Log in

Relevant bibliographies by topics / Slow transit constipation

Contents

Journal articles
Dissertations / Theses
Books
Book chapters

Academic literature on the topic 'Slow transit constipation'

Author: Grafiati

Published: 4 June 2021

Last updated: 1 February 2022

Create a spot-on reference in APA, MLA, Chicago, Harvard, and other styles

Select a source type:

Consult the lists of relevant articles, books, theses, conference reports, and other scholarly sources on the topic 'Slow transit constipation.'

Next to every source in the list of references, there is an 'Add to bibliography' button. Press on it, and we will generate automatically the bibliographic reference to the chosen work in the citation style you need: APA, MLA, Harvard, Chicago, Vancouver, etc.

You can also download the full text of the academic publication as pdf and read online its abstract whenever available in the metadata.

Journal articles on the topic "Slow transit constipation"

1

Bharucha, Adil E., and Sidney F. Phillips. "SLOW TRANSIT CONSTIPATION." Gastroenterology Clinics of North America 30, no. 1 (2001): 77–96. http://dx.doi.org/10.1016/s0889-8553(05)70168-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Bharucha, Adil E., and Sidney F. Philips. "Slow-transit constipation." Current Treatment Options in Gastroenterology 4, no. 4 (2001): 309–15. http://dx.doi.org/10.1007/s11938-001-0056-9.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Wald, Arnold. "Slow transit constipation." Current Treatment Options in Gastroenterology 5, no. 4 (2002): 279–83. http://dx.doi.org/10.1007/s11938-002-0050-x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Altomare, Donato F., Piero Portincasa, Marcella Rinaldi, et al. "Slow-transit constipation." Diseases of the Colon & Rectum 42, no. 2 (1999): 231–40. http://dx.doi.org/10.1007/bf02237134.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

Roe, A. M., D. C. C. Bartolo, and N. J. M. Mortensen. "Slow transit constipation." Digestive Diseases and Sciences 33, no. 9 (1988): 1159–63. http://dx.doi.org/10.1007/bf01535794.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Macdonald, A., J. N. Baxter, and I. G. Finlay. "Idiopathic slow-transit constipation." British Journal of Surgery 80, no. 9 (1993): 1107–11. http://dx.doi.org/10.1002/bjs.1800800909.

Full text

APA, Harvard, Vancouver, ISO, and other styles

7

Wheatley, J. M., J. M. Hutson, C. W. Chow, M. Oliver, and M. R. Hurley. "Slow-transit constipation in childhood." Journal of Pediatric Surgery 34, no. 5 (1999): 829–33. http://dx.doi.org/10.1016/s0022-3468(99)90381-0.

Full text

APA, Harvard, Vancouver, ISO, and other styles

8

Hutson, JM, J. McNamara, S. Gibb, and Y.-M. Shin. "Slow transit constipation in children." Journal of Paediatrics and Child Health 37, no. 5 (2001): 426–30. http://dx.doi.org/10.1046/j.1440-1754.2001.00692.x.

Full text

APA, Harvard, Vancouver, ISO, and other styles

9

Tillou, John, and Vitaliy Poylin. "Functional Disorders: Slow-Transit Constipation." Clinics in Colon and Rectal Surgery 30, no. 01 (2016): 076–86. http://dx.doi.org/10.1055/s-0036-1593436.

Full text

APA, Harvard, Vancouver, ISO, and other styles

10

Mollen, Roland M., Han C. Kuijpers, and Ad T. Claassen. "Colectomy for slow-transit constipation." Diseases of the Colon & Rectum 44, no. 4 (2001): 577–80. http://dx.doi.org/10.1007/bf02234332.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Dissertations / Theses on the topic "Slow transit constipation"

1

Knowles, Charles H. "Slow transit constipation : clinical and aetiological studies." Thesis, Queen Mary, University of London, 2000. http://qmro.qmul.ac.uk/xmlui/handle/123456789/1515.

Full text

Abstract:

Constipation is the second most commonly self-reported gastrointestinal symptom. On the basis of anorectal physiological investigations and colonic transit studies, a subgroup of patients with several intractable symptoms, but without organic disease will be found to have slow transit constipation (STC). STC is a condition of gut dysmotility which predominantly affects young women, and may result in surgical intervention with variable, often unsatisfactory results. The aetiology remains elusive. New aetiological hypotheses for STC were examined following full clinical and pathophysiological characterisation of a large cohort of 130 patients referred to our institution over the last 10 years. Aspects of nerve and muscle dysfunction were studied. A new scoring system demonstrated some ability of multiple symptoms to discriminate STC from other forms of constipation. Detailed clinical and gastrointestinal physiological studies confirmed the heterogeneity of STC patients. Some significant physiological differences were detectable between clinically defined sub-groups of patients and refuted previous assumptions based on smaller numbers. Detailed neurophysiological studies, including quantitative peripheral sensory and autonomic testing, provided evidence of a small fibre neuropathy in a proportion of patients with STC. Mutational screening of some early-onset cases for a possible congenital pathogenetic mechanism, based on the observation that some STC patients had relatives with Hirschsprung's disease demonstrated that mutation of 2 important genes now implicated in this disorder were not a frequent cause of STC. Serum immunoprecipitation assays showed that anti-neuronal ion channel autoantibodies may have an as yet unrecognised role in the development of STC in a small proportion of acquired cases. An inclusion body myopathy was identifiable in colonic tissue of patients with STC, and this appeared to arise secondary to denervation. Further knowledge of the single or multiple pathogenetic mechanisms leading to this clinical condition may allow more rational or directed therapies aimed at the correction of the disease process or processes themselves.

APA, Harvard, Vancouver, ISO, and other styles

2

Lundin, Erik. "Slow Transit Constipation : Aspects of Diagnosis and Treatment." Doctoral thesis, Uppsala : Acta Universitatis Upsaliensis : Univ.-bibl. [distributör], 2005. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-5770.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Preston, David Michael. "The chronic constipation of young women." Thesis, King's College London (University of London), 1985. https://kclpure.kcl.ac.uk/portal/en/theses/the-chronic-constipation-of-young-women(72636ff6-ebe9-4aee-b0bf-db57a4b22a5d).html.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

MacDonald, Angus. "Aspects of colonic motility in idiopathic slow transit constipation." Thesis, University of Glasgow, 1995. http://theses.gla.ac.uk/1906/.

Full text

Abstract:

This thesis sets out to examine the hypothesis that some patients with idiopathic constipation, notably those who develop their symptoms following childbirth or hysterectomy, have regional as opposed to total colonic dysmotility. Such a group may be amenable to segmental rather than total colonic resection. Several clinical studies are presented which establish postchildbirth/hysterectomy constipation as a distinct subgroup of idiopathic constipation. Studies of gastric emptying demonstrate that patients with postchildbirth/hysterectomy constipation have normal motility in the proximal gastrointestinal tract. In contrast, patients with idiopathic constipation have prolonged gastric emptying indicating that proximal GI dysmotility may form a significant component of the presenting symptoms. Having identified that the proximal GI tract appears normal in patients with postchildbirth/hysterectomy constipation the next task was to identify in which region of the colon the dysmotility was most severe. Segmental colonic transit studies, using radio-opaque markers, identify delayed transit in the left colon, while dynamic radio-isotope studies localise the area of abnormality to the sigmoid colon. Colonic manometry studies, using a water-perfusion catheter point to a region of hindgut dysmotility which manifests as an excess of low pressure waves at rest and a specific failure to generate high pressure propagative waves. The usefulness of prostigmine provocation testing is examined critically in this group of patients and the pitfalls of this technique are presented.

APA, Harvard, Vancouver, ISO, and other styles

5

Mohammed, Sahar D. Mohammed. "Colonic motility in health and in slow transit constipation." Thesis, Queen Mary, University of London, 2017. http://qmro.qmul.ac.uk/xmlui/handle/123456789/24737.

Full text

Abstract:

Introduction: Our knowledge of normal human colonic motility remains incomplete. Historically, this has been due to the relative inaccessibility of this organ for study, and the lack of standardisation of methods used to investigate it. Recent device development has provided us with advanced tools by which to assess colonic motility, namely pancolonic manometry, and the wireless motility capsule (WMC). Using traditional diagnostic tests, a subgroup of patients presenting with severe intractable symptoms, but without organic disease, are found to have slow transit constipation (STC). This is believed to be primarily due to colonic dysmotility, although colonic motor functions remain poorly understood in this group also. Aims: The principal aims of this thesis were to: (1) explore the effect of pancolonic manometric recording technique on colonic motility; (2) describe pancolonic motility in STC, compared to healthy control subjects; (3) using the wireless motility capsule (WMC), validate the precise location of the pH fall around the ileo-caecal junction as a landmark for measuring colonic motility; (4) obtain normative data for colonic motility (transit and contractility) and intraluminal pH in a large cohort of healthy volunteers using the WMC, and compare this to patients with STC. Methods: The following methods were used: (1) prolonged pancolonic manometry in healthy volunteers and patients with STC; (2) a dual scintigraphic technique, involving radioactive-labelling of the WMC in healthy volunteers; (3) wireless motility capsule studies of colonic motility in healthy volunteers and in patients with STC. Results: Colonic manometric recording technique (bowel preparation or not, and different catheter types) significantly influences some characteristics of propagating sequence (PS) activity, including frequency, amplitude, polarity, relationship between consecutive PSs, and circadian rhythm. Patients with STC display dysregulated colonic motor function represented by disorganised spatiotemporal patterning and loss of 'regional linkage' among PSs. The fall in pH measured by the WMC was confirmed to be either in the caecum, ascending colon, or as the capsule moved from the caecum to the ascending colon. Using the WMC, the upper limit of normal colonic transit time (CTT) was found to be 51 h; however, CTT is not a continuous variable and exhibits peaks every 24 h. CTT is significantly prolonged in females and affected by the study protocol employed. In patients with STC, colonic contractility (motility index) is increased in comparison with healthy controls, and intraluminal pH is more acidic in the proximal colon, and more alkaline in the distal colon. Conclusions: The method of pancolonic manometry requires standardisation. However, novel metrics derived from prolonged pancolonic recordings have improved our understanding of the physiology of colonic motor function in health, and also pathophysiology in constipation. The WMC provides an alternative, less invasive method to investigate colonic motility; this technique also requires standardisation, but early results in patients with STC complement those from manometry, and also reveal alterations in intraluminal pH that may be of pathophysiological significance.

APA, Harvard, Vancouver, ISO, and other styles

6

Sutcliffe, Jonathan Richard. "Interstitial cell of Cajal Abnormalities in children with Slow Transit Constipation." Thesis, University of Leeds, 2009. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.536116.

Full text

APA, Harvard, Vancouver, ISO, and other styles

Books on the topic "Slow transit constipation"

1

Keshav, Satish, and Alexandra Kent. Constipation. Edited by Patrick Davey and David Sprigings. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199568741.003.0027.

Full text

Abstract:

Patients and doctors often define constipation differently. The normal frequency of defaecation is once every 3 days to three times per day, and constipation may be defined as abnormally infrequent defaecation. A change in the normal pattern and frequency for the particular patient is pertinent. There are numerous causes of constipation, and most can be encountered in both primary and secondary care. In patients with chronic constipation without an evident cause, irritable bowel syndrome (IBS) is the cause in 59%, pelvic floor dysfunction in 25%, slow transit in 13%, and a combination of pelvic floor dysfunction and slow transit in 3%. Constipation affects twice as many women as men, with a higher prevalence in pregnant women. Prevalence is also greater in the elderly, affecting ~20% in the community.

APA, Harvard, Vancouver, ISO, and other styles

2

Hakim, Alan J., and Rodney Grahame. Hypermobility syndromes. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0159.

Full text

Abstract:

Hypermobility-related syndromes constitute a family of heritable disorders of connective tissue (HDCT) that derive from abnormalities affecting genes that encode for the connective tissue matrix proteins such as collagen, fibrillin, and tenascin. They range from such commonplace though poorly recognized conditions such as the joint hypermobility syndrome (JHS) to the better-known, if more rare, eponymous syndromes such as Marfan's syndrome (MFS) and the different types of the Ehlers-Danlos syndrome (EDS). The more common presentations are with skin pathology (bruising, scaring), joint or spinal and/or muscle pain and instability with vulnerability to injury and chronic widespread pain, cardiac valve pathologies, and in MFS and vascular EDS, arterial dilatation with the risk of dissection and rupture. JHS (widely considered synonymous with the EDS hypermobility type) is further complicated by cardiovascular autonomic dysfunction such as orthostatic intolerance, palpitations, and syncope, and the recently described and commonly encountered pangastrointestinal dysmotility. The latter can manifest as gastro-oesophageal reflux, gastroparesis, slow-transit constipation, or rectal evacuatory dysfunction with rectal intussusception. In addition, HDCT are associated with bladder and uterine problems as a consequence of pelvic floor weakness. Such multisystemic conditions need to be managed by a multidisciplinary team able to draw on medical, surgical, physical, and psychological interventions by appropriately experienced specialists and therapists. This chapter introduces the reader to the epidemiology, genetics, classification, and clinical presentation of JHS, EDS, and MFS. It also describes the key investigations required to support a diagnosis and assess complications of an HDCT, as well as the multidisciplinary approach to management.

APA, Harvard, Vancouver, ISO, and other styles

Book chapters on the topic "Slow transit constipation"

1

Leung, Alexander K. C., Cham Pion Kao, Andrew L. Wong, et al. "Slow-Transit Constipation." In Encyclopedia of Molecular Mechanisms of Disease. Springer Berlin Heidelberg, 2009. http://dx.doi.org/10.1007/978-3-540-29676-8_8214.

Full text

APA, Harvard, Vancouver, ISO, and other styles

2

Kessler, H., B. L. Jerby, J. W. Milsom, and W. Hohenberger. "Colektomie bei chronischer Obstipation: Vergleich laparoskopischen und konventionellen Vorgehens / Total Colectomy for Slow Transit Constipation: A Comparison of Laparoscopic and Open Procedures." In Deutsche Gesellschaft für Chirurgie. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-56458-1_65.

Full text

APA, Harvard, Vancouver, ISO, and other styles

3

Hedayat, Kamyar M., Jean-Claude Lapraz, and Ben Schuff. "Constipation, slow transit." In The Theory of Endobiogeny. Elsevier, 2020. http://dx.doi.org/10.1016/b978-0-12-816965-0.00022-6.

Full text

APA, Harvard, Vancouver, ISO, and other styles

4

Wijffels, Niels. "Slow transit constipation." In Pelvic Floor Disorders for the Colorectal Surgeon. Oxford University Press, 2010. http://dx.doi.org/10.1093/med/9780199579624.003.0013.

Full text

APA, Harvard, Vancouver, ISO, and other styles

5

"30 Operative Therapie der Darmträgheit („slow transit constipation“)." In Management der gastrointestinalen und kolorektalen Motilitätsstörungen. De Gruyter, 2020. http://dx.doi.org/10.1515/9783110643633-030.

Full text

APA, Harvard, Vancouver, ISO, and other styles

6

Hakim, Alan J., and Rodney Grahame. "Hypermobility syndromes." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0159_update_003.

Full text

Abstract:

Hypermobility-related syndromes constitute a family of heritable disorders of connective tissue (HDCT) that derive from abnormalities affecting genes that encode for the connective tissue matrix proteins such as collagen, fibrillin, and tenascin. They range from such commonplace though poorly recognized conditions such as the joint hypermobility syndrome (JHS) to the better-known, if rarer, eponymous syndromes such as the Marfan syndrome (MFS) and the different types of the Ehlers-Danlos syndrome (EDS). The more common presentations are with skin pathology (bruising, scaring), joint or spinal and/or muscle pain and instability with vulnerability to injury and chronic widespread pain, cardiac valve pathologies, and in MFS and vascular EDS, arterial dilatation with the risk of dissection and rupture. JHS (widely considered synonymous with the EDS hypermobility type) is further complicated by cardiovascular autonomic dysfunction such as orthostatic intolerance, palpitations, and syncope, and the recently described and commonly encountered pan-gastrointestinal dysmotility. The latter can manifest as gastro-oesophageal reflux, gastroparesis, slow-transit constipation, or rectal evacuatory dysfunction with rectal intussusception. In addition, HDCT are associated with bladder and uterine problems as a consequence of pelvic floor weakness. Such multisystemic conditions need to be managed by a multidisciplinary team able to draw on medical, surgical, physical, and psychological interventions by appropriately experienced specialists and therapists. This chapter introduces the reader to the epidemiology, genetics, classification, and clinical presentation of JHS, EDS, and MFS. It also describes the key investigations required to support a diagnosis and assess complications of an HDCT, as well as the multidisciplinary approach to management.

APA, Harvard, Vancouver, ISO, and other styles

7

Hakim, Alan J., and Rodney Grahame. "Hypermobility syndromes." In Oxford Textbook of Rheumatology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0159_update_004.

Full text

Abstract:

Hypermobility-related syndromes constitute a family of heritable disorders of connective tissue (HDCT) that derive from abnormalities affecting genes that encode for the connective tissue matrix proteins such as collagen, fibrillin, and tenascin. They range from such commonplace though poorly recognized conditions such as hypermobility spectrum disorder (HSD), formerly within the joint hypermobility syndrome (JHS), to the better-known, if rarer, eponymous syndromes such as the Marfan syndrome (MFS), Loeys–Dietz syndrome, and the different types of the Ehlers-Danlos syndrome (EDS). The more common presentations are with skin pathology (bruising, scaring), joint or spinal and/or muscle pain and instability with vulnerability to injury and chronic widespread pain, cardiac valve pathologies, and in MFS and vascular EDS, in particular, arterial dilatation with the risk of dissection and rupture. The hypermobile variant of EDS and HSD are further complicated by cardiovascular autonomic dysfunction such as orthostatic intolerance, palpitations, and syncope, and the recently described and commonly encountered pan-gastrointestinal dysmotility. The latter can manifest as gastro-oesophageal reflux, gastroparesis, slow-transit constipation, or rectal evacuatory dysfunction with rectal intussusception. In addition, HDCT are associated with bladder and uterine problems as a consequence of pelvic floor weakness. Such multisystemic conditions need to be managed by a multidisciplinary team able to draw on medical, surgical, physical, and psychological interventions by appropriately experienced specialists and therapists. This chapter introduces the reader to the epidemiology, genetics, classification, and clinical presentation of HSD, EDS, and MFS. It also describes the key investigations required to support a diagnosis and assess complications of an HDCT, as well as the multidisciplinary approach to management.

APA, Harvard, Vancouver, ISO, and other styles

We offer discounts on all premium plans for authors whose works are included in thematic literature selections. Contact us to get a unique promo code!