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Books on the topic 'Signal EMG du muscle'

Author: Grafiati

Published: 8 June 2024

Last updated: 31 July 2025

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1

B, Bolton T., and Tomita T, eds. Smooth muscle excitation. Harcourt Brace, 1996.

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2

Raeburn, David, and Mark A. Giembycz, eds. Airways Smooth Muscle: Neurotransmitters, Amines, Lipid Mediators and Signal Transduction. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7504-2.

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3

Raeburn, David, and Mark A. Giembycz, eds. Airways Smooth Muscle: Peptide Receptors, Ion Channels and Signal Transduction. Birkhäuser Basel, 1995. http://dx.doi.org/10.1007/978-3-0348-7362-8.

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4

1953, Raeburn D., and Giembycz M. A. 1961-, eds. Airways smooth muscle: Neurotransmitters, amines, lipid mediators, and signal transduction. Birkhauser Verlag, 1995.

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5

Kelly, James Anthony. Aspects of signal transduction in bovine lymphatic smooth muscle cells. University College Dublin, 1996.

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6

1953-, Raeburn D., and Giembycz M. A. 1961-, eds. Airways smooth muscle: Peptide receptors, ion channels, and signal transduction. Birkhäuser Verlag, 1995.

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7

Yamada Conference on Calcium as Cell Signal (1994 Tokyo, Japan). Calcium as cell signal: Proceedings of the Yamada Conference XXXIX on Calcium as Cell Signal, April 26-28, 1994, Tokyo, Japan. Igaku-Shoin, 1996.

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8

Oldenhof, Alexandra Dianne. Effects of mechanical stretch on signal transduction and gene expression in myometrial smooth muscle cells. National Library of Canada, 2001.

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9

A, Sassoon D., ed. Stem cells and cell signalling in skeletel myogenesis. Elsevier, 2002.

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10

Maximum speed of forearm flexion practice effects upon surface EMG signal characteristics. 1985.

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11

Gunjan, Vinit Kumar, and Bita Mokhlesabadifarahani. EMG Signals Characterization in Three States of Contraction by Fuzzy Network and Feature Extraction. Springer, 2015.

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12

Gunjan, Vinit Kumar, and Bita Mokhlesabadifarahani. EMG Signals Characterization in Three States of Contraction by Fuzzy Network and Feature Extraction. Springer London, Limited, 2015.

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13

Stålberg, Erik. Electromyography. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0007.

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Electromyography (EMG) has been used since the 1940s in the diagnosis of neuromuscular disorders. It has particularly developed with the advent of computers and recording equipment with integrated software. This has made methods of analysis fast, robust, and precise, helping to deal with increasing numbers of patients. Indications have changed dynamically over the years, with the development of new EMG methods themselves and complementary methods used in this field for diagnosis such as histochemistry, genetics, and imaging techniques. This chapter focuses mainly on the routine methods based o

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14

Shaibani, Aziz. Myotonia. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0021.

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Myotonia is a slow relaxation phase after normal contraction. Patients report dystonia as muscle stiffness and sometimes pain. They usually adapt to it well. Falls due to myotonia may lead to accidents. Examination for percussion myotonia should be part of neuromuscular examination. Percussion of the thenar muscles with the reflex hammer is the most productive method. Electrically silent myotonia is a sign of Brody myopathy. Myotonia may be incidentally discovered during electromyography (EMG). The most important task is to differentiate between myotonia from paramyotonia clinically and electr

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15

Headley, Barbara J. Muscle scanning: Interpreting EMG scans. Pain Resources, 1990.

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16

Shaibani, Aziz. Myotonia. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0021.

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Myotonia is a slow relaxation phase of a muscle after normal contraction. Patients report myotonia as muscle stiffness and sometimes pain. They usually adapt to it well. Falls due to myotonia may lead to accidents. Checking for percussion and action myotonia should be part of neuromuscular examination. Electrically silent myotonia is a sign of Brody’s syndrome. Myotonia may be incidentally discovered during EMG. The most important task is to differentiate between myotonia and paramyotonia clinically and electromyographically. Most myotonic disorders are caused by mutations of sodium, and chlor

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17

Schwartz, Mark, ed. EMG Methods for Evaluating Muscle and Nerve Function. InTech, 2012. http://dx.doi.org/10.5772/1465.

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18

EMG Methods for Evaluating Muscle and Nerve Function. InTech, 2012.

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19

Trebak, Mohamed, and Scott Earley. Signal Transduction and Smooth Muscle. Taylor & Francis Group, 2021.

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20

Trebak, Mohamed, and Scott Earley. Signal Transduction and Smooth Muscle. Taylor & Francis Group, 2018.

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21

Trebak, Mohamed. Signal Transduction and Smooth Muscle. Taylor & Francis Group, 2018.

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22

Trebak, Mohamed, and Scott Earley. Signal Transduction and Smooth Muscle. Taylor & Francis Group, 2018.

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23

Trebak, Mohamed, and Scott Earley. Signal Transduction and Smooth Muscle. Taylor & Francis Group, 2018.

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24

Kennett, Robin P., and Sidra Aurangzeb. Primary muscle diseases. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0024.

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This chapter on primary muscle diseases explains how analysis of compound muscle action potential (CMAP) amplitude, abnormal spontaneous activity on needle electromyography (EMG), and motor unit action potentials (MUAP) characteristics may be used to give an indication of pathophysiological processes, and goes on to describe the combination and distribution of abnormalities that may be expected in the more commonly encountered myopathies. The conditions considered in detail are inflammatory myopathy (including myositis), critical illness myopathy, disorders with myotonia, inherited myopathy (i

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25

Shaibani, Aziz. Muscle Atrophy and Hypertrophy. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0017.

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Muscle atrophy is usually caused by interruption of axonal flow [axonal neuropathies, motor neuron diseases (MNDs), etc.]. If weakness is out of proportion to atrophy, demyelinating neuropathy should be suspected. Chronic myopathies and immobility also may cause atrophy, but no electromyography (EMG) evidence of denervation or myopathy is found. The pattern of atrophy is often helpful to localize the lesions. Atrophy of the interossi and preservation of the bulk of the thenar muscles suggest ulnar neuropathy, but atrophy of both would suggest a C8 or plexus pathology. Muscle enlargement may be

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26

Shaibani, Aziz. Muscle Atrophy and Hypertrophy. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0017.

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Muscle atrophy is usually caused by interruption of axonal flow (axonal neuropathies, motor neuron diseases, etc.). If weakness is out of proportion to atrophy, conduction block due to demyelinating neuropathy should be suspected. Chronic myopathies and immobility may also cause atrophy, but no EMG evidence of denervation or myopathy is respectively found. The pattern of atrophy is often helpful to localize the lesion. Atrophy of the interossi and preservation of the bulk of the thenar muscles suggest ulnar neuropathy, but atrophy of both would suggest a C8 or plexus pathology. Muscle enlargem

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27

Shaibani, Aziz. Muscle Twitching. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0019.

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Muscle twitching is a pianless involuntary movement of muscles, usually focal short lived. Patients may confuse it with restlessness of the legs and jerking of extremities unless specifically asked. Tremor,especially of the tongue, is also commonly confused with twitching, but its regular nature should be noticed. Fasciculations and rippling are the most important neuromuscular causes of twitching. Reproduction of the symptoms in the clinic, if possible, is very useful for the diagnosis. Otherwise, a video taken by the patient or family members showing these twitchings is equally good. Fascicu

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28

Pitt, Matthew. Needle EMG findings in different pathologies. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780198754596.003.0007.

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In this chapter, the inability of electromyography (EMG) to be able to further progress the diagnosis of myopathy on its own—requiring muscle biopsy and other modalities such as genetics to complete this process—is emphasized. The role of EMG particularly in the era of genetics is discussed. Findings in neurogenic abnormality are next described and the important hereditary conditions such as spinal muscular atrophy (SMA), distal SMA, Brown–Vialetto–Van Laere syndrome, segmental anterior horn cell disease, conditions with progressive bulbar palsy, SMARD1, and pontocerebellar hypoplasia with spi

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29

Misra, V. Peter, and Santiago Catania. EMG-guided botulinum toxin therapy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199688395.003.0026.

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This chapter explains the mechanism by which botulinum neurotoxin (BoNT) causes its neuromuscular paralytic effects, and reviews the developments that led these effects to be harnessed therapeutically. It specifically focuses upon the conditions of dystonia and spasticity. Within the spectrum of these diseases, it discusses those situations where BoNT injections are the treatment of choice. The very accurate targeting of BoNT into specific muscles in many situations is both desirable and crucial in some situations BoNT’s therapeutic neuroparalytic effect may need to be restricted to a single m

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30

Pfurtscheller, Gert, Clemens Brunner, and Christa Neuper. EEG-Based Brain–Computer Interfaces. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0047.

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A brain–computer interface (BCI) offers an alternative to natural communication and control by recording brain activity, processing it online, and producing control signals that reflect the user’s intent or the current user state. Therefore, a BCI provides a non-muscular communication channel that can be used to convey messages and commands without any muscle activity. This chapter presents information on the use of different electroencephalographic (EEG) features such as steady-state visual evoked potentials, P300 components, event-related desynchronization, or a combination of different EEG

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31

Katirji, Bashar. The Scope of the EMG Examination. Edited by Bashar Katirji. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190603434.003.0001.

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Clinical electromyography (EMG) refers to the diagnostic tool in the electrophysiological evaluation of disorders of peripheral nerve and muscle. This introductory chapter defines the terms of the discipline and its scope. Clinical EMG used in the evaluation of Clinical EMG is utilized by a variety of physicians, including specialists in the field of neurology, physical medicine and rehabilitation, orthopedics, hand surgery, neurosurgery, spine, rheumatology and pain management. The scope of the EMG Examination includes nerve conduction studies and needle EMG. It also includes other specialize

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32

The effect of short term EMG biofeedback on neck muscle relaxation for rotary pursuit performance. 1990.

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33

Li, Chien-min. The effect of short term EMG biofeedback on neck muscle relaxation for rotary pursuit performance. 1990.

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34

Raeburn, D. Ed. Airways Smooth Muscle: Neurotransmitters, Amines, Lipid Mediators & Signal Transduction (Exs). Birkhauser, 1996.

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35

Airways Smooth Muscle: Peptide Receptors, Ion Channels and Signal Transduction. Birkhäuser, 2012.

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36

Raeburn, David, and Mark A. Giembycz. Airways Smooth Muscle: Neurotransmitters, Amines, Lipid Mediators and Signal Transduction. Birkhauser Verlag, 2012.

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37

Raeburn, David, and Mark A. Giembycz. Airways Smooth Muscle: Peptide Receptors, Ion Channels and Signal Transduction. Birkhauser Verlag, 2012.

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38

Raeburn, David. Airways Smooth Muscle: Neurotransmitters, Amines, Lipid Mediators And Signal Transduction. Birkhäuser, 2012.

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39

Raeburn, David. "Airways Smooth Muscle: Peptide Receptors, Ion Channels and Signal Transduction". Springer, 2012.

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40

Kimura, Jun, and Jeffrey A. Strakowski. Electrodiagnosis in Diseases of Nerve and Muscle. 5th ed. Oxford University PressNew York, 2025. https://doi.org/10.1093/med/9780197658017.001.0001.

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Abstract Electrodiagnosis in Diseases of Nerve and Muscle: Principles and Practice provides an overview of electromyography (EMG) and electrodiagnosis. It discusses the key concepts of nerve conduction studies, electromyography, intraoperative monitoring, and somatosensory and motor evoked potential. Jun Kimura’s fifth edition of Electrodiagnosis in Diseases of Nerve and Muscle offers a comprehensive text on electromyography that will be useful to both beginning practitioners and experts alike. The fifth edition is expected to be important as a world-class reference in the subspecialty of neur

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41

Calcium as cell signal: Proceedings of the Yamada Conference XXXIX on Calcium as Cell Signal, April 26-28, 1994, Tokyo, Japan. Igaku-Shoin, 1995.

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42

Fashanu, Billy. Exploring EMG-Torque relationship in the quadriceps femoris and the hamstring muscle group and muscle activity duringthe sit-to-stand movement in female subjects: A methodological study. UEL, 1994.

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43

Raeburn, D. Airways Smooth Muscle: Peptide Receptors, Ion Channels, and Signal Transduction (Agents and Actions Supplements). Birkhauser, 1995.

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44

(Editor), E. Raeburn, and M. A. Giembycz (Editor), eds. Airways Smooth Muscle: Peptide Receptors, Ion Channels and Signal Transduction (Respiratory Pharmacology and Pharmacotherapy). Birkhauser Boston, 1995.

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45

Airways Smooth Muscle: Neurotransmitters, Amines, Lipid Mediators and Signal Transduction (Respiratory Pharmacology and Pharmacotherapy). Birkhauser Boston, 1995.

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46

Nuwer, Marc R., Ronald G. Emerson, and Cecil D. Hahn. Principles and Techniques for Long-Term EEG Recording (Epilepsy Monitoring Unit, Intensive Care Unit, Ambulatory). Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0031.

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Long-term monitoring is a set of methods for recording electroencephalographic (EEG) signals over a period of 24 hours or longer. Patient video recording is often synchronized to the EEG. Interpretation aids help physicians to identify events, which include automated spike and seizure detection and various trending displays of frequency EEG content. These techniques are used in epilepsy monitoring units for presurgical evaluations and differential diagnosis of seizures versus nonepileptic events. They are used in intensive care units to identify nonconvulsive seizures, to measure the effective

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47

Shaibani, Aziz. Proximal Arm Weakness. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199898152.003.0012.

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Proximal arm muscles include supra and infra spinatii, pectoralis major and minor, teres major and minor, rhomboids, serratus anterior, deltoids, biceps, and triceps. The main function of these muscles is to lift the arms. The first sign of proximal weakness is difficulty in raising the arms above a horizontal level. Shoulder conditions like supraspinatus tendonitis are often confused as proximal weakness. In myopathies, usually proximal arm weakness is associated with proximal leg weakness. Motor neuron diseases like ALS and SMA and neuropathies like CIDP may present with symmetrical proximal

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48

Sassoon, D. A. Stem Cells and Cell Signalling in Skeletal Myogenesis (Advances in Developmental Biology and Biochemistry, V. 11). Elsevier Science, 2002.

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49

Shaibani, Aziz. Proximal Arm Weakness. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780190661304.003.0012.

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Proximal arm muscles include supra and infra spinatii, pectoralis major and minor, teres major and minor, rhomboids, serratus anterior, deltoids, biceps, and triceps. The main function of these muscles is to abduct the arms. The first sign of proximal weakness is difficulty raising arms above the horizontal level. Shoulder conditions like supraspinatus tendonitis are often confused as proximal weakness. In myopathies, usually proximal arm weakness is associated with proximal leg weakness. Motor neuron diseases (MNDs) like amyotrophic lateral sclerosis (ALS) and spinal muscular atrophy (SMA) an

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50

Chest Pain: Education for Patients and the Public. Exon Publications, 2025. https://doi.org/10.36255/chest-pain.

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Chest pain is a common symptom that can range from mild discomfort to a sign of a serious medical emergency. This article provides a comprehensive guide to understanding chest pain, its causes, symptoms, diagnosis, treatment, and management. It explains how chest pain can originate from the heart, lungs, digestive system, muscles, or nerves, making it essential to recognize when the pain is harmless and when it requires urgent medical attention. The article highlights common causes such as heart disease, acid reflux, lung conditions, and muscle strain, detailing their symptoms and risk factors

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