Academic literature on the topic 'Skeletal muscle'

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Journal articles on the topic "Skeletal muscle"

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Zhang, Tan, Xin Feng, Bo Feng, et al. "CARDIAC TROPONIN T MEDIATED AUTOIMMUNE RESPONSE AND ITS ROLE IN SKELETAL MUSCLE AGING." Innovation in Aging 3, Supplement_1 (2019): S882. http://dx.doi.org/10.1093/geroni/igz038.3231.

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Abstract Cardiac troponin T (cTnT), a key component of contractile machinery essential for muscle contraction, is also expressed in skeletal muscle under certain conditions (e.g. neuromuscular diseases and aging). We have reported that skeletal muscle cTnT regulates neuromuscular junction denervation preferentially in fast skeletal muscle of old mice. Here, we further report that cTnT is also enriched within some myofibers, and/or along microvascular walls in old mice fast skeletal muscle. Strikingly, immunoglobulin G (IgG), together with markers of complement system activation, cell death (ne
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Kholodnyi, R. D. "MODELING THE SKELETAL MUSCLE INJURY IN RATS." International Journal of Veterinary Medicine, no. 3 (October 18, 2022): 253–57. http://dx.doi.org/10.52419/issn2072-2419.2022.3.253.

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Muscles are the most important executive organs - effectors. Both according to morphological and functional characteristics, muscles are divided into two types - striated and smooth. Striated muscles, in turn, are usually divided into skeletal and cardiac. Striated muscles form the motor apparatus of the skeleton, oculomotor, chewing and other motor systems in animals. The striated muscles, with the exception of the heart muscle, are completely controlled by the central nervous system, they are devoid of automatism.The problem of damage to skeletal muscles is very relevant and widespread. Thes
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Heo, Jun-Won, Su-Zi Yoo, Mi-Hyun No, et al. "Exercise Training Attenuates Obesity-Induced Skeletal Muscle Remodeling and Mitochondria-Mediated Apoptosis in the Skeletal Muscle." International Journal of Environmental Research and Public Health 15, no. 10 (2018): 2301. http://dx.doi.org/10.3390/ijerph15102301.

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Obesity is characterized by the induction of skeletal muscle remodeling and mitochondria-mediated apoptosis. Exercise has been reported as a positive regulator of skeletal muscle remodeling and apoptosis. However, the effects of exercise on skeletal muscle remodeling and mitochondria-mediated apoptosis in obese skeletal muscles have not been clearly elucidated. Four-week-old C57BL/6 mice were randomly assigned into four groups: control (CON), control plus exercise (CON + EX), high-fat diet (HFD), and HFD plus exercise groups (HFD + EX). After obesity was induced by 20 weeks of 60% HFD feeding,
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Sandage, Mary J., and Audrey G. Smith. "Muscle Bioenergetic Considerations for Intrinsic Laryngeal Skeletal Muscle Physiology." Journal of Speech, Language, and Hearing Research 60, no. 5 (2017): 1254–63. http://dx.doi.org/10.1044/2016_jslhr-s-16-0192.

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PurposeIntrinsic laryngeal skeletal muscle bioenergetics, the means by which muscles produce fuel for muscle metabolism, is an understudied aspect of laryngeal physiology with direct implications for voice habilitation and rehabilitation. The purpose of this review is to describe bioenergetic pathways identified in limb skeletal muscle and introduce bioenergetic physiology as a necessary parameter for theoretical models of laryngeal skeletal muscle function.MethodA comprehensive review of the human intrinsic laryngeal skeletal muscle physiology literature was conducted. Findings regarding intr
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Azab, Azab. "Skeletal Muscles: Insight into Embryonic Development, Satellite Cells, Histology, Ultrastructure, Innervation, Contraction and Relaxation, Causes, Pathophysiology, and Treatment of Volumetric Muscle I." Biotechnology and Bioprocessing 2, no. 4 (2021): 01–17. http://dx.doi.org/10.31579/2766-2314/038.

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Background: Skeletal muscles are attached to bone and are responsible for the axial and appendicular movement of the skeleton and for maintenance of body position and posture. Objectives: The present review aimed to high light on embryonic development of skeletal muscles, histological and ultrastructure, innervation, contraction and relaxation, causes, pathophysiology, and treatment of volumetric muscle injury. The heterogeneity of the muscle fibers is the base of the flexibility which allows the same muscle to be used for various tasks from continuous low-intensity activity, to repeated subma
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Chen, Wan-Jing, I.-Hsuan Lin, Chien-Wei Lee, and Yi-Fan Chen. "Aged Skeletal Muscle Retains the Ability to Remodel Extracellular Matrix for Degradation of Collagen Deposition after Muscle Injury." International Journal of Molecular Sciences 22, no. 4 (2021): 2123. http://dx.doi.org/10.3390/ijms22042123.

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Aging causes a decline in skeletal muscle function, resulting in a progressive loss of muscle mass, quality, and strength. A weak regenerative capacity is one of the critical causes of dysfunctional skeletal muscle in elderly individuals. The extracellular matrix (ECM) maintains the tissue framework structure in skeletal muscle. As shown by previous reports and our data, the gene expression of ECM components decreases with age, but the accumulation of collagen substantially increases in skeletal muscle. We examined the structural changes in ECM in aged skeletal muscle and found restricted ECM
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Ramamani, A., M. M. Aruldhas, and P. Govindarajulu. "Differential response of rat skeletal muscle glycogen metabolism to testosterone and estradiol." Canadian Journal of Physiology and Pharmacology 77, no. 4 (1999): 300–304. http://dx.doi.org/10.1139/y99-016.

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Although reports on sex steroids have implicated them as promoting protein synthesis and also providing extra strength to the skeletal muscle, it remains unclear whether sex steroids affect glycogen metabolism to provide energy for skeletal muscle functions, since glycogen metabolism is one of the pathways that provides energy for the skeletal muscle contraction and relaxation cycle. The purpose of the current study was to show that testosterone and estradiol act differentially on skeletal muscles from different regions, differentially with reference to glycogen metabolism. To study this hypot
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Shiina, Takahiko, Takeshi Shima, Kazuaki Masuda, et al. "Contractile Properties of Esophageal Striated Muscle: Comparison with Cardiac and Skeletal Muscles in Rats." Journal of Biomedicine and Biotechnology 2010 (2010): 1–7. http://dx.doi.org/10.1155/2010/459789.

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The external muscle layer of the mammalian esophagus consists of striated muscles. We investigated the contractile properties of esophageal striated muscle by comparison with those of skeletal and cardiac muscles. Electrical field stimulation with single pulses evoked twitch-like contractile responses in esophageal muscle, similar to those in skeletal muscle in duration and similar to those in cardiac muscle in amplitude. The contractions of esophageal muscle were not affected by an inhibitor of gap junctions. Contractile responses induced by high potassium or caffeine in esophageal muscle wer
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Brooks, Susan V. "CURRENT TOPICS FOR TEACHING SKELETAL MUSCLE PHYSIOLOGY." Advances in Physiology Education 27, no. 4 (2003): 171–82. http://dx.doi.org/10.1152/advan.2003.27.4.171.

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Contractions of skeletal muscles provide the stability and power for all body movements. Consequently, any impairment in skeletal muscle function results in some degree of instability or immobility. Factors that influence skeletal muscle structure and function are therefore of great interest both scientifically and clinically. Injury, disease, and old age are among the factors that commonly contribute to impairment in skeletal muscle function. The goal of this article is to update current concepts of skeletal muscle physiology. Particular emphasis is placed on mechanisms of injury, repair, and
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Wu, G. Y., and J. R. Thompson. "Is methionine transaminated in skeletal muscle?" Biochemical Journal 257, no. 1 (1989): 281–84. http://dx.doi.org/10.1042/bj2570281.

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Methionine transamination is extensive in rat and chick skeletal-muscle homogenates, but is barely detectable in intact rat, but not chick, skeletal muscles. Branched-chain amino acids essentially block methionine transamination in intact muscles and homogenates from both species. The physiological significance of methionine transamination in skeletal muscle is questioned.
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Dissertations / Theses on the topic "Skeletal muscle"

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Foxton, Ruth. "Dysferlin in skeletal muscle and skeletal muscle disease." Thesis, University of Newcastle Upon Tyne, 2002. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.268429.

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Pathare, Neeti C. "Metabolic adaptations following disuse and their impact on skeletal muscle function." [Gainesville, Fla.] : University of Florida, 2005. http://purl.fcla.edu/fcla/etd/UFE0010024.

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Thesis (Ph.D.)--University of Florida, 2005.<br>Typescript. Title from title page of source document. Document formatted into pages; contains 171 pages. Includes Vita. Includes bibliographical references.
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Peoples, Gregory Edward. "Skeletal muscle fatigue can omega-3 fatty acids optimise skeletal muscle function? /." Access electronically, 2004. http://www.library.uow.edu.au/adt-NWU/public/adt-NWU20041217.123607.

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Thesis (Ph.D.)--University of Wollongong, 2004.<br>Typescript. This thesis is subject to a 12 month embargo (06/09/05 - 14/09/05) and may only be viewed and copied with the permission of the author. For further information please contact the Archivist. Includes bibliographical references: leaf 195-216.
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Salman, Mahmoud M. "Preconditioning in skeletal muscle." Thesis, University College London (University of London), 2008. http://discovery.ucl.ac.uk/1446109/.

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Ischaemia reperfusion injury of skeletal muscle is a major cause of morbidity and mortality in various surgical specialities. Developing a protective method or pharmacological agent that will limit this damage will be of considerable benefit to both patients and doctors. I have used potassium channel openers and calcium as preconditioning agents. The results show that potassium channel openers are a viable option whereas the use of calcium can exacerbate muscle damage. I looked at various protocols of ischaemic and pharmacological preconditioning. The results from both ischaemic and pharmacolo
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Blackwell, Danielle. "The role of Talpid3 in skeletal muscle satellite cells and skeletal muscle regeneration." Thesis, University of East Anglia, 2017. https://ueaeprints.uea.ac.uk/66948/.

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The primary cilium has recently been recognised as an essential regulator of the Sonic hedgehog (Shh) signalling pathway. Mutations that disrupt cilia function in humans can cause conditions known as ciliopathies. A wide range of phenotypes is observed in chick and mouse ciliopathy models,including polydactyly, craniofacial defects and polycystic kidneys. The Shh pathway and therefore primary cilia are vital for many developmental processes, including embryonic muscle development, with recent evidence suggesting they may also play a role in adult muscle regeneration. Our studies focus on the T
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Baker, Brent A. "Characterization of skeletal muscle performance and morphology following acute and chronic mechanical loading paradigms." Morgantown, W. Va. : [West Virginia University Libraries], 2007. https://eidr.wvu.edu/etd/documentdata.eTD?documentid=5325.

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Thesis (Ph. D.)--West Virginia University, 2007.<br>Title from document title page. Document formatted into pages; contains xii, 270 p. : ill. (some col.). Includes abstract. Includes bibliographical references.
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Zhang, Yan. "Cytokines and skeletal muscle wasting." Thesis, National Library of Canada = Bibliothèque nationale du Canada, 1999. http://www.collectionscanada.ca/obj/s4/f2/dsk2/ftp03/MQ47124.pdf.

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Oude, Vrielink Hubertus Hermanus Egbert. "Vasomotion and skeletal muscle perfusion." Maastricht : Maastricht : Rijksuniversiteit Limburg ; University Library, Maastricht University [Host], 1988. http://arno.unimaas.nl/show.cgi?fid=5409.

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Walsh, Garrett Lyndon. "Skeletal muscle powered cardiac assist." Thesis, McGill University, 1988. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=63879.

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Kochamba, Gary. "Skeletal muscle powered cardiac assist." Thesis, McGill University, 1988. http://digitool.Library.McGill.CA:80/R/?func=dbin-jump-full&object_id=61746.

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Books on the topic "Skeletal muscle"

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L, Mastaglia Frank, and Walton John Nicholas, eds. Skeletal muscle pathology. 2nd ed. Churchill Livingstone, 1992.

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Schmalbruch, Henning. Skeletal muscle. Springer-Verlag, 1985.

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Schmalbruch, Henning. Skeletal Muscle. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-82551-4.

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Walton, John N. Skeletal muscle pathology. 2nd ed. Churchill Livingstone, 1992.

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1957-, Prilutsky Boris I., ed. Biomechanics of skeletal muscle. Human Kinetics, 2012.

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Ryall, James G., ed. Skeletal Muscle Development. Springer New York, 2017. http://dx.doi.org/10.1007/978-1-4939-7283-8.

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Myers, Christopher, ed. Skeletal Muscle Physiology. Springer Nature Switzerland, 2024. http://dx.doi.org/10.1007/978-3-031-47065-3.

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Robert, Wortmann, ed. Diseases of the skeletal muscle. Lippincott Williams & Wilkins, 2000.

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Asakura, Atsushi, ed. Skeletal Muscle Stem Cells. Springer US, 2023. http://dx.doi.org/10.1007/978-1-0716-3036-5.

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1945-, Reznick A. Z., ed. Oxidative stress in skeletal muscle. Birkhäuser Verlag, 1998.

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Book chapters on the topic "Skeletal muscle"

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Schmalbruch, H. "Muscle Fibre Types in Mammalian Muscles." In Skeletal Muscle. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-82551-4_4.

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Schmalbruch, H. "General Overview." In Skeletal Muscle. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-82551-4_1.

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Schmalbruch, H. "Microanatomy of Muscle." In Skeletal Muscle. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-82551-4_2.

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Schmalbruch, H. "Skeletal Muscle Fibres." In Skeletal Muscle. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-82551-4_3.

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Schmalbruch, H. "Slow Muscle Fibres." In Skeletal Muscle. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-82551-4_5.

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Schmalbruch, H. "Non-Skeletal Muscles." In Skeletal Muscle. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-82551-4_6.

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Schmalbruch, H. "Development, Regeneration, Growth." In Skeletal Muscle. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-82551-4_7.

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Schmalbruch, H. "Muscle Fibres as Members of Motor Units." In Skeletal Muscle. Springer Berlin Heidelberg, 1985. http://dx.doi.org/10.1007/978-3-642-82551-4_8.

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Fung, Yuan-Cheng. "Skeletal Muscle." In Biomechanics. Springer New York, 1993. http://dx.doi.org/10.1007/978-1-4757-2257-4_9.

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Blottner, Dieter, and Michele Salanova. "Skeletal Muscle." In The NeuroMuscular System: From Earth to Space Life Science. Springer International Publishing, 2014. http://dx.doi.org/10.1007/978-3-319-12298-4_2.

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Conference papers on the topic "Skeletal muscle"

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Gao, Xuankai, Kohei Okasaki, Hirono Ohashi, Takeshi Sakurai, and Yuya Morimoto. "Tongue-Like Bioactuator with Multiple Skeletal Muscle Tissues." In 2025 IEEE 38th International Conference on Micro Electro Mechanical Systems (MEMS). IEEE, 2025. https://doi.org/10.1109/mems61431.2025.10918061.

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Odegard, G. M., T. L. Haut Donahue, D. A. Morrow, and K. R. Kaufman. "Constitutive Modeling of Skeletal Muscle Tissue." In ASME 2007 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2007. http://dx.doi.org/10.1115/sbc2007-175848.

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The main functions of the human musculoskeletal system are to sustain loads and provide mobility. Bones and joints themselves cannot produce movement; skeletal muscles provide the ability to move. Knowledge of muscle forces during given activities can provide insight into muscle mechanics, muscle physiology, musculoskeletal mechanics, neurophysiology, and motor control. However, clinical examinations or instrumented strength testing only provides information regarding muscle groups. Musculoskeletal models are typically needed to calculate individual muscle forces.
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NAGATOMI, RYOICHI. "SKELETAL MUSCLE AND HEALTH." In Proceedings of the Tohoku University Global Centre of Excellence Programme. IMPERIAL COLLEGE PRESS, 2012. http://dx.doi.org/10.1142/9781848169067_0003.

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Krivoi, Igor. "ENDOGENOUS OUABAIN AND SKELETAL MUSCLE." In XV International interdisciplinary congress "Neuroscience for Medicine and Psychology". LLC MAKS Press, 2019. http://dx.doi.org/10.29003/m446.sudak.ns2019-15/245-246.

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Ramirez, Angelica Maria, Begoña Calvo Calzada, and Jorge Grasa. "The Effect of the Fascia on the Stress Distribution in Skeletal Muscle." In ASME 2010 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2010. http://dx.doi.org/10.1115/sbc2010-19696.

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The human and vertebrate interaction with the environment is done primarily through the movement. This is possible due the skeletal muscle: anatomical structure able to contract voluntarily. The skeletal muscles are made up of contractile proteins which slide one over another allowing the muscle shortening and the body force generation. This protein structure of actin and myosin maintains its organization through the connective tissue that surrounds it (endomysium, perimysium and epimysium), creating arrays of myofibrils, fibre bundles, fascicles until conform the whole muscle. All this connec
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Neal, Devin, Mahmut Selman Sakar, and H. Harry Asada. "Bioengineered Fascicle-Like Skeletal Muscle Tissue Constructs." In ASME 2012 Summer Bioengineering Conference. American Society of Mechanical Engineers, 2012. http://dx.doi.org/10.1115/sbc2012-80228.

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Tissue engineered skeletal muscle constructs have and will continue to be valuable in treating, and testing various muscle injuries and diseases. However a significant drawback to numerous methods of producing 3D skeletal muscle constructs grown in vitro is that muscle cell density as a fraction of total volume or mass, is often significantly lower than muscle found in vivo. Therefore a method to increase muscle cell density within a construct is needed.
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Koeppen, Ryan, Meghan E. Huber, Dagmar Sternad, and Neville Hogan. "Controlling Physical Interactions: Humans Do Not Minimize Muscle Effort." In ASME 2017 Dynamic Systems and Control Conference. American Society of Mechanical Engineers, 2017. http://dx.doi.org/10.1115/dscc2017-5202.

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Physical interaction with tools is ubiquitous in functional activities of daily living. While tool use is considered a hallmark of human behavior, how humans control such physical interactions is still poorly understood. When humans perform a motor task, it is commonly suggested that the central nervous system coordinates the musculo-skeletal system to minimize muscle effort. In this paper, we tested if this notion holds true for motor tasks that involve physical interaction. Specifically, we investigated whether humans minimize muscle forces to control physical interaction with a circular kin
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Burmeister and Lehman. "Force Relaxation In Human Skeletal Muscle." In Proceedings of the Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.589829.

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Burmeister, E. E., and S. L. Lehman. "Force relaxation in human skeletal muscle." In 1992 14th Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE, 1992. http://dx.doi.org/10.1109/iembs.1992.5761946.

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Pohle, Regina, Ludwig von Rohden, and Dagmar Fisher. "Skeletal muscle sonography with texture analysis." In Medical Imaging 1997, edited by Kenneth M. Hanson. SPIE, 1997. http://dx.doi.org/10.1117/12.274164.

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Reports on the topic "Skeletal muscle"

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Walters, Thomas. Engineered Skeletal Muscle for Craniofacial Reconstruction. Defense Technical Information Center, 2011. http://dx.doi.org/10.21236/ada601864.

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C. Uy, Genevieve, Raymond L. Rosales, and Satish Khadilkar. Myopathies in Clinical Care: A Focus on Treatable Causes. Progress in Neurobiology, 2024. http://dx.doi.org/10.60124/j.pneuro.2024.10.01.

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Myopathies present a wide range of clinical symptoms that affect the skeletal muscles, including weakness, fatigue, and pain. While acquired myopathies receive significant attention due to the availability of treatment options, it is important to note that some inherited myopathies can also be effectively managed. These myopathies can be classified based on their underlying causes, such as infectious agents, autoimmune disorders leading to muscle inflammation, granulomatous inflammation, metabolic abnormalities within the muscle cells, skeletal muscle channel dysfunctions, prolonged ICU stay,
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Yang, Hui, Xi-Xi Wan, Hui Ma, et al. Prevalence and mortality risk of low skeletal muscle mass in critically ill patients: an updated systematic review and meta-analysis. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2022. http://dx.doi.org/10.37766/inplasy2022.11.0132.

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Review question / Objective: The PICOS principle was adopted when we confirmed the study eligibility. The inclusion criteria were as follows: (1) patients were critically ill, which was defined as adult patients who were from the ICU department; (2) exposure: patients had a clear definition of LSMM based on CT scans, anthropometric methods and ultrasound; (3) presented the prevalence of LSMM or could be calculated by the available data from the article; and (4) study design: observational study (cohort study or cross-sectional study). Articles that were reviews, case reports, comments, corresp
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Kuo, Meng-Hsuan, Chih-Wei Tseng, Ching-Sheng Hsu, Yen-Chun Chen, I.-Ting Kao, and Chen-Yi Wu. Protocol for systematic review and meta-analysis of prognostic value of sarcopenia in advanced HCC patients treating with systemic therapy. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2023. http://dx.doi.org/10.37766/inplasy2023.2.0011.

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Review question / Objective: P: Advanced HCC patients under systemic therapy; I: low skeletal muscle mass (LSMM); C: Non-LSMM; O:overall survival or mortality. Eligibility criteria: (1) cohort studies or cross sectional studies investigations with HCC patients treated with systemic therapy; (2) the articles estimated pretreatment skeletal muscle mass measured by CT-images; (3) studies provided statistical data about the prevalence pretreatment LSMM or influence of LSMM on OS orPFS.
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Buck, Edmond. Mechanism of Calcium Release from Skeletal Muscle Sarcoplasmic Reticulum. Portland State University Library, 2000. http://dx.doi.org/10.15760/etd.1306.

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Phillips, Stuart, Kyle Lau, Alysha D'Souza та Everson Nunes. An umbrella review of systematic reviews of β-hydroxy-β-methyl butyrate (HMB) supplementation in promoting skeletal muscle mass and function in aging and clinical practice. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.10.0072.

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Review question / Objective: An umbrella review of systematic reviews of the use of β-hydroxy-β-methyl butyrate (HMB) supplementation in promoting skeletal muscle mass and function in aging and clinical practice. Condition being studied: Muscle mass (and various proxies thereof), strength, and physical function. Information sources: Pubmed, Web of Science, Embase.
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Holdsworth, Clark, Steven Copp, Tadakatsu Inagaki, et al. Chronic (-)-epicatechin administration does not affect contracting skeletal muscle microvascular oxygenation. Peeref, 2022. http://dx.doi.org/10.54985/peeref.2206p3750191.

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Jalil, Yorschua, and Ruvistay Gutierrez. Myokines secretion and their role in critically ill patients. A scoping review protocol. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, 2021. http://dx.doi.org/10.37766/inplasy2021.9.0048.

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Review question / Objective: 1-How and by which means stimulated muscle from critically ill patients can liberate myokines?, 2-Which are the main characteristics of the critically ill population studied and if some of these influenced myokine´s secretion?, 5-Can myokines exert local or distant effects in critically ill patients?, 5-Which are the potential effects of myokines in critically ill patients? Eligibility criteria: Participants and context: We will include primary studies (randomized or non-randomized trials, observational studies, case series or case report) that consider hospitalize
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Koh, Timothy J. Enhancement of Skeletal Muscle Repair by the Urokinase-Type Plasminogen Activator System. Defense Technical Information Center, 2006. http://dx.doi.org/10.21236/ada448526.

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Xiong, Hui. Modification of the CA²⁺ Release Channel from Sarcoplasmic Reticulum of Skeletal Muscle. Portland State University Library, 2000. http://dx.doi.org/10.15760/etd.1303.

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