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Relevant bibliographies by topics / SK-BR-3

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Journal articles
Dissertations / Theses
Conference papers

Academic literature on the topic 'SK-BR-3'

Author: Grafiati

Published: 4 June 2021

Last updated: 18 February 2022

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Journal articles on the topic "SK-BR-3"

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Yin, Guoling, Xiaoping Li, and Xiaofei Chang. "Paclitaxel liposomes exert radio-sensitization effect on breast cancer cell line SK-BR-3 by regulating expressions of apoptotic proteins." Tropical Journal of Pharmaceutical Research 19, no. 5 (2020): 1009–13. http://dx.doi.org/10.4314/tjpr.v19i5.15.

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Purpose: To study the radio-sanitization effect of paclitaxel liposomes on breast cancer cells, SK-BR-3.Methods: Breast cancer cell line SK-BR-3 was cultured and made into a cell suspension. Four groups of cells were used: control and radiotherapy groups, paclitaxel group, and paclitaxel liposome + radiotherapy group (combination group). The growth inhibitory effects of the different treatments on SKBR-3 cells were determined with CCK-8 method. Apoptosis in each group was evaluated with flow cytometry, while Western blotting was employed to assay Bcl-2, Caspase-3 and Bax protein levels.Results

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Özben, RŞ, and D. Cansaran-Duman. "The expression profiles of apoptosis-related genes induced usnic acid in SK-BR-3 breast cancer cell." Human & Experimental Toxicology 39, no. 11 (2020): 1497–506. http://dx.doi.org/10.1177/0960327120930257.

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This study aims to determine whether usnic acid (UA) could induce the expression of apoptosis-related genes in apoptosis pathway. The current study has enabled us to better understand the target of UA in the treatment of breast cancer. Cell viability was determined by 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide assay. Based on the previous study and the results of this study, UA had the most antiproliferative effect on SK-BR-3 breast cancer cell line. We examined differential expression of 88 apoptosis-related genes by quantitative real-time polymerase chain reaction using the

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Lai, Hung-Wen, Su-Yu Chien, Shou-Jen Kuo, et al. "The Potential Utility of Curcumin in the Treatment of HER-2-Overexpressed Breast Cancer: AnIn VitroandIn VivoComparison Study with Herceptin." Evidence-Based Complementary and Alternative Medicine 2012 (2012): 1–12. http://dx.doi.org/10.1155/2012/486568.

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HER-2 is an important oncoprotein overexpressed in about 15–25% of breast cancers. We hypothesized that the ability of curcumin to downregulate HER-2 oncoprotein and inhibit the signal transduction pathway of PI3K/Akt, MAPK, and NF-κB activation may be important in the treatment of HER-2-overexpressed breast cancer. To examine the effect of curcumin on breast cancer cells, MCF-7, MDA-MB-231, MCF-10A, BT-474, and SK-BR-3-hr (a herceptin resistant strain from SK-BR-3) cells were used forin vitroanalysis. Thein vivoeffect of curcumin on HER-2-overexpressed breast cancer was investigated with the

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Xiao, Yan, Xiugong Gao, Samantha Maragh, William G. Telford, and Alessandro Tona. "Cell Lines as Candidate Reference Materials for Quality Control of ERBB2 Amplification and Expression Assays in Breast Cancer." Clinical Chemistry 55, no. 7 (2009): 1307–15. http://dx.doi.org/10.1373/clinchem.2008.120576.

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Abstract Background: Human epidermal growth factor receptor 2 (HER2) is an important biomarker whose status plays a pivotal role in therapeutic decision-making for breast cancer patients and in determining their clinical outcomes. Ensuring the accuracy and reproducibility of HER2 assays by immunohistochemistry (IHC) and by fluorescence in situ hybridization (FISH) requires a reliable standard for monitoring assay sensitivity and specificity, and for assessing methodologic variation. A prior NIST workshop addressed this need by reaching a consensus to create cell lines as reference materials fo

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Thakur, Archana, Zaid Al-Kadhimi, Cassara Pray, et al. "Transfer of Cellular and Humoral Anti-Tumor Immunity with Immune T Cells After Stem Cell Transplant (SCT) for Metastatic Breast Cancer Following “Vaccination” with Anti-CD3 x Anti-Her2/Neu Bispecific Antibody (Her2Bi) Armed Activated T Cells." Blood 118, no. 21 (2011): 1914. http://dx.doi.org/10.1182/blood.v118.21.1914.1914.

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Abstract Abstract 1914 Novel therapeutic approaches are needed for women with metastatic breast cancer (BrCa). In our phase I clinical trial, infusions of anti-CD3 activated T cells (ATC) armed with anti-CD3 x anti-Her2/neu bispecific antibody (Her2Bi) induced specific cytotoxicity (SC) directed at SK-BR-3 breast cancer cells by fresh peripheral blood lymphocytes (PBL) and induced elevated serum levels of Th1 cytokines. In this study, we took advantage of armed ATC induced anti-tumor immune responses by infusing “immune” T cells collected by leukopheresis. We expanded “immune T cells” with ant

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Do, Ha Thi Thu, та Jungsook Cho. "Involvement of the ERK/HIF-1α/EMT Pathway in XCL1-Induced Migration of MDA-MB-231 and SK-BR-3 Breast Cancer Cells". International Journal of Molecular Sciences 22, № 1 (2020): 89. http://dx.doi.org/10.3390/ijms22010089.

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Chemokine–receptor interactions play multiple roles in cancer progression. It was reported that the overexpression of X-C motif chemokine receptor 1 (XCR1), a specific receptor for chemokine X-C motif chemokine ligand 1 (XCL1), stimulates the migration of MDA-MB-231 triple-negative breast cancer cells. However, the exact mechanisms of this process remain to be elucidated. Our study found that XCL1 treatment markedly enhanced MDA-MB-231 cell migration. Additionally, XCL1 treatment enhanced epithelial–mesenchymal transition (EMT) of MDA-MB-231 cells via E-cadherin downregulation and upregulation

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Kumar, Naresh, Pankaj Attri, Eun Ha Choi, and Han Sup Uhm. "Influence of water vapour with non-thermal plasma jet on the apoptosis of SK-BR-3 breast cancer cells." RSC Advances 5, no. 19 (2015): 14670–77. http://dx.doi.org/10.1039/c4ra15879b.

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Afrakhteh, Moslem, Alireza Kheirollah, Aminollah Pourshohod, Mohammad Ali Ghaffari, Mostafa Jamalan, and Majid Zeinali. "Cytotoxicity of Sodium Arsenite-loaded Anti-HER2 Immunoliposomes Against HER2-expressing Human Breast Cancer Cell Lines." Letters in Drug Design & Discovery 16, no. 5 (2019): 556–62. http://dx.doi.org/10.2174/1570180815666180803120409.

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Background: Chemotherapy is a routine approach in treatment of patients with cancer, while side effects of chemotherapeutic drugs are inevitable. To minimize side effects, specific targeting of neoplastic cells is a promising strategy in cancer therapy. Sodium arsenite is a metalloid toxin with anti-neoplastic properties, but low selectivity and carcinogenic activity have limited its clinical usage. Methods: Targeting of HER2-overexpressing (SK-BR-3) and HER2-low expressing (MCF-7) cancerous breast cell lines by two different liposomal forms of sodium arsenite (bare liposome and trastuzumab-co

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Hossain, Md Imran, Ajit G. Thomas, Fakhri Mahdi та ін. "An efficient synthetic route to l-γ-methyleneglutamine and its amide derivatives, and their selective anticancer activity". RSC Advances 11, № 13 (2021): 7115–28. http://dx.doi.org/10.1039/d0ra08249j.

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l-γ-Methyleneglutamic acid amides selectively inhibit the growth of MCF-7 (ER<sup>+</sup>/PR<sup>+</sup>/HER2<sup>−</sup>), SK-BR-3 (ER<sup>−</sup>/PR<sup>−</sup>/HER2<sup>+</sup>), and triple negative MDA-MB-231 cancer cell lines.

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Hester, Anna, Barbara Salzmann, Martina Rahmeh, et al. "EP3 receptor antagonist L798,106 reduces proliferation and migration of SK-BR-3 breast cancer cells." OncoTargets and Therapy Volume 12 (July 2019): 6053–68. http://dx.doi.org/10.2147/ott.s204919.

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Dissertations / Theses on the topic "SK-BR-3"

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Schwab, Carsten. "Untersuchungen zur zellulären Antwort von BT474- und SK-BR-3-Mammakarzinomzelllinien auf die Behandlung mit den therapeutischen Antikörpern Trastuzumab und Pertuzumab und die Rolle des PTEN-Proteins für das Ansprechen." kostenfrei, 2010. http://epub.uni-regensburg.de/14994/.

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Huang, Chang-Yu, and 黃昶諭. "Mechanism of METCAM/MUC18-promoted progression of human breast cancer cells: METCAM/MUC18 promoted tumorigenesis of human breast cancer SK-BR-3 cells in a dosage specific manner." Thesis, 2014. http://ndltd.ncl.edu.tw/handle/d9rh66.

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碩士<br>中原大學<br>生物科技研究所<br>102<br>Breast cancer is still the most prevalent and deadly cancer in women worldwide. The death reason of recurrent breast cancer is due to distant metastasis even after treatment. After many years of extensive studies, the mechanism and biology of breast cancer tumor formation and metastasis is still poorly understood. METCAM/MUC18, an Ig-like cell adhesion molecule, plays an important role in promoting angiogenesis, tumorigenesis and metastasis in several epithelial tumors. Over-expression of METCAM/MUC18, an Ig-like cell adhesion molecule, promotes tumorigenesis an

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Kopperová, Dana. "Molekulární mechanismy rezistence buněk nádorů prsu k taxanům: úloha ABC transportérů." Master's thesis, 2014. http://www.nusl.cz/ntk/nusl-332179.

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Resistance to chemotherapeutics is a widespread phenomenon in cancer cells that may counteract the successful therapy of many patients. In resistant cells, higher level of ABC transporters, among others, often can be detected. This high level of ABC transporters represents a suspected mechanism of acquired cancer resistance. We studied the molecular mechanism of resistance to taxanes in cancer cells using SK-BR-3 and MCF-7 breast cancer cell lines. We analyzed the effect of paclitaxel on apoptosis induction in the originally sensitive cells of these lines as compared to their counterpart resis

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Schwab, Carsten [Verfasser]. "Untersuchungen zur zellulären Antwort von BT474- und SK-BR-3-Mammakarzinomzelllinien auf die Behandlung mit den therapeutischen Antikörpern Trastuzumab und Pertuzumab und die Rolle des PTEN-Proteins für das Ansprechen / vorgelegt von Carsten Schwab." 2010. http://d-nb.info/1003237312/34.

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Conference papers on the topic "SK-BR-3"

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Salzmann, B., M. Rahmeh, B. Czogalla, et al. "Der EP3-Antagonist L798,106 reduziert Proliferation und Migration von SK-BR-3 Mammakarzinom-Zellen." In Abstracts zum Kongress 2019 der Bayerischen Gesellschaft für Geburtshilfe und Frauenheilkunde (BGGF) und der Österreichischen Gesellschaft für Gynäkologie und Geburtshilfe (OEGGG). Georg Thieme Verlag KG, 2019. http://dx.doi.org/10.1055/s-0039-1693866.

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Pirsko, V., I. Cakstina, M. Priedīte, et al. "PO-227 The effect of chronic mild hypoxia on genomic instability in HER2-overexpression breast cancer cell line SK-BR-3." In Abstracts of the 25th Biennial Congress of the European Association for Cancer Research, Amsterdam, The Netherlands, 30 June – 3 July 2018. BMJ Publishing Group Ltd, 2018. http://dx.doi.org/10.1136/esmoopen-2018-eacr25.744.

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Thomas, T. J., Sripriya Venkiteswaran, Thresia Thomas, PingAr Yang, and Hui-Chen Hsu. "Abstract 4625: Curcumin suppresses the expression of HER-2 gene and interferes with redox regulated pathways in SK-BR-3 breast cancer cells." In Proceedings: AACR 102nd Annual Meeting 2011‐‐ Apr 2‐6, 2011; Orlando, FL. American Association for Cancer Research, 2011. http://dx.doi.org/10.1158/1538-7445.am2011-4625.

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Rose, Ashley, Daniel Wann, Janet Lightner, et al. "Abstract 3567: Metformin suppresses synthesis of pro-survival sphingolipid, sphingosine-1-phosphate, by inhibition of sphingosine kinase-1, in MCF-7 and SK-BR-3 breast cancer cell lines." In Proceedings: AACR 107th Annual Meeting 2016; April 16-20, 2016; New Orleans, LA. American Association for Cancer Research, 2016. http://dx.doi.org/10.1158/1538-7445.am2016-3567.

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