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Books on the topic 'Spinal cord Sex role'

Author: Grafiati

Published: 4 June 2021

Last updated: 13 February 2022

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1

Hooper, Mandy. Sexuality and spinal cord injury. Spinal Injuries Association, 1995.

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2

Ducharme, Stanley H. Sexuality after spinal cord injury: Answers to your questions. Paul H. Brookes Pub., 1997.

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3

White, Suzanne. Women, sexuality and spinal cord injury: Women living with the challenge of spinal cord injury speak out on issues of sexual and reproductive health. Edited by Crump Judith M, Raven Pauline 1954-, and Planned Parenthood Association of Nova Scotia. Planned Parenthood Association of Nova Scotia, 1993.

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4

Baer, Robert W. Is Fred dead?: A manual on sexuality for men with spinal cord injuries. Dorrance Pub. Co., 2003.

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5

Eichner, McDonald Sylvia, ed. Sexuality and spinal cord injury. SpinalCord Injury Center, Froedtert Memorial Lutheran Hospital, 1993.

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6

C, Polosa, and Weaver Lynne C. 1945-, eds. Autonomic dysfunction after spinal cord injury. Elsevier, 2006.

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7

J, Leyson Jose Florante, ed. Sexual rehabilitation of the spinal-cord-injured patient. Humana Press, 1991.

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8

University of Ottawa Evidence-based Practice Center. and United States. Agency for Healthcare Research and Quality., eds. Sexuality and reproductive health following spinal cord injury. Agency for Healthcare Research and Quality, Public Health Service, U.S. Dept. of Health and Human Services, 2004.

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9

Stanley H., Ph.D. Ducharme and Kathleen M., Ph.D. Gill. Sexuality After Spinal Cord Injury : Answers to Your Questions. Brookes Publishing Company, 1996.

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10

Cassidy, Jim, Donald Bissett, Roy A. J. Spence OBE, Miranda Payne, and Gareth Morris-Stiff. Spinal cord compression and bone marrow suppression. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780199689842.003.0030.

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Childhood cancer in adults outlines the uncommon challenge presented by the occurrence of these pathologies in young adults, and the need for shared expertise in their successful management. The embryonal tumours, medulloblastoma, retinoblastoma, neuroblastoma, and Wilms’ tumour are considered first, emphasising similarities and differences in the pathology and management of each when adults are compared with children. The soft tissue sarcoma, rhabdomyosarcoma, is frequently best managed with chemotherapy and surgery. The important role of specialist cytogenetics in all these tumours is emphas

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11

The Circuitry of the Human Spinal Cord: Its Role in Motor Control and Movement Disorders. Cambridge University Press, 2005.

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12

Killen, Joan Marie. PARENTAL AND SPOUSAL ROLE CHANGES AFTER SPINAL CORD INJURY OF A CHILD OR SPOUSE. 1987.

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13

Schiff, David, Jonathan Sherman, and Paul D. Brown. Metastatic tumours: spinal cord, plexus, and peripheral nerve. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780199651870.003.0020.

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Systemic cancers produce substantial neurological morbidity when they spread to the spinal epidural space, producing epidural spinal cord compression—a neurological emergency. Less often, metastases spread directly to spinal cord parenchyma to manifest as intramedullary spinal cord metastasis or result in peripheral nerve dysfunction via compression of the brachial, lumbosacral, or, rarely, the cervical plexus. This chapter reviews the clinical manifestations and risk factors for development of these entities, the diagnostic approach, management options including the role of surgery, radiation

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14

Nashmi, Raad. Role of voltage-gated potassium channels in mediating posttraumatic axonal dysfunction after acute and chronic spinal cord injury. 2000.

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15

Streijger, Femke, Ward T. Plunet, and Wolfram Tetzlaff. Ketogenic Diet and Ketones for the Treatment of Traumatic Brain and Spinal Cord Injury. Edited by Jong M. Rho. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780190497996.003.0016.

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Ketogenic diets (KD)—high in fat, adequate in protein, and very low in carbohydrates—were developed almost a century ago and are still used clinically for drug-resistant epilepsy and some rare metabolic disorders. Possible new indications for cancers, diabetes, obesity, and neurodegenerative disorders are being trialed in humans based on a growing body of preclinical data showing efficacy. However the underlying mechanisms of KD remain incompletely understood. This chapter focuses on the neuroprotective effects of KD after spinal cord injury (SCI) and traumatic brain injury (TBI), and discusse

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16

Allen, Colin, James W. Grau, and Mary W. Meagher. The Lower Bounds of Cognition: What Do Spinal Cords Reveal? Edited by John Bickle. Oxford University Press, 2009. http://dx.doi.org/10.1093/oxfordhb/9780195304787.003.0006.

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This article examines the role of the spinal cords in cognition. It reviews animal science research that challenges the view that behavioral responses to sensory stimuli that do not involve brain mediation are fixed, automatic, and non-cognitive in nature. This research has shown the spinal cord to be a flexible and interesting learning system in its own right. This article discusses the consequences of these findings for philosophical understanding of the relationship between learning, cognition, and even consciousness. The article also explains the relevant concepts of instrumental condition

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17

Mason, Peggy. Homeostatic Systems. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190237493.003.0027.

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The roles of the hypothalamus in regulating fluid balance and supporting the calm affective state needed for maternal care are described. Hypothalamic control of hormone release is reviewed and related disorders such as Addison’s disease and acromegaly are introduced. Basic thermoregulatory principles are presented and the biological danger of ambient heat is emphasized. The concept of set point is explained in the context of fever, antipyresis and hyperthermia. Neural regulation of blood pressure and orthostatic hypotension are briefly described. The patterns and neural circuits involved in b

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18

Bromley, Lesley. The physiology of acute pain. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199234721.003.0001.

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Acute pain as a result of tissue damage is self-limiting. Impulses are generated in primary sensory nerves by chemical mediators released from the damaged tissues. The spinal cord receives these impulses in the dorsal horn. At the level of the spinal cord, the impulses can be amplified or reduced in amplitude by descending inputs. At the level of the spinal cord, the representation of the painful area and the sensitivity of other, surrounding areas can be modified. At the level of the brainstem and thalamus, further modification can take place. The final perception of the pain can be modified

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19

Gugino, Laverne D., Rafael Romero, Marcella Rameriz, Marc E. Richardson, and Linda S. Aglio. TMS in the perioperative period. Edited by Charles M. Epstein, Eric M. Wassermann, and Ulf Ziemann. Oxford University Press, 2012. http://dx.doi.org/10.1093/oxfordhb/9780198568926.013.0020.

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Two stimulation approaches developed for selectively exciting descending motor pathways are, transcranial electrical (TES) and transcranial magnetic (TMS) stimulation. This article highlights the comparison between electrical and magnetic transcranial stimulation. Magnetic stimulation is relatively painless; therefore it is the more preferred technique. The article reviews the use of TMS for monitoring the functional integrity of the descending motor systems during surgery and discusses the potential role of TMS in the preoperative period for conscious patients planning to undergo neurosurgica

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20

Sell, Philip. Thoracolumbar, lumbar, and sacral fractures. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780199550647.003.012043.

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♦ High-energy trauma often results in serious spinal fractures. The junctional zone between the relatively stiff thoracic spine and the more mobile lumbar spine is particularly susceptible to injury♦ The role of decompression in spinal cord injury remains uncertain at level three or four evidence♦ Unstable fractures may be stabilized using modern fracture fixation methods enabling easier nursing care in polytrauma and earlier mobilization than non-surgical treatment♦ There is level two evidence that stable thoracolumbar fractures have similar outcomes with surgical and non-surgical treatment♦

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21

Malcangio, Marzia. Glia. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0035.

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The landmark review discussed in this chapter, published in 2003 by Watkins and Maier, showed how glia have a major role in the modulation of pain mechanisms in the spinal cord and act remotely from peripheral nerve injury. This review led the way to a substantial body of literature demonstrating the pivotal role played by both microglia and astrocytes in chronic pain mechanisms. Since 2003 the modalities underlying neuron–microglia communication (e.g. chemokines, proteases, the translocator protein TSPO) have been dissected, and novel pathways of interactions delineated. Concrete molecular ta

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22

Feinman, Jared W., and John G. Augoustides. Neuroprotection for Aortic Surgery and Stenting. Edited by David L. Reich, Stephan Mayer, and Suzan Uysal. Oxford University Press, 2017. http://dx.doi.org/10.1093/med/9780190280253.003.0017.

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Despite recent advances, aortic surgery and stenting for an array of diseases still pose a significant risk of permanent and severe injury to the brain and/or spinal cord. These neurological risks are best understood in terms of the primary disease pathology, the extent of aortic involvement, mechanisms and risk factors, the role of neuromonitoring modalities, and the surgical techniques required for repair. This chapter will present an overview of perioperative practice in aortic surgery and stenting based on this framework and the latest guidelines and trials in order to describe best practi

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23

Guillery, Ray. The pathways for action. Oxford University Press, 2017. http://dx.doi.org/10.1093/oso/9780198806738.003.0003.

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Early nineteenth-century studies demonstrated, on the basis of clinical, experimental, and anatomical evidence, that a motor pathway, the corticospinal or pyramidal tract, passes from a specific area of the cortex, the precentral motor cortex, to the brainstem and spinal cord. The motor cortex can be seen as a topographic map of the movable body parts, and damage to the cortex or pathways produces correspondingly localized paralysis. However, there are a great many other pathways that link other areas of the cortex to parts of the brain active in the control of movements. These still play a pu

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24

Aziz, Qasim, and James K. Ruffle. The neurobiology of gut feelings. Oxford University Press, 2018. http://dx.doi.org/10.1093/oso/9780198811930.003.0005.

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“It’s a gut feeling.” Indeed, how and why do we get “gut feelings?” After the brain, the gut is the second most innervated bodily organ, diffusely interconnected with gastrointestinal afferent neurons. Whilst sensory neurons from the gut ascend by means of the spinal cord and vagal nerve to subcortical and higher cortical areas of the brain, caudally descending motor efferents from brain to gut seek to modulate gastrointestinal function. Such is the construct of the “brain–gut axis,” a bi-directional body nexus permitting constant information transfer between both brain and gut so as to provid

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25

Black, Sheila. The original description of central sensitization. Edited by Paul Farquhar-Smith, Pierre Beaulieu, and Sian Jagger. Oxford University Press, 2018. http://dx.doi.org/10.1093/med/9780198834359.003.0040.

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The landmark study discussed in this chapter is ‘The contribution of excitatory amino acids to central sensitization and persistent nociception after formalin-induced tissue injury’, published by Coderre and Melzack in 1992. Previous studies in this field implicate a contribution of excitatory amino acids (EAAs), specifically l-glutamate and l-aspartate, to injury-induced sensitization of nociceptive responses in the dorsal horn of the spinal cord. Repetitive stimulation of primary afferent fibres demonstrated that l-glutamate and NMDA can produce ‘wind-up’ of neuronal dorsal horn activity, an

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26

Esch, Megan, and Nancy L. Sicotte. Neuroimaging in Multiple Sclerosis. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199341016.003.0007.

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Magnetic resonance imaging (MRI) of the brain and spinal cord plays an integral role in establishing the diagnosis of multiple sclerosis (MS). The use of MRI leads to earlier recognition of MS, allowing for earlier treatment initiation and more efficient monitoring of disease treatment and progress. This chapter outlines conventional MRI imaging sequences that are used to evaluate MS white matter lesions in the central nervous system. It also addresses the incorporation of new imaging techniques that have increased understanding of clinically definite MS, its variants, and how various diseases

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27

Cooper, Jill, and Nina Kite. Occupational therapy in palliative care. Oxford University Press, 2015. http://dx.doi.org/10.1093/med/9780199656097.003.0046.

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Occupational therapy in palliative care aims to help patients achieve their optimum independence in activities that are important to them. The specific functional difficulties may include extreme fatigue, anxiety and shortness of breath due to advanced disease including metastatic spinal cord compression, fractures, or peripheral neuropathies. Patients may have problems with cognition and perception, body image, life role, and spiritual issues as well as physical disabilities. This chapter describes the broad range of areas in which the occupational therapist works in palliative care together

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28

Bodart, Olivier, and Steven Laureys. Imaging the central nervous system in the critically ill. Oxford University Press, 2016. http://dx.doi.org/10.1093/med/9780199600830.003.0224.

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Imaging techniques play a major role in managing patients with acute severe neurological signs. Initial evaluation of patients with traumatic brain injuries is best performed with a computed tomography (CT) scan, both for its ability to demonstrate most of the significant lesions and for logistical reasons. Magnetic resonance imaging (MRI) is able to provide more subtle information, as well as prognosis indicators, but is impractical until the patient’s condition has been stabilized. MRI has the same advantages for assessing anoxic brain injuries. In strokes, MRI has become the technique of ch

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29

Benarroch, Eduardo E. Neuroscience for Clinicians. Oxford University Press, 2021. http://dx.doi.org/10.1093/med/9780190948894.001.0001.

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The aim of this book is to provide the clinician with a comprehensive and clinical relevant survey of emerging concepts on the organization and function of the nervous system and neurologic disease mechanisms, at the molecular, cellular, and system levels. The content of is based on the review of information obtained from recent advances in genetic, molecular, and cell biology techniques; electrophysiological recordings; brain mapping; and mouse models, emphasizing the clinical and possible therapeutic implications. Many chapters of this book contain information that will be relevant not only

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30

Schaible, Hans-Georg, and Rainer H. Straub. Pain neurophysiology. Oxford University Press, 2013. http://dx.doi.org/10.1093/med/9780199642489.003.0059.

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Physiological pain is evoked by intense (noxious) stimuli acting on healthy tissue functioning as a warning signal to avoid damage of the tissue. In contrast, pathophysiological pain is present in the course of disease, and it is often elicited by low-intensity stimulation or occurs even as resting pain. Causes of pathophysiological pain are either inflammation or injury causing pathophysiological nociceptive pain or damage to nerve cells evoking neuropathic pain. The major peripheral neuronal mechanism of pathophysiological nociceptive pain is the sensitization of peripheral nociceptors for m

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31

Donaghy, Michael. The clinical approach. Oxford University Press, 2011. http://dx.doi.org/10.1093/med/9780198569381.003.0030.

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This chapter describes the appropriate clinical approach to take when presented with a patient reporting a neurological symptom. Just under 10 per cent of the population consult their general practitioner about a neurological symptom each year in the United Kingdom. About 10 per cent of these are referred for a specialist opinion, usually to a neurologist. Nine conditions account for roughly 75 per cent of general neurological referrals and are diagnosed initially on purely clinical grounds, with the other 25 per cent representing the full range of other, potentially very rare, neurological di

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32

(Editor), T. Kumazawa, L. Kruger (Editor), and K. Mizumura (Editor), eds. The Polymodal Receptor - A Gateway to Pathological Pain (Progress in Brain Research). Elsevier Science, 1996.

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33

Takao, Kumazawa, Kruger Lawrence, and Mizumura Kazue, eds. The polymodal receptor: A gateway to pathological pain. Elsevier, 1996.

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