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Books on the topic 'Skeletal tuberculosis'

Author: Grafiati

Published: 4 June 2025

Last updated: 23 June 2025

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1

Tuli, S. M. Tuberculosis of the skeletal system: Bones, joints, spine, and bursal sheaths. Jaypee Brothers Medical Publishers, 1993.

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2

Thijn, Cornelis J. P., and Jieldouw T. Steensma. Tuberculosis of the Skeleton. Springer Berlin Heidelberg, 1990. http://dx.doi.org/10.1007/978-3-642-74665-9.

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3

1942-, Steensma J. T., ed. Tuberculosis of the skeleton: Focus on radiology. Springer-Verlag, 1990.

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4

Tuli, SM. Tuberculosis of the Skeletal System. Jaypee Brothers Medical Publishers (P) Ltd., 2004. http://dx.doi.org/10.5005/jp/books/10993.

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5

Tuberculosis of the Skeletal System. Jaypee Brothers Medical Publishers, 2016.

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6

Tuli, S. M. Tuberculosis of the Skeletal System. 3rd ed. Jaypee Brothers Medical Pub, 2004.

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7

Tuberculosis of the Skeletal System Hb. Jaypee Brothers Medical Publishers, 2020.

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8

Tuli, SM. Tuberculosis of the Skeletal System , (Fourth Edition). Jaypee Brothers Medical Publishers (P) Ltd., 2010. http://dx.doi.org/10.5005/jp/books/11046.

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9

Tuli, S. M. Tuberculosis of the Skeletal System: Bones, Joints, Spine and Bursal Sheaths. Jaypee Brothers Medical Publishers, 2008.

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10

Tuli, SM. Tuberculosis of the Skeletal System (Bones, Joints, Spine and Bursal Sheaths). Jaypee Brothers Medical Publishers (P) Ltd., 2016. http://dx.doi.org/10.5005/jp/books/12726.

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11

Thijn, Cornelis J. P., and Jieldouw T. Steensma. Tuberculosis of the Skeleton. Island Press, 1989.

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12

Lewis, Mary E. Disease and Trauma in the Children from Roman Britain. Edited by Sally Crawford, Dawn M. Hadley, and Gillian Shepherd. Oxford University Press, 2018. http://dx.doi.org/10.1093/oxfordhb/9780199670697.013.25.

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Abstract:

This chapter explores our current knowledge of pathology and trauma in Romano-British non-adult samples focusing on the children from the late Roman cemetery of Poundbury Camp, Dorset. Evidence for metabolic diseases (rickets, scurvy, iron deficiency anaemia), fractures, thalassemia, congenital disorders and tuberculosis, are presented with emphasis on what their presence tells us about the impact of the Romans in Britain. Many of the large Roman sites from the UK were excavated long before diagnostic criteria for recognizing pathology in child remains were fully developed, and European studies tend only to focus on anaemia and its link to malaria. A lack of environmental evidence for the sites from which our skeletal remains are derived is also problematic, and this chapter hopes to set the agenda for future research into the health and life of children living in the Roman World.

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13

Theros, E. G., Jieldouw T. Steensma, and Cornelis J. P. Thijn. Tuberculosis of the Skeleton: Focus on Radiology. Springer London, Limited, 2012.

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14

Morasso, Pietro G., and Cornelis J. P. Thijn. Tuberculosis of the Skeleton: Focus on Radiology. Springer, 2011.

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15

Abdulkader, Rita, and Richard A. Watts. Mycobacterial diseases. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0103.

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The main diseases caused by mycobacterial infection are tuberculosis (TB) and leprosy. Despite a fall in the prevalence of these diseases over the last decade, they are still significant causes of morbidity and mortality worldwide. Atypical mycobacterial infections are encountered less frequently. Immigration patterns, the frequency of human immunodeficiency infection, and the increased numbers of patients on immunosuppressive treatments render mycobacterial infections relevant not only to physicians in the developing world where they traditionally occurred but also in the developed world. Skeletal TB occurs in 1–3% of cases of TB infection, and is more frequently encountered in the immunocompromised. A high index of suspicion is required, diagnosis relies on a combination of clinical features and radiological, histological, and microbiological tests. Multidrug regimens are required for treatment with surgery in selected cases. Leprosy is caused by M. leprae infection. The disease is still a leading cause of disability worldwide. Diagnosis is usually clinical. The course of the disease is indolent but may be interrupted by acute inflammatory reactions, which contribute to nerve damage and disability. Treatment aims at eliminating the mycobacteria using multidrug regimens, and management of complications including leprosy reactions and long-term nerve damage. Atypical mycobacterial infections affecting bone and joints are uncommon; they usually follow direct inoculation of the pathogen. Haematogenous dissemination is encountered in immunocompromised patients. These microorganisms are not usually susceptible to the same drug regimens used in the treatment of tuberculosis.

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