Relevant bibliographies by topics / Signalling protein MHC Class I

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  1. Journal articles
  2. Dissertations / Theses
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  4. Conference papers

Academic literature on the topic 'Signalling protein MHC Class I'

Author: Grafiati
Published: 4 June 2021
Last updated: 18 February 2022

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Journal articles on the topic "Signalling protein MHC Class I"

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Slade, J. W. G., M. J. Watson, T. R. Kelly, G. B. Gloor, M. A. Bernards, and E. A. MacDougall-Shackleton. "Chemical composition of preen wax reflects major histocompatibility complex similarity in songbirds." Proceedings of the Royal Society B: Biological Sciences 283, no. 1842 (2016): 20161966. http://dx.doi.org/10.1098/rspb.2016.1966.

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In jawed vertebrates, genes of the major histocompatibility complex (MHC) play a key role in immunity by encoding cell-surface proteins that recognize and bind non-self antigens. High variability at MHC suggests that these loci may also function in social signalling such as mate choice and kin recognition. This requires that MHC genotype covaries with some perceptible phenotypic trait. In mammals and fish, MHC is signalled chemically through volatile and non-volatile peptide odour cues, facilitating MHC-dependent mate choice and other behaviours. In birds, despite evidence for MHC-dependent ma
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Ellwanger, Kornelia, Christine Kienzle, Sylke Lutz, et al. "Protein kinase D controls voluntary-running-induced skeletal muscle remodelling." Biochemical Journal 440, no. 3 (2011): 327–35. http://dx.doi.org/10.1042/bj20101980.

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Skeletal muscle responds to exercise by activation of signalling pathways that co-ordinate gene expression to sustain muscle performance. MEF2 (myocyte enhancer factor 2)-dependent transcriptional activation of MHC (myosin heavy chain) genes promotes the transformation from fast-twitch into slow-twitch fibres, with MEF2 activity being tightly regulated by interaction with class IIa HDACs (histone deacetylases). PKD (protein kinase D) is known to directly phosphorylate skeletal muscle class IIa HDACs, mediating their nuclear export and thus derepression of MEF2. In the present study, we report
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Krummel, Matthew, Christoph Wülfing, Cenk Sumen, and Mark M. Davis. "Thirty–six views of T–cell recognition." Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 355, no. 1400 (2000): 1071–76. http://dx.doi.org/10.1098/rstb.2000.0644.

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While much is known about the signalling pathways within lymphocytes that are triggered during activation, much less is known about how the various cell surface molecules on T cells initiate these events. To address this, we have focused on the primary interaction that drives T–cell activation, namely the binding of a particular T–cell receptor (TCR) to peptide–MHC ligands, and find a close correlation between biological activity and off–rate; that is, the most stimulatory TCR ligands have the slowest dissociation rates. In general, TCRs from multiple histocompatibility complex (MHC) class–II–
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SINGH, MANISH K., FAUZIA JAMAL, AMIT K. DUBEY, et al. "Co-factor-independent phosphoglycerate mutase of Leishmania donovani modulates macrophage signalling and promotes T-cell repertoires bearing epitopes for both MHC-I and MHC-II." Parasitology 145, no. 3 (2017): 292–306. http://dx.doi.org/10.1017/s0031182017001494.

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SUMMARYImmunoactivation depends upon the antigen potential to modulate T-cell repertoires. The present study has enumerated the effect of 61 kDa recombinant Leishmania donovani co-factor-independent phosphoglycerate mutase (rLd-iPGAM) on mononuclear cells of healthy and treated visceral leishmaniasis subjects as well as on THP-1 cell line. rLd-iPGAM stimulation induced higher expression of interleukin-1β (IL-1β) in the phagocytic cell, its receptor and CD69 on T-cell subsets. These cellular activations resulted in upregulation of host-protective cytokines IL-2, IL-12, IL-17, tumour necrosis fa
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Athanassakis, Irene. "Analysis of the IFN-γ-induced signal transduction pathway in fetal rejection". Mediators of Inflammation 4, № 3 (1995): 209–16. http://dx.doi.org/10.1155/s0962935195000342.

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The placenta, one of the most important fetal tissues during gestation, ensures nutrition, development and protection of the fetus. Although placenta lacks expression of class II MHC antigens, they can be induced either by interferon-gamma (IFN-γ) on the spongiotrophoblast zone, or by 5-azacytidine (5-azaC) on the labyrinthine trophoblast zone, two agents actively participating in a plethora of immunological and inflammatory reactions. This induction is correlated with fetal abortion and fetal developmental abnormalities. In this work thein vitroandin vivosignal transduction pathways followed
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Fairley, J. A., J. Baillie, M. Bain, and J. H. Sinclair. "Human cytomegalovirus infection inhibits epidermal growth factor (EGF) signalling by targeting EGF receptors." Journal of General Virology 83, no. 11 (2002): 2803–10. http://dx.doi.org/10.1099/0022-1317-83-11-2803.

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Infection with human cytomegalovirus (HCMV) is known to involve complex interactions between viral and cellular factors resulting in perturbation of a number of cellular functions. Specifically, HCMV infection targets control of the cell cycle, cellular transcription and immunoregulation, presumably to optimize the cellular environment for virus persistence and productive infection. Here, we show that HCMV infection also prevents external signalling to the cell by disrupting the function of epidermal growth factor receptor (EGFR). Infection with HCMV resulted in a decrease in cell-surface expr
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Vassiliadis, S., N. Kyrpides, D. Stravopodis, M. Grigoriou, I. Athanassakis та J. Papamatheakis. "Investigation of intracellular signals generated by γ-interferon and IL-4 leading to the induction of class II antigen expression". Mediators of Inflammation 2, № 5 (1993): 343–48. http://dx.doi.org/10.1155/s096293519300047x.

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Signal transduction plays a vital role in cellular behaviour as cells respond to various stimuli in different ways and utilize diverse pathways for accomplishing their task. Determination of the pathway followed by various cytokines can be achieved using specific inhibitors which include theophylline (TPH), TMB-8 and W7 that hinder calmodulin binding to Ca2+; sphingosine (SPH), H7 and staurosporine that inhibit protein kinase C (PKC) activation; and mevalonate (MEV) or the anti-p21rasantibody which block G-proteins. This study shows that the immunologically important class II antigens in human
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Tan, H., J. A. West, J. P. Ramsay, et al. "Comprehensive overexpression analysis of cyclic-di-GMP signalling proteins in the phytopathogen Pectobacterium atrosepticum reveals diverse effects on motility and virulence phenotypes." Microbiology 160, no. 7 (2014): 1427–39. http://dx.doi.org/10.1099/mic.0.076828-0.

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Bis-(3′-5′)-cyclic dimeric guanosine monophosphate (c-di-GMP) is a ubiquitous bacterial signalling molecule produced by diguanylate cyclases of the GGDEF-domain family. Elevated c-di-GMP levels or increased GGDEF protein expression is frequently associated with the onset of sessility and biofilm formation in numerous bacterial species. Conversely, phosphodiesterase-dependent diminution of c-di-GMP levels by EAL- and HD-GYP-domain proteins is often accompanied by increased motility and virulence. In this study, we individually overexpressed 23 predicted GGDEF, EAL or HD-GYP-domain proteins enco
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van Loenen, Marleen M., Renate S. Hagedoorn, Roelof Willemze, J. H. Frederik Falkenburg та Mirjam H. M. Heemskerk. "Extracellular Domains of CD8a and β Subunits Are Required and Sufficient for HLA Class I Restricted Helper Activity of TCR-Engineered CD4+ T Cells." Blood 114, № 22 (2009): 3574. http://dx.doi.org/10.1182/blood.v114.22.3574.3574.

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Abstract Abstract 3574 Poster Board III-511 Adoptive transfer of T cell receptor (TCR)-transferred T cells may be an attractive strategy to treat patients with hematological malignancies relapsing after allogeneic stem cell transplantation. Transfer of HLA class I restricted TCRs into CD8+ T cells demonstrated redirected antigen specificity. However, for persistence of anti-leukemic responses in vivo, CD4+ T cells may be important. Therefore, redirecting specificity of CD4+ T cells with well defined HLA class I restricted TCRs might be an attractive strategy for providing help. HLA class I res
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Lacey, Melissa M., Jonathan D. Partridge, and Jeffrey Green. "Escherichia coli K-12 YfgF is an anaerobic cyclic di-GMP phosphodiesterase with roles in cell surface remodelling and the oxidative stress response." Microbiology 156, no. 9 (2010): 2873–86. http://dx.doi.org/10.1099/mic.0.037887-0.

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The Escherichia coli K-12 yfgF gene encodes a protein with domains associated with cyclic di-GMP signalling: GGDEF (associated with diguanylate cyclase activity) and EAL (associated with cyclic di-GMP phosphodiesterase activity). Here, it is shown that yfgF is expressed under anaerobic conditions from a class II FNR (regulator of fumarate and nitrate reduction)-dependent promoter. Anaerobic expression of yfgF is greatest in stationary phase, and in cultures grown at 28 °C, suggesting that low growth rates promote yfgF expression. Mutation of yfgF resulted in altered cell surface properties and
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Dissertations / Theses on the topic "Signalling protein MHC Class I"

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Kress, Alla. "Probing molecular orientational order of lipid reporters and MHC Class I protein in cell membranes using polarization-resolved fluorescence imaging." Thesis, Aix-Marseille 3, 2011. http://www.theses.fr/2011AIX30046.

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L'organisation orientationnelle bio-moléculaire des lipides et des protéines dans la membrane plasmique constitue un facteur important dans les processus biologiques au cours desquelles les fonctions peuvent être reliées aux mécanismes d'orientation et d'organisation. Le concept de séparation transitoire des phases à l'échelle nanométrique dans les domaines ordonnés et désordonnés, aussi appelé « radeau lipidique », est maintenant largement accepté. De plus, les domaines ordonnés contiennent des protéines de signalisation, ce qui souligne l'importance des séparations de phase au cours des proc
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Persson, Karina. "Structural studies of protein assemblies : MHC class I and lumazine synthase /." Uppsala : Swedish Univ. of Agricultural Sciences (Sveriges lantbruksuniv.), 1999. http://epsilon.slu.se/avh/1999/91-576-5499-9.pdf.

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Gruneberg, Ulrike. "The interaction of HLA-DM with conventional MHC class II molecules." Thesis, University College London (University of London), 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.322410.

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Bandyopadhyay, Arunima. "Investigating the binding interactions between peptides and the MHC class II protein I-A(k) /." Thesis, Connect to this title online; UW restricted, 2005. http://hdl.handle.net/1773/8561.

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McFarland, Benjamin James. "Dissecting the cooperative energetics of the binding interactions between peptides and MHC class II proteins /." Thesis, Connect to this title online; UW restricted, 2001. http://hdl.handle.net/1773/8540.

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Lindgren, Cecilia. "Design and Synthesis of Glycopeptides Targeting the Class II MHC DR4 Protein Associated with Rheumatoid Arthritis." Thesis, Umeå universitet, Kemiska institutionen, 2012. http://urn.kb.se/resolve?urn=urn:nbn:se:umu:diva-61847.

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Ghosh, Shohini. "Analysis of the binding interactions between peptides and the MHC class II protein I-A(d) /." Thesis, Connect to this title online; UW restricted, 2004. http://hdl.handle.net/1773/8541.

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Parkinson, Michael David James. "The protein machinery in the endocytic pathway required for downregulation of virally ubiquitinated MHC class I." Thesis, University of Cambridge, 2010. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.608859.

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Michaëlsson, Jakob. "Decoding NK cell receptor specificity : functional and structural studies of MHC class 1 subcomponents /." Stockholm : [Karolinska institutets bibl.], 2002. http://diss.kib.ki.se/2002/91-7349-286-8.

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Murthy, Venkatesh Locharla. "Three dimensional structure of a human Class II MHC protein HLA-DR1 bound to an endogenous peptide." Thesis, Massachusetts Institute of Technology, 1996. http://hdl.handle.net/1721.1/10561.

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Book chapters on the topic "Signalling protein MHC Class I"

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Hunt, Donald F., Jeffrey Shabanowitz, Hanspeter Michel, et al. "Sequence Analysis of Peptides Presented to the Immune System by Class I and Class II MHC Molecules." In Methods in Protein Sequence Analysis. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4899-1603-7_16.

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Gorga, Joan C., Aichun Dong, Mark C. Manning, Robert W. Woody, Winslow S. Caughey, and Jack L. Strominger. "Comparison of the Secondary Structures of Human Class I and Class II MHC Antigens by Ftir and CD Spectroscopy." In Protein Structure and Engineering. Springer US, 1989. http://dx.doi.org/10.1007/978-1-4684-5745-2_23.

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Appella, E., J. G. Omichinski, G. M. Clore, A. M. Gronenborn, and K. Sakaguchi. "Zinc Fingers Involved in MHC Class I Gene Regulation: Use of Synthetic Peptides for Structural Analysis." In Methods in Protein Sequence Analysis. Birkhäuser Basel, 1991. http://dx.doi.org/10.1007/978-3-0348-5678-2_18.

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Gradehandt, Gernot, Johannes Hampl, Nadja Kleber, et al. "Requirements of Exogenous Protein Antigens for Presentation to CD4+ T lymphocytes By MHC Class II-Positive APC." In Advances in Experimental Medicine and Biology. Springer US, 1993. http://dx.doi.org/10.1007/978-1-4615-2930-9_4.

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Wagner, Barbara, Karl-Hermann Schmidt, Bernhard Fleischer, Werner Reichardt, and Manfred Wagner. "Group A Streptococcal M Protein Binds to Several Human Cell Types but not via MHC Class II Molecules." In Streptococci and the Host. Springer US, 1997. http://dx.doi.org/10.1007/978-1-4899-1825-3_129.

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Cox, J. H., J. R. Bennink, L. C. Eisenlohr, and J. W. Yewdell. "Blockade of MHC Class I Molecule Transport from the Endoplasmic Reticulum Inhibits Presentation of Protein Antigens to Cytotoxic T Lymphocytes." In Progress in Immunology. Springer Berlin Heidelberg, 1989. http://dx.doi.org/10.1007/978-3-642-83755-5_125.

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Berzofsky, J. A., J. Cornette, H. Margalit, I. Berkower, K. Cease, and C. DeLisi. "Molecular Features of Class II MHC-Restricted T-Cell Recognition of Protein and Peptide Antigens: The Importance of Amphipathic Structures." In Current Topics in Microbiology and Immunology. Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-71440-5_2.

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Tuchscherer, G., C. Servis, G. Corradin, U. Blum, J. Rivier, and M. Mutter. "Total chemical synthesis, characterization and immunological properties of a MHC class I model using the TASP concept for protein de novo design." In Peptides 1992. Springer Netherlands, 1993. http://dx.doi.org/10.1007/978-94-011-1470-7_389.

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Hassan, MD Imtaiyaz, and Faizan Ahmad. "Structural diversity of class I MHC-like molecules and its implications in binding specificities." In Protein Structure and Diseases. Elsevier, 2011. http://dx.doi.org/10.1016/b978-0-12-381262-9.00006-9.

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Rock, Kenneth L., Ian A. York, Tomo Saric, and Alfred L. Goldberg. "Protein degradation and the generation of MHC class I-presented peptides." In Advances in Immunology. Elsevier, 2002. http://dx.doi.org/10.1016/s0065-2776(02)80012-8.

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Conference papers on the topic "Signalling protein MHC Class I"

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Gentaro, Ito, Tanaka Hiroaki, Yoshi Mami, et al. "Abstract 2687: Immunopotentiator, protein-bound polysaccharide K, is efficient for advanced gastric cancer with negative expression of MHC Class I." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-2687.

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Fromm, George J., Danish Memon, Suresh de Silva, et al. "Abstract 1697: The development of an in vivo model of checkpoint acquired resistance, reveals a program of interferon hyperstimulation, resulting in dysregulation of MHC class I, protein translation/trafficking, and other unique pathways, that may be useful for guiding clinical strategy in patients with phenotypic similarities." In Proceedings: AACR Annual Meeting 2021; April 10-15, 2021 and May 17-21, 2021; Philadelphia, PA. American Association for Cancer Research, 2021. http://dx.doi.org/10.1158/1538-7445.am2021-1697.

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Teitel, J. M., A. Shore, and J. McBarron. "REGULATION OF INTERLEUKIN-2 (IL-2) PRODUCTION BY ENDOTHELIAL CELL (EC) DERIVED SUBSTANCES." In XIth International Congress on Thrombosis and Haemostasis. Schattauer GmbH, 1987. http://dx.doi.org/10.1055/s-0038-1642865.

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We recently reported that vascular EC support the staphylococcal protein A (SPA)-induced production of IL-2 by T cells, and furthermore do so synergistically with monocytes. We have now assessed the contributions of EC membrane-associated and secreted factors to these functions. IL-2 was measured by either human tonsil blast of CTLL cell assay. Separation of EC from T cells by a permeable membrane (.45μ pore size) prevented IL-2 production. Consistent with this, supernatant from resting EC (ECs) was also unable to induce IL-2 generation. In addition, neither a crude EC plasma membrane preparat
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