia gene (sina) is required for formation of the R7 photoreceptor cell in the developing eye of Drosophila melanogaster. The sina protein contains a putative zinc finger domain and localises to the cell nucleus in Drosophila. We report here the identification of a family of genes in the mouse (designated Siah) with extensive sequence homology to Drosophila sina. The Siah genes fall into two main groups: Siah-1, which consists of four closely related members, two of which appear to be functional, and Siah-2, which contains a single functional member. The predicted Siah proteins show an unusually high degree of conservation with sina over the majority of their lengths, diverging significantly only at their amino terminal ends. The Siah-1 and Siah-2 genes are widely expressed at a low level in the embryo and adult. Analysis of Siah-2 by hybridisation histochemistry shows that it is expressed at a higher level in a restricted number of sites during development, including the olfactory epithelium, retina, forebrain and proliferating cartilage of developing bone. The striking degree of sequence homology observed between the Drosophila and murine genes implies strong conservation pressure on the Siah genes and suggests that they play a significant role in vertebrate development.
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Briant, Douglas J., Derek F. Ceccarelli, and Frank Sicheri. "I Siah Substrate!" Structure 14, no. 4 (April 2006): 627–28. http://dx.doi.org/10.1016/j.str.2006.03.003.

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Moghayedi, Alireza, Bankole Awuzie, Temitope Omotayo, Karen Le Jeune, Mark Massyn, Christiana Okobi Ekpo, Manfred Braune, and Paimaan Byron. "A Critical Success Factor Framework for Implementing Sustainable Innovative and Affordable Housing: A Systematic Review and Bibliometric Analysis." Buildings 11, no. 8 (July 23, 2021): 317. http://dx.doi.org/10.3390/buildings11080317.

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The actualization of affordable housing remains a challenge. This challenge is exacerbated by the increasing societal demand for the incorporation of sustainability principles into such housing types to improve levels of occupant health and well-being whilst avouching the desired levels of affordability. Innovative technologies and practices have been described as beneficial to the effectuation of sustainable affordable housing. However, knowledge concerning the deployment of innovative technologies and practices in sustainable affordable housing (sustainable, innovative, affordable housing—SIAH) delivery remains nascent. Consequently, there is a lack of a common ontology among stakeholders concerning how to realize SIAH. This study aims to contribute toward the development of this body of knowledge through the establishment of the critical success factors (CSFs) for effective SIAH implementation. To achieve this objective, a systematic review and bibliometric analysis focusing on a juxtaposition of sustainable, innovative and affordable housing concepts was carried out based on the relevant literature. This led to the identification and clustering of CSFs for these housing concepts at individual levels and as a collective (SIAH). The findings of the study consisted of the establishment of four distinct yet interrelated facets through which SIAH can be achieved holistically, namely, housing design, house element, housing production method and housing technology. A total of 127 CSFs were found to be aligned to these facets, subsequently clustered, and conclusively used for the development of a SIAH CSF framework. The most frequently occurring CSFs with predominant interconnections were the utilization of energy-efficient systems/fittings, tenure security, a comfortable and healthy indoor environment, affordable housing price in relation to income and using water-efficient systems/fittings CSFs, and establishing the emergent SIAH CSF framework. The framework in this study is useful in the documentation of SIAH features for construction projects and further studies into SIAH CSFs.
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Samartsev, V. A., V. A. Gavrilov, and B. S. Pushkarev. "INTRAABDOMINAL HYPERTENSION SYNDROME: CURRENT STATE OF THE PROBLEM." Surgical practice, no. 2 (September 10, 2020): 35–42. http://dx.doi.org/10.38181/2223-2427-2020-2-35-42.

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Syndrome of intra-abdominal hypertension (SIAH) is a dangerous complication that can occur in patients with a surgical profile after surgery in the abdominal cavity. Only in the past 25 years has the SIAH been recognized. According to W. Ertel et al., In one third of patients with peritonitis, pancreatic necrosis, injuries of the abdominal cavity and after extensive surgery, there is an increase in intra-abdominal pressure (IAP), while SIAH develops in 5.5% of such patients. Mortality in SIAH is 42–68%. The reason for the development of so many deaths is the presence of diagnostic, preventive and therapeutic measures, as well as differences in the interpretation of terms. Today, much attention is paid to the study of SIAH, official attempts are being made to standardize and define terms and recommended methods of treatment, but this problem needs further study. The review presents data on the current state of the problem, current trends in the diagnosis, prevention and treatment of patients with IAP and SIAH, their use in practical research.
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Poh, Wee-Han, Jianqing Lin, Brendan Colley, Nicolai Müller, Boon Chong Goh, David Schleheck, Abbas El Sahili, et al. "The SiaABC threonine phosphorylation pathway controls biofilm formation in response to carbon availability in Pseudomonas aeruginosa." PLOS ONE 15, no. 11 (November 6, 2020): e0241019. http://dx.doi.org/10.1371/journal.pone.0241019.

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The critical role of bacterial biofilms in chronic human infections calls for novel anti-biofilm strategies targeting the regulation of biofilm development. However, the regulation of biofilm development is very complex and can include multiple, highly interconnected signal transduction/response pathways, which are incompletely understood. We demonstrated previously that in the opportunistic, human pathogen P. aeruginosa, the PP2C-like protein phosphatase SiaA and the di-guanylate cyclase SiaD control the formation of macroscopic cellular aggregates, a type of suspended biofilms, in response to surfactant stress. In this study, we demonstrate that the SiaABC proteins represent a signal response pathway that functions through a partner switch mechanism to control biofilm formation. We also demonstrate that SiaABCD functionality is dependent on carbon substrate availability for a variety of substrates, and that upon carbon starvation, SiaB mutants show impaired dispersal, in particular with the primary fermentation product ethanol. This suggests that carbon availability is at least one of the key environmental cues integrated by the SiaABCD system. Further, our biochemical, physiological and crystallographic data reveals that the phosphatase SiaA and its kinase counterpart SiaB balance the phosphorylation status of their target protein SiaC at threonine 68 (T68). Crystallographic analysis of the SiaA-PP2C domain shows that SiaA is present as a dimer. Dynamic modelling of SiaA with SiaC suggested that SiaA interacts strongly with phosphorylated SiaC and dissociates rapidly upon dephosphorylation of SiaC. Further, we show that the known phosphatase inhibitor fumonisin inhibits SiaA mediated phosphatase activity in vitro. In conclusion, the present work improves our understanding of how P. aeuruginosa integrates specific environmental conditions, such as carbon availability and surfactant stress, to regulate cellular aggregation and biofilm formation. With the biochemical and structural characterization of SiaA, initial data on the catalytic inhibition of SiaA, and the interaction between SiaA and SiaC, our study identifies promising targets for the development of biofilm-interference drugs to combat infections of this aggressive opportunistic pathogen.
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Dissertations / Theses on the topic "Siah"

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Iwai, Akio. "Siah-1L, a novel transcript variant belonging to the human Siah family of proteins, regulates β-catenin activity in a p53-dependent manner." Kyoto University, 2005. http://hdl.handle.net/2433/144711.

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Lehmann-Horn, Klaus. "Funktionelle Expression von Parkin, Siah-1 und Dorfin unter definierten, mit Morbus Parkinson assoziierten, Zellstressbedingungen." Diss., lmu, 2007. http://nbn-resolving.de/urn:nbn:de:bvb:19-69986.

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Susini, Laurent. "Implication de Siah-1 dans la dégradation des protéines et dans l' expression des gènes durant la réversion tumorale." Paris 7, 2003. http://www.theses.fr/2003PA077115.

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Haag, Daniela [Verfasser]. "Rolle der Siah-Ubiquitinligasen in der Entstehung der pulmonalen Hypertonie und daraus resultierenden rechtsventrikulären Hypertrophie / Daniela Haag." Gießen : Universitätsbibliothek, 2014. http://d-nb.info/1068591455/34.

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Beaucourt, Séverine. "Etude de 2 gènes cibles de la protéine P53 : siah (seven in absentia human homologue) et tsap6 (tumor suppressor activated pathway 6)." Phd thesis, Université Paris Sud - Paris XI, 2005. http://tel.archives-ouvertes.fr/tel-00157749.

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Notre travail a consisté à étudier 2 gènes impliqués dans la réversion tumorale et régulés par P53.
Nous avons identifié dans le promoteur du gène siah-1b une séquence sur laquelle P53 se lie in vitro et in vivo. De plus, nous avons montré la fonctionnalité de cet élément de réponse suite à l'induction de P53.
La particularité de ce site, dont la structure diffère fortement du consensus de liaison de P53 à l'ADN, ouvre de nouvelles perspectives dans l'analyse des mécanismes régulés par P53.
L'étude de la protéine TSAP6 murine a permis de caractériser son expression au niveau tissulaire et intracellulaire. La fonction de TSAP6 n'étant pas bien connue, nous avons entrepris de l'analyser dans des modèles animaux portant un gène tsap6 muté ou invalidé.
La mutation du gène tsap6 Gln-395-Lys induit l'apparition d'une microcytose et d'un emphysème pulmonaire chez la souris. La caractérisation des mécanismes conduisant à ces phénotypes est en cours et nous permettra de mieux comprendre la fonction de TSAP6.
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Khalil, Najat. "L'activation des récepteurs activés par les proliférateurs de peroxysomes beta/delta, stimule l'expression de Siah-1L et entraîne la prolifération et la migration de cellules de glioblastomes." Thesis, Université de Lorraine, 2013. http://www.theses.fr/2013LORR0286/document.

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Les Glioblastomes représentent les tumeurs cérébrales primaires les plus fréquentes chez l'adulte. Nous avons mis en évidence le rôle du récepteur PPARß/delta dans le caractère tumoral des glioblastomes. Nous avons montré que l'activation de ce récepteur stimule la prolifération et la migration des cellules de glioblastome T98G. Nous avons également montré que l'activation de PPARß/delta induit l'expression du gène Siah-1L et que la surexpression de ce gène stimule la prolifération et la migration cellulaire des glioblastomes. Nous avons ensuite étudié la régulation du gène Siah-1L humain et montré que son expression est induite par PPARß/delta. Le clonage du promoteur de ce gène et l'étude de sa régulation a permis de montrer que son induction par PPARß/delta est médiée par un élément de réponse situé au niveau du promoteur
Glioblastoma represent the most frequent primary brain tumor in adults. We highlighted the role of PPARß/delta in glioblastoma tumorigenesis. We have shown that activation of this receptor stimulates proliferation and migration of glioblastoma cells T98G. We also showed that activation of PPARß/delta induces the expression of Siah-1L gene and that overexpression of Siah-1L stimulates cell proliferation and migration of glioblastoma cells. We also studied the regulation of Siah-1L gene and showed that its expression is induced by PPARß/delta. Cloning of Siah-1L gene promoter sequence and the study of its regulation showed that the induction of the expression of Siah-1L by PPARß/delta is mediated by a response element located at the promoter sequence
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Tuijnder, Marcel. "Biological models of tumor reversion." Paris 7, 2005. http://www.theses.fr/2005PA077085.

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BRUZZONI-GIOVANELLI, Heriberto. "Caracterisation fonctionnelle de siah-1, un gene induit par p53 implique dans le controle de la proliferation cellulaire et l'apoptose." Paris 7, 1999. http://www.theses.fr/1999PA077263.

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Le gene sina (seven in absentia) de la drosophile est necessaire pour la specification cellulaire du photorecepteur r7 pendant le developpement oculaire. Siah-1, un des homologues murins de sina, est active dans un modele d'apoptose induite par p53. Son homologue humain, siah-1, code pour une proteine de 282 acides amines avec un domaine en doigt de zinc, localise sur le chromosome 16q12-q13. Nous demontrons que siah-1 est active au cours de la mort cellulaire programmee physiologique dans l'epithelium intestinal. Des cellules humaines selectionnees pour la suppression de leur phenotype tumoral presentent un niveau eleve d'arn messager de siah-1. Dans ces modeles on observe le meme type d'activation de l'expression du gene p21 w a f - 1. La transfection de la lignee cellulaire u937 par p21 w a f - 1 active l'expression de siah-1, rb1 et rb2, associee a des modifications de la l'organisation nucleaire en interphase. Des changements dans la distribution des sites exposes a la digestion par la dnase i et le repositionnement des chromosomes sont observes. Dans les modeles etudies, l'activation de siah-1 s'accompagne d'une perte ou d'une forte reduction du potentiel tumorigene des cellules chez la souris scid/scid. La surexpression du gene siah-1 dans des cellules mcf-7, entraine des alterations profondes de la croissance cellulaire dues a des anomalies majeures de la mitose et associes a l'induction de p21 w a f - 1 et a une augmentation du pourcentage de cellules apoptotiques. La proteine siah-1 est redistribuee dans la matrice nucleaire. Des cellules multinucleees sont observees, ainsi que des defauts dans la formation du fuseau mitotique, dans la separation des noyaux filles et durant la cytokinese. Ces resultats sont en
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Hildebrandt, Leandri. "The significance and status of Social Impact Assessment (SIA) in a South African context / Leandri Hildebrandt." Thesis, North-West University, 2012. http://hdl.handle.net/10394/8700.

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Social Impact Assessment (SIA) identifies the intended and unintended impacts that proposed projects or developments are likely to have on a community or individuals and suggest mitigation measures to prevent these impacts and enhance the positive impacts. The main aim of this dissertation is to explore the significance and the status of SIA in a South African context. EIA is currently in its third era of mandatory practice and with an increasing number of SIAs, it is essential that the practice of SIA should be investigated. The aim of this dissertation was firstly reached by exploring the perspectives of SIA practitioners in South Africa through a questionnaire. The practitioners’ perspectives showed that despite distinct weaknesses in the practice of SIA, i.e. the lack of a fixed set of guidelines and a shortage of SIA skill in the practitioner community, SIA practice has improved since 1997 with the promulgation of ECA, although there is still room for improvement. Secondly a quality review was conducted on a sample of SIARs in South Africa using an adapted review package. The results revealed relatively weak report quality compared to EIA report quality, but with an improvement, in report quality since 1997. The quality review findings appear to confirm the perspectives of the practitioners regarding the state of SIA practice in South Africa. Despite the weaknesses in the SIA process, it appears that the SIA practitioner community is driven by best practice considerations, and that SIA practice is in line with international trends. It appears therefore that despite the observed weaknesses, SIA practice in South Africa is relatively healthy. It is recommended that instead of seeking to strengthen SIA practice by means of regulation and guidelines, SIA practitioners should rather ensure that SIA delivers what it is intended to deliver by ongoing pursuance of best practice, and by improved training and skills development.
Thesis (MSc (Geography and Environmental Studies))--North-West University, Potchefstroom Campus, 2013
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Czechowicz, Julia Stephanie [Verfasser], and Wilhelm [Akademischer Betreuer] Schäfer. "Charakterisierung der Interaktion des Herpes-Simplex-Virus Typ 2 Proteins ICP0 mit der zellulären E3 Ubiquitinligase SIAH-1 und deren Rolle für den viralen Infektionsverlauf / Julia Stephanie Czechowicz ; Betreuer: Wilhelm Schäfer." Hamburg : Staats- und Universitätsbibliothek Hamburg, 2018. http://d-nb.info/1161847391/34.

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Books on the topic "Siah"

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Hashmi, Saira. Siah baif. Lahore: India-e-Naqush, 1986.

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Jean-Christophe, Ammann, Suter Margrit, Kunsthalle Basel, and Amsterdam (Netherlands) Stedelijk Museum, eds. Siah Armajani. Basel: Kunsthalle Basel, 1987.

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Armajani, Siah. Siah Armajani: Elements. New York: Max Protetch Gallery, 1991.

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Armajani, Siah. Siah Armajani, elements. New York, N.Y: Max Protetch Gallery, 1991.

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Weck, Ziba de. Siah Armajani: An ingenious world. London: Parasol Unit Foundation for Contemporary Art, 2013.

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Armajani, Siah. Siah Armajani, Kunsthalle Basel, Stedelijk Museum Amsterdam. Amsterdam: Stedelijk Museum, 1987.

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Armajani, Siah. Siah Armajani: Reading room Sacco & Vanzetti [exhibition]. Münster: The Museum, 1987.

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1956-, Brunner Hannes, ed. Common houses: Siah Armajani [and] Hannes Brunner. New York, N.Y: Swiss Institute, 1993.

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Armajani, Siah. Siah Armajani: Espacios de lectura = reading spaces. Barcelona: Museu d'Art Contemporani de Barcelona, 1995.

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1939-, Armajani Siah, ed. Nicht Stil: Konstruktion : Siah Armajanis Aneignung der Moderne. Frankfurt am Main: Das Museum, 1990.

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Book chapters on the topic "Siah"

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Krämer, Oliver H. "SIAH." In Encyclopedia of Cancer, 1–5. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-642-27841-9_7093-6.

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Krämer, Oliver H. "SIAH." In Encyclopedia of Cancer, 4205–9. Berlin, Heidelberg: Springer Berlin Heidelberg, 2014. http://dx.doi.org/10.1007/978-3-662-46875-3_7093.

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Sharifzadeh, M., R. Masoudi, and M. Ghorbani. "Siah Bisheh powerhouse cavern design modification using observational method and back analysis." In Rock Mechanics and Engineering, 153–80. Leiden, The Netherlands; Boca Raton: CRC Press/Balkema, [2017]– |Includes bibliographical references and index. Contents: volume 1. Principles: CRC Press, 2017. http://dx.doi.org/10.1201/9781315364223-6.

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Hagenbucher, Jan, Hilda Stekman, Alfonso Rodriguez-Gil, Michael Kracht, and M. Lienhard Schmitz. "Testing the Effects of SIAH Ubiquitin E3 Ligases on Lysine Acetyl Transferases." In Methods in Molecular Biology, 297–312. New York, NY: Springer New York, 2016. http://dx.doi.org/10.1007/978-1-4939-6527-4_22.

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Souri, Elham, Jahanshah Pakzad, and Hooman Foroughmand Arabi. "Challenges of Participatory Urban Design: Suggestions for Socially Rooted Problems in Sang-e Siah, Shiraz." In The Urban Book Series, 247–59. Cham: Springer International Publishing, 2019. http://dx.doi.org/10.1007/978-3-030-22762-3_16.

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Hawellek, Christian. "»Sich beobachten heißt sich verändern«." In Systemische Horizonte, 167–78. Göttingen: Vandenhoeck & Ruprecht, 2011. http://dx.doi.org/10.13109/9783666404382.167.

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Thomé, Ulrich. "ATL „Sich pflegen, sich kleiden“." In Neurochirurgische und neurologische Pflege, 81–92. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-55782-8_4.

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Thomé, Ulrich. "ATL „Sich pflegen, sich kleiden“." In Neurochirurgische und neurologische Pflege, 79–90. Berlin, Heidelberg: Springer Berlin Heidelberg, 1997. http://dx.doi.org/10.1007/978-3-662-08944-6_4.

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Porter, Venetia. "Meem 1958, By Siah Armajani." In Metalwork and Material Culture In the Islamic World. I.B.Tauris, 2012. http://dx.doi.org/10.5040/9780755694099.ch-028.

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Shahverdiloo, M. "Estimation of minimum insitu stress by hydrojacking method case study of Siah bisheh power plant." In Rock Stress and Earthquakes, 125–28. CRC Press, 2010. http://dx.doi.org/10.1201/9780415601658-20.

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Conference papers on the topic "Siah"

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Svyatova, Elizaevta. "Abstract 3403: SIAH and SIAH-interacting proteins as potential prognostic and predictive biomarkers in pancreatic adenocarcinoma." In Proceedings: AACR 106th Annual Meeting 2015; April 18-22, 2015; Philadelphia, PA. American Association for Cancer Research, 2015. http://dx.doi.org/10.1158/1538-7445.am2015-3403.

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Fallah, H., R. P. Brenner, and H. R. Haji Hassani. "Design of Instrumentation for the Siah Bishe Concrete Face Rockfill Dams." In GeoCongress 2006. Reston, VA: American Society of Civil Engineers, 2006. http://dx.doi.org/10.1061/40803(187)151.

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Svyatova, Elizaevta. "Abstract 4540: Use of SIAH E3 ubiquitin ligase as a biomarker in pancreatic adenocarcinoma." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-4540.

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Babaei, Mehdi, Maziar Babaei, and Mohsen Niasati. "Parametric analysis of overvoltages caused by back-flashover in “Siah-bishe” 400kV GIS substation." In 2013 3rd International Conference on Electric Power and Energy Conversion Systems (EPECS). IEEE, 2013. http://dx.doi.org/10.1109/epecs.2013.6713049.

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Urban, Zena M., Lauren van Reesema, Minglei Bian, Thomas C. Smyrk, Gloria Peterson, and Amy H. Tang. "Abstract 4442: Inhibition of SIAH E3 ligase function leads to increased cell death in metastatic human cancer cells." In Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1538-7445.am2014-4442.

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Horikawa, Izumi, Kaori Fujita, Abdul M. Mondal, Borivoj Vojtesek, Jean-Christophe Bourdon, David P. Lane, and Curtis C. Harris. "Abstract 3199: p53 represses TRF2 through E3 ubiquitin ligase Siah-1: Feedback regulation in telomere-initiated damage signaling." In Proceedings: AACR 101st Annual Meeting 2010‐‐ Apr 17‐21, 2010; Washington, DC. American Association for Cancer Research, 2010. http://dx.doi.org/10.1158/1538-7445.am10-3199.

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Hassani, H., Sh Arshadnejad, and H. Sarkheil. "Stability Analysis of Fault Rock Heterogeneity on the Left Pressure Shaft Collapse of Siah Bisheh Dam, North Iran." In GeoFlorida 2010. Reston, VA: American Society of Civil Engineers, 2010. http://dx.doi.org/10.1061/41095(365)225.

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Liao, Yang, Cheol Hong Park, Rebecca L. Schmidt, Atique U. Ahmed, Rie Takahashi, and Amy H. Tang. "Abstract A1: SIAH E3 ligase as a novel anticancer drug target to inhibit RAS‐mediated tumorigenesis and metastasis in human cancer cells." In Abstracts: First AACR International Conference on Frontiers in Basic Cancer Research--Oct 8–11, 2009; Boston MA. American Association for Cancer Research, 2009. http://dx.doi.org/10.1158/0008-5472.fbcr09-a1.

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Tang, Amy H., Yang Liao, Maryanne Edens, Ralf Janknecht, and Edward B. Leof. "Abstract B40: SIAH-dependent SMAD2/3/4 ubiquitination and degradation in mediating the K-RAS-TGFβ antagonism during tumorigenesis and metastasis in human pancreatic cancer cells." In Abstracts: AACR Special Conference on Pancreatic Cancer: Progress and Challenges; June 18-21, 2012; Lake Tahoe, NV. American Association for Cancer Research, 2012. http://dx.doi.org/10.1158/1538-7445.panca2012-b40.

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Bian, Minglei, Yang Liao, Vasilena Zheleva, Zena Urban, Monicah Njogu, Justin J. Odanga, Andrew J. Isbell, et al. "Abstract A26: Attacking the most downstream “gatekeeper,” the SIAH-dependent proteolytic machinery, in the oncogenic ERBB/K-RAS signaling pathway to block tumorigenesis and control metastasis in human cancer." In Abstracts: AACR Special Conference on RAS Oncogenes: From Biology to Therapy; February 24-27, 2014; Lake Buena Vista, FL. American Association for Cancer Research, 2014. http://dx.doi.org/10.1158/1557-3125.rasonc14-a26.

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Reports on the topic "Siah"

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BAILEY, DAVID H., and JONATHAN M. BORWEIN. PROPOSED SIAM PROBLEM. Office of Scientific and Technical Information (OSTI), August 2008. http://dx.doi.org/10.2172/983316.

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Giaquinto, Jj N., H. Dutchyshyn, J. W. Wilson, and M. G. Mathis. Single Integrated Air Picture (SIAP) Integrated Assessment Plan (IAP). Fort Belvoir, VA: Defense Technical Information Center, July 2002. http://dx.doi.org/10.21236/ada405128.

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Lee, Hyoung-Koo. Application of a-Si:H radiation detectors in medical imaging. Office of Scientific and Technical Information (OSTI), June 1995. http://dx.doi.org/10.2172/100242.

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Perez-Mendez, V., G. Cho, I. Fujieda, S. N. Kaplan, S. Qureshi, and R. A. Street. Proposed thin film electronics for a-Si:H PIXEL detectors. Office of Scientific and Technical Information (OSTI), April 1989. http://dx.doi.org/10.2172/6105550.

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Lewis, J. G., J. R. Gilbert, and B. N. Parlett. Fifth SIAM conference on applied linear algebra. Final report. Office of Scientific and Technical Information (OSTI), November 1994. http://dx.doi.org/10.2172/105011.

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Dimri, Goberdhan. MiR-146-SIAH2-AR Signaling in Castration-Resistant Prostate Cancer. Fort Belvoir, VA: Defense Technical Information Center, September 2015. http://dx.doi.org/10.21236/ada624004.

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Cho, Gyuseong. Signal and noise analysis of a-Si:H radiation detector-amplifier system. Office of Scientific and Technical Information (OSTI), March 1992. http://dx.doi.org/10.2172/10159346.

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Author, Not Given. SIAM Conference on Life Sciences Portland, OR July 11-14, 2004. Office of Scientific and Technical Information (OSTI), September 2004. http://dx.doi.org/10.2172/829900.

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Author, Not Given. SIAM Conference on Numerical Combustion Sedona, AZ May 9-12, 2004. Office of Scientific and Technical Information (OSTI), September 2004. http://dx.doi.org/10.2172/831175.

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Cho, Gyuseong. Signal and noise analysis of a-Si:H radiation detector-amplifier system. Office of Scientific and Technical Information (OSTI), March 1992. http://dx.doi.org/10.2172/7270898.

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