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Books on the topic 'Spinal cord injured users'

Author: Grafiati

Published: 4 June 2021

Last updated: 4 February 2022

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1

Borgens, Richard Ben. Restoring Function to the Injured Human Spinal Cord. Berlin, Heidelberg: Springer Berlin Heidelberg, 2003. http://dx.doi.org/10.1007/978-3-642-59361-1.

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2

Leyson, Jose Florante J., ed. Sexual Rehabilitation of the Spinal-Cord-Injured Patient. Totowa, NJ: Humana Press, 1991. http://dx.doi.org/10.1007/978-1-4612-0467-1.

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3

Rosenfeld, Marla. Vocational counselling and the inpatient spinal cord injured population. [Hamilton, Ont.]: McMaster University, 1988.

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4

Lee, Amy E. Experiences of injured workers. [Austin, Tex.]: Research and Oversight Council on Workers' Compensation, 1997.

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5

McKenna, Monica Erin. The experience of a spinal cord injured person in the acute setting. Ottawa: National Library of Canada, 1994.

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6

Donovan, William H. Justification for the prescription of durable medical equipment for spinal cord injured persons. Chicago, Ill: American Spinal Injury Association, 1986.

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7

Virginia. General Assembly. Joint Subcommittee Studying the Needs of Head and Spinal Cord Injured Citizens and the Need for Research Pursuant to House Joint Resolution No. 135. Report of the Joint Subcommittee Studying the Needs of Head and Spinal Cord Injured Citizens and the Need for Research Pursuant to House Joint Resolution No. 135: To the Governor and the General Assembly of Virginia. Richmond, Va: Commonwealth of Virginia, 1989.

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8

Virginia. General Assembly. Joint Subcommittee Studying the Needs of Head and Spinal Cord Injured Citizens, the Need for Research, and the Needs of All Physically Handicapped Persons. Report of the Joint Subcommittee Studying the Needs of Head and Spinal Cord Injured Citizens, the Need for Research, and the Needs of All Physically Handicapped Persons to the governor and the General Assembly of Virginia. Richmond: Commonwealth of Virginia, 1991.

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9

1928-, Lee Bok Y., and Ostrander Lee E, eds. The spinal cord injured patient. 2nd ed. New York: Demos, 2002.

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10

Lee, Bok Y. Spinal Cord Injured Patient: Comprehensive Management. W.B. Saunders Company, 1991.

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11

1928-, Lee Bok Y., ed. The Spinal cord injured patient: Comprehensive management. Philadelphia: Saunders, 1991.

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12

(Editor), Bok Y. Lee, and Lee E., Ph.D. Ostrander (Editor), eds. The Spinal Cord Injured Patient, 2nd Edition. 2nd ed. Demos Medical Publishing, 2001.

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13

J, Leyson Jose Florante, ed. Sexual rehabilitation of the spinal-cord-injured patient. Clifton, N.J: Humana Press, 1991.

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14

Keane, Anne Mary. Spasticity and electrical stimulation in the spinal cord injured. 1994.

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15

The effect of body position on spinal cord injured swimmers. 1990.

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16

Physiotherapy management in the rehabilitation of the spinal cord injured. 3rd ed. Toronto, Ont: Lyndhurst, The Spinal Cord Centre, Physiotherapy Dept., 1995.

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17

Inniss, Patrick George. A STUDY OF DAILY LIFE ACTIVITIES OF SPINAL CORD-INJURED WOMEN. 1994.

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18

Gupta, Dimpy. Fast-gelling injectable drug delivery system for the injured spinal cord. 2005.

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19

Circulorespiratory responses of spinal cord injured, quadriplegic men to dynamic physical exercise. 1987.

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20

Payne, Janis A. CONTRIBUTIONS OF GROUP LEARNING IN THE REHABILITATION OF SPINAL CORD INJURED ADULTS. 1991.

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21

Circulorespiratory responses of spinal cord injured, quadriplegic men to dynamic physical exercise. 1989.

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22

Jones, Catherine Allyson. The behaviour of spinal reflexes evoked by cutaneous stimuli during walking in incomplete spinal cord injured subjects. UMI, 1992.

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23

Fode, Mikkel, and Jens Sønksen. The management of fertility in spinal cord injury. Edited by David John Ralph. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199659579.003.0100.

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While spinal cord injury (SCI) does not affect female fertility, the condition most often results in infertility in males due to anejaculation and reduced semen quality. Anejaculation is caused by disruption of the autonomic nerve fibres, which are normally responsible for the ejaculation. The reason for the poor sperm quality has not been firmly established. If spinal cord injured men cannot ejaculate by sexual intercourse or masturbation, ejaculation can be induced by either penile vibratory stimulation or electroejaculation. Only if these methods fail should surgical sperm retrieval be considered. The method of insemination depends largely on the total motile sperm count and patient preference. With the right treatment, it is possible for most SCI men to have children.

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24

Hamann, Maria Claudia Jimenez. A novel strategy for localized delivery of therapeutic agents to the injured spinal cord. 2004.

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25

Hurlbert, R. John. An evaluation of direct current stimulation in the normal and injured rodent spinal cord. 1993.

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26

Finfer, Simon, and Oliver Flower. Assessment and immediate management of spinal cord injury. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0344.

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Spinal cord injury is a potentially devastating injury, which may occur in isolation, but more commonly occurs in the setting of multiple injuries. Motor vehicle accidents and falls are the most common causes. Depending on the level of the injury and its completeness, patients may be left with paraplegia or tetraplegia. The injury may be immediately obvious based on history and clinical examination, but may have to be actively excluded in multiply-injured patients. Thoracolumbar spine fractures are almost always evident on plain X-rays, whereas computed tomography (CT) or magnetic resonance imaging (MRI) is frequently required to exclude cervical spine injuries. Immediate management should be directed at the detection and treatment of life-threatening injuries. Patients should be transferred to a facility specializing in the management of spinal cord injury as soon as feasible. Acute management of the spinal injury itself is largely supportive and aimed at avoiding preventable secondary injury. Respiratory complications are common, and high thoracic or cervical injuries may lead to neurogenic shock. Early identification of the injury and appropriate management results in improved outcome, reducing disability and costs of long-term management.

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27

Deutsch. Life Care Planning for the Spinal Cord Injured: A Step by Step Guide (Life Care). CRC, 1989.

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28

(Illustrator), K. J. Harrington, ed. Restoring Function to the Injured Human Spinal Cord (Advances in Anatomy, Embryology and Cell Biology). Springer, 2003.

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29

Daniels, Richard Donald. EXPLORING THE SELF-CARE VARIABLES THAT EXPLAIN A WELLNESS LIFESTYLE IN SPINAL CORD INJURED WHEELCHAIR BASKETBALL ATHLETES. 1994.

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30

Sullivan, Maureen Patricia. THE RELATIONSHIP OF THE SENSE OF COHERENCE AND LONELINESS TO PSYCHOSOCIAL ADJUSTMENT IN SPINAL CORD INJURED INDIVIDUALS. 1995.

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31

Lucke, Kathleen T. THE MEANING, PROCESS, AND CONSEQUENCES OF NURSE CARING AS PERCEIVED BY SPINAL CORD INJURED INDIVIDUALS DURING REHABILITATION. 1995.

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32

Pressure sore prevention self-efficacy and outcome expectations in the spinal cord-injured: A validity and reliability study. 1992.

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33

Basta, Susan Marie. PRESSURE SORE PREVENTION SELF-EFFICACY AND OUTCOME EXPECTATIONS IN THE SPINAL CORD-INJURED: A VALIDITY AND RELIABILITY STUDY. 1992.

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34

Kalsi-Ryan, Sukhvinder. The Quadriplegia Hand Assessment Tool (Q-HAT): The development of a clinical assessment measure of the hand for the cervical spinal cord injured individual. 2006.

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35

Cavanna, Andrea E. Gabapentin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198791577.003.0006.

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Gabapentin is a second-generation antiepileptic drug characterized by few antiepileptic indications, with very good interaction profile in polytherapy. The therapeutic indications of gabapentin for the treatment of epileptic seizures have been largely overshadowed by its widespread use for the treatment of neuropathic pain (especially post-herpetic neuralgia, diabetic neuropathy, and pain caused by a spinal cord injury). Gabapentin has a good behavioural tolerability profile and a good range of psychiatric uses (unlicensed indications for anxiety disorders and alcohol withdrawal symptoms). Despite the widespread use of gabapentin for behavioural conditions, its potential usefulness as adjunctive treatment of bipolar affective disorder is still controversial.

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36

Cavanna, Andrea E. Pregabalin. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198791577.003.0011.

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Pregabalin is a second-generation antiepileptic drug characterized by few antiepileptic indications, with a very good interaction profile in polytherapy. The therapeutic indications of pregabalin for the treatment of epileptic seizures have been largely overshadowed by its widespread use for the treatment of anxiety disorders (especially generalized anxiety disorder and social anxiety disorder) and neuropathic pain (especially post-herpetic neuralgia, diabetic neuropathy, and pain caused by a spinal cord injury). Pregabalin has a very good behavioural tolerability profile and a wide range of psychiatric uses. Specifically, it has a licensed indication for generalized anxiety disorder for which it is currently widely prescribed.

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37

Black, Sheila. The original description of central sensitization. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0040.

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The landmark study discussed in this chapter is ‘The contribution of excitatory amino acids to central sensitization and persistent nociception after formalin-induced tissue injury’, published by Coderre and Melzack in 1992. Previous studies in this field implicate a contribution of excitatory amino acids (EAAs), specifically l-glutamate and l-aspartate, to injury-induced sensitization of nociceptive responses in the dorsal horn of the spinal cord. Repetitive stimulation of primary afferent fibres demonstrated that l-glutamate and NMDA can produce ‘wind-up’ of neuronal dorsal horn activity, and this is blocked by application of NMDA antagonists. This study uses the formalin test as a behavioural model to investigate the mechanisms underlying central sensitization and the role of EAAs, NMDA, their receptors, and their antagonists in this process.

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38

Ellis, Kathryn. A Brief Overview of the Effect of War Injuries on Sexual Health and Intimacy. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190461508.003.0001.

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This chapter establishes definitions of sexual health and intimacy, provides insight into how they interact and affect successful relationships, and communicates the importance of addressing the sexual and intimacy needs of injured service members and their partners. Common deployment-related diagnoses, including post-traumatic stress disorder, traumatic brain injury, orthopedic injuries, spinal cord injuries, genital injuries, burns, and facial injuries, and their possible corresponding limitations are reviewed, along with how such limitations can result in sexual avoidance and intimacy avoidance, thus leading to more relationship issues and poor quality of life. The experience of partners, including perceptions about the dependence of the injured service member, is discussed as well.

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39

Abcejo, Arnoley S., and Jeffrey J. Pasternak. Neurogenic Shock. Edited by Matthew D. McEvoy and Cory M. Furse. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190226459.003.0072.

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Neurogenic shock is a pathophysiologic state of systemic hypoperfusion characterized by a significant decrease in systemic vascular resistance secondary to loss of sympathetic tone. Neurogenic shock is most commonly seen in the setting of acute spinal cord injury (SCI) but can also occur following significant brain injury. Interruption of sympathetic fibers causes loss of basal vascular sympathetic tone, commonly allowing unopposed parasympathetic tone. As a result, severe hypotension and bradycardia can further exacerbate neurologic injury and organ perfusion. Understanding the physiologic and anatomic changes of neurogenic shock can help direct appropriate resuscitation efforts. Physiologic goals should focus on reversing hypotension, preventing hypoxia, and optimizing perfusion of the injured central nervous system and other critical organs.

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40

Subhas, Kamalakkannan, and Martin Smith. Intensive care management after neurosurgery. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0369.

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The post-operative management of neurosurgical patients is directed towards the prevention, prompt detection, and management of surgical complications, and other factors that put the brain or spinal cord at risk. Close monitoring is required in the first 6–12 post-operative hours as deterioration in clinical status is usually the first sign of a potentially fatal complication. The majority of patients do not require complex monitoring or management beyond the first 12 hours after elective surgery, although prolonged intensive care unit management may be required for those who develop complications, or after acute brain injury. Cardiovascular and respiratory disturbances adversely affect the injured or ‘at risk’ brain, and meticulous blood pressure control and prevention of hypoxia are key aspects of management. Hypertension is particularly common after intracranial neurosurgery and may cause complications, such as intracranial bleeding and cerebral oedema, or be a consequence of them. A moderate target for glycaemic control (7.0–10 mmol/L) is recommended, avoiding hypoglycaemia and large swings in blood glucose concentration. Pain, nausea, and vomiting occur frequently after neurosurgery, and a multimodal approach to pain management and anti-emesis is recommended. Adequate analgesia not only ensures patient comfort, but also avoids pain-related hypertension. Disturbances of sodium and water homeostasis can lead to serious complications, and a structured approach to diagnosis and management minimizes adverse outcomes. Post-operative seizures must be brought rapidly under control because of the risks of secondary cerebral damage and/or progression to status epilepticus.

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41

Schaible, Hans-Georg, and Rainer H. Straub. Pain neurophysiology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0059.

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Physiological pain is evoked by intense (noxious) stimuli acting on healthy tissue functioning as a warning signal to avoid damage of the tissue. In contrast, pathophysiological pain is present in the course of disease, and it is often elicited by low-intensity stimulation or occurs even as resting pain. Causes of pathophysiological pain are either inflammation or injury causing pathophysiological nociceptive pain or damage to nerve cells evoking neuropathic pain. The major peripheral neuronal mechanism of pathophysiological nociceptive pain is the sensitization of peripheral nociceptors for mechanical, thermal and chemical stimuli; the major peripheral mechanism of neuropathic pain is the generation of ectopic discharges in injured nerve fibres. These phenomena are created by changes of ion channels in the neurons, e.g. by the influence of inflammatory mediators or growth factors. Both peripheral sensitization and ectopic discharges can evoke the development of hyperexcitability of central nociceptive pathways, called central sensitization, which amplifies the nociceptive processing. Central sensitization is caused by changes of the synaptic processing, in which glial cell activation also plays an important role. Endogenous inhibitory neuronal systems may reduce pain but some types of pain are characterized by the loss of inhibitory neural function. In addition to their role in pain generation, nociceptive afferents and the spinal cord can further enhance the inflammatory process by the release of neuropeptides into the innervated tissue and by activation of sympathetic efferent fibres. However, in inflamed tissue the innervation is remodelled by repellent factors, in particular with a loss of sympathetic nerve fibres.

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