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Academic literature on the topic 'Snow-White syndrome'

Author: Grafiati
Published: 4 June 2021
Last updated: 16 February 2022

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Journal articles on the topic "Snow-White syndrome"

1

Puledda, Francesca, Christoph Schankin, and Peter J. Goadsby. "Visual snow syndrome." Neurology 94, no. 6 (January 15, 2020): e564-e574. http://dx.doi.org/10.1212/wnl.0000000000008909.

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ObjectiveTo validate the current criteria of visual snow and to describe its common phenotype using a substantial clinical database.MethodsWe performed a web-based survey of patients with self-assessed visual snow (n = 1,104), with either the complete visual snow syndrome (n = 1,061) or visual snow without the syndrome (n = 43). We also describe a population of patients (n = 70) with possible hallucinogen persisting perception disorder who presented clinically with visual snow syndrome.ResultsThe visual snow population had an average age of 29 years and had no sex prevalence. The disorder usually started in early life, and ≈40% of patients had symptoms for as long as they could remember. The most commonly experienced static was black and white. Floaters, afterimages, and photophobia were the most reported additional visual symptoms. A latent class analysis showed that visual snow does not present with specific clinical endophenotypes. Severity can be classified by the amount of visual symptoms experienced. Migraine and tinnitus had a very high prevalence and were independently associated with a more severe presentation of the syndrome.ConclusionsClinical characteristics of visual snow did not differ from the previous cohort in the literature, supporting validity of the current criteria. Visual snow likely represents a clinical continuum, with different degrees of severity. On the severe end of the spectrum, it is more likely to present with its common comorbid conditions, migraine and tinnitus. Visual snow does not depend on the effect of psychotropic substances on the brain.
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2

Puledda, Francesca, Muriel Bruchhage, Owen O'Daly, Dominic Ffytche, Steven C. R. Williams, and Peter J. Goadsby. "Occipital cortex and cerebellum gray matter changes in visual snow syndrome." Neurology 95, no. 13 (August 5, 2020): e1792-e1799. http://dx.doi.org/10.1212/wnl.0000000000010530.

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ObjectiveTo determine whether regional gray and white matter differences characterize the brain of patients with visual snow syndrome, a newly defined neurologic condition, we used a voxel-based morphometry approach.MethodsIn order to investigate whole brain morphology directly, we performed an MRI study on patients with visual snow syndrome (n = 24) and on age- and sex-matched healthy volunteers (n = 24). Voxel-based morphometry was used to determine volumetric differences in patients with visual snow. We further analyzed cerebellar anatomy directly using the high-resolution spatially unbiased atlas template of the cerebellum.ResultsCompared to healthy controls, patients with visual snow syndrome had increased gray matter volume in the left primary and secondary visual cortices, the left visual motion area V5, and the left cerebellar crus I/lobule VI area. These anatomical alterations could not be explained by clinical features of the condition.ConclusionPatients with visual snow syndrome have subtle, significant neuroanatomical differences in key visual and lateral cerebellar areas, which may in part explain the pathophysiologic basis of the disorder.
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3

Daly, Christina M. S., Jason Willer, Ronald Gregg, and Jeffrey M. Gross. "snow white, a Zebrafish Model of Hermansky-Pudlak Syndrome Type 5." Genetics 195, no. 2 (July 26, 2013): 481–94. http://dx.doi.org/10.1534/genetics.113.154898.

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4

Gupta, Bhawani. "Amelogenin x linked chromosome." International Journal of Research in Medical Sciences 5, no. 10 (September 28, 2017): 4214. http://dx.doi.org/10.18203/2320-6012.ijrms20174549.

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The AMELX gene provides instructions for making a protein called amelogenin, which is essential for normal tooth development. Amelogenin is involved in the formation of enamel, which is the hard, white material that forms the protective outer layer of each tooth. Using molecular genetic techniques, we have shown that there is no evidence that the AMGX gene is deleted in this case of the Nance-Horan syndrome. In affected members of a Michigan kindred of Eastern European ancestry segregating X-linked amelogenesis imperfecta with a characteristic snow-capped enamel phenotype.
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5

Michels, Lars, Philipp Stämpfli, Njoud Aldusary, Marco Piccirelli, Patrick Freund, Konrad P. Weber, Fabienne C. Fierz, Spyros Kollias, and Ghislaine Traber. "Widespread White Matter Alterations in Patients With Visual Snow Syndrome." Frontiers in Neurology 12 (September 21, 2021). http://dx.doi.org/10.3389/fneur.2021.723805.

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Background: Visual snow is considered a disorder of central visual processing resulting in a perturbed perception of constant binocular flickering or pixilation of the whole visual field. The underlying neurophysiological and structural alterations remain elusive.Methods: In this study, we included patients (final n = 14, five dropouts; five females, mean age: 32 years) with visual snow syndrome (VSS) and age- and sex-matched controls (final n = 20, 6 dropouts, 13 females, mean age: 28.2 years). We applied diffusion tensor imaging to examine possible white matter (WM) alterations in patients with VSS.Results: The patient group demonstrated higher (p-corrected < 0.05, adjusted for age and sex) fractional anisotropy (FA) and lower mean diffusivity (MD) and radial diffusivity (RD) compared to controls. These changes were seen in the prefrontal WM (including the inferior fronto-occipital fascicle), temporal and occipital WM, superior and middle longitudinal fascicle, and sagittal stratum. When additionally corrected for migraine or tinnitus—dominant comorbidities in VSS—similar group differences were seen for FA and RD, but less pronounced.Conclusions: Our results indicate that patients with VSS present WM alterations in parts of the visual cortex and outside the visual cortex. As parts of the inferior fronto-occipital fascicle and sagittal stratum are associated with visual processing and visual conceptualisation, our results suggest that the WM alterations in these regions may indicate atypical visual processing in patients with VSS. Yet, the frequent presence of migraine and other comorbidities such as tinnitus in VSS makes it difficult to attribute WM disruptions solely to VSS.
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6

Hollis, Leah. "The Procrustes Figure, the Curmudgeon, and the Snow White Syndrome Styles of Higher Education Workplace Bullying." SSRN Electronic Journal, 2021. http://dx.doi.org/10.2139/ssrn.3884494.

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7

Latini, Francesco, Markus Fahlström, Niklas Marklund, and Amalia Feresiadou. "White matter abnormalities in a patient with visual snow syndrome: New evidence from a diffusion tensor imaging study." European Journal of Neurology, May 28, 2021. http://dx.doi.org/10.1111/ene.14903.

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8

Islam, Mahibul, Mahmudul Hasan, Bhaskar Chandra Majudmar, and Sulav Indra Paul. "The Ecotoxicological Effects of Microplastics on Primary Producers in the Marine Environment." Journal of Fisheries Science 2, no. 2 (July 31, 2020). http://dx.doi.org/10.30564/jfsr.v2i2.1906.

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Plastic debris is an emerging environmental threat all over the world. But its effect and distribution in the marine ecosystem is barely known. Microplastics abundance in the marine vegetated area is about 2 to 3 times higher than the bare site in the ocean. Although seagrass meadows trap huge amount of microplastics over the ocean floor, a considerable amount of microplastics are also sink incorporating with the marine aggregates from the epipelagic zone of the ocean. Scavenging of microplastics by diatom aggregation decreases the sinking rate of them rather than cryptophyte. As we know, marine snow is the leading carbon source for zoobenthos, but the ubiquitous presence of microplastics damages cell of different microalgae which may alter the food webs of marine ecosystems. Additionally, microplastics releases immense amount of dissolved organic carbons (DOC) in the surrounding seawater that stimulates the growth of heterotrophic microorganisms as well as their functional activity. Plastic debris result in outbreaks of disease in the marine environment and coral reefs are highly affected by it. When coral reef comes in contact with microplastics, the disease infestation rate of the reef increases massively. Three major disease viz., skeletal eroding band, white syndrome and black band of coral reef causes approximately 46% of reef mortality due to microplastics consumption. Due to complex structure and size, the corals accumulates huge amount of microplastics that increases growth of pathogens by hampering the coral immune system. Existing scientific evidence presents that exposure of microplastics in aquatic environments triggers a wide variety of toxic insult from feeding disruption to reproductive performance, disturbances in energy metabolism throughout the ocean. The present review focused on the ecotoxicological effect of microplastics on primary producers of ocean, its uptake, accumulation, and excretion, and its probable toxicity with risk assessment approaches.
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Books on the topic "Snow-White syndrome"

1

The Snow White syndrome: All about envy. New York: Macmillan, 1986.

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2

Snow White Syndrome. Jove, 1989.

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3

Cohen, Betsy. The Snow White Syndrome: All About Envy. Macmillan Pub Co, 1987.

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Cohen, Betsy. The Snow White Syndrome: All About Envy. Macmillan Pub Co, 1987.

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5

Textual Analysis of a Recovered Memory Trial, Assisted by Computer Search for Keywords. Uppsala University, 2009.

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Book chapters on the topic "Snow-White syndrome"

1

Hollis, Leah P. "The Procrustes Figure, the Curmudgeon, and the Snow White Syndrome." In Human Resource Perspectives on Workplace Bullying in Higher Education, 93–109. New York, NY : Routledge, 2021. |: Routledge, 2021. http://dx.doi.org/10.4324/9781003051923-9.

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