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Journal articles on the topic 'Somatosensory nervous system'

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1

KIMURA, AKIO. "Electrophysiological test for nervous system. Somatosensory evoked potential. (SEP)." JOURNAL OF JAPAN SOCIETY FOR CLINICAL ANESTHESIA 8, no. 1 (1988): 11–21. http://dx.doi.org/10.2199/jjsca.8.11.

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2

Singh, Harminder, Richard W. Vogel, Robert M. Lober, et al. "Intraoperative Neurophysiological Monitoring for Endoscopic Endonasal Approaches to the Skull Base: A Technical Guide." Scientifica 2016 (2016): 1–20. http://dx.doi.org/10.1155/2016/1751245.

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Intraoperative neurophysiological monitoring during endoscopic, endonasal approaches to the skull base is both feasible and safe. Numerous reports have recently emerged from the literature evaluating the efficacy of different neuromonitoring tests during endonasal procedures, making them relatively well-studied. The authors report on a comprehensive, multimodality approach to monitoring the functional integrity of at risk nervous system structures, including the cerebral cortex, brainstem, cranial nerves, corticospinal tract, corticobulbar tract, and the thalamocortical somatosensory system du
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3

Katz, Richard T. "Electrodiagnosis of the Peripheral Nervous System: An Introduction." Guides Newsletter 19, no. 3 (2014): 10–14. http://dx.doi.org/10.1001/amaguidesnewsletters.2014.mayjun02.

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Abstract This article is an introduction to electrodiagnosis of the peripheral nervous system, including electromyography, electroneurography (nerve conduction studies), and somatosensory evoked potentials. Electromyography involves the introduction of a special recording needle into a muscle body in search of spontaneous activity (electrical potentials that occur while the muscle is at rest). Three types of spontaneous activity are of greatest relevance: positive sharp waves, fibrillation potentials, and fasciculations. Electromyography can help assess the status of nerve fibers indirectly, b
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4

Sólyom, A., S. Tóth, I. Holczinger, J. Vajda, Z. Tóth, and R. Kálmánchey. "The Spread of Somatosensory-Evoked Potentials Within the Nervous System." Stereotactic and Functional Neurosurgery 48, no. 1-6 (1985): 222–25. http://dx.doi.org/10.1159/000101131.

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5

Fustes, Otto Jesus Hernández, Cláudia Suemi Kamoi Kay, Paulo José Lorenzoni, Renata Dal-Prá Ducci, Lineu Cesar Werneck, and Rosana Herminia Scola. "Somatosensory evoked potentials in clinical practice: a review." Arquivos de Neuro-Psiquiatria 79, no. 9 (2021): 824–31. http://dx.doi.org/10.1590/0004-282x-anp-2020-0427.

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Abstract The authors present a review of the current use of somatosensory evoked potentials (SSEPs) in neurological practice as a non-invasive neurophysiological technique. For this purpose we have reviewed articles published in English or Portuguese in the PubMed and LILACS databases. In this review, we address the role of SSEPs in neurological diseases that affect the central nervous system and the peripheral nervous system, especially in demyelinating diseases, for monitoring coma, trauma and the functioning of sensory pathways during surgical procedures. The latter, along with new areas of
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6

Hao, Han, Rosmaliza Ramli, Caixue Wang, et al. "Dorsal root ganglia control nociceptive input to the central nervous system." PLOS Biology 21, no. 1 (2023): e3001958. http://dx.doi.org/10.1371/journal.pbio.3001958.

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Accumulating observations suggest that peripheral somatosensory ganglia may regulate nociceptive transmission, yet direct evidence is sparse. Here, in experiments on rats and mice, we show that the peripheral afferent nociceptive information in mice undergoes dynamic filtering within the dorsal root ganglion (DRG) and suggest that this filtering occurs at the axonal bifurcations (t-junctions). Using synchronous in vivo electrophysiological recordings from the peripheral and central processes of sensory neurons (in the spinal nerve and dorsal root), ganglionic transplantation of GABAergic proge
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7

Aminoff, Michael J. "Use of Somatosensory Evoked Potentials to Evaluate the Peripheral Nervous System." Journal of Clinical Neurophysiology 4, no. 2 (1987): 135–44. http://dx.doi.org/10.1097/00004691-198704000-00003.

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8

Jeffry, Joseph, Seungil Kim, and Zhou-Feng Chen. "Itch Signaling in the Nervous System." Physiology 26, no. 4 (2011): 286–92. http://dx.doi.org/10.1152/physiol.00007.2011.

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Itch is a major somatic sensation, along with pain, temperature, and touch, detected and relayed by the somatosensory system. Itch can be an acute sensation, associated with mosquito bite, or a chronic condition, like atopic dermatitis ( 29 , 59 ). The origins of the stimulus can be localized in the periphery or systemic, and associated with organ failure or cancer. Itch is also a perception originating in the brain. Itch is broadly characterized as either histamine-dependent (histaminergic) or histamine-independent (nonhistaminergic), both of which are relayed by subsets of C fibers and by th
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9

Fang, Fang, Qian Luo, Ren-Bin Ge, et al. "Decreased Microstructural Integrity of the Central Somatosensory Tracts in Diabetic Peripheral Neuropathy." Journal of Clinical Endocrinology & Metabolism 106, no. 6 (2021): 1566–75. http://dx.doi.org/10.1210/clinem/dgab158.

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Abstract Context Although diabetic peripheral neuropathy (DPN) is predominantly considered a disorder of the peripheral nerves, some evidence for central nervous system involvement has recently emerged. However, whether or to what extent the microstructure of central somatosensory tracts may be injured remains unknown. Objective This work aimed to detect the microstructure of central somatosensory tracts in type 2 diabetic patients and to correlate it with the severity of DPN. Methods A case-control study at a tertiary referral hospital took place with 57 individuals with type 2 diabetes (25 w
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10

Lee, Wang Wei, Yu Jun Tan, Haicheng Yao, et al. "A neuro-inspired artificial peripheral nervous system for scalable electronic skins." Science Robotics 4, no. 32 (2019): eaax2198. http://dx.doi.org/10.1126/scirobotics.aax2198.

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The human sense of touch is essential for dexterous tool usage, spatial awareness, and social communication. Equipping intelligent human-like androids and prosthetics with electronic skins—a large array of sensors spatially distributed and capable of rapid somatosensory perception—will enable them to work collaboratively and naturally with humans to manipulate objects in unstructured living environments. Previously reported tactile-sensitive electronic skins largely transmit the tactile information from sensors serially, resulting in readout latency bottlenecks and complex wiring as the number
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11

Micu, Elena Claudia, and Laszlo Irsay. "The Rehabilitation of Oncological Patients Presenting Neuropathies." Medicine and Pharmacy Reports 87, no. 2 (2014): 67–72. http://dx.doi.org/10.15386/cjmed-278.

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The International Association for the Study of Pain (IASP 2011) defines neuropathic pain as “the pain caused by an injury or disease of the somatosensory portion of the nervous system”. The central neuropathic pain is defined as “the pain caused by an injury or disease of the central somatosensory central nervous system”, whereas the peripheral neuropathic pain is defined as “the pain caused by an injury or disease of the peripheral somatosensory nervous system”. The peripheral neuropathy describes any affection of the peripheral nervous system. The etiology is vast, there being a number of ov
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12

Guilbaud, Gisèle. "Central neurophysiological processing of joint pain on the basis of studies performed in normal animals and in models of experimental arthritis." Canadian Journal of Physiology and Pharmacology 69, no. 5 (1991): 637–46. http://dx.doi.org/10.1139/y91-095.

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On the basis of anatomical and electrophysiological studies, this review summarizes first, the data dealing with the transmission of joint inputs in the central nervous system of normal animals at the spinal and supraspinal levels. It appears that in these conditions neuronal responses to mechanical noxious stimuli of the joints are relatively few and (or) weak. Second, in sharp contrast, the studies performed in polyarthritic rats have emphasized the profound changes in the activities (spontaneous firing and responsiveness) of the somatosensory neurones at various levels of the central nervou
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13

Gomez-Ramirez, Manuel, Kristjana Hysaj, and Ernst Niebur. "Neural mechanisms of selective attention in the somatosensory system." Journal of Neurophysiology 116, no. 3 (2016): 1218–31. http://dx.doi.org/10.1152/jn.00637.2015.

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Selective attention allows organisms to extract behaviorally relevant information while ignoring distracting stimuli that compete for the limited resources of their central nervous systems. Attention is highly flexible, and it can be harnessed to select information based on sensory modality, within-modality feature(s), spatial location, object identity, and/or temporal properties. In this review, we discuss the body of work devoted to understanding mechanisms of selective attention in the somatosensory system. In particular, we describe the effects of attention on tactile behavior and correspo
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14

Nakamura, R., M. Noritake, Y. Hosoda, K. Kamakura, N. Nagata, and H. Shibasaki. "Somatosensory Conduction Delay in Central and Peripheral Nervous System of Diabetic Patients." Diabetes Care 15, no. 4 (1992): 532–35. http://dx.doi.org/10.2337/diacare.15.4.532.

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15

Ackerley, Rochelle, Michael Borich, Calogero Maria Oddo, and Silvio Ionta. "Insights and Perspectives on Sensory-Motor Integration and Rehabilitation." Multisensory Research 29, no. 6-7 (2016): 607–33. http://dx.doi.org/10.1163/22134808-00002530.

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The present review focuses on the flow and interaction of somatosensory-motor signals in the central and peripheral nervous system. Specifically, where incoming sensory signals from the periphery are processed and interpreted to initiate behaviors, and how ongoing behaviors produce sensory consequences encoded and used to fine-tune subsequent actions. We describe the structure–function relations of this loop, how these relations can be modeled and aspects of somatosensory-motor rehabilitation. The work reviewed here shows that it is imperative to understand the fundamental mechanisms of the so
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16

Fox, Kevin, Helen Wallace, and Stanislaw Glazewski. "Is there a thalamic component to experience–dependent cortical plasticity?" Philosophical Transactions of the Royal Society of London. Series B: Biological Sciences 357, no. 1428 (2002): 1709–15. http://dx.doi.org/10.1098/rstb.2002.1169.

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Sensory deprivation and injury to the peripheral nervous system both induce plasticity in the somatosensory system of adult animals, but in different places. While injury induces plasticity at several locations within the ascending somatosensory pathways, sensory deprivation appears only to affect the somatosensory cortex. Experiments have been performed to detect experience–dependent plasticity in thalamic receptive fields, thalamic domain sizes and convergence of thalamic receptive fields onto cortical cells. So far, plasticity has not been detected with sensory deprivation paradigms that ca
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17

Stiefel, Klaus M., Jonathan Tapson, and André van Schaik. "Temporal Order Detection and Coding in Nervous Systems." Neural Computation 25, no. 2 (2013): 510–31. http://dx.doi.org/10.1162/neco_a_00400.

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This letter discusses temporal order coding and detection in nervous systems. Detection of temporal order in the external world is an adaptive function of nervous systems. In addition, coding based on the temporal order of signals can be used as an internal code. Such temporal order coding is a subset of temporal coding. We discuss two examples of processing the temporal order of external events: the auditory location detection system in birds and the visual direction detection system in flies. We then discuss how somatosensory stimulus intensities are translated into a temporal order code in
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18

Fox, Kevin. "Experience-dependent plasticity mechanisms for neural rehabilitation in somatosensory cortex." Philosophical Transactions of the Royal Society B: Biological Sciences 364, no. 1515 (2008): 369–81. http://dx.doi.org/10.1098/rstb.2008.0252.

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Functional rehabilitation of the cortex following peripheral or central nervous system damage is likely to be improved by a combination of behavioural training and natural or therapeutically enhanced synaptic plasticity mechanisms. Experience-dependent plasticity studies in the somatosensory cortex have begun to reveal those synaptic plasticity mechanisms that are driven by sensory experience and might therefore be active during behavioural training. In this review the anatomical pathways, synaptic plasticity mechanisms and structural plasticity substrates involved in cortical plasticity are e
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19

Elbert, Thomas, Herta Flor, Niels Birbaumer, et al. "Extensive reorganization of the somatosensory cortex in adult humans after nervous system injury." NeuroReport 5, no. 18 (1994): 2593–97. http://dx.doi.org/10.1097/00001756-199412000-00047.

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20

Pasluosta, Cristian, Patrick Kiele, and Thomas Stieglitz. "Paradigms for restoration of somatosensory feedback via stimulation of the peripheral nervous system." Clinical Neurophysiology 129, no. 4 (2018): 851–62. http://dx.doi.org/10.1016/j.clinph.2017.12.027.

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21

Cole, Daniel J., Delia M. Lin, John C. Drummond, and Harvey M. Shapiro. "Spinal tetracaine decreases central nervous system metabolism during somatosensory stimulation in the rat." Canadian Journal of Anaesthesia 37, no. 2 (1990): 231–37. http://dx.doi.org/10.1007/bf03005475.

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22

Ivanichev, G. A., and A. V. Ovchinnikov. "Dinamics of somatosensory generated potentials in myofascial pain syndromes." Kazan medical journal 82, no. 5 (2001): 336–40. http://dx.doi.org/10.17816/kazmj84059.

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In pronounced pain syndrome the most important functional shifts (the generators of pathologically increased excitement) by recording somatosensory generated potentials arc revealed at higher levels than in slight tenderness. At the same time in slight painfulness tenderness a great number of the generators of mild activity arc found, showing insufficient activity of antinociceptive system. After treatment a number of the generators of pathologically increased excitement decrease, at the same time the migration to the caudal parts of nervous system occurs. The neurophysiologic basis for possib
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23

Yakimova, Natalya L., Vladimir A. Pankov, Aleksandr V. Lizarev, et al. "Neurophysiological and morphological effects in the post-exposure vibration period during experimental modeling." Russian Journal of Occupational Health and Industrial Ecology, no. 5 (May 31, 2019): 284–90. http://dx.doi.org/10.31089/1026-9428-2019-59-5-284-290.

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Introduction. Vibration disease continues to occupy one of the leading places in the structure of professional pathology. In workers after the termination of contact with vibration generalization and progression of violations in an organism is noted. The pathogenetic mechanisms of the progredient course of disturbances in the nervous system in the post-contact period of vibration exposure remain insufficiently studied.The aim of the study was to test an experimental model of vibration exposure to assess the neurophysiological and morphological effects of vibration in rats in the dynamics of th
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24

Matsumura, Masashi, and Toshihisa Murofushi. "Vestibular Rehabilitation after Vestibulopathy Focusing on the Application of Virtual Reality." Journal of Otorhinolaryngology, Hearing and Balance Medicine 2, no. 2 (2021): 5. http://dx.doi.org/10.3390/ohbm2020005.

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Human postural control is regulated by the vestibular, somatosensory, and visual systems. These types of sensory information are integrated in the central nervous system to ascertain the body’s position in space. Proper functioning of the vestibular, somatosensory, and visual senses is necessary for the body to maintain equilibrium. Bilateral vestibulopathy (BVP) is a condition in which bilateral peripheral vestibular function is reduced. Its treatment includes vestibular rehabilitation (VeR), balance training, counseling, treating the underlying cause, and avoiding further damage to the vesti
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Uppal, Neha, John J. Foxe, John S. Butler, Frantzy Acluche, and Sophie Molholm. "The neural dynamics of somatosensory processing and adaptation across childhood: a high-density electrical mapping study." Journal of Neurophysiology 115, no. 3 (2016): 1605–19. http://dx.doi.org/10.1152/jn.01059.2015.

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Young children are often hyperreactive to somatosensory inputs hardly noticed by adults, as exemplified by irritation to seams or labels in clothing. The neurodevelopmental mechanisms underlying changes in sensory reactivity are not well understood. Based on the idea that neurodevelopmental changes in somatosensory processing and/or changes in sensory adaptation might underlie developmental differences in somatosensory reactivity, high-density electroencephalography was used to examine how the nervous system responds and adapts to repeated vibrotactile stimulation over childhood. Participants
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Aminoff, Michael J. "The Use of Somatosensory Evoked Potentials in the Evaluation of the Central Nervous System." Neurologic Clinics 6, no. 4 (1988): 809–23. http://dx.doi.org/10.1016/s0733-8619(18)30844-2.

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27

Eisen, Andrew. "The Use of Somatosensory Evoked Potentials for the Evaluation of the Peripheral Nervous System." Neurologic Clinics 6, no. 4 (1988): 825–38. http://dx.doi.org/10.1016/s0733-8619(18)30845-4.

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28

Strijers, R. L. M., S. L. Visser, and E. J. Jonkman. "Central nervous system involvement in uremia; a study using visual and somatosensory evoked potentials." Electroencephalography and Clinical Neurophysiology 87, no. 2 (1993): S72. http://dx.doi.org/10.1016/0013-4694(93)91175-z.

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29

Suresh, Aneesha K., Jeremy E. Winberry, Christopher Versteeg, et al. "Methodological considerations for a chronic neural interface with the cuneate nucleus of macaques." Journal of Neurophysiology 118, no. 6 (2017): 3271–81. http://dx.doi.org/10.1152/jn.00436.2017.

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While the response properties of neurons in the somatosensory nerves and anterior parietal cortex have been extensively studied, little is known about the encoding of tactile and proprioceptive information in the cuneate nucleus (CN) or external cuneate nucleus (ECN), the first recipients of upper limb somatosensory afferent signals. The major challenge in characterizing neural coding in CN/ECN has been to record from these tiny, difficult-to-access brain stem structures. Most previous investigations of CN response properties have been carried out in decerebrate or anesthetized animals, thereb
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30

Suarez-Roca, Heberto, Rebecca Y. Klinger, Mihai V. Podgoreanu, et al. "Contribution of Baroreceptor Function to Pain Perception and Perioperative Outcomes." Anesthesiology 130, no. 4 (2019): 634–50. http://dx.doi.org/10.1097/aln.0000000000002510.

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Abstract Baroreceptors are mechanosensitive elements of the peripheral nervous system that maintain homeostasis by coordinating physiologic responses to external and internal stimuli. While it is recognized that carotid and cardiopulmonary baroreceptor reflexes modulate autonomic output to mitigate excessive fluctuations in arterial blood pressure and to maintain intravascular volume, increasing evidence suggests that baroreflex pathways also project to key regions of the central nervous system that regulate somatosensory, somatomotor, and central nervous system arousal. In addition to maintai
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31

Vareshniuk, O. V., and V. V. Vyun. "Clinical-neurological features of prematurely born children with hypoxic-ischemic lesions of the central nervous system." Медицина сьогодні і завтра 85, no. 4 (2019): 55–60. http://dx.doi.org/10.35339/msz.2019.85.04.08.

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The clinical and neurological features of prematurely born children with perinatal hypoxic-ischemic lesions of the central nervous system were studied. A comprehensive examination of 79 preterm infants of both sexes who underwent perinatal hypoxic-ischemic lesions of the central nervous system was performed. 37.9 % of the surveyed were diagnosed with spastic cerebral palsy, 20.3 % were diagnosed with spastic diplegia, 27.8 % were diagnosed with childhood hemiplegia, hydrocephalus was acquired in 13.9 % as a result of non-traumatic intraventricular hemorrhage. In 10.3 % of children, severe move
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32

Rega, Dalia, Mika Aiko, Nicolás Peñaranda, et al. "Patients with Minimal Hepatic Encephalopathy Show Altered Thermal Sensitivity and Autonomic Function." Journal of Clinical Medicine 10, no. 2 (2021): 239. http://dx.doi.org/10.3390/jcm10020239.

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Cirrhotic patients may experience alterations in the peripheral nervous system and in somatosensory perception. Impairment of the somatosensory system could contribute to cognitive and motor alterations characteristic of minimal hepatic encephalopathy (MHE), which affects up to 40% of cirrhotic patients. We assessed the relationship between MHE and alterations in thermal, vibration, and/or heat pain sensitivity in 58 cirrhotic patients (38 without and 20 with MHE according to Psychometric Hepatic Encephalopathy Score) and 39 controls. All participants underwent attention and coordination tests
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Rega, Dalia, Mika Aiko, Nicolás Peñaranda, et al. "Patients with Minimal Hepatic Encephalopathy Show Altered Thermal Sensitivity and Autonomic Function." Journal of Clinical Medicine 10, no. 2 (2021): 239. http://dx.doi.org/10.3390/jcm10020239.

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Cirrhotic patients may experience alterations in the peripheral nervous system and in somatosensory perception. Impairment of the somatosensory system could contribute to cognitive and motor alterations characteristic of minimal hepatic encephalopathy (MHE), which affects up to 40% of cirrhotic patients. We assessed the relationship between MHE and alterations in thermal, vibration, and/or heat pain sensitivity in 58 cirrhotic patients (38 without and 20 with MHE according to Psychometric Hepatic Encephalopathy Score) and 39 controls. All participants underwent attention and coordination tests
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34

Verley, R. "Reorganization of the Somatosensory Cerebral CortexAfter Peripheral Damage." Physiology 1, no. 1 (1986): 15–19. http://dx.doi.org/10.1152/physiologyonline.1986.1.1.15.

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If a sensory nerve is sectioned, it is expected that the centrally deprived projection area should be reduced to silence and atrophy. However, in recent years the capacity of the central nervous system for reorganization has received increasing attention. This article concerns elimination of input from the tactile system on the head of rodents, which has two components, the long vibrissae and the common fur. After early destruction of the vibrissae in newborn rats, the deprived cortex unexpectedly was not significantly reduced because substitution occurred. If only the vibrissae were destroyed
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35

Ward, Harry, and Steven J. West. "Microglia: sculptors of neuropathic pain?" Royal Society Open Science 7, no. 6 (2020): 200260. http://dx.doi.org/10.1098/rsos.200260.

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Neuropathic pain presents a huge societal and individual burden. The limited efficacy of current analgesics, diagnostic markers and clinical trial outcome measures arises from an incomplete understanding of the underlying mechanisms. A large and growing body of evidence has established the important role of microglia in the onset and possible maintenance of neuropathic pain, and these cells may represent an important target for future therapy. Microglial research has further revealed their important role in structural remodelling of the nervous system. In this review, we aim to explore the evi
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36

Rusanova, Dina V., Oleg L. Lakhman, Natalya V. Slivnitsyna, and Alla N. Kuks. "State of central and peripheral conducting structures in patients with vibration disease, burdened with metabolic syndrome." Hygiene and sanitation 99, no. 10 (2020): 1093–99. http://dx.doi.org/10.47470/0016-9900-2020-99-10-1093-1099.

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Introduction. Vibration disease (VD) is associated with an imbalance of endocrine regulation, the development of metabolic syndrome (MS), which leads to the development of neurological complications. One of the reasons is microcirculation disorders that involve the CNS and peripheral nerves in the pathological process. The aim of the study is to provide a comprehensive assessment of the state of peripheral nerves and afferent pathways in workers with VD, and patients with VD, burdened with MS. Material and methods. We examined: 41 patients with VD associated with combined local and general vib
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Dmytriiev, Dmytro, Pylyp Prudius, Olesia Zaletskaya, Yevhen Lisak, Yurii Rudnitsky, and Nadiia Korenchuk. "Neuropathic pain: mechanisms of development, principles of diagnostics and treatment." Pain medicine 4, no. 2 (2019): 4–32. http://dx.doi.org/10.31636/pmjua.v4i2.1.

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Neuropathic pain is a pain caused by a disease or focal damage to the somatosensory nervous system. The prevalence of chronic pain with neuropathic features in different countries is estimated at 7–10 %. Damages to the nervous system can occur at the level of peripheral nerves, plexus and dorsal roots (peripheral neuropathic pain) or spinal cord and brain (central neuropathic pain). Neuropathic pain is based on pathological activation of pain pathways. Neuropathic pain occurs with diabetic polyneuropathy more often than with all polyneuropathies of another etiology. Hyperglycemia is the major
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Ivica, Nedjeljka, Luciano Censoni, Joel Sjöbom, Ulrike Richter, and Per Petersson. "Differential effects of skilled reaching training on the temporal and spatial organization of somatosensory input to cortical and striatal motor circuits." Journal of Neurophysiology 127, no. 1 (2022): 225–38. http://dx.doi.org/10.1152/jn.00464.2021.

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Sensorimotor transformations are fundamental to the function of the nervous system and determine how patterns of sensory input are converted into appropriate movements. We here investigated the extent to which experience-dependent processes can reshape the organization of somatosensory input feeding into cortico-basal ganglia motor structures. Our data point to a particularly important role for the primary motor cortex in the functional adaptions associated with skilled motor learning.
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Giblin, Denis R. "SOMATOSENSORY EVOKED POTENTIALS IN HEALTHY SUBJECTS AND IN PATIENTS WITH LESIONS OF THE NERVOUS SYSTEM*†." Annals of the New York Academy of Sciences 112, no. 1 (2006): 93–142. http://dx.doi.org/10.1111/j.1749-6632.1964.tb26744.x.

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40

Urasaki, E. "Effect of transcutaneous electrical nerve stimulation (TENS) on central nervous system amplification on somatosensory input." Electroencephalography and Clinical Neurophysiology 103, no. 1 (1997): 186. http://dx.doi.org/10.1016/s0013-4694(97)88878-9.

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41

Urasaki, Eiichirou, Shin-ichi Wada, Hideoki Yasukouchi, and Akira Yokota. "Effect of transcutaneous electrical nerve stimulation (TENS) on central nervous system amplification of somatosensory input." Journal of Neurology 245, no. 3 (1998): 143–48. http://dx.doi.org/10.1007/s004150050194.

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42

Catania, Kenneth C. "The sense of touch in the star-nosed mole: from mechanoreceptors to the brain." Philosophical Transactions of the Royal Society B: Biological Sciences 366, no. 1581 (2011): 3016–25. http://dx.doi.org/10.1098/rstb.2011.0128.

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Star-nosed moles are somatosensory specialists that explore their environment with 22 appendages that ring their nostrils. The appendages are covered with sensory domes called Eimer's organs. Each organ is associated with a Merkel cell–neurite complex, a lamellated corpuscle, and a series of 5–10 free nerve endings that form a circle of terminal swellings. Anatomy and electrophysiological recordings suggest that Eimer's organs detect small shapes and textures. There are parallels between the organization of the mole's somatosensory system and visual systems of other mammals. The centre of the
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Forouzanfar, Fatemeh, and Hamid R. Sadeghnia. "Fibroblast Growth Factors as Tools in the Management of Neuropathic Pain Disorders." Current Drug Targets 21, no. 10 (2020): 1034–43. http://dx.doi.org/10.2174/1389450121666200423084205.

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Neuropathic pain is caused by a damage to or dysfunction of the somatosensory nervous system. The main mechanisms underlying neuropathic pain include ectopic activity in nociceptive nerves, peripheral and central sensitization, impaired inhibitory modulation, and microglial activation. Fibroblast growth factors (FGFs) make up a large family of growth factors that mediate neural development, metabolism, and function through three main key signaling pathways, including RAS/MAP kinase pathway, PI3 kinase/Akt pathway, and PLCγ. An association between the members of the FGF system and the
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Crawford, LaTasha K., and Michael J. Caterina. "Functional Anatomy of the Sensory Nervous System: Updates From the Neuroscience Bench." Toxicologic Pathology 48, no. 1 (2019): 174–89. http://dx.doi.org/10.1177/0192623319869011.

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The simple tripartite classification of sensory neurons as A-beta, A-delta, and C fibers fails to convey the complexity of the neurons that encode stimuli as diverse as the texture of a surface, the location of a pinprick, or the direction of hair movement as a breeze moves across the skin. It has also proven to be inadequate when investigating the molecular mechanisms underlying pain, which can encompass any combination of chemical, tactile, and thermal modalities. Beginning with a brief overview of visceral and sensory neuroanatomy, this review expands upon sensory innervation of the skin as
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Takács, Szabolcs, Sarolta Bankó, and András Papp. "Effect of mitochondrial toxins on evoked somatosensory activity in rats." Open Life Sciences 5, no. 3 (2010): 293–98. http://dx.doi.org/10.2478/s11535-010-0006-4.

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AbstractMitochondrial toxins represent an interesting group of neurotoxicants related both to causation and modelling of central nervous damage. 3-nitropropionic acid (3NP), a neurotoxin of herbal and microfungal origin, inhibits succinate dehydrogenase leading thereby to various biochemical and morphological alterations in the brain. Experimental animals treated by 3NP are used to model Huntington’s disease. Manganese, often present in occupational settings and as environmental pollutant, inhibits complex II and III of the mitochondria and is known to cause Parkinson-like CNS damage. In this
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Lai, Tsu-Hsien, Bing-Wen Soong, Jen-Tse Chen, et al. "Multimodal Evoked Potentials of Kennedy's Disease." Canadian Journal of Neurological Sciences / Journal Canadien des Sciences Neurologiques 34, no. 3 (2007): 328–32. http://dx.doi.org/10.1017/s0317167100006764.

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Background:Kennedy's disease (KD) is an X-linked recessive polyglutamine disease. Traditionally, it is a lower motor neuron syndrome with additional features such as gynecomastia and tremor. Sensory symptoms are minimal if ever present. We used multimodal evoked potential (EPs) tests to study the distribution of the involvement of the disease.Methods:Visual, brainstem auditory, somatosensory and motor EPs were studied in six KD patients. All of them had typical presentations and had been proved genetically.Results:Abnormal findings were noted as follows: prolonged peak latencies of visual EPs,
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Barbosa-Torres, Carlos, and Sixto Cubo-Delgado. "Efficacy of the SMR protocol in women with fibromyalgia for the improvement of chronic pain, sleep, and quality of life." Behavioral Psychology/Psicología Conductual 29, no. 3 (2021): 549–60. http://dx.doi.org/10.51668/bp.8321302n.

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The objective of the study was to analyze how the brain shaping provided by the sensorimotor rhythm protocol (SMR), applied on somatosensory areas, affects pain, sleep and the quality of life in women with fibromyalgia. Thirty-seven women with fibromyalgia who received an SMR protocol in 20 sessions participated and were evaluated before and after treatment. The data showed an increase in the amplitude of the SMR (p= .026) and a decrease in the amplitude of the theta band (p= .011) in the somatosensory cortex after the application of therapy, which caused an increase in the SMR/theta ratio (p=
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Cheng, Chia-Hsiung, Pei-Ying S. Chan, Sylvain Baillet, and Yung-Yang Lin. "Age-Related Reduced Somatosensory Gating Is Associated with Altered Alpha Frequency Desynchronization." Neural Plasticity 2015 (2015): 1–9. http://dx.doi.org/10.1155/2015/302878.

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Sensory gating (SG), referring to an attenuated neural response to the second identical stimulus, is considered as preattentive processing in the central nervous system to filter redundant sensory inputs. Insufficient somatosensory SG has been found in the aged adults, particularly in the secondary somatosensory cortex (SII). However, it remains unclear which variables leading to the age-related somatosensory SG decline. There has been evidence showing a relationship between brain oscillations and cortical evoked excitability. Thus, this study used whole-head magnetoencephalography to record r
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Filatova, E. S., A. M. Lila, and V. A. Parfenov. "Neuropathic pain in rheumatoid arthritis." Neurology, Neuropsychiatry, Psychosomatics 12, no. 5 (2020): 60–65. http://dx.doi.org/10.14412/2074-2711-2020-5-60-65.

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Objective: to identify the signs of neuropathic pain (NP) in patients with rheumatoid arthritis (RA) on the basis of the PainDETECT questionnaire and neurological examination.Patients and methods. A total of 208 RA patients (39 men and 169 women; mean age, 47.7 years) with chronic pain syndrome were examined. The patients underwent rheumatological and neurological examinations; NP was diagnosed using the PainDETECT questionnaire; inflammation severity (DAS28 index), pain intensity (VAS), affective disorders (HADS), and quality of life (EQ-5D) were assessed.Results and discussion. 172 (82.7%) p
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Brooks, Charles N. "Impairment Tutorial: Rating Sensory and Motor Deficits of the Upper Extremity." Guides Newsletter 5, no. 2 (2000): 5–7. http://dx.doi.org/10.1001/amaguidesnewsletters.2000.marapr04.

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Abstract The three components of electrodiagnosis useful in evaluation of the peripheral nervous system and spinal cord include electromyography (EMG), electroneurography (nerve conduction studies), and somatosensory evoked potentials. EMG examination involves introduction of a special recording needle into a muscle belly. Electrical potentials located within a few millimeters of the needle are picked up by an electrode and are transmitted from the muscle to amplifiers that filter and display results visually for the electromyographer. Three types of spontaneous activity in electrical potentia
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