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1

1927-, D'Arcy P. F., and Griffin J. P, eds. Iatrogenic diseases. 3rd ed. Oxford University Press, 1986.

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2

F, D'Arcy P., and Griffin J. P, eds. Iatrogenic diseases. 3rd ed. Oxford University Press, 1986.

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3

D, Cooper J. Allen, ed. Drug-induced pulmonary disease. W. B. Saunders, 1990.

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4

Neil, Kaplowitz, and DeLeve Laurie D. 1955-, eds. Drug-induced liver disease. 2nd ed. Informa Healthcare USA, 2007.

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5

Modicare Foundation (New Delhi, India), ed. The positive side. Roli Books in association with Modicare Foundation, 2002.

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6

N, Sladen Robert, ed. Anesthesia and co-existing disease. Cambridge University Press, 2007.

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7

Neil, Kaplowitz, and DeLeve Laurie D. 1955-, eds. Drug-induced liver disease. Marcel Dekker, 2003.

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8

Neil, Kaplowitz, and DeLeve Laurie D. 1955-, eds. Drug-induced liver disease. Marcel Dekker, 2003.

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9

D, Hellier Michael, ed. Disease, drugs, and the dentist. 2nd ed. Wright, 1989.

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10

Vithoulkas, George. A new model for health and disease. Health and Habitat, 1991.

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11

Zonderman, Jon. Drugs & disease. Chelsea House Publishers, 1987.

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12

C, Piscitelli Stephen, and Rodvold Keith, eds. Drug interactions in infectious disease. Humana Press, 2000.

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13

American Society of Health-System Pharmacists, ed. Drug-induced diseases: Prevention, detection, and management. 2nd ed. American Society of Health-System Pharmacists, 2010.

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14

Davis, C. N. Field guide to tree diseases of Ontario. Great Lakes Forestry Centre, 1997.

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15

1964-, Otley Clark C., ed. Skin disease in organ transplantation. Cambridge University Press, 2008.

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16

Stern, Ronald J. Drugs, diseases, and anesthesia. Lippincott-Raven Publishers, 1997.

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17

Bari Seminar in Nephrology (3rd 1988). Drugs, systemic diseases, and the kidney. Plenum Press, 1989.

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18

Otley, Clark C. Skin disease in organ transplantation. Cambridge University Press, 2008.

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19

C, Petrie J., ed. Gastrointestinal, haematological, and infectious disease therapy. Elsevier, 1985.

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20

(Foreword), Henry Masur, Stephen C. Piscitelli (Editor), and Keith A. Rodvold (Editor), eds. Drug Interactions in Infectious Diseases (Infectious Disease). Humana Press, 2000.

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21

Keshav, Satish, and Alexandra Kent. Psychiatry in gastrointestinal medicine. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0206.

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This chapter discusses psychiatric conditions with gastrointestinal (GI) consequences (including eating disorders, depression, and side effects of psychiatric medications), and GI diseases with psychiatric symptoms (including hepatic encephalopathy, coeliac disease, Wilson’s disease, acute intermittent porphyria, functional GI disease, and inflammatory bowel disease).
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22

Hatfield, Anthea. Cardiovascular disease. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199666041.003.0018.

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Cardiovascular disease is common and patients coming to recovery room with any of these common problems will need special care. The essential signs and symptoms of hypertension, cardiac failure, ischaemic heart disease, and valvular heart disease are outlined. The actions and side-effects of the drugs that these patients take to control their symptoms are described. Recognizing and treating hypotension and myocardial ischaemia are very important and relevant, and they are fully discussed in this chapter.
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23

Kaplowitz, Neil, and Laurie D. DeLeve. Drug-Induced Liver Disease. Taylor & Francis Group, 2020.

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24

Kaplowitz, Neil, and Laurie D. DeLeve. Drug-Induced Liver Disease. Taylor & Francis Group, 2007.

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25

Kaplowitz, Neil. Drug-Induced Liver Disease. Taylor & Francis Group, 2002.

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26

Kaplowitz, Neil, and Laurie D. DeLeve. Drug-Induced Liver Disease. Elsevier Science & Technology Books, 2013.

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27

Gang, Lu, and Philippe Sionneau. The Treatment of Disease in TCM, Vol. 5: Diseases of the Chest, Abdomen & Rib-side (The Treatment of Disease in Tcm). Blue Poppy Press, 1998.

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28

Kaplowitz, Neil, and Laurie D. Deleve. Drug-Induced Liver Disease. Taylor & Francis Group, 2002.

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29

Kaplowitz, Neil, and Laurie D. DeLeve. Drug-Induced Liver Disease. Taylor & Francis Group, 2007.

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30

Reynard, John, and Ben Turney. Watchful waiting for stone disease. Edited by John Reynard. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0020.

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Renal stones are highly prevalent and while many cause symptoms or are of a size that requires treatment even if asymptomatic, the natural history of stones suggests that treatment may not be necessary in all cases. Stone size, position, and number are related, to a degree, to the likelihood of a subsequent stone event such as stone migration causing ureteric colic or increase in stone size, but the predictive power of ‘natural history’ studies is limited by the small number of patients in these studies. In this chapter, the evidence for watchful and waiting for asymptomatic stone disease is e
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31

Drug-Induced Liver Disease. Elsevier Science & Technology Books, 2013.

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32

Niaudet, Patrick, and Alain Meyrier. Minimal change disease. Edited by Neil Turner. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0056_update_001.

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Minimal change disease is characteristically responsive to high-dose corticosteroids. As this is the most common cause of nephrotic syndrome in children, and responses are usually prompt, response to 60 mg/m2/day of oral prednisolone (max. 80 mg) is often used as a diagnostic test. Adults respond more slowly and have a wider differential diagnosis, and often a high risk of side effects, so therapy is not recommended without confirmation by renal biopsy. Then first-line treatment is again prednisolone or prednisone, at 1 mg/kg/day (max. 60 mg). KDIGO and other treatment protocols recommend 6 we
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33

Longmore, Murray, Ian B. Wilkinson, Andrew Baldwin, and Elizabeth Wallin. Infectious diseases. Oxford University Press, 2014. http://dx.doi.org/10.1093/med/9780199609628.003.0009.

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Classification of pathogens –An approach to infectious diseases:Travel adviceAntibiotics: penicillinsCephalosporins and othersEmpirical ‘blind’ treatmentUsing a side-room laboratoryDrug abuse & infectious diseasesPUO and the tropical traveller –GastroenteritisImmunizationNosocomial infectionsSome specific infections:MalariaTuberculosis (MDR-TB )Infectious mononucleosis...
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34

Raje, R. R., and Ply D. Wong. Iatrogenic Diseases (Textbooks for Students). Pjd Publications Limited, 1999.

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35

Stern, Ronald J. Drugs, Diseases & Anesthesia. Lippincott Williams & Wilkins, 1996.

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36

Anesthesia and co-existing disease. Cambridge University Press, 2007.

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37

Sladen, Robert, Douglas B. Coursin, Jonathan T. Ketzler, and Hugh Playford. Anesthesia and Co-Existing Disease. Cambridge University Press, 2007.

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38

Sladen, Robert, Douglas B. Coursin, Jonathan T. Ketzler, and Hugh Playford. Anesthesia and Co-Existing Disease. Cambridge University Press, 2010.

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39

Hoskin, Peter. Radiotherapy planning for metastatic disease. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199696567.003.0021.

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Chapter 16 discusses radiotherapy planning for metastatic disease, predominantly for patients with bone metastasis, spinal cord compression, and brain metastasis. The techniques for such treatments are specific to this indication rather than the primary tumour site.
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40

Keshav, Satish, and Palak Trivedi. Drug-induced liver disease. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0215.

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Drugs are an important and common cause of hepatic injury. This is unsurprising, as the liver is a major site for drug clearance, biotransformation, and excretion. A careful history of drugs taken (prescribed, over the counter, herbal, or illicit) is vital when assessing anyone with abnormal liver function tests. Although toxic or idiosyncratic adverse reactions may occur with many therapeutic agents, drug-induced jaundice is not so common.
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41

(Editor), Neil Kaplowitz, and Laurie D. DeLeve (Editor), eds. Drug-Induced Liver Disease. Informa Healthcare, 2002.

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42

Swales, Catherine. Inflammatory connective tissue disease. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.010005.

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♦ Systemic lupus erythematosus (SLE) is the commonest multisystem connective tissue disease♦ All patients with SLE are antinuclear antibody (ANA) positive, but not all ANA-positive patients have SLE♦ Renal lupus carries a high mortality and requires aggressive immunosuppression♦ Dermatomyositis may be associated with malignancy in the elderly♦ The vasculitides are classified according to vessel size and ANCA (antineutrophil cytoplasmic antibody) profile♦ Therapy for vasculitis includes corticosteroids, steroid-sparing agents, and cytotoxics.
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43

Eisen, Andrew. Motor neurone disease. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199658602.003.0009.

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In this chapter, the following ten key events in motor neurone disease, also known as amyotrophic lateral sclerosis (ALS), are considered: the first description of ALS by Cruveilhier; discovery of the first SOD1 mutation; use of the ALSFRS (functional rating scale) for determining therapeutic trial outcomes; the contentious issue of establishing the site of onset of ALS; clinical, pathological, and molecular evidence indicating that frontotemporal dementia and ALS are closely related; demonstration that ALS bears some resemblance to the transmissible spongiform encephalopathies; use of Riluzol
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44

Thornton, Clare, and Justin Mason. Vascular biology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0057.

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Vascular biology is the study of the physiology of the vasculature and how it may be the target for disease processes. An understanding of vascular biology is central to the study of rheumatic disease for three reasons: it is an integral part of a functioning immune system; it is the primary site of pathology in many conditions; and it is the site of the important secondary complications of chronic inflammation, endothelial dysfunction and atherosclerosis. Vascular biology requires a detailed knowledge of the anatomy and physiology of the vasculature and its constituent vessels. The multistep
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45

Thornton, Clare, and Justin Mason. Vascular biology. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199642489.003.0057_update_001.

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Vascular biology is the study of the physiology of the vasculature and how it may be the target for disease processes. An understanding of vascular biology is central to the study of rheumatic disease for three reasons: it is an integral part of a functioning immune system; it is the primary site of pathology in many conditions; and it is the site of the important secondary complications of chronic inflammation, endothelial dysfunction and atherosclerosis. Vascular biology requires a detailed knowledge of the anatomy and physiology of the vasculature and its constituent vessels. The multistep
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46

Rosenhek, Raphael, Robert Feneck, and Fabio Guarracino. Aortic valve disease. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199599639.003.0014.

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Echocardiography is the gold standard for the assessment of patients with aortic valve (AoV) disease. It allows a detailed morphological assessment of the AoV and thereby makes determination of the aetiology possible. In general, the quantification of aortic stenosis is based on the measurement of transaortic jet velocities and the calculation of AoV area, thus combining a flow-dependent and a flow-independent variable. In the setting of low-flow low-gradient AS, dobutamine echocardiography is of particular diagnostic and prognostic importance. The quantification of aortic regurgitation is bas
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47

Heart disease: Drug-free alternatives to prevent and reverse heart disease. Hay House, 2016.

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48

Heart Disease: Drug-Free Alternatives to Prevent and Reverse Heart Disease. Hay House UK, Limited, 2016.

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49

Hagendorff, Andreas, Elie Chammas, and Mohammed Rafique Essop. Diseases with a main influence on heart valves. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0058.

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The high spatial and temporal resolution, widespread availability, and non-invasive nature of echocardiography make it the imaging modality of choice for assessment of cardiac valvular disease. Echocardiography allows not only detailed evaluation of valve morphology, but also makes possible assessment of the haemodynamic consequences and impact on left and right ventricular size and function. Based on this data, a more informed decision may be made on the nature and timing of surgical or percutaneous intervention. A wide variety of diseases may afflict the cardiac valves. In some such as rheum
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50

Hagendorff, Andreas, and Laura Ernande. Diseases with a main influence on pericardium. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780198726012.003.0059.

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The high spatial and temporal resolution, widespread availability, and non-invasive nature of echocardiography make it the imaging modality of choice for assessment of cardiac valvular disease. Echocardiography allows not only detailed evaluation of valve morphology, but also makes possible assessment of the haemodynamic consequences and impact on left and right ventricular size and function. Based on this data, a more informed decision may be made on the nature and timing of surgical or percutaneous intervention. A wide variety of diseases may afflict the cardiac valves. In some such as rheum
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