Academic literature on the topic 'Society of St. Vincent de Paul. United States'

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Journal articles on the topic "Society of St. Vincent de Paul. United States"

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Austin, Roy L. "Family Environment, Educational Aspiration and Performance in St. Vincent." Review of Black Political Economy 17, no. 3 (January 1989): 101–22. http://dx.doi.org/10.1007/bf02901104.

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Father-absence occurs with unusual frequency among people of African descent in the Caribbean. Yet concern over possibly harmful effects of this condition to children and society which is most obvious in the United States is not informed by scientific findings from this region. The present study yielded no evidence that father-absence retards the aspiration or performance of secondary school students in St. Vincent, West Indies, although twelve different groupings of the available cases were analyzed. Findings from this and some American studies suggest that father-absence is not harmful if it is not strongly condemned by the culture with which youths identify.
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2

Ambrey, Christopher L., Cameron Parsell, Melanie Spallek, and Richard N. S. Robinson. "An Investigation Into Repeat Requests for Charity: Evidence From the St Vincent de Paul Society Queensland, Australia." Nonprofit and Voluntary Sector Quarterly 48, no. 1 (August 22, 2018): 91–107. http://dx.doi.org/10.1177/0899764018794300.

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In advanced industrialized economies, charitable organizations work alongside formal social services provided through welfare states to assist people living in poverty. The work of charities with socially and economically marginalized people, however, often takes place in the absence of robust evidence about what impact charity has on people’s lives. This study draws on a large administrative database to investigate what determines repeat requests for charity and how people may achieve dignity. Our findings show that frequent residential address changes seem to make people more reliant on charity, whereas the more time spent with people receiving charity significantly decreases repeat requests for charity. We propose that the provision of charity can be an opportunity to promote connectedness.
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3

Holcomb, G. E. "First Report of Petunia Blight Caused by Choanephora cucurbitarum in the United States." Plant Disease 87, no. 6 (June 2003): 751. http://dx.doi.org/10.1094/pdis.2003.87.6.751c.

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A blight (wet rot) of petunia (Petunia ×hybrida Hort. Vilm.-Andr.) was observed in a wholesale propagation nursery in Baton Rouge, LA in September 2002. The grower reported that plants wilted and then completely rotted. The disease occurred during a period of hot, humid, and cloudy weather. Approximately 100 flats of flowering-age plants of cvs. Rose and White Madness were destroyed. No fungal sporulation was noticed on dead plants, but occasional strands of white mycelium were observed. The grower's use of azoxystrobin, iprodione, and thiophanate methyl plus mancozeb fungicides during current and past outbreaks of this disease did not prevent disease spread, but disease activity stopped after temperature and humidity dropped in early October. A fungus that produced white aerial mycelia that later developed light yellow areas and also black aerial spore masses was consistently isolated from diseased tissue placed on acidified potato dextrose agar (APDA). The fungus was identified as Choanephora cucurbitarum (Berk. & Ravenel) Thaxt. on the basis of cultural and morphological characteristics (3). Sporangiola were ellipsoid, pale brown to reddish brown with distinct longitudinal striations and measured 15 to 20 × 9 to 14 μm. Sporangiospores were broadly ellipsoid, pale brown to reddish brown, indistinctly striate with fine, hyaline polar appendages, and measured 16 to 34 × 7 to 12 μm. Spore measurements were within the range previously given for C.cucurbitarum (3). Pathogenicity tests were performed by misting a mixture of sporangiola and sporangiospores (25,000 to 70,000 per ml of water taken from 7- to 10-day-old cultures grown on APDA) on flowering-age petunia plants (cvs. Rose Madness, White Madness, and Dreams Pink). Tests were repeated twice. Inoculated plants and uninoculated control plants (2 to 4 of each treatment in each test) were held in a dew chamber at 28°C for 48 h and then moved to a greenhouse. Within 48 h after inoculation, plants developed water-soaked lesions on flowers, leaves, and stems, then wilted and rotted. Uninoculated plants remained disease free except for several that developed disease symptoms in the first test, apparently from the presence of natural inoculum on healthy-appearing plants that were obtained from the nursery where the disease was found. Koch's postulates were completed by reisolation of the pathogen from diseased inoculated plants. C. cucurbitarum (1) and C. infundibulifera (Curr.) Sacc. (2) have been reported to cause flower blight of petunia in the United States and whole plant blight (wet rot) of petunia in Japan (4). To our knowledge, this is the first report of C. cucurbitarum causing whole plant blight of petunia in the United States. References: (1) M. L. Daughtrey et al. Choanephora wet rot of poinsettia. Page 15 in: Compendium of Flowering Potted Plant Diseases. The American Phytopathological Society, St. Paul, MN, 1995. (2) D. F. Farr et al. Fungi on Plants and Plant Products in the United States. The American Phytopathological Society, St. Paul, MN, 1989. (3) P. M. Kirk. Mycological Paper 152:1, 1984. (4) J. Takeuchi and H. Horie. Jpn. J. Phytopathol. 66:72, 2000.
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Tsahouridou, P. C., and C. C. Thanassoulopoulos. "First Report of Hendersonula toruloidea as a Foliar Pathogen of Strawberry-tree (Arbutus unedo) in Europe." Plant Disease 84, no. 4 (April 2000): 487. http://dx.doi.org/10.1094/pdis.2000.84.4.487c.

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During spring 1997 and 1998 in the area of Chalkidiki, in northern Greece, leaves of wild strawberry-tree (Arbutus unedo) were heavily spotted. Small, necrotic brown spots with light gray centers appeared on leaves, and when intense spotting was present, strong defoliation was observed. Isolations from leaves on potato dextrose agar consistently yielded a fungus that was identified as Hendersonula toruloidea (2). Pathogenicity tests on wild strawberry-tree plants were performed, yielding symptoms identical to those originally observed, and H. toruloidea was isolated consistently from inoculated leaves. No cankers appeared on the twigs of the plants, which is a consistent symptom caused by this fungus on strawberry-tree in the United States. Leaf spotting caused by H. toruloidea has been observed in Musa and Rhus spp. (1). This is the first report of H. toruloidea causing leaf spotting and defoliation of strawberry-trees in Europe. References: (1) D. F. Farr et al. 1989. Fungi on Plants and Plant Products in the United States. The American Phytopathological Society, St. Paul, MN. (2) R. M. Nattrass. Br. Mycol. Soc. Trans. 18:189, 1945.
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Seebold, K. W., D. B. Langston, R. C. Kemerait, and J. E. Hudgins. "First Report of a Leaf Spot and Stem Canker Caused by Myrothecium roridum on Watermelon in the United States." Plant Disease 89, no. 3 (March 2005): 342. http://dx.doi.org/10.1094/pd-89-0342a.

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Myrothecium roridum Tode:Fr, pathogenic to a number of cucurbit species, causes fruit rots, cankers on crowns and stems, and leaf spots. Hosts include cantaloupe and honeydew (Cucurbita melo) and cucumber (Cucumis sativus) (1,3). In June 2004, following a period of heavy rainfall, numerous round-to-oblong, brown lesions with concentric rings were observed on leaves of watermelon (Citrullus lanatus) cv. Desert King at the Blackshank Farm in Tifton, GA. Disease was localized in the field and severity was low (<5% of leaf area affected). No symptoms were observed on fruit. Sections of tissue were removed from the margin between healthy and diseased tissue and plated on acidified, 25% potato dextrose agar (aPDA). A small plug of agar and mycelium were removed from colonies that emerged from lesions and were transferred to aPDA. Isolated colonies were characterized by a white, floccose mycelium with concentric, dark green-to-black rings of sporodochia bearing viscid masses of conidia. Conidia were cylindrical with rounded ends and measured 6 to 8 × 1.5 to 2.5 μm. The features of the fungus were consistent with the description of Myrothecium roridum (1,2). Pathogenicity tests were conducted in a temperature-controlled greenhouse. Twenty-five watermelon plants (cv. Desert King) were inoculated with a conidial suspension of M. roridum (5 × 105 conidia per ml) plus 0.1% vol/vol Tween 20. Inoculum was applied on leaves and stems until runoff with a hand-held mister, and plants were placed in a dew chamber for 72 h. Ten plants were sprayed with sterile, distilled water to serve as controls. Inoculated and noninoculated control plants were removed from the dew chamber and maintained at 25 to 28°C. Symptoms appeared 8 days after inoculation and were characterized by round, dark lesions with concentric rings; noninoculated plants were symptomless. Sections of symptomatic tissue were plated, and M. roridum was reisolated. Although M. roridum is a common pathogen of melons and cucumber, to our knowledge, this is the first field report of a leaf spot caused by M. roridum on watermelon in the United States. No further occurrences of the disease on watermelon have been observed in Georgia since the initial discovery of M. roridum in 2004; however, losses could be potentially severe if widespread infection of fruit were to occur. References: (1) B. D. Bruton. Crater Rot. Pages 49–50 in: Compendium of Cucurbit Diseases. T. A. Zitter et al., eds. The American Phytopathological Society, St. Paul, MN, 1996. (2) M. B. Ellis. Page 552 in: Dematiaceous Hyphomycetes. CAB International, Wallingford, UK, 1971. (3) D. F. Farr et al. Page 809 in: Fungi on Plants and Plant Products in the United States. The American Phytopathological Society, St. Paul, MN, 1989.
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6

Hernández, J. R., M. E. Palm Hernández, and T. Tidwell. "First Report of Puccinia vincae on Vinca spp. in California." Plant Disease 86, no. 1 (January 2002): 75. http://dx.doi.org/10.1094/pdis.2002.86.1.75b.

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In 2000, chlorotic leaves with inconspicuous leaf spots were observed on several Vinca major L. and V. minor L. plants in a 0.8-ha area in Woodside, CA. In August 2001, 80 to 90% of the plants were symptomatic. Uredinia measuring 2 to 3 × 1 mm were present on the lower surface of leaves. Urediniospores were ellipsoidal to oblong, 27 to 36 × 17 to 27 μm, with cinnamon-brown walls 1 to 2 μm thick, echinulate, and with three or four equatorial or slightly supraequatorial germ pores. Telia were produced in the uredinia. Teliospores were 1-septate, ellipsoidal to clavate, slightly constricted at the septum, and 34 to 45 × 19 to 30 μm. Teliospore walls were chestnut brown, 1.5 to 2.5 μm thick, and verrucose, with the verrucae sometimes in longitudinal lines. One germ pore covered by a hyaline papilla was present in each cell, at the apex in the upper cell and adjacent to the short, hyaline pedicel in the lower cell. The rust was identified as Puccinia vincae Berk. (1). P. vincae is widespread in Europe on Vinca species and is common on V. major in the eastern United States, Washington, and Idaho (2). To our knowledge, this is the first report of P. vincae on V. major in California (vouchers BPI 841363, 841364) and on V. minor in the United States (voucher BPI 841365). References: (1) J. C. Arthur. Page 324 in: Manual of Rusts in the United States and Canada. Purdue Research Foundation, Lafayette, IN, 1934. (2) D. F. Farr et al. Pages 35 and 916 in: Fungi on Plants and Plant Products in the United States. American Phytopathological Society, St. Paul, MN, 1989.
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Newcombe, G. "Southerly Extension of Poplar Leaf Blight (Linospora tetraspora) in the Pacific Northwest." Plant Disease 82, no. 5 (May 1998): 590. http://dx.doi.org/10.1094/pdis.1998.82.5.590b.

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Poplar leaf blight caused by Linospora tetraspora G. E. Thompson (Ascomycetes, Valsaceae) is widespread on Populus balsamifera in Canada from Quebec to British Columbia (1). The only United States records of this northerly fungus are from Vermont, Wisconsin, and Alaska (1,2). There are no records of this fungus on the Pacific Coast south of British Columbia, despite the presence of susceptible hosts (i.e., Populus trichocarpa and its hybrids). However, in September of 1997, the disease was observed in a hybrid poplar plantation at latitude 47.9°N and longitude 122.1°W near Snohomish, Washington. Blight affected the lower crown of trees in their second year of growth. Leaf lesions, with their characteristic black stromata, were easily distinguished from symptoms of other diseases. Some individual lesions of L. tetraspora affected entire leaf laminae, but there appeared to be little premature defoliation at the time of observation. Populus trichocarpa × P. deltoides hybrids were more commonly blighted than were P. trichocarpa × P. maximowiczii hybrids (i.e., 13/18 clones affected versus 4/11, respectively). A voucher specimen was deposited in the Herbarium at the Pacific Forestry Centre (DAVFP 25289). References: (1) M. E. Barr. Mycol. Mem. No. 7:130, 1978. (2) D. F. Farr. et al. 1989. Fungi on Plants and Plant Products in the United States. American Phytopathological Society, St. Paul, MN.
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8

Putnam, M. L. "First Report of Leaf Blight and Stem Dieback of St. John's-Wort Caused by Diploceras hypericinum in Oregon." Plant Disease 84, no. 11 (November 2000): 1250. http://dx.doi.org/10.1094/pdis.2000.84.11.1250b.

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St. John's-wort, Hypericum perforatum L., was formerly considered a noxious weed in the Pacific Northwest and is now grown commercially for its medicinal properties. In May 1999, plants from a 5-ha field in Jefferson County, OR, were observed with yellowing leaves and stem dieback. Lower leaves showed marginal necrosis or circular, expanding, uniformly brown, unremarkable leaf lesions that appeared randomly over the lamina and consumed from a quarter to nearly the entire leaf area. Remaining leaf tissue was chlorotic, and affected leaves eventually abscised. Infection of the stems resulted in girdling lesions 0.5 to 1.0 cm in length that caused chlorosis, wilting, and eventual dieback of tissues distal to the lesion. Diploceras hypericinum (Cesati) Diedicke was sporulating on affected stems and leaves. The fungus was isolated from surface-disinfested tissue onto 1.5% water agar. A single-spore isolate was used to inoculate 10-month-old plants raised from seed in sand. Spores from 6-week-old cultures grown on 50% potato-dextrose agar were harvested, suspended in phosphate buffer with 0.2% gelatin (PBG), and sprayed onto three plants using a DeVilbiss atomizer. Inoculum concentration was 7 × 103 and 3 ml per plant were used (plants were 8 to 10 cm tall). Three control plants were sprayed with sterile PBG. Inoculated and control plants were separately bagged to retain moisture and maintained at 22 to 25°C. Four days later, inoculated plants exhibited leaf spots similar to those originally observed, followed by stem dieback. D. hypericinum was isolated from all inoculated plants but not from control plants. The known distribution of D. hypericinum is France, Germany, Portugal, Sweden, Greece, and Ontario, Canada (1,2). This is the first report of D. hypericinum causing leaf blight and stem dieback of St. John's-wort in the United States. References: (1) D. F. Farr et al. 1989. Fungi on Plants and Plant Products in the United States. American Phytopathological Society, St. Paul, MN. (2) T. R. Nag Raj. 1993. Coelomycetous Anamorphs with Appendage-bearing Conidia. Mycologue Publications, Waterloo, Canada.
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9

Paulitz, T. C., F. Dugan, W. Chen, and N. J. Grünwald. "First Report of Pythium irregulare on Lentils in the United States." Plant Disease 88, no. 3 (March 2004): 310. http://dx.doi.org/10.1094/pdis.2004.88.3.310a.

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In late June and early July 2002, stunted, chlorotic, and partially defoliated lentils (Lens culinaris Medik.) were observed throughout the lentil-growing areas of eastern Washington. These symptoms were investigated in two fields near Garfield, WA and one field near Genesee, ID. Cv. Mason was more affected than cv. Brewer. Roots were dry and brittle with black discoloration in some cases. Isolates of Fusarium oxysporum and F. solani were obtained from washed roots plated on water agar, but they were nonpathogenic in greenhouse testing in pasteurized field soil and peat-based growing mixes. On 21 April 2003, volunteer lentils growing in the same fields showed symptoms of root rot, and Pythium oospores were observed in the roots. Pythium spp. were isolated by using a selective medium (2). Oospores were aplerotic, intercalary, 12.6 to 21 μm long × 11.2 to 18.2 μm wide, mostly smooth, and often formed in chains. Isolates resembled P. paroecandrum Drechs. and P. irregulare Buisman on the basis of morphological characters (3), but DNA sequences of the internal transcribed spacer region were closer to P. irregulare on the basis of a comparison with a worldwide database of Pythium sequences (C. A. Lévesque, personal communication). Isolates were deposited with the USDA-ARS Western Regional Plant Introduction Station, Pullman, WA. Four hyphal-tip isolates were tested in the greenhouse with inoculum grown in autoclaved sandy loam amended with 1% ground rolled oats. Experiments were performed twice in Thatuna silt loam, first in pasteurized and then in nonpasteurized soil. Inoculum was added to the soil at 500 CFU/g, and seeds were planted on the same day. Each isolate was tested on cvs. Brewer and Mason, with five replicates per treatment. Plants were grown in 4- × 20.5-cm plastic tubes (two plants per tube) for 1 month at 16 to 22°C and supplemented with 14 h of light per day. P. irregulare was reisolated from infected roots in both experiments. Damping-off, stunting, chlorosis, and root rot were observed in the Pythium-inoculated treatments, which corresponded to symptoms observed in the field in 2002. In pasteurized soil, only one isolate reduced the whole, dry, plant weight of Brewer, but the other three isolates reduced the dry weight of Mason. All isolates reduced the root dry weight of Mason in natural soil, but only two isolates reduced the root dry weight of Brewer. To our knowledge, Pythium spp., but not P. irregulare, have been reported previously from lentils (1). P. irregulare also causes root rot on winter wheat, which is rotated with lentils, and this pathogen likely causes yield reduction in both crops. References: (1) D. F. Farr et al. Fungi on Plants and Plant Products in the United States. The American Phytopathological Society, St. Paul, MN, 1989. (2) S. M. Mircetich and J. M. Kraft. Mycopathol. Mycol. Appl. 50:151, 1973. (3) A. J. van der Plaats-Niterink. Stud. Mycol. 21:1, 1981.
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10

Briere, S. C., and G. D. Franc. "First Report of Leaf and Stem Rust Caused by Puccinia sherardiana on Sphaeralcea grossulariaefolia in North America and S. munroana in Wyoming." Plant Disease 82, no. 7 (July 1998): 831. http://dx.doi.org/10.1094/pdis.1998.82.7.831a.

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Diseased samples of globe mallow, Sphaeralcea grossulariaefolia and S. munroana, were submitted by an ornamental seed producer in Wyoming to our Extension Plant Pathology Laboratory in July 1997. Dark brown, amphigenous telia surrounded by chlorotic halos were present on both foliage and stems. Mean teliospore dimensions observed were 30.8 × 44.8 μm. The teliospores germinated readily on water agar at 20°C and formed basidiospores within 24 h. Aecia and uredinia were not found. Based on characteristics mentioned above, this fungus was identified as Puccinia sherardiana Körn (1). This microcyclic rust was previously described on 12 other Sphaeralcea spp. plus other plant species in the Malvaceae family (1,2). Stem and foliar symptoms were reproduced in a greenhouse on 8-week-old plants of S. grossulariaefolia and S. munroana. These plants were inoculated with teliospores removed from the original diseased plant material. Immediately after inoculation, plants were misted and placed in plastic bags and incubated for 36 h at 100% relative humidity and 20°C. Plants continued growth with natural lighting and with day and night temperatures of 20 and 15°C, respectively. Symptoms developed within 12 days with initial telia rupturing the host epidermis 13 days after inoculation. Telia were examined microscopically to complete Koch's postulates. References: (1) J. C. Arthur. Manual of Plant Rusts in United States and Canada. Hafner Pub., 1962. (2) D. F. Farr et al. Fungi on Plants and Plant Products in the United States. American Phytopatholical Society, St. Paul, MN, 1989.
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Dissertations / Theses on the topic "Society of St. Vincent de Paul. United States"

1

Coon, Katherine E. "The Sisters of Charity in Nineteenth-Century America: Civil War Nurses and Philanthropic Pioneers." Thesis, Connect to resource online, 2010. http://hdl.handle.net/1805/2185.

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Thesis (M.A.)--Indiana University, 2010.
Title from screen (viewed on July 19, 2010). Departments of History and Philanthropic Studies, School of Liberal Arts, Indiana University-Purdue University Indianapolis (IUPUI). Advisor(s): Nancy Marie Robertson, Jane E. Schultz, Patricia Wittberg. Includes vitae. Includes bibliographical references (leaves 158-169).
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Books on the topic "Society of St. Vincent de Paul. United States"

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Society of St. Vincent de Paul. Council of the United States. United States manual of the Society of St. Vincent de Paul. Green Bay, Wis: Published for the Council of the United States by Alt Pub. Co., 1995.

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Society of St. Vincent de Paul. Manual of the Society of St. Vincent de Paul in the United States. St. Louis, Mo: Council of the United States, Society of St. Vincent de Paul, 2002.

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Hannefin, Daniel. Daughters of the church: A popular history of the Daughters of Charity in the United States, 1809-1987. Brooklyn, N.Y: New City Press, 1989.

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Kohn, Mary Rose. Rooted in faith: East Central Province, Daughters of Charity of Saint Vincent de Paul in the United States, 1981-1990. Evansville, Ind: Mater Dei Provincialate, 2006.

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Kingsbury, Virginia. Come to the water: The first twelve years of the East Central Province Daughters of Charity of Saint Vincent de Paul in the United States 1969-1981. Evansville, Ind: Mater Dei Provincialate, 1988.

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M, Kelly Ellin, and Melville Annabelle M. 1910-, eds. Elizabeth Seton: Selected writings. New York: Paulist Press, 1987.

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M, Melito Jack, O'Donnell Robert E, Estopare Debbie Sloan, Faherty William Barnaby 1914-, and Society of St. Vincent de Paul., eds. Society of St. Vincent de Paul: 150 years of service in the United States. St. Louis, MO: The Society, Metropolitan Central Council of St. Louis, 1995.

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Hannefin, Daniel. Daughters of the Church: A Popular History of the Daughters of Charity in the United States, 1809-1987. New City Pr, 1990.

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Bunkley, Josephine M. The Testimony Of An Escaped Novice From The Sisterhood Of St. Joseph, Emmettsburg, Maryland, The Mother-house Of The Sisters Of Charity In The United States. University of Michigan Library, 2001.

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