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Journal articles on the topic 'Skeletal muscular system'

Author: Grafiati

Published: 10 January 2023

Last updated: 31 July 2025

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1

Gerçek, Cem. "Modelling the Subjects of Skeletal and Muscular System: Mobile Applications." Journal of Qualitative Research in Education 7, no. 1 (2019): 1–16. http://dx.doi.org/10.14689/issn.2148-2624.1.7c1s.10m.

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2

Endo, Hiroshi, and Mitsuo Wada. "A Musculo-skeletal Mechanism Simulating Human Forearm and Its Control Method." Journal of Robotics and Mechatronics 5, no. 3 (1993): 248–52. http://dx.doi.org/10.20965/jrm.1993.p0248.

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In the human muscular system, each muscle can regulate its compliance, all of the muscles act in opposition to each other, and certain muscles act on several joints. This mechanism serves for skillful human motions. To realize an improved manipulator, we implemented this mechanism into our manipulator which has a musculo-skeletal mechanism similar to a human forearm. The manipulator is driven by the tendon-driven system where the actions of the tendon-driven system are similar to those of the human muscular system. The manipulator has 2 D.O.F and is driven by three contending wires. In additio

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3

Gimranova, Galina G., A. B. Bakirov, E. R. Shaikhlislamova, L. K. Karimova, N. A. Beigul, and L. N. Mavrina. "MUSCULO-SKELETAL AND PERIPHERAL NERVOUS DISEASES IN EMPLOYEES OF THE OIL INDUSTRY IN CONDITIONS OF THE COMBINED IMPACT OF VIBRATION AND THE HEAVY WORKING PROCESS." Hygiene and sanitation 96, no. 6 (2019): 552–55. http://dx.doi.org/10.18821/0016-9900-2017-96-6-552-555.

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There were studied working conditions and the state of the muscular-skeletal system in employees of the oil industry. Working conditions of employees of basic occupations are referred to the hazard Class 3.1 - 3.3. Significant physical exertion with the predominant load on the lumbar-sacral area, constrained posture, the exposure to the general vibration and adverse weather conditions contribute to the development of diseases of the muscular-skeletal and peripheral nervous system. The most perspective trend in solving problems of diseases of the muscular-skeletal system is the implementation o

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4

Schoenau, Eckhard. "Muscular System Is the Driver of Skeletal Development." Annales Nestlé (English ed.) 64, no. 2 (2006): 55–61. http://dx.doi.org/10.1159/000093012.

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5

Elhamrawy, Amr, Islam Elmitwalli, Candice Burrier, Giorgio Veneziano, and Joseph D. Tobias. "Regional anesthesia and muscle-wasting diseases in pediatrics: A focused educational review." Saudi Journal of Anaesthesia 19, no. 1 (2025): 86–91. https://doi.org/10.4103/sja.sja_567_24.

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The muscular dystrophies or muscle-wasting diseases include a diverse group of genetic disorders, which result in progressive degeneration of skeletal muscles, progressive muscle weakness, and comorbid multi-system involvement. Duchenne muscle dystrophy is the most common type of muscular dystrophy with a reported incidence of 1 in every 3500–6000 male live births in the United States. Given the progressive nature of these disorders, skeletal muscle weakness frequently progresses to loss of the ability to ambulate and perform functions of daily life. In addition to affecting the skeletal muscu

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6

Akhtaruzzaman, M., A. A. Shafie, and M. R. Khan. "A REVIEW ON LOWER APPENDICULAR MUSCULOSKELETAL SYSTEM OF HUMAN BODY." IIUM Engineering Journal 17, no. 1 (2016): 83–102. http://dx.doi.org/10.31436/iiumej.v17i1.571.

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Rehabilitation engineering plays an important role in designing various autonomous robots to provide better therapeutic exercise to disabled patients. Hence it is necessary to study human musculoskeletal system and also needs to be presented in scientific manner in order to describe and analyze the biomechanics of human body motion. This review focuses on lower appendicular musculoskeletal structure of human body to represent joints and links architectures; to identify muscle attachments and functions; and to illustrate muscle groups which are responsible for a particular joint movement. First

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Piróg, Katarzyna A., and Michael D. Briggs. "Skeletal Dysplasias Associated with Mild Myopathy—A Clinical and Molecular Review." Journal of Biomedicine and Biotechnology 2010 (2010): 1–13. http://dx.doi.org/10.1155/2010/686457.

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Musculoskeletal system is a complex assembly of tissues which acts as scaffold for the body and enables locomotion. It is often overlooked that different components of this system may biomechanically interact and affect each other. Skeletal dysplasias are diseases predominantly affecting the development of the osseous skeleton. However, in some cases skeletal dysplasia patients are referred to neuromuscular clinics prior to the correct skeletal diagnosis. The muscular complications seen in these cases are usually mild and may stem directly from the muscle defect and/or from the altered interac

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8

Staroseltseva, Natalia. "Muscular pain phenomena." Manual Therapy, no. 1 (May 3, 2022): 78–84. http://dx.doi.org/10.54504/1684-6753-2022-1-78-84.

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Noted was an apparent overdiagnosis of osteochondrosis as a major cause of pain syndrome, and along with it, underestimation of the role of functional disorders of the musculoskeletal system with the appearance of a reversible block in the small and large joints and the formation of musculo-skeletal pain syndromes. The main cause of back, neck and limb pain are myofascial pain syndromes, including manifestations of the fibro-myalgic syndrome.

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9

Tripodi, Luana, Chiara Villa, Davide Molinaro, Yvan Torrente, and Andrea Farini. "The Immune System in Duchenne Muscular Dystrophy Pathogenesis." Biomedicines 9, no. 10 (2021): 1447. http://dx.doi.org/10.3390/biomedicines9101447.

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Growing evidence demonstrates the crosstalk between the immune system and the skeletal muscle in inflammatory muscle diseases and dystrophic conditions such as Duchenne Muscular Dystrophy (DMD), as well as during normal muscle regeneration. The rising of inflammation and the consequent activation of the immune system are hallmarks of DMD: several efforts identified the immune cells that invade skeletal muscle as CD4+ and CD8+ T cells, Tregs, macrophages, eosinophils and natural killer T cells. The severity of muscle injury and inflammation dictates the impairment of muscle regeneration and the

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10

Golovskoy, B. V., M. D. Berg, I. A. Bulatova, E. I. Voronova, and Ya B. Khovaeva. "Muscular system in maintaining health and preventing chronic non-infectious diseases." Perm Medical Journal 38, no. 1 (2021): 72–86. http://dx.doi.org/10.17816/pmj38172-86.

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Chronic non-infectious diseases are the leading cause of premature death according to the WHO data. This review considers the evolution of studying and modern views of domestic and foreign authors on the role of skeletal muscles in maintaining health and preventing chronic non-infectious diseases. An idea regarding the nervous and reflex influence from the working muscles on both individual organs and the body as a whole is presented. The role of myokines, specific proteins produced by skeletal muscles, capable of influencing the state of many organs and systems, is described.

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11

Powell, Jeanne A. "Muscular dysgenesis: a model system for studying skeletal muscle development." FASEB Journal 4, no. 10 (1990): 2798–808. http://dx.doi.org/10.1096/fasebj.4.10.2197156.

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12

Lu, Yuxuan. "Review on the development of occupation-related assistive exoskeletons." Applied and Computational Engineering 9, no. 1 (2023): 82–85. http://dx.doi.org/10.54254/2755-2721/9/20230053.

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Many different types of beneficial exoskeletons have been developed in recent years. The majority of them are intended to lessen the likelihood of a variety of lumbar compression injuries and other types of ailments that workers may be subjected to when they are engaged in repetitive single-activity labour. Work-related muscular and skeletal disorders, often known as WMSDs, are the most prevalent type of muscular and skeletal disorder found in the working population. It is brought on by a single motion that is repeated over and over again. This condition of the muscular and skeletal system sig

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13

Kalinin, R. E., I. A. Suchkov, N. D. Mzhavanadze, and N. V. Korotkova. "Endothelial dysfunction in muscular dystrophies." NAUKA MOLODYKH (Eruditio Juvenium) 9, no. 2 (2021): 326–34. http://dx.doi.org/10.23888/hmj202192326-334.

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Endotheliocytes are the key elements of the vascular wall and are involved in regulation of vascular tone and permeability, inflammation, hemostasis, angiogenesis etc. Impaired function of endothelial cells universally recognized as endothelial dysfunction is associated with a number of common diseases such as ischemic heart disease, arterial hypertension, atherosclerosis, peripheral arterial disease, septic shock, chronic kidney disease, obesity, oncological and autoimmune diseases. Less is known about the role of endothelial cells in pathogenesis of development and progression of rare diseas

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14

Granado, Miriam, Teresa Priego, Ana I. Martín, Mª Ángeles Villanúa, and Asunción López-Calderón. "Ghrelin receptor agonist GHRP-2 prevents arthritis-induced increase in E3 ubiquitin-ligating enzymes MuRF1 and MAFbx gene expression in skeletal muscle." American Journal of Physiology-Endocrinology and Metabolism 289, no. 6 (2005): E1007—E1014. http://dx.doi.org/10.1152/ajpendo.00109.2005.

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Chronic arthritis is a catabolic state associated with an inhibition of the IGF system and a decrease in body weight. Cachexia and muscular wasting is secondary to protein degradation by the ubiquitin-proteasome pathway. The aim of this work was to analyze the effect of adjuvant-induced arthritis on the muscle-specific ubiquitin ligases muscle ring finger 1 ( MuRF1) and muscle atrophy F-box ( MAFbx) as well as on IGF-I and IGF-binding protein-5 ( IGFBP-5) gene expression in the skeletal muscle. We also studied whether the synthetic ghrelin receptor agonist, growth hormone releasing peptide-2 (

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15

Rahman, Fasih Ahmad, and Matthew Paul Krause. "PAI-1, the Plasminogen System, and Skeletal Muscle." International Journal of Molecular Sciences 21, no. 19 (2020): 7066. http://dx.doi.org/10.3390/ijms21197066.

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The plasminogen system is a critical proteolytic system responsible for the remodeling of the extracellular matrix (ECM). The master regulator of the plasminogen system, plasminogen activator inhibitor-1 (PAI-1), has been implicated for its role in exacerbating various disease states not only through the accumulation of ECM (i.e., fibrosis) but also its role in altering cell fate/behaviour. Examination of PAI-1 has extended through various tissues and cell-types with recent investigations showing its presence in skeletal muscle. In skeletal muscle, the role of this protein has been implicated

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16

Chaitow, Leon. "The Muscular System Manual: The Skeletal Muscles of the Human Body." Journal of Bodywork and Movement Therapies 8, no. 1 (2004): 72. http://dx.doi.org/10.1016/s1360-8592(03)00059-7.

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17

Bozzi, Manuela, Francesca Sciandra, Maria Giulia Bigotti, and Andrea Brancaccio. "Misregulation of the Ubiquitin–Proteasome System and Autophagy in Muscular Dystrophies Associated with the Dystrophin–Glycoprotein Complex." Cells 14, no. 10 (2025): 721. https://doi.org/10.3390/cells14100721.

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The stability of the sarcolemma is severely impaired in a series of genetic neuromuscular diseases defined as muscular dystrophies. These are characterized by the centralization of skeletal muscle syncytial nuclei, the replacement of muscle fibers with fibrotic tissue, the release of inflammatory cytokines, and the disruption of muscle protein homeostasis, ultimately leading to necrosis and loss of muscle functionality. A specific subgroup of muscular dystrophies is associated with genetic defects in components of the dystrophin–glycoprotein complex (DGC), which plays a crucial role in linking

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18

Khabirov, R. A. "Muscular syndrome in patients with inflammatory and degenerative diseases of joints and vertebral column." Kazan medical journal 80, no. 2 (1999): 113–16. http://dx.doi.org/10.17816/kazmj65368.

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The manifestations of muscular syndrome affecting the gravity and prediction of the disease take place in the most widespread and invalidizing rheumatic diseases: osteoarthrosis, rheumatoid arthritis and ankylosing spondylarthritis. Paraclinical studies showed heterogeneity of pathogenetic mechanisms in lesion of skeletal muscles in rheumatic diseases. The differentiated methods of the treatment of patients with osteoarthrosis, rheumatoid arthritis and ankylosing spondylarthritis taking into account the clinical picture and pathogenesis of muscular syndrome, as well as the diagnosis criteria a

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19

Chernova, O. N., I. N. Korsakov, D. P. Samchuk, et al. "Experimental models for studying of skeletal muscles regeneration." Genes & Cells 10, no. 4 (2015): 127–40. http://dx.doi.org/10.23868/gc120536.

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Striated muscles play an important role in the maintenance in the maintenance of locomotion, ventilation, mechanical protection, the inner organs support, a common system of energy exchange etc. Skeletal muscle tissue is exposed to various external factors which cause notable damage to skeletal tissue as a result of mechanical injury (contusion, compression, laceration), inflammation as an implication of infectious agents and autoimmune process, toxic effects of various chemical substances. Besides the external causes, genes' defects that code muscle protein components have influence on the mu

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20

Mirzoev, T. M., and B. S. Shenkman. "Mechanosensory Structures in the Mechanotransduction System of Muscle Fibers." Российский физиологический журнал им И М Сеченова 109, no. 8 (2023): 1005–27. http://dx.doi.org/10.31857/s0869813923080083.

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The ability of skeletal muscles to sense mechanical stimuli and respond to them by changing intracellular electrochemical and biochemical processes (mechanotransduction) is of crucial importance for the regulation of physiological processes in muscle fibers. This review describes the main sarcolemmal, sarcomeric, and cytoskeletal mechanosensitive structures and analyzes mechano-dependent signaling pathways and mechanisms involved in the regulation of gene expression as well as muscle protein synthesis and degradation. The final part of the review formulates specific questions in the field of m

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21

Gamucci, Fiorenza, Marcello Pallante, Sybille Molle, Enrico Merlo, and Andrea Bertuglia. "A Preliminary Study on the Use of HD-sEMG for the Functional Imaging of Equine Superficial Muscle Activation during Dynamic Mobilization Exercises." Animals 12, no. 6 (2022): 785. http://dx.doi.org/10.3390/ani12060785.

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Superficial skeletal muscle activation is associated with an electric activity. Bidimensional High-Density Surface Electromyography (HD-sEMG) is a non-invasive technique that uses a grid of equally spaced electrodes applied on the skin surface to detect and portray superficial skeletal muscle activation. The goal of the study was to evaluate the feasibility of HD-sEMG to detect electrical activation of skeletal muscle and its application during rehabilitation exercises in horses. To fulfil this aim, activation of the superficial descending pectoral and external abdominal oblique core muscles w

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22

Chikani, Viral, and Ken K. Y. Ho. "Action of GH on skeletal muscle function: molecular and metabolic mechanisms." Journal of Molecular Endocrinology 52, no. 1 (2013): R107—R123. http://dx.doi.org/10.1530/jme-13-0208.

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Skeletal muscle is a target tissue of GH. Based on its anabolic properties, it is widely accepted that GH enhances muscle performance in sports and muscle function in the elderly. This paper critically reviews information on the effects of GH on muscle function covering structure, protein metabolism, the role of IGF1 mediation, bioenergetics and performance drawn from molecular, cellular and physiological studies on animals and humans. GH increases muscle strength by enhancing muscle mass without affecting contractile force or fibre composition type. GH stimulates whole-body protein accretion

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23

Ábrigo, Johanna, Alvaro A. Elorza, Claudia A. Riedel, et al. "Role of Oxidative Stress as Key Regulator of Muscle Wasting during Cachexia." Oxidative Medicine and Cellular Longevity 2018 (2018): 1–17. http://dx.doi.org/10.1155/2018/2063179.

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Skeletal muscle atrophy is a pathological condition mainly characterized by a loss of muscular mass and the contractile capacity of the skeletal muscle as a consequence of muscular weakness and decreased force generation. Cachexia is defined as a pathological condition secondary to illness characterized by the progressive loss of muscle mass with or without loss of fat mass and with concomitant diminution of muscle strength. The molecular mechanisms involved in cachexia include oxidative stress, protein synthesis/degradation imbalance, autophagy deregulation, increased myonuclear apoptosis, an

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24

Mandal, Tapati, Pupree Mutsuddy, Mezbah Ahmed, Khokon Kumar Nath, Abu Bakker Siddique, and Shamim MF Begum. "Rare Presentations of Muscular Infiltrations in Classical Hodgkin’s lymphoma-A Case Series." Bangladesh Journal of Nuclear Medicine 25, no. 2 (2023): 143–46. http://dx.doi.org/10.3329/bjnm.v25i2.64653.

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Hodgkin’s lymphoma (HL) is a malignant condition of the lymphoid system that involves supradiaphragmatic recesses, and typically presents with lymphadenopathy and spreads from one nodal group to the other. Skeletal muscle infiltration in HL is uncommon. Two cases of classical HL evaluated by an 18F-FDG PET-CT scan detecting multiple extranodal metastases along with skeletal muscle involvement are reported here, and their outcomes are discussed along with the importance of the PET-CT scan as a good imaging modality. Bangladesh J. Nuclear Med. 25(2): 143-146, 2022

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Waldrop, T. G., and J. H. Mitchell. "Effects of barodenervation on cardiovascular responses to static muscular contraction." American Journal of Physiology-Heart and Circulatory Physiology 249, no. 4 (1985): H710—H714. http://dx.doi.org/10.1152/ajpheart.1985.249.4.h710.

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The purpose of this study was to measure blood flow to various tissues during static muscular contraction in anesthetized cats and to evaluate if the baroreflex modulates the cardiovascular responses to muscular contraction. Contraction of the hindlimb muscles induced by ventral root stimulation caused increases in arterial pressure (delta 37.8 +/- 5.5 mmHg) and heart rate (delta 13.9 +/- 3.1 beats/min). Increases in blood flow to the heart, working skeletal muscles, and selected areas of the central nervous system occurred during muscular contraction. Blood flow to visceral organs did not cha

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Suzuki, Naoki, Asaki Hattori, Mitsuhito Hayashibe, Yoshito Otake, Shigeyuki Suzuki, and Susumu Kobayashi. "4D analysis of skeletal and muscular system during locomotion using dynamic spatial video camera system." International Congress Series 1268 (June 2004): 1239. http://dx.doi.org/10.1016/j.ics.2004.03.317.

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27

Ohlendorf, Daniela, Yvonne Haas, Antonia Naser, et al. "Prevalence of Muscular Skeletal Disorders among Qualified Dental Assistants." International Journal of Environmental Research and Public Health 17, no. 10 (2020): 3490. http://dx.doi.org/10.3390/ijerph17103490.

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The occupation of dental assistants (DAs) involves many health risks of the musculoskeletal system due to static and prolonged work, which can lead to musculoskeletal disorders (MSDs). The aim of the study was to investigate the prevalence of MSDs in DAs in Germany. Methods: For this purpose, an online questionnaire analyzed 406 (401 female participants and 5 male participants, 401w/5m) DAs. It was based on the Nordic Questionnaire (lifetime, 12-month, and seven-day MSDs’ prevalence separated into neck, shoulder, elbow, wrist, upper back, lower back, hip, knee, and ankle), and occupational and

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28

IVANCEVIC, VLADIMIR. "LIE–LAGRANGIAN MODEL FOR REALISTIC HUMAN BIODYNAMICS." International Journal of Humanoid Robotics 03, no. 02 (2006): 205–18. http://dx.doi.org/10.1142/s0219843606000680.

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We present a sophisticated Lagrangian model for anatomically and physiologically realistic human biodynamics (RHB), to accompany the recently reported Hamiltonian formulation.1 The present RHB formulation is designed around three main modules: (i) A Riemannian configuration manifold, composed of gauge Lie groups of constrained 3D rotations and translations, which includes more than 300 degrees of freedom (DOF); (ii) exterior Lagrangian dynamics of the human musculo-skeletal system, including all natural conservative, dissipative and driving forces, powered by 600 equivalent muscles; and (iii)

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Zavyalova, A. N., V. P. Novikova, and P. D. Ignatova. "Axis “microbiota - muscle”." Experimental and Clinical Gastroenterology, no. 11 (January 23, 2023): 60–69. http://dx.doi.org/10.31146/1682-8658-ecg-207-11-60-69.

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The luminal intestinal microflora affects the state of the muscular system of experimental animals and humans. The role of nutrition in the development of skeletal muscles is considered. The role of metabolically active substances secreted by the microbiota in the development of cachexia, sarcopenia, and kwashiorkor is considered. The effect of exercise on the gut microbiome was assessed. Possible variants of the influence of probiotics on the state of skeletal muscles and its functional activity in the experiment in animals and humans are presented.

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dos Santos, Leandro, Alex Ribeiro, Edilaine Cavalcante, et al. "Effects of Modified Pyramid System on Muscular Strength and Hypertrophy in Older Women." International Journal of Sports Medicine 39, no. 08 (2018): 613–18. http://dx.doi.org/10.1055/a-0634-6454.

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AbstractThis study aimed to analyze the effects of a pyramid system performed with two repetition zones on muscular strength and skeletal muscle mass (SMM) in older women. Thirty-nine physically independent older women (67.8±5.4 years) were randomly assigned into one of two of groups that performed an 8-week resistance training program in an ascending pyramid fashion. Both groups performed 3 sets: a narrow repetition zone (NPR, n=20) with 12/10/8 repetitions, and a wide repetition zone (WPR, n=19) with 15/10/5 repetitions. The program consisted of 8 whole-body exercises, performed 3 times a we

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Aizawa, Katsuji, Motoyuki Iemitsu, Takeshi Otsuki, Seiji Maeda, Takashi Miyauchi, and Noboru Mesaki. "Sex differences in steroidogenesis in skeletal muscle following a single bout of exercise in rats." Journal of Applied Physiology 104, no. 1 (2008): 67–74. http://dx.doi.org/10.1152/japplphysiol.00558.2007.

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Sex steroid hormones, such as testosterone and estradiol, play important roles in developing both strength and mass of skeletal muscle. Recently, we demonstrated that skeletal muscle can synthesize sex steroid hormones. Whether there are sex differences in basal steroidogenesis or acute exercise-induced alterations of steroidogenesis in the skeletal muscle is unknown. We examined sex differences in the levels of testosterone, estradiol, and steroidogenesis-related enzymes, such as 17β-hydroxysteroid dehydrogenase (HSD), 3β-HSD, and aromatase cytochrome P-450 (P450arom), in the skeletal muscle

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32

Kindler, Urs, Lampros Mavrommatis, Franziska Käppler, et al. "Duchenne Muscular Dystrophy Patient iPSCs—Derived Skeletal Muscle Organoids Exhibit a Developmental Delay in Myogenic Progenitor Maturation." Cells 14, no. 13 (2025): 1033. https://doi.org/10.3390/cells14131033.

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Background: Duchenne muscular dystrophy (DMD), which affects 1 in 3500 to 5000 newborn boys worldwide, is characterized by progressive skeletal muscle weakness and degeneration. The reduced muscle regeneration capacity presented by patients is associated with increased fibrosis. Satellite cells (SCs) are skeletal muscle stem cells that play an important role in adult muscle maintenance and regeneration. The absence or mutation of dystrophin in DMD is hypothesized to impair SC asymmetric division, leading to cell cycle arrest. Methods: To overcome the limited availability of biopsies from DMD p

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Mosca, Nicola, Sara Petrillo, Sara Bortolani, et al. "Redox Homeostasis in Muscular Dystrophies." Cells 10, no. 6 (2021): 1364. http://dx.doi.org/10.3390/cells10061364.

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In recent years, growing evidence has suggested a prominent role of oxidative stress in the pathophysiology of several early- and adult-onset muscle disorders, although effective antioxidant treatments are still lacking. Oxidative stress causes cell damage by affecting protein function, membrane structure, lipid metabolism, and DNA integrity, thus interfering with skeletal muscle homeostasis and functionality. Some features related to oxidative stress, such as chronic inflammation, defective regeneration, and mitochondrial damage are shared among most muscular dystrophies, and Nrf2 has been sh

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Lezhenko, G., O. Pashkova, K. Samoylyk, and A. Brutman. "Functional and biochemical characteristics of the muscle system in children with type I diabetes." CHILD`S HEALTH 17, no. 5 (2022): 249–55. http://dx.doi.org/10.22141/2224-0551.17.5.2022.1525.

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Background. The purpose of the study was to determine possible markers of skeletal muscle damage in children with type 1 diabetes mellitus (T1DM) and their relationship with the features of disease course. Materials and methods. The observation group consisted of 98 children with type 1 diabetes mellitus: the first group included 22 people without disorders of the muscular system; the second — 42 patients with dynapenia; the third — 34 children with diabetic myopathy. Control group — 30 relatively healthy children. Assessment of the static endurance of skeletal muscles, determination of the le

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Arora, Shivani, and Chirag Arora. "A REVIEW ON CONCEPTUAL STUDY OF MUSCULAR DYSTROPHIES: AN AYURVEDIC PERSPECTIVE." International Journal of Research in Ayurveda and Pharmacy 13, no. 01 (2022): 92–95. http://dx.doi.org/10.7897/2277-4343.130121.

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Muscular dystrophies (M.D.) are a category of hereditary illnesses defined by degeneration of the skeletal muscles that control mobility, culminating in muscle atrophy, weakness, confinement to a wheelchair, and eventually death. The most common muscular dystrophy is Duchenne muscular dystrophy (DMD), caused by a lack of the dystrophin gene. In Ayurvedic scriptures, muscular dystrophy is not explicitly linked to any ailment. The notion of Adibala pravritta vyadhi in Ayurveda helps to explain this aetiology. Pathogenesis occurs here due to Beejobhagavayava dushti, which leads to Mamsa Vata dush

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Dalle Carbonare, Luca, Arianna Minoia, Sharazed Zouari, et al. "Crosstalk between Bone and Muscles during Physical Activity." Cells 12, no. 16 (2023): 2088. http://dx.doi.org/10.3390/cells12162088.

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Bone–muscle crosstalk is enabled thanks to the integration of different molecular signals, and it is essential for maintaining the homeostasis of skeletal and muscle tissue. Both the skeletal system and the muscular system perform endocrine activity by producing osteokines and myokines, respectively. These cytokines play a pivotal role in facilitating bone–muscle crosstalk. Moreover, recent studies have highlighted the role of non-coding RNAs in promoting crosstalk between bone and muscle in physiological or pathological conditions. Therefore, positive stimuli or pathologies that target one of

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Pomeshchikova, Irina. "INFLUENCE OF EXERCISES AND GAMES WITH BALL ON VESTIBULAR STABILITY OF STUDENTS WITH MUSCULAR-SKELETAL APPARATUS DISORDERS." Sport Science INFLUENCE OF EXERCISES AND GAMES WITH BALL ON VESTIBULAR STABILITY OF STUDENTS WITH MUSCULAR-SKELETAL APPARATUS DISORDERS, no. 1(9) (2016): 75–83. https://doi.org/10.5281/zenodo.2580353.

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Purpose: to work out approaches to increase of vestibular stability of students with muscular skeletal apparatus disorders by means of outdoor games and exercises with ball. Material: in the research students with disorders of muscular-skeletal apparatus (n=63, age 16 – 19 years, boys and girls) participated. The research was being fulfilled during 1.5 years (three academic semesters). The students had disablement with mild or average disorders. All students were under medical observation and had no counter indications to physical education. Testing of vestibular stability was conducted

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38

K, Sathyavikasini, and Vijaya M S. "Identification of Rare Genetic Disorder from Single Nucleotide Variants Using Supervised Learning Technique." IAES International Journal of Artificial Intelligence (IJ-AI) 6, no. 4 (2017): 174. http://dx.doi.org/10.11591/ijai.v6.i4.pp174-184.

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Muscular dystrophy is a rare genetic disorder that affects the muscular system which deteriorates the skeletal muscles and hinders locomotion. In the finding of genetic disorders such as Muscular dystrophy, the disease is identified based on mutations in the gene sequence. A new model is proposed for classifying the disease accurately using gene sequences, mutated by adopting positional cloning on the reference cDNA sequence. The features of mutated gene sequences for missense, nonsense and silent mutations aims in distinguishing the type of disease and the classifiers are trained with commonl

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Pinheiro, João Augusto Recliski, Luiz Augusto Da Silva, Vinicius Muller Reis Weber, Carlos Ricardo Maneck Malfatti, and Marcos Roberto Brasil. "Muscular Hypertrophic Effects of Oral Contraceptive Consumption in Women." Journal of Pharmacy and Nutrition Sciences 13 (December 31, 2023): 75–81. http://dx.doi.org/10.29169/1927-5951.2023.13.08.

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Hormones have several actions in the body; among their effects on effector organs, they can control the volume of skeletal muscle. The excess of hormones, in this case exogenous ones, could be altering the effects of physical exercise on skeletal muscle hypertrophy, especially after the consumption of contraceptives. Thus, we have a general objective in this work to evaluate the effects of contraceptive consumption on hypertrophy in women. We sought an integrative literature review with studies within 2001-2021, using the PubMed and Scielo databases. Seven articles were selected for a complete

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Cartier, L. J., and P. D. Gollnick. "Sympathoadrenal system and activation of glycogenolysis during muscular activity." Journal of Applied Physiology 58, no. 4 (1985): 1122–27. http://dx.doi.org/10.1152/jappl.1985.58.4.1122.

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Comparisons were made of the appearance of phosphorylase (PHOS) a and lactate (LA) during electrical stimulation of the gastrocnemius (GM) and soleus (SM) muscles of normal and sympathectomized (SYMPX) rats. Ten-second stimulation at 3 Hz increased PHOS a approximately fourfold in the GM of normal rats, whereafter it declined during stimulation until at 60 s it was similar to rest. The increase in PHOS a of GM from SYMPX rats after 10 s of stimulation was approximately 50% that of normal rats. Stimulation of the SM produced smaller and slower increases in PHOS a with the peak occurring after 6

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Nguyen, Vinh-Lac, Kwang-Bok Lee, and Young Jae Moon. "Parathyroid Hormone as a Modulator of Skeletal Muscle: Insights into Bone–Muscle and Nerve–Muscle Interactions." International Journal of Molecular Sciences 26, no. 15 (2025): 7060. https://doi.org/10.3390/ijms26157060.

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Parathyroid hormone (PTH) has been studied to determine its broader role in musculoskeletal health, particularly its effects on skeletal muscle. Bone and muscle are inextricably linked via mechanical loading and biochemical signaling, with both processes playing important roles in muscular metabolism and function. Furthermore, the nervous system must maintain muscle mass and function, as neuromuscular transmission controls muscle contraction, protein synthesis, and energy metabolism. As a systemic endocrine regulator, PTH influences the physiology of skeletal muscle—both directly and through i

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Coles, Chantal A., Ian Woodcock, Daniel G. Pellicci, and Peter J. Houweling. "A Spotlight on T Lymphocytes in Duchenne Muscular Dystrophy—Not Just a Muscle Defect." Biomedicines 10, no. 3 (2022): 535. http://dx.doi.org/10.3390/biomedicines10030535.

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The lack of dystrophin in Duchenne muscular dystrophy (DMD) results in membrane fragility resulting in contraction-induced muscle damage and subsequent inflammation. The impact of inflammation is profound, resulting in fibrosis of skeletal muscle, the diaphragm and heart, which contributes to muscle weakness, reduced quality of life and premature death. To date, the innate immune system has been the major focus in individuals with DMD, and our understanding of the adaptive immune system, specifically T cells, is limited. Targeting the immune system has been the focus of multiple clinical trial

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Volonte, Daniela, Aaron J. Peoples, and Ferruccio Galbiati. "Modulation of Myoblast Fusion by Caveolin-3 in Dystrophic Skeletal Muscle Cells: Implications for Duchenne Muscular Dystrophy and Limb-Girdle Muscular Dystrophy-1C." Molecular Biology of the Cell 14, no. 10 (2003): 4075–88. http://dx.doi.org/10.1091/mbc.e03-03-0161.

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Caveolae are vesicular invaginations of the plasma membrane. Caveolin-3 is the principal structural component of caveolae in skeletal muscle cells in vivo. We have recently generated caveolin-3 transgenic mice and demonstrated that overexpression of wild-type caveolin-3 in skeletal muscle fibers is sufficient to induce a Duchenne-like muscular dystrophy phenotype. In addition, we have shown that caveolin-3 null mice display mild muscle fiber degeneration and T-tubule system abnormalities. These data are consistent with the mild phenotype observed in Limb-girdle muscular dystrophy-1C (LGMD-1C)

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Al-Sabri, Mohamed H., Neha Behare, Ahmed M. Alsehli, et al. "Statins Induce Locomotion and Muscular Phenotypes in Drosophila melanogaster That Are Reminiscent of Human Myopathy: Evidence for the Role of the Chloride Channel Inhibition in the Muscular Phenotypes." Cells 11, no. 22 (2022): 3528. http://dx.doi.org/10.3390/cells11223528.

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The underlying mechanisms for statin-induced myopathy (SIM) are still equivocal. In this study, we employ Drosophila melanogaster to dissect possible underlying mechanisms for SIM. We observe that chronic fluvastatin treatment causes reduced general locomotion activity and climbing ability. In addition, transmission microscopy of dissected skeletal muscles of fluvastatin-treated flies reveals strong myofibrillar damage, including increased sarcomere lengths and Z-line streaming, which are reminiscent of myopathy, along with fragmented mitochondria of larger sizes, most of which are round-like

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Ching Sia, Lim, Maharshi Dave, Jagdish Prasad Sharma, and Tyrus A. McCarty. "Stability Analysis of Muscular and Skeletal System of Human’s leg segments (Thigh and Shank)." International Journal of Darshan Institute on Engineering Research & Emerging Technology 9, no. 1 (2020): 1–8. http://dx.doi.org/10.32692/ijdi-eret/9.1.2020.2001.

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Schönau, Eckhard. "The Development of the Skeletal System in Children and the Influence of Muscular Strength." Hormone Research in Paediatrics 49, no. 1 (1997): 27–31. http://dx.doi.org/10.1159/000023122.

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S, Abarna Shivani, and Leoney A. "Neuromuscular Disorders And Prosthodontic Strategies In Complete Denture Patients." Journal of Clinical Prosthodontics and Implantology 5, no. 2 (2023): 41–44. http://dx.doi.org/10.55995/j-cpi.2023009.

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Neuromuscular disorders leads to loss of muscle function because they affect the musculature and/or the nervous system. Etiology could be due to autoimmune disorders, genetic/hereditary disorders. The process of mastication is controlled and coordinated by a well-defined neurological system which primarily consists of skeletal muscles, motor and sensory nerve fibers. This neuromuscular control involves in every phase of complete denture prosthesis construction. Skeletal muscles directly play a vital role in complete denture prosthesis function. A good muscular control and coordination is neces

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Lo Mauro, Antonella, and Andrea Aliverti. "Physiology of respiratory disturbances in muscular dystrophies." Breathe 12, no. 4 (2016): 318–27. http://dx.doi.org/10.1183/20734735.012716.

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Muscular dystrophy is a group of inherited myopathies characterised by progressive skeletal muscle wasting, including of the respiratory muscles. Respiratory failure,i.e. when the respiratory system fails in its gas exchange functions, is a common feature in muscular dystrophy, being the main cause of death, and it is a consequence of lung failure, pump failure or a combination of the two. The former is due to recurrent aspiration, the latter to progressive weakness of respiratory muscles and an increase in the load against which they must contract. In fact, both the resistive and elastic comp

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Hammerer-Lercher, Angelika, Petra Erlacher, Reginald Bittner, et al. "Clinical and Experimental Results on Cardiac Troponin Expression in Duchenne Muscular Dystrophy." Clinical Chemistry 47, no. 3 (2001): 451–58. http://dx.doi.org/10.1093/clinchem/47.3.451.

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Abstract Background: Because of controversial earlier studies, the purpose of this study was to provide novel experimental and additional clinical data regarding the possible reexpression of cardiac troponin T (cTnT) in regenerating skeletal muscle in Duchenne muscular dystrophy (DMD). Methods: Plasma from 14 patients (mean age, 7.5 years; range, 5.7–19.4 years) with DMD was investigated for creatine kinase (CK), the CK MB isoenzyme (CKMB), cTnT and cardiac troponin I (cTnI), and myoglobin. cTnT concentrations were measured by an ELISA (second-generation assay; Roche) using the ES 300 Analyzer

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Cifuentes-Diaz, Carmen, Tony Frugier, Francesco D. Tiziano, et al. "Deletion of Murine SMN Exon 7 Directed to Skeletal Muscle Leads to Severe Muscular Dystrophy." Journal of Cell Biology 152, no. 5 (2001): 1107–14. http://dx.doi.org/10.1083/jcb.152.5.1107.

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Spinal muscular atrophy (SMA) is characterized by degeneration of motor neurons of the spinal cord associated with muscle paralysis and caused by mutations of the survival motor neuron gene (SMN). To determine whether SMN gene defect in skeletal muscle might have a role in SMA pathogenesis, deletion of murine SMN exon 7, the most frequent mutation found in SMA, has been restricted to skeletal muscle by using the Cre-loxP system. Mutant mice display ongoing muscle necrosis with a dystrophic phenotype leading to muscle paralysis and death. The dystrophic phenotype is associated with elevated lev

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