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Books on the topic 'Sodium phosphates'

Author: Grafiati
Published: 4 June 2021
Last updated: 7 February 2022

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1

Szuszkiewicz, Władysława. Struktura fazowa i właściwości krystalochemiczne fosforanów itru z wapniem i sodem. Wydawn. Akademii Ekonomicznej we Wrocławiu, 1991.

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2

Liu, George Zhigang. Studies on itaconyl bis (sodium methyl phosphate) cross-linked hemoglobin and the synthesis of acyl methyl phosphonates. National Library of Canada = Bibliothèque nationale du Canada, 1993.

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3

Tri-sodium phosphate: Manufactured by the Canada Chemical Manufacturing Co. Limited, London, Ontario. London Print. & Litho. Co., 1991.

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4

Relative Efficacy of Sodium Fluoride and Sodium Monofluorophosphate as Anti-Caries Agents in Dentifrices (International Congress & Symposium). Royal Society of Medicine Press Ltd, 1995.

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5

1933-, Bowen William H., ed. Relative efficacy of sodium fluoride and sodium monofluorophosphate as anti-caries agents in dentifrices: Proceedings of a conference sponsored by Unilever Research, held at the Royal Society of Medicine, London, 5 November 1994. Royal Society of Medicine Press, 1995.

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6

H, Bowen William, and Unilever Research Laboratory (Isleworth, London, England), eds. Relative efficacy of sodium fluoride and sodium monofluorophosphate as anti-caries agents in dentifrices: Proceedings of a conference sponsored by Unilever Research, held at the Royal Society of Medicine, London, 5 November 1994. Royal Society of Medicine Press, 1995.

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7

Murer, Heini, Jürg Biber, and Carsten A. Wagner. Phosphate homeostasis. Edited by Robert Unwin. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0025.

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Inorganic phosphate ions (H2PO4−/ HPO42−) (abbreviated as Pi) are involved in formation of bone and generation of high-energy bonds (e.g. ATP), metabolic pathways, and regulation of cellular functions. In addition, Pi is a component of biological membranes and nucleic acids. Only about 1% of total body Pi content is present in extracellular fluids, at a plasma concentration in adults within the range 0.8–1.4 mMol/L (at pH 7.4 mostly as HPO42−), with diurnal variations of approximately 0.2 mM. A small amount of plasma Pi is bound to proteins or forms complexes with calcium. Under normal, balanc
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8

Shotliffe, Dr Kevin, Dr Annabel Fountain, Dr Mike Jones, et al. Endocrinology and metabolic disorders. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199565979.003.00010.

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Chapter 10 covers endocrinology and metabolic disorders, including diabetes and diabetic coma, abnormalities of sodium and potassium, calcium, magnesium, and phosphate, metabolism, acid-base balance, thyroid emergencies, pituitary emergencies, adrenal emergencies, and toxin-induced hyperthermic syndromes.
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9

Parker, Philip M. The 2007-2012 World Outlook for Meta Sodium Phosphate (100 Percent NaPO3). ICON Group International, Inc., 2006.

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10

Parker, Philip M. The 2007-2012 World Outlook for Tripoly Sodium Phosphate (100 Percent Na5P3O10). ICON Group International, Inc., 2006.

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11

The 2006-2011 World Outlook for Meta Sodium Phosphate (100 Percent NaPO3). Icon Group International, Inc., 2005.

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12

Parker, Philip M. The 2007-2012 World Outlook for Tribasic Sodium Phosphate (100 Percent Na3PO4). ICON Group International, Inc., 2006.

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13

The 2006-2011 World Outlook for Dibasic Sodium Phosphate (100 Percent Na2HPO4). Icon Group International, Inc., 2005.

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14

Parker, Philip M. The 2007-2012 World Outlook for Dibasic Sodium Phosphate (100 Percent Na2HPO4). ICON Group International, Inc., 2006.

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15

The 2006-2011 World Outlook for Tribasic Sodium Phosphate (100 Percent Na3PO4). Icon Group International, Inc., 2005.

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16

The 2006-2011 World Outlook for Acid Pyro Sodium Phosphate (100 Percent Na2H2P2O7). Icon Group International, Inc., 2005.

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17

The 2006-2011 World Outlook for Tetrabasic Sodium Phosphate (pyro, 100 Percent Na4P2O7). Icon Group International, Inc., 2005.

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18

Jörres, Achim, Dietrich Hasper, and Michael Oppert. Electrolyte and acid–base disorders in AKI. Edited by Norbert Lameire. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199592548.003.0230.

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Electrolyte disturbances are common in patients with acute kidney injury (AKI) and should be corrected. In particular, hyperkalaemia above 6–6.5 mmol/L (especially with electrocardiogram changes) constitutes a medical emergency and warrants immediate intervention. Both hypo- and hypernatraemia may occur during AKI. Chronic changes in serum sodium need to be corrected bearing in mind the underlying pathology; however, when severe and evolving rapidly they should be corrected faster, irrespective of the cause. Acid–base disorders are also common in AKI and need to be treated in the context of un
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19

Covic, Adrian, Mugurel Apetrii, Luminita Voroneanu, and David J. Goldsmith. Vascular calcification. Edited by David J. Goldsmith. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199592548.003.0120_update_001.

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Vascular calcification (VC) is a common feature of patients with advanced CKD and it could be, at least in part, the cause of increased cardiovascular mortality in these patients. From a morphologic point of view, there are at least two types of pathologic calcium phosphate deposition in the arterial wall—namely, intima calcification (mostly associated with atherosclerotic plaques) and media calcification (associated with stiffening of the vasculature, resulting in significantly adverse cardiovascular outcomes). Although VC was viewed initially as a passive phenomenon, it appears to be a cell-
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