Relevant bibliographies by topics / Spine plasticity

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  2. Dissertations / Theses
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Academic literature on the topic 'Spine plasticity'

Author: Grafiati
Published: 4 June 2021
Last updated: 9 February 2022

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Journal articles on the topic "Spine plasticity"

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Power, John M., and Pankaj Sah. "Dendritic spine heterogeneity and calcium dynamics in basolateral amygdala principal neurons." Journal of Neurophysiology 112, no. 7 (2014): 1616–27. http://dx.doi.org/10.1152/jn.00770.2013.

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Glutamatergic synapses on pyramidal neurons are formed on dendritic spines where glutamate activates ionotropic receptors, and calcium influx via N-methyl-d-aspartate receptors leads to a localized rise in spine calcium that is critical for the induction of synaptic plasticity. In the basolateral amygdala, activation of metabotropic receptors is also required for synaptic plasticity and amygdala-dependent learning. Here, using acute brain slices from rats, we show that, in basolateral amygdala principal neurons, high-frequency synaptic stimulation activates metabotropic glutamate receptors and
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Lee, Kevin F. H., Cary Soares, and Jean-Claude Béïque. "Examining Form and Function of Dendritic Spines." Neural Plasticity 2012 (2012): 1–9. http://dx.doi.org/10.1155/2012/704103.

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The majority of fast excitatory synaptic transmission in the central nervous system takes place at protrusions along dendrites called spines. Dendritic spines are highly heterogeneous, both morphologically and functionally. Not surprisingly, there has been much speculation and debate on the relationship between spine structure and function. The advent of multi-photon laser-scanning microscopy has greatly improved our ability to investigate the dynamic interplay between spine form and function. Regulated structural changes occur at spines undergoing plasticity, offering a mechanism to account f
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Sala, Carlo, and Menahem Segal. "Dendritic Spines: The Locus of Structural and Functional Plasticity." Physiological Reviews 94, no. 1 (2014): 141–88. http://dx.doi.org/10.1152/physrev.00012.2013.

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The introduction of high-resolution time lapse imaging and molecular biological tools has changed dramatically the rate of progress towards the understanding of the complex structure-function relations in synapses of central spiny neurons. Standing issues, including the sequence of molecular and structural processes leading to formation, morphological change, and longevity of dendritic spines, as well as the functions of dendritic spines in neurological/psychiatric diseases are being addressed in a growing number of recent studies. There are still unsettled issues with respect to spine formati
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Mendez, Pablo, Mathias De Roo, Lorenzo Poglia, Paul Klauser, and Dominique Muller. "N-cadherin mediates plasticity-induced long-term spine stabilization." Journal of Cell Biology 189, no. 3 (2010): 589–600. http://dx.doi.org/10.1083/jcb.201003007.

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Excitatory synapses on dendritic spines are dynamic structures whose stability can vary from hours to years. However, the molecular mechanisms regulating spine persistence remain essentially unknown. In this study, we combined repetitive imaging and a gain and loss of function approach to test the role of N-cadherin (NCad) on spine stability. Expression of mutant but not wild-type NCad promotes spine turnover and formation of immature spines and interferes with the stabilization of new spines. Similarly, the long-term stability of preexisting spines is reduced when mutant NCad is expressed but
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Desai, Niraj S., Tanya M. Casimiro, Stephen M. Gruber, and Peter W. Vanderklish. "Early Postnatal Plasticity in Neocortex of Fmr1 Knockout Mice." Journal of Neurophysiology 96, no. 4 (2006): 1734–45. http://dx.doi.org/10.1152/jn.00221.2006.

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Fragile X syndrome is produced by a defect in a single X-linked gene, called Fmr1, and is characterized by abnormal dendritic spine morphologies with spines that are longer and thinner in neocortex than those from age-matched controls. Studies using Fmr1 knockout mice indicate that spine abnormalities are especially pronounced in the first month of life, suggesting that altered developmental plasticity underlies some of the behavioral phenotypes associated with the syndrome. To address this issue, we used intracellular recordings in neocortical slices from early postnatal mice to examine the e
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Rangamani, Padmini, Michael G. Levy, Shahid Khan, and George Oster. "Paradoxical signaling regulates structural plasticity in dendritic spines." Proceedings of the National Academy of Sciences 113, no. 36 (2016): E5298—E5307. http://dx.doi.org/10.1073/pnas.1610391113.

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Transient spine enlargement (3- to 5-min timescale) is an important event associated with the structural plasticity of dendritic spines. Many of the molecular mechanisms associated with transient spine enlargement have been identified experimentally. Here, we use a systems biology approach to construct a mathematical model of biochemical signaling and actin-mediated transient spine expansion in response to calcium influx caused by NMDA receptor activation. We have identified that a key feature of this signaling network is the paradoxical signaling loop. Paradoxical components act bifunctionall
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Nevian, T., and B. Sakmann. "Spine Ca2+ Signaling in Spike-Timing-Dependent Plasticity." Journal of Neuroscience 26, no. 43 (2006): 11001–13. http://dx.doi.org/10.1523/jneurosci.1749-06.2006.

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Yusifov, Rashad, Anja Tippmann, Jochen F. Staiger, Oliver M. Schlüter, and Siegrid Löwel. "Spine dynamics of PSD-95-deficient neurons in the visual cortex link silent synapses to structural cortical plasticity." Proceedings of the National Academy of Sciences 118, no. 10 (2021): e2022701118. http://dx.doi.org/10.1073/pnas.2022701118.

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Critical periods (CPs) are time windows of heightened brain plasticity during which experience refines synaptic connections to achieve mature functionality. At glutamatergic synapses on dendritic spines of principal cortical neurons, the maturation is largely governed by postsynaptic density protein-95 (PSD-95)-dependent synaptic incorporation of α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors into nascent AMPA-receptor silent synapses. Consequently, in mouse primary visual cortex (V1), impaired silent synapse maturation in PSD-95-deficient neurons prevents the closure of
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Mulholland, Patrick J., and L. Judson Chandler. "The Thorny Side of Addiction: Adaptive Plasticity and Dendritic Spines." Scientific World JOURNAL 7 (2007): 9–21. http://dx.doi.org/10.1100/tsw.2007.247.

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Dendritic spines are morphologically specialized structures that receive the vast majority of central excitatory synaptic inputs. Studies have implicated changes in the size, shape, and number of dendritic spines in activity-dependent plasticity, and have further demonstrated that spine morphology is highly dependent on the dynamic organizational and scaffolding properties of its postsynaptic density (PSD).In vitroandin vivomodels of experience-dependent plasticity have linked changes in the localization of glutamate receptors at the PSD with a molecular reorganization of the PSD and alteratio
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Yu, Wendou, and Bingwei Lu. "Synapses and Dendritic Spines as Pathogenic Targets in Alzheimer’s Disease." Neural Plasticity 2012 (2012): 1–8. http://dx.doi.org/10.1155/2012/247150.

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Synapses are sites of cell-cell contacts that transmit electrical or chemical signals in the brain. Dendritic spines are protrusions on dendritic shaft where excitatory synapses are located. Synapses and dendritic spines are dynamic structures whose plasticity is thought to underlie learning and memory. No wonder neurobiologists are intensively studying mechanisms governing the structural and functional plasticity of synapses and dendritic spines in an effort to understand and eventually treat neurological disorders manifesting learning and memory deficits. One of the best-studied brain disord
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Dissertations / Theses on the topic "Spine plasticity"

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Critchlow, Hannah Marion. "The role of dendritic spine plasticity in schizophrenia." Thesis, University of Cambridge, 2008. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.612238.

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Pfeiffer, Thomas. "Super-resolution STED and two-photon microscopy of dendritic spine and microglial dynamics." Thesis, Bordeaux, 2017. http://www.theses.fr/2017BORD0743/document.

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Les changements des connections neuronales interviendraient dans la formation de la mémoire. J’ai développé de nouvelles approches basées sur l’imagerie photonique pour étudier (i) les interactions entre les microglies et les épines dendritiques, et (ii) le renouvellement des épines dans l’hippocampe in vivo. Ces deux phénomènes contribueraient au remodelage des circuits synaptiques intervenant dans la mémoire. (i) Les microglies sont impliquées dans de nouvelles fonctions en condition saine. J’ai examiné l’effet de la plasticité synaptique sur la dynamique morphologique des microglies, et sur
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Chiang, Chih-Yuan. "Cortical development & plasticity in the FMRP KO mouse." Thesis, University of Edinburgh, 2016. http://hdl.handle.net/1842/22055.

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Autism is one of the leading causes of human intellectual disability (ID). More than 1% of the human population has autism spectrum disorders (ASDs), and it has been estimated that over 50% of those with ASDs also have ID. Fragile X syndrome (FXS) is the most common inherited form of mental retardation and is the leading known genetic cause of autism, affecting approximately 1 in 4000 males and 1 in 8000 females. Approximately 30% of boys with FXS will be diagnosed with autism in their later lives. The cause of FXS is through an over-expansion of the CGG trinucleotide repeat located at the 5’
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Coiro, Pierluca [Verfasser]. "Plasticity-related gene 5 induces spine formation in immature primary neurons / Pierluca Coiro." Berlin : Freie Universität Berlin, 2011. http://d-nb.info/1025355571/34.

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Theis, Anne-Kathrin [Verfasser]. "Ryanodine receptor activation induces long-term plasticity of spine calcium dynamics / Anne-Kathrin Theis." Berlin : Medizinische Fakultät Charité - Universitätsmedizin Berlin, 2016. http://d-nb.info/1119803357/34.

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Knopp, Marcus. "Analysis of spine plasticity in CA1 hippocampal pyramidal neurons employing live cell nanoscopic imaging." Diss., Ludwig-Maximilians-Universität München, 2014. http://nbn-resolving.de/urn:nbn:de:bvb:19-173975.

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In der Großhirnrinde von Säugetieren befindet sich die Mehrheit erregender Synapsen auf Dornfortsätzen, kleinen dendritischen Ausbuchtungen, die in Größe und Form stark variieren. Die Auslösung aktivitätsabhängiger synaptischer Langzeitplastizität geht mit strukturellen Veränderungen dendritischer Dornen einher. Da das beugungsbegrenzte Auflösungsvermögen konventioneller Lichtmikroskope nicht ausreicht um die Morphologie der Dornen verlässlich zu untersuchen, stellte die Elektronenmikroskopie bisher das wichtigste bildgebende Verfahren zur Erforschung von struktureller Plastizität dar, blieb da
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O'Donnell, Cian. "Implications of stochastic ion channel gating and dendritic spine plasticity for neural information processing and storage." Thesis, University of Edinburgh, 2012. http://hdl.handle.net/1842/5886.

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On short timescales, the brain represents, transmits, and processes information through the electrical activity of its neurons. On long timescales, the brain stores information in the strength of the synaptic connections between its neurons. This thesis examines the surprising implications of two separate, well documented microscopic processes — the stochastic gating of ion channels and the plasticity of dendritic spines — for neural information processing and storage. Electrical activity in neurons is mediated by many small membrane proteins called ion channels. Although single ion channels a
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Zhang, Shengxiang. "Imaging dendritic spine structural plasticity during development in vitro and after acute stroke in vivo." Thesis, University of British Columbia, 2006. http://hdl.handle.net/2429/31194.

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The plasticity of dendritic spine structure is important for neural development and synaptic function and is altered in many pathological conditions. In this study, we investigated the mechanisms underlying spine structural plasticity during development and the pathological changes in spine structure during ischemic stroke by using confocal and two-photon microscopy. We first investigated spine structural dynamics during development and the role of intracellular Ca²⁺ in determining basal spine motility in cultured hippocampal neurons. We found that young cultured neurons displayed significantl
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Dhanrajan, T. M. "Morphological correlates of long-term potentiation and ageing in the hippocampus of rats." Thesis, Open University, 1999. http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.340704.

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Bauer, Rachel J. "THE EFFECTS OF LONG-TERM DEAFNESS ON DENSITY AND DIAMETER OF DENDRITIC SPINES ON PYRAMIDAL NEURONS IN THE DORSAL ZONE OF THE FELINE AUDITORY CORTEX." VCU Scholars Compass, 2019. https://scholarscompass.vcu.edu/etd/6028.

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Neuroplasticity has been researched in many different ways, from the growing neonatal brain to neural responses to trauma and injury. According to recent research, neuroplasticity is also prevalent in the ability of the brain to repurpose areas that are not of use, like in the case of a loss of a sense. Specifically, behavioral studies have shown that deaf humans (Bavalier and Neville, 2002) and cats have increased visual ability, and that different areas of the auditory cortex enhance specific kinds of sight. One such behavioral test demonstrated that the dorsal zone (DZ) of the auditory cort
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Books on the topic "Spine plasticity"

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Sjöström, Per Jesper. Spike-timing dependent plasticity. Edited by Henry Markram and Wulfram Gerstner. Frontiers Media SA, 2012. http://dx.doi.org/10.3389/978-2-88919-043-0.

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Rasia-Filho, Alberto A., Rochelle S. Cohen, and Oliver von Bohlen und Halbach, eds. Frontiers in Synaptic Plasticity: Dendritic Spines, Circuitries and Behavior. Frontiers Media SA, 2016. http://dx.doi.org/10.3389/978-2-88919-947-1.

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Spike Timing: Mechanisms and Function. Taylor & Francis Group, 2013.

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Koch, Christof. Biophysics of Computation. Oxford University Press, 1998. http://dx.doi.org/10.1093/oso/9780195104912.001.0001.

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Neural network research often builds on the fiction that neurons are simple linear threshold units, completely neglecting the highly dynamic and complex nature of synapses, dendrites, and voltage-dependent ionic currents. Biophysics of Computation: Information Processing in Single Neurons challenges this notion, using richly detailed experimental and theoretical findings from cellular biophysics to explain the repertoire of computational functions available to single neurons. The author shows how individual nerve cells can multiply, integrate, or delay synaptic inputs and how information can b
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Book chapters on the topic "Spine plasticity"

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Rall, Wilfrid, and Idan Segev. "Dendritic Spine Synapses, Excitable Spine Clusters, and Plasticity." In Cellular Mechanisms of Conditioning and Behavioral Plasticity. Springer US, 1988. http://dx.doi.org/10.1007/978-1-4757-9610-0_22.

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De Rubeis, Silvia, Esperanza Fernández, Andrea Buzzi, Daniele Di Marino, and Claudia Bagni. "Molecular and Cellular Aspects of Mental Retardation in the Fragile X Syndrome: From Gene Mutation/s to Spine Dysmorphogenesis." In Synaptic Plasticity. Springer Vienna, 2012. http://dx.doi.org/10.1007/978-3-7091-0932-8_23.

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Kreutz, M. R., I. König, M. Mikhaylova, C. Spilker, and W. Zuschratter. "Molecular Mechanisms of Dendritic Spine Plasticity in Development and Aging." In Handbook of Neurochemistry and Molecular Neurobiology. Springer US, 2008. http://dx.doi.org/10.1007/978-0-387-32671-9_10.

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Wang, Zejun, and Henriette van Praag. "Exercise and the Brain: Neurogenesis, Synaptic Plasticity, Spine Density, and Angiogenesis." In Functional Neuroimaging in Exercise and Sport Sciences. Springer New York, 2012. http://dx.doi.org/10.1007/978-1-4614-3293-7_1.

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Song, SooMin, Jeany Son, and Myoung-Hee Kim. "Stitching of Microscopic Images for Quantifying Neuronal Growth and Spine Plasticity." In Advances in Visual Computing. Springer Berlin Heidelberg, 2010. http://dx.doi.org/10.1007/978-3-642-17289-2_5.

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Fukunaga, K., N. Shioda, and E. Miyamoto. "The Function of CaM Kinase II in Synaptic Plasticity and Spine Formation." In Handbook of Neurochemistry and Molecular Neurobiology. Springer US, 2009. http://dx.doi.org/10.1007/978-0-387-30370-3_9.

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Johansson, B. B., and P. V. Belichenko. "Environmental Influence on Neuronal and Dendritic Spine Plasticity After Permanent Focal Brain Ischemia." In Maturation Phenomenon in Cerebral Ischemia IV. Springer Berlin Heidelberg, 2001. http://dx.doi.org/10.1007/978-3-642-59446-5_10.

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Girault, Jean-Antoine. "Integrating Neurotransmission in Striatal Medium Spiny Neurons." In Synaptic Plasticity. Springer Vienna, 2012. http://dx.doi.org/10.1007/978-3-7091-0932-8_18.

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Martin, Eric, Samuel Kaski, Fei Zheng, et al. "Spike-Timing-Dependent Plasticity." In Encyclopedia of Machine Learning. Springer US, 2011. http://dx.doi.org/10.1007/978-0-387-30164-8_774.

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Penzes, Peter, and Igor Rafalovich. "Regulation of the Actin Cytoskeleton in Dendritic Spines." In Synaptic Plasticity. Springer Vienna, 2012. http://dx.doi.org/10.1007/978-3-7091-0932-8_4.

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Conference papers on the topic "Spine plasticity"

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Dockendorf, Karl P., and Thomas B. DeMarse. "Amplitude and Spike Timing Dependent Plasticity." In 2007 International Joint Conference on Neural Networks. IEEE, 2007. http://dx.doi.org/10.1109/ijcnn.2007.4371231.

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Ramakrishnan, Shubha, Paul Hasler, and Christal Gordon. "Floating gate synapses with spike time dependent plasticity." In 2010 IEEE International Symposium on Circuits and Systems - ISCAS 2010. IEEE, 2010. http://dx.doi.org/10.1109/iscas.2010.5537768.

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Ochs, Karlheinz, Eloy Hernandez-Guevara, and Enver Solan. "Wave digital emulation of spike-timing dependent plasticity." In 2017 IEEE 60th International Midwest Symposium on Circuits and Systems (MWSCAS). IEEE, 2017. http://dx.doi.org/10.1109/mwscas.2017.8052883.

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Ferri, A. Cisternas, A. Rapoport, P. I. Fierens, and G. A. Patterson. "Mimicking Spike-Timing-Dependent Plasticity with Emulated Memristors." In 2019 Argentine Conference on Electronics (CAE). IEEE, 2019. http://dx.doi.org/10.1109/cae.2019.8709281.

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Taylor, Brady, Amar Shrestha, Qinru Qiu, and Hai Li. "1S1R-Based Stable Learning through Single-Spike-Encoded Spike-Timing-Dependent Plasticity." In 2021 IEEE International Symposium on Circuits and Systems (ISCAS). IEEE, 2021. http://dx.doi.org/10.1109/iscas51556.2021.9401644.

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Mostafa, Hesham, Christian Mayr, and Giacomo Indiveri. "Beyond spike-timing dependent plasticity in memristor crossbar arrays." In 2016 IEEE International Symposium on Circuits and Systems (ISCAS). IEEE, 2016. http://dx.doi.org/10.1109/iscas.2016.7527393.

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Ohno, Shuhei, Hideyuki Kato, and Tohru Ikeguchi. "Neuronal avalanche induced by multiplicative spike-timing-dependent plasticity." In 2011 International Joint Conference on Neural Networks (IJCNN 2011 - San Jose). IEEE, 2011. http://dx.doi.org/10.1109/ijcnn.2011.6033405.

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Jin, Xin, Alexander Rast, Francesco Galluppi, Sergio Davies, and Steve Furber. "Implementing spike-timing-dependent plasticity on SpiNNaker neuromorphic hardware." In 2010 International Joint Conference on Neural Networks (IJCNN). IEEE, 2010. http://dx.doi.org/10.1109/ijcnn.2010.5596372.

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Azghadi, Mostafa Rahimi, Said Al-Sarawi, Nicolangelo Iannella, and Derek Abbott. "Efficient design of triplet based Spike-Timing Dependent Plasticity." In 2012 International Joint Conference on Neural Networks (IJCNN 2012 - Brisbane). IEEE, 2012. http://dx.doi.org/10.1109/ijcnn.2012.6252820.

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Guo, Binbin, Yimao Cai, Yue Pan, Zhenxing Zhang, Yichen Fang, and Ru Huang. "Associative learning based on symmetric spike time dependent plasticity." In 2014 IEEE 12th International Conference on Solid -State and Integrated Circuit Technology (ICSICT). IEEE, 2014. http://dx.doi.org/10.1109/icsict.2014.7021615.

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