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Journal articles on the topic 'Silent tutor'

Author: Grafiati

Published: 7 June 2025

Last updated: 2 August 2025

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1

Danku, Benedicta Enid Mawuse. "EVALUATING ORGANISATIONAL FACTORS AND TUTOR BURNOUT IN AKATSI COLLEGE OF EDUCATION, GHANA." International Journal of Applied Research in Social Sciences 4, no. 5 (2022): 190–202. http://dx.doi.org/10.51594/ijarss.v4i5.357.

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Pickering (2008) observed that administration is a major determinant of a teacher’s perception of stress in the school. Besides, work overload is one of the factors causing burnout among teachers that causes reduced physical and emotional energy (Malik, 2019). The study sought to assess the extent to which administrative support and workload contribute to tutor burnout. The study employed descriptive survey design. A structured questionnaire was used to collect data from thirty-five tutors who were purposively sampled for the study. Concerning administrative support, the Principal often explai

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Crandall, Shane R., Murtaza Adam, Amanda K. Kinnischtzke, and Teresa A. Nick. "HVC Neural Sleep Activity Increases With Development and Parallels Nightly Changes in Song Behavior." Journal of Neurophysiology 98, no. 1 (2007): 232–40. http://dx.doi.org/10.1152/jn.00128.2007.

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Sleep abnormalities are coexpressed with human communication disorders. Recent data from the birdsong system, the best model for human speech, indicate that sleep has a critical role in vocal learning. To understand the neural mechanisms that underlie behavioral changes during sleep, we recorded sleep activity in the song control area HVC longitudinally during song development in zebra finches. We focused on the sensorimotor phase of song learning, when the finch shapes his song behavior toward a learned tutor song model. Direct comparison of sleep activity in adults and juveniles revealed tha

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Tingang, Laurensius, Aunurrahman Aunurrahman, and Ageung Darajat. "Speaking Anxiety of Students of Parit Baru English Village." Journal of English Language Teaching and Education (JELTE) 3, no. 2 (2022): 1–11. http://dx.doi.org/10.31571/jelte.v3i2.148.

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This Research aims to find out the kinds and sources of speaking anxiety of the Students of English Parit Baru Village, Sungai Raya Regency in the academic year of 2021. This research used the descriptive qualitative method. Ten students from different grades of primary school around Parit Baru village were involved in this research. This research used a semi-structured interview and field note as the tool of collection data. data collection used an observation about students’ behavior during class that direct students to become Anxiety. The result showed that the kinds of speaking anxiety of

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Bottjer, Sarah W. "Silent Synapses in a Thalamo-Cortical Circuit Necessary for Song Learning in Zebra Finches." Journal of Neurophysiology 94, no. 6 (2005): 3698–707. http://dx.doi.org/10.1152/jn.00282.2005.

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Developmental changes in synaptic properties may act to limit neural and behavioral plasticity associated with sensitive periods. This study characterized synaptic maturation in a glutamatergic thalamo-cortical pathway that is necessary for vocal learning in songbirds. Lesions of the projection from medial dorsolateral nucleus of the thalamus (DLM) to the cortical nucleus lateral magnocellular nucleus of the anterior nidopallium (LMAN) greatly disrupt song behavior in juvenile birds during early stages of vocal learning. However, such lesions lose the ability to disrupt vocal behavior in norma

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Budiharto, Agus. "The Role of Silent Way Method to English Teaching in a Private Islamic Middle School." Jurnal SOLMA 7, no. 2 (2018): 161. http://dx.doi.org/10.29405/solma.v7i2.1396.

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In the third millennium of quickly globalized world, teaching English as a Foreign Language (EFL) in Indonesia has become a considerable important thing. In this regard, the English teacher plays a key role to attain success in the process of EFL teaching-learning in the classroom. To reach it, the teacher surely needs an appropriate and effective teaching method to be implemented. The present investigation attempts to discuss concerning the roles of Silent Way Method (SWM) as the appropriate method to English teaching. The SWM was implemented in MTS Mifatahul Ulum Pagendingan, as one of priva

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Shinde, Ms. Cynthia Nitin. "AI-Driven Education: Balancing Cognitive Enhancement and Creative Thinking." International Journal of Advance and Applied Research 6, no. 25 (2025): 188–92. https://doi.org/10.5281/zenodo.15295061.

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<strong>Abstract:</strong> Technology is climbing the ladder of rapid advancement, and at its core lies artificial intelligence, a transformative force reshaping industries and redefining human potential. In the realm of education, AI-driven tools are emerging as game changers, offering personalized learning experiences, instant feedback, and adaptive teaching methodologies. With AI seamlessly integrated into classrooms, students can access information effortlessly, sharpen problem-solving skills, and cultivate critical thinking. However, as AI becomes a silent tutor in students' academic jour

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Mostow, Jack, Jessica Nelson-Taylor, and Joseph E. Beck. "Computer-Guided Oral Reading versus Independent Practice: Comparison of Sustained Silent Reading to an Automated Reading Tutor That Listens." Journal of Educational Computing Research 49, no. 2 (2013): 249–76. http://dx.doi.org/10.2190/ec.49.2.g.

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Tsai, Shu-Chiao. "Courseware integration into task-based learning: a case study of multimedia courseware-supported oral presentations for non-English major students." ReCALL 23, no. 2 (2011): 117–34. http://dx.doi.org/10.1017/s0958344011000048.

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AbstractThis study reports on the integration of English for Specific Purposes (ESP) multimedia courseware for oral presentations into a self-learning and elective program for non-English major students in an English as a Foreign Language (EFL) setting. A computer-aided instruction approach, combined with a task-based learning approach, was adopted. Computers played a central role as the means of information delivery. The courseware acted as a silent partner and played the role of a tutor and adjunct teacher to provide students with authentic materials for learning to give English oral present

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Allen, Ira. "Falling Apart Together." Screen Bodies 2, no. 2 (2017): 78–90. http://dx.doi.org/10.3167/screen.2017.020206.

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I could not hear weeping as the screen fell dark. But the glistening cheeks and too-bright eyes spoke loudly enough when the lights rose. We had confronted together a loss of revolutionary possibility, and a refusal of that same loss, that touched nearly every body in the theater. To view Syrian filmmaker Mohammad Ali Atassi’s Our Terrible Country (2014) in Beirut, scarcely seventy-five miles from a Damascus whose dying and lost flowed continuously into Lebanon even as we watched for Syrian hope on the screen, was to be swept up in the civil war’s terrible pull of collective suffering and loss

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Dhar, Arjun, and Apurva Prasad. "Silent Spinal Cord Tumor." Journal of Spinal Surgery 4, no. 1 (2017): 22–25. http://dx.doi.org/10.5005/jp-journals-10039-1120.

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Necek, Magdalena, and Ewelina Biskup. "The Silent Giant." Praxis 104, no. 24 (2015): 1343–45. http://dx.doi.org/10.1024/1661-8157/a002197.

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Stiller Riese Zusammenfassung. Der solitäre fibröse Tumor (SFT) ist eine seltene Neoplasie. Am häufigsten nimmt er seinen Ausgang von der pulmonalen Pleura. Klinische Symptome treten oft erst auf, nachdem der Tumor eine erstaunliche Dimension erreicht hat und benachbarte Strukturen komprimiert. Die Diagnose erfolgt (immuno-) histologisch. Therapeutisch steht die chirurgische Totalresektion im Vordergrund. Wichtig ist eine langfristige und regelmässige Nachsorge, da Rezidive vorkommen und mit einer schlechten Prognose assoziiert sind.

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Ioachimescu, Adriana G., Leslie Eiland, Vaninder S. Chhabra, et al. "Silent Corticotroph Adenomas." Neurosurgery 71, no. 2 (2012): 296–304. http://dx.doi.org/10.1227/neu.0b013e318257c1f0.

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Abstract BACKGROUND: Silent corticotroph adenomas (SCAs) are clinically nonfunctioning pituitary adenomas (NFPAs) with positive staining for corticotropin (ACTH) by immunohistochemistry. Whether SCAs behave more aggressively than NFPAs without ACTH immunoreactivity (ACTH negative) remains controversial. OBJECTIVE: To compare characteristics and outcomes of SCAs with ACTH-negative NFPAs and to identify predictors of aggressive outcome. Primary composite endpoint included the first of any of the following events: progression, recurrence, or death. METHODS: We reviewed all cases of SCAs and all A

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Zhou, Shuaiyang, Rui Zhan, Zhenguo Qiao, and Jianzhong Wu. "Giant solitary fibrous tumor: A clinically silent tumor." Asian Journal of Surgery 44, no. 8 (2021): 1085–86. http://dx.doi.org/10.1016/j.asjsur.2021.05.018.

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Noorulla, Anisa, Hemalatha Ramakrishna, Kandasamy Murugan, and Duraiselvi Paneerselvam. "Diagnostic dilemma of a silent tumor." Journal of Indian Academy of Oral Medicine and Radiology 32, no. 3 (2020): 315. http://dx.doi.org/10.4103/jiaomr.jiaomr_153_19.

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Gil, Júlio, Bruno Marmelo, Luís Abreu, Hugo Antunes, Luís Ferreira dos Santos, and José Costa Cabral. "Silent cardiac tumor with neurological manifestations." Journal of Cardiology Cases 15, no. 4 (2017): 132–35. http://dx.doi.org/10.1016/j.jccase.2016.12.005.

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Kumar, Supreet, Rigved Gupta, Neerav Goyal, Alok Kumar Pandey, Vivek Tandon, and Neerav Goyal. "Silent neuroendocrine tumor of biliary confluence." International Surgery Journal 11, no. 3 (2024): 502–6. http://dx.doi.org/10.18203/2349-2902.isj20240586.

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Neuroendocrine tumors (NETs) of the extrahepatic bile ducts are extremely rare. They are heterogeneous entities of varied histopathological features. The pathological types vary greatly with regards to biological behavior and prognosis. We presented such a case of silent tumor of the biliary tract of neuroendocrine origin in a middle-aged female with nonspecific abdominal pain without any clinically obvious signs which on radiological imaging revealed a tumor of hepatic confluence with metastatic deposits in liver for which she underwent left trisectionectomy with segment 7 metastatectomy. As

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Zhang, Dongyun, Willy Hugo, Marvin Bergsneider, et al. "New Transcriptional Insights into Silent and Active Corticotroph Pituitary Tumors at Single Cell Resolution." Journal of the Endocrine Society 5, Supplement_1 (2021): A646—A647. http://dx.doi.org/10.1210/jendso/bvab048.1318.

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Abstract Silent pituitary corticotroph tumors derive from the Tpit (aka TBX19) pituitary lineage. Accounting for ~ 30% of corticotroph tumors, they are not infrequently clinically aggressive and invade locally into adjacent sellar structures, making complete surgical resection challenging and contributing to their higher recurrence rates. How silent and active corticotroph tumor subtypes differ is not clear although some studies reported that silent corticotroph tumors exhibit reduced PC1 expression causing impaired POMC processing. We used single cell RNAseq to compare the transcriptome betwe

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García-Martínez, A., D. A. Cano, A. Flores-Martínez, et al. "Why don’t corticotroph tumors always produce Cushing’s disease?" European Journal of Endocrinology 181, no. 3 (2019): 351–61. http://dx.doi.org/10.1530/eje-19-0338.

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Objective Silent corticotroph tumors are a pituitary neuroendocrine tumor subtype of corticotroph lineage that do not clinically express Cushing’s disease. The silencing of this type of tumor is not fully understood. The aim of the present study was to delve into the lack of secretory activity, studying the post-transcriptional and post-translational regulation of POMC/ACTH in a series of molecularly identified functioning and silent corticotroph tumors. Design We analyzed 24 silent corticotroph, 23 functioning corticotroph and 25 silent gonadotroph tumors. Methods We used Sanger sequencing, q

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Ricklefs, Franz, Krys Fita, Roman Rotermund, Manfred Westphal, Ulrich Schüller, and Joerg Flitsch. "GENE-23. GENOME-WIDE DNA METHYLATION PROFILES DISTINGUISH SILENT FROM NON-SILENT ACTH ADENOMAS." Neuro-Oncology 21, Supplement_6 (2019): vi102. http://dx.doi.org/10.1093/neuonc/noz175.425.

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Abstract BACKGROUND Corticotroph adenomas are immunopositive for adrenocorticotrophic hormone (ACTH) associated with elevated blood ACTH levels leading to Cushing disease (CD). Yet silent ACTH adenomas (SCA) immunostain for ACTH but do not cause hypercortisolism. SCA have consistently been shown to have a more aggressive postoperative course, then nonfunctioning and ACTH adenomas. Here we show that genome-wide methylation profiles can be used to distinguish ACTH adenomas from SCA. METHODS16 SCA patients and 19 CD patients that underwent transsphenoidal resection were included. Tumor size was m

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Zhou, LanLan, David T. Dicker, Elizabeth Matthew, Wafik S. El-Deiry, and R. Katherine Alpaugh. "Circulating tumor cells: silent predictors of metastasis." F1000Research 6 (August 14, 2017): 1445. http://dx.doi.org/10.12688/f1000research.11313.1.

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Circulating tumor cells (CTCs) were added to the arsenal of clinical testing in 2004 for three cancer types: metastatic breast, prostate, and colorectal cancer. CTCs were found to be an independent prognostic indicator of survival for these three diseases. Multiple enrichment/isolation strategies have been developed and numerous assay applications have been performed using both single and pooled captured/enriched CTCs. We have reviewed the isolation techniques and touched on many analyses. The true utility of a CTC is that it acts as a “silent” predictor of metastatic disease. The mere presenc

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Catalano, Martina, Giandomenico Roviello, Raffaella Santi, et al. "Inflammation in Urological Malignancies: The Silent Killer." International Journal of Molecular Sciences 24, no. 1 (2023): 866. http://dx.doi.org/10.3390/ijms24010866.

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Several studies have investigated the role of inflammation in promoting tumorigenesis and cancer progression. Neoplastic as well as surrounding stromal and inflammatory cells engage in well-orchestrated reciprocal interactions to establish an inflammatory tumor microenvironment. The tumor-associated inflammatory tissue is highly plastic, capable of continuously modifying its phenotypic and functional characteristics. Accumulating evidence suggests that chronic inflammation plays a critical role in the development of urological cancers. Here, we review the origins of inflammation in urothelial,

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Bantu, Sravani, Shirisha R. Vallepu, Mouna Gunda, and Vaishali Thudi. "Incidental Pheochromocytoma: Silent but Violent." Journal of the Endocrine Society 5, Supplement_1 (2021): A137—A138. http://dx.doi.org/10.1210/jendso/bvab048.277.

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Abstract Background: Pheochromocytoma is a rare catecholamine secreting neuroendocrine tumor. It arises from the chromaffin cells of adrenal medulla. It is diagnosed in 5–6.5% of adrenal incidentalomas which is not common. The usual clinical presentation includes the classic triad of sweating, headache and tachycardia. However, asymptomatic cases are seen in 8% of the patients with pheochromocytoma. We present a clinically asymptomatic patient diagnosed during work up of adrenal incidentaloma. The possible etiology for silent presentation includes one of the following:(i) Presence of a smaller

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Levy, S., E. Mendel, S. Kon, Z. Avnur, and R. Levy. "Mutational hot spots in Ig V region genes of human follicular lymphomas." Journal of Experimental Medicine 168, no. 2 (1988): 475–89. http://dx.doi.org/10.1084/jem.168.2.475.

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The genes coding for the Ig light chains expressed in two cases of human follicular lymphoma were cloned and sequenced. In each case, multiple independent isolates of the tumor population were compared. Although each tumor represented a single clone of B cells with a unique V/J joint, different cells within each tumor had accumulated multiple point mutations in the V gene during clonal expansion. Most of the mutations observed were silent, but some resulted in amino acid replacements. Identical silent mutations were often observed in independent isolates of each tumor. By combining the current

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Chinezu, Laura, Emmanuel Jouanneau, Alexandre Vasiljevic, Jacqueline Trouillas, and Gérald Raverot. "Silent GH pituitary tumor: Diagnostic and therapeutic challenges." Annales d'Endocrinologie 74, no. 5-6 (2013): 491–95. http://dx.doi.org/10.1016/j.ando.2013.09.003.

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Cao, L. T. "Mystery of tumor specific immunity: LIGHT turns on silent tumor specific immunity." Journal of Clinical Oncology 24, no. 18_suppl (2006): 20107. http://dx.doi.org/10.1200/jco.2006.24.18_suppl.20107.

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20107 Background: More than 2000 tumor antigens have been identified so far. However, none of these tumor antigens are approved to be immunogenic. Genetic alteration which activates uncontrolled cell growth transforms normal cells to tumor cells. Tumor cells, therefore, do not carry any foreign antigens and thus tumor antigens are not immunogenic (unless in the presence of viral infection). The so-called tumor specific immunity is actually the host versus graft effect, not specific for tumors. Methods and Results: Using proper controlled tissue graft as control, our previous data indicated tha

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Hind, Asbar, Rafi Sana, El Mghari Ghizlane, and El Ansari Nawal. "A rare case of silent pituitary macroadenoma with positive TSH and prolactin immunostaining." World Journal of Advanced Research and Reviews 14, no. 2 (2022): 112–14. https://doi.org/10.5281/zenodo.7186126.

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Silent thyroid stimulating hormone (TSH)-immunostaining pituitary adenomas are rare tumors, they can be either pure or immunoreactive to other pituitary hormones. We report a case of a silent macroadenoma with both TSH and prolactin immunostaining but with no clinical manifestations of hyperthyroidism or hyperprolactinemia. Pituitary magnetic resonance imaging revealed a macroadenoma. Transsphenoidal surgery was incomplete. The immunohistochemical staining showed that tumor cells were reactive to TSH (60%) and to prolactin (40%). Control pituitary imaging revealed a residual macroadenoma, and

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Marisavljevic, Dragomir. "An unusual presentation of “silent” disseminated pancreatic neuroendocrine tumor." World Journal of Gastroenterology 10, no. 19 (2004): 2919. http://dx.doi.org/10.3748/wjg.v10.i19.2919.

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Nishioka, Hiroshi, Asao Hirano, and Sylvia L. Asa. "Silent corticotroph adenoma with multiple cysts: Pars intermedia tumor?" Endocrine Pathology 3, no. 1 (1992): 47–51. http://dx.doi.org/10.1007/bf02921343.

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Tewari, Rohit, Rajat Bajaj, and Reena Bharadwaj. "Medical renal disease in tumor nephrectomies: The silent killer." Saudi Journal of Kidney Diseases and Transplantation 29, no. 1 (2018): 50. http://dx.doi.org/10.4103/1319-2442.225211.

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Wade, Alisha N., Jennifer Baccon, M. Sean Grady, Kevin D. Judy, Donald M. O’Rourke, and Peter J. Snyder. "Clinically silent somatotroph adenomas are common." European Journal of Endocrinology 165, no. 1 (2011): 39–44. http://dx.doi.org/10.1530/eje-11-0216.

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ObjectiveSomatotroph adenomas are typically recognized when they secrete GH excessively and cause acromegaly. Both ‘silent’ somatotroph adenomas (immunohistochemical evidence of GH excess without biochemical or clinical evidence) and ‘clinically silent’ somatotroph adenomas (immunohistochemical and biochemical evidence but no clinical evidence) have occasionally been reported. The relative frequency of each presentation is unknown. The goal of this study was, therefore, to determine the frequency of clinically silent somatotroph adenomas, a group that is potentially recognizable in vivo.Design

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Lamback, Elisa Baranski, Carlos Henrique de Azeredo Lima, Renan Lyra Miranda, et al. "USP8 Somatic Mutations in Cushing’s Disease and Silent Corticotropinomas." Journal of the Endocrine Society 5, Supplement_1 (2021): A651. http://dx.doi.org/10.1210/jendso/bvab048.1328.

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Abstract Background: Somatic mutations in the ubiquitin-specific peptidase 8 (USP8) gene have been described in Cushing’s disease (CD). These mutations increase proopiomelanocortin transcription resulting in ACTH production and seem to correlate with somatostatin receptor type 5 (SST5) expression. Aims: Screen USP8 in patients with corticotropinomas and correlate USP8 mutational status with SST5 expression in CD. Methods: Tumor DNA was extracted and then exon 14 amplified by PCR. SST5 was assessed by immunohistochemistry (clone UMB4) and quantified multiplying the percentage of positive cells

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Hedner, Pavo, and Stig Valdemarsson. "Reduced Size of a Hormonally Silent Pituitary Adenoma during Treatment with CV 205-502, a New Dopamine Agonist Mainly Stimulating D2 Receptors." Neurosurgery 25, no. 6 (1989): 948–50. http://dx.doi.org/10.1227/00006123-198912000-00015.

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Abstract A 39-year-old woman with secondary amenorrhea and visual field defects underwent craniotomy for a large pituitary tumor that was hormonally silent according to measurement of plasma hormone levels and immunohistochemical analysis. During the preoperative investigation, bromocriptine was administered for 1 month, but there was no change in the tumor size as seen on computed tomographic scans. One month after surgery, visual field defects recurred, and a tumor mass comparable to the preoperative state was found on computed tomographic scan. The tumor size gradually diminished during tre

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Himstead, Alexander, Gianna Fote, Nischal Acharya, Edward Kuan, Frank Hsu, and Ahmed Mohyeldin. "SURG-56. TRANSCRIPTION FACTOR CELL LINEAGE DETERMINES AGGRESSIVE BEHAVIOR AMONG NONFUNCTIONAL PITUITARY ADENOMAS." Neuro-Oncology 26, Supplement_8 (2024): viii286—viii287. http://dx.doi.org/10.1093/neuonc/noae165.1135.

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Abstract INTRODUCTION Pituitary neuroendocrine tumors (PitNETs) are the second most common intracranial neoplasm. Silent or non-functional PitNETs represent a common neuro-oncological challenge due to their size and unpredictable invasion of parasellar structures. This makes their complete surgical resection difficult and puts patients at higher risk for recurrence. OBJECTIVE To characterize differences in tumor volume, cavernous sinus invasion, and recurrence in nonfunctional PitNETs. METHODS Out of 615 transsphenoidal surgeries from 2012 to 2024, there were 309 overall PitNETs and 181 nonfun

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Gonzalez, Adriana, Daniel Brat, Emir Veledar, Nelson Oyesiku, Daniel Barrow, and Adriana Ioachimescu. "Abstract #832 Tumor Markers in Functional and Silent Corticotroph Adenomas." Endocrine Practice 24 (April 2018): 189–90. http://dx.doi.org/10.1016/s1530-891x(20)47319-7.

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Klibanski, Anne, Nicholas T. Zervas, Kalman Kovacs, and E. Chester Ridgway. "Clinically silent hypersecretion of growth hormone in patients with pituitary tumors." Journal of Neurosurgery 66, no. 6 (1987): 806–11. http://dx.doi.org/10.3171/jns.1987.66.6.0806.

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✓ Hypersecretion of growth hormone (GH) was found in three women aged 25 to 35 years old, with somatotroph adenomas without clinical stigmata of acromegaly. The patients had previously been diagnosed as having nonfunctioning pituitary macroadenomas, with extrasellar extension. Concentrations of GH were elevated preoperatively in all subjects and could not be suppressed during oral glucose tolerance testing. Somatomedin-C concentrations were elevated in two patients. Immunocytochemical studies of surgically obtained tumor tissue demonstrated sparse positive staining for GH in all subjects. Gel-

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Strauss, Bernard S. "Hypermutability in Carcinogenesis." Genetics 148, no. 4 (1998): 1619–26. http://dx.doi.org/10.1093/genetics/148.4.1619.

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Abstract The presence of numerous chromosomal changes and point mutations in tumors is well established. At least some of these changes play a role in the development of the tumors. It has been suggested that the number of these genetic changes requires that tumorigenesis involves an increase in mutation rate. However, the presence of numerous changes can also be accounted for by efficient selection. What is required to settle the issue is some measure of nonselected mutations in tumors. In order to determine whether the tumor suppressor TP53 (coding for the protein p53) is hypermutable at som

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Maragkos, Georgios A., Georgios Mantziaris, Stylianos Pikis, et al. "499 Silent Corticotroph Staining Pituitary Neuroendocrine Tumors: Prognostic Significance in Radiosurgery." Neurosurgery 70, Supplement_1 (2024): 153. http://dx.doi.org/10.1227/neu.0000000000002809_499.

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INTRODUCTION: There is conflicting evidence on the significance of adrenocorticotrophic hormone (ACTH) staining in the prognosis of nonfunctioning pituitary neuroendocrine tumors (NFpitNETs). METHODS: This retrospective, multicenter study included patients managed with SRS for NFpitNET residuals. The patients were divided into two cohorts: 1) silent corticotroph (SC) for NFpitNETs with positive ACTH immunostaining and 2) non-SC NFpitNETs. Rates of local tumor control, as well as the incidence of post-treatment pituitary and neurological dysfunction were documented. Factors associated with radi

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Gütl, Christian. "Editorial." JUCS - Journal of Universal Computer Science 29, no. (8) (2023): 836–37. https://doi.org/10.3897/jucs.109658.

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Dear Readers, It gives me great pleasure to announce the eighth regular issue of 2023. In this issue, five papers by 24 authors from nine countries cover various topical aspects of computer science. In an ongoing effort to further strengthen our journal, I would like to expand the editorial board: If you are a tenured associate professor or above with a strong publication record, you are welcome to apply to join our editorial board. We are also interested in high-quality proposals for special issues on new topics and trends. As always, I would like to thank all the authors for their sound rese

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Lu, Cheng-Chieh, Chien-Jui Cheng, Yu-Chien Kao, and Mei-Chien Chen. "Secretory Carcinoma: A Silent Mass Increasing in the Parotid Gland." Open Access Macedonian Journal of Medical Sciences 8, no. C (2020): 191–94. http://dx.doi.org/10.3889/oamjms.2020.4603.

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BACKGROUND: Secretory carcinoma (SC) of the salivary gland, also known as mammary analog secretory carcinoma, is a rare tumor in the parotid gland. This kind of tumor is characterized by generally indolent clinical behavior and expression of a break in the ETV6 gene.  CASE REPORT: We present a unique case of secretory carcinoma and show its favorable prognoses. CONCLUSION: Secretory carcinoma of the salivary gland is a low-grade carcinoma with a favorable prognosis. It has low regional lymph node and distant metastasis potential. Due to the possibility of misdiagnosis, immunohistoc

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Muijlwijk, Tara, Naomi Remkes, Jos Poell, René Leemans, Ruud Brakenhoff, and Rieneke van de Ven. "935 Attraction of immune cells by head and neck cancer cell lines and primary tumor-conditioned supernatants." Journal for ImmunoTherapy of Cancer 9, Suppl 2 (2021): A981. http://dx.doi.org/10.1136/jitc-2021-sitc2021.935.

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BackgroundHead and neck squamous cell carcinomas (HNSCC) are classified in human papillomavirus (HPV)-positive and HPV-negative tumors. In general, HPV-negative HNSCC are genetically characterized by many chromosomal gains and losses.1 Previously, we and others identified a HPV-negative subgroup with few or absent copy number alterations (CNA-silent), and a more favorable prognosis.2 3 Tumors with low copy number changes have generally been associated with high immune infiltration scores,4 but for CNA-silent versus CNA-high HPV-negative HNSCC such data are lacking.In this study we aim to unrav

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Batisse, Marie, Gérald Raverot, Salwan Maqdasy, et al. "Aggressive Silent GH Pituitary Tumor Resistant to Multiple Treatments, Including Temozolomide." Cancer Investigation 31, no. 3 (2013): 190–96. http://dx.doi.org/10.3109/07357907.2013.775293.

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Maltempi, Marzia, Stefano Sforna, Elisabetta Girella, et al. "A startling discovery: The case of a giant “silent” cardiac tumor." Journal of Cardiovascular Magnetic Resonance 27 (2025): 101706. https://doi.org/10.1016/j.jocmr.2024.101706.

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Chen, Lieping. "Immunological ignorance of silent antigens as an explanation of tumor evasion." Immunology Today 19, no. 1 (1998): 27–30. http://dx.doi.org/10.1016/s0167-5699(97)01180-8.

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Maschek, U., W. Pülm, S. Segal, and G. J. Hämmerling. "Major histocompatibility complex class I genes in murine fibrosarcoma IC9 are down regulated at the level of the chromatin structure." Molecular and Cellular Biology 9, no. 7 (1989): 3136–42. http://dx.doi.org/10.1128/mcb.9.7.3136-3142.1989.

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The fibrosarcoma IC9 is deficient in the expression of the major histocompatibility complex class I genes Kb, Kk, and Dk and expresses only the Db molecule. Because class I deficiency may enable tumor cells to escape the immune response by cytotoxic T lymphocytes, we investigated why the class I genes are not expressed. Expression of the silent class I genes could not be induced, but all known DNA-binding factors specific for class I genes could be detected in nuclear extracts of IC9 cells. After cloning of the silent Kb gene from the IC9 cells and subsequent transfection of this cloned Kb gen

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Maschek, U., W. Pülm, S. Segal, and G. J. Hämmerling. "Major histocompatibility complex class I genes in murine fibrosarcoma IC9 are down regulated at the level of the chromatin structure." Molecular and Cellular Biology 9, no. 7 (1989): 3136–42. http://dx.doi.org/10.1128/mcb.9.7.3136.

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Abstract:

The fibrosarcoma IC9 is deficient in the expression of the major histocompatibility complex class I genes Kb, Kk, and Dk and expresses only the Db molecule. Because class I deficiency may enable tumor cells to escape the immune response by cytotoxic T lymphocytes, we investigated why the class I genes are not expressed. Expression of the silent class I genes could not be induced, but all known DNA-binding factors specific for class I genes could be detected in nuclear extracts of IC9 cells. After cloning of the silent Kb gene from the IC9 cells and subsequent transfection of this cloned Kb gen

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Hind Asbar, Sana Rafi, Ghizlane El Mghari, and Nawal El Ansari. "A rare case of silent pituitary macroadenoma with positive TSH and prolactin immunostaining." World Journal of Advanced Research and Reviews 14, no. 2 (2022): 112–14. http://dx.doi.org/10.30574/wjarr.2022.14.2.0419.

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Silent thyroid stimulating hormone (TSH)-immunostaining pituitary adenomas are rare tumors, they can be either pure or immunoreactive to other pituitary hormones. We report a case of a silent macroadenoma with both TSH and prolactin immunostaining but with no clinical manifestations of hyperthyroidism or hyperprolactinemia. Pituitary magnetic resonance imaging revealed a macroadenoma. Transsphenoidal surgery was incomplete. The immunohistochemical staining showed that tumor cells were reactive to TSH (60%) and to prolactin (40%). Control pituitary imaging revealed a residual macroadenoma, and

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Yamaguchi-Okada, Mitsuo, Naoko Inoshita, Hiroshi Nishioka, Noriaki Fukuhara, and Shozo Yamada. "Clinicopathological analysis of nonfunctioning pituitary adenomas in patients younger than 25 years of age." Journal of Neurosurgery: Pediatrics 9, no. 5 (2012): 511–16. http://dx.doi.org/10.3171/2012.1.peds11330.

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Object The authors evaluated the pathological and clinical characteristics of young patients with clinically nonfunctioning pituitary adenomas (NFPAs). Methods Twenty-one patients (13 males and 8 females) with NFPAs who were 25 years of age or younger (mean 20 years, range 13–25 years) were retrospectively investigated. The following factors were examined: results of conventional light microscopy, immunohistochemistry, and electron microscopy; clinical symptoms; tumor size and invasion on MRI; and clinical course after therapeutic procedures such as surgery and adjuvant radiotherapy. Results T

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Ozisik, Hatice, Banu Sarer Yurekli, Nilufer Ozdemir Kutbay, Ilker Altun, Yesim Ertan, and Fusun Saygılı. "Acromegaly with negative immunostaining for growth hormone on the contrary to silent somatotroph tumor." International Surgery Journal 4, no. 4 (2017): 1506. http://dx.doi.org/10.18203/2349-2902.isj20171174.

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Silent somatotropinomas are not rare. On the contrary to that, the absence of GH immunostaining in acromegaly has not been well defined. So, we would like to to draw the clinician’s attention to such an entity through a case of acromegaly with negative GH staining.

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García-Martínez, Araceli, Antonio C. Fuentes-Fayos, Carmen Fajardo, et al. "Differential Expression of MicroRNAs in Silent and Functioning Corticotroph Tumors." Journal of Clinical Medicine 9, no. 6 (2020): 1838. http://dx.doi.org/10.3390/jcm9061838.

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The potential role of miRNAs in the silencing mechanisms of pituitary neuroendocrine tumors (PitNETs) has not been addressed. The aim of the present study was to evaluate the expression levels and the potential associated role of some miRNAs, pathways, and transcription factors in the silencing mechanisms of corticotroph tumors (CTs). Accordingly, the expression of miR-375, miR-383, miR-488, miR-200a and miR-103; of PKA, MAP3K8, MEK, MAPK3, NGFIB, NURR1, PITX1, and STAT3 were analyzed via qRT-PCR in 23 silent and 24 functioning CTs. miR-200a and miR-103 showed significantly higher expression i

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Singh, Sukarma. "Is Stress a Contributor to Tumor Metastasis?" Science Insights 44, no. 5 (2024): 1375–80. http://dx.doi.org/10.15354/si.24.re1007.

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Stress, often considered a silent yet pervasive force in our daily lives, has increasingly been recognized for its significant impact on human health. In the realm of oncology, the connection between stress and cancer development, particularly tumor metastasis, has become a subject of intense research and clinical interest. This review discusses the intricate relationship between stress and tumor metastasis, exploring the biological mechanisms, clinical implications, and potential therapeutic approaches at the intersection of these two complex phenomena. By understanding the role of stress in

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