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Relevant bibliographies by topics / Somatosensory nervous system

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Academic literature on the topic 'Somatosensory nervous system'

Author: Grafiati

Published: 2 February 2023

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Journal articles on the topic "Somatosensory nervous system"

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KIMURA, AKIO. "Electrophysiological test for nervous system. Somatosensory evoked potential. (SEP)." JOURNAL OF JAPAN SOCIETY FOR CLINICAL ANESTHESIA 8, no. 1 (1988): 11–21. http://dx.doi.org/10.2199/jjsca.8.11.

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Singh, Harminder, Richard W. Vogel, Robert M. Lober, Adam T. Doan, Craig I. Matsumoto, Tyler J. Kenning, and James J. Evans. "Intraoperative Neurophysiological Monitoring for Endoscopic Endonasal Approaches to the Skull Base: A Technical Guide." Scientifica 2016 (2016): 1–20. http://dx.doi.org/10.1155/2016/1751245.

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Intraoperative neurophysiological monitoring during endoscopic, endonasal approaches to the skull base is both feasible and safe. Numerous reports have recently emerged from the literature evaluating the efficacy of different neuromonitoring tests during endonasal procedures, making them relatively well-studied. The authors report on a comprehensive, multimodality approach to monitoring the functional integrity of at risk nervous system structures, including the cerebral cortex, brainstem, cranial nerves, corticospinal tract, corticobulbar tract, and the thalamocortical somatosensory system during endonasal surgery of the skull base. The modalities employed include electroencephalography, somatosensory evoked potentials, free-running and electrically triggered electromyography, transcranial electric motor evoked potentials, and auditory evoked potentials. Methodological considerations as well as benefits and limitations are discussed. The authors argue that, while individual modalities have their limitations, multimodality neuromonitoring provides a real-time, comprehensive assessment of nervous system function and allows for safer, more aggressive management of skull base tumors via the endonasal route.

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Katz, Richard T. "Electrodiagnosis of the Peripheral Nervous System: An Introduction." Guides Newsletter 19, no. 3 (May 1, 2014): 10–14. http://dx.doi.org/10.1001/amaguidesnewsletters.2014.mayjun02.

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Abstract This article is an introduction to electrodiagnosis of the peripheral nervous system, including electromyography, electroneurography (nerve conduction studies), and somatosensory evoked potentials. Electromyography involves the introduction of a special recording needle into a muscle body in search of spontaneous activity (electrical potentials that occur while the muscle is at rest). Three types of spontaneous activity are of greatest relevance: positive sharp waves, fibrillation potentials, and fasciculations. Electromyography can help assess the status of nerve fibers indirectly, but the integrity of large myelinated sensory and motor neurons can be evaluated directly by nerve conduction studies (NCS), also known as electroneurography. NCS involves the introduction of an electrical stimulus, either by surface electrode or needle, and recording an evoked response. NCS can assess motor neurons, sensory neurons, or mixed nerve trunks, depending on the strategy employed. Somatosensory evoked potentials (SSEP) sometimes are useful as an adjunct to EMG and NCS in the diagnosis of peripheral nervous system pathology and are obtained by stimulating a peripheral mixed nerve at a frequency of approximately 2-5 Hz. Several manufacturers have created automated, hand-held units for performing nerve conduction studies, and neuromuscular ultrasound is noninvasive and painless, and ultrasound of nerve entrapment has identified nerve enlargement just proximal to the site of entrapment. Physicians should know or learn the qualifications of the physician to whom they refer their patients for electrodiagnostic assessment.

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Sólyom, A., S. Tóth, I. Holczinger, J. Vajda, Z. Tóth, and R. Kálmánchey. "The Spread of Somatosensory-Evoked Potentials Within the Nervous System." Stereotactic and Functional Neurosurgery 48, no. 1-6 (1985): 222–25. http://dx.doi.org/10.1159/000101131.

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Fustes, Otto Jesus Hernández, Cláudia Suemi Kamoi Kay, Paulo José Lorenzoni, Renata Dal-Prá Ducci, Lineu Cesar Werneck, and Rosana Herminia Scola. "Somatosensory evoked potentials in clinical practice: a review." Arquivos de Neuro-Psiquiatria 79, no. 9 (September 2021): 824–31. http://dx.doi.org/10.1590/0004-282x-anp-2020-0427.

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Abstract The authors present a review of the current use of somatosensory evoked potentials (SSEPs) in neurological practice as a non-invasive neurophysiological technique. For this purpose we have reviewed articles published in English or Portuguese in the PubMed and LILACS databases. In this review, we address the role of SSEPs in neurological diseases that affect the central nervous system and the peripheral nervous system, especially in demyelinating diseases, for monitoring coma, trauma and the functioning of sensory pathways during surgical procedures. The latter, along with new areas of research, has become one of the most important applications of SSEPs.

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Hao, Han, Rosmaliza Ramli, Caixue Wang, Chao Liu, Shihab Shah, Pierce Mullen, Varinder Lall, et al. "Dorsal root ganglia control nociceptive input to the central nervous system." PLOS Biology 21, no. 1 (January 5, 2023): e3001958. http://dx.doi.org/10.1371/journal.pbio.3001958.

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Accumulating observations suggest that peripheral somatosensory ganglia may regulate nociceptive transmission, yet direct evidence is sparse. Here, in experiments on rats and mice, we show that the peripheral afferent nociceptive information in mice undergoes dynamic filtering within the dorsal root ganglion (DRG) and suggest that this filtering occurs at the axonal bifurcations (t-junctions). Using synchronous in vivo electrophysiological recordings from the peripheral and central processes of sensory neurons (in the spinal nerve and dorsal root), ganglionic transplantation of GABAergic progenitor cells, and optogenetics, we demonstrate existence of tonic and dynamic filtering of action potentials traveling through the DRG. Filtering induced by focal application of GABA or optogenetic GABA release from the DRG-transplanted GABAergic progenitor cells was specific to nociceptive fibers. Light-sheet imaging and computer modeling demonstrated that, compared to other somatosensory fiber types, nociceptors have shorter stem axons, making somatic control over t-junctional filtering more efficient. Optogenetically induced GABA release within DRG from the transplanted GABAergic cells enhanced filtering and alleviated hypersensitivity to noxious stimulation produced by chronic inflammation and neuropathic injury in vivo. These findings support “gating” of pain information by DRGs and suggest new therapeutic approaches for pain relief.

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Aminoff, Michael J. "Use of Somatosensory Evoked Potentials to Evaluate the Peripheral Nervous System." Journal of Clinical Neurophysiology 4, no. 2 (April 1987): 135–44. http://dx.doi.org/10.1097/00004691-198704000-00003.

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Jeffry, Joseph, Seungil Kim, and Zhou-Feng Chen. "Itch Signaling in the Nervous System." Physiology 26, no. 4 (August 2011): 286–92. http://dx.doi.org/10.1152/physiol.00007.2011.

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Itch is a major somatic sensation, along with pain, temperature, and touch, detected and relayed by the somatosensory system. Itch can be an acute sensation, associated with mosquito bite, or a chronic condition, like atopic dermatitis ( 29 , 59 ). The origins of the stimulus can be localized in the periphery or systemic, and associated with organ failure or cancer. Itch is also a perception originating in the brain. Itch is broadly characterized as either histamine-dependent (histaminergic) or histamine-independent (nonhistaminergic), both of which are relayed by subsets of C fibers and by the second-order neurons expressing gastrin-releasing peptide receptor (GRPR) and spinothalamic track (STT) neurons in the spinal cord of rodents. Historically, itch research has been primarily limited to clinical and psychophysical studies and to histamine-mediated mechanisms. In contrast, little is known about the signaling mechanisms underlying nonhistaminergic itch, despite the fact that the majority of chronic itch are mediated by nonhistaminergic mechanisms. During the past few years, important progress has been made in understanding the molecular signaling of itch, largely due to the introduction of mouse genetics. In this review, we examine some of the molecular mechanisms underlying itch sensation with an emphasis on recent studies in rodents.

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Fang, Fang, Qian Luo, Ren-Bin Ge, Meng-Yu Lai, Yu-Jia Gong, Mei Kang, Ming-Ming Ma, et al. "Decreased Microstructural Integrity of the Central Somatosensory Tracts in Diabetic Peripheral Neuropathy." Journal of Clinical Endocrinology & Metabolism 106, no. 6 (March 12, 2021): 1566–75. http://dx.doi.org/10.1210/clinem/dgab158.

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Abstract Context Although diabetic peripheral neuropathy (DPN) is predominantly considered a disorder of the peripheral nerves, some evidence for central nervous system involvement has recently emerged. However, whether or to what extent the microstructure of central somatosensory tracts may be injured remains unknown. Objective This work aimed to detect the microstructure of central somatosensory tracts in type 2 diabetic patients and to correlate it with the severity of DPN. Methods A case-control study at a tertiary referral hospital took place with 57 individuals with type 2 diabetes (25 with DPN, 32 without DPN) and 33 nondiabetic controls. The fractional anisotropy (FA) values of 2 major somatosensory tracts (the spinothalamic tract and its thalamocortical [spino-thalamo-cortical, STC] pathway, the medial lemniscus and its thalamocortical [medial lemnisco-thalamo-cortical, MLTC] pathway) were assessed based on diffusion tensor tractography. Regression models were further applied to detect the association of FA values with the severity of DPN in diabetic patients. Results The mean FA values of left STC and left MLTC pathways were significantly lower in patients with DPN than those without DPN and controls. Moreover, FA values of left STC and left MLTC pathways were significantly associated with the severity of DPN (expressed as Toronto Clinical Scoring System values) in patients after adjusting for multiple confounders. Conclusion Our findings demonstrated the axonal degeneration of central somatosensory tracts in type 2 diabetic patients with DPN. The parallel disease progression of the intracranial and extracranial somatosensory system merits further attention to the central nerves in diabetic patients with DPN.

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Lee, Wang Wei, Yu Jun Tan, Haicheng Yao, Si Li, Hian Hian See, Matthew Hon, Kian Ann Ng, Betty Xiong, John S. Ho, and Benjamin C. K. Tee. "A neuro-inspired artificial peripheral nervous system for scalable electronic skins." Science Robotics 4, no. 32 (July 17, 2019): eaax2198. http://dx.doi.org/10.1126/scirobotics.aax2198.

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The human sense of touch is essential for dexterous tool usage, spatial awareness, and social communication. Equipping intelligent human-like androids and prosthetics with electronic skins—a large array of sensors spatially distributed and capable of rapid somatosensory perception—will enable them to work collaboratively and naturally with humans to manipulate objects in unstructured living environments. Previously reported tactile-sensitive electronic skins largely transmit the tactile information from sensors serially, resulting in readout latency bottlenecks and complex wiring as the number of sensors increases. Here, we introduce the Asynchronously Coded Electronic Skin (ACES)—a neuromimetic architecture that enables simultaneous transmission of thermotactile information while maintaining exceptionally low readout latencies, even with array sizes beyond 10,000 sensors. We demonstrate prototype arrays of up to 240 artificial mechanoreceptors that transmitted events asynchronously at a constant latency of 1 ms while maintaining an ultra-high temporal precision of <60 ns, thus resolving fine spatiotemporal features necessary for rapid tactile perception. Our platform requires only a single electrical conductor for signal propagation, realizing sensor arrays that are dynamically reconfigurable and robust to damage. We anticipate that the ACES platform can be integrated with a wide range of skin-like sensors for artificial intelligence (AI)–enhanced autonomous robots, neuroprosthetics, and neuromorphic computing hardware for dexterous object manipulation and somatosensory perception.

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Dissertations / Theses on the topic "Somatosensory nervous system"

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Whittle, Ian Roger. "Clinical applications of somatosensory evoked potentials in pediatric neurosurgery /." Title page, contents and summary only, 1985. http://web4.library.adelaide.edu.au/theses/09MD/09mdw627.pdf.

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Li, Jianying. "Quantitative Analysis of the Gabaergic System in Cat Primary Somatosensory Cortex and Its Relation to Receptive Field Properties." Thesis, University of North Texas, 1995. https://digital.library.unt.edu/ark:/67531/metadc279190/.

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Sensory neocortex contains a significant number of inhibitory neurons that use gamma-aminobutyric acid (GABA) as their neurotransmitter. Functional roles for these neurons have been identified in physiological studies. For example, in primary somatosensory cortex (SI), blockade of GABAa receptors with bicuculline leads to expansion of receptive fields (RFs). The magnitude of RF enlargement varies between SIpopulations of GABAergic neurons were identified by labeling specific calcium binding proteins.

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Salian, Dilip. "En jämförelse av kortikal registrering mellan olika registreringspunkter vid Somatosensory evoked potentials." Thesis, Örebro universitet, Institutionen för hälsovetenskaper, 2020. http://urn.kb.se/resolve?urn=urn:nbn:se:oru:diva-84599.

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Bakgrund- Sensorisk evoked potentials (SEP) är en neurofysiologisk undersökningsmetod som används för att monitorera svarspotentialer från kroppens sensoriska nervsystem efter en sensorisk stimulering. Registreringen av sensorisk evoked potentials sker med små elektriska stimuleringar över en perifer sensorisk nerv och registreras från tre olika registreringskanaler som benämns N9 över plexus brachialis, N13 Erb’s punkt och N20 för det primär sensoriska cortexområdet. Metod- I denna studie bearbetas data från 20 registreringar för N20-kanalen. Med registreringen av den klinisk använda standardmontaget C3’-Fz som används vid Karolinska universitetssjukhuset, som jämförs mot nya registreringsmontagen C3’-CPz, CP3-Fz CP3-CPz. Stimulering skedde unilateralt över höger nervus medianus på handledsnivå. Syftet med studien var att ta reda på om det fanns någon statistisk signifikant skillnad mellan standardmontaget C3’-Fz mot de alternativa montagen med avseende på amplitud, duration och latenstid mellan två registreringsomgångar. Den statistiska analysen genomfördes med Wilcoxsons teckenranktest för differenserna av registreringsomgångarna i amplitud, duration och latenstid. Spearmans rangkorrelationstest användes för att visa sambandet mellan standardmontaget och de nya registreringsmontagen i amplitud. Resultat- Resultatet visade ingen statistisk signifikant skillnad mellan standardmontaget mot de alternativa montagen för differenserna av amplitud, duration och latenstid mellan de två registringsomgångarna. Korrelationen för amplituderna visade att montaget CP3-Fz hade en starkare grad av samband mot standardmontaget C3’-Fz jämfört med registreringsmontagen C3’-CPz och CP3-CPz. Slutsats- Slutsatsen av denna studie är att det inte fanns någon statistisk signifikant skillnad i differenserna för amplitud, duration och latenstiderna vid jämförelse av standardmontaget mot de nya alternativa montagen. Dock visade montage CP3-Fz på ett starkare samband mot den klinisk använda C3-Fz jämfört med resterande montage med avseende på amplituden.
Background-Sensory evoked potentials (SEP) are a neurophysiological examination method used to monitor electrical response potentials from the body’s sensory nervous system. The registration follows three recording channels throughout the sensory pathway as N9 over plexus brachialis, N13 over cervical vertebrae mentioned as Erb’ point and N20 represented for the primary somatosensory cortex area. Method- In this study data was collected from 20 registrations for N20 channel. Registration for this study measured the clinical used cortical registration montage at Karolinska university hospital C3’-Fz against new registration montages C3'-CPz, CP3-Fz and CP3-CPz, with stimulation on the right median nerve at wrist level unilateral. The purpose of the study was to see if there exists any significant difference between the standard montage C3’-Fz against the new alternative registration montages in regard to amplitude, duration and latency after two registration rounds. Wilcoxson’s singed rank test were used to compare the difference in amplitude, duration and latency between registration rounds. Spearman’s correlation test were used to show the correlation between the standard montage and the new registration montages in amplitude. Result-The result showed no statistical significant difference between the standard montage and the new alternative montages in amplitude, duration and latency for the two registration rounds. The correlation showed registration montage CP3-Fz with a greater correlation towards the standard montage C3’-Fz compared to registration montages C3’-CPz and CP3-CPz in amplitude. Conclusions- This study showed no significant difference in amplitude, duration and latency when it compared the standard montage C3’-Fz against the new alternative montages. The correlation in amplitude showed montage Cp3-Fz with a stronger correlation towards the clinical used registration montage compared to the other new alternative montages.

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LaPrairie, Jamie L. "The Impact of Neonatal Inflammatory Insult on Adult Somatosensory Processing: The Role of the Descending Nociceptive Circuit." Atlanta, Ga. : Georgia State University, 2008. http://digitalarchive.gsu.edu/biology_diss/42/.

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Thesis (Ph. D.)--Georgia State University, 2008.
Title from title page (Digital Archive@GSU, viewed June 16, 2010) Anne Z. Murphy, committee chair; Timothy Bartness, Matthew Grober, Michael Gold, Charles Derby, committee members. Includes bibliographical references (p. 148-164).

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Alves, da Motta Mauricy. "Contribution a l'etude des correlations existant entre les potentiels evoques somesthesiques et le debit sanguin cerebral." Toulouse 3, 1988. http://www.theses.fr/1988TOU30065.

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Hofstetter, Christoph. "Cell therapy for spinal cord injury, studies of motor and sensory systems /." Stockholm, 2005. http://diss.kib.ki.se/2005/91-7140-382-5/.

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Derre, Alexandre. "Douleurs chroniques : implication et potentiel thérapeutique des membres de la famille FXYD." Thesis, Université de Montpellier (2022-….), 2022. http://www.theses.fr/2022UMONT006.

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Douleurs chroniques : Implication et potentiel thérapeutique des membres de la famille FXYDLes douleurs chroniques constituent un problème majeur de santé publique affectant près de 18% de la population mondiale. Elles ont des conséquences néfastes sur la qualité de vie des patients et engendrent des situations critiques sur le plan médical, sociologique et économique. Les traitements actuels sont relativement limités, souvent inefficaces et/ou présentent des effets secondaires délétères. De fait, une meilleure connaissance et une meilleure prise en charge de cette pathologie constituent des enjeux importants en recherche fondamentale et clinique.Dans ce contexte, mon projet de thèse a porté sur deux protéines, Fxyd2 et Fxyd7, qui appartiennent à la famille des protéines à motif FXYD qui contient 7 membres. Ces deux protéines sont décrites comme modulateurs de l’activité de la pompe Na,K-ATPase, et sont présentes dans des sous-types très spécifiques de neurones somatosensensoriels au sein des ganglions rachidiens dorsaux. La pompe Na,K-ATPase est décrite dans de nombreux phénomènes physiologiques et joue un rôle important dans l’excitabilité neuronale par le maintien du potentiel membranaire grâce à un jeu d'entrée et de sortie de sodium (Na+) et de potassium (K+). Le maintien de cet équilibre ionique est d’autant plus important que des phénomènes d’hyperexcitabilité neuronale sont souvent décrits dans le cadre des douleurs chroniques.Dans un premier objectif de mon projet de thèse, j’ai participé au développement d’une stratégie thérapeutique transposable à l’Homme basée sur une approche innovante de thérapie génique d’extinction. Ainsi, nous avons montré que l’usage d’oligonucléotides antisens lipidomodifiés dirigés contre le gène Fxyd2 et administrés par voie intrathécale permet un effet analgésique puissant chez des rats en condition de douleur neuropathique ou inflammatoire, aboutissant au retour à une sensibilité mécanique normale. De plus, des modifications chimiques conférant une meilleure stabilité de notre molécule thérapeutique permettent de prolonger son efficacité jusqu’à 10 jours.Mes travaux se sont portés, dans un deuxième objectif, sur la compréhension des modes d’action de Fxyd2 dans la physiopathologie des neurones somatosensoriels des ganglions rachidiens dorsaux notamment en identifiant ses partenaires protéiques par une approche protéomique. Ainsi, nous avons montré par spectrométrie de masse en tandem et par Proximity Ligation Assay, que Fxyd2 pouvait interagir de manière directe avec d’autres protéines que la sous unité ɑ1 de la pompe Na,K-ATPase en condition physiologique chez la souris. En effet, Fxyd2 semble interagir également avec la sous unité ɑ3 de cette pompe et aussi avec la PMCA, la GST et la Prdx6.Dans un troisième objectif, j’ai également étudié le rôle du gène Fxyd7 dans le système somatosensoriel aussi bien en condition normale que pathologique. Dans un premier temps, j’ai confirmé son profil d’expression au sein de sous populations neuronales de nocicepteurs peptidergiques, de mécanocepteurs et de neurones proprioceptifs dans les GRD de souris. Ensuite, en utilisant la souris Knock-Out pour le gène Fxyd7, j’ai procédé à des différents tests de motricité, d’équilibre et de sensibilité qui n’ont montré aucun défaut majeur chez ces souris en condition naïve. En condition de douleur neuropathique, avec le modèle SNL (Spinal Nerve Ligation), les tests de sensibilité mécanique ont montré aucune influence de la mutation, ni sur la phase aigüe de la douleur, ni sur sa chronicisation alors qu’en condition de douleur inflammatoire, avec le modèle CFA (Complete Freund’s Adjuvant), l’absence du gène empêche la chronicisation de la douleur inflammatoire de manière remarquable.Nos résultats montrent ainsi un potentiel thérapeutique majeur de deux membres de la famille FXYD pour traiter les douleurs chroniques
Chronic pain: Implication and therapeutic potential of FXYD protein members Chronic pain is a major public health problem affecting nearly 18% of the world’s population. It has deleterious consequences on patient’s quality of life and generates critical situations on the medical, sociological and economic levels. Current treatments are relatively limited, often ineffective and/or have deleterious side effects. In fact, better knowledge and an improved management of these pathologies is a major challenge for fundamental and clinical research.In this context, my thesis project is based on two different proteins, Fxyd2 and Fxyd7, which are members of a family of 7 proteins which contain a characteristic FXYD amino-acid motif. These two proteins have been described as modulators of the Na,K-ATPases’ activity, and are present in very specific somatosensory neurons of the dorsal root ganglia. The Na,K-ATPase pump is implicated in a large variety of physiological phenomena with a critical role in neuronal excitability by maintaining membrane potential thanks to the transfer of sodium (Na+) and potassium (K+). The maintenance of this ionic equilibrium is a crucial point since neuronal hyperexcitability has often been described in chronic pain.The first objective of my thesis was to develop a therapeutic strategy suitable for human therapy based on a very innovative gene extinction strategy. Thus, we showed that lipidomodified antisense oligonucleotides directed against the Fxyd2 gene and administered intrathecally induce a strong analgesic effect in neuropathic pain or in inflammatory pain models of rats, leading to normal mechanical sensitivity. Moreover, we showed that specific chemical modifications induce a better stability of our therapeutic molecule which prolongs its efficacy up to 10 days.In the second objective, my work was directed toward understanding the mechanisms of action of Fxyd2 in neuronal physiopathology in dorsal root ganglia, especially by identifying its protein partners using a proteomic approach. Thus, I showed by tandem mass spectrometry and by Proximity Ligation Assay that Fxyd2 could interact directly with proteins other than the ɑ1 subunit of the Na,K-ATPase in physiological conditions in mice. Indeed, Fxyd2 seems to interact also with the ɑ3 subunit of this pump and also with PMCA, GST and Prdx6.My third objective was to study the role of the Fxyd7 gene in the somatosensory system in normal and pathological conditions. In the first place, I used in situ hybridization to show its expression in specific neuronal subpopulations including peptidergic nociceptors, mechanoreceptors and in proprioceptive neurons in the mouse DRG. Then, using motor, equilibrium and mechanical sensitivity tests in Fxyd7 KO mice, I demonstrated the absence of major behavioral defects in these mice in normal conditions. In neuropathic pain conditions, using the SNL (Spinal Nerve Ligation) model, mechanical sensitivity tests did not reveal any influence of this mutation, neither in the acute nor chronic phases. However, in chronic inflammatory pain conditions induced by injection of CFA (Complete Freund’s Adjuvant), Fxyd7 null mutants failed to maintain pain responses. Thus Fxyd7 expression in DRG neurons appears to be specifically required for the maintenance of chronic inflammatory pain.Our results thus show a major therapeutic potential of two FXYD family members to treat chronic pain

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Gioanni, Yves. "Organisation fonctionnelle du cortex moteur et epilepsie experimentale : implication des reflexes transcorticaux dans l'elaboration des decharges paroxystiques." Paris 7, 1987. http://www.theses.fr/1987PA077206.

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FORESTIER-BEN, HAMIDA CHRISTIANE. "Etude qualitative et quantitative de l'ontogenese post-natale du gyrus supra-sylvien du chat : correlations spatio-temporelles de differents indicateurs morphologiques de developpement." Paris 6, 1987. http://www.theses.fr/1987PA066167.

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Martínez, Jauand Mercedes. "Factores genéticos y psicosociales implicados en la modulación del dolor." Doctoral thesis, Universitat de les Illes Balears, 2013. http://hdl.handle.net/10803/125008.

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La La sensibilidad al dolor y el riesgo sufrir dolor crónico representan fenómenos complejos de naturaleza multidimensional, con una importante variabilidad interindividual. El objetivo de esta tesis doctoral se centró en explorar los factores genéticos y psicosociales implicados en la modulación del dolor y el riesgo a sufrir dolor crónico mediante seis estudios. Los dos primeros exploraron el efecto de factores genéticos y de la edad de inicio de la menopausia en la sensibilidad al dolor en pacientes con fibromialgia en comparación con voluntarias sanas. El primer estudio reveló un incremento de la frecuencia de alelos asociados a una reducida actividad de la enzima COMT en pacientes con síndrome de fibromialgia, junto con una elevada sensibilidad al dolor en estos grupos. El segundo estudio mostró que las pacientes con fibromialgia presentaron una edad de inicio de la menopausia más temprana que las controles. Asimismo, se encontró que las pacientes con menopausia temprana mostraban mayor sensibilidad al dolor que las pacientes con aparición tardía de la menopausia. Los dos siguientes examinaron el papel de factores genéticos en la actividad metabólica cerebral asociada al dolor, la respuesta de analgesia por placebo y en una tarea de funciones ejecutivas. El tercer estudio reveló que el alelo met66 del polimorfismo Val66Met en el gen BDNF se asociaba a un fenotipo de vulnerabilidad, resistencia a la analgesia por placebo, incrementos en la actividad dopaminérgica durante el procesamiento de dolor y reducciones durante la condición placebo. Asimismo, se observó que estos efectos eran dependientes del género del sujeto, con una mayor exacerbación en mujeres y con efectos nulos en hombres. El cuarto estudio mostró aumentos en la actividad del sistema opioide asociados a la respuesta de analgesia por placebo en portadores del alelo C del polimorfismo funcional C385A del gen FAAH. Por otro lado, no se encontraron diferencias significativas debidas a este polimorfismo en la respuesta al dolor en ausencia de placebo, ni en la activación del sistema dopaminérgico. Finalmente, los dos últimos estudios exploraron la modulación social analizando cambios en la actividad eléctrica cerebral como consecuencia de la observación de dolor y tacto en otros. El quinto estudio mostró diferencias en los potenciales evocados visuales en función de la percepción de expresiones faciales de dolor y enfado. El sexto estudio reveló que la observación de experiencias somatosensoriales dolorosas y no dolorosas en otras personas modulaba la amplitud de los potenciales somatosensoriales. Todos estos datos subrayan la naturaleza multidimensional de la respuesta al dolor y resaltan el papel de los factores genéticos y psicosociales en la persistencia del dolor a lo largo del tiempo.
Pain sensitivity and risk for chronic pain constitute complex multidimensional phenomena that vary significantly among individuals. The objective of the present Doctoral Thesis was focused on exploring genetic and psychosocial factors involved in the modulation of pain and chronic pain risk throughout six studies. The first two studies explored the effect of genetic factors and age-of-onset of menopause in pain sensitivity in fibromyalgia patients as compared to healthy volunteers. The first study showed an increased frequency of alleles associated with a reduced activity of COMT enzyme in patients with fibromyalgia syndrome, coupled with high sensitivity to pain in these groups. The second study showed that patients with fibromyalgia had an ageof- onset of menopause earlier than controls. We also found that patients with early menopause showed higher pain sensitivity than patients with late age-of-onset of menopause. The next two studies explored brain metabolic activity in response to pain and placebo analgesia and during an executive function task. The third study revealed that met66 allele of the Val66Met polymorphism in the BDNF gene was associated with a phenotype of vulnerability, strength, placebo analgesia, increases in dopaminergic activity during the processing of pain and reductions during the placebo condition. It was also noted that these effects were dependent on gender, being exacerbated in women as compared to men. The fourth study showed increases in placebo analgesia and in placebo-induced opioid activity in the C385 allele of the functional polymorphism C385A of the FAAH gene. Furthermore, there were no significant differences due to this polymorphism in the pain response in absence of placebo, or in dopaminergic system activation. Finally, the latter two studies explored the social modulation of brain electrical activity during observation of pain and somatosensory experiences in other´s. The fifth study showed differences in visual evoked potentials during the sight of pain and anger faces. The sixth study showed that observation of painful and non-painful experiences in others modulated the amplitude of somatosensory evoked potentials in the onlooker. These data underscore the multidimensional nature of pain response and highlight the role of genetic and psychosocial factors in the persistence of pain.

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Books on the topic "Somatosensory nervous system"

1

Baumgartner, Christoph. Clinical Electrophysiology of the Somatosensory Cortex: A Combined Study Using Electrocorticography, Scalp-EEG, and Magnetoencephalography. Vienna: Springer Vienna, 1993.

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Evoked potential primer: Visual, auditory, and somatosensory evoked potentials in clinical diagnosis. Boston: Butterworth, 1985.

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Spehlmann, Rainer. Evoked potential primer: Visual, auditory, and somatosensory evoked potentials in clinical diagnosis. Boston: Butterworth, 1985.

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Snow, Peter J., and Peter Wilson. Plasticity in the Somatosensory System of Developing and Mature Mammals — The Effects of Injury to the Central and Peripheral Nervous System. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75701-3.

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1931-, Spehlmann Rainer, ed. Spehlmann's evoked potential primer: Visual, auditory, and somatosensory evoked potentials in clinical diagnosis. 2nd ed. Boston: Butterworth-Heinemann, 1994.

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Kuhn, H., G. Breithardt, Peter J. Snow, U. Gleichmann, J. Schoenmackers, H. H. Dahm, H. Gillmann, R. M. Jungblut, W. Krelhaus, and F. Loogen. Plasticity in the Somatosensory System of Developing and Mature Mammals - The Effects of Injury to the Central and Peripheral Nervous System. Springer, 2011.

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Peter, Wilson, and Peter J. Snow. Plasticity in the Somatosensory System of Developing and Mature Mammals -- the Effects of Injury to the Central and Peripheral Nervous System. Springer, 2012.

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Mauguière, François, and Luis Garcia-Larrea. Somatosensory and Pain Evoked Potentials. Edited by Donald L. Schomer and Fernando H. Lopes da Silva. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190228484.003.0043.

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This chapter discusses the use of somatosensory evoked potentials (SEPs) and pain evoked potentials for diagnostic purposes. The generators of SEPs following upper limb stimulation have been identified through intracranial recordings, permitting the analysis of somatosensory disorders caused by neurological diseases. Laser activation of fibers involved in thermal and pain sensation has extended the applications of evoked potentials to neuropathic pain disorders. Knowledge of the effects of motor programming, paired stimulations, and simultaneous stimulation of adjacent somatic territories has broadened SEP use in movement disorders. The recording of high-frequency cortical oscillations evoked by peripheral nerve stimulation gives access to the functioning of SI area neuronal circuitry. SEPs complement electro-neuro-myography in patients with neuropathies and radiculopathies, spinal cord and hemispheric lesions, and coma. Neuroimaging has overtaken SEPs in detecting and localizing central nervous system lesions, but SEPs still permit assessment of somatosensory and pain disorders that remain unexplained by anatomical investigations.

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(Foreword), Thomas Woolsey, ed. Barrel Cortex. Cambridge University Press, 2008.

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Fox, Kevin. Barrel Cortex. Cambridge University Press, 2008.

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Book chapters on the topic "Somatosensory nervous system"

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Brandt, Thomas. "Somatosensory Vertigo." In Clinical Medicine and the Nervous System, 277–88. London: Springer London, 1991. http://dx.doi.org/10.1007/978-1-4471-3342-1_23.

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Snow, Peter J., and Peter Wilson. "Plasticity in the Peripheral Somatosensory Nervous System." In Plasticity in the Somatosensory System of Developing and Mature Mammals — The Effects of Injury to the Central and Peripheral Nervous System, 6–57. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75701-3_2.

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Becker, Aimee, and Deborah A. Rusy. "Somatosensory Evoked Potentials." In Monitoring the Nervous System for Anesthesiologists and Other Health Care Professionals, 3–26. Boston, MA: Springer US, 2011. http://dx.doi.org/10.1007/978-1-4614-0308-1_1.

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Becker, Aimee, Corey Amlong, and Deborah A. Rusy. "Somatosensory-Evoked Potentials." In Monitoring the Nervous System for Anesthesiologists and Other Health Care Professionals, 3–18. Cham: Springer International Publishing, 2017. http://dx.doi.org/10.1007/978-3-319-46542-5_1.

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Kochs, E., and P. Bischoff. "Anesthesia and Somatosensory Evoked Responses." In Central Nervous System Monitoring in Anesthesia and Intensive Care, 146–75. Berlin, Heidelberg: Springer Berlin Heidelberg, 1986. http://dx.doi.org/10.1007/978-3-642-78441-5_12.

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Amlong, Corey, Whitney Fallahian, Aimee Becker, and Deborah A. Rusy. "Somatosensory-Evoked Potentials." In Koht, Sloan, Toleikis's Monitoring the Nervous System for Anesthesiologists and Other Health Care Professionals, 3–19. Cham: Springer International Publishing, 2022. http://dx.doi.org/10.1007/978-3-031-09719-5_1.

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Tononi, Giulio. "Functional segregation and integration in the nervous system: Theory and models." In Somesthesis and the Neurobiology of the Somatosensory Cortex, 409–18. Basel: Birkhäuser Basel, 1996. http://dx.doi.org/10.1007/978-3-0348-9016-8_34.

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Snow, Peter J., and Peter Wilson. "Plasticity and the Somatosensory Thalamus." In Plasticity in the Somatosensory System of Developing and Mature Mammals — The Effects of Injury to the Central and Peripheral Nervous System, 286–311. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75701-3_6.

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Snow, Peter J., and Peter Wilson. "Plasticity and the Somatosensory Cerebral Cortex." In Plasticity in the Somatosensory System of Developing and Mature Mammals — The Effects of Injury to the Central and Peripheral Nervous System, 312–93. Berlin, Heidelberg: Springer Berlin Heidelberg, 1991. http://dx.doi.org/10.1007/978-3-642-75701-3_7.

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Colella, Micol, Micaela Liberti, Francesca Apollonio, and Giorgio Bonmassar. "A Miniaturized Ultra-Focal Magnetic Stimulator and Its Preliminary Application to the Peripheral Nervous System." In Brain and Human Body Modeling 2020, 167–76. Cham: Springer International Publishing, 2020. http://dx.doi.org/10.1007/978-3-030-45623-8_9.

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AbstractTranscranial magnetic stimulation (TMS) is a noninvasive brain stimulation technique used in the clinic to treat several neurological disorders and psychiatric diseases. One of TMS’s significant limitations is its low spatial resolution, which often results in a mismatch between the target area in the brain and the stimulation site on the scalp. To enhance its spatial resolution, we designed and built a complete stimulation system complete with a millimetric-diameter coil and microscopic traces (μCoil). The first tests conducted on healthy volunteers showed that the μCoil stimulation of the radial nerve in the wrist could indeed evoke somatosensory nerve action potentials (SNAPs). In this chapter, we study this nerve stimulation system with electromagnetic and neuron simulators on a neurofunctionalized model from the Virtual Population (ViP v.4) and a μCoil figure-8 geometry. In particular, we study how changes in the μCoil geometry, such as the number of layers, shape, and length of an iron or air core, may help to promote the generation of somatosensory nerve action potentials.

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Conference papers on the topic "Somatosensory nervous system"

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Sugimoto a, Junya, and Hiroshi Hagiwara b. "Effect of fNIRS on Physiological Index and Performance Under Vibratory Stimulus." In Applied Human Factors and Ergonomics Conference. AHFE International, 2019. http://dx.doi.org/10.54941/ahfe100217.

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Many traffic accidents are caused by human error. In order to help prevent human such error, we investigated brain hemodynamics in both the frontal and the somatosensory areas by functional near-infrared spectroscopy (fNIRS), electrocardiogram (ECG), and the following two types of task performance with vibratory stimulation: a Tracking task and the Stroop test. To evaluate changes in oxygenated hemoglobin concentration (oxyHb), we used δoxyHb as previously defined (H. Iwasaki: Availability and future prospects of functional near-infrared spectroscopy in usability evaluation, Human Factors and Ergonomics). Briefly, the waveform data of oxyHb are passed through a differential filter. A sum of more than zero is defined as a positive component, whereas a sum of less than zero is defined as a negative component. δoxyHb is defined as a positive component minus a negative component. δoxyHb > 0 indicates an increasing trend of oxyHb and δoxyHb < 0 indicates a decreasing trend of oxyHb. Our results show that tracking error and the variance of tracking error were reduced when vibratory stimulation was present. Marginally statistically significant (p < 0.1) differences for both tracking and Stroop indices were observed when comparing measures with and without vibratory stimulation. These results suggest that subjects were able to track targets more stably with than without vibratory stimulation. On the other hand, performance on the Stroop test (reaction time, variance of Stroop test, and percentage of correct answers) was not affected by vibratory stimulation. ECG HF (high frequency) in both tasks was lower with than without vibratory stimuli. ECG LF (low frequency)/HF in both tasks was higher with than without vibratory stimuli. The results of HF and LF/HF stimulation imply the predominance of both the sympathetic nervous system during vibratory stimulation and the parasympathetic nervous system with no stimulus. δoxyHb showed differences in the somatosensory area during the Tracking task between vibratory stimulation and no stimulation. In summary, presentation of vibratory stimuli improved performance in the Tracking task. Therefore, use of vibratory stimulation during driving may decrease traffic accidents caused by human error.

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Valenti, Fabio. "Use of Insoles to Enhance Postural Control." In Socratic Lectures 7. University of Lubljana Press, 2022. http://dx.doi.org/10.55295/psl.2022.d3.

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Abstract: Postural control is a complex process involving sensory inputs from visual, vestibular, proprioceptive and tactile receptors, processed by the central nervous system (CNS). Sensory information provided by muscle and cutaneous afferents in the foot optimize the ability to stand upright and control the postural sway. The foot, as a direct and often only interface between the body and the ground, constitutes an essential functional whole participating in mechanisms of postural control and regulation, allowing the body to sense and interact with the surrounding environment. Among many somatosensory stimulations designed to improve balance, wearing shoe insoles presents one of the easiest and most cost-effective ways. This method can be used both amongst elderly population for fall prevention and amongst athletes to reach better performance and furthermore prevent injuries. With the growing interest in insole use, several prototypes have been developed to monitor movement during day to day use. For therapeutic purposes, the type of insoles used in the studies was often not clarified, and the term insole was used as a general term.The proposed theme of the discussion is to review already existing data on insole use for treatment of postural balance. Keywords: Postural control; Postural sway balance; Insoles; Foot stimulation

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Reports on the topic "Somatosensory nervous system"

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Zheng, Ruo-xiang, Jia-wei Xu, Bi-yao Jiang, Wei Tang, Chun-li Lu, Xiao-yang Hu, and Jian-ping Liu. Mind-body therapies in traditional Chinese medicine for neuropathic pain: a systematic review of randomized controlled trials. INPLASY - International Platform of Registered Systematic Review and Meta-analysis Protocols, April 2022. http://dx.doi.org/10.37766/inplasy2022.4.0016.

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Review question / Objective: The purpose of this review is to comprehensively evaluate the effectiveness and safety on mind-body therapies of traditional Chinese medicine for neuropathic pain. Condition being studied: According to the definition by the International Association for the Study of Pain (IASP), neuropathic pain is a kind of pain caused by lesions or diseases affecting the somatosensory nervous system. It has brought considerable negative impacts on patients and society. Neuropathic pain is a prevalent disease and can be induced by a variety of clinical conditions such as spinal cord injury (prevalence rate: 53%), induced peripheral neuropathic pain (prevalence rate: 38%), diabetic peripheral neuropathic pain (prevalence rate: 10%-26%), chemotherapy postherpetic neuralgia (3.9-42.0/10,000 people per year), prosopalgia (3-5/10,000 people per year), and so on. However, current recommended medicines for neuropathic pain management could cause dependence and adverse events. Thus, alternatives would be helpful for both patients and clinicians. Mind-body therapy in traditional Chinese medicine (TCM) has a long history in clinical practice for relieving pain and their effectiveness has not been systematically reviewed.The purpose of this review is to comprehensively evaluate the effectiveness and safety on mind-body therapies of traditional Chinese medicine for neuropathic pain.

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