Relevant bibliographies by topics / Sickle cell pathology

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  1. Journal articles
  2. Dissertations / Theses
  3. Books
  4. Book chapters
  5. Conference papers

Academic literature on the topic 'Sickle cell pathology'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Sickle cell pathology"

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Malowany, Janet I., and Jagdish Butany. "Pathology of sickle cell disease." Seminars in Diagnostic Pathology 29, no. 1 (2012): 49–55. http://dx.doi.org/10.1053/j.semdp.2011.07.005.

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Rice-Evans, C., S. C. Omorphos, and E. Baysal. "Sickle cell membranes and oxidative damage." Biochemical Journal 237, no. 1 (1986): 265–69. http://dx.doi.org/10.1042/bj2370265.

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Sickle erythrocytes and their membranes are susceptible to endogenous free-radical-mediated oxidative damage which correlates with the proportion of irreversibly sickled cells. The suppression of incubation-induced oxidative stress by antioxidants, free radical scavengers and an iron chelator suggest that oxidation products of membrane-bound haemoglobin contribute towards the pathology of the disease.
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&NA;. "Liver Pathology In Sickle Cell Diseases." Advances in Anatomic Pathology 2, no. 5 (1995): 336. http://dx.doi.org/10.1097/00125480-199509000-00009.

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Diwan, B. A., M. T. Gladwin, C. T. Noguchi, J. M. Ward, A. L. Fitzhugh, and G. S. Buzard. "Renal Pathology in Hemizygous Sickle Cell Mice." Toxicologic Pathology 30, no. 2 (2002): 254–62. http://dx.doi.org/10.1080/019262302753559597.

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Hawley, Dean A., and Leo J. McCarthy. "Sickle Cell Disease." American Journal of Forensic Medicine and Pathology 30, no. 1 (2009): 69–71. http://dx.doi.org/10.1097/paf.0b013e3181873c90.

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Manci, E. A. "Pathology of Berkeley sickle cell mice: similarities and differences with human sickle cell disease." Blood 107, no. 4 (2006): 1651–58. http://dx.doi.org/10.1182/blood-2005-07-2839.

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Sheshanna, Nanditha, Gurpreet Sethi, and Sumitha M. Prakash. "Sudden Death in Sickle Cell Disease: An Autopsy Diagnosis." Journal of Medical Sciences 3, no. 4 (2017): 113–15. http://dx.doi.org/10.5005/jp-journals-10045-0069.

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ABSTRACT Sickle cell disease is a common hereditary hemoglobinopathy with high prevalence in the central and northeastern regions of India. A 24-year-old male patient with 3 days’ history of fever was brought dead to the hospital. Morphology showed clogging of blood vessels with sickled red blood cells (RBCs) in all the organs, and an autopsy diagnosis of sickle cell disease was made. As the cause may not be obvious in many cases, most patients remain undiagnosed. It is important to note the circumstances of death, gross finding, and histopathology, with hemoglobin electrophoresis if available
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Gheorghe, Gabriela, Marian Rollins-Raval, and Miguel Reyes-Múgica. "Sickle Cell Disease." International Journal of Surgical Pathology 17, no. 3 (2008): 270–71. http://dx.doi.org/10.1177/1066896908328578.

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Jacobs, A. "Sickle Cell Disease." Journal of Clinical Pathology 39, no. 7 (1986): 815. http://dx.doi.org/10.1136/jcp.39.7.815-a.

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Rudy, Hayeem L., David Yang, Andrew D. Nam, and Woojin Cho. "Review of Sickle Cell Disease and Spinal Pathology." Global Spine Journal 9, no. 7 (2018): 761–66. http://dx.doi.org/10.1177/2192568218799074.

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Study Design:Sickle cell disease (SCD) is a relatively common blood disorder that has profound implications on the musculoskeletal system and particularly the spine; however, there is a paucity of data in the literature discussing this important topic.Objectives:(1) To elucidate common spinal pathologies affecting patients with SCD, as well as the medical and surgical treatments available for these patients. (2) To discuss indications for surgical management of spinal complications of SCD and important for orthopedic surgeons when taking patients with SCD to the operating room.Methods:A narrat
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Dissertations / Theses on the topic "Sickle cell pathology"

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Kiessling, Katrin. "The importance of amino acid transport for human red blood cells." Thesis, University of Oxford, 1997. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.389052.

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Wright, Adrian C. "Three transgenic hemoglobin SAD (S-Antilles, -Punjab), partial-"knockout" murine models of human sickle cell disease : the generation and phenotypic analysis of SAD mice with (1) heterozygous-null deletion of the murine a-globin genes, (2) homozygous-null deletion of the murine a-globin genes, and (3) heterozygous null deletions of the murine a- and b-globin genes." Thesis, McGill University, 2000. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=31559.

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To further our understanding of the complex pathophysiology of human sickle cell disease (SCD) and to develop better therapies, three transgenic mouse lines have been produced that survive on mostly human hemoglobin (Hb). These lines have been generated by crossing HbSAD (SAD) transgenic mice with other mice carrying a null deletion of the endogenous globin genes (alpha or beta), SAD/homozygous alpha-globin null (SADalpha -/-) mice display a SCD-like phenotype and have a dramatically reduced mean lifespan; therefore, this line provides a novel model for assessment of anti-sickling protocols or
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Diarra, Amadou. "LA DRÉPANOCYTOSE ET LES ASPECTS TRANSFUSIONNELS AU MALI: Problématique de la transfusion sanguine chez les drépanocytaires à Bamako." Doctoral thesis, Universite Libre de Bruxelles, 2015. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/220037.

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IntroductionLa drépanocytose est l’affection génétique la plus répandue dans le monde plus particulièrement en Afrique subsaharienne, considérée à juste titre comme l’une des régions originaires de la mutation drépanocytaire avec de fortes prévalences. Elle est reconnue comme un problème de santé majeur par la communauté internationale en raison de sa morbidité et de sa mortalité élevées. Au Mali, environ 5 à 6 milles naissances de drépanocytaires sont enregistrées par an [Diallo, 2008] et ils auront besoin d’une prise en charge médicale spécifique. Le Centre de Recherche et de Lutte contre la
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Paule, Inès. "Adaptation of dosing regimen of chemotherapies based on pharmacodynamic models." Phd thesis, Université Claude Bernard - Lyon I, 2011. http://tel.archives-ouvertes.fr/tel-00846454.

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There is high variability in response to cancer chemotherapies among patients. Its sources are diverse: genetic, physiologic, comorbidities, concomitant medications, environment, compliance, etc. As the therapeutic window of anticancer drugs is usually narrow, such variability may have serious consequences: severe (even life-threatening) toxicities or lack of therapeutic effect. Therefore, various approaches to individually tailor treatments and dosing regimens have been developed: a priori (based on genetic information, body size, drug elimination functions, etc.) and a posteriori (that is us
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Mukisi, Mukaza Martin. "Contribution à l'étude de l'ostéonécrose drépanocytaire de la tête fémorale de l'adulte: épidémiologie, diagnostic et traitement." Doctoral thesis, Universite Libre de Bruxelles, 2010. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/210075.

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La drépanocytose est la maladie moléculaire et héréditaire (transmission mendélienne récessive et autosomique) la plus répandue au monde. Elle est un problème de santé publique par sa gravité et ses implications socio-économiques dans de nombreux pays. Seuls les sujets homozygotes (SS) ou hétérozygotes composites (SC) sont malades, les hétérozygotes (AS) ne sont que des transmetteurs du gène S. Elle est la première cause d’OstéoNécrose de la Tête Fémorale (ONTF), douloureuse évoluant vers l’arthrose, en l’absence de traitement chez un patient jeune.<p>La Guadeloupe compte 450.000 habitants, do
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Noubouossie, Fondjie-Denis. "Contribution du test de génération de thrombine in vitro à l'étude des troubles de la coagulation dans le drépanocytose." Doctoral thesis, Universite Libre de Bruxelles, 2013. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209459.

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La drépanocytose est associée à un état d’hypercoagulabilité qui se manifeste sur le plan clinique par un risque augmenté de thromboses artérielles et veineuses. L’exploration de la coagulation chez les patients drépanocytaires montrait surtout une activation de la coagulation et des altérations des acteurs pro- et anticoagulants du système hémostatique. Les tests de coagulation globale de routine tels que l’aPTT et le PT sont peu sensibles aux états d’hypercoagulabilité. La fonction hémostatique globale des patients drépanocytaires était donc peu connue. Le test de génération de thrombine est
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Batina, Agasa Salomon. "Drépanocytose et transfusion sanguine: étude réalisée à Kisangani en République Démocratique du Congo." Doctoral thesis, Universite Libre de Bruxelles, 2011. http://hdl.handle.net/2013/ULB-DIPOT:oai:dipot.ulb.ac.be:2013/209894.

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Introduction<p><p>La drépanocytose, affection génétique concernant 1 à 2% de la population en Afrique sub-saharienne, est une maladie chronique dont l’un des traitements essentiels est la transfusion sanguine. Kisangani, une ville du Nord-est de la République Démocratique du Congo, compte environ un million d’habitants et près de 30 000 naissances par an. Elle est caractérisée entre autre par l’endémie malarienne, la fréquence élevée dans la population des virus de l’immunodéficience humaine (VIH), des virus des hépatites B (VHB) et C (VHC) et la carence d’autosuffisance en sang. Le don de san
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Natarajan, Maya. "Receptor mediated adhesion of sickle erythrocytes and damage to IL-1 beta stimulated endothelial cells under flow conditions in vitro." Thesis, 1996. http://hdl.handle.net/1911/16917.

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Enhanced, abnormal adherence of sickle erythrocytes to the endothelium lining blood vessels is hypothesized to contribute to vasoocclusion in sickle cell patients, but the specific molecular mechanisms by which these erythrocyte-endothelial interactions occur have not yet been fully elucidated. Alteration of endothelial function, or frank endothelial damage, may also be involved in the pathogenesis of vascular occlusion. In this thesis, these two different aspects of vascular occlusion, namely adhesion and damage, were studied. A parallel plate flow chamber and computerized phase contrast micr
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Books on the topic "Sickle cell pathology"

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Ferrari, Lynne R. Sickle Cell Disease. Edited by Kirk Lalwani, Ira Todd Cohen, Ellen Y. Choi, and Vidya T. Raman. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190685157.003.0051.

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Sickle cell anemia is a disease that combines molecular biology, clinical features, biochemistry, pathology, natural selection, population genetics, gene expression, and genomics and is the world’s most common life-threatening monogenic disorder. Clinical features include anemia; painful crisis especially in fingers, chest, and long bones; hemolysis; splenic infarction resulting in functional asplenia; and microinfarction leading to neurologic and renal impairment. The maintenance of adequate body temperature with active warming devices and warmed intravenous fluids, monitoring hydration and u
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Tsuyoshi, Ohnishi S., and Ohnishi Tomoko, eds. Membrane abnormalities in sickle cell disease and in other red blood cell disorders. CRC Press, 1994.

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Book chapters on the topic "Sickle cell pathology"

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"Sickle Cell Nephropathy." In Diagnostic Pathology: Kidney Diseases. Elsevier, 2016. http://dx.doi.org/10.1016/b978-0-323-37707-2.50122-1.

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Dravid, NV. "Sickle Cell Anemia." In Manual of Autopsy Pathology. Jaypee Brothers Medical Publishers (P) Ltd., 2004. http://dx.doi.org/10.5005/jp/books/10461_8.

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"Sickle Cell Anemia." In Diagnostic Pathology: Blood and Bone Marrow. Elsevier, 2018. http://dx.doi.org/10.1016/b978-0-323-39254-9.50030-1.

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Nayak, Ramadas, Sharada Rai, and Astha Gupta. "Sickle cell disease." In Essentials in Hematology and Clinical Pathology. Jaypee Brothers Medical Publishers (P) Ltd., 2012. http://dx.doi.org/10.5005/jp/books/11513_8.

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Nayak, Ramadas, and Sharada Rai. "Sickle Cell Disease." In Essentials in Hematology and Clinical Pathology. Jaypee Brothers Medical Publishers (P) Ltd., 2017. http://dx.doi.org/10.5005/jp/books/12955_9.

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Maheshwari, Nanda. "Sickle Cell Preparation." In Clinical Pathology, Haematology and Blood Banking (For DMLT Students). Jaypee Brothers Medical Publishers (P) Ltd., 2008. http://dx.doi.org/10.5005/jp/books/10143_14.

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Raptis, Constantine. "Pulmonary Complications of Sickle Cell Anemia." In Chest Imaging. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780199858064.003.0025.

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“Sickle cell disease” describes the spectrum of pathology in patients with at least one HbS chain and one other abnormal β‎ globin chain. Although patients with sickle cell disease often present with a simple community acquired pneumonia, acute chest syndrome must be considered in patients presenting with chest pain and fever, as it carries an increased risk of mortality, especially in adults. A few other entities, including rib infarction and subdiaphragmatic pathologies, can mimic the symptoms of acute chest syndrome. Finally, the findings of sickle cell disease on chest radiography will be discussed. Radiologists must be familiar with these findings in order to accurately interpret imaging studies, especially when the history of sickle cell is not provided.
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Öztaş, Yeşim, and İffet İpek Boşgelmez. "Oxidative stress in sickle cell disease and emerging roles for antioxidants in treatment strategies." In Pathology. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-815972-9.00006-8.

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Inusa, Baba, Maddalena Casale, and Nicholas Ward. "Introductory Chapter: Introduction to the History, Pathology and Clinical Management of Sickle Cell Disease." In Sickle Cell Disease - Pain and Common Chronic Complications. InTech, 2016. http://dx.doi.org/10.5772/65648.

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"Haematology." In Oxford Handbook for Medical School, edited by Kapil Sugand, Miriam Berry, Imran Yusuf, et al. Oxford University Press, 2019. http://dx.doi.org/10.1093/med/9780199681907.003.0018.

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Haematology is the speciality encompassing diseases of the blood, bone marrow, and lymphatic system. It requires skills in both medicine and pathology. Haematology departments are also responsible for providing a hospital’s laboratory and transfusion service. Common diagnoses include myeloma, lymphoma, leukaemia, idiopathic thrombocytopaenia, sickle cell disease, clotting and bleeding disorders, and transfusion-related conditions. Important haematological investigations include the blood film, bone marrow studies with increasing use of cytogenetics, and clotting studies. A summary of important red cell morphologies is included, in addition to summaries of haematopoietic cell lineage and clotting pathways (and their related disorders). Venous thromboembolic disease is discussed, as well as the variety of anticoagulant therapies currently available. Important elements of transfusion immunology and medicine are outlined with a clinical guide to transfusion reactions and their management. The section on haematology in the emergency department describes emergency management of neutropaenic sepsis. A practical guide to the interpretation of investigations is included.
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Conference papers on the topic "Sickle cell pathology"

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Chamone, D. A. F., A. Y. Hoshikawa-Fujimura, C. Massumoto, G. Bellotti, F. Arashiro, and M. Jamra. "ABNORMALITIES OF PLATELET AGGREGATION AND ENHANCED FACTOR X ACTIVATOR ACTIVITY OF WASHED PLATELETS IN SICKLE CELL DISEASE." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1644544.

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The occurence of microvascular occlusion is one of the most prominent pathologic features of sickle cell anemia. The mechanism of vaso occlusion has generally been attributed to the abnormal shape and reduced deformability of the sickled erithrocy tes. However, the involvement of vascular endothelium, platelets and their interactions with coagulation factors may also be of pathogenic significance in microvascular occlusive crises.We investigated the interaction between vascular endothelium, platelets and blood coagulation factors in 23 patients with Sickle Cell Disease (SCD) and in normal volu
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