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Relevant bibliographies by topics / Sickle cell anemia

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Academic literature on the topic 'Sickle cell anemia'

Author: Grafiati

Published: 4 June 2021

Last updated: 30 November 2024

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Journal articles on the topic "Sickle cell anemia"

1

Fabrizio Nifosì, Antonio, Pablo Castelló, Lorenzo Nifosí, and Gianfilippo Nifosí. "Dental alterations in sickle cell anemia." Edorium Journal of Dentistry 4, no. 1 (June 1, 2017): 1–5. http://dx.doi.org/10.5348/d01-2017-23-ra-3.

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The epidemiology of the dental disease in sickle cell anemia is not adequately described. The aim of this work is either the evaluation of literature on the topic, or to describe if the disease itself predisposes to a dental pathology, and eventually also if the latter can influence its course. We selected many cases and reviews in order to identify the dental diseases related to sickle cell anemia. Caries and periodontitis are not directly caused by disease. However, an inflammatory state caused by a dental infection can trigger or precipitate a sickle crisis. Malocclusion angle class II is particularly frequent, as a result of bone facial changes and/or related to muscle imbalance. Temporomandibular joint disorder is possible. Asymptomatic pulp necrosis is due to the sickling that causes vasocclusion within microcirculation of the dental pulp. Large population-based studies are needed in order to clarify the dental involvement in this disease. A strict collaboration between the dentist and the hematologist is essential.

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Eagle, Kim, and Donald C. Doll. "Sickle Cell Anemia." New England Journal of Medicine 328, no. 2 (January 14, 1993): 105. http://dx.doi.org/10.1056/nejm199301143280206.

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Riley, Kiernan, Michael M. Evans, Kaléi Kowalchik, Lucy Adams, and Megan Lucey. "Sickle cell anemia." Nursing Made Incredibly Easy! 18, no. 6 (November 2020): 26–33. http://dx.doi.org/10.1097/01.nme.0000717640.57159.b5.

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Mitchell, Ronald. "Sickle Cell Anemia." American Journal of Nursing 99, no. 5 (May 1999): 36–37. http://dx.doi.org/10.1097/00000446-199905000-00045.

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Lonergan, Gael J., David B. Cline, and Susan L. Abbondanzo. "Sickle Cell Anemia." RadioGraphics 21, no. 4 (July 2001): 971–94. http://dx.doi.org/10.1148/radiographics.21.4.g01jl23971.

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Schrier, S. "Sickle Cell Anemia." ASH Image Bank 2001, no. 1208 (December 8, 2001): 100248. http://dx.doi.org/10.1182/ashimagebank-2001-100248.

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7

Sydenstricked [sic], V. P. "Sickle Cell Anemia." American Journal of Diseases of Children 141, no. 6 (June 1, 1987): 612. http://dx.doi.org/10.1001/archpedi.1987.04460060030025.

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8

Mason, V. R. "Sickle Cell Anemia." JAMA: The Journal of the American Medical Association 254, no. 14 (October 11, 1985): 1955. http://dx.doi.org/10.1001/jama.1985.03360140113037.

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9

Johnson, Cage S. "Sickle Cell Anemia." JAMA: The Journal of the American Medical Association 254, no. 14 (October 11, 1985): 1958. http://dx.doi.org/10.1001/jama.1985.03360140116038.

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Powars, Darleen. "Sickle Cell Anemia." American Journal of Diseases of Children 147, no. 11 (November 1, 1993): 1197. http://dx.doi.org/10.1001/archpedi.1993.02160350071011.

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Dissertations / Theses on the topic "Sickle cell anemia"

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Saleh, A. W. "Modulation of fetal hemoglobin in sickle cell anemia." Maastricht : Maastricht : Universiteit Maastricht ; University Library, Maastricht University [Host], 1998. http://arno.unimaas.nl/show.cgi?fid=8498.

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Winn, Vaniethia R. "The psychosocial adjustment of adolescents with Sickle Cell Anemia." DigitalCommons@Robert W. Woodruff Library, Atlanta University Center, 1994. http://digitalcommons.auctr.edu/dissertations/381.

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The overall objective of this study is to investigate the psychosocial problems experienced by adolescents with sickle cell anemia. To attain this objective, the following issues were addressed by the researcher: (a) peer relationships; (b) independence; (c) stress; and (d) self-concept. A descriptive research design was used in the study. A questionnaire developed by the researcher was utilized and administered via telephone conversation/interview to 13 adolescents with sickle cell anemia located in the Metropolitan Atlanta area. The results of the study indicated that the participants diagnosed with sickle cell anemia maintain good peer relationships, experienced high levels of stress, have low self-concept and, have not achieved an appropriate level of independence with regard to a sense of personal freedom.

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Yeboah, Sybil A. Parise Leslie V. "Do CD40L and CD40 contribute to sickle cell anemia?" Chapel Hill, N.C. : University of North Carolina at Chapel Hill, 2008. http://dc.lib.unc.edu/u?/etd,1640.

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Thesis (M.S.)--University of North Carolina at Chapel Hill, 2008.
Title from electronic title page (viewed Sep. 16, 2008). "... in partial fulfillment of the requirements for the degree of Master of Science in the Department of Biochemistry." Discipline: Biochemistry and Biophysics; Department/School: Medicine.

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Brown, Marena Dessette. "Sickle cell-endothelial interactions : modulation of cell adhesion molecule expression." Diss., Georgia Institute of Technology, 1995. http://hdl.handle.net/1853/11306.

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Weng, Weijun Ferrone Frank A. "Universal metastability of sickle hemoglobin polymerization /." Philadelphia, Pa. : Drexel University, 2008. http://hdl.handle.net/1860/2832.

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6

Reed, Caroline. "Increasing Hydroxyurea Adherence for Pediatric Patients With Sickle Cell Anemia." Thesis, Walden University, 2016. http://pqdtopen.proquest.com/#viewpdf?dispub=10141603.

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Sickle cell disease is a disabling chronic autosomal recessive blood disease characterized by abnormal hemoglobin, pain crises, and frequent emergency department visits. Adherence to hydroxyurea therapy has been shown to improve these patient outcomes. Guided by the theory of comfort, the purpose of this project was to determine if an educational intervention would increase adherence to hydroxyurea therapy in pediatric patients between 2 and 17 years of age recruited from an urban university hospital hematology clinic. The RE-AIM model was used to support the translation of evidence and the change process. An educational video produced by AFLAC was viewed by patients’ parents 4 weeks after enrollment into this pretest/posttest design project. A total of 22 African-American parent participants completed the 8-item Morisky Medication Adherence Scale at baseline and again at 8 weeks to assess hydroxyurea adherence. The Short Test of Functional Health Literacy in Adults tool was used to assess parents’ health learning needs; all parents met the adequate literacy level at baseline. Using t test statistics, no statistically significant differences were found pretest to posttest on the Morisky Medication Adherence Scale scores, mean corpuscular hemoglobin, and fetal hemoglobin percentages. Wilcoxon Signed Rank tests showed no significant differences in emergency room visits nor number of pain crisis. Although no significant changes emerged in short-term hematologic findings, emergency room visits, and pain crises, social change in the health care setting was promoted by confirming parents were able to understand education and a high level of hydroxyurea adherence was maintained; literature indicated that long-term adherence to hydroxyurea limits severe attacks.

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Zakharov, Mikhail N. Ferrone Frank A. "A microrheological study of sickle hemoglobin polymerization /." Philadelphia, Pa. : Drexel University, 2009. http://hdl.handle.net/1860/3075.

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Moxley, Kristan Michelle. "Impact of Carrier Screening on Pregnant Women’s Knowledge of Sickle Cell Anemia." Case Western Reserve University School of Graduate Studies / OhioLINK, 2008. http://rave.ohiolink.edu/etdc/view?acc_num=case1201280917.

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Cytlak, Urszula Malgorzata. "Phosphatidylserine exposure in red blood cells from patients with sickle cell disease." Thesis, University of Cambridge, 2015. https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.708601.

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Wagner, Matthew Christian. "Histamine as a Potential Initiator of Sickle Pain crisis by Mediation of Sickle Erythrocyte Adherence in a Shear-Dependent Manner." Diss., Georgia Institute of Technology, 2006. http://hdl.handle.net/1853/14478.

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The genetic disorder sickle cell anemia causes hemolytic anemia and sickle pain crisis, episodes of microvascular occlusion resulting in painful ischemic tissue damage. Pain crisis is thought to occur when sickle erythrocytes adhere in the post-capillary venule, partially occluding the vessel. The resulting slowed blood flow causes more extensive cell adherence and entrapment of rigid, deoxygenated erythrocytes until the vessel is entirely occluded. It was hypothesized that the inflammatory mediators histamine and tumor necrosis factor-, factors known to cause endothelial expression of adhesive ligands, might significantly increase sickle erythrocyte adhesion, and thus be capable of initiating sickle pain crisis. It was also hypothesized that the perfusion shear stress environment of the endothelium, known to be oscillatory and reduced in sickle cell patients, was a significant mediating factor of sickle cell adhesion. An in-vitro flow chamber using cultured endothelial cells and erythrocytes from blood samples of sickle cell anemic patients was used to quantify sickle erythrocyte adherence to stimulated and unstimulated endothelial cells under shear stresses from 1.0 to 0.1 dyne/cm2. Results showed that both endothelial stimulation and reduction of the perfusion shear stress increased sickle erythrocyte adherence. In combination, the use of inflammatory stimulation with reduced shear stress resulted in further increased adhesion, but only when above the range of 0.1 V 0.2 or 0.4 dyne/cm2, depending on the inflammatory mediator. Adhesion below this level of shear is not significantly increased by endothelial stimulation. The mechanism by which histamine mediates adhesion was investigated, and found to involve the endothelial H2 and H4 receptors and expression of the P-selectin ligand. These data suggest that irregular flow, typical of sickle microvasculature, may act in conjunction with the pro-inflammatory state of sickle vasculature and the histaminergic nature of some pain treatments to initiate or propagate sickle vaso-occlusion. Findings concerning histamine, tumor necrosis factor-alpha, and shear stress effects on adherence are discussed in relation to their possible applicability to patient health, future studies are outlined to confirm the relation of in vitro data to in vivo patient condition, and proposals are made for applying these methodologies to other potential mediators of sickle erythrocyte adhesion.

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Books on the topic "Sickle cell anemia"

1

Costa, Fernando Ferreira, and Nicola Conran, eds. Sickle Cell Anemia. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-06713-1.

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2

National Institutes of Health (U.S.), ed. Sickle cell anemia. [Bethesda, Md.]: U.S. Dept. of Health and Human Services, Public Health Service, National Institutes of Health, 1990.

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3

B, Silverstein Virginia, and Nunn Laura Silverstein, eds. Sickle cell anemia. Springfield, NJ, USA: Enslow Publishers, 1997.

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4

Massachusetts. Department of Public Health. Sickle cell anemia. Boston, MA: Massachusetts Dept. of Public Health, 1989.

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5

Beshore, George. Sickle cell anemia. New York: F. Watts, 1994.

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National Institutes of Health (U.S.). Sickle cell anemia. Bethesda, Md.]: U.S. Department of Health and Human Services, Public Health Service, National Institutes of Health, 1990.

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7

K, Ballas Samir. Sickle cell pain. 2nd ed. Washington, D.C: IASP Press, 2014.

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8

Jones, Phill. Sickle cell disease. New York: Chelsea House, 2008.

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9

B, Silverstein Virginia, and Nunn Laura Silverstein, eds. The sickle cell anemia update. Berkeley Heights, NJ: Enslow Publishers, 2006.

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10

Peak, Lizabeth. Sickle cell disease. Detroit: Lucent Books, 2008.

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Book chapters on the topic "Sickle cell anemia"

1

Roberts, Scott. "Sickle Cell Anemia." In Queenan's Management of High-Risk Pregnancy, 93–97. Oxford, UK: Wiley-Blackwell, 2012. http://dx.doi.org/10.1002/9781119963783.ch12.

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Taylor, Jeffrey S. "Sickle Cell Anemia." In Pediatric Surgery, 305–10. Cham: Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-04340-1_55.

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3

Morse, Linda W., and Agnes E. Shine. "Sickle cell anemia." In Health-related disorders in children and adolescents: A guidebook for understanding and educating., 596–602. Washington: American Psychological Association, 1998. http://dx.doi.org/10.1037/10300-083.

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4

Morse, Linda W., Melanie C. Morse, and Sarah E. Bills. "Sickle cell anemia." In Health-related disorders in children and adolescents: A guidebook for educators and service providers (2nd ed.)., 83–89. Washington: American Psychological Association, 2023. http://dx.doi.org/10.1037/0000349-010.

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Jorge, Susan E., Daniela M. Ribeiro, Magnun N. N. Santos, and Maria de Fátima Sonati. "Hemoglobin: Structure, Synthesis and Oxygen Transport." In Sickle Cell Anemia, 1–22. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-06713-1_1.

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Abdennour, Rouba, and Miguel R. Abboud. "Treatment of Childhood Sickle Cell Disease." In Sickle Cell Anemia, 231–67. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-06713-1_10.

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Claudino, Mário A., Carla F. Franco Penteado, and Kleber Yotsumoto Fertrin. "Priapism in Sickle Cell Disease: New Aspects of Pathophysiology." In Sickle Cell Anemia, 269–83. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-06713-1_11.

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Costa, Fernando Ferreira, and Kleber Yotsumoto Fertrin. "Clinical Manifestations and Treatment of Adult Sickle Cell Disease." In Sickle Cell Anemia, 285–318. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-06713-1_12.

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Saad, Sara T. Olalla, and Simone O. Gilli. "Hemoglobin Sβ Thalassemia, SC Disease and SD Disease: Clinical and Laboratorial Aspects." In Sickle Cell Anemia, 319–37. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-06713-1_13.

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Adekile, Adekunle, and Julie Makani. "Sickle Cell Disease in Africa and the Arabian Peninsula: Current Management and Challenges." In Sickle Cell Anemia, 339–70. Cham: Springer International Publishing, 2016. http://dx.doi.org/10.1007/978-3-319-06713-1_14.

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Conference papers on the topic "Sickle cell anemia"

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Albayrak, Batuhan, Muazzez Buket Darici, Furkan Kiraci, Arif Selcuk Ogrenci, Atilla Ozmen, and Kerem Ertez. "Orak Hücreli Anemi Tespiti Sickle Cell Anemia Detection." In 2018 Medical Technologies National Congress (TIPTEKNO). IEEE, 2018. http://dx.doi.org/10.1109/tiptekno.2018.8597092.

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CHEUNG, ANTHONY TZE-WAI. "BLOOD FLOW ABNORMALITIES IN SICKLE CELL ANEMIA." In Advances in Engineering Mechanics - Reflections and Outlooks - In Honor of Theodore Y.-T. Wu. WORLD SCIENTIFIC, 2005. http://dx.doi.org/10.1142/9789812702128_0029.

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Wynter, Clive I. "Paramagnetic Europium Salen Complex and Sickle-Cell Anemia." In INDUSTRIAL APPLICATIONS OF THE MOSSBAUER EFFECT: International Symposium on the Industrial Applications of the Mossbauer Effect. AIP, 2005. http://dx.doi.org/10.1063/1.1923673.

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Tengshe, Richa, Aishwarya U. N, Akarsh Raj, Akshaya K, Apeksha A. Pattanshetty, and Binish Fatimah. "Sickle cell anemia detection using convolutional neural network." In 2021 12th International Conference on Computing Communication and Networking Technologies (ICCCNT). IEEE, 2021. http://dx.doi.org/10.1109/icccnt51525.2021.9580165.

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Wahed, Fareen Farzana, Anitha Juliette A., P. Sinthia, and G. Aloy Anuja Mary. "Detection of sickle cell anemia using SVM classifier." In EIGHTH INTERNATIONAL CONFERENCE NEW TRENDS IN THE APPLICATIONS OF DIFFERENTIAL EQUATIONS IN SCIENCES (NTADES2021). AIP Publishing, 2022. http://dx.doi.org/10.1063/5.0074138.

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Lodonio, Larissa Lacerda, Livia Romana Lima Gonçalves Arrais, David Nilson Gondim Alves, Valéria Sampaio Freire Alencar, Eva Cristina Lopes Vieira Torres, Estefani Gonçalves de Almeida Grangeiro, Jackeline Lima Vidal, Orleudo Ferreira Teixeira, Gessyca Tavares Feitosa, and Joanderson Nunes Cardoso. "Acute chest syndrome in sickle cell anemia: Diagnostic challenges and therapeutic strategies." In VI Seven International Multidisciplinary Congress. Seven Congress, 2024. http://dx.doi.org/10.56238/sevenvimulti2024-066.

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Sickle cell disease is a genetic blood disorder caused by a mutation in the amino acid glutamic acid, which is replaced by valine in the beta chain of the hemoglobin molecule. In this context, acute chest syndrome is a serious disease with a high rate of morbidity and mortality, accounting for approximately 25% of deaths in patients with sickle cell anemia. Thus, the objective of this study is to describe the main aspects of Acute Chest Syndrome (ACS) in patients with sickle cell anemia. This is a review study of a scoping review in which data collection was carried out on the research portal of the Virtual Health Library (BVS). The databases used were: Online Medical Literature Search and Analysis System ( MEDLINE ) and Latin American and Caribbean Literature in Health Sciences (LILACS ) , where the following descriptors were used for the search: “Sickle Cell Anemia” AND“Acute Chest Syndrome”. Using the descriptors, 451 articles were found. The inclusion criteria were: articles published between 2019 and 2024, available in full and free of charge; and the exclusion criteria were repeated articles, paid articles, and methods with an emphasis on literature review. Through the established criteria, 20 studies were included in the final sample. Acute chest syndrome is characterized by fever and/or respiratory symptoms with pulmonary infiltrates, which can lead to sepsis and cause stroke in patients with sickle cell anemia. In addition, hemolysis increases during sickle cell crises, causing a faster depletion of nitric oxide, which is a potent vasodilator of metabolism and a factor for cardiopulmonary hemodynamics. In this context, ultrasound is recognized as the gold standard in the diagnosis of the syndrome, due to the absence of radiation, with high accuracy, sensitivity, and specificity. Changes in lung function in acute chest syndrome cause variations in the levels of inflammatory markers that can help to recognize the condition and treat it more effectively, such as phosphatidylserine, a specific type of phospholipid that is essential in cell membranes, serum ferritin, which increases in an attempt to compensate for the increase in hemolysis, and IL-6, which reflects the recruitment of monocytes and other innate immune cells in the lungs. In view of the intense hemolysis, red blood cell transfusions can be seen as a protective factor for acute chest syndrome and can even be considered the definitive therapy for the syndrome, as it improves the supply of oxygen to the tissues, increases the overall level of hemoglobin and reduces the fraction of sickle-shaped red blood cells. This allows the patient to have relief from respiratory symptoms more quickly. Therefore, acute chest syndrome in patients with sickle cell anemia requires appropriate interventions and should be treated as rare diseases in health units, whether in the basic health unit or in emergency care, in order to ensure adequate treatment without delays. Thus, the management that should be carried out is analgesia, hydration, antibiotic therapy, bronchodilators, ventilation, which may be invasive or non-invasive, oxygen and blood transfusion.

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Solomon, Rance, James Cooper, Gabriel Welker, Elaura Aguilar, Brooke Flanagan, Chelsey Pennycuff, David Scott, et al. "Relative deformability of red blood cells in sickle cell trait and sickle cell anemia by trapping and dragging." In European Conferences on Biomedical Optics, edited by Lothar D. Lilge and Ronald Sroka. SPIE, 2013. http://dx.doi.org/10.1117/12.2032591.

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Chy, Tajkia Saima, and Mohammad Anisur Rahaman. "Automatic Sickle Cell Anemia Detection Using Image Processing Technique." In 2018 International Conference on Advancement in Electrical and Electronic Engineering (ICAEEE). IEEE, 2018. http://dx.doi.org/10.1109/icaeee.2018.8642984.

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Soni, Samiksha, Hardik Thakkar, and Bikesh Kumar Singh. "Transfer Learning for Sickle Cell Anemia and Trait Classification." In 2022 Second International Conference on Power, Control and Computing Technologies (ICPC2T). IEEE, 2022. http://dx.doi.org/10.1109/icpc2t53885.2022.9776656.

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Wachem, Oussema. "195 Echocardiographic evaluation in children with sickle cell anemia." In British Cardiovascular Society Annual Conference, ‘Back to the patient’, 3–5 June 2024. BMJ Publishing Group Ltd and British Cardiovascular Society, 2024. http://dx.doi.org/10.1136/heartjnl-2024-bcs.190.

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Reports on the topic "Sickle cell anemia"

1

Goddard, Sharon. Sickle Cell Anemia : a Psychosocial Study of Attitudes and Effect. Portland State University Library, January 2000. http://dx.doi.org/10.15760/etd.2054.

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